cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.7284/149428
title: GNSS (Global Navigation Satellite System) data collected during research cruise NBP0302 using a Trimble Centurion P(Y) instrument system onboard the platform RV Nathaniel B. Palmer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/149387
title: Gyrocompass data collected during research cruise NBP0302 using a Yokogawa CMZ700B instrument system onboard the platform RV Nathaniel B. Palmer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/149079
title: INS (Inertial Navigation System) data collected during research cruise NBP0302 using a Kongsberg Seapath 200 instrument system onboard the platform RV Nathaniel B. Palmer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/149416
title: Multibeam Echosounder data collected during research cruise NBP0302 using a Kongsberg EM120 instrument system onboard the platform RV Nathaniel B. Palmer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.5281/zenodo.6984708
title: Empirical impact of FDI Screening Mechanisms on FDI inflows

description: Data file for dissertation
DOI: 10.5281/zenodo.6984709
title: Empirical impact of FDI Screening Mechanisms on FDI inflows

description: Data file for dissertation
DOI: 10.5281/zenodo.6986031
title: Empirical impact of FDI Screening Mechanisms on FDI inflows

description: Data file for dissertation
DOI: 10.6073/pasta/a946904960bb11f44915b80fb4fc5981
title: Model Simulations of The Effects of Shifts in High-frequency Weather Variability (No Long-term Weather Trend) Control Carbon Loss from Land to the Atmosphere, Toolik Lake, Alaska, 2022-2122

description: Climate change is increasing extreme weather events, but effects on high-frequency weather variability and the resultant impacts on ecosystem function are poorly understood. We assessed ecosystem responses of arctic tundra to changes in day-to-day weather variability using a biogeochemical model and stochastic simulations of daily temperature, precipitation, and light. Changes in weather variability altered ecosystem carbon, nitrogen, and phosphorus stocks and cycling rates. Some responses of processes (e.g., respiration) were inconsistent with expectations, indicating that whole-ecosystem interactions and feedbacks moderate or even reverse responses to weather variability. More weather variability led to greater carbon losses from land to atmosphere, and less variability led to higher carbon sequestration on land. The magnitude of response to weather variability was similar to that predicted from climate mean trend effects. This dataset consists of the MEL parameter file, driver files and output files for simulations without a long term weather trend.
DOI: 10.5281/zenodo.6373649
title: PLEC: A Target Enabling Package

description: Little is known about how PLEC biology is linked to AD pathophysiology, despite the multifactorial indicators pointing to PLEC as a prominent risk factor for AD. This project is intended to develop resources to help the scientific community delve deeper into PLEC biology within the central nervous system, where PLEC is prominently expressed by both excitatory and inhibitory neurons, as well as astrocytes. The resource development for PLEC is a basic target enabling package (TEP), which includes the identification and characterization of viable antibodies against PLEC for biochemistry and immunohistology (the antibody report is linked below). We anticipate that the development of additional resources to investigate PLEC’s role in AD pathogenesis will result in the discovery of new therapeutic approaches to treat AD.
description: Future versions of this TEP will contain experimental data on the PLEC TEP.
DOI: 10.5281/zenodo.6374187
title: PLEC: A Target Enabling Package

description: Little is known about how PLEC biology is linked to AD pathophysiology, despite the multifactorial indicators pointing to PLEC as a prominent risk factor for AD. This project is intended to develop resources to help the scientific community delve deeper into PLEC biology within the central nervous system, where PLEC is prominently expressed by both excitatory and inhibitory neurons, as well as astrocytes. The resource development for PLEC is a basic target enabling package (TEP), which includes the identification and characterization of viable antibodies against PLEC for biochemistry and immunohistology (the antibody report is linked below). We anticipate that the development of additional resources to investigate PLEC’s role in AD pathogenesis will result in the discovery of new therapeutic approaches to treat AD.
description: Future versions of this TEP will contain experimental data on the PLEC TEP.
DOI: 10.5281/zenodo.6373648
title: PLEC: A Target Enabling Package

description: Little is known about how PLEC biology is linked to AD pathophysiology, despite the multifactorial indicators pointing to PLEC as a prominent risk factor for AD. This project is intended to develop resources to help the scientific community delve deeper into PLEC biology within the central nervous system, where PLEC is prominently expressed by both excitatory and inhibitory neurons, as well as astrocytes. The resource development for PLEC is a basic target enabling package (TEP), which includes the identification and characterization of viable antibodies against PLEC for biochemistry and immunohistology (the antibody report is linked below). We anticipate that the development of additional resources to investigate PLEC’s role in AD pathogenesis will result in the discovery of new therapeutic approaches to treat AD.
description: Future versions of this TEP will contain experimental data on the PLEC TEP.
DOI: 10.5281/zenodo.6373610
title: NDUFS2: A Target Enabling Package

description: The molecular role of NDUFS2 in mitochondrial metabolism is through its participation in complex I of the respiratory chain within the inner membrane of mitochondria. Complex I function is critical for mitochondrial ATP production, but is also involved in reactive oxygen species production. As a core component of complex I, NDUFS2 resides within two hypotheses of opposing mechanisms with the potential to foster AD pathology: AD Hypometabolism Hypothesis and the AD Oxidative Stress Hypothesis. Each hypothesis is subordinate to a separately identified biological domain (mitochondrial metabolism and oxidative stress). Consequently, decreasing complex I integrity via down-regulation of NDUFS2 could either promote or decrease the propagation of AD pathology. The primary aim of this project is to generate resources to more effectively study the role of NDUFS2 in AD pathogenesis, through the generation of a basic TEP suite of resources, including a full antibody characterization report linked below. Future studies leveraging these resources may shed light onto the molecular mechanism associated with NDUFS2 linkage with AD risk.
description: Future versions of this TEP will contain experimental data on the NDUFS2 TEP.
DOI: 10.5281/zenodo.6373609
title: NDUFS2: A Target Enabling Package

description: The molecular role of NDUFS2 in mitochondrial metabolism is through its participation in complex I of the respiratory chain within the inner membrane of mitochondria. Complex I function is critical for mitochondrial ATP production, but is also involved in reactive oxygen species production. As a core component of complex I, NDUFS2 resides within two hypotheses of opposing mechanisms with the potential to foster AD pathology: AD Hypometabolism Hypothesis and the AD Oxidative Stress Hypothesis. Each hypothesis is subordinate to a separately identified biological domain (mitochondrial metabolism and oxidative stress). Consequently, decreasing complex I integrity via down-regulation of NDUFS2 could either promote or decrease the propagation of AD pathology. The primary aim of this project is to generate resources to more effectively study the role of NDUFS2 in AD pathogenesis, through the generation of a basic TEP suite of resources, including a full antibody characterization report linked below. Future studies leveraging these resources may shed light onto the molecular mechanism associated with NDUFS2 linkage with AD risk.
description: Future versions of this TEP will contain experimental data on the NDUFS2 TEP.
DOI: 10.5281/zenodo.6374134
title: NDUFS2: A Target Enabling Package

description: The molecular role of NDUFS2 in mitochondrial metabolism is through its participation in complex I of the respiratory chain within the inner membrane of mitochondria. Complex I function is critical for mitochondrial ATP production, but is also involved in reactive oxygen species production. As a core component of complex I, NDUFS2 resides within two hypotheses of opposing mechanisms with the potential to foster AD pathology: AD Hypometabolism Hypothesis and the AD Oxidative Stress Hypothesis. Each hypothesis is subordinate to a separately identified biological domain (mitochondrial metabolism and oxidative stress). Consequently, decreasing complex I integrity via down-regulation of NDUFS2 could either promote or decrease the propagation of AD pathology. The primary aim of this project is to generate resources to more effectively study the role of NDUFS2 in AD pathogenesis, through the generation of a basic TEP suite of resources, including a full antibody characterization report linked below. Future studies leveraging these resources may shed light onto the molecular mechanism associated with NDUFS2 linkage with AD risk.
description: Future versions of this TEP will contain experimental data on the NDUFS2 TEP.
DOI: 10.5281/zenodo.6373581
title: Cathepsin H: A Target Enabling Package

description: CTSH participates in multiple AD linked biological domains, as noted above, and plays a role in both proteostasis and apoptosis, as well as immune response–three biological domains linked with AD pathology. Little is known about the precise role that CTSH may play in AD pathology, but it participates in hypotheses linked with all three biological domains. Given the genetic and omic association with AD risk, elucidating the mechanisms of biological contribution to AD risk may inform future therapeutic development. The goal of this project is to develop a basic target enabling package to facilitate the future exploration of the role of CTSH in AD biology.
description: Future versions of this TEP will contain experimental data on the CTSH TEP.
DOI: 10.5281/zenodo.6373582
title: Cathepsin H: A Target Enabling Package

description: CTSH participates in multiple AD linked biological domains, as noted above, and plays a role in both proteostasis and apoptosis, as well as immune response–three biological domains linked with AD pathology. Little is known about the precise role that CTSH may play in AD pathology, but it participates in hypotheses linked with all three biological domains. Given the genetic and omic association with AD risk, elucidating the mechanisms of biological contribution to AD risk may inform future therapeutic development. The goal of this project is to develop a basic target enabling package to facilitate the future exploration of the role of CTSH in AD biology.
description: Future versions of this TEP will contain experimental data on the CTSH TEP.
DOI: 10.5281/zenodo.6374095
title: Cathepsin H: A Target Enabling Package

description: CTSH participates in multiple AD linked biological domains, as noted above, and plays a role in both proteostasis and apoptosis, as well as immune response–three biological domains linked with AD pathology. Little is known about the precise role that CTSH may play in AD pathology, but it participates in hypotheses linked with all three biological domains. Given the genetic and omic association with AD risk, elucidating the mechanisms of biological contribution to AD risk may inform future therapeutic development. The goal of this project is to develop a basic target enabling package to facilitate the future exploration of the role of CTSH in AD biology.
description: Future versions of this TEP will contain experimental data on the CTSH TEP.
DOI: 10.6084/m9.figshare.20482518
title: Erythroxylum lineolatum decontaminated FSCR

description: .....
DOI: 10.6084/m9.figshare.20482518.v1
title: Erythroxylum lineolatum decontaminated FSCR

description: .....
DOI: 10.6084/m9.figshare.19798882
title: KCNJ6 Supplemental Tables

description: Supplemental tables for Popova et al., " <strong>Alcohol reverses the effects of </strong><em><strong>KCNJ6</strong></em><strong> (GIRK2) noncoding variants on excitability of human glutamatergic neurons"</strong> <br> <strong>Supplemental Table 1. </strong>SNPs identified in RNAseq samples. Variants were identified in the region surrounding KCNJ6 (chr21: 37499112-38245792) using samtools mpileup to bcftools call, followed by filtering to remove low quality. Results were loaded into the Ensembl VEP tool. A dot (“.”) indicates missing data and/or low quality. The reference allele and alternate alleles were obtained from the UCSC Genome Table Browser. <br> <strong>Supplemental Table 2. </strong>Differentially expressed genes (DEG) comparing AF to UN without exposure to ethanol. The table contains all output from DESeq2 results. Excel filters are set to show only genes significantly different (padj &lt;= 0.05) and at least 1.5-fold different (abs(log2FoldChange) &gt; 0.585). Release the filters to see the full list. <br> <strong>Supplemental Table 3. </strong>Enriched Gene Ontology-Biological Process (GO-BP) terms from genes increased in AF relative to UN. <br> <strong>Supplemental Table 4. </strong>Enriched Gene Ontology-Biological Process (GO-BP) terms from genes decreased in AF relative to UN.
DOI: 10.6084/m9.figshare.19798882.v2
title: KCNJ6 Supplemental Tables

description: Supplemental tables for Popova et al., " <strong>Alcohol reverses the effects of </strong><em><strong>KCNJ6</strong></em><strong> (GIRK2) noncoding variants on excitability of human glutamatergic neurons"</strong> <br> <strong>Supplemental Table 1. </strong>SNPs identified in RNAseq samples. Variants were identified in the region surrounding KCNJ6 (chr21: 37499112-38245792) using samtools mpileup to bcftools call, followed by filtering to remove low quality. Results were loaded into the Ensembl VEP tool. A dot (“.”) indicates missing data and/or low quality. The reference allele and alternate alleles were obtained from the UCSC Genome Table Browser. <br> <strong>Supplemental Table 2. </strong>Differentially expressed genes (DEG) comparing AF to UN without exposure to ethanol. The table contains all output from DESeq2 results. Excel filters are set to show only genes significantly different (padj &lt;= 0.05) and at least 1.5-fold different (abs(log2FoldChange) &gt; 0.585). Release the filters to see the full list. <br> <strong>Supplemental Table 3. </strong>Enriched Gene Ontology-Biological Process (GO-BP) terms from genes increased in AF relative to UN. <br> <strong>Supplemental Table 4. </strong>Enriched Gene Ontology-Biological Process (GO-BP) terms from genes decreased in AF relative to UN.
DOI: 10.6084/m9.figshare.19798882.v1
title: KCNJ6 Supplemental Tables

description: Supplemental tables for Popova et al., " <strong>Alcohol reverses the effects of </strong><em><strong>KCNJ6</strong></em><strong> (GIRK2) noncoding variants on excitability of human glutamatergic neurons"</strong> <br> <strong>Supplemental Table 1. </strong>SNPs identified in RNAseq samples. Variants were identified in the region surrounding KCNJ6 (chr21: 37499112-38245792) using samtools mpileup to bcftools call, followed by filtering to remove low quality. Results were loaded into the Ensembl VEP tool. A dot (“.”) indicates missing data and/or low quality. The reference allele and alternate alleles were obtained from the UCSC Genome Table Browser. <br> <strong>Supplemental Table 2. </strong>Differentially expressed genes (DEG) comparing AF to UN without exposure to ethanol. The table contains all output from DESeq2 results. Excel filters are set to show only genes significantly different (padj &lt;= 0.05) and at least 1.5-fold different (abs(log2FoldChange) &gt; 0.585). Release the filters to see the full list. <br> <strong>Supplemental Table 3. </strong>Enriched Gene Ontology-Biological Process (GO-BP) terms from genes increased in AF relative to UN. <br> <strong>Supplemental Table 4. </strong>Enriched Gene Ontology-Biological Process (GO-BP) terms from genes decreased in AF relative to UN.
DOI: 10.34740/kaggle/dsv/4063562
title: Elon Musk Tweets
DOI: 10.5281/zenodo.6110278
title: Single-cell datasets for temporal gene expression integration

description: Contains loom files and preprocessed adata objects to compare methods for temporal gene expression integration. Loom files can be accessed using the 'read' function in Scvelo. Preprocessed adata objects can be accessed using the 'read_h5ad' function in Scanpy. The raw single-cell RNA sequencing datasets can be found under the following accession codes. Mouse embryonic cell cycle dataset from Ref. (https://doi.org/10.1038/nbt.3102) was originally downloaded from ArrayExpress with the accession code E-MTAB-2805 Hematopoiesis differentiation dataset from Ref. (https://doi.org/10.1182/blood-2016-05-716480) was originally downloaded from the Gene Expression Omnibus with the accession code GSE81682 NKT cell differentiation dataset from Ref. (https://doi.org/10.1038/ni.3437) was originally downloaded from the Gene Expression Omnibus with the accession code GSE74596. Hematopoiesis differentiation dataset from Ref. (https://doi.org/10.1038/nature19348) was originally downloaded from the Gene Expression Omnibus with the accession codes GSE70236, GSE70240, GSE70244 LPS stimulation dataset from Ref. (https://doi.org/10.1016/j.cels.2017.03.010) was originally downloaded from the Gene Expression Omnibus with the accession code GSE94383. INF-gamma stimulation dataset from Ref. (https://doi.org/10.1038/s41587-020-00803-5) was originally downloaded from the Gene Expression Omnibus with the accession code GSE161465. AML chemotherapy dataset from Ref. (https://doi.org/10.1038/s41591-018-0233-1) was originally downloaded from the Gene Expression Omnibus with the accession code GSE116481. AML diagnosis/relapse dataset from Ref. (https://doi.org/10.1038/s41375-021-01338-7) was originally downloaded from the Gene Expression Omnibus with the accession code GSE126068. MS case control PBMC and CSF datasets from Ref. (https://doi.org/10.1038/s41467-019-14118-w) was originally downloaded from the Gene Expression Omnibus with the accession code GSE138266.
DOI: 10.7284/141170
title: Multiplexed data collected during research cruise AE2015 using a BIOS METOC instrument system onboard the platform RV Atlantic Explorer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/141171
title: Multiplexed data collected during research cruise AE2015 using a BIOS METOC instrument system onboard the platform RV Atlantic Explorer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/141172
title: Multiplexed data collected during research cruise AE2015 using a BIOS METOC instrument system onboard the platform RV Atlantic Explorer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.5281/zenodo.6783086
title: Datasets accompanying 'Downscaling using Deep Convolutional Autoencoders, a case study for South East Asia'

description: Datasets required to replicate the experiment described in the paper: 'Downscaling using Deep Convolutional Autoencoders, a case study for South East Asia' This includes NetCDF files used to generate training data and processed climate data for model training, trained models and outputs from model predictions. It also contains the datasets used for comparisons (CORDEX and CMIP6)
DOI: 10.5281/zenodo.6783085
title: Datasets accompanying 'Downscaling using Deep Convolutional Autoencoders, a case study for South East Asia'

description: Datasets required to replicate the experiment described in the paper: 'Downscaling using Deep Convolutional Autoencoders, a case study for South East Asia' This includes NetCDF files used to generate training data and processed climate data for model training, trained models and outputs from model predictions. It also contains the datasets used for comparisons (CORDEX and CMIP6)
DOI: 10.7284/141174
title: Multiplexed data collected during research cruise AE2015 using a BIOS METOC instrument system onboard the platform RV Atlantic Explorer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/141169
title: ADCP (Acoustic Doppler Current Profiler) data collected during research cruise AE2015 using a Hawaii UHDAS instrument system onboard the platform RV Atlantic Explorer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/141173
title: CTD (Conductivity, Temperature, Depth) data collected during research cruise AE2015 using a SeaBird SBE-911+ instrument system onboard the platform RV Atlantic Explorer

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.6084/m9.figshare.16816081
title: Datasets for the manuscript "Pore-scale visualization and quantification of dissolution in microfluidic rough channels" by Zhou et al.

description: This is the dataset for the dissolution experiments in microfluidics.
DOI: 10.6084/m9.figshare.16816081.v4
title: Datasets for the manuscript "Pore-scale visualization and quantification of dissolution in microfluidic rough channels" by Zhou et al.

description: This is the dataset for the dissolution experiments in microfluidics.
DOI: 10.6084/m9.figshare.16816081.v3
title: Datasets for the manuscript "Pore-scale visualization and quantification of dissolution in microfluidic rough channels" by Zhou et al.

description: This is the dataset for the dissolution experiments in microfluidics.
DOI: 10.7910/dvn/xtejre/jpiys8
title: 911 Ecometrics BLK Longitudinal, Monthly, External.tab

description: CSV holding ecometric indices for Census Blocks across months. External version for public use.
DOI: 10.7910/dvn/xtejre/rb6jme
title: 911 Call Type Description 2010-14.tab

description: Descriptions of call types by year for 2010 - 2014 and annual call type frequencies for 2011.
DOI: 10.7910/dvn/xtejre/uhsoay
title: 911 Ecometrics BLK Longitudinal, Yearly, External.tab

description: CSV holding ecometric indices for Census Blocks across years. External version for public use.
DOI: 10.7910/dvn/xtejre/2luudx
title: 911 Call Type Description 2014-19.tab

description: Descriptions of call types by year for 2014 - 2019 and annual call type frequencies for 2015-2018.
DOI: 10.7910/dvn/xtejre/kvmuyt
title: 911 Ecometrics CBG Longitudinal, Yearly, External.tab

description: CSV holding ecometric indices for Census Block Groups across years. External version for public use.
DOI: 10.7910/dvn/xtejre/qwt9mf
title: 911 Ecometrics CBG Longitudinal, Monthly, External.tab

description: CSV holding ecometric indices for Census Block Groups across months. External version for public use.
DOI: 10.7910/dvn/xtejre/turkyo
title: 911 Ecometrics CT Longitudinal, Yearly, External.tab

description: CSV holding ecometric indices for Census Tracts across years. External version for public use.
DOI: 10.7910/dvn/xtejre/j44uy4
title: 911 Ecometrics CT Longitudinal, Monthly, External.tab

description: CSV holding ecometric indices for Census Tracts across months. External version for public use.
DOI: 10.7910/dvn/xtejre/to1fbj
title: 911 BLK Yearly Ecometrics 2010-2018 Shapefile.zip

description: Zipped shapefile holding ecometric indices for Census Blocks
DOI: 10.7910/dvn/xtejre/2lhezn
title: 911 Call Type Frequency by CT and Year.tab
DOI: 10.7910/dvn/xtejre/qgujrb
title: 911 CT Yearly Ecometrics 2010-2018 Shapefile.zip

description: Zipped shapefile holding ecometric indices for Census Tracts
DOI: 10.7910/dvn/xtejre/xsso8m
title: 911 CBG Yearly Ecometrics 2010-2018 Shapefile.zip

description: Zipped shapefile holding ecometric indices for Census Block Groups