title: Place attachment of Ngāi Te Ahi to Hairini Marae

authors: Teddy, Lani

subjects: Hairini

abstract: Twelve members of Ngāi Te Ahi, a hapū from Tauranga, were interviewed to explore how they talk about their place attachment to Hairini Marae.  This was organized around five key dimensions of place attachment taken from the literature—continuity, distinctiveness, symbolism, attachment and familiarity.  We found that in discussing all dimensions, place attachment was equally about social and cultural relationships, history, and socialization.  Place attachment for groups such as Māori is complex because it encompasses all social relationships past and present.  The implication for those working with Māori is to take seriously the wider connotations of place when talking to Māori about marae, traditional homelands, and their land.

text: Place attachment of Ngāi Te Ahi to Hairini Marae Twelve members of Ngāi Te Ahi, a hapū from Tauranga, were interviewed to explore how they talk about their place attachment to Hairini Marae.  This was organized around five key dimensions of place attachment taken from the literature—continuity, distinctiveness, symbolism, attachment and familiarity.  We found that in discussing all dimensions, place attachment was equally about social and cultural relationships, history, and socialization.  Place attachment for groups such as Māori is complex because it encompasses all social relationships past and present.  The implication for those working with Māori is to take seriously the wider connotations of place when talking to Māori about marae, traditional homelands, and their land.

year: 2008

title: The physical health and premature mortality of Indigenous Māori following first-episode psychosis diagnosis: A 15-year follow-up study

authors: Monk, Nathan

subjects: First-episode psychosis, New Zealand, Māori, indigenous health, health equity, physical health, cardiometabolic health, premature mortality, cohort study

abstract: Background:
People experiencing psychosis are at greater risk of physical health conditions and premature mortality. It is likely that Indigenous Māori youth, who experience additional systemic inequities caused by settler-colonisation, face even greater physical health and mortality risks following a diagnosis of first-episode psychosis.
Objective:
Compare Māori and non-Māori for risk of hospitalisation and mortality for up to 15 years following first-episode psychosis diagnosis.
Methods:
A cohort (N = 14,122) of young people (16–24 years) with first-episode psychosis diagnosis between 2001 and 2019 were identified. Using crude Kaplan–Meier and adjusted Cox proportional hazards models, Māori (n = 5211) and non-Māori (n = 8911) were compared on hospitalisation and mortality outcomes for up to 15 years.
Results:
In the 15 years following first-episode psychosis diagnosis, Māori had higher adjusted risk of all-cause mortality (hazard ratio = 1.21, 95% confidence interval = [1.01, 1.45]), hospitalisation with diabetes (hazard ratio = 1.44, 95% confidence interval = [1.15, 1.79]), injury/poisoning (hazard ratio = 1.11, 95% confidence interval = [1.05, 1.16]), general physical health conditions (hazard ratio = 1.07, 95% confidence interval = [1.02, 1.13]) and also appeared to be at greater risk of cardiovascular hospitalisations (hazard ratio = 1.34, 95% confidence interval = [0.97, 1.86]). Kaplan–Meier plots show hospitalisation and mortality inequities emerging approximately 4–7 years following first-episode psychosis diagnosis.
Conclusions:
Māori are at greater risk for hospitalisation and premature mortality outcomes following first-episode psychosis. Early screening and intervention, facilitated by culturally safe health service delivery, is needed to target these inequities early.

text: The physical health and premature mortality of Indigenous Māori following first-episode psychosis diagnosis: A 15-year follow-up study Background:
People experiencing psychosis are at greater risk of physical health conditions and premature mortality. It is likely that Indigenous Māori youth, who experience additional systemic inequities caused by settler-colonisation, face even greater physical health and mortality risks following a diagnosis of first-episode psychosis.
Objective:
Compare Māori and non-Māori for risk of hospitalisation and mortality for up to 15 years following first-episode psychosis diagnosis.
Methods:
A cohort (N = 14,122) of young people (16–24 years) with first-episode psychosis diagnosis between 2001 and 2019 were identified. Using crude Kaplan–Meier and adjusted Cox proportional hazards models, Māori (n = 5211) and non-Māori (n = 8911) were compared on hospitalisation and mortality outcomes for up to 15 years.
Results:
In the 15 years following first-episode psychosis diagnosis, Māori had higher adjusted risk of all-cause mortality (hazard ratio = 1.21, 95% confidence interval = [1.01, 1.45]), hospitalisation with diabetes (hazard ratio = 1.44, 95% confidence interval = [1.15, 1.79]), injury/poisoning (hazard ratio = 1.11, 95% confidence interval = [1.05, 1.16]), general physical health conditions (hazard ratio = 1.07, 95% confidence interval = [1.02, 1.13]) and also appeared to be at greater risk of cardiovascular hospitalisations (hazard ratio = 1.34, 95% confidence interval = [0.97, 1.86]). Kaplan–Meier plots show hospitalisation and mortality inequities emerging approximately 4–7 years following first-episode psychosis diagnosis.
Conclusions:
Māori are at greater risk for hospitalisation and premature mortality outcomes following first-episode psychosis. Early screening and intervention, facilitated by culturally safe health service delivery, is needed to target these inequities early.

year: 2024

title: Morphodynamics of the Whitianga Tidal Inlet and Buffalo Bay, New Zealand

authors: Steeghs, Lauren

subjects: Eddy formation

abstract: The primary aim of this study was to investigate the sedimentation processes within Buffalo Bay, particularly within and adjacent to the Whitianga tidal inlet, in order to ascertain reasons for the shoaling at both the inlet, and the identified shallow zone around Pandora Rock. 

Comparison of historic bathymetries suggests the ebb delta and ebb discharge channel of the Whitianga tidal inlet are rapidly accreting and the ebb tidal discharge channel is gradually migrating northeast towards Whakapenui Point.  Accretion rates of up to 25 cm y-1 were calculated in the ebb delta and inlet discharge channel area between 1979 and 1995 and aerial photo comparisons suggest the ebb delta area had increased by 400 % between 1990 and 2002.  Results of the hydrodynamic and sediment transport modelling suggest the rapid accretion in the ebb delta vicinity is likely to be caused by a combination of catchment estuary inputs, which are deposited on the ebb tide as the ebb flow decelerates over the ebb delta, and inputs that have been moved south along Buffalo Beach by flood currents and an eddy that forms landward of the ebb tidal discharge.  Residual tidal velocities further suggest a deposition zone in the ebb delta vicinity resulting from opposing currents and the deceleration of currents.  

Hydrodynamic modelling results indicate the isolated shallow zone around Pandora rock appears to be caused by a transient eddy in the southern section of Buffalo Bay.  The eddy is formed by the ebb tidal discharge from the inlet.  Accretion probably occurs in the centre of the eddy which moves north as the ebb tide progresses.  

Results obtained from a current meter and sediment trap deployed in northern Buffalo Bay suggest suspended sediment transport is minimal in northern Buffalo Bay, only occurring with large wave activity.   Results of the hydrodynamic and sediment transport modelling further demonstrate that this area experiences low flow velocities, and has little interaction with the rest of Buffalo Bay.  The minimal sediment input to this area, combined with the occasional erosion of the seafloor, primarily by wave activity, is thought to have resulted in long term erosion of northwestern Buffalo Bay between 1938 and 1979.  Although the beach and nearshore is eroding, it is likely the addition of sediment would act to stabilise this section of eroding beach.  Renourishment material could be provided by the ebb delta, the southern tip of Buffalo Bay or the isolated sandbar northeast of the inlet entrance.

text: Morphodynamics of the Whitianga Tidal Inlet and Buffalo Bay, New Zealand The primary aim of this study was to investigate the sedimentation processes within Buffalo Bay, particularly within and adjacent to the Whitianga tidal inlet, in order to ascertain reasons for the shoaling at both the inlet, and the identified shallow zone around Pandora Rock. 

Comparison of historic bathymetries suggests the ebb delta and ebb discharge channel of the Whitianga tidal inlet are rapidly accreting and the ebb tidal discharge channel is gradually migrating northeast towards Whakapenui Point.  Accretion rates of up to 25 cm y-1 were calculated in the ebb delta and inlet discharge channel area between 1979 and 1995 and aerial photo comparisons suggest the ebb delta area had increased by 400 % between 1990 and 2002.  Results of the hydrodynamic and sediment transport modelling suggest the rapid accretion in the ebb delta vicinity is likely to be caused by a combination of catchment estuary inputs, which are deposited on the ebb tide as the ebb flow decelerates over the ebb delta, and inputs that have been moved south along Buffalo Beach by flood currents and an eddy that forms landward of the ebb tidal discharge.  Residual tidal velocities further suggest a deposition zone in the ebb delta vicinity resulting from opposing currents and the deceleration of currents.  

Hydrodynamic modelling results indicate the isolated shallow zone around Pandora rock appears to be caused by a transient eddy in the southern section of Buffalo Bay.  The eddy is formed by the ebb tidal discharge from the inlet.  Accretion probably occurs in the centre of the eddy which moves north as the ebb tide progresses.  

Results obtained from a current meter and sediment trap deployed in northern Buffalo Bay suggest suspended sediment transport is minimal in northern Buffalo Bay, only occurring with large wave activity.   Results of the hydrodynamic and sediment transport modelling further demonstrate that this area experiences low flow velocities, and has little interaction with the rest of Buffalo Bay.  The minimal sediment input to this area, combined with the occasional erosion of the seafloor, primarily by wave activity, is thought to have resulted in long term erosion of northwestern Buffalo Bay between 1938 and 1979.  Although the beach and nearshore is eroding, it is likely the addition of sediment would act to stabilise this section of eroding beach.  Renourishment material could be provided by the ebb delta, the southern tip of Buffalo Bay or the isolated sandbar northeast of the inlet entrance.

year: 2007