Other pulmonary diseases ruled out. The initial asthmatic response to the inhalation of the irritant is likely due to airways inflammation. The persistent bronchial hyperreactivity is not well explained by biopsy material of bronchial tissue from RADS patients. These biopsies show only a mild inflammatory response with no eosinophilic infiltration of the airway tissue. The absence of eosinophils suggests that the persistent asthmatic symptoms of RADS are not due to an inflammatory disorder.
Treatment is the same as for any asthmatic patient.
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