Given a context passage, generate a question from the passage such that its answer is shortest continous span from the passage.

Input: Consider Input: Instituto de Administracao de Projetos Educacionais Futebol Clube play their home games at Estadio Nhozinho Santos.

Output: The Instituto de Administração de Projetos Educacionais Futebol Clube played home games where?


Input: Consider Input: That year the chocolate magnate Peter Ludwig agreed to endow 350 modern artworks--then valued at $45 million --and in return the City of Cologne committed itself to build a dedicated ''Museum Ludwig'' for works made after the year 1900.

Output: The Museum Ludwig was named for whom?


Input: Consider Input: The APP/PS1ki mouse model for Alzheimer's disease (AD) exhibits robust brain and spinal cord axonal degeneration and hippocampal CA1 neuron loss starting at 6 months of age. It expresses human mutant APP751 with the Swedish and London mutations together with two FAD-linked knocked-in mutations (PS1 M233T and PS1 L235P) in the murine PS1 gene. The present report covers a phenotypical analysis of this model using either behavioral tests for working memory and motor performance, as well as an analysis of weight development and body shape. At the age of 6 months, a dramatic, age-dependent change in all of these properties and characteristics was observed, accompanied by a significantly reduced ability to perform working memory and motor tasks. The APP/PS1ki mice were smaller and showed development of a thoracolumbar kyphosis, together with an incremental loss of body weight. While 2-month-old APP/PS1ki mice were inconspicuous in all of these tasks and properties, there is a massive age-related impairment in all tested behavioral paradigms. We have previously reported robust axonal degeneration in brain and spinal cord, as well as abundant hippocampal CA1 neuron loss starting at 6 months of age in the APP/PS1ki mouse model, which coincides with the onset of motor and memory deficits described in the present report.


Output: Which disease the London mutation involved in?