Fisherman & farmer. [volume] (Edenton, N.C.) 1887-19??, 1901-09-12

nor infectious. 1t is likely the
cause is connected with the food
either developed in it through
some Preventative process, or
upon it, in the form ot one OF the
many parasitic fungi which may
be found on plants and grain.
These, when consumed at Ger
tain stages Of development make
a poisonous impression upon the
brain and ultimately, induce
structural changes. THIS is shown
by the history Of the outbreaks.
Feeding upon hay, oats In the
straw, new fodder, grazing all
night, together with bad santa,
conditions about stables are all
causes which help to produce
the trouble.
The symptoms may appear
gradually and this is, by far, the
least fatal form = The horse at
first will appear dull and off his
feed; appear weak about the
loins; staggering gait, with diff
Guilty i'll swallowing = and i'll
some cases total inability. Some
times spasms ensue, followed by
paralysis of the whole body.
In other cases there is delirium
indicated by the animals going
through a series Of automatic
movements, as if running or trot
ting. The delirium may take vio-
lent form and the animal bruise
himself seriously, Ill his strug~
giles, but usually deep coma rem
ders him quiet until he expires.
In violent cases, death usually
occurs in from four, to twenty>
four hours, after attack IS first
noticed.
The pulse is variab'e. notice
that they are below normal until
the disease progresses then they
work their way up slowly until
they become imperceptible. The
pulse in the beginning of she
disease helps very little IN dag
noslng the case, veIy little fever
at first, no pain. Respiration
Igenerally quick and catchy.
In the next form in which it
may develop it fist becomes
manifest by difficulty in. swal-
lowing and siowuess in mastica-
ton and weakness which max
be first noticed in the strength,
of the tail, the animal seems to
be unable to- switch it or to Otter
resistance. There is no evitle nee
of pain. The respirations are un-
changed, the temperature less
than normal, the bowels consti>
| pated, sometimes | SID trouble
IN moving them, These symp-
| toms may rcr22I1r unchanged
for several DAYS, then gradually
get better or tha power to swal-
low may Become entirely lost
and the weakness and uucertain-
ty IN gait become more perceptl.
BILE, then sleepiness or coma
may appear, the pulse becomes
depressed, slow and weak, the
breathing sterto,ous ana parox~
ilons of delirium develop. witll
inability to stand and some rig>
ldltlY of the spinal. muscles OF
partial cranp Of the neck end
Jaws. In such cases death ,mav
occur III from six. to ten abavs,
rom the commencement Of tile
attack. II, many cases there Is
no evidence: of pain, spasrn OF
fever at any time during thS
progress OF the disease and final,
! ~ profound coma develops and
] death follows, painless and with
out struggle.
| ID the last or sniidest forEa the
ability to swallow is never en-
tlrely lost. Generally the ANNA
, will begin to improve about the
fourth Or fifth day and recover
In all cases where COME remains
iabsent for six or seven days
there iSa good chance for re
covery. There is great DANCE:
III local paralysis, which vii
take tlrRe to subsiGe.
PATHOLOGY.
The poison having becom,
absorbed. acts upon the nerv,
centres i'll three ways:
Ist. BY shock Or collapse, from
the extreme amount of virus alb
> sorbed.
end. More slowly by bloc
poisoning.
; 3rd. FROM a mal-nututive {
parts, consequent upon derar
yed nervous function.
TREATMENT.
In the worst class of case!
treatment is very seldom success
full. In the other two there is
: chance for recovery, if treate
: correctly, by a man that has ha
soue experience with the d.
taSe. This is what Morris til
Veterinary Surgeon has to sa