cyanic-selkie/wikianc huggingface dataset:
Ciwon Kwayoyin Halitta
Multifactorial cuta
Cututtukan kwayoyin halitta na iya zama hadaddun, multifactorial, ko polygenic, ma'ana ana iya danganta su da tasirin kwayoyin halitta da yawa a hade tare da salon rayuwa da abubuwan muhalli. Cututtuka masu yawa sun haɗa da cututtukan zuciya da ciwon sukari. Kodayake rikice-rikice masu rikitarwa sukan taru a cikin iyalai, ba su da tsayayyen tsarin gado. Wannan yana da wahala a iya tantance haɗarin da mutum ke da shi na gado ko kuma kamuwa da wannan cuta. Cututtuka masu rikitarwa suma suna da wahalar nazari da kuma magance su domin har yanzu ba a gano takamaiman abubuwan da ke haifar da mafi yawan wadannan cututtuka ba. Nazarin da ke nufin gano dalilin rikice-rikice masu rikitarwa na iya amfani da hanyoyi da yawa don ƙayyade ƙungiyoyin genotype - phenotype. Hanya ɗaya, tsarin genotype-farko, yana farawa ta hanyar gano bambance-bambancen kwayoyin halitta a cikin marasa lafiya sannan kuma ƙayyade alamun bayyanar cututtuka. Wannan ya saba wa mafi al'ada phenotype-farko hanya, kuma zai iya gano dalilan haddasawa da a baya an rufe su da asibiti iri -iri, shiga, da kuma bayyanawa.
A kan ƙayyadaddun ƙayyadaddun ƙayyadaddun cututtuka, cututtuka na polygenic sun kasance suna "gudu a cikin iyalai", amma gadon bai dace da tsari mai sauƙi ba kamar cututtukan Mendelian. Wannan baya nufin cewa kwayoyin halitta ba za a iya gano su a ƙarshe kuma su yi nazari ba. Hakanan akwai wani bangaren muhalli mai ƙarfi ga yawancin su (misali, hawan jini ). Sauran abubuwan sun haɗa da:
Cutar chromosomal
Rashin lafiyar chromosomal wani yanki ne na chromosomal ɓatacce, ƙari, ko mara daidaituwa na DNA. Yana iya kasancewa daga adadi mai ƙima na chromosomes ko rashin daidaituwar tsari a cikin ɗaya ko fiye da chromosomes. Misalin waɗannan cututtuka shine trisomy 21 ( Down syndrome ), wanda a ciki akwai ƙarin kwafin chromosome 21.
Bincike
Saboda nau'ikan cututtukan ƙwayoyin cuta da aka sani, ganewar asali ya bambanta kuma ya dogara da cutar. Yawancin cututtukan ƙwayoyin cuta ana gano su kafin haihuwa, a lokacin haihuwa, ko kuma lokacin ƙuruciyar yara duk da haka wasu, irin su cutar Huntington, na iya tserewa ganowa har sai majiyyaci ya fara girma.
Abubuwan asali na rashin lafiyar kwayoyin halitta sun dogara ne akan gadon kayan gado. Tare da zurfin tarihin iyali, yana yiwuwa a hango yiwuwar rashin lafiya a cikin yara wanda ke jagorantar ƙwararrun likitocin zuwa takamaiman gwaje-gwaje dangane da rashin lafiya da ba da damar iyaye su shirya don canje-canjen salon rayuwa, tsammanin yiwuwar haihuwa, ko tunanin ƙarewa. Ciwon ciki na iya gano alamun rashin daidaituwa a cikin ci gaban tayin ta hanyar duban dan tayi, ko gano kasancewar sifofin halayen ta hanyoyin ɓarke waɗanda suka haɗa da shigar da bincike ko allura a cikin mahaifa kamar na amniocentesis.
Hasashen
Ba duk cututtukan kwayoyin halitta ba ne kai tsaye ke haifar da mutuwa; duk da haka, babu wasu sanannun magunguna na cututtukan ƙwayoyin cuta. Yawancin cututtuka na kwayoyin halitta suna shafar matakan ci gaba, irin su Down syndrome, yayin da wasu ke haifar da bayyanar cututtuka na jiki kawai kamar dystrophy na muscular. Sauran cututtuka, irin su cutar Huntington, ba su nuna alamun ba har sai sun girma. A lokacin aiki na rashin lafiyar kwayoyin halitta, marasa lafiya galibi suna dogara ne akan kiyayewa ko rage jinkirin lalacewar ingancin rayuwa da kiyaye ikon kai na haƙuri. Wannan ya haɗa da jiyya na jiki, kula da ciwo, kuma yana iya haɗawa da zaɓi na madadin shirye-shiryen magani.
Magani
Maganin cututtukan ƙwayoyin cuta yaƙi ne da ke gudana, tare da fiye da 1,800 na gwajin asibiti da aka kammala, ana ci gaba, ko kuma an amince da su a duk duniya. Duk da haka, yawancin zaɓuɓɓukan magani sun haɗa da magance alamun rashin lafiya a ƙoƙarin inganta rayuwar marasa lafiya.
Maganin kwayoyin halitta yana nufin wani nau'i na magani inda aka gabatar da kwayar halitta mai lafiya ga majiyyaci. Wannan ya kamata ya rage lahani da ke haifar da kuskuren ƙwayar cuta ko rage ci gaban cutar. Babban cikas shine isar da kwayoyin halitta zuwa ga tantanin halitta, nama, da gabobin da cutar ta shafa. Masu bincike sun bincika yadda za su iya shigar da kwayar halitta a cikin yuwuwar triliyoyin sel waɗanda ke ɗauke da gurɓataccen kwafin. Samun amsar wannan ya kasance shingen hanya tsakanin fahimtar cutar kwayar halitta da kuma gyara matsalar kwayoyin halitta.
Epidemiology
Kusan 1 a cikin 50 mutane suna fama da sanannen rashin lafiya-kwayoyin halitta, yayin da kusan 1 cikin 263 ke fama da cutar chromosomal. Kusan kashi 65% na mutane suna da wata irin matsalar lafiya sakamakon maye gurbi na haihuwa. Saboda babban adadin cututtukan kwayoyin halitta, kusan 1 cikin mutane 21 suna fama da cutar ta kwayoyin halitta da aka rarraba a matsayin " rauni " (wanda aka fi sani da yana shafar ƙasa da 1 cikin mutane 2,000). Yawancin cututtuka na kwayoyin halitta ba su da yawa a kansu. Akwai sanannun cututtukan ƙwayoyin cuta sama da 6,000, kuma ana bayyana sabbin cututtukan ƙwayoyin cuta koyaushe a cikin littattafan likitanci.
Tarihi
Sanannen yanayin kwayoyin halitta na farko a cikin hominid yana cikin nau'in burbushin halittu Paranthropus robustus , tare da sama da kashi uku na mutane suna nuna amelogenesis imperfecta.
Kwalejin Ilimi da Nazarin Shari'a ta Jihar Jigawa
Kwalejin Ilimi da Nazarin Shari'a ta Jihar Jigawa babbar cibiyar ilimi ce ta gwamnatin jihar da ke a Ringim, Jihar Jigawa, Najeriya. Shugaban ta na yanzu shine Abbas A. Abbas.
Tarihi
An kafa Kwalejin Ilimi da Nazarin Shari’a ta Jihar Jigawa a shekarar 1992.
Saminu Ɗorayi
Malam Saminu Ibrahim Ɗorayi wanda akafi sani da Malam Saminu Ɗorayi an haife shi ne a birnin Kano a shekarar 1940. shi ne babban limamin masallacin Ɗorayi Babba a Jihar Kano da ke arewa maso yammacin Najeriya.
Gidan Kayan Tarihi Na Birnin Benin
Gidan tarihi na birnin Benin babban gidan kayan tarihi ne na kasa a cikin birnin Benin, Najeriya. dake tsakiyar birnin kan dandalin Sarki. Gidan kayan tarihin yana da adadi mai yawa na kayan tarihi masu alaƙa da daular Benin kamar terracotta, sifofin tagulla da simintin ƙarfe. Har ila yau, yana da tsohuwar fasaha da ke da alaƙa da zamanin farko.
Saminu Ɗorayi
Farkon Rayuwa
Malam Saminu an haife shi a Kano a shekarar 1940. Ya rayu tare da mahaifinsa (wato Malam Ibrahim Ɗorayi babba) ya kuma fara karatu a wajen mahaifinsa har ya sauke Al-Ƙur'ani mai tsarki kafin rasuwar mahaifinsa. Mahaifinsa ya rasu a shekara ta 1959. Bayan rasuwar mahaifinsa ya koma karatu wurin almajiran mahaifin sa inda ya koyi Ilimin fikihu da Ilimin tafsiri dana Hadisi.
Gidan Kayan Tarihi Na Birnin Benin
Wuri
Gidan tarihi na birnin Benin yana wani wuri da ake kira Ring Road wanda a da mutanen Benin ke kiransa da dandalin Sarki, amma Kwamared Adams Oshiomhole ya canza shi zuwa dandalin Oba Ovonramwen a lokacin yana gwamnan jihar.
Saminu Ɗorayi
Karatun Boko
Bayan wasu lokuta malam yaga cewar bayajin dadin irin kallon da akeyiwa malaman soro wato ana musu kallon kamar su ba komai bane. Wannan tasa ya shiga SAS (kwalejin harshen Larabci). Bayan ya gama kuma ya shiga Jami'ar Bayero, inda a nan ya yi difiloma da digiri baki daya.
Rele Art Gallery
Rele Gallery wuri ne na fasaha na zamani da kuma zane a cikin Legas, Najeriya. Yana kan titin Military, Onikan Legas da kuma Victoria Island, Legas.
Tarihi
Gidan hoton wani yanki ne na Rele Arts Foundation, wacce kungiya ce mai zaman kanta.
A cikin shekarar 2021, Gallery ɗin ya buɗe wani wuri a Los Angeles, California. Ita ce taswirar Afirka ta farko da aka buɗe a Los Angeles.
Artists
Rele Gallery ya fito da wasu mawakan duniya na Najeriya da suka hada da Victor Ehikhamenor da Kelechi Amadi-Obi.
Okaukuejo Airport
Filin jirgin sama na Okaukuejo ( filin jirgin sama ne a yankin Oshana na Namibiya, wanda ke hidima ga wuraren shakatawa na Etosha da cibiyar gudanarwa, Okaukuejo.Titin jirgin sama 3 km arewacin kauye.
Shire of Calliope
Shere na kalliofe karamar hukuma ce da ke yankin kapricornia na Queensland, Ostiraliya. Ya kasance a tsakiyar garin kalliope.
Tarihi
An ƙirƙiri Ƙungiyar kalliope a ranar 11 ga watan Nuwamba 1879 a matsayin ɗaya daga cikin rarrabuwa 74 a kusa da Queensland a ƙarƙashin Dokar Alƙalai na 1879 tare da yawan jama'a 1044.
A ranar 7 ga watan Janairu 1902 wani ɓangare na kalliope ya rabu don ƙirƙirar Miriam Vale Division.
Tare da nassi na sanan Hukumomin Dokar 1902, Calliope Division ya zama Shire na Calliope a kan 31 Maris 1903.
A cikin shekara ta 1927, zauren majalisa ya kasance a Gladstone.
Biyo bayan rahoton da Hukumar Gyaran Karamar Hukuma ta fitar a watan Yulin 2007, tsoffin kananan hukumomi uku:
An hade su don samar da yankin Gladstone akan 15 Maris 2008.
Garuruwa da unguwanni
Shire na kalliope ya haɗa da ƙauyuka masu zuwa:
Garuruwa:
Yankunan birni:
Sauran al'ummomi:
Ciwon Daji
Ciwon daji, ko ciwon daji na iyali, cuta ce ta kwayoyin halitta wanda gadaje maye gurbi a cikin daya ko fiye da kwayoyin halitta ya sa wadanda abin ya shafa ga ci gaban ciwon daji kuma yana iya haifar da farkon farkon wadannan ciwon daji. Ciwon daji sau da yawa yana nuna ba kawai babban haɗarin kamuwa da cutar kansa ba, har ma da haɓakar ciwace-ciwacen farko masu zaman kansu.
Yawancin waɗannan cututtukan suna haifar da maye gurbi a cikin ƙwayoyin cuta masu hana ƙari, kwayoyin halittar da ke da hannu wajen kare tantanin halitta daga juyewar kansa. Sauran kwayoyin halittar da za a iya shafa su ne kwayoyin gyaran DNA, oncogenes da kwayoyin da ke cikin samar da jini ( angiogenesis ). Misalai na gama-gari na ciwon daji da aka gada sune ciwon daji na nono-ovarian na gado da ciwon daji marasa polyposis colon cancer (Lynch syndrome).
Fage
Ciwon daji na gado yana ƙarƙashin kashi 5 zuwa 10 cikin ɗari na duk cututtukan daji kuma akwai nau'ikan ciwon daji sama da 50 waɗanda za a iya gane su. Fahimtar ilimin kimiyya game da cututtukan ciwon daji na haɓakawa sosai: ana samun ƙarin cututtukan cututtuka, ilimin halitta yana ƙara bayyana, kuma gwajin ƙwayoyin cuta yana inganta ganowa, jiyya, da rigakafin cututtukan daji. Idan aka yi la’akari da yawaitar ciwon nono da ciwon hanji, cututtukan da aka fi sani da su sun haɗa da ciwon daji na nono-ovarian na gado da kuma ciwon daji marasa polyposis colon cancer (Lynch syndrome).
Wasu cututtukan da ba a saba gani ba suna da alaƙa mai ƙarfi tare da cututtukan cututtukan daji na gado. Ya kamata a yi la'akari da gwajin kwayoyin halitta tare da adrenocortical carcinoma ; ciwon daji na carcinoid ; yaduwa ciwon daji ; tube fallopian / ciwon daji na peritoneal na farko; leiomyosarcoma ; medullary thyroid ciwon daji ; paraganglioma / pheochromocytoma; ciwon daji na koda na chromophobe, hybrid oncocytic, ko oncocytoma histology; ciwon daji na sebaceous ; da ciwace-ciwacen jima'i tare da tubules na annular. Likitocin kulawa na farko na iya gano mutanen da ke cikin haɗarin kamuwa da ciwon daji na gado.
Genetics na ciwon daji
Kwafi biyu na kowane kwayar halitta suna nan a cikin dukkan sel na jiki kuma kowannensu ana kiransa allele. Yawancin ciwon daji na ciwon daji ana yaɗa su ta hanyar ƙwararrun ƙwararrun ƙwayoyin cuta. A cikin waɗannan lokuta, allele guda ɗaya kawai dole ne ya kasance a wurin don mutum ya sami yanayin cutar kansa. Mutanen da ke da allele guda ɗaya na al'ada da kuma allele ɗaya mara kyau ana kiran su heterozygous. Mutum mai heterozygous da mutumin da ke da allura biyu na al'ada ( homozygous ) za su sami damar 50% na haifar da yaron da abin ya shafa. An san maye gurbin da ke cikin kwayoyin halittar da aka gada a matsayin maye gurbi na germline da kuma ƙarin maye gurbi a cikin al'ada na al'ada yana haifar da ci gaban ciwon daji. Wannan shi ake kira da Knudson ta biyu-hit hasashe, inda farkon bugu na kwayar halitta shine maye gurbi kuma bugun na biyu yana faruwa daga baya a rayuwa. Kamar yadda kawai allele ɗaya ke buƙatar canzawa (kamar idan aka kwatanta da duka a cikin abin da ake kira "ciwon daji na lokaci-lokaci"), mutum yana da babbar dama ta bunkasa ciwon daji fiye da yawan jama'a.
Kadan sau da yawa, ana iya yaɗuwar ciwon siga a matsayin halin koma baya na autosomal. Dole ne a canza nau'ikan cututtukan da kuma da cutar kansa. Mutumin da ke da allale guda biyu ana kiransa da homozygous recessive. Duk iyaye biyu dole ne su sami aƙalla kuskure guda ɗaya don yaro ya zama mai ɗaukar homozygous. Idan iyaye biyu suna da allele guda ɗaya da allele guda ɗaya ( heterozygous ) to suna da damar 25% na samar da ɗan luwaɗi na homozygous (yana da tsinkaya), 50% damar samar da ɗan heterozygous (mai ɗaukar kwayar cutar) da 25% damar haifar da yaro tare da alleles guda biyu na al'ada.
Misalan cututtukan ciwon daji na autosomal sune autoimmune lymphoproliferative syndrome (Canale-Smith Syndrome), ciwon Beckwith-Wiedemann (ko da yake kashi 85 cikin 100 na lokuta na ɗan lokaci ne), -Dubé ciwo, Carney ciwo, familial chordoma, Cowden ciwo, dysplastic nevus ciwo tare da familial melanoma, iyali adenomatous polyposis, gadon nono-ovarian ciwon daji, gadon yaduwa na ciki ciwon daji (HDGC), Gada nonpolyectal cancer color. (Lynch ciwo), Howel-Evans ciwo na esophageal ciwon daji tare da tylosis, yara polyposis ciwo, Li-Fraumeni ciwo, mahara endocrine neoplasia type 1/2, mahara osteochondromatosis, neurofibromatosis type 1/2, babu basal-cell carcinoma ciwo (Gorlin ciwo). ), Peutz–Jeghers ciwo, Familial prostate ciwon daji, hereditary leiomyomatosis renal cell ciwon daji (LRCC), hereditary papillary renal ciwon daji, hereditary paraganglioma -pheochromocytoma ciwo, retinoblastoma, tuberous sclerosis, von Hippel-Lindau cuta da kuma Wilm's tumor cuta.
Okaukuejo
Okaukuejo ita ce cibiyar gudanarwa na Etosha National Park a Namibia. Yana da kusan kilomita 650 daga Windhoek babban birnin kasar. Wurin yana samun matsakaicin ruwan sama na shekara-shekara na kusan 350 mm, kodayake a lokacin damina na 2010/2011 676 mm aka auna.
Ciwon Daji
Genetics na ciwon daji
Misalan cututtuka na ciwon daji na autosomal sune ataxia-telangiectasia, Bloom syndrome, Fanconi anemia, MUTYH polyposis, Rothmund-Thomson ciwo, Werner ciwo da Xeroderma pigmentosum.
Okaukuejo
Hukumar kula da dajin Namibia kuma tana kula da sansanin yawon bude ido. Akwai wuraren shakatawa iri-iri daga wuraren zango zuwa gidajen kula da gida tare da kayan aikin braai. Akwai kuma babban wurin wanka, babban gidan abinci da mashaya. Akwai kananan shaguna guda biyu. Shago ɗaya yana sayar da abinci na yau da kullun da itacen wuta don braai.
Nassoshi
-19.18333°N, 15.93333°W<templatestyles src="Module:Coordinates/styles.css"> -19.18333°N, 15.93333°W
Ciwon Daji
Misalai
Ko da yake ciwon daji yana nuna haɗarin ciwon daji, haɗarin ya bambanta. Ga wasu daga cikin waɗannan cututtuka, ciwon daji ba shine farkon fasalinsu ba.
Amelungsburg (Süntel)
Amelungsburg ya kasan ce wata tsohuwar sautin ringi ce wacce ke kan tudun Amelungsberg a kan tudun Süntel a gundumar Hameln-Pyrmont a jihar Lower Saxony ta Jamus.
Ciwon Daji
Fanconi anemia
Fanconi anemia cuta ce da ke da fa'idar bakan asibiti, gami da: farkon farawa da haɗarin ciwon daji; gazawar kasusuwa ; da rashin haihuwa. Babban bayyanar cututtuka na wannan cuta shine wadanda ke da alaka da hematopoeisis (samar da jini ta hanyar kasusuwa ); waɗannan sun haɗa da anemia aplastic, myelodysplastic syndrome da kuma m myeloid cutar sankarar bargo. Ciwace-ciwacen hanta da squamous cell carcinomas na esophagus, oropharynx da uvula sune ciwace-ciwacen ciwace-ciwacen da aka danganta da FA. Abubuwan da aka haifa sun haɗa da: skeletal anomalies (musamman wadanda ke shafar hannu), cafe au lait spots da hypopigmentation. Har zuwa yau, kwayoyin halittar da aka sani suna haifar da FA sune: FANCA, FANCB, FANCC, FANCD2, FANCE, FANCF, FANCG, FANCI, FANCJ, FANCL, FANCM, FANCN, FANCO, FANCP da BRCA2 (wanda aka sani da FANCD1). Gadon wannan ciwo da farko shine recessive autosomal, amma FANCB za a iya gado daga uwa ko uba x-chromosome ( x-linked recessive gadon ). Hanyar FA tana da hannu wajen gyaran DNA lokacin da aka haɗa nau'i biyu na DNA ba daidai ba ( interstrand crosslinks ). Hanyoyi da yawa ana daidaita su ta hanyar FA don wannan ciki har da gyaran ɓangarorin nucleotide, fassarar fassarar da sake hadewar homologous.
Amelungsburg (Süntel)
Bayani
Shafin yana kusa da kadada 15 kuma ya ta'allaka ne akan tudun Amelungsberg, wanda tsayinsa ya kai mita 900 da faɗin mita 300, kuma ya faɗi ƙasa sosai a kowane bangare. Kafaffun sun ƙunshi shinge a ɓangarorin biyu na ƙasa tare da tsawon kusan mita 1000. A yau tana da tsayin kusan mita 1.5 kuma tana da faɗin mita 4. A sauran ɓangarorin biyu akwai alamun ramuka. Akwai ragowar tsohuwar hanyar shiga a gefen kudu maso gabas na tsaunin da ke ƙasa da mita 70 yamma da ɓarkewar yau a cikin kariya ta hanyar gandun daji.
A ƙasa rukunin yanar gizon yana gudanar da tsauni mai tsawan mita 300 da tudu wanda ke nuna shaidar ƙofar. Wannan aikin ƙasa yana da faɗin kusan mita 7 kuma tsayinsa ya kai mita 2. Ginin da ke gabansa yana da faɗin mita 4 kuma har yanzu yana da zurfin mita.
Hakowa
A cikin 1954, 1955, da 2005, an yi haƙa ƙasa. Waɗannan sun tabbatar da cewa a cikin bangon akwai busasshen bangon dutse mai nisan mita 3 kuma tsayinsa ya kai mita 2.5. Kasusuwan dabbobi waɗanda wataƙila sun kasance ɓangaren sharar da rundunar gini ta bari, an ƙera carbon ɗin zuwa kusan 400 BC Kafin kayan aikin 300, gami da kayan aiki, sassan keken ƙarfe da tayoyin an sanya su zuwa lokacin La Tène. An gina shinge na gaba zuwa karni na 8. Abubuwan da aka samo daga lokacin Saxon sun haɗa da fibulae guda biyu, spurs, gatari, wuka da kan lance. Anvils, hammers da shavings tagulla shaida ce ta masu aikin ƙarfe.
Tarihi
Dangane da ramuka, an gina katangar a ƙarni na ƙarni na ƙarni kafin zamanin Roman, masu binciken kayan tarihi suna lissafin lokacin ginin har zuwa makonni shida. An tsawaita rukunin a lokacin amfani na kashi na biyu yayin Yaƙin Saxon. A cikin 782 a cikin Süntel an kashe sojojin Frank a ƙarƙashin sarkin Saxon Widukind, wanda ya kai ga kisan gillar Verden. Amelungsburg ba filin yaƙi ba ne - ba a sami makamai kaɗan a nan ba - amma yana iya kasancewa wani ɓangare na dabarun soja na Saxon gaba ɗaya.
Ciwon Daji
Familial adenomatous polyposis
Familial adenomatous polyposis (FAP) wani ciwo ne mai rinjaye na autosomal wanda ke ƙara haɗarin kansar launin fata. Kusan 1 cikin 8000 mutane za su kamu da wannan cuta kuma tana da kusan 100% shiga. Mutumin da ke da wannan cuta zai sami ɗaruruwa zuwa dubbai na adenoma mara kyau a ko'ina cikin hanjin su, wanda a mafi yawan lokuta zai ci gaba zuwa ciwon daji. Sauran ciwace-ciwacen da ke karuwa a yawan sun hada da; osteomas, adrenal adenomas da carcinomas, thyroid ciwace-ciwacen daji da kuma desmoid ciwace-ciwacen daji. Dalilin wannan rashin lafiya shine maye gurbin APC gene, wanda ke shiga cikin tsarin β-catenin. Kuskuren APC yana haifar da β-catenin don tarawa a cikin sel kuma kunna abubuwan rubutun da ke cikin yaduwar kwayar halitta, ƙaura, bambance-bambance da apoptosis (mutuwar kwayar halitta).
Gadon nono da ciwon daji na kwai
Ciwon daji na ciwon nono-ovarian na gado cuta ce da ta mamaye ta ta hanyar maye gurbi na kwayoyin halittar BRCA1 da BRCA2. A cikin mata wannan cuta da farko yana ƙara haɗarin nono da kansar kwai, amma kuma yana ƙara haɗarin kamuwa da cutar sankara na fallopian tube da papillary serous carcinoma na peritoneum. A cikin maza ana ƙara haɗarin cutar kansar prostate. Sauran cututtukan daji da ke da alaƙa da wannan ciwon sun haɗa da ciwon daji na pancreatic, kansar nono na namiji, ciwon daji na launin fata da kuma ciwon daji na mahaifa da cervix. Maye gurbin kwayoyin halitta sun kai kusan kashi 7% da 14% na ciwon nono da ovarian, bi da bi, kuma BRCA1 da BRCA2 ke da kashi 80% na waɗannan lokuta. BRCA1 da BRCA2 duka kwayoyin cutar ciwon tumo ne da ke da hannu wajen kiyayewa da gyara DNA, wanda hakan ke haifar da rashin zaman lafiyar kwayoyin halitta. Maye gurbi a cikin waɗannan kwayoyin halitta suna ba da damar ƙarin lalacewa ga DNA, wanda zai haifar da ciwon daji.
Gadon ciwon daji mara polyposis
Ciwon daji wanda ba shi da polyposis na gado, wanda kuma aka sani da cutar Lynch, ciwo ne na kansa wanda ya mamaye kansa wanda ke ƙara haɗarin kansar launin fata. Ana haifar da maye gurbi a cikin kwayoyin halittar DNA mismatch gyara (MMR), musamman MLH1, MSH2, MSH6 da PMS2. Bugu da ƙari, ciwon daji na launin fata da yawa sauran ciwon daji suna karuwa a mita. Wadannan sun hada da; ciwon daji na endometrial, ciwon ciki, ciwon daji na ovarian, ciwon daji na ƙananan hanji da ciwon daji na pancreatic. Ciwon daji na hanji wanda ba shi da polyposis kuma yana da alaƙa da farkon farawar kansar launin fata. Kwayoyin halittar MMR suna da hannu wajen gyara DNA lokacin da tushe akan kowane madaidaicin DNA ba su daidaita ba. Ƙwayoyin halittar MMR marasa lahani suna ba da damar ci gaba da sakawa da goge maye gurbi a yankuna na DNA da aka sani da microsatellites. Waɗannan gajerun jerin DNA masu maimaitawa sun zama marasa ƙarfi, suna haifar da yanayin rashin zaman lafiya na microsatellite (MSI). Ana samun maye gurbin microsatellites sau da yawa a cikin kwayoyin halittar da ke da hannu wajen farawa da ci gaba, kuma MSI na iya inganta rayuwar sel, wanda zai haifar da ciwon daji.
Paraganglioma-pheochromocytoma ciwo na gado
Yawancin lokuta na paraganglioma na iyali suna haifar da maye gurbi a cikin succinate dehydrogenase (succinate: ubiquinone oxidoreductase) ƙananan kwayoyin halitta ( SDHD, SDHAF2, SDHC, SDHB ).
PGL-1 yana da alaƙa da maye gurbin SDHD, kuma yawancin mutanen PGL-1 tare da paraganglioma sun shafi ubanni maimakon iyaye mata. PGL1 da PGL2 sun mamaye autosomal tare da bugawa. PGL-4 yana hade da maye gurbin SDHB kuma yana da alaƙa da haɗarin pheochromocytoma mafi girma, da kuma ciwon daji na renal cell da ciwon daji na thyroid wanda ba medullary ba.
Li-Fraumeni ciwo
Li-Fraumeni ciwo ne mai autosomal rinjaye ciwo da farko lalacewa ta hanyar maye gurbi a cikin TP53 gene, wanda ƙwarai yana ƙara haɗarin ciwon daji da yawa kuma yana da alaƙa sosai da farkon farkon waɗannan ciwon daji. Ciwon daji masu alaka da wannan cuta sun hada da; sarcomas nama mai laushi (sau da yawa ana samun su a cikin yara), osteosarcoma, ciwon nono, ciwon daji na kwakwalwa, cutar sankarar bargo da adrenocortical carcinoma. Mutanen da ke da ciwon Li-Fraumeni sau da yawa suna da cututtukan daji na farko masu zaman kansu da yawa. Dalilin babban nau'in asibiti na wannan cuta na iya kasancewa saboda wasu maye gurbi da ke gyara cutar. Sunadaran da aka samar ta hanyar TP53 gene, p53, yana da hannu a cikin kama sake zagayowar tantanin halitta, gyaran DNA da apoptosis. Ƙila p53 bazai iya aiwatar da waɗannan matakai yadda ya kamata ba, wanda zai iya zama dalilin samuwar ƙwayar cuta. Saboda kawai 60-80% na mutanen da ke da cutar suna da maye gurbi a cikin TP53, sauran maye gurbi a cikin hanyar p53 na iya shiga cikin ciwo na Li-Fraumeni.
polyposis mai alaƙa da MUTYH
polyposis mai alaƙa da MUTYH yana raba mafi yawan fasalulluka na asibiti tare da FAP; Bambance-bambancen shine cewa cuta ce ta autosomal recessive cuta ta hanyar maye gurbi a cikin kwayar gyaran DNA na MUTYH. Ciwon daji tare da ƙarin haɗari a cikin wannan cuta sune ciwon daji na launi, adenoma na ciki da adenoma duodenal.
Nevoid basal cell carcinoma syndrome
Nevoid basal cell carcinoma ciwo, wanda kuma aka sani da Gorlin ciwo, ya kasan ce kuma wani autosomal rinjaye ciwon daji ciwo a cikin abin da hadarin basal cell carcinoma yana da yawa sosai. Cutar tana da alamun basal cell nevi, jaw keratocysts da skeletal abnormalities. Ƙididdiga na ciwon basal cell carcinoma na rashin lafiya ya bambanta, amma yana da kusan 1 a cikin 60000. Kasancewar basal cell carcinoma ya fi girma a fari fiye da mutane baƙi; 80% da 38%, bi da bi. Odontogenic keratocysts ana samun su a cikin kusan 75% na mutanen da ke da cutar kuma galibi suna faruwa a farkon rayuwarsu. Mafi yawan cututtukan kwarangwal suna faruwa a kai da fuska, amma sau da yawa wasu wuraren suna shafa kamar kejin haƙarƙari. Abubuwan da ke haifar da maye gurbi na wannan cuta yana faruwa a cikin kwayar halittar PTCH, kuma samfurin PTCH shine mai hana ƙari da ke cikin siginar tantanin halitta. Kodayake ainihin aikin wannan furotin a cikin ciwon basal cell carcinoma ba a san shi ba, yana da hannu a cikin hanyar siginar bushiya, wanda aka sani don sarrafa ci gaban kwayar halitta da ci gaba.
Von Hippel-Lindau cuta
Cutar von Hippel-Lindau cuta ce mai wuyar gaske, yanayin halitta mai ƙarfi da ke haifar da ɗaiɗaikun mutane ga ciwace-ciwace marasa kyau da marasa kyau. Mafi yawan ciwace-ciwacen daji a cikin cutar Von Hippel-Lindau sune tsarin juyayi na tsakiya da hemangioblastomas na retinal, carcinomas na renal fili, pheochromocytomas, ciwace-ciwacen ƙwayar cuta na pancreatic, cysts pancreatic, ciwace-ciwacen endolymphatic sac da epididymal papillary cystadenomas. Cutar Von Hippel-Lindau ta samo asali ne daga maye gurbi a cikin kwayar cutar ta von Hippel-Lindau a kan chromosome 3p25.3.
Xeroderma pigmentosum
Xeroderma pigmentosum cuta ce ta autosomal recessive cuta wacce ke da hankali ga hasken ultraviolet (UV), haɓakar haɗarin kunar rana a jiki da haɗarin cututtukan fata. Haɗarin ciwon daji na fata ya fi sau 10000 fiye da na mutane na yau da kullun kuma ya haɗa da nau'ikan kansar fata da yawa, gami da melanoma da cututtukan fata waɗanda ba melanoma ba. Hakanan, wuraren da aka fallasa rana na harshe, lebe da idanu suna da haɗarin kamuwa da cutar kansa. Xeroderma pigmentosum na iya haɗuwa da wasu cututtuka na ciki da kuma ciwace-ciwacen daji. Baya ga ciwon daji, wasu sauye-sauyen kwayoyin halitta waɗanda ke haifar da xeroderma pigmentosum suna hade da neurodegeneration. Xeroderma pigmentosum na iya haifar da maye gurbin kwayoyin halitta a cikin kwayoyin halitta 8, wanda ke samar da enzymes masu zuwa: XPA, XPB, XPC, XPD, XPE, XPF, XPG da Pol η. XPA-XPF su ne enzymes gyare-gyaren cirewar nucleotide waɗanda ke gyara DNA mai lalacewa da hasken UV da kuskuren sunadaran zasu ba da damar gina maye gurbin da hasken UV ya haifar. Pol η shine polymerase, wanda shine enzyme wanda ke shiga cikin kwafin DNA. Akwai polymerases da yawa, amma pol η shine enzyme wanda ke yin kwafin UV mai lalacewa DNA. Maye gurbi a cikin wannan kwayar halitta zai haifar da kuskuren pol η enzyme wanda ba zai iya kwafin DNA tare da lalacewar hasken UV ba. Mutanen da ke da maye gurbi na wannan kwayar halitta suna da juzu'in XP; Cutar bambance-bambancen XP.
Binciken kwayoyin halitta
Ana iya amfani da gwajin kwayoyin halitta don gano maye gurbin kwayoyin halitta ko chromosomes da ke wucewa ta cikin tsararraki. Mutanen da suka gwada ingancin samun maye gurbin kwayoyin halitta ba lallai ba ne a la'anta su don haɓaka cutar kansa da ke da alaƙa da maye gurbin, duk da haka suna da haɗarin kamuwa da cutar kansa idan aka kwatanta da yawan jama'a. Ana ba da shawarar cewa mutane su sami gwajin kwayoyin halitta idan tarihin lafiyar danginsu ya haɗa da: Iyali da yawa masu ciwon daji, wani a cikin danginsu wanda ya kamu da cutar kansa tun yana ƙanana ko kuma ta wata ƙabila. Tsarin binciken kwayoyin halitta hanya ce mai sauƙi, mara lalacewa. Duk da haka, kafin a gwada kwayoyin halitta don maye gurbin mai haƙuri yawanci dole ne ya je wurin mai ba da lafiya kuma ya bi ta hanyar shawarwari daya-daya, inda suke tattauna tarihin kansa da na iyali na ciwon daji. Kwararren likita zai iya tantance yiwuwar majiyyaci ya sami maye gurbin kuma zai iya jagorance su ta hanyar gwajin kwayoyin halitta. Yana da mahimmanci cewa wannan shawarwarin ya faru saboda yana tabbatar da cewa mutumin ya ba da izini don shiga gwajin kwayoyin halitta, ya sani kuma ya fahimci matakai, fa'idodi da iyakancewar hanya kuma ya fi sanin sakamakon sakamakon gwajin ji. Ana iya yin gwajin ta hanyar amfani da ruwan jiki ko ƙwayoyin majiyyaci, wannan ya haɗa da; jini (wanda shine ya fi kowa), jini, ruwan amniotic har ma da sel daga ciki na bakin da aka samo daga buccal swab. Ana aika wannan kayan zuwa dakin gwaje-gwaje na musamman na kwayoyin halitta inda masu fasaha za su bincika shi, ana mayar da sakamakon gwajin zuwa ma'aikacin lafiya wanda ya nemi bincike kuma an tattauna sakamakon tare da mai haƙuri.
Za a iya samun gwajin kai tsaye ga mabukaci ba tare da ƙwararren likita ba amma ba a ba da shawarar ba saboda mabukaci ya rasa damar tattauna shawararsu tare da ƙwararrun ilimi. Dangane da Laburaren Magunguna na Ƙasa a cikin gwajin ƙwayoyin halittar Amurka a Amurka farashin farashi daga $100-$2000 ya danganta da nau'in da ƙaƙƙarfan gwaji.
Ayyukan rigakafi
Gwajin kwayoyin halitta yana da mahimmanci kamar idan gwajin ya fito tabbatacce sun fi sanin lafiyar kansu da lafiyar dangin dangi. Tare da taimako da shawarwari daga kwararrun likitoci za su iya ɗaukar matakai don rage girman haɗarin ci gaban ciwon daji ta hanyar:
Akwai wasu nau'o'in ayyukan rigakafi, misali ga nono na gado da kuma Ciwon daji na Ovarian zai kasance ta hanyar tiyata: A hysterectomy shine cire duk ko wasu daga cikin mahaifa, yayin da mastectomy yana cire nono ( mastectomy biyu ma'ana cewa nono biyu). an cire su), wannan na iya ƙara shekaru akan tsawon rayuwarsu . Wani ma'auni na rigakafi shine gwajin cutar kansa na yau da kullun da kuma duba. Idan mutum yana da cutar Lynch to sai a yi masa tiyatar colonoscopy na yau da kullun don bincika idan akwai wani canji a cikin sel ɗin da ke rufe bangon hanji, an tabbatar da binciken akai-akai yana ƙara matsakaicin shekaru 7 akan tsawon rayuwar mutumin da ke fama da shi. daga cutar Lynch kamar yadda gano wuri da wuri yana nufin daidaitattun matakan rigakafi da tiyata za a iya ɗauka cikin sauri. Ana kuma ba da shawarar duba nono akai-akai ga matan da aka gano suna da maye gurbi na BRCA, haka kuma, binciken da aka yi kwanan nan ya nuna cewa maza masu haɗarin kamuwa da cutar kansar prostate saboda maye gurbi na BRCA na iya rage haɗarinsu ta hanyar shan aspirin . Aspirin yana da amfani sosai wajen rage yawan ciwon daji; duk da haka, dole ne a sha shi akai-akai sama da aƙalla tsawon shekaru biyar don yin tasiri.
Yawaitar maye gurbi a cikin kabilu daban-daban
Sau da yawa maye gurbi ya fi zama ruwan dare a wasu ƙabilu, wannan saboda jinsi na iya bin kakanninsu zuwa wuri ɗaya, rikitattun kwayoyin halitta daga kakanni sukan bi ta cikin tsararraki wanda shine dalilin da ya sa wasu ƙabilun suka fi kamuwa da maye gurbi, ta haka sai karuwa suke. yiwuwar kamuwa da cutar kansa [61]. Kamar yadda aka ambata a sama, wannan na iya zama da amfani saboda yana iya taimakawa ƙwararrun kiwon lafiya tantance haɗarin majiyyaci na maye gurbi kafin a gwada su. Ciwon daji na Werner yana da yawan 1 cikin 200,000 masu rai a cikin Amurka, amma yana shafar mutane a Japan a cikin 1 cikin 20,000-40,000. 1 a cikin 40 Ashkenazi Yahudawa suna da maye gurbi na BRCA, wannan babban bambanci ne daga yawan jama'a a Amurka inda 1 a cikin 400 ke shafar. Yahudawan Ashkenazi na cikin hatsarin kamuwa da ciwon nono na gado da kuma ciwon daji na kwai kuma ana ba da shawarar cewa su yi gwajin kwayoyin halitta don ganin ko suna da maye gurbi da kuma duba cutar kansa akai-akai.
Kwalejin ilimi ta jihar Jigawa
Kwalejin Ilimi ta Jihar Jigawa babbar cibiyar ilimi ce ta gwamnatin jiha wacce ke Gumel, Jihar Jigawa, Najeriya. Yana da alaƙa da Jami'ar Bayero ta Kano don shirye -shiryen digiri. Shugaban makarantar na yanzu shine Saidu Barau Ahmad.
Tarihi
An kafa Kwalejin Ilimi ta Jihar Jigawa a 1976.
Darussan
Cibiyar tana ba da darussan masu zuwa;
Sultan Al-Ghannam
Sultan bin Abdullah bin Salem Al-Ghannam (Arabic; an haife shi a ranar 6 ga watan Mayu 1994) ɗan wasan ƙwallon ƙafa ne na Saudi Arabia wanda ke taka leda a matsayin mai yin wasa a gefen dama ga ƙungiyar Saudi Professional League Al Nassr da ƙungiyar Saudi Arabia.
Ayyukan kulob
Al-Ghannam ya fara aikinsa a kulob din Al-Zulfi. A ranar 27 ga watan Agustan shekara ta 2015, Al-Ghannam ya shiga kungiyar Pro League ta Al-Faisaly.
A ranar 12 ga watan Maris na shekara ta 2018, Al-Ghannam ya shiga Al-Nassr a kan canja wurin kyauta, ya sanya hannu kan kwangilar shekaru hudu tare da kulob din. A kakar wasa ta farko a kulob din, Al-Ghannam ya lashe gasar Pro League ta 2018-19. A ranar 21 ga watan Satumba 2021, Al-Ghannam ya sabunta kwantiraginsa da Al-Nassr har zuwa karshen kakar 2023-24.
Ayyukan ƙasa da ƙasa
An haɗa Al-Ghannam a cikin tawagar Saudi Arabia don gasar cin kofin Asiya ta AFC ta 2019 a Hadaddiyar Daular Larabawa.
A ranar 20 ga watan Nuwamba 2019, an ambaci sunan Al-Ghannam a cikin tawagar gasar cin kofin Gulf ta Larabawa ta 24.
A ranar 11 ga watan Nuwamba 2022, an sanya Al-Ghannam a cikin tawagar gasar cin kofin duniya ta FIFA ta 2022.
Kungiyar
Al-Nassr
Jami'ar kimiya da fasaha ta jihar kano dake wudil
Jami’ar kimiya da fasaha ta jihar kano dake wudil ko (KUST) an fari kirkiran tane a matsayin "Jami’ar fasaha" (KUT) a shekara ta 2001. Jami’ar tazo na 123 cikin jerin fitattun jami’oin da muke dasu a kasarmu Nigeria. Rabiu Musa Kwankwaso shiya samar da wannan jami’a ta farko wacce take itace mallakin jihar lokacin da yayi mulkinshi na farko a matsayin gwamna a jihar ta kano(Kano State), daga shekara ta 1999 zuwa 2003. Wacce a ka samar da ita a wudil, daya daga cikin kananun hulumomin na jihar kano a Nigeria. Jami’ar wacce akasani da Jami’ar fasaha dake Wudil, ta samu sauyin suna inda akamayar da ita Jami’ar kimiya da fasaha ta jihar kano Wudil a shekarar 2005 cikin tsari da bin dokoki kamar yarda shugabancin hukumar jami’oi sukatanada. Jami’ar tashiga daga cikin jerin jami’oi masu arziki anan gaba.
Tsari
KUST, Jami’ar kimiya da fasaha ta jihar kano ta fara da bangarori guda biyu ne (Bangaren kimiya ta noma da bunqasa tattalin arziki na fasaha da kuma Bangaren kimiya) amma yanzu Akwai bangarori guda shida (6) bangarorin - Bangaren noma da bunkasa tattalin arziki na fasaha (FAAT); Bangaren kimiya ta Computa da lissafi (FACAMS) bangaren kimiyar ilimin kasa da kula da tattalin arzikin kasa (FAEES); Bangaren injiniyoyi (FAENG); Bangaren koyon kimiya (FASSE) da kuma bangaren koyarwa na karantarwa na musamman (FASTE).
Hausa mouse
Mouse Hausa (Mus haussa) wani nau'in ɓera ne a cikin gidan Muridae. Ana samunsa a ƙasashe kamar su: Benin, Burkina Faso, Ivory Coast, Ghana, Mali, Mauritania, Niger, Nigeria, da Senegal. halitta habitats ne bushe savanna, arable ƙasar, yankunan karkara da gidãjen Aljanna, kuma birane.
2011 zaɓen gwamnan jihar Taraba
Zaɓen gwamnan jihar Taraba a shekara ta 2011 shine zaɓen gwamnan jihar Taraba karo na 5. Wanda aka gudanar a ranar 26 ga Afrilu, 2011, ɗan takarar jam’iyyar People’s Democratic Party Danbaba Suntai ya lashe zaben, inda ya doke Ahmed Yusuf na Congress for Progressive Change.
Manuela Aguilera
Aiki
A wasannin nakasassu na lokacin bazara na 1984, ta sami lambar zinare a tseren mita 100 na baya L6, da mita 200 na kowane mutum L6. Ta ci lambar azurfa a tseren tseren mita 100 na L6.
2011 zaɓen gwamnan jihar Taraba
Sakamako
ƴan takara 11 ne suka fafata a zaben. Danbaba Suntai daga jam’iyyar People’s Democratic Party ne ya lashe zaɓen, inda ya doke Ahmed Yusuf na jam’iyyar Congress for Progressive Change. Ingantattun kuri'u 716,769.
Kwale-kwalen Dufuna
Kwale-Kwalen Dufuna wani Kwale-Kwale ne da wani makiyayi (Bafulatani) ya gano shi a shekarar Alif da dari tara da tamanin da bakwai (1987), mai tazarar kilomita kaɗan daga ƙauyen Dufuna da ke a Ƙaramar Hukumar Fune, nesa kaɗan da kogin Komadugu Gana, a jihar Yobe, da ke Arewa Maso Gabashin Najeriya.
Tarihi
A ranar 4 ga watan Mayun (1987), wani makiyayi (Bafulatani) mai suna Malam Ya'u a yayin da yake tona rijiya ya gano wannan Kwale-Kwalen. Nan da nan kuma sai ya yi hanzari  ya sanar da sarkin ƙauyensu game da lamarin.
A cikin shekara ta 1989 da 1990, Jami'ar Maiduguri ta fara binciken wurin don tabbatar da ko Kwale-Kwale ne.
Jami'ar'ar Frankfurt da ta Maiduguri wadda Farfesa Peter Breunig da Garba Abubakar, suka gudanar da binciken wurin, in da kuma aka sake ɗaukar samfurin Kwale-kwalen da wasu sassa domin aiwatar da gwaje-gwaje a Jamus a karo na biyu.
A shekara ta Alif da Ɗari Tara da Casa'in da Huɗu (1994), wata tawagar binciken kayan tarihi daga Jamus da Najeriya ta tona wurin, in da ma'aikata Hamsin suka aikin haƙo Kwale-Kwalen, wadda hakan ya ɗauke su sama da mako biyu, kuma an gano tsawonsa ya kai mita 8.4, faɗinsa kuma mita 0.5 sannan yana da kauri 5cm.
An gano Kwale-kwalen a cikin wani ruwa da aka tona da yumɓu ke kwance tsakaninsa da bisa wanda ke kare shi a cikin yanayi mara amfani da iskar oxygen.
Wataƙila an ƙirƙire shi a cikin al'adar yin kwale-kwale a wancan lokacin, kuma an yi amfani da shi wajen kamun kifi a gefen kogin Komadugu Gana. Wataƙila jama'a ne suka ƙera shi daga yankin Yammacin Sahara, zuwa kogin Nilu na tsakiyar Sudan, zuwa yankin Arewa na Kenya.
A yanzu haka dai, Kwale-kwalen yana cikin gidan Adana Kayan Tarihi na Damaturu a jihar Yobe, Najeriya.
A ranar Huɗu ga watan Mayun Alif da Ɗari Tara da Tamanin da Bakwai (1987), wani makiyayi (Bafulatani) mai suna Malam Ya'u a yayn da yake tona rijiya.
Jami'ar Frankfurt da na Maiduguri wadda Farfesa Peter Breunig da Garba Abubakar, suka gudanar da binciken wurin, in da kuma aka sake ɗaukar samfurin Kwale-kwalen da wasu sassa domin aiwatar da gwaje-gwaje a Jamus a karo na biyu.
A shekara ta Alif da Ɗari Tara da Casa'in da Huɗu (1994), wata tawagar binciken kayan tarihi daga Jamus da Najeriya ta tono wurin, in da ma'aikata Hamsin suka haƙo in da ake zargin Kwale-Kwalen yake wadda ya ɗauke su sama da mako biyu, kuma an gano tsawonsa ya kai mita 8.4, faɗinsa mita 0.5 da kauri 5cm.
Kwale-kwalen wanda ya kasance sanannen kuma mafi tsufa a nahiyar Afirka, sannan na biyu a tsufa a duniya. An haƙƙace kan cewar ya kai tsakanin shekaru 8,000 zuwa 8,500 kafin haihuwar Annabi Isa AS.
Wataƙila an ƙirƙire shi a cikin al'adar yin kwale-kwale a wancan lokacin, kuma an yi amfani da shi wajen kamun kifi a gefen kogin Komadugu Gana.
Wataƙila jama'a ne suka ƙera shi daga yankin Yammacin Sahara, zuwa kogin Nilu na tsakiyar Sudan, zuwa yankin Arewa na Kenya.
1004 Estate
1004 Housing Estate wani yanki ne mai girman hekta 11 a cikin Victoria Island, Legas. Asalin sunan Estate Federal Housing Estate, Legas kuma Isaac Fola-Alade ya tsara shi, an gina shi a cikin shekarar 1970s a matsayin mafi girman nau'in sa a lokacin. Gidan yana da gungu 4 na gidaje masu gidaje da yawa; Gine-gine masu tsayi 6 da ƙananan gine-gine 4 waɗanda suka ƙunshi gidaje sama da 1000. An gina shi azaman wurin zama na alfarma ga iyalan Sanatoci da membobin Majalisar Wakilai, an buɗe wannan kadara a cikin shekarar 1979. Bayan da aka koma Abuja babban birnin tarayya, manyan ma’aikatan gwamnatin tarayya suka mamaye shi. Gidan kuma ya kasance mazaunin ƴan ƙasar waje. A cikin shekarar 2004, an sayar da kadarorin ga Kamfanin Haɓaka Dukiyar UACN. A shekara ta 2007, wannan kadara ta shiga cikin tsarin ba da izini da gwamnati ta yi don mika kulawa ga masu haɓaka kadarorin masu zaman kansu. Yarjejeniyar da ta kai Naira biliyan 7 ita ce hada-hadar kadarorin da ta fi kowacce girma a Najeriya a wannan shekarar.
Rigingimu
Mazauna yankin sun yi ikirarin cewa jami’an tsaro da ke kula da masu aikata miyagun laifukan da ke lalata wutar lantarki da dukiyoyinsu. Kukan taimako ya kai ga gwamnati. Kwamitin majalissar dokokin jihar Legas mai kula da gidaje ya kai ziyarar ba-zata domin gudanar da bincike kan rikicin da ya barke gidajen.
Pilar Javaloyas
Pilar Javaloyas 'yar wasan nakasassu ce ta kasar Sipaniya wacce ta fafata a Para iyo. Ta lashe lambobin yabo goma sha ɗaya a wasannin nakasassu na lokacin bazara na 1980, na nakasassu na bazara na 1984, da na nakasassu na lokacin rani na 1988.
Aiki
A wasannin nakasassu na lokacin bazara na 1980, ta lashe lambobin azurfa a cikin bugun ƙirjin 6 na mita 100, mita 100 na baya 6, mita 100 mara nauyi 6, mita 100 malam buɗe ido 6, da 200 na kowane mutum 6.
A wasannin nakasassu na lokacin bazara na 1984, ta sami lambar azurfa ta mita 100 na baya L5, da lambobin tagulla a cikin mita 100 malam buɗe ido L5, da mita 200 na kowane mutum L5.
A wasannin nakasassu na bazara na 1988, a Seoul, ta ci lambobin zinare, a cikin mita 100 na baya na 6, mita 100 malam buɗe ido 6, da lambar tagulla mai tsayin mita 400.
Muhammad Taqi-ud-Din al-Hilali
Muhammad Taqi-ud-Din bin Abdil-Qadir Al-Hilali (Arabic: محمد تقي الدين الهلالي, : ; 1893-1987) ya kasance Salafi na Maroko na ƙarni na 20, wanda ya fi shahara da fassarorin Ingilishi na Sahih Bukhari kuma, tare da Muhsin Khan, Alkur'ani, mai taken Alkur'an Mai Girma
Rayuwa ta farko da ilimi
An haifi Hilali a Rissani, Morocco, kusa da oasis na Tafilalt a cikin kwarin kusa da Sajalmasah a cikin 1893 (1311 AH).
A cikin shekaru ashirin, Hilali ya koma Aljeriya don nazarin Shari'ar Musulmi, ya ci gaba zuwa Masar a 1922. Yayinda yake can, Hilali ya shiga Jami'ar Al-Azhar kawai don ya fita bayan ya yi takaici da tsarin karatun. Maimakon haka, Hilali ya shafe lokaci a ƙarƙashin kulawar Rashid Rida, sannan ya koma Maroko a wannan shekarar don kammala digiri na farko a Jami'ar al-Karaouine. Da yake amsa kiran da ya kafa Muslim Brotherhood Hassan al-Banna ya yi wa masu ilimi Musulmi na Maroko su raba ra'ayoyi tare da waɗanda ke wasu wurare, Hilali ya rubuta wasiƙu da yawa ga mujallar kungiyar waɗanda hukumomin mulkin mallaka na Faransa suka tsayar da su. An kama shi kuma an tsare shi na kwana uku ba tare da tuhuma ba, an sami sakin Hilali kuma ya tsere daga Morocco. Ba da daɗewa ba bayan ya tsere daga ƙasar, an yanke masa hukuncin kisa ba tare da izini ba saboda aikin tawaye da Faransa mai kare Morocco.
A Asiya da Turai
Bayan yin aikin hajji zuwa Makka, Hilali ya koma Indiya don bin karatun Hadith. Yayinda yake can, ya yi aiki a matsayin shugaban nazarin Larabci a Darul-uloom Nadwatul Ulama a Lucknow. Bayan kammala karatunsa a Indiya, Hilali ya shafe shekaru uku a Iraki kafin Sarki na farko na Saudi Arabia Ibn Saud ya gayyace shi da kansa don koyarwa a ƙasar musulmi mai tsarki. Hilali ya koyar kuma ya jagoranci addu'a a Madina a Al-Masjid an-Nabawi, wuri na biyu mafi tsarki na Islama, na tsawon shekaru biyu kuma ya koyar a Makka a Masjid al-Haram, wurin da ya fi tsarki na Islami, na tsawon shekara guda.
Bayan kammala karatunsa a Makka, Hilali ya shiga Jami'ar Baghdad; ya kuma yi aiki a matsayin mataimakin farfesa yayin da yake can. Hilali ya koma Indiya a takaice a karo na biyu, kuma ya shiga Jami'ar Lucknow a matsayin dalibi da malami, wanda ya fi shahara shi ne Abul Hasan Ali Hasani Nadwi. Shakib Arslan, wanda ya kasance babban aboki na Hilali, ya shiga cikin hulɗa a Ofishin Harkokin Waje na Jamus kuma ya taimaka wa Hilali ya yi rajista (kuma, a matsayin dalibi da malami) a Jami'ar Bonn. Wani almajirin Rashid Rida, masanin Salafi kuma mai fafutukar mulkin mallaka wanda ya fara koyar da Larabci a Jami'ar Bonn a 1936 kuma ya zama shugaban sashen al'adu na Cibiyar Nazarin Musulunci ta Babban Ofishin Harkokin Waje, da kuma mai watsa shirye-shiryen Rediyo Berlin a Larabci. A cikin 1942, Amin al-Husayni ya tura shi zuwa Maroko don shirya ayyukan ɓoye.
Komawa zuwa Maroko, sannan Iraki, sannan Maroko, sa'an nan Saudi Arabia, sannan Marok
Kusan ƙarshen Yaƙin Duniya na II, Hilali ya bar Jamus zuwa Morocco ta Faransa, wanda ya girgiza da kira ga 'yancin kai. Ya koma Iraki a shekara ta 1947, ya sake komawa matsayin malami a jami'ar da ke Baghdad. Bayan juyin juya halin 14 ga Yuli, Hilali ya koma masarautar Maroko mai zaman kanta sau ɗaya. An nada shi a matsayin malami a Jami'ar Mohammed V da ke Rabat a 1959 sannan daga baya a reshe a Fes.
A shekara ta 1968, Babban Mufti na Saudi Arabia Abd al-Aziz ibn Baz ya rubuta wa Hilali yana neman ya dauki matsayin koyarwa a Jami'ar Musulunci ta Madina, wanda Bin Baz shine shugaban. Hilali ya yarda, ya zauna a Saudi Arabia na wani lokaci tsakanin 1968 da 1974.
A shekara ta 1974, Hilali ya yi ritaya daga koyarwa har abada, ya koma Meknes da farko kuma daga baya zuwa Casablanca, inda ya mallaki gida. Hilali ya mutu a ranar 22 ga Yuni, 1987 (25th na Shawal a cikin shekara ta 1408 AH). An binne shi a unguwar Sbata.
Karɓar baƙi
Yawancin malaman musulmai da malaman Yammacin Turai sun soki Hilali saboda fassarar Alkur'ani. Dokta Ahmed Farouk Musa, masanin kimiyya a Jami'ar Monash, ya ɗauki fassarar Hilali-Khan a matsayin babban dalilin tsattsauran ra'ayi da aikin farfaganda da hukumomin addinin Saudiyya suka rarraba tare da kuɗi daga gwamnatin mai arzikin mai. Hakazalika, Imad-ad-Dean Ahmad, shugaban Cibiyar Minaret of Freedom ta Bethesda, ya yi iƙirarin ƙarya cewa fassarar Wahabi ce ta Alkur'ani kuma Musulmai a Amurka ba su yarda da ita ba.
Tun da fassarar Hilali ta dogara ne akan tafsir na gargajiya (bayani na Alkur'ani), yawancin waɗanda suka soki fassararsa suna da dalilai na baya.
Bugu da ƙari, Khaled Abou El Fadl da Khaleel Mohammed sun soki fassarar Hilali a matsayin karkatar da ma'anar Alkur'ani
Ayyuka
Hilali ya yi aiki tare da Muhammad Muhsin Khan a cikin fassarar Turanci na ma'anar Alkur'ani da Sahih Al-Bukhari. An bayyana fassararsu ta Alkur'ani a matsayin mai girman kai, wanda ya haɗa da sharhi daga Tafsir al-Tabari, Tafsir ibn Kathir, Tafsar al-Qurtubi da Sahih al-Bukhari. An kuma soki shi saboda saka fassarorin makarantar Salafi kai tsaye a cikin fassarar Alkur'ani ta Turanci. An zarge shi da koyar da Musulmai da masu yuwuwa tare da fassarorin Musulunci ta hanyar parenthesis, a matsayin koyarwar Alkur'ani kanta.
Mohammed bin Khalid Al Saud
[[File:Secretary Pompeo Meets with Saudi Crown Prince Mohammed bin Salman (44637935134).jpg|thumb|Sakatare Pompeo ya gana da Yarima mai jiran gado na Saudiyya Mohammed bin Salman
]] Mohammed bin Khalid Al Saud (an haife shi a shekara ta 1967) memba ne na House of Saud kuma ɗan kasuwa ne. Shi ne shugaban da kuma darektan kungiyar Al Faisaliah. Ya kasance shugaban kwamitin daraktocin Kamfanin Sadarwar Saudiyya tun a shekarar 2018.
Ƙuruciya da ilimi
An haifi Mohammed bin Khalid a shekarar 1967. Shi jikan Abdullah bin Faisal ne sabili da haka, babban jikan Sarki Faisal ne. Mahaifin Muhammadu shine Khalid bin Abdullah. Mahaifiyarsa 'yar Sarki Khalid ce, Al Jawhara.
Mohammed bin Khalid ya kammala karatu a Jami'ar Man Fetur da Ma'adanai ta Sarki Fahd kuma ya sami digiri na farko a cikin gudanar da masana'antu. Ya sami MBA daga Makarantar Kasuwanci ta Harvard a shekarar 1996.
Ayyuka
Mohammed bin Khalid ya fara aikinsa a Citibank a New York da Geneva. Bayan haka ya yi aiki a matsayin mataimakin janar manajan Bankin Saudi Amurka na tsawon watanni bakwai. Sa'an nan a watan Mayu na shekara ta 1997 ya fara aiki a matsayin mataimakin shugaban kasa a kungiyar Al Faisaliah Group bisa bukatar kawunsa, Mohammed bin Abdullah wanda a lokacin shi ne shugaban kamfanin. Kakan Mohammed Abdullah bin Faisal ne ya kafa kamfanin a shekarar 1970. Mohammed bin Khalid ya kasance shugaban da darektan kamfanin tun shekara ta 2002.