As you enter a sunlit room, the photoreceptors in the retina are bombarded with light. Photosensitivity in humans is controlled with the use of melanin. Albino rats lack melanin, in their pupils and epithelium layer, which aids in the ability to withstand sunlight. In addition to insufficient amounts of photopigment, albino rats may have a problem in retinal interaction. The interaction amongst the rods, cones and varying channels are the structures which give us the ability to decipher ranges of light.
Humans use rods for dim light and cones for bright light.
Taking the human model, two main differences between an albino rat and normal human s visual system is the amount of melanin and the proper functioning between rods, cones and their interaction with specific channels. It is interesting to note this role reversal of human model to observe animal behavior because it demonstrates that species other than the Homo Sapiens have a similar network of neurons which perform basically the same function in vision. Trying to connect albino rats and humans exemplifies the manner in which behavioral output is affected by a faulty connection or an insufficient amount of a specific photopigment. Even though abundant research has not been done on the amount of neurotransmitters in the visual system of albino rats, this comparison seems to suggest that humans and rats are interrelated. This demonstrates that two mammals do not need to be equivalent in behavioral productivity but have a common use in an evolutionary pathway.