cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.17639/nott.7149
title: From competition to facilitation: temporal contiguity determines interactions between events in human Action-Outcome performance

description: Three experiments (n=81, n=81, n=82 respectively) explored how temporal contiguity influences Action-Outcome learning, assessing whether an intervening signal competed, facilitated, or had no effect on performance and causal attribution in undergraduate participants. Across experiments, we observed competition and facilitation as a function of the temporal contiguity between Action and Outcome. When there was a strong temporal relationship between Action and Outcome, the signal competed with the action, hindering instrumental performance but not causal attribution (Experiments 1 and 3). However, with weak temporal contiguity, the same signal facilitated both instrumental performance and causal attribution (Experiments 1 and 2). Finally, the physical intensity of the signal determined the magnitude of competition. As anticipated by associative learning models, a more salient signal disrupted to a greater extent instrumental performance (Experiment 3). The current results can be accounted for by a recent adaptation of configural theory of learning (Herrera et al., 2021).
DOI: 10.17639/nott.7154
title: Raw Data for Customisable Tablet Printing: The Development of Multimaterial Hot Melt Inkjet 3D Printing to Produce Complex and Personalised Dosage Forms

description: Dataset contains raw data used for the creation of figures in the publication entitled 'Customisable Tablet Printing: The Development of Multimaterial Hot Melt Inkjet 3D Printing to Produce Complex and Personalised Dosage Forms'
DOI: 10.17639/nott.7166
title: Ink-jet 3D printing as a strategy for developing bespoke non-eluting biofilm resistant medical devices

description: Chronic infection as a result of bacterial biofilm formation on implanted medical devices is a major global healthcare problem requiring new biocompatible, biofilm-resistant materials. Here we demonstrate how bespoke devices can be manufactured through ink-jet-based 3D printing using bacterial biofilm inhibiting formulations without the need for eluting antibiotics or coatings. Candidate monomers were formulated and their processability and reliability demonstrated. Formulations for in vivo evaluation of the 3D printed structures were selected on the basis of their in vitro bacterial biofilm inhibitory properties and lack of mammalian cell cytotoxicity. In vivo in a mouse implant infection model, Pseudomonas aeruginosa biofilm formation on poly-TCDMDA was reduced by ∼99% when compared with medical grade silicone. Whole mouse bioluminescence imaging and tissue immunohistochemistry revealed the ability of the printed device to modulate host immune responses as well as preventing biofilm formation on the device and infection of the surrounding tissues. Since 3D printing can be used to manufacture devices for both prototyping and clinical use, the versatility of ink-jet based 3D-printing to create personalised functional medical devices is demonstrated by the biofilm resistance of both a finger joint prosthetic and a prostatic stent printed in poly-TCDMDA towards P. aeruginosa and Staphylococcus aureus.
DOI: 10.17639/nott.7179
title: Simultaneous conventional and microwave heating for the synthesis of adsorbents for CO2 capture: comparative study to pristine technologies

description: Microwave has become an attractive technology in the valorisation of renewable biomass and in the mitigation of challenges of climate change. In this work, the synergic effects of coupling microwave and mild conventional heating conditions has been investigated in preparing engineered ultra-micropore carbons from lignocellulosic biomass. The processing conditions were systematically investigated and correlated to the physicochemical properties of activated carbons produced and their performance in post-combustion CO2 capture. The highest CO2 uptake (225 mg g-1) was achieved for the hybrid carbon produced at low temperature (600 °C) and modest microwave intensity. The synergic effect of hybrid heating was confirmed by the significant CO2 uptake increase up to 80 and 60 % for the activated carbons prepared by microwave and conventional heating, respectively. The enhanced adsorption was confirmed by cyclic regeneration up to 99 % after 16 adsorption-desorption cycles, showing a linear correlation between the surface area, micropore volume and CO2 uptake. The Pseudo-first order model accurately describes the adsorption phenomena, indicating that physisorption is the primary mechanism governing the process. The results acquired from this study highlight the process intensification in the synthesis of porous materials with comparable properties that are typically attained in conventional heating using energy intensive conditions. Additionally, this approach reveals the benefits of conventional treatment for increasing the material’s microwave susceptibility and as consequence to reduce the processing time by microwave heating. The synergic effects confirms the potential of hybrid heating for applications where fast and selective heating is paramount.
DOI: 10.17639/nott.7190
title: Design and evaluation of new quinazolin-4(3H)-one derived PqsR antagonists as quorum sensing quenchers in Pseudomonas aeruginosa

description: A study focused on the design, synthesis and evaluation of pqs quorum sensing inhibitors
DOI: 10.17639/nott.7052
title: Christian Missions in sub-Saharan Africa

description: The databases share are two shapefiles detailing the location and characteristics of Protestant and Catholic missionaries in Africa in the late- 19th and early 20th centuries. PROTESTANT MISSIONS: Geocoded information at the mission level for missions operating in Africa up to 1903. Among others, the data report at the mission level missionary investments (printing presses, hospitals, schools, etc. Cite as Cagé, Julia and Valeria Rueda (2016), "The Long Term Effects of the Printing Press in sub-Saharan Africa." American Economic Journal: Applied Economics, 2016, vol. 8 (3), pp: 69-99, 2016 Source Beach, Harlam P. (1903), A Geography and Atlas of Protestant Missions: Their Environment, Forces, Distribution, Methods, Problems, Results and Prospects at the Opening of the Twentieth Century", New York: Student Volunteer Movement for Foreign Missions. CATHOLIC MISSIONS: Geocoded information at the mission level for Catholic missions operating in Africa up to 1924. The data reports missions with a hospital and Catholic administrative information Cite as : Cagé, Julia and Valeria Rueda (forthcoming), "Sex and the Mission: The Conflicting Effects of Early Christian Missions on HIV in sub-Saharan Africa", Journal of Demographic Economics. Source Streit, Karl (1929), Atlas Hierarchicus: Descriptio geographica et statistica Sanctae Romanae Ecclesiae tum occidentis tum orientis juxta statum praesentum accedunt nonnullae notae historicae necnon ethnographicae, Paderbornae : sumptibus typographiae Bonifacianae
DOI: 10.17639/nott.7182
title: Quantitative chemical exchange saturation transfer imaging of nuclear overhauser effects in acute ischemic stroke - underpinning data

description: Research originally performed at the Institute of Biomedical Engineering at the University of Oxford
description: Data and code required to generate figures in the paper entitled: Quantitative chemical exchange saturation transfer imaging of nuclear overhauser effects in acute ischemic stroke
DOI: 10.17639/nott.7189
title: Cannabidiol concentrations in individual lymph nodes

description: Cannabidiol concentrations in individual lymph nodes
DOI: 10.17639/nott.7178
title: Scanning Electron Microscope (SEM) raw data of vermiculite-based composite materials

description: Scanning Electron Microscope (SEM) photos for vermiculite-based composite materials to visualise the salt inside of the matrix of a composite. The resolution produced is better than one nanometre. This can be used to make sure that the salt is binding uniformly inside the matrix and therefore maximising surface area and potential for mass and heat transfer.
DOI: 10.17639/nott.7184
title: Data for 'Attitudes to Animal Use of Named Species for Different Purposes: Effects of Speciesism, Individualising Morality, Likeability and Demographic Factors'

description: to be linked to the published article which provides the full context to these data
DOI: 10.17639/nott.7186
title: Feldenkrais video

description: This Feldenkrais session was designed by a Feldenkrais practitioner to be used in a soma design workshop which aimed to improve aircraft passenger comfort.
DOI: 10.17639/nott.7117
title: Raw Egg Data

description: This dataset contains the lead concentrations found in the eggs sampled throughout this study. The farm eggs were produced at a small, private farm located approximately 0.6 km from a derelict lead mine. The control 1 eggs were produced at a private farm near Sutton Bonington, Nottinghamshire. The control 2 eggs were commercially available free range eggs from Wales, purchased from a major supermarket chain.
DOI: 10.17639/nott.7170
title: Elucidating the molecular landscape of the stratum corneum

description: • Characterization of the molecular structure of skin, especially the barrier layer, the stratum corneum using 3D OrbiSIMS. • Understand the chemical variations relating to fundamental biological processes, such as the epidermal cholesterol sulfate. • Investigate the non-homogeneity of the stratum corneum, both laterally and as a function of depth. Including the distribution and localisation of key components in the skin e.g. fatty acids, ceramides and natural moisturising factors. • Demonstrate the permeation of a commercially important anti-aging peptide into human stratum corneum.
DOI: 10.17639/nott.6531
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 6768R-2

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 6768R-2 (III) Source: NIG Name: DNApol-epsilon Full name: DNA polymerase ε 255kD subunit Also known as: DNApol-ε255, DNApol-ε, l(3)pl10, Polε Annotation symbol: CG6768 FlyBase ID: FBgn0264326 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)
DOI: 10.17639/nott.6538
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 25622

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 25622 (III) Source: VDRC Name: Dscam1 (1) Full name: Down syndrome cell adhesion molecule 1 Also known as: Dscam, l(2)43Bc, Dm_2R:13612 , Neu1, Dm_2R:13579 Annotation symbol: CG17800 FlyBase ID: FBgn0033159 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)
DOI: 10.17639/nott.6513
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 55906

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 55906 (III) Source: Bloomington Name: Ddr (2) Full name: Discoidin domain receptor Also known as: Annotation symbol: CG33531 FlyBase ID: FBgn0053531 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)
DOI: 10.17639/nott.6608
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 10293R-1

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 10293R-1 (III) Source: NIG Name: how Full name: held out wings Also known as: who, l(3)j5B5, sci, stru, anon- EST:Liang-2 .39 Annotation symbol: CG10293 FlyBase ID: FBgn0264491 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)
DOI: 10.17639/nott.6552
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 15887

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 15887 (III) Source: VDRC Name: E2F1 (2) Full name: E2F transcription factor 1 Also known as: E2F, dE2F1, dE2F, E(var)3-93E, drosE2F1 Annotation symbol: CG6376 FlyBase ID: FBgn0011766 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)
DOI: 10.17639/nott.6603
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 2040R-1

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 2040R-1 (III) Source: NIG Name: hig (2) Full name: hikaru genki Also known as: CT6282 Annotation symbol: CG2040 FlyBase ID: FBgn0010114 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)
DOI: 10.17639/nott.6606
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 37005

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 37005 (III) Source: VDRC Name: Hml (1) Full name: Hemolectin Also known as: CT21553 Annotation symbol: CG7002 FlyBase ID: FBgn0029167 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)
DOI: 10.15468/dl.6qajnp
title: Occurrence Download

description: A dataset containing 0 species occurrences available in GBIF matching the query:
{
 "and" : [
 "TaxonKey is Scomber alalunga Bonnaterre, 1788",
 "Geometry POLYGON((170 20, 170 50, 120 50, 120 20, 170 20))",
 "HasCoordinate is true"
 ]
}

 The dataset includes 0 records from 0 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0007295-241126133413365/datasets/export for details.
DOI: 10.17639/nott.6579
title: An in vivo systematic genetic analysis of tumour progression in Drosophila - RNAi line: 49960

description: This data set forms part of the larger 'Fly Cancer Screen' data set, which consists of over 700 image sets
description: Metastasis is the leading cause of death for cancer patients. Consequently it is imperative that we improve our understanding of the molecular mechanisms that underlie progression of tumour growth towards malignancy. Advances in genome characterisation technologies have been very successful in identifying commonly mutated or misregulated genes in a variety of human cancers. A major challenge however is the translation of these findings to new biological insight due to the difficulty in evaluating whether these candidate genes drive tumour progression. Using the genetic amenability of Drosophila melanogaster we generated tumours with specific genotypes in the living animal and carried out a detailed systematic loss-of-function analysis to identify numerous conserved genes that enhance or suppress epithelial tumour progression. This enabled the discovery of functional cooperative regulators of invasion and the establishment of a network of conserved ‘invasion suppressors’. RNAi line: 49960 (III) Source: VDRC Name: fkh (2) Full name: fork head Also known as: Sebp2 Annotation symbol: CG10002 FlyBase ID: FBgn0000659 File naming convention: File names typically contain representations of date (DDMMYY), RNAi Line, Animal Number and, in some cases, window (to accommodate larger samples that require multiple image stacks)