## 1 Introduction  
After performing dental extractions, dry socket is one of the most frequently encountered postoperative complications [1]. Dry socket is also referred to as necrotic socket, fibrinolytic socket, alveolar osteeitis (AO), alveolitis, sicca dolorosa, and localized osteeitis [2]. Although dry socket can be encountered in any tooth, it is associated more frequently after mandibular third molar extraction, being the extraction that is essential to the beginning of dry socket [3]. Dry socket develops as a postoperative complication when a blood clot fails to develop, dissolves, or dislodges at the site of the extraction before the socket is healed [4]. Clinically, a dry socket is visible as part or all of the bone around the occlusal perimeter or within the socket exposed a few days after the extraction. The incidence of post-extraction complications overall is 8.4% for the mandibular teeth; among them, 4.2% are reported to be dry socket, while in the maxilla, 5.9% of complications are reported overall, 0.4% of which being dry socket [5, 6]. The incidence of dry socket varies between 1% and 5% of all dental extractions, with up to 38% of dry socket cases associated with mandibular third molars [7, 8]. Studies report that insufficient blood supply is one of the primary reasons for site-specificity of development of dry socket; however, increased bone density, excessive forces during extraction, and decreased capacity of producing granulation tissue are responsible factors too [9, 10, 11]. Moreover, risk factors for increasing the probability of developing dry socket include use of oral contraceptives, bacterial causes, infection, previous history of experiencing dry socket, and smoking [12]. Factors such as age, sex, medical history, extraction site, amount of anesthesia, and operator experience have no effect on the observation of dry socket [13]. The socket that has been left exposed might be accumulated with food debris and bacterial biofilms that further hinder the healing process of the socket. Moreover, bacteria collected inside the socket may even ferment the food particles inside it, which in turn exacerbates the already compromised situation of the socket. Most commonly, patients suspected of suffering from dry socket present with acute pain from the exposed socket that lingers for many days until the socket is completely covered by the epithelium. Before management, clinicians focus on preventing the development of dry sockets with the use of chlorhexidine mouthwash preoperatively, use of antibiotics such as penicillins, clindamycin, metronidazolezole, and erythromycin, antifibrinolytes such as traxanemic acid, and antimicrobial photodynamic therapy (ADPT) [14]. Management of dry sockets has varied among clinicians, depending on their expertise and experience with dealing with dry sockets. Traditionally, the treatment of dry socket has been irrigation, surgical intervention, and placement of medicated dressings. Irrigation is performed which removes the debris, sequestra, and bacteria from the exposed bone surface of the dry socket. Most commonly, the irrigation solutions used in the dry socket are chlorhexidine gluconate and normal saline [15]. Medicated dressings are then placed in the socket, which promotes healing and reduces patient symptoms. Commonly applied dressings include Alvogyl and zinc oxide eugenol (ZOE) after irrigation of the socket. One study reports Alvogyl to be more effective than ZOE by providing a short time of complete pain relief, fewer visits needed to change the dressings, and, lastly, faster healing of the socket [16]. Natural medicines have been used for many decades, as they tend to have few to no side effects as compared to pharmaceutically produced drugs. Honey is one such product that has previously been used to treat many infections, as its use has been found to make the infectious wound sterile in 3-6 days [17]. One of the active ingredients of honey is propolisis.