Helper T s release substances that cause B cells to multiply and produce antibodies. The killer T cells now begin shooting holes in host cells infected by virus. Antibodies released by B cells bind to antigens on the surfaces of free-floating viruses. The binding makes it easier for macrophages to destroy viruses and signals complement to puncture holes in viruses. As infection is controlled, the activated T and B cells are turned off by suppresser T cells.
Memory cells remain behind to respond quickly if the same virus attacks again.