fenil210/Med-Lamma huggingface dataset:
What to do for Prostate Enlargement: Benign Prostatic Hyperplasia ?
- Benign prostatic hyperplasiaalso called BPHis a condition in men in which the prostate gland is enlarged and not cancerous.  - The prostate is a walnut-shaped gland that is part of the male reproductive system.  - The cause of benign prostatic hyperplasia is not well understood; however, it occurs mainly in older men.  - Benign prostatic hyperplasia is the most common prostate problem for men older than age 50.  - Lower urinary tract symptoms suggestive of benign prostatic hyperplasia may include      - urinary frequencyurination eight or more times a day    - urinary urgencythe inability to delay urination    - trouble starting a urine stream    - a weak or an interrupted urine stream    - dribbling at the end of urination    - nocturiafrequent urination during periods of sleep    - urinary retentionthe inability to empty the bladder completely    - urinary incontinencethe accidental loss of urine    - pain after ejaculation or during urination    - urine that has an unusual color or smell    - The complications of benign prostatic hyperplasia may include      - acute urinary retention    - chronic, or long lasting, urinary retention    - blood in the urine    - urinary tract infections (UTIs)    - bladder damage    - kidney damage    - bladder stones    - A health care provider diagnoses benign prostatic hyperplasia based on      - a personal and family medical history    - a physical exam    - medical tests    - Treatment options for benign prostatic hyperplasia may include      - lifestyle changes    - medications    - minimally invasive procedures    - surgery    - The complications of benign prostatic hyperplasia treatment depend on the type of treatment.  - Researchers have not found a way to prevent benign prostatic hyperplasia.  - Researchers have not found that eating, diet, and nutrition play a role in causing or preventing benign prostatic hyperplasia. + NIDDK + Prostate Enlargement: Benign Prostatic Hyperplasia
What is (are) Dumping Syndrome ?
Dumping syndrome occurs when food, especially sugar, moves too fast from the stomach to the duodenumthe first part of the small intestinein the upper gastrointestinal (GI) tract. This condition is also called rapid gastric emptying. Dumping syndrome has two forms, based on when symptoms occur:
                
- early dumping syndromeoccurs 10 to 30 minutes after a meal  - late dumping syndromeoccurs 2 to 3 hours after a meal + NIDDK + Dumping Syndrome
What is (are) Dumping Syndrome ?
The GI tract is a series of hollow organs joined in a long, twisting tube from the mouth to the anusthe opening where stool leaves the body. The body digests food using the movement of muscles in the GI tract, along with the release of hormones and enzymes. The upper GI tract includes the mouth, esophagus, stomach, duodenum, and small intestine. The esophagus carries food and liquids from the mouth to the stomach. The stomach slowly pumps the food and liquids into the intestine, which then absorbs needed nutrients. Two digestive organs, the liver and the pancreas, produce digestive juices that reach the small intestine through small tubes called ducts.
                
The last part of the GI tractcalled the lower GI tractconsists of the large intestine and anus. The large intestine is about 5 feet long in adults and absorbs water and any remaining nutrients from partially digested food passed from the small intestine. The large intestine then changes waste from liquid to a solid matter called stool. Stool passes from the colon to the rectum. The rectum is located between the last part of the coloncalled the sigmoid colonand the anus. The rectum stores stool prior to a bowel movement. During a bowel movement, stool moves from the rectum to the anus. + NIDDK + Dumping Syndrome
What causes Dumping Syndrome ?
Dumping syndrome is caused by problems with the storage of food particles in the stomach and emptying of particles into the duodenum. Early dumping syndrome results from rapid movement of fluid into the intestine following a sudden addition of a large amount of food from the stomach. Late dumping syndrome results from rapid movement of sugar into the intestine, which raises the body's blood glucose level and causes the pancreas to increase its release of the hormone insulin. The increased release of insulin causes a rapid drop in blood glucose levels, a condition known as hypoglycemia, or low blood sugar. + NIDDK + Dumping Syndrome
What are the symptoms of Dumping Syndrome ?
The symptoms of early and late dumping syndrome are different and vary from person to person. Early dumping syndrome symptoms may include
                
- nausea  - vomiting  - abdominal pain and cramping  - diarrhea  - feeling uncomfortably full or bloated after a meal  - sweating  - weakness  - dizziness  - flushing, or blushing of the face or skin  - rapid or irregular heartbeat
                
- hypoglycemia  - sweating  - weakness  - rapid or irregular heartbeat  - flushing  - dizziness
                
About 75 percent of people with dumping syndrome report symptoms of early dumping syndrome and about 25 percent report symptoms of late dumping syndrome. Some people have symptoms of both types of dumping syndrome.1 + NIDDK + Dumping Syndrome
What are the treatments for Dumping Syndrome ?
Treatment for dumping syndrome includes changes in eating, diet, and nutrition; medication; and, in some cases, surgery. Many people with dumping syndrome have mild symptoms that improve over time with simple dietary changes. + NIDDK + Dumping Syndrome
What to do for Dumping Syndrome ?
The first step to minimizing symptoms of dumping syndrome involves changes in eating, diet, and nutrition, and may include
                
- eating five or six small meals a day instead of three larger meals  - delaying liquid intake until at least 30 minutes after a meal  - increasing intake of protein, fiber, and complex carbohydratesfound in starchy foods such as oatmeal and rice  - avoiding simple sugars such as table sugar, which can be found in candy, syrup, sodas, and juice beverages  - increasing the thickness of food by adding pectin or guar gumplant extracts used as thickening agents
                
Some people find that lying down for 30 minutes after meals also helps reduce symptoms.
                
Medication
                
A health care provider may prescribe octreotide acetate (Sandostatin) to treat dumping syndrome symptoms. The medication works by slowing gastric emptying and inhibiting the release of insulin and other GI hormones. Octreotide comes in short- and long-acting formulas. The short-acting formula is injected subcutaneouslyunder the skinor intravenouslyinto a veintwo to four times a day. A health care provider may perform the injections or may train the patient or patient's friend or relative to perform the injections. A health care provider injects the long-acting formula into the buttocks muscles once every 4 weeks. Complications of octreotide treatment include increased or decreased blood glucose levels, pain at the injection site, gallstones, and fatty, foul-smelling stools.
                
Surgery
                
A person may need surgery if dumping syndrome is caused by previous gastric surgery or if the condition is not responsive to other treatments. For most people, the type of surgery depends on the type of gastric surgery performed previously. However, surgery to correct dumping syndrome often has unsuccessful results. + NIDDK + Dumping Syndrome
What to do for Dumping Syndrome ?
- Dumping syndrome occurs when food, especially sugar, moves too fast from the stomach to the duodenumthe first part of the small intestinein the upper gastrointestinal (GI) tract.  - Dumping syndrome has two forms, based on when symptoms occur:      - early dumping syndromeoccurs 10 to 30 minutes after a meal    - late dumping syndromeoccurs 2 to 3 hours after a meal    - People who have had surgery to remove or bypass a significant part of the stomach are more likely to develop dumping syndrome. Other conditions that impair how the stomach stores and empties itself of food, such as nerve damage caused by esophageal surgery, can also cause dumping syndrome.  - Early dumping syndrome symptoms include      - nausea    - vomiting    - abdominal pain and cramping    - diarrhea    - feeling uncomfortably full or bloated after a meal    - sweating    - weakness    - dizziness    - flushing, or blushing of the face or skin    - rapid or irregular heartbeat    - The symptoms of late dumping syndrome include      - hypoglycemia    - sweating    - weakness    - rapid or irregular heartbeat    - flushing    - dizziness    - Treatment for dumping syndrome includes changes in eating, diet, and nutrition; medication; and, in some cases, surgery. Many people with dumping syndrome have mild symptoms that improve over time with simple dietary changes. + NIDDK + Dumping Syndrome
What is (are) Renal Tubular Acidosis ?
Renal tubular acidosis (RTA) is a disease that occurs when the kidneys fail to excrete acids into the urine, which causes a person's blood to remain too acidic. Without proper treatment, chronic acidity of the blood leads to growth retardation, kidney stones, bone disease, chronic kidney disease, and possibly total kidney failure.
                
The body's cells use chemical reactions to carry out tasks such as turning food into energy and repairing tissue. These chemical reactions generate acids. Some acid in the blood is normal, but too much acidacidosiscan disturb many bodily functions. Healthy kidneys help maintain acid-base balance by excreting acids into the urine and returning bicarbonatean alkaline, or base, substanceto the blood. This "reclaimed" bicarbonate neutralizes much of the acid that is created when food is broken down in the body. The movement of substances like bicarbonate between the blood and structures in the kidneys is called transport.
                
One researcher has theorized that Charles Dickens may have been describing a child with RTA in the character of Tiny Tim from A Christmas Carol. Tiny Tim's small stature, malformed limbs, and periods of weakness are all possible consequences of the chemical imbalance caused by RTA.1 In the story, Tiny Tim recovers when he receives medical treatment, which would likely have included sodium bicarbonate and sodium citrate, alkaline agents to neutralize acidic blood. The good news is that medical treatment can indeed reverse the effects of RTA. + NIDDK + Renal Tubular Acidosis
How to diagnose Renal Tubular Acidosis ?
To diagnose RTA, doctors check the acid-base balance in blood and urine samples. If the blood is more acidic than it should be and the urine less acidic than it should be, RTA may be the reason, but additional information is needed to rule out other causes. If RTA is the reason, additional information about the sodium, potassium, and chloride levels in the urine and the potassium level in the blood will help identify which type of RTA a person has. In all cases, the first goal of therapy is to neutralize acid in the blood, but different treatments may be needed to address the different underlying causes of acidosis. + NIDDK + Renal Tubular Acidosis
What to do for Renal Tubular Acidosis ?
- Renal tubular acidosis (RTA) is a disease that occurs when the kidneys fail to excrete acids into the urine, which causes a person's blood to remain too acidic.  - Without proper treatment, chronic acidity of the blood leads to growth retardation, kidney stones, bone disease, chronic kidney disease, and possibly total kidney failure.  - If RTA is suspected, additional information about the sodium, potassium, and chloride levels in the urine and the potassium level in the blood will help identify which type of RTA a person has.  - In all cases, the first goal of therapy is to neutralize acid in the blood, but different treatments may be needed to address the different underlying causes of acidosis. + NIDDK + Renal Tubular Acidosis
What is (are) Kidney Dysplasia ?
Kidney dysplasia is a condition in which the internal structures of one or both of a fetus kidneys do not develop normally while in the womb. During normal development, two thin tubes of muscle called ureters grow into the kidneys and branch out to form a network of tiny structures called tubules. The tubules collect urine as the fetus grows in the womb. In kidney dysplasia, the tubules fail to branch out completely. Urine that would normally flow through the tubules has nowhere to go. Urine collects inside the affected kidney and forms fluid-filled sacs called cysts. The cysts replace normal kidney tissue and prevent the kidney from functioning.
                
Kidney dysplasia can affect one kidney or both kidneys. Babies with severe kidney dysplasia affecting both kidneys generally do not survive birth. Those who do survive may need the following early in life:
                
- blood-filtering treatments called dialysis  - a kidney transplant
                
Children with dysplasia in only one kidney have normal kidney function if the other kidney is unaffected. Those with mild dysplasia of both kidneys may not need dialysis or a kidney transplant for several years.
                
Kidney dysplasia is also called renal dysplasia or multicystic dysplastic kidney. + NIDDK + Kidney Dysplasia
What is (are) Kidney Dysplasia ?
The kidneys are two bean-shaped organs, each about the size of a fist. They are located just below the rib cage, one on each side of the spine. Every day, the two kidneys filter about 120 to 150 quarts of blood to produce about 1 to 2 quarts of urine, which is composed of wastes and extra fluid. Children produce less urine than adultsthe amount they produce depends on their age. The urine flows from the kidneys to the bladder through the two ureters, one on each side of the bladder. The bladder stores urine. The muscles of the bladder wall remain relaxed while the bladder fills with urine. As the bladder fills to capacity, signals sent to the brain tell a person to find a toilet soon. When the bladder empties, urine flows out of the body through a tube called the urethra, located at the bottom of the bladder.
                
The kidneys, ureters, bladder, and urethra are parts of the urinary tract. More information is provided in the NIDDK health topics, the kidneys and the urinary tract. + NIDDK + Kidney Dysplasia
What causes Kidney Dysplasia ?
Genetic factors can cause kidney dysplasia. Genes pass information from both parents to the child and determine the childs traits. Sometimes, parents may pass a gene that has changed, or mutated, causing kidney dysplasia.
                
Genetic syndromes that affect multiple body systems can also cause kidney dysplasia. A syndrome is a group of symptoms or conditions that may seem unrelated yet are thought to have the same genetic cause. A baby with kidney dysplasia due to a genetic syndrome might also have problems of the digestive tract, nervous system, heart and blood vessels, muscles and skeleton, or other parts of the urinary tract.
                
A baby may also develop kidney dysplasia if his or her mother takes certain prescription medications during pregnancy, such as some used to treat seizures and high blood pressure. A mothers use of illegal drugs, such as cocaine, during pregnancy may also cause kidney dysplasia in her unborn child. + NIDDK + Kidney Dysplasia
How many people are affected by Kidney Dysplasia ?
Kidney dysplasia is a common condition. Scientists estimate that kidney dysplasia affects about one in 4,000 babies.1 This estimate may be low because some people with kidney dysplasia are never diagnosed with the condition. About half of the babies diagnosed with this condition have other urinary tract defects.2 + NIDDK + Kidney Dysplasia
What are the symptoms of Kidney Dysplasia ?
Many babies with kidney dysplasia in only one kidney have no signs of the condition. In some cases, the affected kidney may be enlarged at birth and may cause pain. + NIDDK + Kidney Dysplasia
What are the complications of Kidney Dysplasia ?
The complications of kidney dysplasia can include
                
- hydronephrosis of the working kidney. A baby with kidney dysplasia in only one kidney might have other urinary tract defects. When other defects in the urinary tract block the flow of urine, the urine backs up and causes the kidneys and ureters to swell, a condition called hydronephrosis. If left untreated, hydronephrosis can damage the working kidney and reduce its ability to filter blood. Kidney damage may lead to chronic kidney disease (CKD) and kidney failure.  - a urinary tract infection (UTI). A urine blockage may increase a babys chance of developing a UTI. Recurring UTIs can also lead to kidney damage.  - high blood pressure.  - a slightly increased chance of developing kidney cancer.
                
More information is provided in the NIDDK health topics, urine blockage in newbornsand UTIs in children. + NIDDK + Kidney Dysplasia
How to diagnose Kidney Dysplasia ?
Health care providers may be able to diagnose kidney dysplasia during a womans pregnancy using a fetal ultrasound, also called a fetal sonogram. Ultrasound uses a device, called a transducer, that bounces safe, painless sound waves off organs to create an image of their structure. Fetal ultrasound is a test done during pregnancy to create images of the fetus in the womb. A specially trained technician performs the procedure in a health care providers office, an outpatient center, or a hospital, and an obstetrician or a radiologist interprets the images. An obstetrician is a doctor who specializes in pregnancy and childbirth. A radiologist is a doctor who specializes in medical imaging. The patientin this case, the fetus motherdoes not need anesthesia for this procedure. The images can show defects in the fetus kidneys and other parts of the urinary tract.
                
Health care providers do not always diagnose kidney dysplasia before a baby is born. After birth, health care providers often diagnose kidney dysplasia during an evaluation of the child for a UTI or another medical condition. A health care provider uses ultrasound to diagnose kidney dysplasia after the baby is born. + NIDDK + Kidney Dysplasia
What are the treatments for Kidney Dysplasia ?
If the condition is limited to one kidney and the baby has no signs of kidney dysplasia, no treatment may be necessary. However, the baby should have regular checkups that include
                
- checking blood pressure.  - testing blood to measure kidney function.  - testing urine for albumin, a protein most often found in blood. Albumin in the urine may be a sign of kidney damage.  - performing periodic ultrasounds to monitor the damaged kidney and to make sure the functioning kidney continues to grow and remains healthy. + NIDDK + Kidney Dysplasia
How to prevent Kidney Dysplasia ?
Researchers have not found a way to prevent kidney dysplasia caused by genetic factors or certain genetic syndromes. Pregnant women can prevent kidney dysplasia by avoiding the use of certain prescription medications or illegal drugs during pregnancy. Pregnant women should talk with their health care provider before taking any medications during pregnancy. + NIDDK + Kidney Dysplasia
What is the outlook for Kidney Dysplasia ?
The long-term outlook for a child with kidney dysplasia in only one kidney is generally good. A person with one working kidney, a condition called solitary kidney, can grow normally and may have few, if any, health problems.
                
The affected kidney may shrink as the child grows. By age 10,3 the affected kidney may no longer be visible on x-ray or ultrasound. Children and adults with only one working kidney should have regular checkups to test for high blood pressure and kidney damage. A child with urinary tract problems that lead to failure of the working kidney may eventually need dialysis or a kidney transplant.
                
More information is provided in the NIDDK health topics, solitary kidney, dialysis, and kidney transplants. + NIDDK + Kidney Dysplasia
What is the outlook for Kidney Dysplasia ?
The long-term outlook for a child with kidney dysplasia in both kidneys is different from the long-term outlook for a child with one dysplastic kidney. A child with kidney dysplasia in both kidneys
                
- is more likely to develop CKD.  - needs close follow-up with a pediatric nephrologista doctor who specializes in caring for children with kidney disease. Children who live in areas that dont have a pediatric nephrologist available can see a nephrologist who cares for both children and adults.  - may eventually need dialysis or a kidney transplant. + NIDDK + Kidney Dysplasia
What to do for Kidney Dysplasia ?
Researchers have not found that eating, diet, and nutrition play a role in causing or preventing kidney dysplasia. + NIDDK + Kidney Dysplasia
What to do for Kidney Dysplasia ?
- Kidney dysplasia is a condition in which the internal structures of one or both of a fetus kidneys do not develop normally while in the womb.  - Genetic factors can cause kidney dysplasia.  - Genetic syndromes that affect multiple body systems can also cause kidney dysplasia.  - A baby may also develop kidney dysplasia if his or her mother takes certain prescription medications during pregnancy, such as some used to treat seizures and high blood pressure.  - Many babies with kidney dysplasia in only one kidney have no signs of the condition.  - Health care providers may be able to diagnose kidney dysplasia during a womans pregnancy using a fetal ultrasound, also called a fetal sonogram.  - Health care providers do not always diagnose kidney dysplasia before a baby is born.  - If the condition is limited to one kidney and the baby has no signs of kidney dysplasia, no treatment may be necessary.  - Researchers have not found a way to prevent kidney dysplasia caused by genetic factors or certain genetic syndromes.  - Pregnant women can prevent kidney dysplasia by avoiding the use of certain prescription medications or illegal drugs during pregnancy.  - The long-term outlook for a child with kidney dysplasia in only one kidney is generally good.  - The long-term outlook for a child with kidney dysplasia in both kidneys is different from the long-term outlook for a child with one dysplastic kidney. A child with kidney dysplasia in both kidneys      - is more likely to develop chronic kidney disease (CKD)    - needs close follow-up with a pediatric nephrologist    - may eventually need dialysis or a kidney transplant + NIDDK + Kidney Dysplasia
What is (are) Viral Gastroenteritis ?
Viral gastroenteritis is inflammation of the lining of the stomach, small intestine, and large intestine. Several different viruses can cause viral gastroenteritis, which is highly contagious and extremely common. Viral gastroenteritis causes millions of cases of diarrhea each year.
                
Anyone can get viral gastroenteritis and most people recover without any complications, unless they become dehydrated. + NIDDK + Viral Gastroenteritis
What are the symptoms of Viral Gastroenteritis ?
The main symptoms of viral gastroenteritis are
                
- watery diarrhea  - vomiting
                
Other symptoms include
                
- headache  - fever  - chills  - abdominal pain
                
Symptoms usually appear within 12 to 48 hours after exposure to a gastroenteritis-causing virus and last for 1 to 3 days. Some viruses cause symptoms that last longer. + NIDDK + Viral Gastroenteritis