## 4 Discussion
Again, the literature is inconclusive in assessing the impact of white cereals on obesity risk. Studies have shown no direct correlation between the consumption of white grains and cardiometabolic risk [78; 79]. Results contradicting this were obtained in a cohort study, which confirmed that weight gain was positively related to the intake of refined-grain foods [80]. This result may be further evidence that the risk of metabolic diseases cannot be assessed from the perspective of the consumption of selected food groups. To determine such a risk or to find the cause of android fatness, it is necessary to consider dietary patterns, genetic implications and disease burden, which in the case of our group may have been important. We believe that self-esteem was not irrelevant here. In addition, our study's HAF and NAF women had adequate nutritional knowledge and reported comparable physical activity levels. Therefore, we should consider perceived barriers to healthy eating and dietary behaviours: 'lack of willpower, 'time constraints' and 'taste preferences', especially among young women [81]. The group that consumes dairy less frequently consumes meat and cereals more often. In summary, we would like to emphasise that the relationship between self-esteem and eating behaviours is mediated by complex biological mechanisms, including the brain's reward system, hormonal regulation, neurotransmitter balance, and the body's stress response. Low self-esteem can contribute to unhealthy eating patterns, including emotional eating and eating disorders, through these biological pathways. From neurobiology, it is known that areas of the brain that are involved in self-esteem, such as the prefrontal cortex (involved in self-regulation and decision-making) and the ventral striatum (which plays a role in reward processing), are also involved in eating behaviour [82]. The interaction between these brain regions can influence how we perceive ourselves and respond to food cues. Low self-esteem can lead to reduced activity in the prefrontal cortex, impairing the ability to make healthy decisions about eating [83]. The brain's reward system, particularly the dopaminergic system, plays a significant role in both self-esteem and eating behaviour [84]. When self-esteem is low, people may seek external sources of pleasure or reward, which can lead to overestimating or eating unhealthy foods to \"feel better\", triggering dopamine release in the brain and reinforcing unhealthy eating habits. Dopamine is involved in the brain's reward system, and eating can trigger the release of dopamine. Individuals with low self-esteem may seek out foods that activate the dopamine system. Low self-esteem can affect serotonin levels in the brain [85]. Serotonin is a neurotransmitter that plays a role in mood regulation, appetite control, and impulse control [85; 86]. Low self-esteem is often associated with higher levels of stress, and chronic stress can lead to elevated cortisol levels [87]. Cortisol, a stress hormone, can influence eating behaviour by increasing appetite, particularly for high-calorie, \"comfort\" foods [88]. Leptin and ghrelin regulate the appetite [89]. Leptin signals satiety (feeling of fullness), while ghrelin stimulates hunger. Research suggests that individuals with low self-esteem may experience imbalances in these hormones, potentially leading to either overeating or under-eating [90]. For example, low self-esteem might cause emotional distress that alters normal hunger cues, leading to disrupted eating patterns. In conclusion, our study achieved our goal and answered whether self-esteem drives dietary behaviours and fat distribution in women with menstrual bleeding disorders. However, the answer to this question was surprising because women with higher levels of self-esteem over-modified their diets and, at the same time, presented higher android fatness. Our work has some limitations.