Far Infrared Rays are invisible wavelengths of light. Invisible because they are longer than the regular light we see. This invisible wavelength of light absorbed by the body can be felt as heat. Our bodies also radiate Far-Infrared warmth. When the body absorbs Far Infrared Rays it is like being in the sunshine the body is warmed up, you feel nice and relaxed full of energy.  1. Far-infrared are more efficient, penetrate quicker, and provide faster response in the body. Far-Infrared is absorbed by the molecules in six vibrational modes (scissoring, rocking, wagging, twisting, symmetric and asymmetric stretching). The National Library of Medicine PMC369987 2. Increases surface blood flow without increasing blood pressure, by promoting dilation of the blood vessels. 3. Improved flexibility. Warming up the muscle and connective tissue can help it become more pliable, restoring its elasticity * 4. Studies showed FIR benefits the Nitric oxide, one of the most important molecules for blood vessel health  Nitric Oxide Synthase - an overview Far-infrared radiation acutely increases nitric oxide production by increasing Ca(2+) mobilization and Ca(2+)/calmodulin-dependent protein kinase II-mediated phosphorylation of endothelial nitric oxide synthase at serine 1179 Using FIR as a thermal therapy agent, we investigate the molecular mechanism of its effect on endothelial nitric oxide synthase (eNOS) activity and NO production. FIR increased the phosphorylation of eNOS at serine 1179 (eNOS-Ser(1179)) in a time-dependent manner (up to 40min of FIR radiation) in bovine aortic endothelial cells (BAEC) without alterations in eNOS expression. This increase was accompanied by increases in NO production and intracellular Ca(2+) levels. Treatment with KN-93, a selective inhibitor of Ca(2+)/calmodulin-dependent protein kinase II (CaMKII) and H-89, a protein kinase A inhibitor, inhibited FIR radiation-stimulated eNOS-Ser(1179) phosphorylation. FIR radiation itself also increased the temperature of culture medium. As transient receptors potential vanilloid (TRPV) ion channels are known to be temperature-sensitive calcium channels, we explore whether TRPV channels mediate these observed effects. Reverse transcription-PCR assay revealed two TRPV isoforms in BAEC, TRPV2 and TRPV4. Although ruthenium red, a pan-TRPV inhibitor, completely reversed the observed effect of FIR radiation, a partial attenuation (∼20%) was found in cells treated with Tranilast, TRPV2 inhibitor. However, ectopic expression of siRNA of TRPV2 showed no significant alteration in FIR radiation-stimulated eNOS-Ser(1179) phosphorylation. This study suggests that FIR radiation increases NO production via increasing CaMKII-mediated eNOS-Ser(1179