gebhart/patrepeval huggingface dataset:
Survey of dry SO2 control systems (3rd). Final report 1980 (1981)

The report is the third on the status of dry flue gas desulfurization (FGD) processes in the U.S. It reviews current and recently completed research, development, and commercial activities. Dry FGD systems covered include: (1) spray dryers with a fabric filter or an electrostatic precipitator (ESP), (2) dry injection of alkaline material into flue gas, combined with particulate collection in an ESP or a fabric filter, and (3) combustion of coal/alkali fuel mixtures. Spray drying remains the only commercially applied FGD process. With two new utility and two new industrial spray drying system sales since the last report, dry FGD has been selected for 3600 MW of utility generating capacity and about 600,000 acfm of industrial coal-fired flue gas. All new systems use a lime sorbent and a fabric filter for particulate collection. No new commercial systems have begun operation since the Fall of 1980, but the first utility system was scheduled to start up in the Spring of 1981. Pilot-scale demonstration programs which have been recently completed and several continuing programs are discussed. Significant work on dry FGD waste disposal has begun under both government and industry sponsorship and is addressed in the report.
Transfusion Medicine. Second of Two Parts. Blood Conservation (1999)

No AccessJournal of UrologyUrological Survey: Abstracts: Perioperative Care1 Jul 1999Transfusion Medicine. Second of Two Parts. Blood Conservation L.T. Goodnough, M.E. Brecher, M.H. Kantner, and J.P. AuBochon L.T. GoodnoughL.T. Goodnough More articles by this author , M.E. BrecherM.E. Brecher More articles by this author , M.H. KantnerM.H. Kantner More articles by this author , and J.P. AuBochonJ.P. AuBochon More articles by this author View All Author Informationhttps://doi.org/10.1097/00005392-199907000-00112AboutFull TextPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissionsReprints ShareFacebookLinked InTwitterEmail "Transfusion Medicine. Second of Two Parts. Blood Conservation." The Journal of Urology, 162(1), p. 281 Departments of Medicine and Pathology, Washington University School of Medicine, St. Louis, Missouri, Departments of Pathology and Laboratory Medicine, University of North Carolina, Chapel Hill, North Carolina, Department of Pathology, Southern California Permanente Medical Group, Woodland Hills, California, and Departments of Pathology, Dartmouth-Hitchcock Medical Center, Lebanon, New Hampshire© 1999 by American Urological Association, Inc.FiguresReferencesRelatedDetails Volume 162Issue 1July 1999Page: 281 Advertisement Copyright & Permissions© 1999 by American Urological Association, Inc.MetricsAuthor Information L.T. Goodnough More articles by this author M.E. Brecher More articles by this author M.H. Kantner More articles by this author J.P. AuBochon More articles by this author Expand All Advertisement PDF downloadLoading ...
Introducing A Podand Motif to Alkyne Metathesis Catalyst Design: A Highly Active Multidentate Molybdenum(VI) Catalyst that Resists Alkyne Polymerization (2011)

Podand prevents polymers: The molybdenum(VI) propylidyne catalyst 1 with a podand triphenolamine ligand shows high activity in the metathesis of a variety of alkyne substrates, including heterocycles that contain donor moieties. With one substrate-binding site blocked by the multidentate ligand, the undesired polymerization of small alkynes that occurs with non-podand-ligand complex 2 is completely inhibited.
Toll-like receptor 4 agonistic monoclonal antibody reverses new onset type 1 diabetes (BA12P.106) (2014)

Abstract Viral, parasitic or bacterial infections can prevent T1D in NOD mice. The “hygiene hypothesis” proposes that the incidence of autoimmunity is increasing due to decreased immune stimulation in childhood, offering a rationale for stimulation of innate immunity as a treatment for T1D. We demonstrate that a TLR4/MD-2 specific agonistic monoclonal antibody, UT18, can not only prevent T1D, but also reverse new onset T1D (diagnosed by polyuria, weight loss, and elevated blood glucose). UT15, a control TLR4/MD-2 non-agonistic monoclonal antibody, neither prevented nor reversed T1D. In the prevention studies, UT18 treatment resulted in decreased insulitis vs. UT15 treatment. UT18 treatment permanently reversed new onset T1D in 9/13 mice, while UT15 treated mice rapidly progressed to end-stage diabetes (&gt;600 mg/dl). Mice successfully treated with UT18 had less severe islet infiltration and increased insulin positive beta-cell area compared to UT15 controls. Transfer of CD4+ and CD8+ T-cells (from mice successfully treated with UT18) did not cause diabetes for up to 75 days post transfer, whereas cells transferred from UT15 and non-diabetic NOD mice rapidly caused disease. The lack of T cell mediated diabetes transfer suggests that T cell tolerance was restored in new onset diabetic mice treated with UT18. Restoration of T cell tolerance via TLR-4/MD-2 mediated innate immune stimulation is a promising new pathway for treatment of T1D and other T cell mediated autoimmune diseases.
Polarization-Independent Receivers for Low-Cost Coherent OOK Systems (2014)

We demonstrate analytically a novel scheme to obtain polarization-independent operation in low-cost coherent OOK receivers, suitable for access networks. We move from a well-known phase-diversity receiver, exploiting a 3 × 3 coupler, three photodiodes, and basic analogue processing. If that receiver is modified to inject the local oscillator (or the signal) into two inputs with proper polarization states, we show, by both mathematical derivation and numerical simulations, that the resulting electrical signal can be polarization-independent; this is attained only if the frequency-detuning between the signal carrier and the local oscillator is high enough, so that intrinsic second order distortions can be spectrally isolated and suppressed by the low-pass filtering of the receiver.
The soluble pyocins <scp>S2</scp> and <scp>S4</scp> from <i><scp>P</scp>seudomonas aeruginosa</i> bind to the same <scp>FpvAI</scp> receptor (2012)

Soluble (S-type) pyocins are Pseudomonas aeruginosa bacteriocins that kill nonimmune P. aeruginosa cells by gaining entry via a specific receptor, which, in the case of pyocin S2, is the siderophore pyoverdine receptor FpvAI, and in the case of pyocin S3, FpvAII. The nucleic acid sequence at the positions 4327697-4327359 of P. aeruginosa PAO1 genome was not annotated, but it was predicted to encode the immunity gene of the flanking pyocin S4 gene (PA3866) based on our analysis of the genome sequence. Using RT-PCR, the expression of the immunity gene was detected, confirming the existence of an immunity gene overlapping the S4 pyocin gene. The PA3866 coding for pyocin S4 and the downstream gene coding for the immunity protein were cloned and expressed in Escherichia coli and the His-tagged S4 pyocin was obtained in pure form. Forty-three P. aeruginosa strains were typed via PCR to identify their ferripyoverdine receptor gene (fpvAI-III) and were tested for their sensitivity to pyocin S4. All S4-sensitive strains had the type I ferripyoverdine receptor fpvA gene. Some S4-resistant type I fpvA-positive strains were detected, but all of them had the S4 immunity gene, and, following the deletion of the immunity gene, became S4-sensitive. The fpvAI receptor gene was deleted in a S4-sensitive strain, and, as expected, the mutant became resistant to S4. The N-terminal receptor binding domain (RBD) of pyocin S2, which also uses the FpvAI receptor to enter the cell, was cloned in the pET-15b vector, and expressed in E. coli. When the purified RBD was mixed with pyocin S4 at different ratios, an inhibition of killing was observed, indicating that S2 RBD competes with the pyocin S4 for the binding to the FpvAI receptor. The S2 RBD was also shown to enhance the expression of the pvdA pyoverdine gene, suggesting that it, like pyoverdine, works via the known siderophore-mediated signalization pathway.
INTRODUCTORY TO BOOK I (2011)

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Alarm reduction and correlation in defence of IP networks (2005)

Society's critical infrastructures are increasingly dependent on IP networks. Intrusion detection and tolerance within data networks is therefore imperative for dependability in other domains such as telecommunications and future energy management networks. Today's data networks are protected by human operators who are overwhelmed by the massive information overload through false alarm rates of the protection mechanisms. This paper studies the role of alarm reduction and correlation in supporting the security administrator in an enterprise network. We present an architecture that incorporates intrusion detection systems as sensors, and provides improved alarm data to the human operator or to automated actuators. Alarm reduction and correlation via static and adaptive filtering, normalisation, and aggregation is demonstrated on the output from three sensors (Snort, Samhain and Syslog) used in a telecom test network.
Bicycle helmets and injury prevention: A formal review (2000)

Australia is one of the few countries with current uniform helmet legislation (introduced in the early 1990s), but many cyclists continue to ride without head protection. Many peer-reviewed epidemiological studies published over the past decade have investigated the efficacy of helmets in preventing serious injury to bicycle riders involved in crashes. This report presents a formal summary quantification of these results and provides overwhelming evidence in support of helmets for preventing head injury and fatal injury. (a)
Adaptive and dynamic RFID tag anti-collision based on secant iteration (2018)

Radio frequency identification (RFID) has recently experienced unprecedented development. Among many other areas, it has been widely applied in blood station management, automatic supermarket checkout, and logistics. In the application of RFID for large-scale passive tags, tag collision is inevitable owing to the non-cooperation mechanism among tags. Therefore, a tag anti-collision method is a key factor affecting the identification efficiency. In this paper, we propose a tag anti-collision method based on Aloha technology for RFID. It estimates the number of remaining tags using the secant iteration method. To achieve optimal identification efficiency, it adaptively and dynamically adjusts the lengths of the subsequent frames according to the principle that the length of a frame should be the same as the number of tags to be identified. For pseudo-solutions of tag population estimation while using secant iteration, we present an elimination method by two probing frames. The simulation results show that the estimation precision of our method can reach above 97%. Thus, it can meet the requirement of the tag anti-collision estimation accuracy. Its global throughput is obviously superior to the Q algorithm adopted by the current international standard, and it is close to the ideal system. It consequently outperforms existing schemes.
Automated optimization of key WCDMA parameters (2004)

This paper validates the feasibility of automated optimization of key wideband code division multiple access (WCDMA) radio resource management parameters using control methods. The parameters are regularly adjusted in order to improve performance. The parameters examined in this study include the total cell transmission power target, the received total interference target, the downlink radio link power maximums, the handover windows and the pilot channel powers. The control was based on expert-defined rules, which applied specific trade-off policies and statistics of poor quality calls, blocking, packet queuing, power and interference levels and terminal measurements to qualify the parameter values. The approach was validated using a dynamic WCDMA system simulator with a deployment of macro and micro cells on a city region. Results on automated optimization of single parameters on cell level and results on simultaneous multi-parameter optimization on cell-cluster level are presented in this paper. The use of the automated parameter optimization methods was shown to result in a significant increase of capacity in comparison to the default parameter settings. Copyright © 2004 John Wiley & Sons, Ltd.
POLYMER-POLYMER ADHESION IN MELT-PROCESSED LAYERED STRUCTURES (2000)

Improving the adhesion of polyolefins to glassy polymers is complicated by the semicrystalline nature of the polyolefins. Traditional methods used in glassy polymers to increase the interlayer adhesion, including the addition of a diblock copolymer or the formation of a copolymer through in situ reaction, are still successful with semicrystalline polymers. However, melt miscibility of the adhesion promoting molecules is no longer sufficient; they must also co-crystallize. Even when co-crystallization is achieved, the reactive method is shown to provide greater fracture toughness than the addition of a pre-made diblock copolymer. In the latter case, the formation of micelles limits the efficiency of the diblock copolymer. Finally, significant adhesion enhancement is attainable in reactive systems with contact times as short as 45 seconds as demonstrated through a multilayer coextrusion of amorphous nylon against a polypropylene-maleated polypropylene blend.
Propagation mechanisms for UHF wave transmission through walls and windows (2003)

In this paper, both simulations and measurements are used to explore the propagation properties of ultrahigh-frequency (UHF) waves traveling through building structures and, in particular, windows. The propagation influence of several features of a typical window structure is examined, including the electrical properties of the surrounding wall, the homogeneity and roughness of this wall, and the incidence angle of the UHF wave. In these studies, ray tracing is benchmarked against both narrow-band measurements and the results of a full-wave moment method technique. Some of the inherent limitations of ray tracing are assessed and corrective methods are proposed in order to improve its accuracy.
Impact of endotoxin on UCP homolog mRNA abundance, thermoregulation, and mitochondrial proton leak kinetics (2000)

Linking tissue uncoupling protein (UCP) homolog abundance with functional metabolic outcomes and with expression of putative genetic regulators promises to better clarify UCP homolog physiological function. A murine endotoxemia model characterized by marked alterations in thermoregulation was employed to examine the association between heat production, UCP homolog expression, and mitochondrial proton leak ("uncoupling"). After intraperitoneal lipopolysaccharide (LPS, approximately 6 mg/kg) injection, colonic temperature (T(c)) in adult female C57BL6/J mice dropped to a nadir of approximately 30 degrees C by 8 h, preceded by a four- to fivefold drop in liver UCP2 and UCP5/brain mitochondrial carrier protein 1 mRNA levels, with no change in their hindlimb skeletal muscle (SKM) expression. SKM UCP3 mRNA rose fivefold during development of hypothermia and was correlated with an LPS-induced increase in plasma free fatty acid concentration. UCP2 and UCP5 transcripts recovered about three- to sixfold in both tissues starting at 6-8 h, preceding a recovery of T(c) between 16 and 24 h. SKM UCP3 followed an opposite pattern. Such results are not consistent with an important influence of UCP3 in driving heat production but do not preclude a role for UCP2 or UCP5 in this process. The transcription coactivator PGC-1 displayed a transient LPS-evoked rise (threefold) or drop (two- to fivefold) in SKM and liver expression, respectively. No differences between control and LPS-treated mouse liver or SKM in vitro mitochondrial proton leak were evident at time points corresponding to large differences in UCP homolog expression.
Efficient all-polymer solar cells based on blend of tris(thienylenevinylene)-substituted polythiophene and poly[perylene diimide-<i>alt</i>-bis(dithienothiophene)] (2008)

A narrow band-gap alternating copolymer of perylene diimide and bis(dithienothiophene) (2) and a polythiophene derivative substituted by a tris(thienylenevinylene) conjugated side chain (4) are used as acceptor and donor, respectively, in all-polymer solar cells (SCs). The optimized device based on the blend of 4 and 2 in the ratio 3:1 (w/w) gives a short circuit current (Jsc) of 5.02 mA cm−2 and a power conversion efficiency of 1.48%, under simulated AM 1.5 illumination at 100 mW cm−2. These values are among the highest values reported for all-polymer SCs.
July 2006 version (2006)

Background: Referrals of junior and secondary age children with language and communication impairments attending mainstream schools; 
Evidence that early language and communication problems may not resolve; 
Lack of secondary school SLT provision; 
Lack of knowledge about the nature and extent of difficulties secondary students may encounter; 
Lack of suitable assessments.
An annotated bibliography of GRASP – Part I: Algorithms (2008)

Abstract A greedy randomized adaptive search procedure (GRASP) is a metaheuristic for combinatorial optimization. It is a multi‐start or iterative process, in which each GRASP iteration consists of two phases, a construction phase, in which a feasible solution is produced, and a local search phase, in which a local optimum in the neighborhood of the constructed solution is sought. Since 1989, numerous papers on the basic aspects of GRASP, as well as enhancements to the basic metaheuristic have appeared in the literature. GRASP has been applied to a wide range of combinatorial optimization problems, ranging from scheduling and routing to drawing and turbine balancing. This is the first of two papers with an annotated bibliography of the GRASP literature from 1989 to 2008. This paper covers algorithmic aspects of GRASP.
Enhancement of phorbol diester-induced HL-60-mediated cytotoxicity by retinoic acid, dimethyl sulfoxide, and 5-azacytidine. (1986)

Both peripheral blood monocytes and neutrophils are known to be capable of lysing a variety of extracellular tumor and non-tumor cell targets. The HL-60 human promyelocytic leukemia cell line has served as a useful model of human granulocyte and macrophage differentiation in studies from many laboratories. We have previously reported that phorbol diesters, which induce differentiation along the macrophage pathway, stimulate HL-60 cells to become strikingly cytotoxic to a variety of red cell targets. We now report that agents known to differentiate HL-60 along the granulocyte pathway (retinoic acid, dimethyl sulfoxide, 5-azacytidine) do not, in themselves, induce HL-60 to become cytotoxic. However, previous exposure (3-5 days) to these granulocyte pathway active agents markedly enhances phorbol diester-triggered killing. This enhancement is particularly striking at decreased effector:target ratios (as low as one effector per five targets) and is also demonstrated by a shift to lower concentrations of the phorbol diester dose-response curve. Retinoic acid is the most effective of the three agents tested, although priming (previous exposure) with dimethyl sulfoxide or 5-azacytidine also markedly enhances killing. These studies demonstrate that HL-60-mediated killing may be dissected pharmacologically into at least two distinct steps and further support the utility of this model system in studies of the development of macrophage-like cytotoxic cells. This system has also proven to be useful in the characterization of cytokines which mimic the differentiation effects of retinoic acid and dimethyl sulfoxide (J. A. Leftwich and R. E. Hall, manuscript in preparation).
Evidence for proteolytic cleavage of the 120-kilodalton outer membrane protein of rickettsiae: identification of an avirulent mutant deficient in processing (1992)

The 120-kDa rickettsial outer membrane protein (rOmpB) is encoded by a gene with the capacity to encode a protein of approximately 168 kDa. The carboxy-terminal end of the molecule is apparently cleaved to yield 120- and 32-kDa products. Both polypeptides are surface exposed and remain associated with the outer membrane of intact rickettsiae. All species of rickettsiae examined display similar cleavage of rOmpB. Comparison of diverse species of rickettsiae demonstrate a conserved N terminus of the 32-kDa fragment, with a predicted procaryotic secretory signal peptide immediately upstream of the proposed cleavage site. Coprecipitation of the 120-kDa rOmpB protein and the 32-kDa peptide by monoclonal antibodies specific for the 120-kDa portion of the molecule suggests that the two fragments remain noncovalently associated on the surface of rickettsiae. Analysis of an avirulent mutant of Rickettsia rickettsii revealed reduced amounts of the 120- and 32-kDa fragments, but with a correspondingly larger rOmpB protein that displayed properties expected of the putative precursor. This avirulent mutant grows intracellularly but fails to cause the lysis of infected cells that is typical of R. rickettsii. DNA sequence analysis of the region of the gene encoding the cleavage site of the avirulent strain revealed no difference from the sequence obtained from virulent R. rickettsii. The 168-kDa putative precursor of the avirulent strain of R. rickettsii was not extracted from the surface by dilute buffers, as is the 120-kDa protein of virulent R. rickettsii or R. prowazekii. These latter results suggest that the 32-kDa C-terminal region of the molecule may serve as a membrane anchor domain.
Involvement of class II MHC molecules in the LPS-induction of IL-1/TNF secretions by human monocytes. Quantitative differences at the polymorphic level. (1989)

Anti-class II ag mAb (DR and DQ) inhibited, in a dose-dependent manner, LPS-induced IL-1 and TNF secretions from human monocytes (34 to 95% inhibition). The potentiating effect of IFN-gamma on LPS-induced TNF secretion (15.3 +/- 0.7 to 44 +/- 0.6 ng/ml) was also blocked by anti-class II ag mAb (44 +/- 0.6 to 0.3 +/- 0.03 ng/ml). We also report a relationship between interindividual differences in monocyte IL-1 and TNF secretions and the HLA-D-encoded genetic polymorphism. Heterozygotes were, in general, higher secretors of those cytokines than homozygotes. Analysis of these secretions in heterozygotes demonstrated a differential effect of certain haplotype combinations (i.e., DR2-DR4 vs DR2-DR3) that could be arbitrarily characterized as being "low" or "high" secretors (6,230 +/- 2,950 vs 13,029 +/- 6,541 cpm for IL-1, and 12 +/- 10 vs 25 +/- 15 ng/ml for TNF, p = 0.006 and 0.048). DR-associated Dw subtypes appeared to account for differences within certain haplotype combinations (Dw18 vs Dw19 in DRw13/DR4) (11,227 +/- 3,648 vs 17,166 +/- 3,176 cpm for IL-1, and 13 +/- 9 vs 25 +/- 10 ng/ml for TNF, p = 0.02 and 0.047). Interindividual differences were better explained by differences in LPS sensitivity than by differences in the kinetics of secretion and related not to the secretory process itself but to the rate of cytokine synthesis. Finally, there were no relationships between high secretor genotypes and IDD high risk genotypes. Thus, we conclude that, a) LPS-induced IL-1 and TNF secretions are, at least in part, regulated by class II MHC molecules, b) that HLA-D region-encoded genetic polymorphism accounts for interindividual differences in these secretions, and c) that the HLA-associated risk to develop IDD is not explained by these cytokine secretory differences as previously proposed.