Intraneostriatal administration of glutamate antagonists increases behavioral activation and decreases neostriatal ascorbate via nondopaminergic mechanisms. Acute and long-term amphetamine treatments alter extracellular ascorbate in neostriatum but not nucleus accumbens of freely moving rats. Chronic ascorbate potentiates the effects of chronic haloperidol on behavioral supersensitivity but not D2 dopamine receptor binding. Changes in locomotion and dopamine neurotransmission following amphetamine, haloperidol, and exposure to novel environmental stimuli. Effect of forebrain dopamine depletion on novelty-induced place preference behavior in rats.
Naloxone enhances the expression of morphine-induced conditioned place preference.
The role of nucleus accumbens dopamine and excitatory amino acids in the expression of cocaine-induced behavioral sensitization in rats. An easily-fabricated electrode holder for recordings from awake, behaving animals. Modulation of glutamate-induced excitation of striatal neurons by dopamine iontophoresis in awake, unrestrained rats. Bari, A.A. and R.C. Pierce . Increases in calcium channel mRNA in the ventral tegmental area contribute to behavioral sensitization to cocaine.