## 3 Results  
None of the participants reported side effects or pain during the treatment sessions, and none of them reported persistent pain after the therapy had finished. The expected duration of the whole therapy cycle was 153 h, while the detected duration recorded from the data readings of stimulator memory in T1 was 144 h. Therefore, it could be concluded that most of the patients maintained the therapy regime (Table 1). The proportional decrease in the stimulus strength in milliamps (28 mA on average) was found in T1. The value of the rectangular stimulus duration that needed to be adjusted for patients, based on the results of single motor unit action potential recordings in sEMG studies, was 17.5 ms on average, which is characteristic of the neurogenic type of muscle motor unit injury.  
The data in Table 2 indicate that the parameters of amplitude more than frequency index of sEMG recordings from all muscles on both sides differed significantly at \(p<0.001\) compared to those recorded in the healthy volunteers, both in T0 as well as in T1. However, the data also indicate a significant improvement of the above parameters in T0 versus T1 in iSCI subjects from the K + E group. The examples of recordings from one of the iSCI patients (Figure 3C) showed that before the application of individually selected parameters of electrostimulation to lower extremities nerves, the sEMG amplitudes and frequencies of motor unit action potential requirements recorded from the TA were lower and then increased following the application of personalized electrotherapy.  
\begin{table}
\begin{tabular}{c c c c c c c c c c c c c c c c c c c} \hline \hline  & \multicolumn{3}{c}{**Control**} & \multicolumn{3}{c}{**K \(\pm\) C Group ON + E2)**} & \multicolumn{3}{c}{**K \(\times\) Group ON + 20**} & \multicolumn{3}{c}{**K \(\times\) E Group ON + E20**} & \multicolumn{3}{c}{**K \(\times\) E Group ON + E20**} \\ \cline{3-14} \multirow{-2}{*}{**Parameter**} & **Slide** & **Min-Max** & **Before (Tt)** & **After (Tt)**  
\begin{table}
\begin{tabular}{c c c c c c c c c c c c c c c c c c c} \hline \hline  & \multicolumn{3}{c}{**Control**} & \multicolumn{3}{c}{**K + E Group (N = E2)**} & \multicolumn{3}{c}{K + E Group (N = E3)} \\  & \multicolumn{3}{c}{**(N = E3)**} & \multicolumn{3}{c}{**(N = E3)**} & \multicolumn{3}{c}{**(N = E3)**} & \multicolumn{3}{c}{**(N = E3)**} & \multicolumn{3}{c}{**(N = E3)**} & \multicolumn{3}{c}{**(N = E3)**} \\ \hline \multirow{2}{*}{**Parameter**} & \multirow{2}{*}{**Side**} & **Min-Max** & **Before (TB)** & **After (TB)** & **TB** & **TB** & **TB** & **TB** & **TB** & **TB** &  
The ENG data in Table 2 show that the amplitudes more than the latencies of M-wave recordings performed in iSCI patients differed at \(p<0.001\) from normative parameters in T0 in both groups of patients. Stimuli strengths necessary to elicit compound muscle-evoked potentials were significantly higher in patients than in healthy volunteers at 55 mA on average. Applied electotherapy treatment provided a significant increase in M-wave-evoked potential amplitude parameters and shortening of the latency value with a simultaneous decrease in stimulus strength necessary to evoke them in T1 (only in patients from the K + E group), as can be seen from the examples of recordings in Figure 3B. All of the above axonal-type changes were more detectable and reversible following stimulations of peroneal than tibial nerves in both groups of patients.  
The data in Table 2 indicate the higher percentage of improvement in parameters of sEMG recordings during maximal contraction in the K + E group rather than in the K group. The neurophysiological parameters recorded in both groups of iSCI patients did not differ significantly before the therapy. However, they were significantly different in the final observation at \(p\) from 0.05 to 0.008.