cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.14483049
title: Comparison of outcomes of peritoneal dialysis between patients after failed kidney transplant and transplant-naïve patients: a meta-analysis of observational studies

description: The influence of prior failed kidney transplants on outcomes of peritoneal dialysis (PD) is unclear. Thus, we conducted a systematic review and meta-analysis to compare the outcomes of patients initiating PD after a failed kidney transplant with those initiating PD without a prior history of kidney transplantation. We searched PubMed, Embase, CENTRAL, and Google Scholar databases from inception until 25 November 2020. Our meta-analysis considered the absolute number of events of mortality, technical failures, and patients with peritonitis, and we also pooled multi-variable adjusted hazard ratios (HR). We included 12 retrospective studies. For absolute number of events, our analysis indicated no statistically significant difference in technique failure [RR, 1.14; 95% CI, 0.80–1.61; I<sup>2</sup>=52%; <i>p</i> = 0.48], number of patients with peritonitis [RR, 1.13; 95% CI, 0.97–1.32; I<sup>2</sup>=5%; <i>p</i> = 0.11] and mortality [RR, 1.00; 95% CI, 0.67–1.50; I<sup>2</sup>=63%; <i>p</i> = 0.99] between the study groups. The pooled analysis of adjusted HRs indicated no statistically significant difference in the risk of technique failure [HR, 1.25; 95% CI, 0.88–1.78; I<sup>2</sup>=79%; <i>p</i> = 0.22], peritonitis [HR, 1.04; 95% CI, 0.72–1.50; I<sup>2</sup>=76%; <i>p</i> = 0.85] and mortality [HR, 1.24; 95% CI, 0.77–2.00; I<sup>2</sup>=66%; <i>p</i> = 0.38] between the study groups. Patients with kidney transplant failure initiating PD do not have an increased risk of mortality, technique failure, or peritonitis as compared to transplant-naïve patients initiating PD. Further studies are needed to evaluate the impact of prior and ongoing immunosuppression on PD outcomes.
DOI: 10.6084/m9.figshare.14483049.v1
title: Comparison of outcomes of peritoneal dialysis between patients after failed kidney transplant and transplant-naïve patients: a meta-analysis of observational studies

description: The influence of prior failed kidney transplants on outcomes of peritoneal dialysis (PD) is unclear. Thus, we conducted a systematic review and meta-analysis to compare the outcomes of patients initiating PD after a failed kidney transplant with those initiating PD without a prior history of kidney transplantation. We searched PubMed, Embase, CENTRAL, and Google Scholar databases from inception until 25 November 2020. Our meta-analysis considered the absolute number of events of mortality, technical failures, and patients with peritonitis, and we also pooled multi-variable adjusted hazard ratios (HR). We included 12 retrospective studies. For absolute number of events, our analysis indicated no statistically significant difference in technique failure [RR, 1.14; 95% CI, 0.80–1.61; I<sup>2</sup>=52%; <i>p</i> = 0.48], number of patients with peritonitis [RR, 1.13; 95% CI, 0.97–1.32; I<sup>2</sup>=5%; <i>p</i> = 0.11] and mortality [RR, 1.00; 95% CI, 0.67–1.50; I<sup>2</sup>=63%; <i>p</i> = 0.99] between the study groups. The pooled analysis of adjusted HRs indicated no statistically significant difference in the risk of technique failure [HR, 1.25; 95% CI, 0.88–1.78; I<sup>2</sup>=79%; <i>p</i> = 0.22], peritonitis [HR, 1.04; 95% CI, 0.72–1.50; I<sup>2</sup>=76%; <i>p</i> = 0.85] and mortality [HR, 1.24; 95% CI, 0.77–2.00; I<sup>2</sup>=66%; <i>p</i> = 0.38] between the study groups. Patients with kidney transplant failure initiating PD do not have an increased risk of mortality, technique failure, or peritonitis as compared to transplant-naïve patients initiating PD. Further studies are needed to evaluate the impact of prior and ongoing immunosuppression on PD outcomes.
DOI: 10.6084/m9.figshare.17104549
title: Can warnings curb the spread of fake news? The interplay between warning, trust and confirmation bias

description: Despite attempts by social media companies to curb the spread of fake news with warnings flagging news credibility, the effectiveness of such measures remains unclear. Through the lens of the cognitive dissonance theory and individuals’ trust in the news, this study develops a theoretical model that explains why and how warnings affect an individual’s intention to share fake news. The study empirically assesses the predicted relationships using experimental survey data from 382 individuals. The findings provide evidence for two processes that underlie the effectiveness of warnings in curbing fake news sharing: (1) warnings negatively impact intention to share fake news through the psychological mechanism of lowering people’s cognitive and emotional trust in the news and (2) warnings mitigate the impact of cognitive trust on intention to share fake news. Confirmation bias is found to serve as a boundary condition for the effectiveness of warnings in lowering individuals’ cognitive and emotional trust in the news and in reducing the impacts of trust on an individual’s intention to share fake news.
DOI: 10.6084/m9.figshare.17104549.v1
title: Can warnings curb the spread of fake news? The interplay between warning, trust and confirmation bias

description: Despite attempts by social media companies to curb the spread of fake news with warnings flagging news credibility, the effectiveness of such measures remains unclear. Through the lens of the cognitive dissonance theory and individuals’ trust in the news, this study develops a theoretical model that explains why and how warnings affect an individual’s intention to share fake news. The study empirically assesses the predicted relationships using experimental survey data from 382 individuals. The findings provide evidence for two processes that underlie the effectiveness of warnings in curbing fake news sharing: (1) warnings negatively impact intention to share fake news through the psychological mechanism of lowering people’s cognitive and emotional trust in the news and (2) warnings mitigate the impact of cognitive trust on intention to share fake news. Confirmation bias is found to serve as a boundary condition for the effectiveness of warnings in lowering individuals’ cognitive and emotional trust in the news and in reducing the impacts of trust on an individual’s intention to share fake news.
DOI: 10.6084/m9.figshare.17104595
title: Mass-wasting-inferred Variability of 130,000-year Indian Summer Monsoon Intensity from Deposits in the Southeast Tibetan Plateau

description: A large proportion (~80%)
of the Indian subcontinent’s precipitation comes from the Indian summer monsoon
(ISM), which influences one-fifth of the world’s population. A long-term
reliable proxy for ISM is fundamental to understanding previous global climate
change. We establish a mass-wasting-inferred proxy to examine the paleohydrogeology (river undercutting history) of
the Southeast Tibetan Plateau and reconstruct the variability of ISM intensity
(precipitation) in the past 130,000 years. Our data suggest that mass-wasting
events, which provides us sufficient samples for paleoclimate research, are
prone to dramatic climate changes, especially extreme climate environments. The
Southeast Tibetan Plateau was subjected to at least four distinct ISM intensity
phases in the past 130,000 years. We conclude that ISM intensity has a
cyclicity featured by the Earth’s orbit with obliquity, and that ISM intensity transformation lags
the global ice volume (sea level) change by 8–15 kyrs.
DOI: 10.6084/m9.figshare.22347528
title: Integrative exploration of the mutual gene signatures and immune microenvironment between benign prostate hyperplasia and castration-resistant prostate cancer

description: Benign prostate hyperplasia (BPH) and prostate cancer (CaP) are among the most frequently occurring prostatic diseases. When CaP progressed to castration-resistant CaP (CRPC), the prognosis is poor. Although CaP/CRPC and BPH frequently coexist in prostate, the inter-relational mechanism between them is largely unknown. Single-cell RNA sequencing, bulk-RNA sequencing, and microarray data of BPH, CaP in the Gene Expression Omnibus database were obtained and comprehensively analyzed. Weighted Gene Co-Expression Network Analysis (WGCNA) and lasso regression analysis were performed to explore the potential biomarkers. With WGCNA, five modules in BPH, two in CaP, and three in CRPC were identified as significant modules. Pathway enrichment analysis found that the epigenetics and chromosomal-related signaling were dominantly clustered in the CaP group but not in BPH and CRPC. Lasso regression analysis was used to analyze further the mutual genes between the BPH module and the CRPC module. As a result, DDA1, ERG28, OGFOD1, and OXA1L were significantly correlated with the transcriptomic features in both BPH and CRPC. More importantly, the role of the four gene signatures was validated in two independent anti-PD-1 immunotherapy cohort. This study revealed the shared gene signatures and immune microenvironment between BPH and CRPC. The identified hub genes, including DDA1, ERG28, OGFOD1, and OXA1L, might be potential therapeutic targets for facilitating immunotherapy in prostate cancer.
DOI: 10.6084/m9.figshare.22347528.v1
title: Integrative exploration of the mutual gene signatures and immune microenvironment between benign prostate hyperplasia and castration-resistant prostate cancer

description: Benign prostate hyperplasia (BPH) and prostate cancer (CaP) are among the most frequently occurring prostatic diseases. When CaP progressed to castration-resistant CaP (CRPC), the prognosis is poor. Although CaP/CRPC and BPH frequently coexist in prostate, the inter-relational mechanism between them is largely unknown. Single-cell RNA sequencing, bulk-RNA sequencing, and microarray data of BPH, CaP in the Gene Expression Omnibus database were obtained and comprehensively analyzed. Weighted Gene Co-Expression Network Analysis (WGCNA) and lasso regression analysis were performed to explore the potential biomarkers. With WGCNA, five modules in BPH, two in CaP, and three in CRPC were identified as significant modules. Pathway enrichment analysis found that the epigenetics and chromosomal-related signaling were dominantly clustered in the CaP group but not in BPH and CRPC. Lasso regression analysis was used to analyze further the mutual genes between the BPH module and the CRPC module. As a result, DDA1, ERG28, OGFOD1, and OXA1L were significantly correlated with the transcriptomic features in both BPH and CRPC. More importantly, the role of the four gene signatures was validated in two independent anti-PD-1 immunotherapy cohort. This study revealed the shared gene signatures and immune microenvironment between BPH and CRPC. The identified hub genes, including DDA1, ERG28, OGFOD1, and OXA1L, might be potential therapeutic targets for facilitating immunotherapy in prostate cancer.
DOI: 10.6084/m9.figshare.12910347
title: Additional file 3 of Neuroinvasive Listeria monocytogenes infection triggers accumulation of brain CD8+ tissue-resident memory T cells in a miR-155-dependent fashion

description: Additional file 3: Supplemental data file 1.
DOI: 10.6084/m9.figshare.12910347.v1
title: Additional file 3 of Neuroinvasive Listeria monocytogenes infection triggers accumulation of brain CD8+ tissue-resident memory T cells in a miR-155-dependent fashion

description: Additional file 3: Supplemental data file 1.
DOI: 10.6084/m9.figshare.17104657
title: Efficacy and safety of thrombopoietin receptor agonists in the treatment of thrombocytopenia after hematopoietic stem cell transplantation: a meta-analysis and systematic review

description: Thrombocytopenia is a tough complication after hematopoietic stem cell transplantation (HSCT) with elusive pathogenesis and lack of well-established therapies. Thrombopoietin receptor agonists (TPO-RAs) have been used for thrombocytopenia post HSCT in recent years, but the outcomes remain debatable. We conducted this meta-analysis and systematic-review to evaluate the efficacy and safety of TPO-RAs for platelet recovery after HSCT. We searched PubMed, EMBASE, and Cochrane databases for studies on the application of TPO-RAs (eltrombopag and romiplostim) in the settings of primary or secondary thrombocytopenia after HSCT by 17 March 2021. Efficacy outcomes included response rate and survival rate, and adverse events were also evaluated. A total of 19 studies involving 378 patients were included. The pooled response rate was 73% (95%CI: 68–78%), which was significantly higher than recombinant human thrombopoietin (rhTPO) (27.8%). The pooled survival rate was 66% (95%CI: 54–77%), and infection was found to be the main cause of death. In addition, the pooled rate of adverse events was 3% (95%CI: 1–7%), with no severe adverse events reported. TPO-RAs could effectively and safely promote the recovery of platelets in patients after HSCT.
DOI: 10.6084/m9.figshare.17104657.v1
title: Efficacy and safety of thrombopoietin receptor agonists in the treatment of thrombocytopenia after hematopoietic stem cell transplantation: a meta-analysis and systematic review

description: Thrombocytopenia is a tough complication after hematopoietic stem cell transplantation (HSCT) with elusive pathogenesis and lack of well-established therapies. Thrombopoietin receptor agonists (TPO-RAs) have been used for thrombocytopenia post HSCT in recent years, but the outcomes remain debatable. We conducted this meta-analysis and systematic-review to evaluate the efficacy and safety of TPO-RAs for platelet recovery after HSCT. We searched PubMed, EMBASE, and Cochrane databases for studies on the application of TPO-RAs (eltrombopag and romiplostim) in the settings of primary or secondary thrombocytopenia after HSCT by 17 March 2021. Efficacy outcomes included response rate and survival rate, and adverse events were also evaluated. A total of 19 studies involving 378 patients were included. The pooled response rate was 73% (95%CI: 68–78%), which was significantly higher than recombinant human thrombopoietin (rhTPO) (27.8%). The pooled survival rate was 66% (95%CI: 54–77%), and infection was found to be the main cause of death. In addition, the pooled rate of adverse events was 3% (95%CI: 1–7%), with no severe adverse events reported. TPO-RAs could effectively and safely promote the recovery of platelets in patients after HSCT.
DOI: 10.6084/m9.figshare.12910359
title: Additional file 1 of Alteration of plasma metabolites associated with chemoradiosensitivity in esophageal squamous cell carcinoma via untargeted metabolomics approach

description: Additional file 1.
DOI: 10.6084/m9.figshare.12910359.v1
title: Additional file 1 of Alteration of plasma metabolites associated with chemoradiosensitivity in esophageal squamous cell carcinoma via untargeted metabolomics approach

description: Additional file 1.
DOI: 10.6084/m9.figshare.21298979
title: Additional file 2 of Screening of differentially expressed microRNAs and target genes in two potato varieties under nitrogen stress

description: Additional file 2: Figure 1. Analysis of variability among groups of differential miRNAsHN: excessive N application; LN: no N application; Y: Yanshu4; D: Atlantic;S: seedling stage; A: budding stage (A) LN_YLSvsLN_DLS (B) LN_YLAvsLN_DLA (C) HN_YLSvsHN_DLS. (D)HN_YLAvsHN_DLA (E) HN_DLSvsLN_DLS (F) HN_DLAvsLN_DLA. (G) HN_YLSvsLN_YLS (H)HN_YLAvsLN_YLA (I) LN_DLAvsLN_DLS. (J)HN_DLAvsHN_DLS (K)LN_YLAvsLN_YLS (L)HN_YLAvsHN_YLS.(M)LN_YRSvsLN_DRS (N)LN_YRAvsLN_DRA (O)HN_YRSvsHN_DRS. (P)HN_YRAvsHN_DRA (Q)HN_DRSvsLN_DRS(R)HN_DRAvsLN_DRA. (S)HN_YRSvsLN_YRS (T)HN_YRAvsLN_YRA (U)LN_DRAvsLN_DRS. (V)HN_DRAvsHN_DRS(W)LN_YRAvsLN_YRS (X)HN_YRAvsHN_YRS. Using the values of log2(fold change) as the horizontalcoordinate and -log10(p-value) as the vertical coordinate, volcanoplots were constructed for all miRNAs during differential expression analysis.The horizontal coordinate represents the fold change in the differentialexpression of miRNAs in different samples, and the vertical coordinaterepresents the statistical significance of the difference in the change inexpression levels of miRNAs. The red color represents up-regulatedsignificantly differentially expressed genes, the blue color representsdown-regulated significantly differentially expressed genes, and the grey dots representnon-significant differentially expressed genes. Figure 2. Comparison between groups of differential miRNAs. HN: excess N; LN:no N; Y: Yanshu4; D: Atlantic; S: seedling stage; A: budding stage. Thehorizontal coordinates indicate the data obtained after the comparison ofgroups, and the vertical coordinates indicate the number of up- anddown-regulated miRNAs. The red color represents up-regulated miRNAs, the bluecolor represents down-regulated miRNAs, and the numbers represent the number ofup- and down-regulated miRNAs. Figure 3. Results of clustering analysis ofdifferential miRNAs. HN: excess N; LN: no N; Y: Yanshu4; D: Atlantic; S: seedling;A: bud onset. (A)LN_YLSvsLN_DLS (B)LN_YLAvsLN_DLA (C)HN_YLSvsHN_DLS. (D)HN_YLAvsHN_DL.(E)HN_DLSvsLN_DLS (F)HN_DLAvsLN_DLA. (G)HN_YLSvsLN_YLS(H)HN_YLAvsLN_YLA (I)LN_DLAvsLN_DLS.(J)HN_DLAvsHN_DLS (K)LN_YLAvsLN_YLS (L)HN_YLAvsHN_YLS. (M)LN_YRSvsLN_DRS (N)LN_YRAvsLN_DRA(O)HN_YRSvsHN_DRS. (P)HN_YRAvsHN_DRA (Q)HN_DRSvsLN_DRS (R)HN_DRAvsLN_DRA. (S)HN_YRSvsLN_YR.(T)HN_YRAvsLN_YRA (U)LN_DRAvsLN_DRS. (V)HN_DRAvsHN_DRS (W)LN_YRAvsLN_YRS (X)HN_YRAvsHN_YRS
DOI: 10.6084/m9.figshare.21298979.v1
title: Additional file 2 of Screening of differentially expressed microRNAs and target genes in two potato varieties under nitrogen stress

description: Additional file 2: Figure 1. Analysis of variability among groups of differential miRNAsHN: excessive N application; LN: no N application; Y: Yanshu4; D: Atlantic;S: seedling stage; A: budding stage (A) LN_YLSvsLN_DLS (B) LN_YLAvsLN_DLA (C) HN_YLSvsHN_DLS. (D)HN_YLAvsHN_DLA (E) HN_DLSvsLN_DLS (F) HN_DLAvsLN_DLA. (G) HN_YLSvsLN_YLS (H)HN_YLAvsLN_YLA (I) LN_DLAvsLN_DLS. (J)HN_DLAvsHN_DLS (K)LN_YLAvsLN_YLS (L)HN_YLAvsHN_YLS.(M)LN_YRSvsLN_DRS (N)LN_YRAvsLN_DRA (O)HN_YRSvsHN_DRS. (P)HN_YRAvsHN_DRA (Q)HN_DRSvsLN_DRS(R)HN_DRAvsLN_DRA. (S)HN_YRSvsLN_YRS (T)HN_YRAvsLN_YRA (U)LN_DRAvsLN_DRS. (V)HN_DRAvsHN_DRS(W)LN_YRAvsLN_YRS (X)HN_YRAvsHN_YRS. Using the values of log2(fold change) as the horizontalcoordinate and -log10(p-value) as the vertical coordinate, volcanoplots were constructed for all miRNAs during differential expression analysis.The horizontal coordinate represents the fold change in the differentialexpression of miRNAs in different samples, and the vertical coordinaterepresents the statistical significance of the difference in the change inexpression levels of miRNAs. The red color represents up-regulatedsignificantly differentially expressed genes, the blue color representsdown-regulated significantly differentially expressed genes, and the grey dots representnon-significant differentially expressed genes. Figure 2. Comparison between groups of differential miRNAs. HN: excess N; LN:no N; Y: Yanshu4; D: Atlantic; S: seedling stage; A: budding stage. Thehorizontal coordinates indicate the data obtained after the comparison ofgroups, and the vertical coordinates indicate the number of up- anddown-regulated miRNAs. The red color represents up-regulated miRNAs, the bluecolor represents down-regulated miRNAs, and the numbers represent the number ofup- and down-regulated miRNAs. Figure 3. Results of clustering analysis ofdifferential miRNAs. HN: excess N; LN: no N; Y: Yanshu4; D: Atlantic; S: seedling;A: bud onset. (A)LN_YLSvsLN_DLS (B)LN_YLAvsLN_DLA (C)HN_YLSvsHN_DLS. (D)HN_YLAvsHN_DL.(E)HN_DLSvsLN_DLS (F)HN_DLAvsLN_DLA. (G)HN_YLSvsLN_YLS(H)HN_YLAvsLN_YLA (I)LN_DLAvsLN_DLS.(J)HN_DLAvsHN_DLS (K)LN_YLAvsLN_YLS (L)HN_YLAvsHN_YLS. (M)LN_YRSvsLN_DRS (N)LN_YRAvsLN_DRA(O)HN_YRSvsHN_DRS. (P)HN_YRAvsHN_DRA (Q)HN_DRSvsLN_DRS (R)HN_DRAvsLN_DRA. (S)HN_YRSvsLN_YR.(T)HN_YRAvsLN_YRA (U)LN_DRAvsLN_DRS. (V)HN_DRAvsHN_DRS (W)LN_YRAvsLN_YRS (X)HN_YRAvsHN_YRS
DOI: 10.6084/m9.figshare.21298985
title: Additional file 4 of Screening of differentially expressed microRNAs and target genes in two potato varieties under nitrogen stress

description: Additional file 4: Figure 1. Construction of therecombinant plasmid pCAMBIA1300-LUC-NiR for the luciferase reporter vector. Figure 2. Construction of the recombinant plasmid pCAMBIA1300-35S-396-5p for theluciferase localization report vector.
DOI: 10.6084/m9.figshare.21298985.v1
title: Additional file 4 of Screening of differentially expressed microRNAs and target genes in two potato varieties under nitrogen stress

description: Additional file 4: Figure 1. Construction of therecombinant plasmid pCAMBIA1300-LUC-NiR for the luciferase reporter vector. Figure 2. Construction of the recombinant plasmid pCAMBIA1300-35S-396-5p for theluciferase localization report vector.
DOI: 10.6084/m9.figshare.20250417
title: Additional file 1 of Clinical features and recurrence of Corynebacterium kroppenstedtii infection in patients with mastitis

description: Additional file 1. Corynebacterium kroppenstedtii database and Please insert into the section of Data availability.
DOI: 10.6084/m9.figshare.20250417.v1
title: Additional file 1 of Clinical features and recurrence of Corynebacterium kroppenstedtii infection in patients with mastitis

description: Additional file 1. Corynebacterium kroppenstedtii database and Please insert into the section of Data availability.
DOI: 10.6084/m9.figshare.12910401
title: Additional file 10 of Comparative chloroplast genome analyses of Avena: insights into evolutionary dynamics and phylogeny

description: Additional file 10: Table S10. Codon usage preference of all protein coding genes for eleven Avena plastomes calculated by http://www.bioinformatics.org/sms2/codon_usage . a Codon numbers of 20 amino acids and stop codons. b Codon number percent (%) of 20 amino acids and stop codons
DOI: 10.6084/m9.figshare.12910401.v1
title: Additional file 10 of Comparative chloroplast genome analyses of Avena: insights into evolutionary dynamics and phylogeny

description: Additional file 10: Table S10. Codon usage preference of all protein coding genes for eleven Avena plastomes calculated by http://www.bioinformatics.org/sms2/codon_usage . a Codon numbers of 20 amino acids and stop codons. b Codon number percent (%) of 20 amino acids and stop codons
DOI: 10.6084/m9.figshare.12910404
title: Additional file 11 of Comparative chloroplast genome analyses of Avena: insights into evolutionary dynamics and phylogeny

description: Additional file 11: Table S11. The relative synonymous codon usage (RSCU) values for eleven Avena species computed by CodonW v.1.4.2 [38]. a RSCU values. b RSCU values subtracted by 1.00 (the expected value if no codon bias).
DOI: 10.6084/m9.figshare.12910404.v1
title: Additional file 11 of Comparative chloroplast genome analyses of Avena: insights into evolutionary dynamics and phylogeny

description: Additional file 11: Table S11. The relative synonymous codon usage (RSCU) values for eleven Avena species computed by CodonW v.1.4.2 [38]. a RSCU values. b RSCU values subtracted by 1.00 (the expected value if no codon bias).
DOI: 10.6084/m9.figshare.20250438
title: Additional file 1 of Identification of prognostic stemness biomarkers in colon adenocarcinoma drug resistance

description: Additional file 1: Supplementary Table 1. Stemness Indices Derived for All PanCancer 33 TCGA Cohort.
DOI: 10.6084/m9.figshare.20250438.v1
title: Additional file 1 of Identification of prognostic stemness biomarkers in colon adenocarcinoma drug resistance

description: Additional file 1: Supplementary Table 1. Stemness Indices Derived for All PanCancer 33 TCGA Cohort.
DOI: 10.6084/m9.figshare.20250441
title: Additional file 2 of Identification of prognostic stemness biomarkers in colon adenocarcinoma drug resistance

description: Additional file 2: Supplementary Table 2. The differentially expressed genes of tumor, stage, mRNAsi and chemoresistance.
DOI: 10.6084/m9.figshare.20250441.v1
title: Additional file 2 of Identification of prognostic stemness biomarkers in colon adenocarcinoma drug resistance

description: Additional file 2: Supplementary Table 2. The differentially expressed genes of tumor, stage, mRNAsi and chemoresistance.