cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.8152751
title: Barrier Analysis survey data on handwashing with soap among Internally Displaced People in two camps in Iraq

description: This is a Barrier Analysis dataset. Barrier Analysis is a rapid assessment tool which is part of the Designing for Behaviour Change Framework. The Barrier Analysis approach is intended to be used in advance of designing a behaviour change programme. It allows programme designers to identify key barriers and determinants of behaviour which can then be used to develop strategies for behaviour change. This dataset captures the responses of 90 internally displaced people living in camps in the Kurdistan Region of Iraq. The behaviour of interest is handwashing with soap at critical times. <br>
DOI: 10.6084/m9.figshare.8152751.v1
title: Barrier Analysis survey data on handwashing with soap among Internally Displaced People in two camps in Iraq

description: This is a Barrier Analysis dataset. Barrier Analysis is a rapid assessment tool which is part of the Designing for Behaviour Change Framework. The Barrier Analysis approach is intended to be used in advance of designing a behaviour change programme. It allows programme designers to identify key barriers and determinants of behaviour which can then be used to develop strategies for behaviour change. This dataset captures the responses of 90 internally displaced people living in camps in the Kurdistan Region of Iraq. The behaviour of interest is handwashing with soap at critical times. <br>
DOI: 10.6084/m9.figshare.8152733
title: Assessing copper fertility of intrusive rocks using field portable X-Ray fluorescence (pXRF) data

description: Appendix B. Raw and ICP-calibrated pXRF data.
DOI: 10.6084/m9.figshare.8152733.v1
title: Assessing copper fertility of intrusive rocks using field portable X-Ray fluorescence (pXRF) data

description: Appendix B. Raw and ICP-calibrated pXRF data.
DOI: 10.6084/m9.figshare.8152730
title: Assessing copper fertility of intrusive rocks using field portable X-Ray fluorescence (pXRF) data

description: Appendix A. pXRF and ICP Standards Data.
DOI: 10.6084/m9.figshare.8152730.v1
title: Assessing copper fertility of intrusive rocks using field portable X-Ray fluorescence (pXRF) data

description: Appendix A. pXRF and ICP Standards Data.
DOI: 10.6084/m9.figshare.8152724
title: A New Genetic Based Hyper-heuristic Algorithm for Multitask- and Uncertain-Oriented Service Composition in Cloud Manufacturing

description: This is the simulation data used in the article "A New Genetic Based Hyper-heuristic Algorithm for Multitask- and Uncertain-Oriented Service Composition in Cloud Manufacturing"
DOI: 10.6084/m9.figshare.8152724.v1
title: A New Genetic Based Hyper-heuristic Algorithm for Multitask- and Uncertain-Oriented Service Composition in Cloud Manufacturing

description: This is the simulation data used in the article "A New Genetic Based Hyper-heuristic Algorithm for Multitask- and Uncertain-Oriented Service Composition in Cloud Manufacturing"
DOI: 10.6084/m9.figshare.8152652
title: Supplementary Material for: Hypoxia-Ischemia and Hypothermia Independently and Interactively Affect Neuronal Pathology in Neonatal Piglets with Short-Term Recovery

description: Therapeutic hypothermia is the standard of clinical care for moderate neonatal hypoxic-ischemic encephalopathy. We investigated the independent and interactive effects of hypoxia-ischemia (HI) and temperature on neuronal survival and injury in basal ganglia and cerebral cortex in neonatal piglets. Male piglets were randomized to receive HI injury or sham procedure followed by 29 h of normothermia, sustained hypothermia induced at 2 h, or hypothermia with rewarming during fentanyl-nitrous oxide anesthesia. Viable and injured neurons and apoptotic profiles were counted in the anterior putamen, posterior putamen, and motor cortex at 29 h after HI injury or sham procedure. Terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL) identified genomic DNA fragmentation to confirm cell death. Though hypothermia after HI preserved viable neurons in the anterior and posterior putamen, hypothermia prevented neuronal injury in only the anterior putamen. Hypothermia initiated 2 h after injury did not protect against apoptotic cell death in either the putamen or motor cortex, and rewarming from hypothermia was associated with increased apoptosis in the motor cortex. In non-HI shams, sustained hypothermia during anesthesia was associated with neuronal injury and corresponding viable neuron loss in the anterior putamen and motor cortex. TUNEL confirmed increased neurodegeneration in the putamen of hypothermic shams. Anesthetized, normothermic shams did not show abnormal neuronal cytopathology in the putamen or motor cortex, thereby demonstrating minimal contribution of the anesthetic regimen to neuronal injury during normothermia. We conclude that the efficacy of hypothermic protection after HI is region specific and that hypothermia during anesthesia in the absence of HI may be associated with neuronal injury in the developing brain. Studies examining the potential interactions between hypothermia and anesthesia, as well as with longer durations of hypothermia, are needed.
DOI: 10.6084/m9.figshare.8152652.v1
title: Supplementary Material for: Hypoxia-Ischemia and Hypothermia Independently and Interactively Affect Neuronal Pathology in Neonatal Piglets with Short-Term Recovery

description: Therapeutic hypothermia is the standard of clinical care for moderate neonatal hypoxic-ischemic encephalopathy. We investigated the independent and interactive effects of hypoxia-ischemia (HI) and temperature on neuronal survival and injury in basal ganglia and cerebral cortex in neonatal piglets. Male piglets were randomized to receive HI injury or sham procedure followed by 29 h of normothermia, sustained hypothermia induced at 2 h, or hypothermia with rewarming during fentanyl-nitrous oxide anesthesia. Viable and injured neurons and apoptotic profiles were counted in the anterior putamen, posterior putamen, and motor cortex at 29 h after HI injury or sham procedure. Terminal deoxynucleotidyl transferase dUTP nick end labeling (TUNEL) identified genomic DNA fragmentation to confirm cell death. Though hypothermia after HI preserved viable neurons in the anterior and posterior putamen, hypothermia prevented neuronal injury in only the anterior putamen. Hypothermia initiated 2 h after injury did not protect against apoptotic cell death in either the putamen or motor cortex, and rewarming from hypothermia was associated with increased apoptosis in the motor cortex. In non-HI shams, sustained hypothermia during anesthesia was associated with neuronal injury and corresponding viable neuron loss in the anterior putamen and motor cortex. TUNEL confirmed increased neurodegeneration in the putamen of hypothermic shams. Anesthetized, normothermic shams did not show abnormal neuronal cytopathology in the putamen or motor cortex, thereby demonstrating minimal contribution of the anesthetic regimen to neuronal injury during normothermia. We conclude that the efficacy of hypothermic protection after HI is region specific and that hypothermia during anesthesia in the absence of HI may be associated with neuronal injury in the developing brain. Studies examining the potential interactions between hypothermia and anesthesia, as well as with longer durations of hypothermia, are needed.
DOI: 10.6084/m9.figshare.11859216
title: Extended CRISPRi data for S4 Table

description: This file includes extended CRISPRi data for "High-throughput mapping of the phage resistance landscape in E. coli" by Mutalik et al., 2020
DOI: 10.6084/m9.figshare.11859216.v3
title: Extended CRISPRi data for S4 Table

description: This file includes extended CRISPRi data for "High-throughput mapping of the phage resistance landscape in E. coli" by Mutalik et al., 2020
DOI: 10.6084/m9.figshare.8152622
title: Severity of hot flushes is inversely associated with dietary intake of vitamin B<sub>6</sub> and oily fish

description: <b>Objectives:</b> This study investigated the links between the severity of vasomotor symptoms (VMS) and the dietary consumption of a variety of nutrients. <b>Method:</b> A cross-sectional analysis of the first-visit records of 262 women aged 40–65 years was conducted. The severity of their hot flushes (HF) and night sweats (NS) and their dietary consumption of nutrients were evaluated using the Menopausal Health-Related Quality of Life Questionnaire and the brief-type self-administered Diet History Questionnaire, respectively. The relationships between severity of HF/NS and dietary intake were analyzed separately for 43 major nutrients. We then evaluated different food items as sources of the nutrients. <b>Results:</b> After adjustment for age, body mass index, menopausal status, and background factors significantly related to VMS, only vitamin B<sub>6</sub> (VB<sub>6</sub>) was significantly related to severity of HF (adjusted odds ratio per 10 μg/MJ in VB<sub>6</sub> intake, 0.92; 95% confidence interval, 0.86–0.97). Moreover, a significant inverse relationship was found between the consumption of oily fish as a source of VB<sub>6</sub> and the severity of HF. <b>Conclusions:</b> VB<sub>6</sub> and oily fish intake is inversely associated with the severity of HF in middle-aged women. Therefore, increased intake of VB<sub>6</sub> could help attenuate HF.
DOI: 10.6084/m9.figshare.8152622.v1
title: Severity of hot flushes is inversely associated with dietary intake of vitamin B<sub>6</sub> and oily fish

description: <b>Objectives:</b> This study investigated the links between the severity of vasomotor symptoms (VMS) and the dietary consumption of a variety of nutrients. <b>Method:</b> A cross-sectional analysis of the first-visit records of 262 women aged 40–65 years was conducted. The severity of their hot flushes (HF) and night sweats (NS) and their dietary consumption of nutrients were evaluated using the Menopausal Health-Related Quality of Life Questionnaire and the brief-type self-administered Diet History Questionnaire, respectively. The relationships between severity of HF/NS and dietary intake were analyzed separately for 43 major nutrients. We then evaluated different food items as sources of the nutrients. <b>Results:</b> After adjustment for age, body mass index, menopausal status, and background factors significantly related to VMS, only vitamin B<sub>6</sub> (VB<sub>6</sub>) was significantly related to severity of HF (adjusted odds ratio per 10 μg/MJ in VB<sub>6</sub> intake, 0.92; 95% confidence interval, 0.86–0.97). Moreover, a significant inverse relationship was found between the consumption of oily fish as a source of VB<sub>6</sub> and the severity of HF. <b>Conclusions:</b> VB<sub>6</sub> and oily fish intake is inversely associated with the severity of HF in middle-aged women. Therefore, increased intake of VB<sub>6</sub> could help attenuate HF.
DOI: 10.6084/m9.figshare.8152565
title: urban clusters in mid- to high- latitudes

description: Urban clusters of 2013 in mid- to high- latitudes (&gt;30N)The extents of the urban areas were mapped using a cluster-based method that estimates optimal thresholds using nighttime stable-light data from the Defense Meteorological Satellite Program/Operational Linescan System (DMSP/OLS).<br>Using this data, please refer to these papers: Zhou, Y. et al. A cluster-based method to map urban area from DMSP/OLS nightlights. Remote Sensing of Environment 147, 173-185 (2014).<br>Zhou, Y. et al. A global map of urban extent from nightlights. Environmental Research Letters 10, 054011 (2015)<br>
DOI: 10.6084/m9.figshare.8152565.v2
title: urban clusters in mid- to high- latitudes

description: Urban clusters of 2013 in mid- to high- latitudes (&gt;30N)The extents of the urban areas were mapped using a cluster-based method that estimates optimal thresholds using nighttime stable-light data from the Defense Meteorological Satellite Program/Operational Linescan System (DMSP/OLS).<br>Using this data, please refer to these papers: Zhou, Y. et al. A cluster-based method to map urban area from DMSP/OLS nightlights. Remote Sensing of Environment 147, 173-185 (2014).<br>Zhou, Y. et al. A global map of urban extent from nightlights. Environmental Research Letters 10, 054011 (2015)<br>
DOI: 10.6084/m9.figshare.8152565.v1
title: urban clusters in mid- to high- latitudes

description: Urban clusters of 2013 in mid- to high- latitudes (&gt;30N)The extents of the urban areas were mapped using a cluster-based method that estimates optimal thresholds using nighttime stable-light data from the Defense Meteorological Satellite Program/Operational Linescan System (DMSP/OLS).<br>Using this data, please refer to these papers: Zhou, Y. et al. A cluster-based method to map urban area from DMSP/OLS nightlights. Remote Sensing of Environment 147, 173-185 (2014).<br>Zhou, Y. et al. A global map of urban extent from nightlights. Environmental Research Letters 10, 054011 (2015)<br>
DOI: 10.6084/m9.figshare.8152553
title: Supplementary Material for: IMPULS: Impulsivity-Focused Group Intervention to Reduce Binge Eating Episodes in Patients with Binge Eating Disorder – A Randomised Controlled Trial

description: <b><i>Background:</i></b> Impulsivity is a risk factor for binge eating disorder, and binge eating (BE) equates to impulsive eating behaviour. Hence, we developed IMPULS, a cognitive behavioural group intervention focusing on impulsive eating. <b><i>Methods:</i></b> We randomised 41 patients to the IMPULS group and 39 to the control group. The IMPULS group participated in the IMPULS treatment, while both groups completed weekly self-observations. We compared both groups concerning BE episodes in the past 4 weeks at the end of treatment (primary outcome). As secondary outcomes, we investigated eating pathology, depression, general impulsivity and body mass index (BMI) at the end of treatment and in a 3-month follow-up. <b><i>Results:</i></b> The primary outcome failed, because BE episodes in the past 4 weeks were reduced in both groups at the end of treatment. At follow-up, the IMPULS group showed further improvement, contrary to the control group. The BE days/episodes in the 2 months before were overall reduced in both groups. Eating pathology was reduced in the IMPULS group at the end of treatment and partly in both groups at the follow-up. Depression was only reduced in the IMPULS group. General impulsivity and BMI did not change. <b><i>Conclusions:</i></b> The IMPULS study has a negative primary outcome. However, secondary outcomes indicate that the IMPULS treatment might be promising, as BE, eating pathology and depression were reduced in the IMPULS group. The initially reduced BE in the control group might represent a short-term effect from the self-observations. General impulsivity and BMI might need a longer time or more intensive treatment to change.
DOI: 10.6084/m9.figshare.8152505
title: Fixed area oscillating prey assay data (Fig. 5)

description: Fixed area oscillating prey assay data (Fig. 5). Colours in spreadsheet represent log value of cell.
DOI: 10.6084/m9.figshare.8152505.v1
title: Fixed area oscillating prey assay data (Fig. 5)

description: Fixed area oscillating prey assay data (Fig. 5). Colours in spreadsheet represent log value of cell.
DOI: 10.6084/m9.figshare.8150981
title: Additional file 9: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio dark gene annotations in hg38. (TXT 759 kb)
DOI: 10.6084/m9.figshare.8150981.v1
title: Additional file 9: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio dark gene annotations in hg38. (TXT 759 kb)
DOI: 10.6084/m9.figshare.8150978
title: Additional file 8: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio percent dark genes in hg38. (TXT 145 kb)
DOI: 10.6084/m9.figshare.8150978.v1
title: Additional file 8: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio percent dark genes in hg38. (TXT 145 kb)
DOI: 10.6084/m9.figshare.8150975
title: Additional file 7: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: ONT dark gene annotations in hg38. (TXT 732 kb)
DOI: 10.6084/m9.figshare.8150975.v1
title: Additional file 7: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: ONT dark gene annotations in hg38. (TXT 732 kb)
DOI: 10.6084/m9.figshare.8150972
title: Additional file 6: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: ONT percent dark genes in hg38. (TXT 291 kb)
DOI: 10.6084/m9.figshare.8150972.v1
title: Additional file 6: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: ONT percent dark genes in hg38. (TXT 291 kb)
DOI: 10.6084/m9.figshare.8150969
title: Additional file 5: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: IlluminaRL250 dark gene annotations in hg38. (TXT 1798 kb)
DOI: 10.6084/m9.figshare.8150969.v1
title: Additional file 5: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: IlluminaRL250 dark gene annotations in hg38. (TXT 1798 kb)
DOI: 10.6084/m9.figshare.8150966
title: Additional file 4: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: IlluminaRL250 percent dark genes in hg38. (TXT 282 kb)
DOI: 10.6084/m9.figshare.8150966.v1
title: Additional file 4: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: IlluminaRL250 percent dark genes in hg38. (TXT 282 kb)
DOI: 10.6084/m9.figshare.8150963
title: Additional file 42: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics dark-by-MAPQ gene annotations in hg38. (TXT 730 kb)
DOI: 10.6084/m9.figshare.8150963.v1
title: Additional file 42: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics dark-by-MAPQ gene annotations in hg38. (TXT 730 kb)
DOI: 10.6084/m9.figshare.8150960
title: Additional file 41: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics percent dark-by-MAPQ genes in hg38. (TXT 111 kb)
DOI: 10.6084/m9.figshare.8150960.v1
title: Additional file 41: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics percent dark-by-MAPQ genes in hg38. (TXT 111 kb)
DOI: 10.6084/m9.figshare.8150957
title: Additional file 40: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics dark-by-depth gene annotations in hg38. (TXT 693 kb)
DOI: 10.6084/m9.figshare.8150957.v1
title: Additional file 40: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics dark-by-depth gene annotations in hg38. (TXT 693 kb)
DOI: 10.6084/m9.figshare.8150954
title: Additional file 3: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: IlluminaRL100 dark gene annotations in hg38. (TXT 3152 kb)
DOI: 10.6084/m9.figshare.8150954.v1
title: Additional file 3: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: IlluminaRL100 dark gene annotations in hg38. (TXT 3152 kb)
DOI: 10.6084/m9.figshare.8150951
title: Additional file 39: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics percent dark-by-depth genes in hg38. (TXT 211 kb)
DOI: 10.6084/m9.figshare.8150951.v1
title: Additional file 39: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics percent dark-by-depth genes in hg38. (TXT 211 kb)
DOI: 10.6084/m9.figshare.8150948
title: Additional file 38: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics camouflaged gene annotations in hg38. (TXT 459 kb)
DOI: 10.6084/m9.figshare.8150948.v1
title: Additional file 38: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics camouflaged gene annotations in hg38. (TXT 459 kb)
DOI: 10.6084/m9.figshare.8150945
title: Additional file 37: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics percent camouflaged genes in hg38. (TXT 55 kb)
DOI: 10.6084/m9.figshare.8150945.v1
title: Additional file 37: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: 10x Genomics percent camouflaged genes in hg38. (TXT 55 kb)
DOI: 10.6084/m9.figshare.8150942
title: Additional file 36: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio dark-by-MAPQ gene annotations in hg38. (TXT 477 kb)
DOI: 10.6084/m9.figshare.8150942.v1
title: Additional file 36: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio dark-by-MAPQ gene annotations in hg38. (TXT 477 kb)
DOI: 10.6084/m9.figshare.8150939
title: Additional file 35: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio percent dark-by-MAPQ genes in hg38. (TXT 41 kb)
DOI: 10.6084/m9.figshare.8150939.v1
title: Additional file 35: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio percent dark-by-MAPQ genes in hg38. (TXT 41 kb)
DOI: 10.6084/m9.figshare.8150936
title: Additional file 34: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio dark-by-depth gene annotations in hg38. (TXT 334 kb)
DOI: 10.6084/m9.figshare.8150936.v1
title: Additional file 34: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio dark-by-depth gene annotations in hg38. (TXT 334 kb)
DOI: 10.6084/m9.figshare.8150933
title: Additional file 33: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio percent dark-by-depth genes in hg38. (TXT 113 kb)
DOI: 10.6084/m9.figshare.8150933.v1
title: Additional file 33: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio percent dark-by-depth genes in hg38. (TXT 113 kb)
DOI: 10.6084/m9.figshare.8150930
title: Additional file 32: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio camouflaged gene annotations in hg38. (TXT 348 kb)
DOI: 10.6084/m9.figshare.8150930.v1
title: Additional file 32: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio camouflaged gene annotations in hg38. (TXT 348 kb)
DOI: 10.6084/m9.figshare.8150927
title: Additional file 31: of Systematic analysis of dark and camouflaged genes reveals disease-relevant genes hiding in plain sight

description: PacBio percent camouflaged genes in hg38. (TXT 31 kb)