It creates an 8 bp target site duplication on transposition. Other Pages Interpretations P elements are typical transposable elements , containing terminal inverted repeats and creating target site duplications on transposition. Autonomous P elements encode a protein which is probably the transposase. Hybrid dysgenesis results from activation of the transposition ability of dormant P elements. P elements are maintained in a dormant state by a factor present in P cytoplasm .
Transposition causes a variety of genetic aberrations.
The resulting truncated protein acts as a repressor of transposition. In germ cells, removal of the fourth exon is inhibited by a germ-cell specific protein. The active transposase is made. This is an example of regulation by differential splice site suppression .