Given a context passage, generate a question from the passage such that its answer is shortest continous span from the passage.

Ex Input:
According to Apollodorus, Agenor was born in Memphis of Egypt to Poseidon and Libya and he had a twin brother named Belus.

Ex Output:
What was the name of Agenor mother?


Ex Input:
The Eagles looked to win their 6th game in a row against the winless 49ers. The Eagles got off to a slow start, only leading 3-0 after the first quarter. Late in the second quarter, the Eagles blew the game open. Carson Wentz found tight end Zach Ertz for a 1-yard touchdown in the back of the end zone off of a bootleg pass, and Jalen Mills intercepted rookie QB C.J. Beathard and returned it 37 yards for a touchdown. The 49ers appeared to have some life in the third quarter, following a Carson Wentz interception, leading to a Matt Breida 22 yard touchdown reception. However, the Eagles responded, with Wentz connecting with Alshon Jeffery for a 53-yard touchdown, putting the game out of reach. The Eagles went on to win 33-10 and advanced to 7-1 on the season. Despite the win, the Eagles played a sloppy game in a rainy afternoon at the Linc.

Ex Output:
Which player had the longest touchdown catch?


Ex Input:
Chronic myelogenous leukemia (CML) results from the neoplastic transformation of primitive hematopoietic stem cells, and has been classified as a myeloproliferative disorder. The hallmark of CML is the presence of a balanced translocation between the long arms of chromosomes 9 and 22, t(9;22)(q34;q11.2), which is known as the Philadelphia (Ph) chromosome. This translocation results in the formation of the bcr-abl fusion gene, which, in turn, is translated into a chimeric Bcr-Abl protein with deregulated tyrosine kinase activity. Constitutive Bcr-Abl expression has been shown to be necessary and sufficient for the transformed phenotype of CML cells. CML is unique among human cancers in that a single genetic defect, the Ph chromosome, is responsible for the transformed phenotype. Since this discovery more than 40 years ago, our understanding of the clinical course, therapy, and prognosis of patients with CML has changed significantly. These changes have culminated in the emergence of imatinib, the first rationally designed, molecularly targeted therapy for human malignancy. In this review, the authors describe the molecular biology of CML and the development of imatinib as a therapeutic agent for the treatment of CML.

Ex Output:

What tyrosine kinase, involved in a Philadelphia- chromosome positive chronic myelogenous leukemia, is the target of Imatinib (Gleevec)?