# Pulse oximetry as a Diagnostic Tool to Determine Pulp Vitality: A Systematic Review
## 3 Results
### Study Results
Both studies concluded that the pulp maintained its regeneration potential after RT. \begin{table}
\begin{tabular}{c c c c c c c c c} \hline \hline
**Author** & **UI** & **UC** & **UP** & **UM** & **LI** & **LC** & **LP** & **LM** \\ \hline [PERSON] (1996) [11] & 28.95\% (10) & - & - & - & 50.28\% (10) & - & - & - \\ \hline [PERSON] (2008) [17] & 91.29\% (30) & 90.69\% (30) & - & - & - & - & - & - \\ \hline [PERSON] (2014) [32] & UCI: & & & & & & & \\
2.51\% (15) & 85.11\% (15) & 89.55\% & - & - & - & - & - & - \\
2.51\% (10) & 81.25\% & - & - & - & - & - & - & - \\ \hline [PERSON] (2015) [33] & 81.25\% & - & - & - & - & - & - & - \\ \hline [PERSON] (2015) [31] & UCI: 84.4\% & 83.4\% & - & - & - & LCI: 83.5\% & & - & - \\ \hline [PERSON] (2016) [22] & UCI: 83.4\% & 83.4\% & - & - & LCI: 82\% & 84.5\% & - & - \\ \hline  & SG & & & & SG & & & \\  & UCI: & & & & LCI: & & & \\  & 89-98\% & & & & 89-96\% & & & \\  & ULI: & & & & LLI: & & & \\ [PERSON] & 90–98\% & SG: 89–96\% & & & 88–96\% & & & \\ (2016) [22] & CC & CG: & - & - & CG & 90–96\% & & - \\  & UCI: & 89–98\% & & & & 89–96\% & & & \\  & 89–98\% & & & & LLI: & & & \\  & ULI: & & & & 89–97\% & & & & \\  & 90–98\% & & & & 89–97\% & & & & \\ \hline [PERSON] (2017) A [18] & - & - & - & 83.59\% (61) & - & - & - & 86.89\% (51) \\ \hline  & HbSS & & & & HbSS & & & \\  & UCI: 78\% & HbSS: 87\% & HbSS: 75\% & LCI: 86\% & HbSS: 93\% & HbSS: 88\% & HbSS: 89\% \\ [PERSON] (2017) [28] & UL: 79\% & HbAA: & HbAA: & HbAA: & LH: 93\% & HbAA: & HbAA: & HbAA: \\  & HbAA & 86\% & 91\% & 89\% & HbAA: & 90\% & 88\% & 89\% \\  & UCI: 89\% & & & & LCI:83\% & & & \\  & ULI: 87\% & & & & LLI:91\% & & & \\ \hline [PERSON] (2018) [27] & UCI: 84.9\% & - & - & - & - & - & - & - \\ \hline UI: upper incisors; UCI: upper central incisors; UCI: upper earlines; UC: upper earlines; UP: upper premolars; UM: upper molars; LI: lower incisors; LCI: lower central incisors; LLI: lower lateral incisors; LC: lower earlines; LP: lower premolars; LM: lower molars; SC: study group; CG: control group; HbSS: individuals with sickle cell anemia; HbA: individuals with normal hemoglobin A. & & & & \\ \hline \hline \end{tabular}
\end{table} 
Table 4: Oxygen saturation percentages (%SpO2) by tooth type. \begin{table}
\begin{tabular}{c c c c c c} \hline \hline
**Author** & **PC** & **RP** & **IP** & **PN** & **NC** \\ \hline F. [PERSON] (2012) [26] & 92.2\% & 87.4\% & 83.1\% & 74.6\% & 0\% \\ [PERSON]. [PERSON] (2016) [4] & 93\% & 92\% & 89\% & 71\% & 0\% \\ B. [PERSON] (2017) [15] & 94.6\% & 85.45\% & 81.6\% & 70.7\% & 0\% \\ [PERSON]. [PERSON] (2017) [30] & 81.47\% & 93.81\% & - & - & - \\ \hline \hline \end{tabular}  
* PC: positive control; RP: reversible pulpitis; IP: irreversible pulpitis; PN: pulp necrosis; NC: negative control.  
\end{table} 
Table 5: Oxygen saturation percentages (%SpO2) by pulp status. Various studies assessed the use of pulse oximetry to determine pulp vitality in patients with different pathologies. [PERSON] et al. (2014) [16] studied how the periodontal disease affected SpO\({}_{2}\) levels and found out that patients with periodontal disease had fewer levels of SpO\({}_{2}\). SpO\({}_{2}\) in periodontal patients was 76.7%, whereas in healthy patients it was significantly higher (86.7%). In a similar manner, [PERSON] et al. (2017) [28] investigated SpO\({}_{2}\) on patients with homozygous sickle cell anemia, and [PERSON] et al. (2017) [23] compared PO to EPT and cold tests in canines after alveolar cleft bone grafting. In the first study, they concluded that patients with sickle cell anemia had lower levels of SpO\({}_{2}\) than those without it, except in canines. In the second study, even though both sides had adequate blood flow supply, SpO\({}_{2}\) levels were lower on the cleft side. Singularly, [PERSON] et al.