The Cecil Whig. [volume] (Elkton, Md.) 1841-current, 1884-05-31
ACUTE CHAMBERS.
TO de Editor IF The Cecil Wig:

10 de Editor IF The Cecil Wig:
The acute form of CHAMBERS, being pre-
ceeded by increased temperature, appears
suddenly. The temperature varies from
101 TO 104 ID degrees, Fahrenheit, for Il
week previous to the appearance of the
first symptoms of the disease, the ami
NAL appearing well all this time. When
the more violent symptoms appear they
are recognized by very severe chills of a
persistent character. loss of appetite, a
slight BLUSH white discharge issuing
from one nostril-gencrally the left nos
tril-pulse free and numbering A. to
Go per minute, the reSpirations increased
to - per minute. Sometimes the disease
is ushered in more violently, the chill
continuing for As hours without in
Permission, followed by a sanguineous dis
charge from the nostrils, the visible mu-
cous membrane of the nose violently in
famed, and in some instances small
ulcers are formed. Commonly the chills
are not so persistent, but they are always
observable. TIE sub-maxillary lynn.
phatic glands enlarge; at first they are
soft, but soon become hard and indu-
rated and adhere to the internal aspect
of the sub maxillary Gaw) bone.
Other Emphatic glands enlarge, and
may be seen on the sides Of the neck and
chest, and also on the back, in the form
of round white Jumps, hard and rolling
to the touch. Sometimes these nodules
become inilamed, suppuratc, burst and
expose raw, unhealthy looking sores,
from which runs more or less ichorous
and irritating purulent discharge. The
eyes are weak, ard have thin white
discharge issuing from them. When
the discharge is great the noStrils be-
come swollen, ard the accumulation OF
the glutuious discharge on them impedcs
the act OF breathing. Abcesscs some.
times form along the course of the lynn.
phaticS of the face.
This form of CHAMBERS is rapidly fatal
1t runs its course rapidly, and is very
easily communicated to man. As in tho
clse where acute and rapid inilammations
have pervaded the system, ard especially
when such are the iutllmmlti0ns accom-
panying the development of blood dis
ease, the post mortem LESSONS are very
great. showing rapid destruction and
breaking down of the tissues wherever
the diseased virus has been deposited
The turbinated, nasal and cthmoid
bones Clones of the head are III a state
of necrosis and their surfaces are Gov.
cred with the glardcrous matter. The
lungs are always more or less iutlamcG
in every case of CHAMBERS. Sometimes
one lung OF both lungs: OF perhaps but
one lobulc of a lung is affected, when il
presents the appearance of small tumours
Many of these tumCurs contain pus
hence they have been called tuberclcs
and from this suppositiOn, the nature o
the disease has been termed ''tuber.
culosis." Many writers think that tuber-
cIlosis can be generated by tie depCs;-
ton of glandeTous matter in the lobes OF
the lung. Whether the disease has its
seat in the head or in the lungs, will not
presume to decide, but perhaps The
opinions of our most celebrated patholo
gists on this point may be interesting.
Nirchow maintains that the head is the
seat Of the disease, and that the deposits
in the lungs are only secondary LESSONS
Philippe and Bouley are convinced
that the disease always commences in the
lungs and that the head is only affected
secondalily. These last two observers
claim the strong analogy of CHAMBERS an(
tuberculosis is proof sufficient that they
are but different forms of the one disease
They say 1.-THE g:andular swellings az
exactly the same; the mode of invasion
is likewise the same: now acute, aud de