cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.15468/2xx6uw
title: Gabon Biodiversity Collection - Reptilia

description: English:
Gabon Biodiversity Collection is the largest zoological reference collection known in Central Africa. It consist of 123,000 specimen including Mammals, birds, fish, reptiles and amphibians, plants and mainly arthropods. The collection of reptiles, actually has almost 500 specimens. Reptiles, as amphibian are under studied and our hope is to foster more research in the region by making visible what is already available as information. This open source collection, has also the ambition to educate on the various species occurring in Gabon.
 
Français:
La Collection de la Biodiversité du Gabon est la plus grande collection de référence zoologique connue en Afrique Centrale. Elle est composée de 123 000 spécimens comprenant des mammifères, des oiseaux, des poissons, des reptiles, des amphibiens, des plantes et surtout des arthropodes. La collection de reptiles compte actuellement presque 500 spécimens. Tout comme les amphibiens, les reptiles sont peu étudiés et notre espoir est d'encourager la recherche dans la région en rendant visible les informations déjà disponibles. Cette collection, accessible au public, a aussi la prétention d'informer sur les différentes espèces de reptiles présents au Gabon.
DOI: 10.48597/cvtp-hxwn
title: Measurement of Chloride at Sepstrup Sande

description: Measurement of Chloride at Sepstrup Sande
DOI: 10.6084/m9.figshare.20440658.v1
title: Construction of a High-Density Genetic Linkage Map and QTL Analysis of Fiber Yield Traits in Kenaf (<i>Hibiscus Cannabinus</i> L.) via SLAF-seq

description: High-density genetic maps are vital for quantitative trait loci (QTL) mapping, genome assembly, and marker-assisted selection (MAS) in plants. Meanwhile, kenaf (<i>Hibiscus cannabinus</i> L.) is an important crop that produces raw fiber to many industries. However, both high-density genetic map construction and QTL identification have been limited in this crop due to insufficient molecular markers. Here, we constructed a high-density genetic linkage map in kenaf via specific-locus amplified fragment sequencing (SLAF-seq). An F<sub>7:8</sub> mapping population of 138 recombinant inbred lines was developed by crossing between Alian and Fuhong 992. In total, 220,484 high-quality SLAFs were detected, among which 52,832 were polymorphic; 4,167 polymorphic markers were then utilized to construct a genetic map. The assembled genetic map contained 18 linkage groups, spanning 1,952.68 cM with a mean distance of 0.47 cM among the adjacent markers. Phenotypic data from 10 fiber yield-related traits were used for QTL analysis, and a total of 85 QTLs were detected for 10 traits. We were able to construct the densest linkage map yet reported in kenaf and our findings will aid in further QTL mapping, genome comparisons, and MAS breeding in kenaf.
DOI: 10.48597/cyb8-qz4p
title: Measurement of Sulphate_corrected at Keldsnor

description: Measurement of Sulphate_corrected at Keldsnor
DOI: 10.48597/dczr-uzej
title: Measurement of Inorganics in precipitation at Anholt

description: Measurement of Inorganics in precipitation at Anholt
DOI: 10.48597/dz6w-b2py
title: Measurement of Ammonium at Jarczew

description: Measurement of Ammonium at Jarczew
DOI: 10.48597/eu5j-srpz
title: Measurement of Sodium at Keldsnor

description: Measurement of Sodium at Keldsnor
DOI: 10.48597/fqs4-hyre
title: Measurement of Inorganics in precipitation at Ulborg

description: Measurement of Inorganics in precipitation at Ulborg
DOI: 10.48597/h9dp-fham
title: Measurement of Precipitation_amount at Diabla Gora

description: Measurement of Precipitation_amount at Diabla Gora
DOI: 10.48597/hp79-c8w3
title: Measurement of Inorganics in air and particle phase at Tange

description: Measurement of Inorganics in air and particle phase at Tange
DOI: 10.48597/m3e6-3qm5
title: Measurement of Nitrate at Sniezka

description: Measurement of Nitrate at Sniezka
DOI: 10.48597/msr7-pyfw
title: Measurement of Ammonium at Keldsnor

description: Measurement of Ammonium at Keldsnor
DOI: 10.48597/mx3d-fpuk
title: Measurement of Ammonia at Anholt

description: Measurement of Ammonia at Anholt
DOI: 10.48597/n4z7-ndpm
title: Measurement of Magnesium at Keldsnor

description: Measurement of Magnesium at Keldsnor
DOI: 10.48597/nd3g-4hcx
title: Measurement of Sulphate_corrected at Preila

description: Measurement of Sulphate_corrected at Preila
DOI: 10.48597/negr-kjn4
title: Measurement of Chloride at Diabla Gora

description: Measurement of Chloride at Diabla Gora
DOI: 10.15468/wehca5
title: Gabon Biodiversity Collection - Poisson (Fish)

description: English
Gabon Biodiversity Collection is the largest zoological reference collection known in Central Africa. It consist of 123000 specimen including Mammals, birds, fish, reptiles and amphibians, plants and mainly arthropods. The collection of fish actually has over 400 specimen. Fish are under studied and the actual knowledge need to be updated. Our expectation is to make  recent research data available to researchers and students.
Francais
La Collection de la Biodiversité du Gabon est la plus grande collection de référence zoologique connue en Afrique centrale. Elle est composée de plus de 123 000 spécimens comprenant des mammifères, des oiseaux, des poissons, des reptiles et des amphibiens, des plantes et surtout des arthropodes. La collection de poissons compte actuellement plus de 425 spécimens. Les poissons sont peu étudiés et les connaissances actuelles doivent être mises à jour. Notre objectif est de mettre à la disposition des chercheurs et des étudiants les données de recherches récentes.
DOI: 10.48597/ngar-qc22
title: Measurement of pH at Keldsnor

description: Measurement of pH at Keldsnor
DOI: 10.48597/nuf5-jtue
title: Measurement of Calcium at Anholt

description: Measurement of Calcium at Anholt
DOI: 10.48597/p2p4-gt2a
title: Measurement of Ammonia at Ulborg

description: Measurement of Ammonia at Ulborg
DOI: 10.48597/pgrw-bdf5
title: Measurement of Inorganics in precipitation at Diabla Gora

description: Measurement of Inorganics in precipitation at Diabla Gora
DOI: 10.48597/q3hq-39zz
title: Measurement of Sodium at Keldsnor

description: Measurement of Sodium at Keldsnor
DOI: 10.48597/q3u6-4edk
title: Measurement of Potassium at Anholt

description: Measurement of Potassium at Anholt
DOI: 10.48597/qskq-zngs
title: Measurement of Calcium at Ulborg

description: Measurement of Calcium at Ulborg
DOI: 10.48597/rh32-js2y
title: Measurement of Ammonia at Ulborg

description: Measurement of Ammonia at Ulborg
DOI: 10.48597/rhu4-4xwu
title: Measurement of Calcium at Keldsnor

description: Measurement of Calcium at Keldsnor
DOI: 10.48597/rw77-s636
title: Measurement of Precipitation_amount at Leba

description: Measurement of Precipitation_amount at Leba
DOI: 10.48597/th5s-w5wm
title: Measurement of Pm10_mass at Keldsnor

description: Measurement of Pm10_mass at Keldsnor
DOI: 10.6084/m9.figshare.6809186
title: TFEB-dependent induction of thermogenesis by the hepatocyte SLC2A inhibitor trehalose

description: The macroautophagy/autophagy-inducing disaccharide, trehalose, has been proposed to be a promising therapeutic agent against neurodegenerative and cardiometabolic diseases. We recently showed that trehalose attenuates hepatic steatosis in part by blocking hepatocyte glucose transport to induce hepatocyte autophagic flux. However, although every major demonstration of trehalose action invokes activating autophagic flux as its primary function, the mechanism of action of trehalose in whole-body energy metabolism remains poorly defined. Here, we demonstrate that trehalose induces hepatocyte TFEB (transcription factor EB)-dependent thermogenesis <i>in vivo</i>, concomitant with upregulation of hepatic and white adipose expression of UCP1 (uncoupling protein 1 [mitochondrial, protein carrier]). Mechanistically, we provide evidence that hepatocyte fasting transcriptional and metabolic responses depend upon PPARGC1A (peroxisome proliferative activated receptor, gamma, coactivator 1 alpha), TFEB, and FGF21 (fibroblast growth factor 21) signaling. Strikingly, hepatocyte-selective TFEB knockdown abrogated trehalose induction of thermogenesis and white adipose tissue UCP1 upregulation <i>in vivo</i>. In contrast, we found that trehalose action on thermogenesis was independent of LEP (leptin) and the autophagy pathway, as there was robust thermogenic induction in trehalose-treated <i>ob/ob, Becn1, Atg16l1</i>, and <i>Epg5</i> mutant mice. We conclude that trehalose induces metabolically favorable effects on whole-body thermogenesis in part via hepatocyte-centered fasting-like mechanisms that appear to be independent of autophagic flux. Our findings elucidate a novel mechanism by which trehalose acts as a metabolic therapeutic agent by activating hepatic fasting responses. More broadly, the hepatic glucose fasting response may be of clinical utility against overnutrition-driven disease, such as obesity and type 2 diabetes mellitus.
DOI: 10.48597/tjjr-ut69
title: Measurement of Ammonium at Tange

description: Measurement of Ammonium at Tange
DOI: 10.48597/tust-6yks
title: Measurement of Precipitation_amount at Møsvatn

description: Measurement of Precipitation_amount at Møsvatn
DOI: 10.48597/tzdp-7bqq
title: Measurement of Pm25_mass at Birkenes

description: Measurement of Pm25_mass at Birkenes
DOI: 10.48597/tzjd-zmvu
title: Measurement of Sulphate_total at Keldsnor

description: Measurement of Sulphate_total at Keldsnor
DOI: 10.48597/uugz-53f8
title: Measurement of Magnesium at Sepstrup Sande

description: Measurement of Magnesium at Sepstrup Sande
DOI: 10.6084/m9.figshare.6809186.v1
title: TFEB-dependent Induction of Thermogenesis by the Hepatocyte SLC2A Inhibitor Trehalose

description: The macroautophagy/autophagy-inducing disaccharide, trehalose, has been proposed to be a promising therapeutic agent against neurodegenerative and cardiometabolic diseases. We recently showed that trehalose attenuates hepatic steatosis in part by blocking hepatocyte glucose transport to induce hepatocyte autophagic flux. However, although every major demonstration of trehalose action invokes activating autophagic flux as its primary function, the mechanism of action of trehalose in whole-body energy metabolism remains poorly defined. Here, we demonstrate that trehalose induces hepatocyte TFEB (transcription factor EB)-dependent thermogenesis <i>in vivo</i>, concomitant with upregulation of hepatic and white adipose expression of UCP1 (uncoupling protein 1 [mitochondrial, protein carrier]). Mechanistically, we provide evidence that hepatocyte fasting transcriptional and metabolic responses depend upon PPARGC1A (peroxisome proliferative activated receptor, gamma, coactivator 1 alpha), TFEB, and FGF21 (fibroblast growth factor 21) signaling. Strikingly, hepatocyte-selective TFEB knockdown abrogated trehalose induction of thermogenesis and white adipose tissue UCP1 upregulation <i>in vivo</i>. In contrast, we found that trehalose action on thermogenesis was independent of LEP (leptin) and the autophagy pathway, as there was robust thermogenic induction in trehalose-treated <i>ob/ob, Becn1, Atg16l1</i>, and <i>Epg5</i> mutant mice. We conclude that trehalose induces metabolically favorable effects on whole-body thermogenesis in part via hepatocyte-centered fasting-like mechanisms that appear to be independent of autophagic flux. Our findings elucidate a novel mechanism by which trehalose acts as a metabolic therapeutic agent by activating hepatic fasting responses. More broadly, the hepatic glucose fasting response may be of clinical utility against overnutrition-driven disease, such as obesity and type 2 diabetes mellitus.
DOI: 10.48597/w9kx-q732
title: Measurement of Sodium at Diabla Gora

description: Measurement of Sodium at Diabla Gora
DOI: 10.6084/m9.figshare.6809186.v3
title: TFEB-dependent induction of thermogenesis by the hepatocyte SLC2A inhibitor trehalose

description: The macroautophagy/autophagy-inducing disaccharide, trehalose, has been proposed to be a promising therapeutic agent against neurodegenerative and cardiometabolic diseases. We recently showed that trehalose attenuates hepatic steatosis in part by blocking hepatocyte glucose transport to induce hepatocyte autophagic flux. However, although every major demonstration of trehalose action invokes activating autophagic flux as its primary function, the mechanism of action of trehalose in whole-body energy metabolism remains poorly defined. Here, we demonstrate that trehalose induces hepatocyte TFEB (transcription factor EB)-dependent thermogenesis <i>in vivo</i>, concomitant with upregulation of hepatic and white adipose expression of UCP1 (uncoupling protein 1 [mitochondrial, protein carrier]). Mechanistically, we provide evidence that hepatocyte fasting transcriptional and metabolic responses depend upon PPARGC1A (peroxisome proliferative activated receptor, gamma, coactivator 1 alpha), TFEB, and FGF21 (fibroblast growth factor 21) signaling. Strikingly, hepatocyte-selective TFEB knockdown abrogated trehalose induction of thermogenesis and white adipose tissue UCP1 upregulation <i>in vivo</i>. In contrast, we found that trehalose action on thermogenesis was independent of LEP (leptin) and the autophagy pathway, as there was robust thermogenic induction in trehalose-treated <i>ob/ob, Becn1, Atg16l1</i>, and <i>Epg5</i> mutant mice. We conclude that trehalose induces metabolically favorable effects on whole-body thermogenesis in part via hepatocyte-centered fasting-like mechanisms that appear to be independent of autophagic flux. Our findings elucidate a novel mechanism by which trehalose acts as a metabolic therapeutic agent by activating hepatic fasting responses. More broadly, the hepatic glucose fasting response may be of clinical utility against overnutrition-driven disease, such as obesity and type 2 diabetes mellitus.
DOI: 10.6084/m9.figshare.6809186.v2
title: TFEB-dependent induction of thermogenesis by the hepatocyte SLC2A inhibitor trehalose

description: The macroautophagy/autophagy-inducing disaccharide, trehalose, has been proposed to be a promising therapeutic agent against neurodegenerative and cardiometabolic diseases. We recently showed that trehalose attenuates hepatic steatosis in part by blocking hepatocyte glucose transport to induce hepatocyte autophagic flux. However, although every major demonstration of trehalose action invokes activating autophagic flux as its primary function, the mechanism of action of trehalose in whole-body energy metabolism remains poorly defined. Here, we demonstrate that trehalose induces hepatocyte TFEB (transcription factor EB)-dependent thermogenesis <i>in vivo</i>, concomitant with upregulation of hepatic and white adipose expression of UCP1 (uncoupling protein 1 [mitochondrial, protein carrier]). Mechanistically, we provide evidence that hepatocyte fasting transcriptional and metabolic responses depend upon PPARGC1A (peroxisome proliferative activated receptor, gamma, coactivator 1 alpha), TFEB, and FGF21 (fibroblast growth factor 21) signaling. Strikingly, hepatocyte-selective TFEB knockdown abrogated trehalose induction of thermogenesis and white adipose tissue UCP1 upregulation <i>in vivo</i>. In contrast, we found that trehalose action on thermogenesis was independent of LEP (leptin) and the autophagy pathway, as there was robust thermogenic induction in trehalose-treated <i>ob/ob, Becn1, Atg16l1</i>, and <i>Epg5</i> mutant mice. We conclude that trehalose induces metabolically favorable effects on whole-body thermogenesis in part via hepatocyte-centered fasting-like mechanisms that appear to be independent of autophagic flux. Our findings elucidate a novel mechanism by which trehalose acts as a metabolic therapeutic agent by activating hepatic fasting responses. More broadly, the hepatic glucose fasting response may be of clinical utility against overnutrition-driven disease, such as obesity and type 2 diabetes mellitus.
DOI: 10.48597/wred-pz99
title: Measurement of Conductivity at Ispra

description: Measurement of Conductivity at Ispra
DOI: 10.48597/xch4-x5xp
title: Measurement of Sulphate_total at Cabauw Zijdeweg

description: Measurement of Sulphate_total at Cabauw Zijdeweg
DOI: 10.6084/m9.figshare.7333484.v1
title: Additional file 10: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S10. P65 expression in different TNM stage of RCC samples from TCGA RCCÂ dataset. (XLSX 40Â kb)
DOI: 10.6084/m9.figshare.7333484
title: Additional file 10: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S10. P65 expression in different TNM stage of RCC samples from TCGA RCCÂ dataset. (XLSX 40Â kb)
DOI: 10.6084/m9.figshare.7333496
title: Additional file 11: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S11. P65 expression and patientsâ survival time from TCGA RCCÂ dataset. (XLSX 42Â kb)
DOI: 10.6084/m9.figshare.7333496.v1
title: Additional file 11: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S11. P65 expression and patientsâ survival time from TCGA RCCÂ dataset. (XLSX 42Â kb)
DOI: 10.15468/szqjn2
title: 2015_nsmk_smpl_ms

description: National Science Museum 2015
Marine Life Resources Collection List
DOI: 10.6084/m9.figshare.7333505
title: Additional file 12: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S12. SMAD4 expression in RCC and normal renal samples from TCGA RCCÂ dataset. (XLSX 40Â kb)
DOI: 10.6084/m9.figshare.7333505.v1
title: Additional file 12: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S12. SMAD4 expression in RCC and normal renal samples from TCGA RCCÂ dataset. (XLSX 40Â kb)
DOI: 10.6084/m9.figshare.7333517
title: Additional file 13: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S13. SMAD4 expression and paitientsâ survival time from TCGA RCCÂ dataset according to www.proteinatlas.org. (XLSX 31Â kb)
DOI: 10.6084/m9.figshare.7333517.v1
title: Additional file 13: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S13. SMAD4 expression and paitientsâ survival time from TCGA RCCÂ dataset according to www.proteinatlas.org. (XLSX 31Â kb)
DOI: 10.6084/m9.figshare.7333529
title: Additional file 14: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S14. miR-452-5p expression and corresponding SMAD4 expression in RCC and normal renal samples from TCGA RCCÂ dataset. (XLSX 42Â kb)
DOI: 10.6084/m9.figshare.7333529.v1
title: Additional file 14: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S14. miR-452-5p expression and corresponding SMAD4 expression in RCC and normal renal samples from TCGA RCCÂ dataset. (XLSX 42Â kb)
DOI: 10.6084/m9.figshare.7333538
title: Additional file 1: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S1. Sequences of oligonucleotides used in this study. (XLSX 10Â kb)
DOI: 10.6084/m9.figshare.7333538.v1
title: Additional file 1: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S1. Sequences of oligonucleotides used in this study. (XLSX 10Â kb)
DOI: 10.6084/m9.figshare.7333541
title: Additional file 2: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S2-GSE32099. (XLS 113Â kb)
DOI: 10.6084/m9.figshare.7333541.v1
title: Additional file 2: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S2-GSE32099. (XLS 113Â kb)
DOI: 10.6084/m9.figshare.7333544
title: Additional file 3: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S3-GSE37989. (XLS 72Â kb)
DOI: 10.6084/m9.figshare.7333544.v1
title: Additional file 3: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S3-GSE37989. (XLS 72Â kb)
DOI: 10.6084/m9.figshare.7333547
title: Additional file 4: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S4. miR-452-5p expression in RCC and normal renal samples from TCGA RCCÂ dataset. (XLS 137Â kb)
DOI: 10.6084/m9.figshare.7333547.v1
title: Additional file 4: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S4. miR-452-5p expression in RCC and normal renal samples from TCGA RCCÂ dataset. (XLS 137Â kb)
DOI: 10.6084/m9.figshare.7333553
title: Additional file 5: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S5. Clinical characteristics of 102 RCC patients according to miRNA-452-5p expression levels. (XLSX 12Â kb)
DOI: 10.6084/m9.figshare.7333553.v1
title: Additional file 5: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S5. Clinical characteristics of 102 RCC patients according to miRNA-452-5p expression levels. (XLSX 12Â kb)
DOI: 10.15468/v7shjq
title: Protected Fungi of Poland. Distribution, Threats, Conservation Recommendations

description: This dataset contains data on the distribution of protected fungi from Poland, collected in the book ""Protected Fungi of Poland. Distribution, Threats, Conservation Recommendations"". The book is Poland's first comprehensive and extensive monograph covering all fungal species currently protected in the country. The data on distribution of taxa were gathered from published records (MaterialCitation), experts' observations (HumanObservation), and herbarium specimens deposited in public collections (PreservedSpecimens). In this book, all distribution data were presented in ATMOS square system (10 km by 10 km). In this dataset, the centroid of each square (with coordinate uncertainty in meters to cover whole square) is given as coordinate of specific taxon. The monograph is the result of a project under the auspices of the Polish Mycological Society and produced by team of twenty authors, complies as much information as possible about the 117 species of fungi which are currently under legal protection in Poland.

The book should be cited: Kujawa A., Ruszkiewicz-Michalska M., Kałucka I.L. (2020) Grzyby chronione Polski. Rozmieszczenie, zagrożenia, rekomendacje ochronne. Wyd. Instytut Środowiska Rolniczego i Leśnego PAN, pp. 512.

The whole book can be downloaded under following link: http://www.ptmyk.pl/wp-content/uploads/2021/07/GrzybyChronione.pdf "
DOI: 10.6084/m9.figshare.7333559
title: Additional file 6: of Sunitinib-suppressed miR-452-5p facilitates renal cancer cell invasion and metastasis through modulating SMAD4/SMAD7 signals

description: Table S6. Univariate and Multivariate Cox Regression Analyses of miR-452-5p. (XLSX 10Â kb)