Yes. Researchers now believe that this can be explained by cross-excitation in the trigeminal ganglion. That peripheral trigeminal inflammation in masticatory muscles can trigger migraine pathology is a fascinating phenomenon.
To understand how this happens, it helps to first review trigeminal anatomy. The peripheral afferents of both meningeal tissues and the TMJ apparatus project towards the trigeminal ganglion, the location of their cell bodies. Neural connections from the ganglion then project to the trigeminal nucleus caudalis, which in turn has tertiary connections in the thalamus, cortex, and limbic system. Hence, the area of interest where both neural systems project is the trigeminal ganglion.
As explained above, peripheral insult in the masseter causes CGRP expression in the trigeminal ganglion. When CGRP is expressed in the ganglion, it acts in a paracrine fashion by stimulating satellite microglial cells to release pro-inflammatory mediators, leading to an inflammatory cascade. One study showed that administering CGRP to migroglial cells in culture stimulates the release of over 20 cytokines.