jpd459/healthver_resplit huggingface dataset:
Question: touching a contaminated surface will not make you sick
Claim: there is also growing evidence that smaller particles called aerosols can hang in the air longer and travel farther.
Evidence: In this new study, human coronavirus 229E was rapidly inactivated on a range of copper alloys (within a few minutes for simulated fingertip contamination) and Cu/Zn brasses were very effective at lower copper concentration.
Verdict: Neutral
Evidence: The most common coronaviruses may well survive or persist on surfaces for up to one month.
Verdict: Neutral
Evidence: Frequent touching of contaminated surfaces in public areas is therefore a potential route of SARS-CoV-2 transmission.
Verdict: Neutral
Evidence: Surfaces near patients hospitalized with respiratory infections were frequently contaminated by pathogens, with Klebsiella pneumoniae being most common, highlighting the potential for transmission of respiratory pathogens via surfaces.
Verdict: Neutral
Question: Can drinking hot green tea help in preventing COVID-19?
Claim: Hot matcha green tea (with EGCG) can do magic for covid-19
Evidence: The present study corroborates that compound "I" (EGCG) mostly present in the integrants of green tea, shows the highest potentiality for acting as an inhibitor of SARS-CoV-2
Verdict: Refutes
Evidence: In conclusion, the plant-based foods play a vital role to enhance the immunity of people to control of COVID-19.
Verdict: Refutes
Evidence: The findings of the present study need in vivo experiments to prove the utility of "I", "F","D" and "E" compounds and their further use in making herb-based anti-SARS-CoV-2 product in near future
Verdict: Refutes
Evidence: This analysis may help to create a new ethno-drug formulation for preventing or curing the COVID-19
Verdict: Refutes
Question: Can wearing masks help in preventing the spread of the coronavirus disease?
Claim: There have been extensive randomized controlled trial (RCT) studies, and meta-analysis reviews of RCT studies, which all show that masks and respirators do not work to prevent respiratory influenza-like illnesses, or respiratory illnesses believed to be transmitted by droplets and aerosol particles.
Evidence: Wearing medical masks or N95 masks (namely N95 respirators) can slow the virus spread and reduce the infection risk.
Verdict: Refutes
Question: Does Vitamin C impact COVID-19 prevention and treatment?
Claim: Vitamin C cures COVID-19
Evidence: The majority of documents encouraged the consumption of fruits, vegetables, and whole grain foods. Thirty-one percent of the guidelines highlighted the importance of minerals and vitamins such as zinc and vitamins C, A, and D to maintain a well-functioning immune system.
Verdict: Refutes
Evidence: CONCLUSION The use of IV vitamin C in patients with moderate to severe COVID-19 disease may be feasible.
Verdict: Refutes
Evidence: We identified a total of 17 patients who received IV vitamin C for COVID-19. The inpatient mortality rate in this series was 12% with 17.6% rates of intubation and mechanical ventilation. We noted a significant decrease in inflammatory markers, including ferritin and D-dimer, and a trend to decreasing FiO2 requirements, after vitamin C administration. 
Verdict: Refutes
Evidence: Some nutrients are actively involved in the proper functioning and strengthening of the human immune system against viral infections including dietary protein, omega-3 fatty acids, vitamin A, vitamin D, vitamin E, vitamin B1, vitamin B6, vitamin B12, vitamin C, iron, zinc, and selenium. Few studies were done on the effect of dietary components on prevention of COVID-19, but supplementation with these nutrients may be effective in improving the health status of patients with viral infections.
Verdict: Refutes
Question: Do antibiotics work against the coronavirus?
Claim: What are the best antibiotics for the coronavirus? None whatsoever.
Evidence: This activity is conserved on the SARS-Cov-2, thus placing teicoplanin as a potential treatment for patients with this virus.
Verdict: Refutes
Evidence: Despite frequent prescription of broad-spectrum empirical antimicrobials in patients with coronavirus associated respiratory infections, there is a paucity of data to support the association with respiratory bacterial/fungal co-infection.
Verdict: Supports
Evidence: Teicoplanin, an antibiotic used to treat staphylococci infection, previously showed efficacy to inhibit the first stage of MERS-coronarivus viral cycle in human cells. 
Verdict: Refutes
Question: will SARS-CoV2 infected people develop immunity?
Claim: in some people with severe COVID-19, the body goes rogue and attacks one of its own key immune defenses instead of the coronavirus.
Evidence: The CD4 + memory exploiting could help immune system to recall immunity of already know antigens against coronaviruses, avoiding or limiting "lung crash" until virus specific immunity develops and making it faster and prolonged. Finally, this administration could be helpful not only in already infected patients, but also before infection. In fact, people could have an immune system more ready when the contact with the Covid-19 will occur.
Verdict: Neutral
Evidence: Based on animal experiments, already acquired data on other coronavirus types and plausibility assumptions, it can be assumed that seroconverted patients have an immunity of limited duration and only a very low risk of reinfection.
Verdict: Neutral
Evidence:  According to this hypothesis vaccine formulations (tetanus, diphtheria, Bordetella pertussis) could be re-administrate after the first contact with Covid-19, better before the development of respiratory severe illness and of course before full-blown ARDS (Acute Respiratory Distress Syndrome). The CD4+ memory exploiting could help immune system to recall immunity of already know antigens against coronaviruses, avoiding or limiting lung crash until virus specific immunity develops and making it faster and prolonged. 
Verdict: Neutral
Evidence: In this study, we collected blood from COVID-19 patients who have recently become virus-free and therefore were discharged, and analyzed their SARS-CoV-2-specific antibody and T cell responses. We observed SARS-CoV-2-specific humoral and cellular immunity in the patients. Both were detected in newly discharged patients, suggesting both participate in immune-mediated protection to viral infection.
Verdict: Neutral
Question: Does Vitamin C impact COVID-19 prevention and treatment?
Claim: Everyone who get the coronavirus will die: No.
Evidence: The majority of documents encouraged the consumption of fruits, vegetables, and whole grain foods. Thirty-one percent of the guidelines highlighted the importance of minerals and vitamins such as zinc and vitamins C, A, and D to maintain a well-functioning immune system.
Verdict: Neutral
Evidence: CONCLUSION The use of IV vitamin C in patients with moderate to severe COVID-19 disease may be feasible.
Verdict: Neutral
Evidence: Dietary supplementation has not been linked to COVID-19 prevention.
Verdict: Neutral
Evidence: We identified a total of 17 patients who received IV vitamin C for COVID-19. The inpatient mortality rate in this series was 12% with 17.6% rates of intubation and mechanical ventilation. We noted a significant decrease in inflammatory markers, including ferritin and D-dimer, and a trend to decreasing FiO2 requirements, after vitamin C administration. 
Verdict: Neutral
Question: Does garlic protect against covid-19
Claim: Drinking boiled garlic water will cure COVID-19.
Evidence: Hoaxes (such as eating garlic or citrus to prevent COVID-19) were detected in 15 videos (10.9%).
Verdict: Refutes
Evidence: In late December 2019 in Wuhan, China, several patients with viral pneumonia were identified as 2019 novel coronavirus (2019-nCoV). So far, there are no specific treatments for patients with coronavirus disease-19 (COVID-19), and the treatments available today are based on previous experience with similar viruses such as severe acute respiratory syndrome-related coronavirus (SARS-CoV), Middle East respiratory syndrome coronavirus (MERS-CoV), and Influenza virus.
Verdict: Refutes
Evidence: 51% will drink ginger with honey and 42.7% eat garlic for infection prevention.
Verdict: Supports
Evidence: compounds derived from Allium sativum (garlic) have the potential to decrease the expression of proinflammatory cytokines and to reverse the immunological abnormalities to more acceptable levels. Allium sativum is suggested as a beneficial preventive measure before being infected with SARS-CoV-2 virus.
Verdict: Refutes
Question: Does Vitamin D impact COVID-19 prevention and treatment?
Claim: Low Vitamin D Levels Tied to Odds for Severe COVID
Evidence: Several studies demonstrated the role of vitamin D in reducing the risk of acute viral respiratory tract infections and pneumonia. These include direct inhibition with viral replication or with anti-inflammatory or immunomodulatory ways. In the meta-analysis, vitamin D supplementation has been shown as safe and effective against acute respiratory tract infections. Thus, people who are at higher risk of vitamin D deficiency during this global pandemic should consider taking vitamin D supplements to maintain the circulating 25(OH)D in the optimal levels (75-125nmol/L).
Verdict: Supports
Evidence: A principal defence against uncontrolled inflammation, and against viral infection in general, is provided by T regulatory lymphocytes (Tregs). Treg levels have been reported to be low in many COVID-19 patients and can be increased by vitamin D supplementation. Low vitamin D levels have been associated with an increase in inflammatory cytokines and a significantly increased risk of pneumonia and viral upper respiratory tract infections. Vitamin D deficiency is associated with an increase in thrombotic episodes, which are frequently observed in COVID-19. Vitamin D deficiency has been found to occur more frequently in patients with obesity and diabetes. These conditions are reported to carry a higher mortality in COVID-19. If vitamin D does in fact reduce the severity of COVID-19 in regard to pneumonia/ARDS, inflammation, inflammatory cytokines and thrombosis, it is our opinion that supplements would offer a relatively easy option to decrease the impact of the pandemic.
Verdict: Supports
Evidence: a small reverse correlation between mortality rate and vitamin D status was observed throughout the globe (r= -0.177). In Asia, a medium direct correlation was observed for recovery rate (r= 0.317) and a significant reveres correlation for mortality rate (r= -0.700) with vitamin D status in such patients.
Verdict: Refutes
Evidence: In Europe, there were no correlations for both recovery (r= 0.040) and mortality rate (r= -0.035).
Verdict: Refutes
Question: how does the coronavirus respond to changes in the weather
Claim: Coronavirus can Be Crushed By Warmer Weather
Evidence: Results indicated that temperatures had a negative linear relationship with the number of confirmed cases.
Verdict: Refutes
Evidence: We show that the delay between exposure and detection of infection complicates the estimation of weather impact on COVID-19 transmission, potentially explaining significant variability in results to-date
Verdict: Neutral
Evidence: temperature is the most influential parameter that reduces the growth at the rate of 13-17 cases/day with a 1C rise in temperature.
Verdict: Supports
Evidence: Correcting for that distributed delay and offering conservative estimates, we find a negative relationship between temperatures above 25 degrees Celsius and estimated reproduction number ([R]), with each degree Celsius associated with a 3.1% (95% CI, 1.5% to 4.8%) reduction in [R]. Higher levels of relative humidity strengthen the negative effect of temperature above 25 degrees.
Verdict: Supports
Question: are heart complications likely in patients with COVID-19?
Claim: Heart damage lingers in COVID-19 patients, even after recovery
Evidence:  consistent reports of cardiac injury and de novo cardiac complications, including possible myocarditis, arrhythmia, and heart failure in patients without prior cardiovascular disease or significant risk factors, are emerging
Verdict: Neutral
Evidence: Myocardial injury is significantly associated with fatal outcome of COVID-19, while the prognosis of patients with underlying CVD but without myocardial injury is relatively favorable. Myocardial injury is associated with cardiac dysfunction and arrhythmias. Inflammation may be a potential mechanism for myocardial injury.
Verdict: Neutral
Evidence: the greater susceptibility of individuals with underlying cardiovascular conditions to develop more severe Covid-19 with higher mortality rate is likely to be confounded, in part, by age and the type of co-morbidities. Patients with heart failure or chronic kidney disease might show an excess risk
Verdict: Neutral
Evidence: The clinical evidence in our study suggested that myocardial injury is more likely related to systemic consequences rather than direct damage by the 2019 novel coronavirus. The elevation in cardiac markers was probably due to secondary and systemic consequences and can be considered as the warning sign for recent adverse clinical outcomes of the patients
Verdict: Neutral
Question: Does heat prevent COVID-19?
Claim: coronavirus can be prevented by having millets in one's diet because it generates heat in the body.
Evidence: It is suggested that Chinese medicine of clearing heat and removing toxin should be used to prevent the uninfected people from COVID-19, and intervene in the early stage of the disease and control the development of it to be severe by inhibiting the cytokine storm.
Verdict: Refutes
Evidence: Since many coronaviruses are sensitive to heat, heating inactivation of samples at 56C prior to testing is considered a possible method to reduce the risk of transmission, but the effect of heating on the measurement of SARS-CoV-2 antibodies is still unclear.
Verdict: Refutes
Evidence: Of note, 44.12% of the detected IgM levels were dropped below the cutoff value after heating, suggesting heat inactivation can lead to false-negative results of these samples.
Verdict: Refutes
Evidence: Considering that SARS-CoV-2 cannot tolerate temperatures above 70 C, here we designed and fabricated efficient filters based on heated nickel (Ni) foam to catch and kill SARS-CoV-2.
Verdict: Refutes
Question: Can animals spread COVID-19?
Claim: A few cats have reportedly tested positive for COVID-19 after they came into contact with people who were infected. They include pet cats and zoo animals.
Evidence: Using data from probable cat-to-cat transmission in Wuhan, China, we estimated the basic reproduction number R0 under this scenario at 1.09 (95% confidence interval: 1.05, 1.13). This value is much lower than the R0 reported for humans and close to one, indicating that the sustained transmission between cats is unlikely to occur.
Verdict: Supports
Evidence: Although there is currently no evidence that animals play a substantial role in spreading COVID-19, CDC advises persons with suspected or confirmed COVID-19 to restrict contact with animals during their illness and to monitor any animals with confirmed SARS-CoV-2 infection and separate them from other persons and animals at home (1).
Verdict: Supports
Evidence: There is currently no indication that feline- or canine-to-human transmission can occur, though there is rising evidence of the reverse.
Verdict: Supports
Evidence: There is evidence that SARS-CoV-2 can infect felines, dogs and minks, and there is evidence of human-to-animal infection.
Verdict: Supports
Question: how does the coronavirus respond to changes in the weather
Claim: The pandemic coronavirus will wane with warmer weather, like some other respiratory
Evidence: COVID-19, due to its regular flu-like symptoms, is also expected to show similar seasonality and subside as the global temperatures rise in the northern hemisphere with the onset of spring
Verdict: Supports
Evidence: Temperature is the most influential parameter that reduces the growth at the rate of 13-16 cases/day with a 1{degrees}C rise in temperature
Verdict: Supports
Evidence: Correcting for that distributed delay and offering conservative estimates, we find a negative relationship between temperatures above 25 degrees Celsius and estimated reproduction number ([R]), with each degree Celsius associated with a 3.1% (95% CI, 1.5% to 4.8%) reduction in [R]. Higher levels of relative humidity strengthen the negative effect of temperature above 25 degrees.
Verdict: Supports
Evidence: Preliminary observations from our team and others suggest that temperature and climatological factors could influence the spread of this novel coronavirus
Verdict: Supports
Question: Which organs are most affected by COVID-19?
Claim: The new coronavirus can infect organs throughout the body, including lungs, throat, heart, liver, brain, kidneys and the intestines
Evidence: Since COVID-19 spread all over the world becoming a pandemic illness, researchers have better characterized route of virus transmissibility and clinical signs and symptoms of the disease.
Verdict: Neutral
Evidence: While COVID-19 primarily affects the lungs, causing interstitial pneumonitis and severe acute respiratory distress syndrome (ARDS), it also affects multiple organs, particularly the cardiovascular system.
Verdict: Supports
Evidence: The symptoms most commonly reported by patients affected by coronavirus disease (COVID-19) include cough, fever, and shortness of breath. However, other major events usually observed in COVID-19 patients (e.g., high blood pressure, arterial and venous thromboembolism, kidney disease, neurologic disorders, and diabetes mellitus) indicate that the virus is targeting the endothelium, one of the largest organs in the human body.
Verdict: Supports
Evidence: Since viral transmission occurs through the droplets emitted during coughing or sneezing, the lungs are primarily affected. However, SARS-CoV-2 can affect several human organs due to high expressions of ACE2 receptor which is the main viral target and the virus may affect not only higher and lower respiratory tracts, but also heart, kidney, gastro enteric tract, liver, pancreas, nervous system and skin.
Verdict: Supports
Question: Is a headache sign of the coronavirus disease?
Claim: A headache isn't a common symptom of the virus, but about 14% of people infected with corna have experienced it
Evidence: Muscle injury or myalgia was the most common (19.2%, 95%CI 15.423.2%) neurologic symptom of COVID-19, followed by headache (10.9%, 95%CI 8.6213.51%);
Verdict: Supports
Evidence: Cough, fever, headache and asthenia were the most prevalent symptoms.
Verdict: Refutes
Evidence: The aim of this manuscript is to investigate transversally Ear Nose Throat (ENT) symptoms COVID-19 infection correlated and to study the neurotropism and neuroinvasiveness of the virus in the head-neck district through the investigation of the sense of smell, taste, tearing, salivation and hearing.
Verdict: Neutral
Evidence: Symptomatic COVID19 patients, around 6%10%, also reported headache as a presenting symptom.
Verdict: Refutes
Question: are patients taking Angiotensin-converting enzyme inhibitors (ACE) inhibitors at increased risk for COVID-19?
Claim: Since patients treated with ACEIs and ARBS will have increased numbers of ACE2 receptors in their lungs for coronavirus S proteins to bind to, they may be at increased risk of severe disease outcomes due to SARS-CoV-2infections
Evidence:  current evidence did not confirm the concern that ACEI/ARB exposure is harmful in patientswith COVID-19 infection. This study supports the current guidelines that discourage discontinuation of ACEIs or ARBs in COVID-19 patients and the setting of the COVID-19 pandemic
Verdict: Refutes
Evidence: This has led to the hypothesis that use of ACEIs and ARBs may increase the risk of developing severe COVID-19.
Verdict: Supports
Evidence: Our results strongly support the recommendation that ACEI and ARB should be continued in the population and in COVID-19 positive patients, reinforcing the position of several scientific societies.
Verdict: Neutral
Evidence: Patients who take ACEIs and ARBS may be at increased risk of severe disease outcomes due to SARS-CoV-2 infections.
Verdict: Supports
Question: Is dexamethasone effective for treating COVID-19?
Claim: budesonide, commonly prescribed in the United States under the name Pulmicort, is a good treatment for COVID-19.
Evidence: In June 2020, a large randomised controlled clinical trial in the UK found that dexamethasone was effective in reducing the number of deaths in patients with severe coronavirus disease 2019 (COVID-19).
Verdict: Neutral
Evidence: Many drugs have shown promise for treatment of COVID-19.
Verdict: Neutral
Evidence: Corticosteroids could be useful in patients with an inflammatory profile, considering that acute respiratory distress syndrome may be the consequence of cytokine storm syndrome. LEARNING POINTS: One of the main pathophysiological hypotheses for severe COVID-19 pneumonia is inappropriate immunological hyperactivation. Corticosteroid therapy may be useful in these patients.
Verdict: Neutral
Evidence: We designed a trial to evaluate the effectiveness of early intravenous dexamethasone administration on the number of days alive and free of mechanical ventilation within 28 days after randomization in adult patients with moderate or severe ARDS due to confirmed or probable COVID-19.
Verdict: Neutral
Question: Does Vitamin D impact COVID-19 prevention and treatment?
Claim: VITAMIN D LEVELS MAY IMPACT COVID-19 MORTALITY RATES
Evidence: testing positive for COVID-19 was associated with increasing age(RR(age<50)=1.05,p<0.021;RR(age[]50)=1.02,p<0.064)), non-white race(RR=2.54,p<0.01) and being likely vitamin D deficient (deficient/treatment-not-increased:RR=1.77,p<0.02) as compared to likely vitamin D sufficient(not-deficient/treatment-not-decreased), with predicted COVID-19 rates in the vitamin D deficient group of 21.6%(95%CI[14.0%-29.2%] ) versus 12.2%(95%CI[8.9%-15.4%]) in the vitamin D sufficient group.
Verdict: Supports
Evidence: Vitamin D deficiency that is not sufficiently treated is associated with COVID-19 risk.
Verdict: Supports
Evidence: A principal defence against uncontrolled inflammation, and against viral infection in general, is provided by T regulatory lymphocytes (Tregs). Treg levels have been reported to be low in many COVID-19 patients and can be increased by vitamin D supplementation. Low vitamin D levels have been associated with an increase in inflammatory cytokines and a significantly increased risk of pneumonia and viral upper respiratory tract infections. Vitamin D deficiency is associated with an increase in thrombotic episodes, which are frequently observed in COVID-19. Vitamin D deficiency has been found to occur more frequently in patients with obesity and diabetes. These conditions are reported to carry a higher mortality in COVID-19. If vitamin D does in fact reduce the severity of COVID-19 in regard to pneumonia/ARDS, inflammation, inflammatory cytokines and thrombosis, it is our opinion that supplements would offer a relatively easy option to decrease the impact of the pandemic.
Verdict: Supports
Evidence: The severity of coronavirus 2019 infection (COVID-19) is determined by the presence of pneumonia, severe acute respiratory distress syndrome (SARS-CoV-2), myocarditis, microvascular thrombosis and/or cytokine storms, all of which involve underlying inflammation.
Verdict: Neutral
Question: What vaccine candidates are being tested for Covid-19?
Claim: Another candidate vaccine, called CoronaVac , being developed by Beijing-based Sinovac Biotech, protected rhesus macaque monkeys from infection with the novel coronavirus,
Evidence: Our findings may contribute to the design of an efficacious vaccine against COVID-19.
Verdict: Neutral
Evidence: Currently there is no known vaccine or specific antiviral treatment for COVID-19
Verdict: Refutes
Evidence: At this time, no vaccine is available to control further dissemination of the disease.
Verdict: Refutes
Evidence: Currently, there is no vaccine or therapeutic available for this disease.
Verdict: Refutes