cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.5517/ccdc.csd.cc1p8wc6
title: CCDC 1557668: Experimental Crystal Structure Determination

description: Related Article: Mohammed A. Abbas, Colin D. McMillen, Julia L. Brumaghim|2017|Inorg.Chim.Acta|468|308|doi:10.1016/j.ica.2017.07.003
description: An entry from the Cambridge Structural Database, the world’s repository for small molecule crystal structures. The entry contains experimental data from a crystal diffraction study. The deposited dataset for this entry is freely available from the CCDC and typically includes 3D coordinates, cell parameters, space group, experimental conditions and quality measures.
DOI: 10.14457/kmutnb.the.2002.164
title: การปรับปรุงคุณภาพของกระบวนการผลิตชิ้นส่วนหัวอ่านฮาร์ดดิสก์ /
DOI: 10.14457/kmutnb.the.2002.165
title: การเปรียบเทียบประสิทธิภาพของแผนภูมิควบคุมเฉลี่ยสำหรับตรวจวัดกระบวนการที่มีอัตสหสัมพันธ์ /
DOI: 10.5255/ukda-sn-6908-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Local Authority District
DOI: 10.5255/ukda-sn-6909-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Westminster Parliamentary Constituencies
DOI: 10.5255/ukda-sn-6910-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census Area Statistics Wards
DOI: 10.5255/ukda-sn-6911-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2001 Lower Layer Super Output Areas
DOI: 10.5255/ukda-sn-6912-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Local Education Authorities
DOI: 10.5255/ukda-sn-7083-8
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access
DOI: 10.5517/ccdc.csd.cc1pb0yz
title: CCDC 1558771: Experimental Crystal Structure Determination

description: Related Article: Satoshi Sugimoto, Hideki Ohtsu, Kiyoshi Tsuge|2018|J.Photochem.Photobiol.A:Chem.|353|602|doi:10.1016/j.jphotochem.2017.08.002
description: An entry from the Cambridge Structural Database, the world’s repository for small molecule crystal structures. The entry contains experimental data from a crystal diffraction study. The deposited dataset for this entry is freely available from the CCDC and typically includes 3D coordinates, cell parameters, space group, experimental conditions and quality measures.
DOI: 10.14457/kmutnb.the.2003.248
title: การพัฒนาโปรแกรมวิเคราะห์หาพารามิเตอร์ ของระบบเชิงกลและศึกษาพฤติกรรมของแผ่นระนาบผิวเรียบที่ติดแผ่นเทปตัวหน่วง โดยวิธีโพลีเรฟเฟอร์เรนซ์ และวิธีอิบราฮิม /
DOI: 10.5255/ukda-sn-6913-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Strategic Health Authorities
DOI: 10.5255/ukda-sn-6914-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Primary Care Organisations
DOI: 10.14457/kmutnb.the.2002.166
title: ระบบฐานข้อมูลดินเชิงวิศวกรรม เทศบาลอำเภอเมืองตาก จังหวัดตาก /
DOI: 10.14457/kmutnb.the.2002.167
title: สถานะและการเพิ่มพูนสมรรถนะ ในการปฏิบัติงานของวิศวกรฝ่ายโทรศัพท์นครหลวงองค์การโทรศัพท์แห่งประเทศไทย /
DOI: 10.5255/ukda-sn-6915-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2001 Output Area Classification
DOI: 10.6073/pasta/70ac6872ba9bf021375c3fc8ff8ffb36
title: Chlorophyll determined by extraction of samples taken approximately weekly from seawater intake starting at Palmer Station by station personnel including during winter-over period, 1991 - 2017
DOI: 10.14457/kmutnb.the.2002.168
title: การวิเคราะห์และออกแบบวิธีการมอดูเลตกระแสแมกนิไทซิงค์สำหรับบัลลาสต์อิเล็กทรอนิกส์ชนิดกระตุ้นการทำงานด้วยตัวเอง /
DOI: 10.14457/kmutnb.the.2002.169
title: การควบคุมการประหยัดพลังงานในการขับเคลื่อนมอเตอร์เหนี่ยวนำด้วยวิธีปรับตามสนามโดยอ้อม /
DOI: 10.20348/storedb/1053/1091
title: A dose-dependent perturbation in cardiac energy metabolism is linked to radiation-induced ischemic heart disease in Mayak nuclear workers
title: Human Patients raw data

description: Epidemiological studies show a significant increase in ischemic heart disease (IHD) incidence associated with total external gamma-ray dose among Mayak plutonium enrichment plant workers. Our previous studies using mouse models suggest that persistent alteration of heart metabolism due to the inhibition of peroxisome proliferator-activated receptor (PPAR) alpha accompanies cardiac damage after high doses of ionising radiation. The aim of the present study was to elucidate the mechanism of radiation-induced IHD in humans. The cardiac proteome response to irradiation was analysed in Mayak workers who were exposed only to external doses of gamma rays. All participants were diagnosed during their lifetime with IHD that also was the cause of death. Label-free quantitative proteomics analysis was performed on tissue samples from the cardiac left ventricles of individuals stratified into four radiation dose groups (0 Gy, &lt; 100 mGy, 100500 mGy, and &gt; 500 mGy).
The groups could be separated using principal component analysis based on all proteomics features. Proteome profiling showed a dose-dependent increase in the number of downregulated mitochondrial and structural proteins. Both proteomics and immunoblotting showed decreased expression of several oxidative stress responsive proteins in the irradiated hearts. The phosphorylation of transcription factor PPAR alpha was increased in a dose-dependent manner, which is indicative of a reduction in transcriptional activity with increased radiation dose. These data suggest that chronic external radiation enhances the risk for IHD by inhibiting PPAR alpha and altering the expression of mitochondrial, structural, and antioxidant components of the heart.
description: Raw data in FASTQ format
DOI: 10.24388/x10001013
title: C., Herzler, J., Griebel, P., Curran, H., Keromnes, A., Mantzaras, I., H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 0.1, dilution 1:1, 4 bar series

description: Initial composition: H2 O2 Ar
DOI: 10.24388/x10001014
title: C., Herzler, J., Griebel, P., Curran, H., Keromnes, A., Mantzaras, I., H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 0.1, dilution 1:1, 1 bar series

description: Initial composition: H2 O2 Ar
DOI: 10.24388/x10001015
title: C., Herzler, J., Griebel, P., Curran, H., Keromnes, A., Mantzaras, I., H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 4.0, dilution 1:5, 16 bar series

description: Initial composition: H2 O2 Ar
DOI: 10.5255/ukda-sn-6916-5
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Travel to Work Areas
DOI: 10.5255/ukda-sn-7339-3
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2011 Lower Layer Super Output Areas
DOI: 10.5255/ukda-sn-7637-2
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2001 Middle Layer Super Output Areas
DOI: 10.20348/storedb/1053/1087
title: A dose-dependent perturbation in cardiac energy metabolism is linked to radiation-induced ischemic heart disease in Mayak nuclear workers
title: Gene Expression Tables

description: Epidemiological studies show a significant increase in ischemic heart disease (IHD) incidence associated with total external gamma-ray dose among Mayak plutonium enrichment plant workers. Our previous studies using mouse models suggest that persistent alteration of heart metabolism due to the inhibition of peroxisome proliferator-activated receptor (PPAR) alpha accompanies cardiac damage after high doses of ionising radiation. The aim of the present study was to elucidate the mechanism of radiation-induced IHD in humans. The cardiac proteome response to irradiation was analysed in Mayak workers who were exposed only to external doses of gamma rays. All participants were diagnosed during their lifetime with IHD that also was the cause of death. Label-free quantitative proteomics analysis was performed on tissue samples from the cardiac left ventricles of individuals stratified into four radiation dose groups (0 Gy, &lt; 100 mGy, 100500 mGy, and &gt; 500 mGy).
The groups could be separated using principal component analysis based on all proteomics features. Proteome profiling showed a dose-dependent increase in the number of downregulated mitochondrial and structural proteins. Both proteomics and immunoblotting showed decreased expression of several oxidative stress responsive proteins in the irradiated hearts. The phosphorylation of transcription factor PPAR alpha was increased in a dose-dependent manner, which is indicative of a reduction in transcriptional activity with increased radiation dose. These data suggest that chronic external radiation enhances the risk for IHD by inhibiting PPAR alpha and altering the expression of mitochondrial, structural, and antioxidant components of the heart.
description: Summary gene expression tables ( Excel file) for individuals 1-5. 
Sample 8 in RNAseq is number 16 in Mayak manuscript
Sample 9 in RNAseq is number 26 in Mayak manuscript
Sample 12 in RNAseq is number 31 in Mayak manuscript (bad RNA quality)
Sample 13 in RNAseq is number 37 in Mayak manuscript
Sample 14 in RNAseq is number 42 in Mayak manuscript
DOI: 10.20348/storedb/1053/1088
title: A dose-dependent perturbation in cardiac energy metabolism is linked to radiation-induced ischemic heart disease in Mayak nuclear workers
title: Library Quality Control

description: Epidemiological studies show a significant increase in ischemic heart disease (IHD) incidence associated with total external gamma-ray dose among Mayak plutonium enrichment plant workers. Our previous studies using mouse models suggest that persistent alteration of heart metabolism due to the inhibition of peroxisome proliferator-activated receptor (PPAR) alpha accompanies cardiac damage after high doses of ionising radiation. The aim of the present study was to elucidate the mechanism of radiation-induced IHD in humans. The cardiac proteome response to irradiation was analysed in Mayak workers who were exposed only to external doses of gamma rays. All participants were diagnosed during their lifetime with IHD that also was the cause of death. Label-free quantitative proteomics analysis was performed on tissue samples from the cardiac left ventricles of individuals stratified into four radiation dose groups (0 Gy, &lt; 100 mGy, 100500 mGy, and &gt; 500 mGy).
The groups could be separated using principal component analysis based on all proteomics features. Proteome profiling showed a dose-dependent increase in the number of downregulated mitochondrial and structural proteins. Both proteomics and immunoblotting showed decreased expression of several oxidative stress responsive proteins in the irradiated hearts. The phosphorylation of transcription factor PPAR alpha was increased in a dose-dependent manner, which is indicative of a reduction in transcriptional activity with increased radiation dose. These data suggest that chronic external radiation enhances the risk for IHD by inhibiting PPAR alpha and altering the expression of mitochondrial, structural, and antioxidant components of the heart.
description: Quality control data
DOI: 10.20348/storedb/1053/1089
title: A dose-dependent perturbation in cardiac energy metabolism is linked to radiation-induced ischemic heart disease in Mayak nuclear workers
title: Sequencing Quality Control

description: Epidemiological studies show a significant increase in ischemic heart disease (IHD) incidence associated with total external gamma-ray dose among Mayak plutonium enrichment plant workers. Our previous studies using mouse models suggest that persistent alteration of heart metabolism due to the inhibition of peroxisome proliferator-activated receptor (PPAR) alpha accompanies cardiac damage after high doses of ionising radiation. The aim of the present study was to elucidate the mechanism of radiation-induced IHD in humans. The cardiac proteome response to irradiation was analysed in Mayak workers who were exposed only to external doses of gamma rays. All participants were diagnosed during their lifetime with IHD that also was the cause of death. Label-free quantitative proteomics analysis was performed on tissue samples from the cardiac left ventricles of individuals stratified into four radiation dose groups (0 Gy, &lt; 100 mGy, 100500 mGy, and &gt; 500 mGy).
The groups could be separated using principal component analysis based on all proteomics features. Proteome profiling showed a dose-dependent increase in the number of downregulated mitochondrial and structural proteins. Both proteomics and immunoblotting showed decreased expression of several oxidative stress responsive proteins in the irradiated hearts. The phosphorylation of transcription factor PPAR alpha was increased in a dose-dependent manner, which is indicative of a reduction in transcriptional activity with increased radiation dose. These data suggest that chronic external radiation enhances the risk for IHD by inhibiting PPAR alpha and altering the expression of mitochondrial, structural, and antioxidant components of the heart.
description: Quality control data for RNAseq
DOI: 10.20348/storedb/1053/1090
title: A dose-dependent perturbation in cardiac energy metabolism is linked to radiation-induced ischemic heart disease in Mayak nuclear workers
title: Patient Comparisons/Analysis

description: Epidemiological studies show a significant increase in ischemic heart disease (IHD) incidence associated with total external gamma-ray dose among Mayak plutonium enrichment plant workers. Our previous studies using mouse models suggest that persistent alteration of heart metabolism due to the inhibition of peroxisome proliferator-activated receptor (PPAR) alpha accompanies cardiac damage after high doses of ionising radiation. The aim of the present study was to elucidate the mechanism of radiation-induced IHD in humans. The cardiac proteome response to irradiation was analysed in Mayak workers who were exposed only to external doses of gamma rays. All participants were diagnosed during their lifetime with IHD that also was the cause of death. Label-free quantitative proteomics analysis was performed on tissue samples from the cardiac left ventricles of individuals stratified into four radiation dose groups (0 Gy, &lt; 100 mGy, 100500 mGy, and &gt; 500 mGy).
The groups could be separated using principal component analysis based on all proteomics features. Proteome profiling showed a dose-dependent increase in the number of downregulated mitochondrial and structural proteins. Both proteomics and immunoblotting showed decreased expression of several oxidative stress responsive proteins in the irradiated hearts. The phosphorylation of transcription factor PPAR alpha was increased in a dose-dependent manner, which is indicative of a reduction in transcriptional activity with increased radiation dose. These data suggest that chronic external radiation enhances the risk for IHD by inhibiting PPAR alpha and altering the expression of mitochondrial, structural, and antioxidant components of the heart.
description: Principal component analysis/Boxplots/Comparison data
DOI: 10.24388/x10001016
title: C., Herzler, J., Griebel, P., Curran, H., Keromnes, A., Mantzaras, I., H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 4.0, dilution 1:5, 4 bar series

description: Initial composition: H2 O2 Ar
DOI: 10.24388/x10001017
title: C., Herzler, J., Griebel, P., Curran, H., Keromnes, A., Mantzaras, I., H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 4.0, dilution 1:5, 1 bar series

description: Initial composition: H2 O2 Ar
DOI: 10.24388/x10001018
title: C., Herzler, J., Griebel, P., Curran, H., Keromnes, A., Mantzaras, I., H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 0.5, dilution 1:5, 16 bar series

description: Initial composition: H2 O2 N2
DOI: 10.24388/x10001019_x
title: C., Herzler, J., Griebel, P., Curran, H., Keromnes, A., Mantzaras, I., H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 0.5, dilution 1:5, 4 bar series

description: Initial composition: H2 O2 N2
DOI: 10.24388/x10001020
title: Naumann, C., Herzler, J., Griebel, P., Curran, H., Keromnes H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / O2 / Ar, Phi = 0.5, dilution 1:5, 1 bar series

description: Initial composition: H2 O2 N2
DOI: 10.24388/x10001021_x
title: C. Naumann; J. Herzler; P. Griebel; H. Curran; A. Keromnes; I. Mantzaras H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / CO / O2 / Ar, Phi = 1.0, dilution 1:5, H2 /CO = 5/95, 16 bar series

description: Initial composition: H2 CO O2 Ar
DOI: 10.24388/x10001022
title: C. Naumann; J. Herzler; P. Griebel; H. Curran; A. Keromnes; I. Mantzaras H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / CO / O2 / Ar, Phi = 1.0, dilution 1:5, H2 /CO = 5/95, 4 bar series

description: Initial composition: H2 CO O2 Ar
DOI: 10.24388/x10001023
title: C. Naumann; J. Herzler; P. Griebel; H. Curran; A. Keromnes; I. Mantzaras H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / CO / O2 / Ar, Phi = 1.0, dilution 1:5, H2 /CO = 5/95, 1 bar series

description: Initial composition: H2 CO O2 Ar
DOI: 10.24388/x10001024_x
title: C. Naumann; J. Herzler; P. Griebel; H. Curran; A. Keromnes; I. Mantzaras H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / CO / O2 / Ar, Phi = 1.0, dilution 1:5, H2 /CO = 50/50, 16 bar series

description: Initial composition: H2 CO O2 Ar
DOI: 10.5255/ukda-sn-7638-2
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2011 Middle Layer Super Output Areas
DOI: 10.5255/ukda-sn-7639-2
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2011 Output Area Classification
DOI: 10.5255/ukda-sn-7640-2
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2011 Rural-Urban Indicators
DOI: 10.5255/ukda-sn-7641-2
title: Understanding Society: Innovation Panel, Waves 1-9, 2008-2016: Special Licence Access, Census 2001 Rural-Urban Indicators
DOI: 10.14457/cmu.res.2004.583
title: ปัญหาและอุปสรรคในการตรวจพิสูจน์ผู้ติดยาเสพติด ตาม พ.ร.บ. ฟื้นฟูสมรรถภาพผู้ติดยาเสพติด พ.ศ.2545 : กรณีศึกษาสำนักงานคุมประพฤติในเขตพื้นที่ สังกัดสำนักงานคุมประพฤติ ภาค 5 และสำนักงานคุมประพฤติ ภาค 6 /
DOI: 10.14457/cmu.res.2007.376
title: ปัญหาและอุปสรรคในการดำเนินงาน ตามมาตรการด้านการช่วยเหลือผู้ประสบอัคคีภัย ของสำนักงานป้องกันและบรรเทาสาธารณภัย จังหวัดเชียงใหม่ /
DOI: 10.14457/cmu.res.2007.377
title: ปัญหาและอุปสรรคในการนำเสนอและการแสวงหาข้อเท็จจริง และพยานหลักฐานในคดีปกครอง /
DOI: 10.14457/cmu.res.2009.454
title: ปัญหาและอุปสรรคในการดำเนินงานของศูนย์อนามัยที่ 10 จังหวัดเชียงใหม่ กรมอนามัย กระทรวงสาธารณสุข /
DOI: 10.24388/x10001025
title: C. Naumann; J. Herzler; P. Griebel; H. Curran; A. Keromnes; I. Mantzaras H2-IGCC project: Results of ignition delay times for hydrogen-rich and syngas fuel mixtures measured; 2011, Table 3.1, H2 / CO / O2 / Ar, Phi = 1.0, dilution 1:5, H2 /CO = 50/50, 4 bar series

description: Initial composition: H2 CO O2 Ar
DOI: 10.5281/zenodo.546106
title: Dataset From 'Age Effects On Saccadic Adaptation: Evidence From Different Paradigms Reveals Specific Vulnerabilities', Journal Of Vision,  17(6):9, 1-18. Doi: 10.1167/17.6.9

description: Dataset associated with the following publication:

Huang, J., Gegenfurtner K., Schütz, A.C., & Billino, J. (2017). Age effects on saccadic adaptation: Evidence from different paradigms reveals specific vulnerabilities. Journal of Vision, 17(6:9, 1-18. DOI: 10.1167/17.6.9 

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For each experiment one folder is provided.

Each folder contains 2 data files and 1 description file  providing column labels.<br>
One data file contains data on saccadic eye movements, the other one gives measures of executive control.

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description: This research was supported by a grant to Jutta Billino and Karl Gegenfurtner from the German Research Foundation, Collaborative Research Centre SFB/TRR 135: Cardinal Mechanisms of Perception.
DOI: 10.5281/zenodo.599151
title: Dataset From 'Age Effects On Saccadic Adaptation: Evidence From Different Paradigms Reveals Specific Vulnerabilities', Journal Of Vision,  17(6):9, 1-18. Doi: 10.1167/17.6.9

description: Dataset associated with the following publication:

Huang, J., Gegenfurtner K., Schütz, A.C., & Billino, J. (2017). Age effects on saccadic adaptation: Evidence from different paradigms reveals specific vulnerabilities. Journal of Vision, 17(6:9, 1-18. DOI: 10.1167/17.6.9 

--------------------------------------------------------------

For each experiment one folder is provided.

Each folder contains 2 data files and 1 description file  providing column labels.<br>
One data file contains data on saccadic eye movements, the other one gives measures of executive control.

-------------------------------------------------------------
description: This research was supported by a grant to Jutta Billino and Karl Gegenfurtner from the German Research Foundation, Collaborative Research Centre SFB/TRR 135: Cardinal Mechanisms of Perception.
DOI: 10.5281/zenodo.840844
title: Transcripts Of Expert Interviews With Writing Center Directors In The Usa In 2012

description: This document contains transcripts of interviews with 16 writing center directors in different writing centers in the USA, conducted in 2012. The overall research question for the interviews was how writing center directors deal with challenges in writing center work and how they conduct institutional work for their writing centers.

The interviews belong to the habilitation thesis <em>“Strategien für die Institutionalisierung von Schreibzentren an Hochschulen. Eine qualitative Analyse der Institutionalisierungsarbeit von Leitungspersonen in Schreibzentren“</em>, as proposed to the <em>Fakultätsrat der Kultur-, Sozial- und Bildungswissenschaftlichen Fakultät</em> of the Humboldt University Berlin, April 1st 2016. The study has been accepted as habilitation by the <em>Fakultätsrat</em> at February 15<sup>th</sup> 2017.

The results of the study will be published as open access publication (e-book) and in print by W.Bertelsmann Verlag (wbv) Bielefeld in October 2017. The study contains a description of demographic details and other details of the interviews and explains the methodology, which clarifies why the interview transcripts eventually change to part-transcriptions due to theoretical saturation.
description: Data gathering for this research project was funded by the German Research Foundation (DFG), GI 861/1-1, project title: "Erfolgreiche Implementierung von Schreibzentren an US-amerikanischen Hochschulen: Eine explorative Studie auf der Basis von Experteninterviews. Ein Beitrag zur Schreibzentrumsforschung mit Transfer zur europäischen Hochentwicklung im Bologna-Prozess."