Should we be successful, our work will provide a novel target for new, more selective, antiarrhythmic drugs. Effects of lignocaine and quinidine on the persistent sodium current in rat ventricular myocytes. A persistent sodium current in rat ventricular myocytes. Inactivation-resistant channels underlying the persistent sodium current in rat ventricular myocytes. Sodium currents in toad cardiac pacemaker cells.
Hypoxia increases persistent sodium current in rat ventricular myocytes.
Inhibition of oxidative metabolism increases persistent sodium current in rat CA1 hippocampal neurons. Nitric oxide increases persistent sodium current in rat hippocampal neurons. Please direct enquiries regarding this page to the Lurline Hardy .