In this task, you're given a paragraph and title from the research paper. Your task is to classify whether the given title is suitable or not for the research paper based on the given paragraph. Return "True" if title is proper according to paragraph else "False".
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Question: Paragraph: Mannose-binding lectin (MBL) is a key soluble pathogen recognition protein of the innate immune system that binds specific mannose-containing glycans on the surfaces of microbial agents and initiates complement activation via the lectin pathway. Prior studies showed that MBL-dependent activation of the complement cascade neutralized insect cell-derived West Nile virus (WNV) in cell culture and restricted pathogenesis in mice. Here, we investigated the antiviral activity of MBL in infection by dengue virus (DENV), a related flavivirus. Using a panel of naïve sera from mouse strains deficient in different complement components, we showed that inhibition of infection by insect cell-and mammalian cell-derived DENV was primarily dependent on the lectin pathway. Human MBL also bound to DENV and neutralized infection of all four DENV serotypes through complement activation-dependent and -independent pathways. Experiments with human serum from naïve individuals with inherent variation in the levels of MBL in blood showed a direct correlation between the concentration of MBL and neutralization of DENV; samples with high levels of MBL in blood neutralized DENV more efficiently than those with lower levels. Our studies suggest that allelic variation of MBL in humans may impact complement-dependent control of DENV pathogenesis. Citation Avirutnan P, et al. 2011. Complement-mediated neutralization of dengue virus requires mannose-binding lectin. mBio 2(6):e00276-11. 
 Title: Complement-Mediated Neutralization of Dengue Virus Requires Mannose-Binding Lectin

Answer: True


Question: Paragraph: Despite being simple eukaryotic organisms, the yeasts Saccharomyces cerevisiae and Schizosaccharomyces pombe have been widely used as a model to study human pathologies and the replication of human, animal, and plant viruses, as well as the function of individual viral proteins. The complete genome of S. cerevisiae was the first of eukaryotic origin to be sequenced and contains about 6,000 genes. More than 75% of the genes have an assigned function, while more than 40% share conserved sequences with known or predicted human genes. This strong homology has allowed the function of human orthologs to be unveiled starting from the data obtained in yeast. RNA plant viruses were the first to be studied in yeast. In this paper, we focus on the use of the yeast model to study the function of the proteins of human immunodeficiency virus type 1 (HIV-1) and the search for its cellular partners. This human retrovirus is the cause of AIDS. The WHO estimates that there are 33.4 million people worldwide living with HIV/AIDS, with 2.7 million new HIV infections per year and 2.0 million annual deaths due to AIDS. Current therapy is able to control the disease but there is no permanent cure or a vaccine. By using yeast, it is possible to dissect the function of some HIV-1 proteins and discover new cellular factors common to this simple cell and humans that may become potential therapeutic targets, leading to a long-lasting treatment for AIDS. 
 Title: Yeast and the AIDS Virus: The Odd Couple

Answer: False


Question: Paragraph: A 69-year-old male with a past medical history of hypertension, diabetes mellitus, and acute myelogenous leukemia (AML) who underwent allogeneic hematopoietic stem cell transplantation (HSCT) presented with recurrent fever and cough. The patient had undergone allogeneic HSCT from a matched unrelated donor 17 months previously, conditioned by fludarabine, melphalan, and alemtuzumab. His posttransplant course was complicated by graft versus host disease and multiple central venous catheter infections with coagulase-negative Staphylococcus spp., vancomycinresistant Enterococcus, Lactobacillus, and alpha-hemolytic Streptococcus. During his initial course, he also developed a nodular pneumonia, and the subsequent workup included a bronchoscopy that was nondiagnostic. The pneumonia resolved with empiric posaconazole treatment. Sixteen months after the transplant, the patient developed fatigue, low-grade fevers, and cough. Chest x-ray (CXR) showed a new, left lower lobe infiltrate. A sputum culture then grew fluoroquinolone-susceptible Pseudomonas, and the Oncology Service subsequently discharged the patient home on moxifloxacin alone. Three weeks later, he presented again with recurrent cough and fever and was readmitted. A CXR showed a new, right lower lobe infiltrate. The patient had a remote history of symptomatic bradycardia for which he had undergone pacemaker placement. He was a former cigarette smoker (40 pack-years), but he denied any intravenous drug use. He was a retired automobile mechanic and lived at home with his wife and a dog. He had traveled recently to Las Vegas for a short trip to visit casinos. His medications on admission included acyclovir, fluconazole, and dapsone prophylaxis. He previously had an adverse reaction to imipenem, which caused a rash, and to voriconazole, which caused visual hallucinations. On physical examination, he was alert and oriented. His temperature was 100.5 °F, heart rate 86 beats per minute, blood pressure 134/94 mmHg, respiratory rate 20 breaths per minute, and oxygen saturation 90% on ambient air. He had mild bilateral crackles at the lung bases and a well-healed, non-tender, nonerythematous surgical scar at the site of the pacemaker pocket on the anterior chest wall. The remainder of his physical examination was unremarkable. Laboratory values were significant for pancytopenia, with WBC count of 3500/μl, (76% neutrophils, 8% lymphocytes, and 12% monocytes), hemoglobin 11.6 g/dL (normal range, 13.5-17.5 g/ dL), and platelet count of 122,000/μL (normal range, 150,000-450,000/μL). Other laboratory 
 Title: Pulmonary Infection in a Patient After Stem Cell Transplantation

Answer:
False