cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.21227/3cqf-ye58
title: An Interesting Observation- C

description: This dataset is in support of my claim on an intersting observation on " " submitted to IEEE Transactions on Circuits and Systems I . The paper is under evaluation but here are the supplementary materials relevant to this paper. It includess multimedia, gif images which are the result of theBelow is the paper Abstract submitted to IEEEif able to do work on this, will submit by dec 2020 o jan 2021,otherwise june or dec 2021
DOI: 10.6073/pasta/a32f4827bf2b967359319b2e5610c2ee
title: Water Quality Data (Grab Samples) from the Taylor Slough, Everglades National Park (FCE LTER), South Florida from September 1999 to Present

description: Water quality samples are being collected using ISCO autosamplers at all wetland sites (that is, all sites except TS/Ph-9, 10, and 11). The autosamplers contain 24 1L bottles. Water is sampled by programming the autosamplers to take composite samples once every 3 days. These samples are a composite of four 250mL subsamples drawn every 18 hours (a sampling scheme that captures a dawn, noon, dusk, and midnight sample in every three day composite). The samples are collected every 3-4 weeks and analyzed for total phosphorus (TP), total nitrogen (TN), and salinity. When sites are visited to collect these samples, we also collect a grab sample that is immediately put on ice. A portion of these grab samples is filtered through a Whatman GF/F filter immediately upon return to the lab, and the filtered samples are analyzed for inorganic nutrients such as NO2-, NO3-, NH4+, SRP, and DOC. The unfiltered fraction of these grab samples is analyzed for TP, TN, and TOC. We use these montly grab samples to generate relationships between TP and SRP, and between TN and NO2- + NO3- + NH4+. Dissolved nutrients are measured using standard rapid flow analyzer (RFA) techniques. TP is analyzed with a modified Solorzano and Sharp (1980) technique. TN is measured with an Antec TN analyzer, TOC and DOC are quantified on a Shimadzu TOC Analyzer, and salinity is measured with a YSI conductivity meter. In addition to the regular water quality monitoring, we use the rain level actuators at all freshwater sites to trigger water sampling after rain events exceed a given threshold of duration and/or intensity. As currently programmed, when the threshold of = 2.5 cm of rain per hour is passed, the autosampler at that site collects a 500mL sample 30 minutes after the threshold has been reached. Rain event samples are collected, treated, and analyzed as all other water quality samples.
DOI: 10.5281/zenodo.4041604
title: Long Timescale X-ray Variability of BAL and mini-BAL quasars

description: Serendipitous and targeted <em>Chandra</em> observations of BAL and mini-BAL quasars identified in SDSS DR16Q. This data set contains 293 observations of 93 BAL and mini-BAL quasars. This data accompanies the research note: Timlin, J., Brandt, W.N., Zhu, S. (2020), RNASS, in press.
DOI: 10.5517/ccdc.csd.cc25rcbn
title: CCDC 2018451: Experimental Crystal Structure Determination

description: Related Article: Yue Gui, Xin Yao, Ilia Guzei, Michael M. Aristov, Junguang Yu, Lian Yu|2020|Chem.Mater.|32|7754|doi:10.1021/acs.chemmater.0c02209
DOI: 10.5517/ccdc.csd.cc25rccp
title: CCDC 2018452: Experimental Crystal Structure Determination

description: Related Article: Yue Gui, Xin Yao, Ilia Guzei, Michael M. Aristov, Junguang Yu, Lian Yu|2020|Chem.Mater.|32|7754|doi:10.1021/acs.chemmater.0c02209
DOI: 10.5517/ccdc.csd.cc25rcfr
title: CCDC 2018454: Experimental Crystal Structure Determination

description: Related Article: Yue Gui, Xin Yao, Ilia Guzei, Michael M. Aristov, Junguang Yu, Lian Yu|2020|Chem.Mater.|32|7754|doi:10.1021/acs.chemmater.0c02209
DOI: 10.5517/ccdc.csd.cc20wwf7
title: CCDC 1873839: Experimental Crystal Structure Determination

description: Related Article: Sandeep Kumar, Sadhika Khullar, Sanjay K. Mandal|2020|ACS Omega|5|21873|doi:10.1021/acsomega.0c03065
DOI: 10.5517/ccdc.csd.cc20wwg8
title: CCDC 1873840: Experimental Crystal Structure Determination

description: Related Article: Sandeep Kumar, Sadhika Khullar, Sanjay K. Mandal|2020|ACS Omega|5|21873|doi:10.1021/acsomega.0c03065
DOI: 10.5517/ccdc.csd.cc24lpk0
title: CCDC 1984172: Experimental Crystal Structure Determination

description: Related Article: Magy A. Mekhail, Kristof Pota, Timothy M. Schwartz, Kayla N. Green|2020|RSC Advances|10|31165|doi:10.1039/D0RA05756H
DOI: 10.5281/zenodo.4044627
title: Secondary Vertex Finding in Jets Dataset

description: This is a dataset for developing secondary vertex finding algorithms. The dataset consists of jets sampled from \( pp\rightarrow t\bar{t}\) events at \(\sqrt{s}=14\) TeV. The events are generated with Pythia8 and a basic detector simulation is performed with Delphes emulating a detector similar to ATLAS. Each jet has a set of associated reconstructed tracks. the task of secondary vertex finding is to partition this set according to the point of origin of the particle that created that track. The dataset includes a label for each track that indicates which vertex it originated from. This label is an arbitrary integer - within one jet a pair of tracks originated from the same point if their labels are equal, and from different points otherwise. charged particle tracks are represented by 6 perigee parameters and their covariance matrix. Noise is added to the track perigee parameters with gaussian smearing. The track parameters resolution depends on the transverse momentum and pseudo-rapidity of the track. The covariance matrix is diagonal in this simplified track smearing model.
DOI: 10.5281/zenodo.4044628
title: Secondary Vertex Finding in Jets Dataset

description: This is a dataset for developing secondary vertex finding algorithms. The dataset consists of jets sampled from \( pp\rightarrow t\bar{t}\) events at \(\sqrt{s}=14\) TeV. The events are generated with Pythia8 and a basic detector simulation is performed with Delphes emulating a detector similar to ATLAS. Each jet has a set of associated reconstructed tracks. the task of secondary vertex finding is to partition this set according to the point of origin of the particle that created that track. The dataset includes a label for each track that indicates which vertex it originated from. This label is an arbitrary integer - within one jet a pair of tracks originated from the same point if their labels are equal, and from different points otherwise. charged particle tracks are represented by 6 perigee parameters and their covariance matrix. Noise is added to the track perigee parameters with gaussian smearing. The track parameters resolution depends on the transverse momentum and pseudo-rapidity of the track. The covariance matrix is diagonal in this simplified track smearing model.
DOI: 10.21227/j15a-8r35
title: Waterloo Quality-of-Experience IV

description: The diversity of video delivery pipeline poses a grand challenge to the evaluation of adaptive bitrate (ABR) streaming algorithms and objective quality-of-experience (QoE) models.
DOI: 10.21227/xh0m-wx62
title: Waterloo Quality-of-Experience IV

description: The diversity of video delivery pipeline poses a grand challenge to the evaluation of adaptive bitrate (ABR) streaming algorithms and objective quality-of-experience (QoE) models.
DOI: 10.6084/m9.figshare.12998954.v1
title: R0229_20181030a_videos

description: Rat 0229, skilled reaching data collected in Leventhal laboratory by Alexandra Bova
DOI: 10.5281/zenodo.4047982
title: A video dataset for wooden box assembly

description: This is a dataset of videos for wooden box assembly. The main strength of this dataset is the design of standard and uniform workflow and the use of multiple cameras capturing videos from different angles. A total of 62 videos of 17 subjects were collected. The duration of the videos is 13.0 hours. The whole workflow is designed into nine steps. The dataset contains the videos of the assembly process and the temporal annotation data for the nine steps in each video. Our dataset could be used to facilitate the studies in different applications such as object recognition, human action classification, intelligent automation etc.
description: Funded by Business Finland (1660/31/2018) European Union's Horizon 2020 Research and Innovation Programme (grant number: 777222
DOI: 10.5281/zenodo.4040604
title: A video dataset for wooden box assembly

description: This is a dataset of videos for wooden box assembly. The main strength of this dataset is the design of standard and uniform workflow and the use of multiple cameras capturing videos from different angles. A total of 62 videos of 17 subjects were collected. The duration of the videos is 13.0 hours. The whole workflow is designed into nine steps. The dataset contains the videos of the assembly process and the temporal annotation data for the nine steps in each video. Our dataset could be used to facilitate the studies in different applications such as object recognition, human action classification, intelligent automation etc.
description: Funded by Business Finland (1660/31/2018) European Union's Horizon 2020 Research and Innovation Programme (grant number: 777222
DOI: 10.6084/m9.figshare.13002977
title: Set 1_R1_molecule_info

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13002977.v2
title: Set 1_R1_molecule_info

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13002977.v1
title: molecule_info.h5

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13002962
title: Set 1_R1_filtered_gene

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13002962.v2
title: Set 1_R1_filtered_gene

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13002962.v1
title: filtered_gene_bc_matrices_h5.h5

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13002989
title: Set 1_R1_raw-gene

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13002989.v3
title: Set 1_R1_raw-gene

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.5281/zenodo.3647820
title: Dynamic Contrast Enhanced MRI Raw Data Acquired with 3D Cones Trajectory

description: This repository contains the raw data for the second dynamic contrast enhanced (DCE) MRI in Extreme MRI: Large-Scale Volumetric Dynamic Imaging from Continuous Non-Gated Acquisitions. The data is stored as numpy arrays, containing k-space data (ksp.npy), coordinates (coord.npy), and density compensation factors (dcf.npy). Code to process and reconstruct the data is available here: https://github.com/mikgroup/extreme_mri For more information about how the data is acquired, please see the linked paper.
description: {"references": ["Dataset associated with arXiv:1909.13482"]}
DOI: 10.5281/zenodo.3672170
title: Lung MRI Raw Data Acquired with 3D UTE Radial Trajectory

description: This repository contains the raw data for the second lung MRI result in Extreme MRI: Large-Scale Volumetric Dynamic Imaging from Continuous Non-Gated Acquisitions. The data is stored as numpy arrays, containing k-space data (ksp.npy), coordinates (coord.npy), and density compensation factors (dcf.npy). Code to process and reconstruct the data is available here: https://github.com/mikgroup/extreme_mri For more information about how the data is acquired, please see the linked paper.
DOI: 10.21227/sgc1-c652
title: An Interesting Observation E

description: This dataset is in support of my Research claim - "dersting observation on " , submitted to IEEE Transactions on Circuits and Systems I .Here is the dataset along with supplementary materials relevant to this paper. It includess multimedia, gif images which are the result of the
DOI: 10.6084/m9.figshare.13003064
title: Set 1_R1_web_summary

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13003064.v1
title: Set 1_R1_web_summary

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.34740/kaggle/dsv/1513049
title: Red Wine data set
DOI: 10.6084/m9.figshare.13003067
title: Set 1_R2_filtered_gene

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13003067.v1
title: Set 1_R2_filtered_gene

description: Renal artery stenosis (RAS) engenders stenotic-kidney ischemia, dysfunction, and injury, but whether these are mediated by cellular senescence has not been elucidated. Using INK-ATTAC transgenic mice, high-resolution imaging, and unbiased scRNA-sequencing of murine kidneys, the authors identified cellular senescence as an important mechanism of progressive injury triggered in renal epithelial/stromal cells within post-stenotic kidneys. Both P16-specific and broad quercetin/dasatinib interventions to blunt senescence improved renal function and structure, underscoring its central role in the pathogenesis of the disease. Furthermore, this mechanism was conserved in human subjects with RAS. These observations reveal new mechanisms that contribute to the pathogenesis of chronic ischemic renal injury, and support development of senolytic therapy to reduce senescent cell burden and delay renal injury.
DOI: 10.6084/m9.figshare.13003076
title: Two datasets and the generated result

description: 1_a.json: 1000 random numbers over the range 0-100 1_b.json: new numbers from the original 1000 numbers in 1_a.json using the equation y=3x+6 the result generated by these two datasets
DOI: 10.6084/m9.figshare.13003076.v1
title: Two datasets and the generated result

description: 1_a.json: 1000 random numbers over the range 0-100 1_b.json: new numbers from the original 1000 numbers in 1_a.json using the equation y=3x+6 the result generated by these two datasets