gal1: :ret1 cells showed a very similar, if slightly broader peak of coatomer in fractions 17 and 18, in which all subunits including ϵ‐cop co‐fractionated (figure 4b).
In GAL1: :RET1 cells, excess monomeric α‐COP is proteolytically degraded and such α‐COP fragments bind to ϵ‐COP.