In this task, you're given a paragraph and title from the research paper. Your task is to classify whether the given title is suitable or not for the research paper based on the given paragraph. Return "True" if title is proper according to paragraph else "False".

Ex Input:
Paragraph: Ebolavirus, a member of the Filoviridae family of negative-sense RNA viruses, causes severe haemorrhagic fever leading up to 90% lethality. Ebolavirus matrix protein VP40 is involved in the virus assembly and budding process. The RNA binding pocket of VP40 is considered as the drug target site for structure based drug design. High Throughput Virtual Screening and molecular docking studies were employed to find the suitable inhibitors against VP40. Ten compounds showing good glide score and glide energy as well as interaction with specific amino acid residues were short listed as drug leads. These small molecule inhibitors could be potent inhibitors for VP40 matrix protein by blocking virus assembly and budding process. ISSN 0973-2063 (online) 0973-8894 (print) Bioinformation 9(6): 286-292 (2013) 287 © 2013 Biomedical Informatics replication [23]. C-terminal domain of VP40 is required for membrane association [24, 25]. open access ISSN 0973-2063 (online) 0973-8894 (print) Bioinformation 9(6): 286-292 (2013) 289 
 Title: Hypothesis High-throughput virtual screening and docking studies of matrix protein vp40 of ebola virus

Ex Output:
False


Ex Input:
Paragraph: Respiratory virus infection (RVI) in pediatric solid organ transplant (SOT) recipients poses a significant risk; however, the epidemiology and effects of an RVI after pediatric SOT in the era of current molecular diagnostic assays are unclear. Methods. A retrospective observational cohort of pediatric SOT recipients (January 2010 to June 2013) was assembled from 9 US pediatric transplant centers. Charts were reviewed for RVI events associated with hospitalization within 1 year after the transplant. An RVI diagnosis required respiratory symptoms and detection of a virus (ie, human rhinovirus/enterovirus, human metapneumovirus, influenza virus, parainfluenza virus, coronavirus, and/or respiratory syncytial virus). The incidence of RVI was calculated, and the association of baseline SOT factors with subsequent pulmonary complications and death was assessed. Results. Of 1096 pediatric SOT recipients (448 liver, 289 kidney, 251 heart, 66 lung, 42 intestine/multivisceral), 159 (14.5%) developed RVI associated with hospitalization within 12 months after their transplant. RVI occurred at the highest rates in intestine/ abdominal multivisceral (38%), thoracic (heart/lung) (18.6%), and liver (15.6%) transplant recipients and a lower rate in kidney (5.5%) transplant recipients. RVI was associated with younger median age at transplant (1.72 vs 7.89 years; P < .001) and among liver or kidney transplant recipients with the receipt of a deceased-donor graft compared to a living donor (P = .01). The all-cause and attributable case-fatality rates within 3 months of RVI onset were 4% and 0%, respectively. Multivariable logistic regression models revealed that age was independently associated with increased risk for a pulmonary complication (odds ratio, 1.24 [95% confidence interval, 1.02-1.51]) and that receipt of an intestine/multivisceral transplant was associated with increased risk of all-cause death (odds ratio, 24.54 [95% confidence interval, 1.69-327.96]). Conclusions. In this study, hospital-associated RVI was common in the first year after pediatric SOT and associated with younger age at transplant. All-cause death after RVI was rare, and no definitive attributable death occurred. 
 Title: A Multicenter Consortium to Define the Epidemiology and Outcomes of Pediatric Solid Organ Transplant Recipients With Inpatient Respiratory Virus Infection

Ex Output:
True


Ex Input:
Paragraph: The rat (Rattus norvegicus) is used in a broad field of biomedical research, and numerous animal models for human diseases have been developed. The rat genetic map, however, remains poorly documented. Hedrich (1990) described a rat linkage map consisting of 13 groups, of which only 6 were assigned to chromosomes. Levan and coworkers (1991, 1992) collected mapping information from the literature, including their own works. Their tables and figures contain gene chromosomal assignments, cytogenetical localizations, and comparisons of the rat G-banding pattern with those of mice and humans, but do not include any information about gene orders or distances. Recently, an informative rat genetic linkage map has been reported by Serikawa and colleagues (1992b). It is based on the segregation of 125 polymorphic markers in seven rat backcrosses and two F 2 crosses, and all of the linkage groups are assigned to particular chromosomes. The linkage map and the information on microsatellite markers became a great help for genetic studies with rat models for human diseases. Since then, a considerable amount of new data for the rat linkage map has appeared in several publications. We, therefore, have collected mapping data published up to the middle of 1993, including our recent reports, and have summarized it here in a list of mapped loci. This genetic map, constructed by combining several linkage data, and two comparative maps for mouse or human homologous rat genes should be very informative to many researchers working on genetic studies in rats, mice, or humans. Rat genetic linkage map 
 Title: Special Report A rat genetic linkage map and comparative maps for mouse or human homologous rat genes

Ex Output:

True