Mice develop normally without tenascin.
Defects of embryonic organogenesis resulting from targeted disruption of the N-myc gene in the mouse. Regulation and role of PDGF receptor alpha subunit expression during embryogenesis. Dynamic expression pattern of the myc proto-oncogene in midgestation mouse embryos. Mice homozygous for a null mutation of activin beta B are viable and fertile. Identification of laminin domains involved in branching morphogenesis - effects of anti laminin monoclonal antibodies on mouse embryonic lung development.