cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.24958824
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. 

Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. 

Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. 

Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.6084/m9.figshare.24958824.v1
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. 

Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. 

Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. 

Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.25919/869t-1387
title: CSMB_808 genomic data, from a coal seam metagenome from the Walloon Subgroup in the Surat Basin, Australia

description: Contig bin, Kelpie-reconstructed 16S rRNA gene sequences, Prokka annotation file, BlastKOALA annotation file, TransportDB annotation file, dbCAN annotation file
DOI: 10.34740/kaggle/dsv/7361255
title: Egypt Gold Prices | Daily Updated
DOI: 10.5281/zenodo.10001071
title: Revisiting Ion-Pair Interactions in Phase Transfer Catalysis: From Ionic Compounds to Real Catalyst Systems

description: Supporting information for the paper: Revisiting Ion-Pair Interactions in Phase Transfer Catalysis: From Ionic Compounds to Real Catalyst Systems
DOI: 10.5281/zenodo.8161222
title: Revisiting Ion-Pair Interactions in Phase Transfer Catal- ysis: From Pure Ionic Compounds to Real Catalyst Sys- tems

description: Supporting information for the paper: Revisiting Ion-Pair Interactions in Phase Transfer Catalysis: From Pure Ionic Compounds to Real Catalyst Systems
DOI: 10.5281/zenodo.8161221
title: Revisiting Ion-Pair Interactions in Phase Transfer Catalysis: From Ionic Compounds to Real Catalyst Systems

description: Supporting information for the paper: Revisiting Ion-Pair Interactions in Phase Transfer Catalysis: From Ionic Compounds to Real Catalyst Systems
DOI: 10.5281/zenodo.8386079
title: Revisiting Ion-Pair Interactions in Phase Transfer Catal- ysis: From Pure Ionic Compounds to Real Catalyst Sys- tems

description: Supporting information for the paper: Revisiting Ion-Pair Interactions in Phase Transfer Catalysis: From Pure Ionic Compounds to Real Catalyst Systems
DOI: 10.25345/c5222rh3v
title: MassIVE MSV000093785 - RORDEPs in supernatants of Rumincoccus torques strains cultures
title: This dataset contains the raw proteomics data acquired using a Orbitrap Exploris 480 from bacterial supernatants in R. torques ATCC 27756 culture, and collected from human plasma for the detection of two bacterial polypeptides called RORDEP1 and 2.
DOI: 10.6084/m9.figshare.24958848
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. 

Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. 

Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. 

Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.6084/m9.figshare.24958848.v1
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. 

Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. 

Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. 

Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.34810/data80/1
title: Database.tab
DOI: 10.6084/m9.figshare.24958851
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. 

Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. 

Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. 

Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.6084/m9.figshare.24958851.v1
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. 

Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. 

Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. 

Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.25919/bty2-hq67
title: Monoaromatic-degrading MAGs from coal seam environments of Australia and North America

description: Four metagenome-assembled genomes (MAGs) from the Krumholzibacteriota phylum and the Deltaproteobacteria class. Each MAG contains one or more benzoyl-CoA reductase genes. The associated metagenomes were obtained through public databases, and the recorded source environments were subsurface coal seams in Australia and North America.

Contigs bins and Prokka annotations (nucleotides) for each MAG is provided in this collection, as well as Kelpie-generated 16S rDNA results from the associated metagenomes, and the Python scripts that were used for producing and analysing the MAGs.
DOI: 10.5281/zenodo.10455004
title: Data underpinning "Measurement-Induced Dark State Phase Transitions in Long-Ranged Fermion Systems"

description: We provide the raw data used to produce the plots shown in our paper "Measurement-Induced Dark State Phase Transitions in Long-Ranged Fermion Systems" and a Jupyter Notebook that was used to plot the data.
description: We identify an unconventional algebraic scaling phase in the quantum dynamics of long-range hopping, free fermions, which are exposed to continuous local measurements. The algebraic phase occurs for hopping decay exponents 1 < p ≲ 3=2, and features an algebraic entanglement entropy growth, and a slow algebraic decay of the density-density correlation function, both with a fractional exponent. It is separated from a critical phase with logarithmic entanglement growth at small, and an area law phase with constant entanglement entropy at large monitoring rates. A perturbative renormalization group analysis predicts that the transitions to the long-range phase correspond to an unconventional, modified sine-Gordon theory. Exact numerical simulations of the monitored wave functions are in excellent agreement with an analytical replica field theory approach, which confirms the view of the measurement-induced phase transition as a quantum phase transition in the dark state of an effective, non-Hermitian Hamiltonian.
DOI: 10.5281/zenodo.10455005
title: Data underpinning "Measurement-Induced Dark State Phase Transitions in Long-Ranged Fermion Systems"

description: We provide the raw data used to produce the plots shown in our paper "Measurement-Induced Dark State Phase Transitions in Long-Ranged Fermion Systems" and a Jupyter Notebook that was used to plot the data.
description: We identify an unconventional algebraic scaling phase in the quantum dynamics of long-range hopping, free fermions, which are exposed to continuous local measurements. The algebraic phase occurs for hopping decay exponents 1 < p ≲ 3=2, and features an algebraic entanglement entropy growth, and a slow algebraic decay of the density-density correlation function, both with a fractional exponent. It is separated from a critical phase with logarithmic entanglement growth at small, and an area law phase with constant entanglement entropy at large monitoring rates. A perturbative renormalization group analysis predicts that the transitions to the long-range phase correspond to an unconventional, modified sine-Gordon theory. Exact numerical simulations of the monitored wave functions are in excellent agreement with an analytical replica field theory approach, which confirms the view of the measurement-induced phase transition as a quantum phase transition in the dark state of an effective, non-Hermitian Hamiltonian.
DOI: 10.22008/fk2/rsccz4
title: Geologisk kort over Danmark, 1:50 000. Kortbladet 1312 II Svendborg. Geologisk basisdatakort

description: Well data, hydrogeology, Paleocene, Eemian, Weichselian, Holocene
DOI: 10.6084/m9.figshare.24912150
title: Supplementary Material for: Duration and efficiency of combined vs. isolated aerobic training interventions in post-stroke cognition: a systematic review.

description: Background: This study aimed to evaluate the effects of physical activity on the cognition of patients with stroke, comparing the effectiveness of using isolated or combined rehabilitation, as well as the duration and intensity of training, to identify the characteristics of optimal training programs for post-stroke cognitive rehabilitation. Methods: For this systematic review, we followed PRISMA guidelines and searched Web of Science, Scopus, PEDRo, Sportdiscus, PubMed, Trial Registries, and Google Scholar for studies published between 2013 and April 12, 2023. We included randomized controlled trials (RCT) of interventions that measured physical activity's effect on cognition in patients with stroke. We restricted our search to reports published in the English language. Evidence from the randomized controlled trials was synthesized. The risk of bias was assessed using the Cochrane risk of bias tool. Results: Of the 1755 records identified, 34 were eligible, and data were available, with mainly low to moderate risk of general bias. The combined rehabilitation training programs proved more efficient when compared with isolated rehabilitation training programs in several cognitive domains. A moderate intensity, 12-week intervention duration, with three weekly sessions, proved adequate. Discussion: It seems that combined rehabilitation training programs are efficient for patients with stroke as these alter neuroplastic conditions due to synergistic or additional onset of action. Future research should investigate combined rehabilitation training programs, with follow-up, to assess how long the recorded improvements last. The protocol of this study is registered in PROSPERO, number (OMITTED FOR BLIND REVIEW).
DOI: 10.6084/m9.figshare.24912150.v1
title: Supplementary Material for: Duration and efficiency of combined vs. isolated aerobic training interventions in post-stroke cognition: a systematic review.

description: Background: This study aimed to evaluate the effects of physical activity on the cognition of patients with stroke, comparing the effectiveness of using isolated or combined rehabilitation, as well as the duration and intensity of training, to identify the characteristics of optimal training programs for post-stroke cognitive rehabilitation. Methods: For this systematic review, we followed PRISMA guidelines and searched Web of Science, Scopus, PEDRo, Sportdiscus, PubMed, Trial Registries, and Google Scholar for studies published between 2013 and April 12, 2023. We included randomized controlled trials (RCT) of interventions that measured physical activity's effect on cognition in patients with stroke. We restricted our search to reports published in the English language. Evidence from the randomized controlled trials was synthesized. The risk of bias was assessed using the Cochrane risk of bias tool. Results: Of the 1755 records identified, 34 were eligible, and data were available, with mainly low to moderate risk of general bias. The combined rehabilitation training programs proved more efficient when compared with isolated rehabilitation training programs in several cognitive domains. A moderate intensity, 12-week intervention duration, with three weekly sessions, proved adequate. Discussion: It seems that combined rehabilitation training programs are efficient for patients with stroke as these alter neuroplastic conditions due to synergistic or additional onset of action. Future research should investigate combined rehabilitation training programs, with follow-up, to assess how long the recorded improvements last. The protocol of this study is registered in PROSPERO, number (OMITTED FOR BLIND REVIEW).
DOI: 10.5281/zenodo.10468116
title: Attachments
DOI: 10.5281/zenodo.10468115
title: Attachments
DOI: 10.15129/7b3da154-f717-4b15-8118-abe3828c7c56
title: Data for: “The effect of laser pulse evolution on down-ramp injection in laser wakefield accelerators”

description: Data produced using the FBPIC particle-in-cell code https://github.com/fbpic/fbpic and used in the corresponding publication "The effect of laser pulse evolution on down-ramp injection in laser wakefield accelerators". Data details available in the README file.