In this task, you're given a paragraph and title from the research paper. Your task is to classify whether the given title is suitable or not for the research paper based on the given paragraph. Return "True" if title is proper according to paragraph else "False".

[Q]: Paragraph: Background: Embryonated chicken eggs have been used since the mid-20th century to grow a wide range of animal viruses to high titers. However, eggs have found so far only limited use in the production of recombinant proteins. We now describe a system, based on a Sendai virus minigenome, to produce large amounts of heterologous viral glycoproteins in the allantoic cavity of embryonated eggs. Results: Soluble forms of human respiratory syncytial virus (HRSV) and human metapneumovirus (HMPV) fusion (F) proteins, devoid of their transmembrane and cytoplasmic domains, were produced in allantoic fluids using the Sendai minigenome system. The first step was rescuing in cell cultures Sendai virus minigenomes encoding the proteins of interest, with the help of wild type Sendai virus. The second step was propagating such recombinant defective viruses, together with the helper virus, in the allantoic cavity of chicken embryonated eggs, and passage to optimize protein production. When compared with the production of the same proteins in the culture supernatant of cells infected with vaccinia recombinants, the yield in the allantoic fluid was 5-10 fold higher. Mutant forms of these soluble proteins were easily constructed by site-directed mutagenesis and expressed in eggs using the same approach. The simplicity and economy of the Sendai minigenome system, together with the high yield achieved in the allantoic fluid of eggs, makes it an attractive method to express soluble glycoproteins aimed for structural studies. 
 Title: High level expression of soluble glycoproteins in the allantoic fluid of embryonated chicken eggs using a Sendai virus minigenome system
[A]: True


[Q]: Paragraph: A hallmark of disease is that most pathogens are able to infect more than one host species. However, for most pathogens, we still have a limited understanding of how this affects epidemiology, persistence and virulence of infections-including several zoonotic pathogens that reside in wild animal reservoirs and spillover into humans. In this chapter, we review the current knowledge of mallard (Anas platyrhynchos) as host for pathogens. This species is widely distributed, often occupying habitats close to humans and livestock, and is an important game bird species and the ancestor to domestic ducks-thereby being an excellent model species to highlight aspects of the wildlife, domestic animal interface and the relevance for human health. We discuss mallard as host for a range of pathogens but focus more in depth of it as a reservoir host for influenza A virus (IAV). Over the last decades, IAV research has surged, prompted in part to the genesis and spread of highly pathogenic virus variants that have been devastating to domestic poultry and caused a number of human spillover infections. The aim of this chapter is to synthesise and review the intricate interactions of virus, host and environmental factors governing IAV epidemiology and evolution. 
 Title: Of Ducks and Men: Ecology and Evolution of a Zoonotic Pathogen in a Wild Reservoir Host
[A]: False


[Q]: Paragraph: We report the first case of transmission of Panton-Valentine leukocidin-producing Staphylococcus aureus to a physician during the resuscitation of an infant with fatal pneumonia. The physician exhibited numerous furuncles. This case highlights the necessity for health care workers to protect themselves against transmission of infectious diseases from patient to care giver. Severe respiratory diseases, in immunocompetent infants, and chronic furunculosis have been associated with Staphylococcus aureus strains producing Panton-Valentine leukocidin (PVL), a cytotoxin that causes leukocyte destruction and tissue necrosis [1] . PVL-producing S. aureus is known to cause rapidly extensive and necrotizing pneumonia in children and young adults. We report the first case, to our knowledge, of transmission of PVL-producing S. aureus to a physician during the resuscitation of an infant with fatal pneumonia. Case report. A 4-month-old boy was admitted to the pediatric ward of Saint-Vincent-de-Paul hospital (Paris) with a history of 3 days of cough and 1 day of moderate fever. Physical examination revealed only mild respiratory distress. Findings on a chest radiogram were normal, as were the WBC count and the C-reactive protein and procalcitonin levels. Bronchiolitis was diagnosed, and the patient did not receive antibiotics. Progressive respiratory failure developed during the 12 h after admission, and the patient required nasal oxygenation. While a physical examination was being performed by a physician, the patient experienced cardiac arrest. Resuscitation measures, including oral intubation, oxygenation, bag-valve-mask ventilation, chest compression, and intravenous administration of adrenaline, were immediately performed and were continued for 1 h, without success. It was notable that blood flow from the trachea was observed during intubation. Repeated oral and tracheal suction was performed. Because the resuscitation took place in the general pediatric ward, health care workers participating in the resuscitation did not wear any protection, such as a facial mask and gloves. A necropsy was performed, and the findings revealed right lobar pneumonia with a necrotizing hemorrhage of the entire right lung and one-half of the left lung. A culture specimen from tracheal aspirates grew methicillin-susceptible S. aureus. Identification of S. aureus was based on colony morphology, coagulation of citrated rabbit plasma (bioMérieux), and production of clumping factor (determined with the Staphyslide test; bioMérieux). The methicillin resistance was determined by the Kirby-Bauer disk-diffusion method, with the use of a 5-mg oxacillin disk, in accordance with the NCCLS recommendations [2], after 24-48 h of incubation at 35ЊC. The S. aureus isolate was only resistant to penicillin and amoxicillin and was susceptible to methicillin, aminosides, macrolides, quinolones, and cotrimoxazole. On day 5, the physician who had performed intubation presented with numerous furuncles of the fingers and face. Samples were obtained from the skin lesions. At the same time, nasal samples were obtained from the 15 health care workers who had participated in the resuscitation. S. aureus strains susceptible to methicillin were isolated from samples from the physician and from 5 other health care workers. The isolate from the infant (S1, isolated from a tracheal sample) and 7 isolates from the 6 health care workers (S2, isolated from skin lesions of 1 physician; S4 and S5, from nasal and anal samples from another physician; and S3, S6, S7, and S8, from nasal samples from 4 nurses) were genotyped and analyzed for the presence of the PVL gene. PFGE, used for genotyping, was performed on DNA fragments obtained by digestion with the restriction enzyme SmaI by use of a CHEF-MAPPER apparatus. The PFGE banding patterns were compared visually. Strains were considered genetically distinguishable if their restriction patterns differed by у3 bands [3] . The isolates from the infant (S1) and from 1 
 Title: Transmission of Panton-Valentine Leukocidin-Producing Staphylococcus aureus to a Physician during Resuscitation of a Child
[A]:
True