Similarly, newer evidence suggests that the VMH is not a satiety center but rather is part of a circuit that regulates feeding and other motivated behaviors via the autonomic nervous system. Eventually, however, fat cells become resistant to insulin. No one stimulus for hunger Gastric extension from weight of food has been proposed. If you cut the neural connections, then the fly will eat till it explodes. CCK tested for nausea through use of conditioned taste aversion learning.
Injections cause decrease in food intake.
Cutting vagus has no effect. In adults, high estrogen levels associated with preference for sweet foods. Believe that leptin regulates body weight by telling the brain how much fat is stored. At least one of these has been identified in hypothalamus.