cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.19959861.v1
title: Morphometric magnetic resonance imaging study of the quadriceps tendon in hemodialysis patients: comparison with non-dialyzed controls

description: Abstract Objective: To evaluate the knees of individuals with renal failure who are on hemodialysis, using magnetic resonance imaging (MRI), comparing them with those of a group of individuals with normal renal function. Materials and Methods: This was a cross-sectional, observational, controlled study conducted between August 2018 and February 2020. The cases consisted of 15 patients who had been on hemodialysis for ≥ 5 years and did not have a quadriceps tendon rupture. The controls consisted of 15 individuals with normal renal function who were matched (1:1) to the cases for sex, age, and physical activity level. The subjects in both groups underwent MRI of the right knee only. Results: The mean ages of the cases and controls were 50 ± 15 years and 49 ± 14 years, respectively. The median time on hemodialysis was 11 years (range, 10-14 years). Serum levels of parathyroid hormone, ferritin, alkaline phosphatase, phosphorus, and creatinine were higher among the cases than among the controls, whereas serum albumin and hemoglobin were lower (p &lt; 0.05 for all). The MRI study showed a hyperintense signal in the quadriceps tendon in 11 of the cases and in three of the controls (p = 0.009). Knee joint effusion was observed in nine of the cases and in three of the controls (p &lt; 0.05). The thickness, length, and width of the tendon did not differ between the groups. A hyperintense signal in the tendon was not associated with the time on hemodialysis; nor with the levels of intact parathyroid hormone, hemoglobin, or alkaline phosphatase. Conclusion: Patients on chronic hemodialysis, even those without a tendon rupture, show a hyperintense signal in the quadriceps tendon on MRI.
DOI: 10.6084/m9.figshare.19959861
title: Morphometric magnetic resonance imaging study of the quadriceps tendon in hemodialysis patients: comparison with non-dialyzed controls

description: Abstract Objective: To evaluate the knees of individuals with renal failure who are on hemodialysis, using magnetic resonance imaging (MRI), comparing them with those of a group of individuals with normal renal function. Materials and Methods: This was a cross-sectional, observational, controlled study conducted between August 2018 and February 2020. The cases consisted of 15 patients who had been on hemodialysis for ≥ 5 years and did not have a quadriceps tendon rupture. The controls consisted of 15 individuals with normal renal function who were matched (1:1) to the cases for sex, age, and physical activity level. The subjects in both groups underwent MRI of the right knee only. Results: The mean ages of the cases and controls were 50 ± 15 years and 49 ± 14 years, respectively. The median time on hemodialysis was 11 years (range, 10-14 years). Serum levels of parathyroid hormone, ferritin, alkaline phosphatase, phosphorus, and creatinine were higher among the cases than among the controls, whereas serum albumin and hemoglobin were lower (p &lt; 0.05 for all). The MRI study showed a hyperintense signal in the quadriceps tendon in 11 of the cases and in three of the controls (p = 0.009). Knee joint effusion was observed in nine of the cases and in three of the controls (p &lt; 0.05). The thickness, length, and width of the tendon did not differ between the groups. A hyperintense signal in the tendon was not associated with the time on hemodialysis; nor with the levels of intact parathyroid hormone, hemoglobin, or alkaline phosphatase. Conclusion: Patients on chronic hemodialysis, even those without a tendon rupture, show a hyperintense signal in the quadriceps tendon on MRI.
DOI: 10.6084/m9.figshare.19959862
title: Effect of the Surgical Safety Checklist on the incidence of adverse events: contributions from a national study

description: ABSTRACT Objective: the study evaluated the effect of using the safe surgery checklist (CL) on the incidence of adverse events (AE). Methods: cross-sectional and retrospective research with 851 patients undergoing surgical procedures in 2012 (n=428) and 2015 (n=423), representing the periods before and after CL implantation. The AE incidences for each year were estimated and compared. The association between the occurrence of AE and the presence of CL in the medical record was analyzed. Results: a reduction in the point estimate of AE was observed from 13.6% (before using the CL) to 11.8% (with the use of the CL). The difference between the proportions of AE in the periods before and after the use of CL was not significant (p=0.213). The occurrence of AE showed association with the following characteristics: anesthetic risk of the patient, length of stay, surgery time and classification of the procedure according to the potential for contamination. Considering the proportion of deaths, there was a significant reduction in deaths (p=0.007) in patients whose CL was used when compared to those without the use of the instrument. There was no significant association between the presence of CL and the occurrence of AE. It was concluded that the presence of CL in the medical record did not guarantee an expected reduction in the incidence of AE. Conclusion: however, it is believed that the use of the instrument integrated with other patient safety strategies can improve the safety/quality of surgical care in the long term.
DOI: 10.6084/m9.figshare.19959862.v1
title: Effect of the Surgical Safety Checklist on the incidence of adverse events: contributions from a national study

description: ABSTRACT Objective: the study evaluated the effect of using the safe surgery checklist (CL) on the incidence of adverse events (AE). Methods: cross-sectional and retrospective research with 851 patients undergoing surgical procedures in 2012 (n=428) and 2015 (n=423), representing the periods before and after CL implantation. The AE incidences for each year were estimated and compared. The association between the occurrence of AE and the presence of CL in the medical record was analyzed. Results: a reduction in the point estimate of AE was observed from 13.6% (before using the CL) to 11.8% (with the use of the CL). The difference between the proportions of AE in the periods before and after the use of CL was not significant (p=0.213). The occurrence of AE showed association with the following characteristics: anesthetic risk of the patient, length of stay, surgery time and classification of the procedure according to the potential for contamination. Considering the proportion of deaths, there was a significant reduction in deaths (p=0.007) in patients whose CL was used when compared to those without the use of the instrument. There was no significant association between the presence of CL and the occurrence of AE. It was concluded that the presence of CL in the medical record did not guarantee an expected reduction in the incidence of AE. Conclusion: however, it is believed that the use of the instrument integrated with other patient safety strategies can improve the safety/quality of surgical care in the long term.
DOI: 10.22008/fk2/sfvsbm/kr1ik2
title: Play_Maps_DCG_2020.mpk
DOI: 10.22008/fk2/sfvsbm
title: Replication data for Hydrocarbon Play Maps in the Danish Central Graben

description: Hydrocarbon play maps in the Danish North Sea. Maps in the format of: Arcgis package version 10.6
DOI: 10.34740/kaggle/dsv/3734439
title: AutoXGB
DOI: 10.6084/m9.figshare.19959900.v1
title: Additional file 1 of What complete mitochondrial genomes tell us about the evolutionary history of the black soldier fly, Hermetia illucens

description: Additional file 1. List of all individuals used during the study. Origin and accession number.
DOI: 10.6084/m9.figshare.19959900
title: Additional file 1 of What complete mitochondrial genomes tell us about the evolutionary history of the black soldier fly, Hermetia illucens

description: Additional file 1. List of all individuals used during the study. Origin and accession number.
DOI: 10.6084/m9.figshare.19959912.v1
title: Additional file 5 of What complete mitochondrial genomes tell us about the evolutionary history of the black soldier fly, Hermetia illucens

description: Additional file 5. Paired blasts of the mitochondrial genomes used in the study.
DOI: 10.6084/m9.figshare.19959912
title: Additional file 5 of What complete mitochondrial genomes tell us about the evolutionary history of the black soldier fly, Hermetia illucens

description: Additional file 5. Paired blasts of the mitochondrial genomes used in the study.
DOI: 10.5281/zenodo.6561491
title: Raw Data for "RASER MRI: Magnetic Resonance Images formed Spontaneously exploiting Cooperative Nonlinear Interaction"

description: This upload contains the raw data used for Fig. 3-5 in "RASER MRI: Magnetic Resonance Images formed Spontaneously exploiting Cooperative Nonlinear Interaction". Experimental conditions and details about the datasets are given in a "ReadMe.txt" file.
description: Consider using version 1.1 instead
description: {"references": ["Lehmkuhl, S\u00f6ren et.al (2022) Raw data associated to https://arxiv.org/abs/2203.00632"]}
DOI: 10.6084/m9.figshare.19959930
title: Additional file 2 of Open chromatin analysis in Trypanosoma cruzi life forms highlights critical differences in genomic compartments and developmental regulation at tDNA loci

description: Additional file 2: Table S1. Repetitive regions at T. cruzi genome. Table S2. Log2 RPGC at core and disruptive compartment. Table S3. RPGC at the indicated features in epimastigotes and metacyclics (Figure 4C). Table S4. RPGC at the indicated features in epimastigotes and metacyclics (Figure 4C). Table S4. Hierarchical clustering output from PTUs (with or without SSRs) and tDNA.
DOI: 10.6084/m9.figshare.19959930.v1
title: Additional file 2 of Open chromatin analysis in Trypanosoma cruzi life forms highlights critical differences in genomic compartments and developmental regulation at tDNA loci

description: Additional file 2: Table S1. Repetitive regions at T. cruzi genome. Table S2. Log2 RPGC at core and disruptive compartment. Table S3. RPGC at the indicated features in epimastigotes and metacyclics (Figure 4C). Table S4. RPGC at the indicated features in epimastigotes and metacyclics (Figure 4C). Table S4. Hierarchical clustering output from PTUs (with or without SSRs) and tDNA.
DOI: 10.6084/m9.figshare.19959933
title: Additional file 3 of Open chromatin analysis in Trypanosoma cruzi life forms highlights critical differences in genomic compartments and developmental regulation at tDNA loci

description: Additional file 3: Videos S1–S4. 3D reconstruction of euchromatin (yellow), heterochromatin (purple) and nucleolus (red) in epimastigotes, intermediate 1 and 2, and metacyclics.https://repositorio.butantan.gov.br/handle/butantan/3965.
DOI: 10.6084/m9.figshare.19959933.v1
title: Additional file 3 of Open chromatin analysis in Trypanosoma cruzi life forms highlights critical differences in genomic compartments and developmental regulation at tDNA loci

description: Additional file 3: Videos S1–S4. 3D reconstruction of euchromatin (yellow), heterochromatin (purple) and nucleolus (red) in epimastigotes, intermediate 1 and 2, and metacyclics.https://repositorio.butantan.gov.br/handle/butantan/3965.
DOI: 10.6084/m9.figshare.19768025
title: Additional file 2 of Genomic profiling of sporadic multiple meningiomas

description: Additional file 2. Supplementary Table 1: Cohort summary table with sequencing and clinical details.
DOI: 10.6084/m9.figshare.19768025.v2
title: Additional file 2 of Genomic profiling of sporadic multiple meningiomas

description: Additional file 2. Supplementary Table 1: Cohort summary table with sequencing and clinical details.
DOI: 10.6084/m9.figshare.19768025.v1
title: Additional file 2 of Genomic profiling of sporadic multiple meningiomas

description: Additional file 2. Supplementary Table 1: Cohort summary table with sequencing and clinical details.
DOI: 10.6084/m9.figshare.16943631.v1
title: Additional file 1 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 1.
DOI: 10.6084/m9.figshare.16943631
title: Additional file 1 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 1.
DOI: 10.6084/m9.figshare.16943634.v1
title: Additional file 2 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 2.
DOI: 10.6084/m9.figshare.16943640
title: Additional file 3 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 3.
DOI: 10.6084/m9.figshare.16943640.v1
title: Additional file 3 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 3.
DOI: 10.6084/m9.figshare.16943634
title: Additional file 2 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 2.
DOI: 10.6084/m9.figshare.16943643
title: Additional file 4 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 4.
DOI: 10.6084/m9.figshare.16943643.v1
title: Additional file 4 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 4.
DOI: 10.6084/m9.figshare.16943646
title: Additional file 5 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 5.
DOI: 10.6084/m9.figshare.16943646.v1
title: Additional file 5 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 5.
DOI: 10.6084/m9.figshare.16943649.v1
title: Additional file 6 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 6.
DOI: 10.6084/m9.figshare.16943649
title: Additional file 6 of Chlorpromazine induces cytotoxic autophagy in glioblastoma cells via endoplasmic reticulum stress and unfolded protein response

description: Additional file 6.
DOI: 10.6084/m9.figshare.19960011.v1
title: Additional file 3 of Facilitating safety evaluation in maternal immunization trials: a retrospective cohort study to assess pregnancy outcomes and events of interest in low-risk pregnancies in England

description: Additional file 3. Codes used to identify exclusion criteria for the low-risk cohort.
DOI: 10.6084/m9.figshare.19960011
title: Additional file 3 of Facilitating safety evaluation in maternal immunization trials: a retrospective cohort study to assess pregnancy outcomes and events of interest in low-risk pregnancies in England

description: Additional file 3. Codes used to identify exclusion criteria for the low-risk cohort.
DOI: 10.6084/m9.figshare.19960017
title: Additional file 5 of Facilitating safety evaluation in maternal immunization trials: a retrospective cohort study to assess pregnancy outcomes and events of interest in low-risk pregnancies in England

description: Additional file 5. Codes used to identify pregnancy outcomes and pregnancy-related events of interest.
DOI: 10.6084/m9.figshare.19960017.v1
title: Additional file 5 of Facilitating safety evaluation in maternal immunization trials: a retrospective cohort study to assess pregnancy outcomes and events of interest in low-risk pregnancies in England

description: Additional file 5. Codes used to identify pregnancy outcomes and pregnancy-related events of interest.
DOI: 10.6084/m9.figshare.19960026
title: Additional file 8 of Facilitating safety evaluation in maternal immunization trials: a retrospective cohort study to assess pregnancy outcomes and events of interest in low-risk pregnancies in England

description: Additional file 8. Codes used to define the variables.
DOI: 10.6084/m9.figshare.19960026.v1
title: Additional file 8 of Facilitating safety evaluation in maternal immunization trials: a retrospective cohort study to assess pregnancy outcomes and events of interest in low-risk pregnancies in England

description: Additional file 8. Codes used to define the variables.
DOI: 10.6084/m9.figshare.19960044
title: Additional file 10 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 10. Comparison of BRG1 and POGZ common target genes.
DOI: 10.6084/m9.figshare.19960044.v1
title: Additional file 10 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 10. Comparison of BRG1 and POGZ common target genes.
DOI: 10.6084/m9.figshare.19960050.v1
title: Additional file 2 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 2. GO terms of POGZ targets.
DOI: 10.6084/m9.figshare.19960053
title: Additional file 3 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 3. KEGG terms of POGZ targets.
DOI: 10.6084/m9.figshare.19960053.v1
title: Additional file 3 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 3. KEGG terms of POGZ targets.
DOI: 10.6084/m9.figshare.19960056.v1
title: Additional file 4 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 4. Comparison of POGZ, ADNP, CBX3 inetractors in ESCS.
DOI: 10.6084/m9.figshare.19960050
title: Additional file 2 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 2. GO terms of POGZ targets.
DOI: 10.6084/m9.figshare.19960056
title: Additional file 4 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 4. Comparison of POGZ, ADNP, CBX3 inetractors in ESCS.
DOI: 10.6084/m9.figshare.19960059
title: Additional file 5 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 5. Uncropped WB data images.
DOI: 10.6084/m9.figshare.19960059.v1
title: Additional file 5 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 5. Uncropped WB data images.
DOI: 10.6084/m9.figshare.19960062
title: Additional file 6 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 6. POGZ, ADNP and CHD4 co-bound sites and genes.
DOI: 10.6084/m9.figshare.19960065
title: Additional file 7 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 7. ATAC-seq and H3K27ac comparison between us and Markenscoff-Papadimitriou et al.
DOI: 10.6084/m9.figshare.19960062.v1
title: Additional file 6 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 6. POGZ, ADNP and CHD4 co-bound sites and genes.
DOI: 10.6084/m9.figshare.19960065.v1
title: Additional file 7 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 7. ATAC-seq and H3K27ac comparison between us and Markenscoff-Papadimitriou et al.
DOI: 10.6084/m9.figshare.19960068
title: Additional file 8 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 8. POGZ targets and down-regulated genes in Figure 4F.
DOI: 10.6084/m9.figshare.19960068.v1
title: Additional file 8 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 8. POGZ targets and down-regulated genes in Figure 4F.
DOI: 10.6084/m9.figshare.19960071.v1
title: Additional file 9 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 9. POGZ targets and up-regulated genes in Figure 4G.
DOI: 10.6084/m9.figshare.19960071
title: Additional file 9 of Autism-associated protein POGZ controls ESCs and ESC neural induction by association with esBAF

description: Additional file 9. POGZ targets and up-regulated genes in Figure 4G.
DOI: 10.6084/m9.figshare.16922764
title: Intra-genomic rRNA gene variability of Nassellaria and Spumellaria (Rhizaria, Radiolaria) assessed by Sanger, MinION and Illumina sequencing

description: Ribosomal DNA (rDNA) genes are known to be valuable markers for the barcoding of eukaryotic life and its phylogenetic classification at various taxonomic levels. The large scale exploration of environmental microbial diversity through metabarcoding approaches have been focused mainly on the hypervariable regions V4 and V9 of the 18S rDNA gene. Yet, the accurate interpretation of such environmental surveys is hampered by technical (e.g., PCR and sequencing errors) and biological biases (e.g., intra-genomic variability). Here we explored the intra-genomic diversity of Nassellaria and Spumellaria specimens (Radiolaria) by comparing Sanger sequencing with two different high-throughput sequencing platforms: Illumina and Oxford Nanopore Technologies (MinION). Our analysis determined that intra-genomic variability of Nassellaria and Spumellaria is generally low, yet in some Spumellaria specimens we found two different copies of the V4 with a similarity lower than 97%. From the different sequencing methods, Illumina showed the highest number of contaminations (i.e., environmental DNA, cross-contamination, tag-jumping), revealed by its high sequencing depth; and Minion showed the highest sequencing rate error (~14%). Yet the long reads produced by MinION (~2900 bp) allowed accurate phylogenetic reconstruction studies. These results, highlight the requirement for a careful interpretation of Illumina based metabarcoding studies, in particular regarding low abundant amplicons, and open future perspectives towards full environmental rDNA metabarcoding surveys.<br> <br> Here you can find 7 files gathering raw data obtained in this study and its associated metadata: <br> Data obtained by <strong>Sanger</strong> sequencing:<br> sanger_raw.fasta.gz: A fasta file containing the sequences with the following sequence names: "Cell-identifier_primer_barcode_well"; please note the underscore delimiting fields. <br> Data obtained by <strong>MinION (ONT)</strong> sequencing: minion_raw.fastq.gz: Fastq file. minion_raw.fast5.tar.gz: Fast5 native MinION raw data for future implementations or re-analysis. info_minion_demultiplex.txt: The information file for demultiplexing MinION data. <br> Data obtained by <strong>Illumina</strong> sequencing: R1.fastq.gz: R1 fastq file. R2.fastq.gz: R2 fastq file. info_illumina_tags_cutadapt.txt: The information file for demultiplexing Illumina data.<br> <br> For further details or if using these datasets, please see/cite the following study:<br> Sandin MM, Romac S, Not F (2022) <strong>Intra-genomic rRNA gene variability of Nassellaria and Spumellaria (Rhizaria, Radiolaria) assessed by Sanger, MinION and Illumina sequencing</strong>. <em>in press, Environmental Microbiology</em>; <strong>https://doi.org/10.1111/1462-2920.16081</strong><br>
DOI: 10.22008/fk2/ycqxtr/jn9tqq
title: 100006.nc
DOI: 10.22008/fk2/ycqxtr/6r8rvi
title: 100009.nc
DOI: 10.22008/fk2/ycqxtr/fhnw8t
title: 100010.nc
DOI: 10.22008/fk2/ycqxtr/vfthf5
title: 100011.nc
DOI: 10.22008/fk2/ycqxtr/yfnek0
title: 100013.nc
DOI: 10.22008/fk2/ycqxtr/m80ub1
title: 100014.nc