## 2 Materials and Methods
\begin{table}
\begin{tabular}{l l} \hline \hline
**\# Case (Page)** & **Diagnosis** \\ \hline
**7 (page 148)** & Out-of-hospital cardiorespiratory arrest due to ventricular fibrillation. Cardiac sarcoidosis with \\
**7 (page 148)** & intramyocardial scarring but no evidence of active intramyocardial inflammation. Preserved left ventricular \\  & systolic function. Coronary arteries without lesions or coronary anomalies. Subcutaneous ICD implantation \\  & for secondary prevention. \\ \hline
**8 (page 211)** & Congestive heart failure in anasarca situation. Moderate left ventricular dysfunction. Type 2 diabetes mellitus \\  & with poor chronic metabolic control (HbA1c 7.2\%) and microvascular involvement. Three-vessel coronary \\
**8 (page 211)** & artery disease: severe stenosis of the proximal anterior descending artery, percutaneously revascularised by \\  & drug-eluting stent implantation. Ischaemic stroke secondary to left carotid artery occlusion, revascularised by \\  & stenting. Initial NIHSS of 2. \\ \hline
**9 (page 219)** & Acute myocardial infarction with ST-segment elevation of inferior location. Three-vessel coronary artery \\  & disease: Mid CD (ARI) 90\%, mid DA 90\%, distal Cx. 40\%. Primary PCI with drug-eluting stent over medial \\  & DC. Elective PCI second outpatient PCI on severe mid LAD lesion. Mild left ventricular dysfunction (LVEF \\  & 50\%). Peripheral arterial disease. Type 2 diabetes mellitus with good metabolic control. LDL cholesterol above \\  & target (\(<\)50 mg/dl). High blood pressure. \\ \hline
**10 (page 230)** & Idiopathic dilated cardiomyopathy with moderate dysfunction (EF 40\%), probably in the context of old \\  & myocarditis. Coronary arteries without angiographically significant lesions. DM2 without optimal control \\  & (Hb1 Ac 7.3\%). \\ \hline
**11 (page 365)** & First episode of heart failure. Severe biventricular dysfunction. Coronary arteries without significant lesions. \\  & Non-sustained monomorphic ventricular tachycardia. Paroxysmal atrial fibrillation. Intracavitary thrombus \\
**11 (page 365)** & (left ventricle), not seen on cardiac MRI after initiation of anticoagulation. Subacute ischaemic stroke in the \\  & right PCA territory of probable cardioembolic aetiology, with subsequent haemorrhagic transformation. HIV \\  & infection under treatment with good control. Consumption of toxic substances. \\ \hline
**12 (page 427)** & Extensive anterior STEACS. Haemodynamic status KK1. Single vessel coronary artery disease (SCAD). \\  & Percutaneous total revascularisation using a drug-eluting stent. Moderate LV dysfunction (LVEF 44\%). \\  & Hyperlipoproteinaemia A. \\ \hline
**13 (page 465)** & Anomalous pulmonary venous connection: drainage from right superior pulmonary vein to superior vena \\  & cava. Significant left-right shunt with right ventricular volume overload. Normal RV systolic function. Focal \\  & atrial tachycardia. Radiofrequency ablation effective. \\ \hline
**14 (page 523)** & AL cardiac amyloidosis. Paroxysmal atrial fibrillation, pharmacologic cardioversion. CHADS2-VA2 SC 1 point. \\  & Moderately impaired LVEF. Moderately low mitral regurgitation. Multiple myeloma with cardiac and renal \\  & involvement in active treatment. Febrile syndrome in a patient with mild bicyclopenia. Chronic kidney disease \\  & secondary to monoclonal gammopathy. \\ \hline
**15 (page 633)** & Possible acute myocardial infarction on underlying heart disease presenting as cardiogenic shock with a favourable \\
**16 (page 633)** & cardiomyopathy secondary to MELAS. WPW syndrome (left lateral tract). Recurrent intraventricular thrombus \\  & with peripheral embolism (necrosis and amputation of lower phalanges). \\ \hline
**16 (page 666)** & Advanced chronic heart failure with severely depressed ejection fraction. Congenitally corrected transposition \\  & of great vessels. Right lower pneumonia due to SARS-CoV-2/COVID-19. Re-exacerbated chronic kidney \\  & disease. Iron deficiency treated with intravenous administration of iron-carboxymaltose. \\ \hline
**17 (page 760)** & Early endocarditis on mechanical prosthetic aortic valve by Staphylococcus epidermidis. Mitroaortic junction \\  & abscess with extension to tricuspid valve. Severe mitral and tricuspid regurgitation. 2:1 atrioventricular block. \\
**17 (page 760)** & Acute myocardial infarction with inferior ST elevation. Septic coronary embolisms in dominant circumflex and \\  & anterior descending coronary artery. Thromboaspiration and balloon angioplasty over LAD and Cx. Mixed \\  & cardiogenic/septic shock. Emergent surgery for aortic, tricuspid and mitral ring valve replacement. Death. \\ \hline
**18 (page 835)** & Paracardiac mass with pericardial and intramural infiltration, compatible with metastasis of renal carcinoma. \\ \hline
**19 (page 860)** & HF with reduced EF. Initial CF III with clinical and analytical improvement after titration of neuhormonal \\  & medication. Currently NYHA II. Moderate left ventricular systolic dysfunction (LVEF 35\%) of ischaemic \\  & aetiology. Surgically revascularised chronic ischaemic heart disease: IMA-ADA and saphenous bypass to ACx. \\  & Subsequent control coronary angiograms with patency of the mammary graft and occlusion of the saphenous \\
**19 (page 860)** & graft (severe and diffuse disease), for conservative management. ACD thin and diffusely diseased vessel, \\  & discouraged for revascularisation. Metabolic syndrome with type III obesity and poor initial control of \\  & classical CVRFs. Improvement of metabolic control and weight reduction with medical treatment (aGLP1). \\  & Current BMI 35.6 kg/m2 (type II obesity). DM2 better controlled after initiation of iSGLT2 and aGLP1. \\  & Multifactorial chronic CKD with creatinine levels stable at around 1.3-1.5 mg/dl. Ex-smoker. \\ \hline \hline \end{tabular}
\end{table}