Generate an explanation for the given claim using the provided supporting material from the paragraph. Please consider the following points while generating an output. 1) The claim will always have supporting proof in the paragraph, and the paragraph will have a clear point of view supporting the claim. 2) There can be instances where direct correlation to the claim is not available. Make use of the sentences in the paragraph to provide a corresponding output supporting the claim. 3) The explanation should be at least one and at most twelve sentences long.

[EX Q]: Paragraph: The 2017 case is the first case of pediatric tetanus in Oregon in more than 30 years and alarmed infectious disease experts who said tetanus is almost unheard of in the U.S. since widespread immunization began in the 1940s. The child received an emergency dose of the tetanus vaccine in the hospital, but his parents declined to give him a second dose — or any other childhood shots — after he recovered, the paper said. “When I read that, my jaw dropped. I could not believe it. That’s a tragedy and a misunderstanding, and I’m just flabbergasted,” said Dr. William Schaffner, an expert in infectious diseases and chair at the Department of Preventive Medicine at the Vanderbilt University School of Medicine in Nashville, Tennessee. “This is an awful disease, but ... we have had a mechanism to completely prevent it, and the reason that we have virtually no cases anymore in the United States is because we vaccinate, literally, everyone.” Doctors in Portland, Oregon, who treated the child declined to provide any further information about the family at a news conference Friday, citing medical privacy laws. It was the first time that Dr. Judith Guzman-Cottrill, the pediatric infectious disease expert who treated the child, had ever seen tetanus because of widespread vaccination against it in the U.S. When the boy arrived at the emergency department, his muscle spasms were so severe he could not talk, could not open his mouth and was struggling to breathe, she said. “We had a hard time taking care of this child — watching him suffer — and it is a preventable disease,” Guzman-Cottrill said. News about the tetanus case comes as lawmakers in Oregon and Washington are considering bills that would end non-medical exemptions for routine childhood vaccines as the Pacific Northwest weathers its third month of a measles outbreak. Seventy people in southwest Washington, most of them unvaccinated children, have been diagnosed with the highly contagious viral illness since Jan. 1, as well as a handful of people in Portland, Oregon. Unlike measles, which is a virus, someone who has survived a case of tetanus is not immune and can get the illness again if they remain unvaccinated. Tetanus also isn’t transmitted person-to-person by sneezing or coughing like the measles, but instead comes from bacterial spores that are found in the environment. Tetanus spores exist everywhere in the soil. When an unvaccinated person gets a deep, penetrating wound, those spores can invade the cut and begin producing the bacteria that causes the illness. The tetanus bacterium secretes a toxin that gets into the bloodstream and latches onto the nervous system. Anywhere from three to 21 days after infection, symptoms appear: muscle spasms, lockjaw, difficulty swallowing and breathing and seizures. The disease can cause death or severe disability in those who survive, Schaffner said. About 30 people contract tetanus each year nationwide, according to the CDC, and 16 people died of it between 2009 and 2015. It’s rare among children; those over 65 are the most vulnerable. In the case in Oregon, the boy cut himself on the forehead while playing, and his family stitched up the wound themselves. Six days later, he began clenching his jaw, arching his neck and back and had uncontrollable muscle spasms. When he began to have trouble breathing, his parents called paramedics and he was transported by air to Oregon Health & Science University’s Doernbecher Children’s Hospital in Portland. When he arrived, he asked for water but could not open his mouth. The child was sedated, put on a ventilator and cared for in a darkened room while wearing ear plugs because any stimulation made his pain and muscle spasms worse. His fever spiked to almost 105 degrees (40.5 Celsius), and he developed high blood pressure and a racing heartbeat. Forty-four days after he was hospitalized, the boy was able to sip clear liquids. Six days later, he was able to walk a short distance with help. After another three weeks of inpatient rehabilitation and a month at home, he could ride a bike and run — a remarkable recovery, experts said. The child’s care — not including the air ambulance and inpatient rehabilitation — cost nearly $1 million, about 72 times the mean for a pediatric hospitalization in the U.S., the paper noted. “The way to treat tetanus is you have to outlast it. You have to support the patient because this poison links on chemically and then it has to be slowly metabolized,” Schaffner said. Cases of tetanus have dropped by 95 percent in the U.S. since widespread childhood vaccination and adult booster shots became routine nearly 80 years ago; deaths have dropped 99 percent. The CDC recommends a five-dose series of tetanus shots for children between the ages of 2 months and 6 years and a booster shot every 10 years for adults. ____ Follow Gillian Flaccus on Twitter at http://www.twitter.com/gflaccus Claim: CDC: Unvaccinated Oregon boy almost dies of tetanus.
[EX A]: An unvaccinated 6-year-old Oregon boy was hospitalized for two months for tetanus and almost died of the bacterial illness after getting a deep cut while playing on a farm, according to a case study published Friday by the U.S. Centers for Disease Control and Prevention.

[EX Q]: Paragraph: In 2004, a virulent form of canine influenza surfaced at greyhound racing parks in Florida. In that outbreak, it infected 24 greyhounds and killed 8 more. The contagion has since been confirmed in seven states, having killed greyhounds at tracks in Florida, Massachusetts, Arizona, West Virginia, Wisconsin, Texas and Iowa. It is highly contagious, spreading through shared items (like toys) or human contact (kennel workers have carried the virus home with them), in addition to dog-to-dog encounters. The virus that has been felling greyhounds is an H3N8 flu closely related to an equine flu strain. It is not related to typical human flus or to the H5N1 avian flu that killed about 100 people in Asia. Several states — including Florida, Minnesota, and Louisiana, among others — reported another rash of cases in late June 2017. According to the American Veterinary Medical Association (AVMA), the outbreaks in those states were caused by a strain known as canine H3N2 influenza, which was first reported in Chicago in 2015. An AVMA spokesperson, Sharon Granskog, told us in a phone interview that such outbreaks can be seasonal in nature: There’s a time of year when more dogs are in contact with one another, they’re at dog parks, it’s highly contagious. We suggest that you make sure if your dog is going to a dog park that it’s healthy and talk to a veterinarian about whether or not a vaccine would be right for your dog. The disease was first reported in the U.S. in 2004 after several racing greyhounds in Florida contracted it. The AVMA said that they believed that the dogs became infected by a strain of H3N8 influenza, which is particular to horses. Dr. Ruben Donis, chief of molecular genetics for the influenza branch of the Centers for Disease Control and Prevention, confirmed that the flu jumped from horses to dogs, “a very rare event of considerable scientific interest” and added “at this point, there is no reason to panic.” How that jump occurred is either not known at this point or is not being commented on, so the email’s assertion that the eating of raw meat was to blame should be regarded as speculation rather than as fact. While there is always the possibility the virus might again jump species, this time to humans, this strain of flu has been present in horses for more than 40 years without any documented cases of humans catching it or the related canine flu. But there are vaccines available for both the equine and canine flus. Many readers have been confused (and unnecessarily frightened) by the difference between the terms “morbidity rate” and “mortality rate.” The morbidity rate describes the percentage of animals that will contract the disease after being exposed to the virus, but, despite its name, the term has nothing to do with the death rate associated with the flu. (Nearly 80 percent of dogs exposed to the canine flu virus will contract only a mild form of the disease which mimics kennel cough, a type of canine bronchitis that is rarely serious.) The mortality rate, which describes the percentage of animals that will die after contracting the disease, is in the much lower range of 5 to 8 percent, according to Dr. Cynda Crawford, the veterinary immunologist who first isolated the canine flu virus: The mortality rate is around 5 to 8 percent, says veterinarian Cynda Crawford at the University of Florida’s College of Veterinary Medicine in Gainesville. “I want to stress that despite the rumors that are out on the Internet and other such sources, this disease is not as deadly as people want to make it,” Crawford said. She says she receives more than 30 calls a day from concerned veterinarians. Dr. Crawford describes the contagion as producing in dogs “a moist, productive cough that ends in a gagging response, that will persist for one to four weeks, despite treatment with antibiotics or cough suppressants. Some dogs develop a thick, yellow discharge from the nose. A very few dogs will spike a high fever, between 105 to 107 degrees Fahrenheit. They become lethargic and weak, with rapid, shallow breathing. This is likely to progress to pneumonia.” Other veterinary experts have estimated the potential death rate as between 1 and 10 percent, with the higher percentage applying to very young, very old, or infirm dogs. Presence of the virus in dogs can be confirmed only through blood tests performed at the Cornell University College of Veterinary Medicine. Results of such blood screens take as long as two weeks. Dr. Crawford recommends keeping dogs showing symptoms of respiratory disease at home and away from other dogs for up to two weeks. The CDC, which is tracking the disease, issued no official recommendations. Because the symptoms of this as yet unnamed virus somewhat mimic bordetella, a less virulent illness commonly known as kennel cough, it is hard to ascertain how widespread the flu has become. On the flipside of that confusion, vets in various parts of the country have been thrown into a panic when encountering run-of-the-mill kennel cough in any of their clients, fearing they are instead confronting cases of the new flu. The Animal Health Diagnostic Center at Cornell University inserted a caution against such hair-trigger diagnosis within a larger advisory about the potential for the flu to have spread to the state of New York: “One should not lose sight of the fact that all respiratory infections in dogs are not due to canine influenza virus. Adhering to the ‘band wagon’ approach could result in the failure to appropriately treat dogs with infections previously known to cause respiratory problems in dogs.” We found this good advice for vets and dog owners in our inbox one day: PLEASE DO NOT PANIC, and do NOT assume that every cough is Canine Influenza. Kennel Cough from parainfluenza and Bordetella is more common. However, the Animal Health Diagnostic Center at Cornell is interested in receiving samples from dogs that appear to have kennel cough. Clinical Signs: Since this is a new pathogen in dogs, there is currently no natural immunity present in the unexposed canine population. Almost all exposed dogs will become infected, and nearly 80% have clinical signs. In the mild form the dogs will have a cough that persists for 10 to 21 days. The cough may be soft and moist or dry. Many dogs will have a nasal discharge from a secondary bacterial infection and low grade fever. The nasal discharge responds to broad spectrum antibiotics. In the severe form with pneumonia there is a high fever (104-106 F) and respiratory difficulties. X-rays may show consolidation. These dogs often have secondary bacterial infections and have responded to broad-spectrum antibiotics and supportive care including intravenous hydration. The incubation period is two to five days and dogs may shed virus for seven to 10 days. The disease can spread rapidly throughout a boarding kennel. Dogs that are coughing SHOULD NOT BE BROUGHT TO SHOWS or Performance EVENTS. In a 1 June 2017 advisory to its members, the AVMA suggested that vaccines against the H3N2 virus strain “might be another prevention option” for dogs who were at least 8 weeks old and also recommended isolating dogs who could be infected, as well as not sharing toys between dogs and disinfecting shelter, veterinary, and boarding facilities. Claim: A virus deadly to dogs has been spreading in the canine population of the U.S.
[EX A]: Has a virus deadly to dogs been spreading in the canine population of the U.S.?

[EX Q]: Paragraph: The scientists reached the conclusion in studying the decline in the harvest of four commercially important species of shellfish in coastal areas from Maine to North Carolina — eastern oysters, northern quahogs, softshell clams and northern bay scallops. They reported that their findings came down squarely on the side of a warming ocean environment and a changing climate, and not excessive harvest by fishermen. One of the ways warming has negatively impacted shellfish is by making them more susceptible to predators, said the lead author of the study, Clyde MacKenzie, a shellfish researcher for the National Oceanic and Atmospheric Administration who is based in Sandy Hook, New Jersey. “Their predation rate is faster in the warmer waters. They begin to prey earlier, and they prey longer into the fall,” MacKenzie said. “These stocks have gone down.” MacKenzie’s findings, the product of a collaboration with Mitchell Tarnowski, a shellfish biologist with the Maryland Department of Natural Resources, appeared recently in the journal Marine Fisheries Review. The findings have implications for consumers of shellfish, because a declining domestic harvest means the prices of shellfish such as oysters and clams could rise, or the U.S. could become more dependent on foreign sources. The scientists observed that the harvest of eastern oysters from Connecticut to Virginia fell from around 600,000 bushels in 1960 to less than 100,000 in 2005. The harvest of the four species declined from 1980 to 2010 after enjoying years of stability from 1950 to 1980, they found. The scientists reported that a positive shift in the North Atlantic Oscillation led to the degradation of shellfish habitat. The oscillation is an irregular fluctuation of atmospheric pressure that impacts weather and climate, which in turn affects things like reproduction and food availability for shellfish. The study mirrors what Maine clam harvesters are seeing on the state’s tidal flats, said Chad Coffin, a clammer and the president of the Maine Clammers Association. Maine’s harvest of softshell clams — the clams used to make fried clams and clam chowder — dwindled to its lowest point since 1930 last year. It will take adopting new strategies, such as shellfish farming, for the fisheries to survive, Coffin said. “Clammers aren’t the reason there’s no clams,” he said. “We need to adapt, we need to focus our efforts on adapting to the environment we have.” Some near-shore shellfish harvests in the U.S. remain consistently productive, such as the Maine sea scallop fishery, which takes place in bays and coastal areas in the winter. The state’s scallop fishery bottomed out at about 33,000 pounds in 2005, but has climbed in recent years, and its 2017 total of almost 800,000 pounds was the most since 1997. Many in Maine attribute the health of the fishery to conservative management, said Alex Todd, a scallop fisherman who also works the waters off Massachusetts. “Up and down the coast, there have been good years recently compared to 10 or 15 years ago,” he said. But the scientists’ findings track with others who have studied the impact of warming waters on shellfish, such as Brian Beal, a professor of marine ecology at the University of Maine at Machias. Beal, who was not involved in the study, has said rising seawater temperature could spell “doom and gloom for the clamming industry and probably for other industries as well.” That’s especially true of valuable species that are important food items, like clams and mussels, he said. “None of this can be attributed to overfishing, a term that is used willy-nilly and applied erroneously to these declines in commercially important shellfish,” Beal said. Claim: Warming hurting shellfish, aiding predators, ruining habitat.
[EX A]:
Valuable species of shellfish have become harder to find on the East Coast because of degraded habitat caused by a warming environment, according to a pair of scientists that sought to find out whether environmental factors or overfishing was the source of the decline.