## 4 Discussion  
Approximately 70 per cent of the general population experiences at least one of the symptoms of a temporomandibular disorder (TMD) [30]. Temporomandibular disorders are generally present in early adulthood. TMD can have many causes, among which the most common are occlusal disharmony, mandibular instability, parafunctional habits, stress, and anxiety [31]. Unconscious teeth clenching causes constant trauma to the masticatory region and represents myofascal pain's main etiological factors. The latter is characterised by trigger points, which cause continuous aches and possible central excitatory effects, usually described by patients as headaches [3]. Studies show that the referred pain elicited by active MTrPs reproduces the headache pattern in TTH patients [8]. The TrPs in the muscles are involved in chronic tension-type headaches (CTTH) [32,33]. A recent study evaluated the effects of four different therapies on TMD patients: night splint, betamethasone, sodium hyalurone, or platelet-rich plasma injections. All four groups had improved mouth opening and pain reduction [34]. Another study compared the degree of stretching of the temporal muscles and masseaters through post-isometric \begin{table}
\begin{tabular}{c c c} \hline \hline  & **DMN Average Connectivity:** & **PN Average Connectivity:** \\  & **T2–T1** & **T2–T1** \\ \hline PT 1 (CS) & 15.83 & 1.14 \\ \hline PT 2 (SS) & \(-6.86\) & 2.03 \\ \hline PT 3 (RF) & \(-7.34\) & 20.24 \\ \hline PT 4 (AN) & \(-6.76\) & 13.41 \\ \hline PT 5 (CT) & \(-8.93\) & 0.20 \\ \hline PT 6 (NM) & \(-1.96\) & 31.18 \\ \hline PT 7 (RE) & \(-0.79\) & 17.95 \\ \hline PT 8 (LM) & \(10\) & 2.2 \\ \hline PT 9 (FA) & \(-8.58\) & 21.82 \\ \hline PT 10 (SG) & 5.13 & \(-9.77\) \\ \hline \hline \end{tabular}
\end{table} 
Table 4: Average connectivity of the DMN and PN. relaxation methods and treatments for myofascal relaxation. A decrease in the electrical activity of the muscles examined in both study groups was noted [35]. Myofascali pain is the most frequent TMD found in patients. There is a correlation between TMD and muscle activity and the tone of the chewing muscles. In this study, we report the effect of pathological therapies on pain and the correlated changes in masseter thickness, pain, and the default mode neural network in subjects with myofascali pain in the masticatory region due to unconscious clenching. Parafunctional activities, such as bruxism and teeth clenching, cause continuous microtrauma to the TMJ. This condition stimulates the fibres of the masticatory muscles for a long time, causing pain in the affected areas. The study protocol consisted of three key points concerning clenching and, therefore, myofascali pain: The splint's thickness did not exceed 0.7 mm, because the brain does not recognise this as a foreign body between the teeth; therefore, the patient did not grind on it. One splint was used during the day and another during the night. There were also stretching exercises for paravertebral musculature (mainly through a lat machine) to reduce the unconscious clenching reflex that activates the paravertebral musculature. Bifofeedback exercises for the tongue give the patient an awareness of the spatial positioning of the arches so that they can become aware of the teeth clenching and stop it. The biofeedback technique allows patients to recognise, correct, and prevent the physiological alterations underlying pathological conditions with consequent reduction or elimination. Qualified panels and meta-analyses assessed biofeedback-related approaches to headache treatment. These reviews indicate that various forms of biofeedback were effective for migraine and tension-type headaches [36]. After the treatment, the patients reported significantly decreased symptomatology (Tables 1-3), pain, and detected trigger points (VAS and palpation), and decreased unconscious teeth clenching and muscular tension. Regardless of the type of joint disorder, modifications in the disc-condyle relation (TMJ resonances) were not observed in all patients.