## 4 Discussion
\begin{table}
\begin{tabular}{c c c c c c} \hline (wt \%) & **O** & **Ca** & **P** & **Si** & **Ca/P Ratio** \\ \hline \multicolumn{6}{c}{Implant/Scaffold} \\ \hline
15 days & 44.73 \(\pm\) 0.12 (44.73) & 35.01 \(\pm\) 0.25 (32.01) & 20.25 \(\pm\) 0.46 (20.25) & 0.11 \(\pm\) 1.62 (0.11) & 1.74 \(\pm\) 0.86 (1.74) \\
30 days & 45.43 \(\pm\) 0.13 (45.43) & 32.12 \(\pm\) 0.24 (32.14) & 22.36 \(\pm\) 0.43 (22.36) & 0.09 \(\pm\) 1.83 (0.09) & 1.44 \(\pm\) 0.84 (1.07) \\
60 days & 48.47 \(\pm\) 0.12 (48.47) & 29.62 \(\pm\) 0.26 (29.62) & 21.84 \(\pm\) 0.35 (21.84) & 0.07 \(\pm\) 1.82 (0.07) & 1.36 \(\pm\) 0.76 (1.36) \\ \hline \multicolumn{6}{c}{Bone Interface} \\ \hline
15 days & 50.68 \(\pm\) 0.11 (50.68) & 32.95 \(\pm\) 0.31 (32.95) & 16.32 \(\pm\) 0.53 (16.32) & 0.05 \(\pm\) 1.63 (0.05) & 2.02 \(\pm\) 0.3 (0.2) \\
30 days & 55.88 \(\pm\) 0.10 (55.88) & 30.23 \(\pm\) 0.29 (30.23) & 13.85 \(\pm\) 0.54 (13.85) & 0.04 \(\pm\) 1.33 (0.04) & \(\pm\) 0.84 (1.04) \\
60 days & 59.15 \(\pm\) 0.12 (59.15) & 28.62 \(\pm\) 0.32 (28.62) & 12.21 \(\pm\) 0.75 (12.21) & 0.02 \(\pm\) 1.25 (0.02) & 2.34 \(\pm\) 0.96 (2.3) \\ \hline \multicolumn{6}{c}{New bone} \\ \hline
15 days & 59.69 \(\pm\) 0.15 (59.69) & 26.78 \(\pm\) 0.16 (26.78) & 13.5 \(\pm\) 0.62 (13.50) & 0.02 \(\pm\) 1.63 (0.02) & 1.98 \(\pm\) 0.80 (1.98) \\
30 days & 60.56 \(\pm\) 0.09 (60.56) & 25.42 \(\pm\) 0.24 (25.42) & 14.01 \(\pm\) 0.53 (14.01) & 0.01 \(\pm\) 1.80 (0.01) & 67 \(\pm\) 0.91 (1.81) \\
60 days & 63.38 \(\pm\) 0.09 (63.38) & 24.36 \(\pm\) 0.15 (24.36) & 12.25 \(\pm\) 0.74 (12.25) & 0.01 \(\pm\) 0.73 (0.01) & \(\pm\) 0.97 (1.91) \\ \hline \end{tabular}
\end{table} 
Table 2: The EDS elemental analysis of the reaction zone at 15, 30 and 60 implantation days. Mean \(\pm\) SD (median). (0.34 A). Therefore, the Si-O bond length (0.161) was longer than that of the P-O bond (0.155) and the ionic radius of the phosphate group (PO\({}_{4}\)[3\({}^{-}\)) was shorter than that of the silicon group (SiO\({}_{4}\)[4\({}^{-}\)) [48; 49; 50; 51]]. This may diminish the stability of calcium phosphates, thus enhancing their solubility, and may explain the greater degradability observed for the Si-\(\alpha\)TCP scaffolds. The histomorphometric results of the present study obtained a value of 60.11% for the Si-\(\alpha\)TCP-treated bone defect, which was filled by newly formed bone by 60 healing days. New L. 1996 growth was located in the vicinity of the implant ceramic particles and within the scaffold. This is possibly owing to the scaffold's open porosity (76%) and crystallinity. High porosity \(\psi\) is seen to facilitate the resorption process as the pores' external and external surface areas were expo, 't to the medium, which brought about an increase in the calcium and phosphorous ion rel - 59.0 (31.57) and 40.52 (0.87 (14.10), respectively--into the intercellular medium for sevfal microns by the scaffold body. The new bone ingrowths in the implant were more evident at 30 and 60 day adds the precess further entered the implant, they advanced into the spaces between the implant's closed scaffold particles to form a characteristic interlocking pattern at the interface. The SEM (Fig. 5) showed massive bone colonization of the implant through the original scaffold \(\psi\) is caused by the structure's gradual dissolution. These advanced processes implied that the scaffold material's free particles were detected in many areas across the restructuring in the implant. The fact that the densities inside the material and at the bone-ceramic interface significatty and gradually redu, do implied that the restorative process not only went from the peripheral to the center, but were initiated in an early material implantation stage by a cellular mechanism [1; 30].