Support is provided during prenatal diagnosis, the child's life and after the child's passing.
Lack ofCorrespondence Between Phenotypic Manifestations and Triplicated Loci Along Chromosome 18. The authors suggest that this case, like others in the literature, support the notion that no one region of chromosome 18 is sufficient to produce the classical phenotype. The case of Binkert and colleagues represents a boy who has trisomy for the entire short arm and the top two bands of the long arm of 18. While the child has a multiple congenital anomaly syndrome with developmental delay, he certainly would not be identified as having the trisomy 18 syndrome. Reed proposes an interesting hypothesis related to the possible role of regulator genes being triplicated and producing the phenotypic finding of excessive arches in dermatoglyphics.