## 1 Introduction  
Ameloblastic fibroodontoma (AFO) is one of the odontogenic tumors (OT), present in the oral cavity and is considered one or rare lesion in the oral cavity, estimated 0.1-3.4% (0.3-2.3%) in some studies [1, 2]. Since the WHO classification in 1992, some crucial changes in OT studies had changed [1, 2]. AFO is common in children and young adults (one to two decades of life) and it might be related to odontogenesis. In almost all cases it grows slowly without any symptoms or other clinical manifestations and is mostly diagnosed on radiographs [2]. [PERSON] et al. conclude that it is still unclear if AFO is an early stage of odontoma [3, 4]. In both cases, impacted teeth at different stages of eruption might be found [5, 6]. In the old World Health Organisation classification (2005), AFO was classified in the same group as ameloblastic fibroma (AF), amelblastic fibrodentinoma (AFD), and others, characteristic of the presence of odontogenic epithelium with odontogenic ectomesenchyme with or without hard tissue formation [7, 8]. Their shape, size, and advancement might vary greatly. Back in 2017, the new WHO classification changed AFO and AFD (ameloblastic fibrodentinoma) from two separate entities, since they represent the early stages of complex odontoma development (developing odontoma group) [9, 10]. This fact was related to little clinical evidence of AOF, and AFD classifying them as independent entities. Similar changes influenced odontoameloblastoma (OA), since its more characteristic of an odontoma with coexisting ameloblastoma but with clinical signs as ameloblastoma [9; 10]. The new classification divides OT and classifies them into epithelial, mesenchymal (ectomesenchymal), or mixed forms depending on which component of the tooth germ gives rise to the neoplasm [7; 8; 9; 10]. Nowadays AFO is considered a benign mixed odontogenic lesion, most present in the mandible, and less commonly in the maxillary region. This origin is related to the presence of hard odontoid tissue with a great variety in the spectrum of developing odontoma [11; 12; 13]. In most cases, an asymptomatic swelling, followed by delayed teeth eruption with well-defined lesion borders remains the most typical symptom of this pathology [8; 9; 10; 11; 12]. More advanced lesions might rarely displace surrounding teeth and the lesion might exceed the cortical bone layer towards the soft tissues [8; 9; 10; 11; 12; 13; 14; 15]. The inferior alveolar nerve is not involved in the lesion and its function loss is not common, similar to surrounding teeth resorption [8; 9; 10; 11; 12; 13; 14; 15]. Bigger lesions might lead to facial disfigurement, mandibular asymmetry, bone swelling, and problems with biting and chewing. Malignant transformation is not common; however, long-term follow-up is necessary. Problems with tooth eruption might arise, or their displacement. In radiological assessment, the lesion is mostly well-defined, unilocular, radiolucent mass with areas of radiopaque material, and teeth-like structures, with no, erupted teeth in the lesion cavity. Irregularity of size and shape of radiolucent areas might correspond with the growth phase of the lesion. Its well-defined margins with the presence of radiopaque areas might suggest its entity; however, other larger lesions might require a biopsy at first to establish the biology of the tumor before any final surgery will take place. The current classification of AFO describes this lesion as of mixed origins of ameloblastic fibroma and a complex odontoma with ectodermal and mesenchymal components and odontogenic tissue such as enamel, dentin with the common presence of an ectopic impacted/retained teeth [7; 8; 9; 10; 11; 12; 13; 14]. Some correlation with the scope of present different types of dental hard tissues is worth remembering [15; 16]. This mixed benign neoplasm is also built of various dental hard tissues with different calcification stages.