cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.15468/dl.a6yx5t
title: Occurrence Download

description: A dataset containing 2931 species occurrences available in GBIF matching the query:
{
 "and" : [
 "TaxonKey is one of (Eusmilia fastigiata (Pallas, 1766))",
 "OccurrenceStatus is Present",
 "Year is greater than or equal to 2000",
 "BasisOfRecord is one of (Human Observation, Observation, Machine Observation)",
 "HasCoordinate is true",
 "HasGeospatialIssue is false",
 {
 "or" : [
 "CoordinateUncertaintyInMeters is less than 100",
 "CoordinateUncertaintyInMeters is null"
 ]
 }
 ]
}

 The dataset includes 2931 records from 58 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0090042-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.j39rbf
title: Occurrence Download

description: A dataset containing 3 species occurrences available in GBIF matching the query:
{
 "and" : [
 "Continent is South America",
 "Country is Colombia",
 "OccurrenceStatus is Present",
 "PublishingOrg is one of (eace4687-50e8-4f9a-829b-29ff8ff1fa8b, eac88d99-9f6c-4031-8fc4-8088f0e0dfe7, 2a7e3080-28a9-11dd-97cd-b8a03c50a862, bc092ff0-02e4-11dc-991f-b8a03c50a862, f7f9717e-9e50-4a00-a30f-7b134390a566, 28eb1a3f-1c15-4a95-931a-4af90ecb574d)",
 "TaxonKey is Fuchsia arborescens La Llave, 1832"
 ]
}

 The dataset includes 3 records from 2 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0090044-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.h9h7kn
title: Occurrence Download

description: A dataset containing 0 species occurrences available in GBIF matching the query:
{
 "and" : [
 {
 "and" : [
 "HasGeospatialIssue is false",
 "HasCoordinate is true",
 "OccurrenceStatus is Present",
 {
 "not" : {
 "BasisOfRecord" : [
 "is one of (Fossil, Living Specimen)"
 ]
 }
 }
 ]
 },
 "SpeciesKey is Cranogona pavida Ribaut, 1951",
 "Year is greater than or equal to 2004",
 "Year is less than or equal to 2023"
 ]
}

 The dataset includes 0 records from 0 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0090043-240626123714530/datasets/export for details.
DOI: 10.15468/dl.hehr8y
title: Occurrence Download

description: A dataset containing 13 species occurrences available in GBIF matching the query:
{
 "and" : [
 {
 "and" : [
 "HasGeospatialIssue is false",
 "HasCoordinate is true",
 "OccurrenceStatus is Present",
 {
 "not" : {
 "BasisOfRecord" : [
 "is one of (Fossil, Living Specimen)"
 ]
 }
 }
 ]
 },
 "SpeciesKey is Euryopis sexalbomaculata (Lucas, 1846)",
 "Year is greater than or equal to 2004",
 "Year is less than or equal to 2023"
 ]
}

 The dataset includes 13 records from 3 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0090046-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.p4kmun
title: Occurrence Download

description: A dataset containing 8 species occurrences available in GBIF matching the query:
{
 "and" : [
 "Country is Mozambique",
 "TaxonKey is Maerua andradae Wild"
 ]
}

 The dataset includes 8 records from 6 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0090047-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.gma34q
title: Occurrence Download

description: A dataset containing 16061 species occurrences available in GBIF matching the query:
{
 "and" : [
 {
 "and" : [
 "HasGeospatialIssue is false",
 "HasCoordinate is true",
 "OccurrenceStatus is Present",
 {
 "not" : {
 "BasisOfRecord" : [
 "is one of (Fossil, Living Specimen)"
 ]
 }
 }
 ]
 },
 "SpeciesKey is Colletes hederae Schmidt & Westrich, 1993",
 "Year is greater than or equal to 2004",
 "Year is less than or equal to 2023"
 ]
}

 The dataset includes 16061 records from 197 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0090045-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.6084/m9.figshare.24087915
title: <i>Co-cultures of cerebellar slices from mice with different reelin genetic backgrounds as a model to</i><i>study cortical lamination</i> - <b>Supplementary Material 6</b>

description: <b>Spatial pattern of Purkinje neuron distribution analysis data</b>
DOI: 10.6084/m9.figshare.24087915.v1
title: <i>Co-cultures of cerebellar slices from mice with different reelin genetic backgrounds as a model to</i><i>study cortical lamination</i> - <b>Supplementary Material 6</b>

description: <b>Spatial pattern of Purkinje neuron distribution analysis data</b>
DOI: 10.6084/m9.figshare.23959119
title: mandatory fields required to publish the item

description: mandatory fields required to publish the item
DOI: 10.6084/m9.figshare.23959119.v2
title: mandatory fields required to publish the item

description: mandatory fields required to publish the item
DOI: 10.6084/m9.figshare.23959119.v1
title: data.sav

description: <b>Objectives:</b> Two earthquakes of 7.7 and 7.6 magnitudes on the Richter scale occurred in Kahramanmaraş on February 6, 2023, have resulted in 50,000 deaths and significant devastation. Our aim is to assess the anxiety levels of emergency healthcare professionals (HPs) providing medical services in earthquake-stricken area following the Kahramanmaraş earthquake.<b>Methods:</b> This study includes survivor HPs and volunteer HPs. Only 89 out of 159 HPs completed the questionnaire, resulting in a response rate of 57.1%.<b>Results:</b> The mean Beck Anxiety Inventory (BAI) score for Survivor HPs was 29.53±13.25, and for Volunteer HPs was 7.62±11.12. The difference between scores was 21.91±2.61, and it was statistically significant (95% CI: 16.77-27.05, p=0.000).<b>Conclusions:</b> Our study indicates that HPs who providing first-line health services in earthquake-stricken areas exhibit high anxiety scores. The BAI scores of Survivor HPs were higher compared to Volunteer HPs. As the duration of work in the earthquake-stricken areas increased, anxiety levels rise accordingly.
DOI: 10.5519/qd.xtwpkgoe
title: Query on the Natural History Museum Data Portal (data.nhm.ac.uk) (448 records)
DOI: 10.6084/m9.figshare.24079362
title: Skin scRNA-seq Data

description: This dataset was generated as part of the study titled "An Optimized Tissue Dissociation Protocol for Flow Cytometry and Single-Cell RNA Sequencing Analysis of Immune Cells in Human Skin". Each sample corresponds to the skin of an individual donor.
DOI: 10.5258/soton/d2763
title: Dataset supporting the University of Southampton Doctoral Thesis "Navigating the landscape: Roles, perspectives, and experiences of psychologists supporting gender diverse children and young people in school settings".

description: Dataset supporting the University of Southampton Doctoral Thesis "Navigating the landscape: Roles, perspectives, and experiences of psychologists supporting gender diverse children and young people in school settings". This dataset includes qualitative survey responses collected on Qualtrics, from trainee and qualified educational psychologists. Also included are information sheet and consent forms sent to participants. Licenses/restrictions placed on the data, or limitations of reuse: Creative Commons Attribution Licence Recommended citation for the data: New-Brown, G.J. (2023). What Guides Us? Exploring Current Practices of Educational Psychologists with gender diverse CYP in the UK.
DOI: 10.6084/m9.figshare.24088041
title: Supplementary Material for: ABCF1/CXCL12/CXCR4 enhances glioblastoma cell proliferation, migration and invasion by activating the PI3K/AKT signal pathway

description: Background: Glioblastoma (GBM) is the most prevalent and fatal form of brain tumor, which is associated with a poor prognosis. ATP-binding cassette subfamily F member 1 (ABCF1), is an E2 ubiquitin-conjugating enzyme, which is implicated in regulating immune responses and tumorigenesis. Aberrant E3 ubiquitylation has been evidenced in GBM. However, the role of ABCF1 in GBM needs to be further explored. Methods: The expression of ABCF1, CXC chemokine ligand 12 (CXCL12) and CXC chemokine receptor 4 (CXCR4) in GBM tissues was examined by the GEPIA tool, real-time PCR and western blotting. HMC3, U251MG and LN-229 cells were cultured and transfected with shRNA targeting ABCF1 and ABCF1 plasmid. The proliferative, migrative and invasive ability of cells was detected. Western blotting was used to detect the levels of phosphorylated phosphatidylinositol 3-kinase (PI3K) and phosphorylated protein kinase B (AKT). Results: We observed that GBM tissues had higher ABCF1, CXCL12, and CXCR4 expression levels. The expression of CXCL12 and CXCR4were enhanced by ABCF1 overexpression, which were significantly reversed by silence of ABCF1 in GBM cells. Silencing ABCF1 or CXCR4 inhibition weakened the capacity of GBM cell growth, migration and invasion, while ectopic ABCF1 expression or CXCL12 treatment enhanced the cellular function of GBM cells. Furthermore, p-PI3K and p-AKT protein levels were downregulated by ABCF1 knockdown or CXCR4 blockade, which were prompted by ABCF1 overexpression or CXCL12 supplement. Conclusion: The ABCF1-CXCL12-CXCR4 axis was identified as a key player in GBM cell survival and metastasis through activating the PI3K/AKT signaling pathway in GBM cells.
DOI: 10.6084/m9.figshare.24088041.v1
title: Supplementary Material for: ABCF1/CXCL12/CXCR4 enhances glioblastoma cell proliferation, migration and invasion by activating the PI3K/AKT signal pathway

description: Background: Glioblastoma (GBM) is the most prevalent and fatal form of brain tumor, which is associated with a poor prognosis. ATP-binding cassette subfamily F member 1 (ABCF1), is an E2 ubiquitin-conjugating enzyme, which is implicated in regulating immune responses and tumorigenesis. Aberrant E3 ubiquitylation has been evidenced in GBM. However, the role of ABCF1 in GBM needs to be further explored. Methods: The expression of ABCF1, CXC chemokine ligand 12 (CXCL12) and CXC chemokine receptor 4 (CXCR4) in GBM tissues was examined by the GEPIA tool, real-time PCR and western blotting. HMC3, U251MG and LN-229 cells were cultured and transfected with shRNA targeting ABCF1 and ABCF1 plasmid. The proliferative, migrative and invasive ability of cells was detected. Western blotting was used to detect the levels of phosphorylated phosphatidylinositol 3-kinase (PI3K) and phosphorylated protein kinase B (AKT). Results: We observed that GBM tissues had higher ABCF1, CXCL12, and CXCR4 expression levels. The expression of CXCL12 and CXCR4were enhanced by ABCF1 overexpression, which were significantly reversed by silence of ABCF1 in GBM cells. Silencing ABCF1 or CXCR4 inhibition weakened the capacity of GBM cell growth, migration and invasion, while ectopic ABCF1 expression or CXCL12 treatment enhanced the cellular function of GBM cells. Furthermore, p-PI3K and p-AKT protein levels were downregulated by ABCF1 knockdown or CXCR4 blockade, which were prompted by ABCF1 overexpression or CXCL12 supplement. Conclusion: The ABCF1-CXCL12-CXCR4 axis was identified as a key player in GBM cell survival and metastasis through activating the PI3K/AKT signaling pathway in GBM cells.
DOI: 10.5519/qd.hbbeqrbn
title: Query on the Natural History Museum Data Portal (data.nhm.ac.uk) (2567 records)
DOI: 10.6084/m9.figshare.24088020
title: Supplement Material for: Pathogenicity of Variant m.13528A&gt;G in MT-ND5 in Leber’s Hereditary Optic Neuropathy Is Unsupported

description: With interest we read the article by Pandya et al. [<strong>1</strong>] on a 57-year-old male who was diagnosed with Leber’s hereditary optic neuropathy (LHON) due to the variant m.13528A&gt;G in <em>MT-ND5</em>. Although the patient did not receive idebenone, visual acuity improved by the 24-month follow-up [<strong>1</strong>]. The study is excellent but has limitations that are cause of concerns and should be discussed. The CARE Checklist has been completed by the authors for this report, attached as supplementary material (for all online suppl. material, see <strong>https://doi.org/10.1159/000531387</strong>).
DOI: 10.6084/m9.figshare.24088047
title: Supplementary Material for: ABCF1/CXCL12/CXCR4 enhances glioblastoma cell proliferation, migration and invasion by activating the PI3K/AKT signal pathway

description: Background: Glioblastoma (GBM) is the most prevalent and fatal form of brain tumor, which is associated with a poor prognosis. ATP-binding cassette subfamily F member 1 (ABCF1), is an E2 ubiquitin-conjugating enzyme, which is implicated in regulating immune responses and tumorigenesis. Aberrant E3 ubiquitylation has been evidenced in GBM. However, the role of ABCF1 in GBM needs to be further explored. Methods: The expression of ABCF1, CXC chemokine ligand 12 (CXCL12) and CXC chemokine receptor 4 (CXCR4) in GBM tissues was examined by the GEPIA tool, real-time PCR and western blotting. HMC3, U251MG and LN-229 cells were cultured and transfected with shRNA targeting ABCF1 and ABCF1 plasmid. The proliferative, migrative and invasive ability of cells was detected. Western blotting was used to detect the levels of phosphorylated phosphatidylinositol 3-kinase (PI3K) and phosphorylated protein kinase B (AKT). Results: We observed that GBM tissues had higher ABCF1, CXCL12, and CXCR4 expression levels. The expression of CXCL12 and CXCR4were enhanced by ABCF1 overexpression, which were significantly reversed by silence of ABCF1 in GBM cells. Silencing ABCF1 or CXCR4 inhibition weakened the capacity of GBM cell growth, migration and invasion, while ectopic ABCF1 expression or CXCL12 treatment enhanced the cellular function of GBM cells. Furthermore, p-PI3K and p-AKT protein levels were downregulated by ABCF1 knockdown or CXCR4 blockade, which were prompted by ABCF1 overexpression or CXCL12 supplement. Conclusion: The ABCF1-CXCL12-CXCR4 axis was identified as a key player in GBM cell survival and metastasis through activating the PI3K/AKT signaling pathway in GBM cells.
DOI: 10.6084/m9.figshare.24088047.v1
title: Supplementary Material for: ABCF1/CXCL12/CXCR4 enhances glioblastoma cell proliferation, migration and invasion by activating the PI3K/AKT signal pathway

description: Background: Glioblastoma (GBM) is the most prevalent and fatal form of brain tumor, which is associated with a poor prognosis. ATP-binding cassette subfamily F member 1 (ABCF1), is an E2 ubiquitin-conjugating enzyme, which is implicated in regulating immune responses and tumorigenesis. Aberrant E3 ubiquitylation has been evidenced in GBM. However, the role of ABCF1 in GBM needs to be further explored. Methods: The expression of ABCF1, CXC chemokine ligand 12 (CXCL12) and CXC chemokine receptor 4 (CXCR4) in GBM tissues was examined by the GEPIA tool, real-time PCR and western blotting. HMC3, U251MG and LN-229 cells were cultured and transfected with shRNA targeting ABCF1 and ABCF1 plasmid. The proliferative, migrative and invasive ability of cells was detected. Western blotting was used to detect the levels of phosphorylated phosphatidylinositol 3-kinase (PI3K) and phosphorylated protein kinase B (AKT). Results: We observed that GBM tissues had higher ABCF1, CXCL12, and CXCR4 expression levels. The expression of CXCL12 and CXCR4were enhanced by ABCF1 overexpression, which were significantly reversed by silence of ABCF1 in GBM cells. Silencing ABCF1 or CXCR4 inhibition weakened the capacity of GBM cell growth, migration and invasion, while ectopic ABCF1 expression or CXCL12 treatment enhanced the cellular function of GBM cells. Furthermore, p-PI3K and p-AKT protein levels were downregulated by ABCF1 knockdown or CXCR4 blockade, which were prompted by ABCF1 overexpression or CXCL12 supplement. Conclusion: The ABCF1-CXCL12-CXCR4 axis was identified as a key player in GBM cell survival and metastasis through activating the PI3K/AKT signaling pathway in GBM cells.
DOI: 10.6084/m9.figshare.24088104
title: Supplementary Material for: Mismatch repair deficiency in biliary tract cancer: prognostic implications and correlation with histology.

description: Introduction: Mismatch repair (MMR) deficiency represents a biomarker and therapeutic target in various neoplasms, but its role in biliary tract cancers (BTCs) remains misunderstood. Methods: MMR status was retrospectively assessed using immunohistochemistry in 163-BTCs patients. We identified MMR proficiency (pMMR)/deficiency (dMMR) according to the loss of MMR proteins (MLH-1, PMS-2, MSH-2, MSH-6). The primary objective of the study was to assess the incidence of dMMR in BTCs; the secondary purpose was to explore its association with prognosis and clinical features. Results: dMMR was recorded in 9 patients and it was strongly associated with mucinous histology (p&lt;0.01). Regarding the prognostic effect, in 122-radically resected patients, disease-free-survival (DFS) resulted significantly shorter in dMMR-patients compared to pMMR-patients (10.7 vs 31.3 months, p = 0.025) and so did nodal status (48.2 vs 15.3 months in N0 vs N+) (p &lt; 0.01). Moreover, dMMR confirmed its prognostic role in terms of DFS at multivariate analysis (p = 0.03), together with nodal status (p = 0.01) and resection margin (p = 0.03). In 103 M+ patients (encompassing 41 metastatic de novo and 62 recurred after surgery patients) there weren’t differences between dMMR and pMMR regarding survival analyses. Discussion/Conclusions: dMMR status is strongly correlated with mucinous histology and represents an independent prognostic factor in terms of disease-relapse in patients with resected BTC.
DOI: 10.6084/m9.figshare.24088104.v1
title: Supplementary Material for: Mismatch repair deficiency in biliary tract cancer: prognostic implications and correlation with histology.

description: Introduction: Mismatch repair (MMR) deficiency represents a biomarker and therapeutic target in various neoplasms, but its role in biliary tract cancers (BTCs) remains misunderstood. Methods: MMR status was retrospectively assessed using immunohistochemistry in 163-BTCs patients. We identified MMR proficiency (pMMR)/deficiency (dMMR) according to the loss of MMR proteins (MLH-1, PMS-2, MSH-2, MSH-6). The primary objective of the study was to assess the incidence of dMMR in BTCs; the secondary purpose was to explore its association with prognosis and clinical features. Results: dMMR was recorded in 9 patients and it was strongly associated with mucinous histology (p&lt;0.01). Regarding the prognostic effect, in 122-radically resected patients, disease-free-survival (DFS) resulted significantly shorter in dMMR-patients compared to pMMR-patients (10.7 vs 31.3 months, p = 0.025) and so did nodal status (48.2 vs 15.3 months in N0 vs N+) (p &lt; 0.01). Moreover, dMMR confirmed its prognostic role in terms of DFS at multivariate analysis (p = 0.03), together with nodal status (p = 0.01) and resection margin (p = 0.03). In 103 M+ patients (encompassing 41 metastatic de novo and 62 recurred after surgery patients) there weren’t differences between dMMR and pMMR regarding survival analyses. Discussion/Conclusions: dMMR status is strongly correlated with mucinous histology and represents an independent prognostic factor in terms of disease-relapse in patients with resected BTC.
DOI: 10.5517/ccdc.csd.cc2bhqp8
title: CCDC 2160070: Experimental Crystal Structure Determination

description: Related Article: Sébastien Néron, Mathieu Morency, Cédric Malveau, Thierry Maris, Radu Iftimie, James D. Wuest|2022|J.Org.Chem.|87|15796|doi:10.1021/acs.joc.2c01805
description: An entry from the Cambridge Structural Database, the world’s repository for small molecule crystal structures. The entry contains experimental data from a crystal diffraction study. The deposited dataset for this entry is freely available from the CCDC and typically includes 3D coordinates, cell parameters, space group, experimental conditions and quality measures.
DOI: 10.5517/ccdc.csd.cc2bhqq9
title: CCDC 2160071: Experimental Crystal Structure Determination

description: Related Article: Sébastien Néron, Mathieu Morency, Cédric Malveau, Thierry Maris, Radu Iftimie, James D. Wuest|2022|J.Org.Chem.|87|15796|doi:10.1021/acs.joc.2c01805
description: An entry from the Cambridge Structural Database, the world’s repository for small molecule crystal structures. The entry contains experimental data from a crystal diffraction study. The deposited dataset for this entry is freely available from the CCDC and typically includes 3D coordinates, cell parameters, space group, experimental conditions and quality measures.
DOI: 10.5517/ccdc.csd.cc2bhqtd
title: CCDC 2160074: Experimental Crystal Structure Determination

description: Related Article: Sébastien Néron, Mathieu Morency, Cédric Malveau, Thierry Maris, Radu Iftimie, James D. Wuest|2022|J.Org.Chem.|87|15796|doi:10.1021/acs.joc.2c01805
description: An entry from the Cambridge Structural Database, the world’s repository for small molecule crystal structures. The entry contains experimental data from a crystal diffraction study. The deposited dataset for this entry is freely available from the CCDC and typically includes 3D coordinates, cell parameters, space group, experimental conditions and quality measures.
DOI: 10.5517/ccdc.csd.cc2bhqrb
title: CCDC 2160072: Experimental Crystal Structure Determination

description: Related Article: Sébastien Néron, Mathieu Morency, Cédric Malveau, Thierry Maris, Radu Iftimie, James D. Wuest|2022|J.Org.Chem.|87|15796|doi:10.1021/acs.joc.2c01805
description: An entry from the Cambridge Structural Database, the world’s repository for small molecule crystal structures. The entry contains experimental data from a crystal diffraction study. The deposited dataset for this entry is freely available from the CCDC and typically includes 3D coordinates, cell parameters, space group, experimental conditions and quality measures.
DOI: 10.5517/ccdc.csd.cc2bhqsc
title: CCDC 2160073: Experimental Crystal Structure Determination

description: Related Article: Sébastien Néron, Mathieu Morency, Cédric Malveau, Thierry Maris, Radu Iftimie, James D. Wuest|2022|J.Org.Chem.|87|15796|doi:10.1021/acs.joc.2c01805
description: An entry from the Cambridge Structural Database, the world’s repository for small molecule crystal structures. The entry contains experimental data from a crystal diffraction study. The deposited dataset for this entry is freely available from the CCDC and typically includes 3D coordinates, cell parameters, space group, experimental conditions and quality measures.
DOI: 10.6084/m9.figshare.24031914.v1
title: Supplementary Material for: Scleroderma Secondary to Pembrolizumab: A Case Report and Review of 19 cases of anti PD-1-induced scleroderma.

description: Immune checkpoint inhibitors are increasingly being used to treat various malignancies. They are known to potentially cause immune-related adverse effects, including dermatological manifestations. A rare cutaneous immune-related adverse effect is scleroderma, which has been reported to occur with anti-programmed cell death-1 (PD-1) agents such as pembrolizumab and nivolumab. This may present with skin tightening and hardening at any point during or after immunotherapy. We present the case of a 54-year-old Caucasian woman who, following 16 cycles of Pembrolizumab for breast cancer, developed clinical features of scleroderma confirmed on histology. She was initially treated with oral corticosteroids, followed by oral psoralen-UVA (PUVA), with poor response, but eventually improved with methotrexate. A literature review revealed 12 other cases of scleroderma following pembrolizumab treatment and 6 cases of scleroderma following nivolumab treatment . Scleroderma developed at different stages of pembrolizumab therapy. Although scleroderma is not commonly drug-induced, pembrolizumab and nivolumab may be a rare causes and it is important to elicit an accurate drug history, including immunotherapy, in such cases.