Given a question and a context passage, generate the answer having the word or phrase from the context passage. Here, the answer should be a shortest continous span from the passage.

[Q]: Context: The Raiders traveled across the bay to Monster Park in San Francisco to take on the 49ers. The 49ers got on the board first with an Alex Smith to Arnaz Battle touchdown pass midway through the first quarter. The Raiders responded with a 33-yard field goal by Sebastian Janikowski. Janikowski then converted a 36 yarder early in the second quarter. Randy Moss scored his 100th career touchdown reception on a 22-yard pass from Andrew Walter, as the Raiders went into halftime with a 13-7 lead. The 49ers then went on to score 24 unanswered points in the second half, as Smith connected on touchdown passes with Battle once again, and Maurice Hicks in the third quarter. Joe Nedney converted a chip shot 19-yard field goal early in the fourth quarter. On the next Raiders play from scrimmage, Melvin Oliver recovered a fumble for a touchdown, as Walter tried to lateral a pass to LaMont Jordan, who let it hit the ground, and assumed it was an incomplete pass. Marques Tuiasosopo hit Courtney Anderson with a touchdown pass late in the game, but it was too little too late. Nedney completed the scoring with a 39-yard field goal. 
 Question: Who scored first in the first quarter?
[A]: the 49ers


[Q]: Context: As of the 2010 United States Census, there were 1,951,269 people, 715,365 households, and 467,916 families residing in the county. The population density was . There were 840,343 housing units at an average density of . The racial makeup of the county was 60.9% white, 10.5% black or African American, 8.7% Asian, 0.7% Pacific islander, 0.7% American Indian, 13.5% from other races, and 5.1% from two or more races. Those of Hispanic or Latino origin made up 29.1% of the population. In terms of ancestry, 11.7% were Germans, 9.1% were Irish people, 7.6% were English people, 6.3% were Italians, and 2.7% were Americans. 
 Question: Which group from the census is larger: Pacific islander or two or more races?
[A]: two or more races


[Q]: Context: Christianson Syndrome, a recently identified X-linked neurodevelopmental disorder, is caused by mutations in the human gene SLC9A6 encoding the recycling endosomal alkali cation/proton exchanger NHE6. The patients have pronounced limitations in cognitive ability, motor skills and adaptive behaviour. However, the mechanistic basis for this disorder is poorly understood as few of the more than 20 mutations identified thus far have been studied in detail. Here, we examined the molecular and cellular consequences of a 6 base-pair deletion of amino acids Glu(287) and Ser(288) (ES) in the predicted seventh transmembrane helix of human NHE6 expressed in established cell lines (CHO/AP-1, HeLa and neuroblastoma SH-SY5Y) and primary cultures of mouse hippocampal neurons by measuring levels of protein expression, stability, membrane trafficking, endosomal function and cell viability. In the cell lines, immunoblot analyses showed that the nascent mutant protein was properly synthesized and assembled as a homodimer, but its oligosaccharide maturation and half-life were markedly reduced compared to wild-type (WT) and correlated with enhanced ubiquitination leading to both proteasomal and lysosomal degradation. Despite this instability, a measurable fraction of the transporter was correctly sorted to the plasma membrane. However, the rates of clathrin-mediated endocytosis of the ES mutant as well as uptake of companion vesicular cargo, such as the ligand-bound transferrin receptor, were significantly reduced and correlated with excessive endosomal acidification. Notably, ectopic expression of ES but not WT induced apoptosis when examined in AP-1 cells. Similarly, in transfected primary cultures of mouse hippocampal neurons, membrane trafficking of the ES mutant was impaired and elicited marked reductions in total dendritic length, area and arborization, and triggered apoptotic cell death. These results suggest that loss-of-function mutations in NHE6 disrupt recycling endosomal function and trafficking of cargo which ultimately leads to neuronal degeneration and cell death in Christianson Syndrome. 
 Question: Mutation of which gene is implicated in the Christianson syndrome?
[A]:
slc9a6