Generate an explanation for the given claim using the provided supporting material from the paragraph. Please consider the following points while generating an output. 1) The claim will always have supporting proof in the paragraph, and the paragraph will have a clear point of view supporting the claim. 2) There can be instances where direct correlation to the claim is not available. Make use of the sentences in the paragraph to provide a corresponding output supporting the claim. 3) The explanation should be at least one and at most twelve sentences long.

[Q]: Paragraph: The announcement by the world’s largest fast-food chain comes as more companies respond to government and consumer pressure to address the global obesity epidemic. McDonald’s, which often bears the brunt of criticism over the restaurant industry’s penchant for tempting diners with indulgent and often high-calorie food, said it would offer the option in all of its 20 major global markets by 2020. McDonald’s also vowed to promote and market only water, milk and juice as the beverages in its popular Happy Meals for children as part of its announcement at the Clinton Global Initiative annual meeting in New York on Thursday. Waist sizes around the world are increasing, setting off alarms in public health circles. In recent years, the U.S. food industry has begun yielding to pressure from government, parents and consumers seeking to slim down adults and children. Sugary sodas have been yanked from public schools; sugar, sodium and calorie levels have been reduced in products, and calorie counts have been posted on some restaurant menus. The Center for Science in the Public Interest, a nonprofit that has tangled with McDonald’s over everything from fattening food to the marketing of Happy Meals, approved of the company’s move to add more fruits and vegetables to the menu. Still, it says the company and its rivals have a long way to go in terms of offering healthier options. “McDonald’s slow march toward healthier meals made a major advance today, but a long road lies ahead for the company,” CSPI said in a statement. McDonald’s is seen as something of a trend setter among restaurant chains, but like many of its peers, it has tended to resist external efforts to force change. About one year ago McDonald’s said it would begin listing calorie information on menus in some 14,000 U.S. restaurants and drive-throughs — ahead of a national rule that would require larger restaurant chains to make such disclosures but months after other chains embraced the idea. In July 2011 it tweaked its popular Happy Meals for children - reducing the french fries portion by more than half and adding apples to every order. The move came after pressure from CSPI, parent groups and others. More than one-third of Americans are obese, and about 10 percent of the nation’s healthcare bill is tied to obesity-related illnesses, such as Type 2 diabetes, heart disease and hypertension, according to the Organization for Economic Co-operation and Development. McDonald’s said its announcement is part of a plan developed with the Alliance for a Healthier Generation, which was founded by the Clinton Foundation and American Heart Association, to increase customers’ access to fruit and vegetables and help families and children to make informed eating and lifestyle choices. Claim: Want fruit with your burger? McDonald's expands anti-obesity push.
[A]: Hold the fries, pass the salad. McDonald’s Corp on Thursday said it would offer healthy options as part of its popular value meals, letting customers choose a side salad, fruit or vegetables instead of french fries.


[Q]: Paragraph: In 2004, a virulent form of canine influenza surfaced at greyhound racing parks in Florida. In that outbreak, it infected 24 greyhounds and killed 8 more. The contagion has since been confirmed in seven states, having killed greyhounds at tracks in Florida, Massachusetts, Arizona, West Virginia, Wisconsin, Texas and Iowa. It is highly contagious, spreading through shared items (like toys) or human contact (kennel workers have carried the virus home with them), in addition to dog-to-dog encounters. The virus that has been felling greyhounds is an H3N8 flu closely related to an equine flu strain. It is not related to typical human flus or to the H5N1 avian flu that killed about 100 people in Asia. Several states — including Florida, Minnesota, and Louisiana, among others — reported another rash of cases in late June 2017. According to the American Veterinary Medical Association (AVMA), the outbreaks in those states were caused by a strain known as canine H3N2 influenza, which was first reported in Chicago in 2015. An AVMA spokesperson, Sharon Granskog, told us in a phone interview that such outbreaks can be seasonal in nature: There’s a time of year when more dogs are in contact with one another, they’re at dog parks, it’s highly contagious. We suggest that you make sure if your dog is going to a dog park that it’s healthy and talk to a veterinarian about whether or not a vaccine would be right for your dog. The disease was first reported in the U.S. in 2004 after several racing greyhounds in Florida contracted it. The AVMA said that they believed that the dogs became infected by a strain of H3N8 influenza, which is particular to horses. Dr. Ruben Donis, chief of molecular genetics for the influenza branch of the Centers for Disease Control and Prevention, confirmed that the flu jumped from horses to dogs, “a very rare event of considerable scientific interest” and added “at this point, there is no reason to panic.” How that jump occurred is either not known at this point or is not being commented on, so the email’s assertion that the eating of raw meat was to blame should be regarded as speculation rather than as fact. While there is always the possibility the virus might again jump species, this time to humans, this strain of flu has been present in horses for more than 40 years without any documented cases of humans catching it or the related canine flu. But there are vaccines available for both the equine and canine flus. Many readers have been confused (and unnecessarily frightened) by the difference between the terms “morbidity rate” and “mortality rate.” The morbidity rate describes the percentage of animals that will contract the disease after being exposed to the virus, but, despite its name, the term has nothing to do with the death rate associated with the flu. (Nearly 80 percent of dogs exposed to the canine flu virus will contract only a mild form of the disease which mimics kennel cough, a type of canine bronchitis that is rarely serious.) The mortality rate, which describes the percentage of animals that will die after contracting the disease, is in the much lower range of 5 to 8 percent, according to Dr. Cynda Crawford, the veterinary immunologist who first isolated the canine flu virus: The mortality rate is around 5 to 8 percent, says veterinarian Cynda Crawford at the University of Florida’s College of Veterinary Medicine in Gainesville. “I want to stress that despite the rumors that are out on the Internet and other such sources, this disease is not as deadly as people want to make it,” Crawford said. She says she receives more than 30 calls a day from concerned veterinarians. Dr. Crawford describes the contagion as producing in dogs “a moist, productive cough that ends in a gagging response, that will persist for one to four weeks, despite treatment with antibiotics or cough suppressants. Some dogs develop a thick, yellow discharge from the nose. A very few dogs will spike a high fever, between 105 to 107 degrees Fahrenheit. They become lethargic and weak, with rapid, shallow breathing. This is likely to progress to pneumonia.” Other veterinary experts have estimated the potential death rate as between 1 and 10 percent, with the higher percentage applying to very young, very old, or infirm dogs. Presence of the virus in dogs can be confirmed only through blood tests performed at the Cornell University College of Veterinary Medicine. Results of such blood screens take as long as two weeks. Dr. Crawford recommends keeping dogs showing symptoms of respiratory disease at home and away from other dogs for up to two weeks. The CDC, which is tracking the disease, issued no official recommendations. Because the symptoms of this as yet unnamed virus somewhat mimic bordetella, a less virulent illness commonly known as kennel cough, it is hard to ascertain how widespread the flu has become. On the flipside of that confusion, vets in various parts of the country have been thrown into a panic when encountering run-of-the-mill kennel cough in any of their clients, fearing they are instead confronting cases of the new flu. The Animal Health Diagnostic Center at Cornell University inserted a caution against such hair-trigger diagnosis within a larger advisory about the potential for the flu to have spread to the state of New York: “One should not lose sight of the fact that all respiratory infections in dogs are not due to canine influenza virus. Adhering to the ‘band wagon’ approach could result in the failure to appropriately treat dogs with infections previously known to cause respiratory problems in dogs.” We found this good advice for vets and dog owners in our inbox one day: PLEASE DO NOT PANIC, and do NOT assume that every cough is Canine Influenza. Kennel Cough from parainfluenza and Bordetella is more common. However, the Animal Health Diagnostic Center at Cornell is interested in receiving samples from dogs that appear to have kennel cough. Clinical Signs: Since this is a new pathogen in dogs, there is currently no natural immunity present in the unexposed canine population. Almost all exposed dogs will become infected, and nearly 80% have clinical signs. In the mild form the dogs will have a cough that persists for 10 to 21 days. The cough may be soft and moist or dry. Many dogs will have a nasal discharge from a secondary bacterial infection and low grade fever. The nasal discharge responds to broad spectrum antibiotics. In the severe form with pneumonia there is a high fever (104-106 F) and respiratory difficulties. X-rays may show consolidation. These dogs often have secondary bacterial infections and have responded to broad-spectrum antibiotics and supportive care including intravenous hydration. The incubation period is two to five days and dogs may shed virus for seven to 10 days. The disease can spread rapidly throughout a boarding kennel. Dogs that are coughing SHOULD NOT BE BROUGHT TO SHOWS or Performance EVENTS. In a 1 June 2017 advisory to its members, the AVMA suggested that vaccines against the H3N2 virus strain “might be another prevention option” for dogs who were at least 8 weeks old and also recommended isolating dogs who could be infected, as well as not sharing toys between dogs and disinfecting shelter, veterinary, and boarding facilities. Claim: A virus deadly to dogs has been spreading in the canine population of the U.S.
[A]: Has a virus deadly to dogs been spreading in the canine population of the U.S.?


[Q]: Paragraph: Dr. Dana Meaney-Delman, who has been leading the inquiry at the U.S. Centers for Disease Control and Prevention (CDC) in Atlanta, wants to know where that oil came from. The answer will help explain whether these cells play a key role in the vaping-related outbreak that has killed seven people and sickened 530 so far. It may also reveal whether some of these cases have been occurring all along, undetected. “We’re looking to partner with any lab that can assist in the identification of what those lipids (fats) are,” Meaney-Delman said in a telephone interview. A group of researchers who have been studying the long-term effects of vaping told Reuters they have taken up the challenge. They have begun to re-examine lung cell samples they have collected in recent years for evidence of these oil-filled immune cells in people who vaped but didn’t get sick. One possibility: The deposits are residue from inhaling vaping oils, such as those containing the marijuana ingredient tetrahydrocannabinol (THC) or vitamin E acetate. Both are considered possible contributors to the current illnesses. Some researchers suspect the oils are formed inside the lungs as part of the body’s natural response to chemicals found in many commercial vaping devices. One theory is that vaping these chemicals may impair the immune system, and make people who vape more vulnerable to respiratory distress, they say. A study published in the Journal of Clinical Investigation earlier this month has fueled the latter theory. It found that mice exposed to aerosols of propylene glycol and vegetable glycerin - common solvents used in conventional nicotine vaping devices - developed these same fat-clogged immune cells even though they were never exposed to vaping oils. These mice also had impaired immune systems compared to mice exposed to room air. The study set off alarm bells for Thomas Eissenberg, co-director of the Center for Tobacco Products at Virginia Commonwealth University. For years, doctors have reported isolated cases of pneumonia-like illnesses in people who vaped. In many cases, patients also had these fat-filled immune cells - called lipid-laden macrophages. Now, these same abnormalities have been found in mice, and in at least some of the people who have fallen ill recently. “For me, the implication is there may be some underlying level of disease, like what we’re seeing in this cluster, that’s been going on all along,” Eissenberg said in a phone interview. Early last week, Eissenberg joined some 25 vaping experts on a hastily arranged conference call. At least three researchers who have already collected lung cell samples from otherwise healthy vapers told Reuters they would see whether these people also had fat-laden pockets within their immune cells. Other researchers, like Eissenberg, are seeking funding to collect new samples of their own. They want to help determine whether these abnormalities have been present for years, and whether they have made vapers generally more vulnerable to severe disease, possibly triggered by some new vaping substance. The group’s members say they have been in regular contact with officials at the CDC and the National Institutes of Health on how they can best help with the multistate investigation. They include Robert Tarran, a physiologist and vaping expert at the University of North Carolina School of Medicine, and Dr. Peter Shields, a lung cancer specialist at The Ohio State University Wexner Medical Center, who has one of the country’s largest sets of lung samples from vapers, smokers and never-smokers. “It could be that we’ll see something in the general, normal population of people who are using e-cigarettes,” Shields said. Investigators at the CDC and the U.S. Food and Drug Administration (FDA) have pointed to THC vaping oils or vitamin E, a substance used in some THC products, as a possible cause of these illnesses. But they have not ruled out anything yet, including conventional nicotine liquids. CDC pathologists are examining hundreds of lung cell samples gathered from patients in the outbreak. Meanwhile, forensic chemists at the FDA are testing more than 150 products to determine whether there is a common ingredient that may help explain the illnesses. “We need to parlay what we’re seeing in product samples with what we’re seeing in lung tissue,” Meaney-Delman said. Dr. Laura Crotty Alexander, a lung specialist at the University of California at San Diego, has been studying vaping’s effect on health since 2013. Two years ago, she treated a patient with the same set of symptoms that are now being described across the country, and is now checking her lung cell samples to look for clues. “It’s possible that everybody who is vaping is at risk,” she said. Claim: U.S. scientists join effort to solve mysterious vaping-related illnesses.
[A]:
The U.S. investigation into hundreds of cases of life-threatening lung illnesses related to vaping has turned up a curious abnormality: Many of the victims had pockets of oil clogging up cells responsible for removing impurities in the lungs.