## 1 Introduction  
Obesity in children and adolescents is a public health problem with a multifactorial origin in which eating behavior plays a role [1]. This behavior, although influenced by the environment, can also present as genetic conditioning [2], which can be analyzed through the detection of single nucleotide polymorphisms (SNPs) that are associated with the obese phenotype, mediated by eating behavior [3].  
Numerous studies support the association between SNPs and the risk of developing overweight or obesity. This is explained by the fact that specific genotypes seem to condition attitudes towards food, such as appetite or satiety, speed of intake, frequency of food consumption, or the tendency to experience emotional hunger. With this approach, research groups such as [PERSON] et al. [4], [PERSON] et al. [5], [PERSON] et al. [6], and [PERSON] et al. [7] have associated eating behavior with polygenic risk, quantified in a range between 8 and 32 SNPs.  
Other investigations have related obesity with lower responsiveness to internal satiety signals and greater receptivity to olfactory or gustatory stimuli, which would lead to a higher caloric intake [8]. It has also been shown that an infant's feeding self-regulation arises from genetic-environmental interactions responsible for behaviors such as rapid eating and delayed satiety [8; 9]. [PERSON] et al. [10] investigated the role of eating behavior in genetic susceptibility to obesity, and their thorough literature analysis over the decade of 2010-2020 concluded that emotional and uncontrolled eating behaviors lead to being overweight.  
A pioneering study from the United Kingdom by [PERSON] et al. [11] investigated the association of the SNP rs9939609 genotype of the FTO gene with adiposity and satiety control indicators in children. It proved the usefulness of psychometric scales ([PERSON] et al. of Food and the Child Eating Behavior Questionnaire) as objective measures of eating behavior. This is also emphasized by [PERSON]'s literature review [10], in which the authors highlight the value of psychometric tests that objectively measure attitudes toward food.  
Undoubtedly, the development of eating habits is a process with a complex etiology, including psychological aspects, particularly the influence of family, social, and affective factors. Some research has highlighted a particular circumstance, reporting that in overweight subjects, there is a positive relationship between an emotional eating pattern and body mass index [12]. Hence, food is eaten to satisfy physiological hunger, to fill an inner emptiness, or to comfort the mood.  
Determining the interactions between genotype and eating behavior in greater depth is particularly interesting in childhood and adolescence because it is at these stages when most future habits are shaped. This is a window of opportunity for prevention, and eventually, for an individualized nutritional approach. Therefore, this study aims to analyze the eating behavior of a sample of Madrid schoolchildren concerning their genetic predisposition to obesity through the conduction of the Children's Eating Behavior Questionnaire (CEBQ), in combination with a genotyping assessment from a panel of 32 SNPs.