cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.23728/b2share.121719afee3b4425b9cb89838b879245
title: Air quality data from the LIFE+RESPIRA project in Pamplona, Spain: PM1, PM2.5 and PM10 in 2016 (07h UTC) (shapefile)

description: Shapefile including air quality data from the LIFE+RESPIRA project (http://www.liferespira.eu/) in Pamplona, Spain, with measures of PM1, PM2.5 and PM10 particles during 2016, aggregated by hour (07h UTC).
DOI: 10.34816/efa.archives.693722
title: Philippes-L4216-032A-Inscription

description: Référence : L4216-032A<br />Publication(s) : Inédit<br />Domaine : Épigraphie<br />
DOI: 10.34816/efa.archives.693721
title: Philippes-L4216-031A-Inscription

description: Référence : L4216-031A<br />Publication(s) : Inédit<br />Domaine : Épigraphie<br />
DOI: 10.23728/b2share.9fdfb5d40247486d8ee8cc70f356b15b
title: Air quality data from the LIFE+RESPIRA project in Pamplona, Spain: PM1 in 2016 (07h UTC) (interpolated GeoTIFF)

description: GeoTIFF file including interpolated air quality data from the LIFE+RESPIRA project (http://www.liferespira.eu/) in Pamplona, Spain, with measures of PM1 during 2016, aggregated by hour (07h UTC).
DOI: 10.34816/efa.archives.693720
title: Philippes-L4216-029A-Inscription

description: Référence : L4216-029A<br />Publication(s) : Inédit<br />Domaine : Épigraphie<br />
DOI: 10.23728/b2share.723e7a6e9da54732acb469499058d352
title: Air quality data from the LIFE+RESPIRA project in Pamplona, Spain: NO in 2016 (09h UTC) (clipped PNG)

description: PNG image representing clipped interpolated air quality data from the LIFE+RESPIRA project (http://www.liferespira.eu/) in Pamplona, Spain, with measures of nitrogen monoxide during 2016, aggregated by hour (09h UTC).
DOI: 10.34816/efa.archives.693719
title: Philippes-L4216-028A-Inscription

description: Référence : L4216-028A<br />Publication(s) : Inédit<br />Domaine : Épigraphie<br />
DOI: 10.34816/efa.archives.693718
title: Philippes-L4216-027A-Inscription

description: Référence : L4216-027A<br />Publication(s) : Inédit<br />Domaine : Épigraphie<br />
DOI: 10.23728/b2share.4909404372ce469fba13226426e4716b
title: Air quality data from the LIFE+RESPIRA project in Pamplona, Spain: PM1 in 2016 (07h UTC) (interpolated PNG)

description: PNG image representing interpolated air quality data from the LIFE+RESPIRA project (http://www.liferespira.eu/) in Pamplona, Spain, with measures of PM1 during 2016, aggregated by hour (07h UTC).
DOI: 10.34816/efa.archives.693717
title: Philippes-L4216-026A-Inscription

description: Référence : L4216-026A<br />Publication(s) : Inédit<br />Domaine : Épigraphie<br />
DOI: 10.34816/efa.archives.693716
title: Philippes-L4216-025A-Inscription

description: Référence : L4216-025A<br />Publication(s) : Inédit<br />Domaine : Épigraphie<br />
DOI: 10.15468/dl.v72b4x
title: Occurrence Download

description: A dataset containing 50 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Leucas suffruticosa Benth."
 ]
}

 The dataset includes 50 records from 12 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0047898-240321170329656/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.6084/m9.figshare.13114922.v1
title: Additional file 13 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 13: Table S13. The biomass of the roots and shoots in JZ18, 35S:miR168a and 35S:rSlAGO1 under the normal and K+ deficiency stress conditions.
DOI: 10.6084/m9.figshare.13114943.v1
title: Additional file 1 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 1: Table S1. Primers used in this study.
DOI: 10.6084/m9.figshare.13114922
title: Additional file 13 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 13: Table S13. The biomass of the roots and shoots in JZ18, 35S:miR168a and 35S:rSlAGO1 under the normal and K+ deficiency stress conditions.
DOI: 10.6084/m9.figshare.13114916
title: Additional file 11 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 11: Table S11. List of the differentially expressed mRNAs in 35S:SlmiR168a plants compared with WT.
DOI: 10.6084/m9.figshare.6823559
title: Additional file 1: of AKT isoform-specific expression and activation across cancer lineages

description: 211 cell lines used in RPPA analysis. (XLSX 17 kb)
DOI: 10.6084/m9.figshare.6823559.v1
title: Additional file 1: of AKT isoform-specific expression and activation across cancer lineages

description: 211 cell lines used in RPPA analysis. (XLSX 17 kb)
DOI: 10.6084/m9.figshare.6823718
title: MOESM4 of MicroRNA expression profiling of goat peripheral blood mononuclear cells in response to peste des petits ruminants virus infection

description: Additional file 4. Differentially expressed miRNA in the PPRV-infected goat PBMC compared to the mock-infected groups. Prediction of 12Â 065 target genes for 103 known miRNA and 213 novel miRNA differentially expressed in the mock-infected and PPRV-infected goat PBMC.
DOI: 10.6084/m9.figshare.13114958
title: Additional file 3 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 3: Table S3. List of all expressed miRNA in 35S:SlmiR168a, 35S:rSlAGO1 and WT.
DOI: 10.6084/m9.figshare.13114970
title: Additional file 7 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 7: Table S7. List of the miRNAs whose target genes are predicted.
DOI: 10.6084/m9.figshare.13114967.v1
title: Additional file 6 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 6: Table S6. List of the differentially expressed miRNAs in 35S:rSlAGO1 plants compared with WT.
DOI: 10.6084/m9.figshare.13114973
title: Additional file 8 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 8: Table S8. Target predict annotation for the differentially expressed miRNAs.
DOI: 10.6084/m9.figshare.13114973.v1
title: Additional file 8 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 8: Table S8. Target predict annotation for the differentially expressed miRNAs.
DOI: 10.6084/m9.figshare.13114943
title: Additional file 1 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 1: Table S1. Primers used in this study.
DOI: 10.6084/m9.figshare.13114964.v1
title: Additional file 5 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 5: Table S5. List of the differentially expressed miRNAs in 35S:SlmiR168a plants compared with WT.
DOI: 10.6084/m9.figshare.13114970.v1
title: Additional file 7 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 7: Table S7. List of the miRNAs whose target genes are predicted.
DOI: 10.6084/m9.figshare.13114976.v1
title: Additional file 9 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 9: Table S9. miRNA/mRNA pairs in the comparison of 35S:SlmiR168a and WT plants, with upregulated/upregulated,downregulated/downregulated, upregulated/downregulated, downregulated/upregulated by integrated analysis of miRNA-Seq and mRNA-Seq.
DOI: 10.6084/m9.figshare.20455183.v1
title: Passive smoking may be associated with bleeding of cerebral arteriovenous malformation in non-smoking women: a retrospective analysis

description: ABSTRACT Background Smoking has been considered to be a risk factor for cardiovascular disease, cancer, depression and other diseases in previous reports, and active smoking is considered to be a risk factor for hemorrhagic stroke. In addition, a retrospective study showed that male smokers were at increased risk of bleeding from arteriovenous malformation (AVM), compared with non-smokers. However, the effect of passive smoking on rupturing of cerebral AVM in non-smoking women has not been addressed. Objective This study aimed to assess the impact of tobacco exposure on AVM bleeding risk in non-smoking women. Methods A total of 393 non-smoking women diagnosed with AVM were included. They were divided into a bleeding group (205 women) and a non-bleeding group (188 women). We conducted univariate and multivariate analysis on these two groups. In univariate analysis, risk factors that might be related to AVM bleeding were analyzed. In multivariate analysis, the relationship between passive smoking and AVM rupture was analyzed by correcting confounding factors. Results Multivariate analysis showed that the proportion of passive smoking was statistically different between the bleeding group and the non-bleeding group (OR = 1.609; CI = 1.031-2.509; p = 0.036). Conclusion Passive smoking may increase the risk of AVM bleeding in non-smoking women. This increased risk may be related to the inflammatory response, vascular wall damage, hemodynamic disorders, changes in atherosclerosis and changes in gene expression caused by passive smoking.
DOI: 10.6084/m9.figshare.13114961
title: Additional file 4 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 4: Table S4. The expressed conserved miRNAs were classified into different miRNAs families.
DOI: 10.6084/m9.figshare.13114964
title: Additional file 5 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 5: Table S5. List of the differentially expressed miRNAs in 35S:SlmiR168a plants compared with WT.
DOI: 10.6084/m9.figshare.13114976
title: Additional file 9 of miR168 targets Argonaute1A mediated miRNAs regulation pathways in response to potassium deficiency stress in tomato

description: Additional file 9: Table S9. miRNA/mRNA pairs in the comparison of 35S:SlmiR168a and WT plants, with upregulated/upregulated,downregulated/downregulated, upregulated/downregulated, downregulated/upregulated by integrated analysis of miRNA-Seq and mRNA-Seq.
DOI: 10.6084/m9.figshare.20455183
title: Passive smoking may be associated with bleeding of cerebral arteriovenous malformation in non-smoking women: a retrospective analysis

description: ABSTRACT Background Smoking has been considered to be a risk factor for cardiovascular disease, cancer, depression and other diseases in previous reports, and active smoking is considered to be a risk factor for hemorrhagic stroke. In addition, a retrospective study showed that male smokers were at increased risk of bleeding from arteriovenous malformation (AVM), compared with non-smokers. However, the effect of passive smoking on rupturing of cerebral AVM in non-smoking women has not been addressed. Objective This study aimed to assess the impact of tobacco exposure on AVM bleeding risk in non-smoking women. Methods A total of 393 non-smoking women diagnosed with AVM were included. They were divided into a bleeding group (205 women) and a non-bleeding group (188 women). We conducted univariate and multivariate analysis on these two groups. In univariate analysis, risk factors that might be related to AVM bleeding were analyzed. In multivariate analysis, the relationship between passive smoking and AVM rupture was analyzed by correcting confounding factors. Results Multivariate analysis showed that the proportion of passive smoking was statistically different between the bleeding group and the non-bleeding group (OR = 1.609; CI = 1.031-2.509; p = 0.036). Conclusion Passive smoking may increase the risk of AVM bleeding in non-smoking women. This increased risk may be related to the inflammatory response, vascular wall damage, hemodynamic disorders, changes in atherosclerosis and changes in gene expression caused by passive smoking.
DOI: 10.6084/m9.figshare.6823718.v1
title: MOESM4 of MicroRNA expression profiling of goat peripheral blood mononuclear cells in response to peste des petits ruminants virus infection

description: Additional file 4. Differentially expressed miRNA in the PPRV-infected goat PBMC compared to the mock-infected groups. Prediction of 12Â 065 target genes for 103 known miRNA and 213 novel miRNA differentially expressed in the mock-infected and PPRV-infected goat PBMC.
DOI: 10.6084/m9.figshare.6823892
title: Additional file 11: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S11. Primers used for RT-PCR. (XLSX 11 kb)
DOI: 10.6084/m9.figshare.6823934.v1
title: Additional file 3: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S3. The numbers and ratios of testicular lncRNAs and mRNAs on each mouse chromosome. (XLSX 11 kb)
DOI: 10.6084/m9.figshare.6823904
title: Additional file 1: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S1. LncRNAs and mRNAs in the mouse brain, heart, kidney, liver and testis. (XLSX 18613 kb)
DOI: 10.6084/m9.figshare.6823904.v1
title: Additional file 1: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S1. LncRNAs and mRNAs in the mouse brain, heart, kidney, liver and testis. (XLSX 18613 kb)
DOI: 10.6084/m9.figshare.6823919
title: Additional file 2: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S2. Tissue-specific lncRNAs and mRNAs in the mouse brain, heart, kidney, liver and testis. (XLSX 4020 kb)
DOI: 10.6084/m9.figshare.6823973.v1
title: Additional file 6: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S6. GO terms of the nearby protein-coding genes. (XLSX 19 kb)
DOI: 10.6084/m9.figshare.6823943
title: Additional file 4: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S4. The numbers and ratios of six types of testicular lncRNA. (XLSX 10 kb)
DOI: 10.6084/m9.figshare.20455284
title: Efficacy and safety of phosphatidylinositol 3-kinase inhibitors in patients with chronic lymphocytic leukemia: a meta-analysis and systematic review

description: Phosphatidylinositol 3-kinase (PI3K) inhibitors demonstrate promising effects in the treatment of relapsed/refractory chronic lymphocytic leukemia (CLL). Emerging evidence of the therapeutic effects of the PI3K inhibitors in various aspects remains controversial. This meta-analysis was used to evaluate the efficacy and safety of PI3K inhibitors based on a synthesis of the data generated by randomized controlled trials (RCTs) of patients with CLL, by searching PubMed, EMBASE, Cochrane Central Register of Controlled Trials, and ClinicalTrials.gov registry. Five RCTs with 1593 patients were included. The PI3K inhibitors significantly improved progression-free survival (PFS), the chance of overall response, and partial response compared to the control arm. Regarding adverse events (AEs), the PI3K inhibitors increased the risk of pyrexia, chills, diarrhea, decreased appetite, vomiting, rash, pneumonia, and upper respiratory tract infection. Available evidence showed significant improvements over conventional therapy in PFS of the PI3K inhibitors in patients with CLL, with relatively manageable AEs.
DOI: 10.6084/m9.figshare.6823976
title: Additional file 7: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S7. Mouse testis-specific lncRNAs that show conservation in human. (XLSX 26 kb)
DOI: 10.6084/m9.figshare.17833892
title: Serum anti-PAD4 autoantibodies are present in cystic fibrosis children and increase with age and lung disease severity

description: Cystic fibrosis (CF) lung disease begins early in childhood and is characterized by neutrophilic inflammation of the airways. Neutrophil extracellular traps (NETs) represent one mechanism by which neutrophils contribute to lung damage. The enzyme peptidylarginine deiminase 4 (PAD4) is required for NET formation. Our overall concept is that NET formation delivers PAD4 outside the neutrophil resulting in autoantibody generation, and this autoimmunity may be a novel mechanism contributing to CF lung disease progression. The aim of this study was to investigate clinical predictors of serum anti-PAD4 autoantibody (PAD4 Ab) levels in CF subjects with a wide range of ages from early childhood through middle age. We measured PAD4 Ab levels in sera from 104 CF subjects. PAD4 Abs were detectable among CF children as young as one year of age and elevated compared to paediatric healthy controls. PAD4 Ab levels increased significantly with age (<i>r</i> = 0.584, <i>p</i> &lt;.001) and correlated with lower lung function (<i>r</i> = −0.481, <i>n</i> = 99, <i>p</i> &lt;.001). PAD4 Abs were elevated in subjects with chronic <i>Pseudomonas aeruginosa</i> airways infection (<i>p</i> &lt;.001), but not with other key clinical CF co-variates including sex, <i>CFTR</i> genotype, sweat chloride, pancreatic enzyme use, nutritional status, recent pulmonary exacerbations, <i>Staphylococcus aureus</i>, or CF-related diabetes. PAD4 Ab levels were also correlated with serum anti-double-stranded DNA IgA autoantibodies, which have similarly been shown to be elevated in CF subjects and associated with lung damage. In multivariable analysis, age and lung function remained correlated with PAD4 Ab levels. In summary, we describe novel findings of anti-PAD4 autoantibodies in CF that are present early in childhood, increase over time with age, and correlate with lung disease severity. Autoimmunity to antigens extruded by NETs appears to be an early event in CF lung disease, and airway autoimmunity related to NET formation is a potential mechanism of lung disease progression in CF.HighlightsSerum anti-PAD4 autoantibodies are detected in paediatric CF serum and are elevated compared to healthy paediatric controlsAnti-PAD4 autoantibodies increase with ageAnti-PAD4 autoantibodies correlate with lower lung function, <i>Pseudomonas</i> aeruginosa airway infection and anti-dsDNA IgA autoantibodies, but not with other key clinical CF co-variatesAge and lung function remain correlated with anti-PAD4 autoantibodies in multivariable analysis Serum anti-PAD4 autoantibodies are detected in paediatric CF serum and are elevated compared to healthy paediatric controls Anti-PAD4 autoantibodies increase with age Anti-PAD4 autoantibodies correlate with lower lung function, <i>Pseudomonas</i> aeruginosa airway infection and anti-dsDNA IgA autoantibodies, but not with other key clinical CF co-variates Age and lung function remain correlated with anti-PAD4 autoantibodies in multivariable analysis
DOI: 10.6084/m9.figshare.17833892.v1
title: Serum anti-PAD4 autoantibodies are present in cystic fibrosis children and increase with age and lung disease severity

description: Cystic fibrosis (CF) lung disease begins early in childhood and is characterized by neutrophilic inflammation of the airways. Neutrophil extracellular traps (NETs) represent one mechanism by which neutrophils contribute to lung damage. The enzyme peptidylarginine deiminase 4 (PAD4) is required for NET formation. Our overall concept is that NET formation delivers PAD4 outside the neutrophil resulting in autoantibody generation, and this autoimmunity may be a novel mechanism contributing to CF lung disease progression. The aim of this study was to investigate clinical predictors of serum anti-PAD4 autoantibody (PAD4 Ab) levels in CF subjects with a wide range of ages from early childhood through middle age. We measured PAD4 Ab levels in sera from 104 CF subjects. PAD4 Abs were detectable among CF children as young as one year of age and elevated compared to paediatric healthy controls. PAD4 Ab levels increased significantly with age (<i>r</i> = 0.584, <i>p</i> &lt;.001) and correlated with lower lung function (<i>r</i> = −0.481, <i>n</i> = 99, <i>p</i> &lt;.001). PAD4 Abs were elevated in subjects with chronic <i>Pseudomonas aeruginosa</i> airways infection (<i>p</i> &lt;.001), but not with other key clinical CF co-variates including sex, <i>CFTR</i> genotype, sweat chloride, pancreatic enzyme use, nutritional status, recent pulmonary exacerbations, <i>Staphylococcus aureus</i>, or CF-related diabetes. PAD4 Ab levels were also correlated with serum anti-double-stranded DNA IgA autoantibodies, which have similarly been shown to be elevated in CF subjects and associated with lung damage. In multivariable analysis, age and lung function remained correlated with PAD4 Ab levels. In summary, we describe novel findings of anti-PAD4 autoantibodies in CF that are present early in childhood, increase over time with age, and correlate with lung disease severity. Autoimmunity to antigens extruded by NETs appears to be an early event in CF lung disease, and airway autoimmunity related to NET formation is a potential mechanism of lung disease progression in CF.HighlightsSerum anti-PAD4 autoantibodies are detected in paediatric CF serum and are elevated compared to healthy paediatric controlsAnti-PAD4 autoantibodies increase with ageAnti-PAD4 autoantibodies correlate with lower lung function, <i>Pseudomonas</i> aeruginosa airway infection and anti-dsDNA IgA autoantibodies, but not with other key clinical CF co-variatesAge and lung function remain correlated with anti-PAD4 autoantibodies in multivariable analysis Serum anti-PAD4 autoantibodies are detected in paediatric CF serum and are elevated compared to healthy paediatric controls Anti-PAD4 autoantibodies increase with age Anti-PAD4 autoantibodies correlate with lower lung function, <i>Pseudomonas</i> aeruginosa airway infection and anti-dsDNA IgA autoantibodies, but not with other key clinical CF co-variates Age and lung function remain correlated with anti-PAD4 autoantibodies in multivariable analysis
DOI: 10.6084/m9.figshare.6823973
title: Additional file 6: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S6. GO terms of the nearby protein-coding genes. (XLSX 19 kb)
DOI: 10.6084/m9.figshare.6823991
title: Additional file 8: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S8. LncRNAs and mRNAs in three mouse germ cell-related cell lines (F9, GC-1 and GC-2). (XLSX 16402 kb)
DOI: 10.6084/m9.figshare.6823976.v1
title: Additional file 7: of Profiling of testis-specific long noncoding RNAs in mice

description: Table S7. Mouse testis-specific lncRNAs that show conservation in human. (XLSX 26 kb)