cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.16943750
title: Salicylic acid and the viral virulence factor 2b regulate the divergent roles of autophagy during cucumber mosaic virus infection

description: Macroautophagy/autophagy is a conserved intracellular degradation pathway that has recently emerged as an integral part of plant responses to virus infection. The known mechanisms of autophagy range from the selective degradation of viral components to a more general attenuation of disease symptoms. In addition, several viruses are able to manipulate the autophagy machinery and counteract autophagy-dependent resistance. Despite these findings, the complex interplay of autophagy activities, viral pathogenicity factors, and host defense pathways in disease development remains poorly understood. In the current study, we analyzed the interaction between autophagy and cucumber mosaic virus (CMV) in <i>Arabidopsis thaliana</i>. We show that autophagy is induced during CMV infection and promotes the turnover of the major virulence protein and RNA silencing suppressor 2b. Intriguingly, autophagy induction is mediated by salicylic acid (SA) and dampened by the CMV virulence factor 2b. In accordance with 2b degradation, we found that autophagy provides resistance against CMV by reducing viral RNA accumulation in an RNA silencing-dependent manner. Moreover, autophagy and RNA silencing attenuate while SA promotes CMV disease symptoms, and epistasis analysis suggests that autophagy-dependent disease and resistance are uncoupled. We propose that autophagy counteracts CMV virulence via both 2b degradation and reduced SA-responses, thereby increasing plant fitness with the viral trade-off arising from increased RNA silencing-mediated resistance. <b>Abbreviations:</b> AGO1: argonaute1; ANOVA: analysis of variance; ATG: autophagy related; AZD: AZD8055; CMV: cucumber mosaic virus; CaMV: cauliflower mosaic virus; Co-IP: Co-immunoprecipitation; ConA: concanamycin A; CP: coat protein; DAI: days after inoculation; DCL2/DCL4: dicer like 2/ dicer like 4; DMSO: dimethyl sulfoxide; FLUC: firefly luciferase; GFP: green fluorescent protein; GUS: β-glucuronidase; h: hours; NahG: salicylate hydroxylase; NBR1: neighbor of BRCA1; NPR1: non-expressor of pathogensis related 1; PR1: pathogenesis related 1; RDR6: RNA dependent RNA polymerase 6; RFP: red fluorescent protein; RLUC: renilla luciferase; SA: salicylic acid; SGS3: suppressor of gene silencing 3; TuMV: turnip mosaic virus; WT: wild type
DOI: 10.6084/m9.figshare.16943750.v1
title: Salicylic acid and the viral virulence factor 2b regulate the divergent roles of autophagy during cucumber mosaic virus infection

description: Macroautophagy/autophagy is a conserved intracellular degradation pathway that has recently emerged as an integral part of plant responses to virus infection. The known mechanisms of autophagy range from the selective degradation of viral components to a more general attenuation of disease symptoms. In addition, several viruses are able to manipulate the autophagy machinery and counteract autophagy-dependent resistance. Despite these findings, the complex interplay of autophagy activities, viral pathogenicity factors, and host defense pathways in disease development remains poorly understood. In the current study, we analyzed the interaction between autophagy and cucumber mosaic virus (CMV) in <i>Arabidopsis thaliana</i>. We show that autophagy is induced during CMV infection and promotes the turnover of the major virulence protein and RNA silencing suppressor 2b. Intriguingly, autophagy induction is mediated by salicylic acid (SA) and dampened by the CMV virulence factor 2b. In accordance with 2b degradation, we found that autophagy provides resistance against CMV by reducing viral RNA accumulation in an RNA silencing-dependent manner. Moreover, autophagy and RNA silencing attenuate while SA promotes CMV disease symptoms, and epistasis analysis suggests that autophagy-dependent disease and resistance are uncoupled. We propose that autophagy counteracts CMV virulence via both 2b degradation and reduced SA-responses, thereby increasing plant fitness with the viral trade-off arising from increased RNA silencing-mediated resistance. <b>Abbreviations:</b> AGO1: argonaute1; ANOVA: analysis of variance; ATG: autophagy related; AZD: AZD8055; CMV: cucumber mosaic virus; CaMV: cauliflower mosaic virus; Co-IP: Co-immunoprecipitation; ConA: concanamycin A; CP: coat protein; DAI: days after inoculation; DCL2/DCL4: dicer like 2/ dicer like 4; DMSO: dimethyl sulfoxide; FLUC: firefly luciferase; GFP: green fluorescent protein; GUS: β-glucuronidase; h: hours; NahG: salicylate hydroxylase; NBR1: neighbor of BRCA1; NPR1: non-expressor of pathogensis related 1; PR1: pathogenesis related 1; RDR6: RNA dependent RNA polymerase 6; RFP: red fluorescent protein; RLUC: renilla luciferase; SA: salicylic acid; SGS3: suppressor of gene silencing 3; TuMV: turnip mosaic virus; WT: wild type
DOI: 10.6084/m9.figshare.16635159.v2
title: Autophagy inhibition by TSSC4 (tumor suppressing subtransferable candidate 4) contributes to sustainable cancer cell growth

description: Cancer cell growth is dependent upon the sustainability of proliferative signaling and resisting cell death. Macroautophagy/autophagy promotes cancer cell growth by providing nutrients to cells and preventing cell death. This is in contrast to autophagy promoting cell death under some conditions. The mechanism regulating autophagy-mediated cancer cell growth remains unclear. Herein, we demonstrate that TSSC4 (tumor suppressing subtransferable candidate 4) is a novel tumor suppressor that suppresses cancer cell growth and tumor growth and prevents cell death induction during excessive growth by inhibiting autophagy. The oncogenic proteins ERBB2 (erb-b2 receptor tyrosine kinase 2) and the activation EGFR mutant (EGFRvIII, epidermal growth factor receptor variant III) promote cell growth and TSSC4 expression in breast cancer and glioblastoma multiforme (GBM) cells, respectively. In EGFRvIII-expressing GBM cells, <i>TSSC4</i> knockout shifted the function of autophagy from a pro-cell survival role to a pro-cell death role during prolonged cell growth. Furthermore, the interaction of TSSC4 with MAP1LC3/LC3 (microtubule associated protein 1 light chain 3) via its conserved LC3-interacting region (LIR) contributes to its inhibition of autophagy. Finally, TSSC4 suppresses tumorsphere formation and tumor growth by inhibiting autophagy and maintaining cell survival in tumorspheres. Taken together, sustainable cancer cell growth can be achieved by autophagy inhibition via TSSC4 expression. <b>Abbreviations:</b> 3-MA: 3-methyladenine; ACTB: actin beta; CQ: chloroquine; EGFRvIII: epidermal growth factor receptor variant III; ERBB2: erb-b2 receptor tyrosine kinase 2; GBM: glioblastoma multiforme; LIR: LC3-interacting region; MAP1LC3/LC3: microtubule Associated protein 1 light chain 3; TSSC4: tumor suppressing subtransferable candidate 4
DOI: 10.6084/m9.figshare.16635159.v1
title: Autophagy inhibition by TSSC4 (tumor suppressing subtransferable candidate 4) contributes to sustainable cancer cell growth

description: Cancer cell growth is dependent upon the sustainability of proliferative signaling and resisting cell death. Macroautophagy/autophagy promotes cancer cell growth by providing nutrients to cells and preventing cell death. This is in contrast to autophagy promoting cell death under some conditions. The mechanism regulating autophagy-mediated cancer cell growth remains unclear. Herein, we demonstrate that TSSC4 (tumor suppressing subtransferable candidate 4) is a novel tumor suppressor that suppresses cancer cell growth and tumor growth and prevents cell death induction during excessive growth by inhibiting autophagy. The oncogenic proteins ERBB2 (erb-b2 receptor tyrosine kinase 2) and the activation EGFR mutant (EGFRvIII, epidermal growth factor receptor variant III) promote cell growth and TSSC4 expression in breast cancer and glioblastoma multiforme (GBM) cells, respectively. In EGFRvIII-expressing GBM cells, <i>TSSC4</i> knockout shifted the function of autophagy from a pro-cell survival role to a pro-cell death role during prolonged cell growth. Furthermore, the interaction of TSSC4 with MAP1LC3/LC3 (microtubule associated protein 1 light chain 3) via its conserved LC3-interacting region (LIR) contributes to its inhibition of autophagy. Finally, TSSC4 suppresses tumorsphere formation and tumor growth by inhibiting autophagy and maintaining cell survival in tumorspheres. Taken together, sustainable cancer cell growth can be achieved by autophagy inhibition via TSSC4 expression. <b>ABBREVIATIONS</b>: 3-MA: 3-methyladenine; ACTB: actin beta; CQ: chloroquine; EGFRvIII: epidermal growth factor receptor variant III; ERBB2: erb-b2 receptor tyrosine kinase 2; GBM: glioblastoma multiforme; LIR: LC3-interacting region; MAP1LC3/LC3: microtubule Associated protein 1 light chain 3; TSSC4: tumor suppressing subtransferable candidate 4
DOI: 10.6073/pasta/beb2eac54c6f02e1ff6206b3ec21e0d3
title: SBC LTER: Beach: Distribution of terrestrial organic material in intertidal and nearshore marine sediment due to debris flow response efforts

description: These data describe the distribution and processing of terrestrial organic material observed in the Santa Barbara Channel (Goleta Bay) during the spring of 2018. Specifically, beach, slough, and marine sediments were sampled during and after the debris disposal event that took place between January and February 2018 following the Thomas Fire and the subsequent Montecito Debris Flow. Data are contained in one table, including organic carbon, carbon isotope, lignin phenol, and pyrogenic carbon measurements analyzed from sediment cores collected at different depths and locations in the nearshore region.
DOI: 10.6084/m9.figshare.20102912
title: BWH_06/2022_7T

description: 7T structural scans acquired with focus on subcortical contrast.
DOI: 10.6084/m9.figshare.20102912.v1
title: BWH_06/2022_7T

description: 7T structural scans acquired with focus on subcortical contrast.
DOI: 10.17894/ucph.66293057-46c5-4d02-a116-dfd597ce5a78
title: Questionnaire data from H2020 INTEGRITY WP2
DOI: 10.5281/zenodo.6687136
title: Hippocampus barbouri decontaminated gx

description: .
DOI: 10.5281/zenodo.6687135
title: Hippocampus barbouri decontaminated gx

description: .
DOI: 10.5281/zenodo.6687146
title: Pandion haliaetus decontaminated gx

description: .
DOI: 10.5281/zenodo.6687145
title: Pandion haliaetus decontaminated gx

description: .
DOI: 10.5281/zenodo.6687164
title: Enypniastes eximia decontaminated gx

description: .
DOI: 10.5281/zenodo.6687163
title: Enypniastes eximia decontaminated gx

description: .
DOI: 10.6084/m9.figshare.19768501
title: Data

description: <strong>Influenza vaccine as predisposing factor for Guillain-Barre syndrome: a systematic review and meta-analysis</strong> <br> Luis M. Herrera-Alvitres<sup>a</sup>, Miguel A. Tresierra-Ayala<sup>b</sup>, Raul M. Herrera-Edquen<sup>c</sup>, Christian R. Mejia<sup>d</sup>. <br> <strong>Affiliations</strong> a. Escuela de Medicina, Universidad Privada Antenor Orrego, Trujillo, Perú. ORCID: 0000-0003-3727-6097. Postal address: Urb. Las Palmas II, Mz. C, Lt. 12. Correspondence: lherreraa1@upao.edu.pe b. Escuela de Medicina, Universidad Privada Antenor Orrego, Trujillo, Perú. ORCID: 0000-0003-2351-527X. Correspondence: mtresierraa@upao.edu.pe c. Hospital de Apoyo Recuay, Ministerio de Salud, Recuay, Perú. ORCID: 0000-0003-0810-2071. d. Centro de Investigación de Medicina Traslacional, Universidad Norbert Wiener, Lima, Perú. ORCID: 0000-0002-5940-7281. <br> <strong>Keywords</strong> Influenza Vaccines, Guillain-Barre Syndrome, Systematic Review, Meta-Analysis, Observational Study. <br>
DOI: 10.6084/m9.figshare.19768501.v1
title: Data

description: <strong>Influenza vaccine as predisposing factor for Guillain-Barre syndrome: a systematic review and meta-analysis</strong> <br> Luis M. Herrera-Alvitres<sup>a</sup>, Miguel A. Tresierra-Ayala<sup>b</sup>, Raul M. Herrera-Edquen<sup>c</sup>, Christian R. Mejia<sup>d</sup>. <br> <strong>Affiliations</strong> a. Escuela de Medicina, Universidad Privada Antenor Orrego, Trujillo, Perú. ORCID: 0000-0003-3727-6097. Postal address: Urb. Las Palmas II, Mz. C, Lt. 12. Correspondence: lherreraa1@upao.edu.pe b. Escuela de Medicina, Universidad Privada Antenor Orrego, Trujillo, Perú. ORCID: 0000-0003-2351-527X. Correspondence: mtresierraa@upao.edu.pe c. Hospital de Apoyo Recuay, Ministerio de Salud, Recuay, Perú. ORCID: 0000-0003-0810-2071. d. Centro de Investigación de Medicina Traslacional, Universidad Norbert Wiener, Lima, Perú. ORCID: 0000-0002-5940-7281. <br> <strong>Keywords</strong> Influenza Vaccines, Guillain-Barre Syndrome, Systematic Review, Meta-Analysis, Observational Study. <br>
DOI: 10.21227/6ff3-9682
title: Educating youth about human impact on freshwater ecosystems using an online serious game 

description: We developed an online serious game to educate adolescents regarding human impact on freshwater ecosystems. Here, we provide the Unity codes for building the game. We also provide a data set containing survey responses of 41 middle school students who participated in a study that assesses the effectiveness of the game, along with MatLab and R codes that analyze these data.
DOI: 10.5281/zenodo.6687225
title: [IO Islamic 868] Akhbâr-alaṣfiyâ

description: <strong>Akhbâr-alaṣfiyâ .</strong> <strong>This manuscript is now IO Islamic 641 in the India Office collections.</strong> <strong>[metadata:</strong> <strong>Hermann Ethé</strong><strong><em>, </em></strong><strong><em>Catalogue of Persian Manuscripts in the Library of the India Office,</em></strong> <strong>2 vols. (Oxford: India Office, 1903): volume 1,</strong> <strong>number 868 here with notations and hyperlinks]</strong>. <br> 641 Akhbâr-alaṣfiyâ (اخبار الاصفيا ) . A much smaller and less known work, of very similar contents, giving likewise biographies of all the famous Saints and Shaikhs who have lived or stayed in <em>India</em>, compiled by ‘Abd-alṣamad bin Afḍal Muḥammad bin Yûsuf Anṣârî, a nephew of the famous Abû-alfḍl and editor of his official letters, the مكاتبات علاّمى , see above, Nos. 271-286 in this Cat., compare also Rieu iii. p. 1087<sup>a</sup>, where as title of the present work is given: اخبارات الاصفيا . It was completed A.H. 1014 (A.D.1605, 1606) and dedicated to the emperor Jahângîr, see fol. 1<sup>b</sup>, fol. 6<sup>b</sup>, last line, fol. 7<sup>a</sup>, ll. 15 and 16, and fol. 7<sup>b</sup>, ll. 14 and 15. It contains short biographical accounts of 245 male and 4 female persons, beginning, like the preceding work, with Ghauth alṣamadânî Ḳuṭb-i-rabbânî Shaikh Muḥyî-aldîn Abû Muḥammad ‘Abd-alḳâdir alḥasanî alḥusainî aljilânî ( born A.H. 471 = A.D. 1079, died A.H. 561 = A.D. 1166 ), see fol. 8<sup>b</sup>, and ends with Fâṭimah Sâlimah, on fol. 70<sup>a</sup>. A complete index on ff. 1<sup>b</sup>-5<sup>a</sup>; the work itself begins thus, on fol. 6<sup>b</sup>: جهان جهان آفرين مرجان و جهان آفرين را كه جهان دانش و جان سخن بصاحب فطرتان الخ , and concludes on fol. 70<sup>b</sup>. Ff. 71<sup>b</sup>-74<sup>b</sup> are filled by another hand with a story of Shaikh Sharaf-aldîn Abû ‘Alî Ḳalandar of Pânîpat (who died A.H. 724=A.D. 1324, see Rieu ii. p. 668, and iii. p. 1090<sup>b</sup> sq.). Copied by Bâḳir Sâmânî, in the thirty-first year of ‘Âlamgîr’s reign (A.H. 1098, 1099 = A.D. 1687, 1688). No. 868, ff. 74, ll. 19-28; very irregularly and unequally written in Shikasta; size, 10<sup>5/8</sup> in. by 6<sup>3/8</sup> in.
DOI: 10.5281/zenodo.6687224
title: [IO Islamic 868] Akhbâr-alaṣfiyâ

description: <strong>Akhbâr-alaṣfiyâ .</strong> <strong>This manuscript is now IO Islamic 641 in the India Office collections.</strong> <strong>[metadata:</strong> <strong>Hermann Ethé</strong><strong><em>, </em></strong><strong><em>Catalogue of Persian Manuscripts in the Library of the India Office,</em></strong> <strong>2 vols. (Oxford: India Office, 1903): volume 1,</strong> <strong>number 868 here with notations and hyperlinks]</strong>. <br> 641 Akhbâr-alaṣfiyâ (اخبار الاصفيا ) . A much smaller and less known work, of very similar contents, giving likewise biographies of all the famous Saints and Shaikhs who have lived or stayed in <em>India</em>, compiled by ‘Abd-alṣamad bin Afḍal Muḥammad bin Yûsuf Anṣârî, a nephew of the famous Abû-alfḍl and editor of his official letters, the مكاتبات علاّمى , see above, Nos. 271-286 in this Cat., compare also Rieu iii. p. 1087<sup>a</sup>, where as title of the present work is given: اخبارات الاصفيا . It was completed A.H. 1014 (A.D.1605, 1606) and dedicated to the emperor Jahângîr, see fol. 1<sup>b</sup>, fol. 6<sup>b</sup>, last line, fol. 7<sup>a</sup>, ll. 15 and 16, and fol. 7<sup>b</sup>, ll. 14 and 15. It contains short biographical accounts of 245 male and 4 female persons, beginning, like the preceding work, with Ghauth alṣamadânî Ḳuṭb-i-rabbânî Shaikh Muḥyî-aldîn Abû Muḥammad ‘Abd-alḳâdir alḥasanî alḥusainî aljilânî ( born A.H. 471 = A.D. 1079, died A.H. 561 = A.D. 1166 ), see fol. 8<sup>b</sup>, and ends with Fâṭimah Sâlimah, on fol. 70<sup>a</sup>. A complete index on ff. 1<sup>b</sup>-5<sup>a</sup>; the work itself begins thus, on fol. 6<sup>b</sup>: جهان جهان آفرين مرجان و جهان آفرين را كه جهان دانش و جان سخن بصاحب فطرتان الخ , and concludes on fol. 70<sup>b</sup>. Ff. 71<sup>b</sup>-74<sup>b</sup> are filled by another hand with a story of Shaikh Sharaf-aldîn Abû ‘Alî Ḳalandar of Pânîpat (who died A.H. 724=A.D. 1324, see Rieu ii. p. 668, and iii. p. 1090<sup>b</sup> sq.). Copied by Bâḳir Sâmânî, in the thirty-first year of ‘Âlamgîr’s reign (A.H. 1098, 1099 = A.D. 1687, 1688). No. 868, ff. 74, ll. 19-28; very irregularly and unequally written in Shikasta; size, 10<sup>5/8</sup> in. by 6<sup>3/8</sup> in.
DOI: 10.5281/zenodo.6687174
title: Harpyhaliaetus solitarius decontaminated gx

description: .
DOI: 10.5281/zenodo.6687173
title: Harpyhaliaetus solitarius decontaminated gx

description: .
DOI: 10.5281/zenodo.6687286
title: Falco deiroleucus decontaminated gx

description: .
DOI: 10.5281/zenodo.6687285
title: Falco deiroleucus decontaminated gx

description: .
DOI: 10.6084/m9.figshare.20126474
title: Factors controlling the effects of mutualistic bacteria on plants associated with fungi

description: Data for Figures 2, 3, and 4.
DOI: 10.5281/zenodo.6687314
title: Tyrannus tyrannus decontaminated gx

description: .
DOI: 10.5281/zenodo.6687313
title: Tyrannus tyrannus decontaminated gx

description: .
DOI: 10.5281/zenodo.6687346
title: Toxicodendron radicans decontaminated gx

description: .
DOI: 10.5281/zenodo.6687345
title: Toxicodendron radicans decontaminated gx

description: .
DOI: 10.6084/m9.figshare.3761562.v2257
title: Online Labour Index: Measuring the Online Gig Economy for Policy and Research

description: Data repository for the data underlying the Online Labour Index. See http://ilabour.oii.ox.ac.uk online-labour-index/ for details.
DOI: 10.5281/zenodo.6687360
title: Leptodoras nelsoni decontaminated gx

description: .
DOI: 10.5281/zenodo.6687359
title: Leptodoras nelsoni decontaminated gx

description: .
DOI: 10.6084/m9.figshare.20126492
title: hTERT RPE cells scEU-seq AnnData

description: A scEU-seq data set from immortalized human retinal pigment epithelium (RPE) cells, published by (Battich et al., 2020).
DOI: 10.6084/m9.figshare.20126492.v1
title: hTERT RPE cells scEU-seq AnnData

description: A scEU-seq data set from immortalized human retinal pigment epithelium (RPE) cells, published by (Battich et al., 2020).
DOI: 10.5281/zenodo.6687391
title: Ceyx melanurus decontaminated gx

description: .
DOI: 10.5281/zenodo.6687390
title: Ceyx melanurus decontaminated gx

description: .
DOI: 10.3886/e120462v1-121440
title: National Neighborhood Data Archive (NaNDA): Socioeconomic Status and Demographic Characteristics of ZIP Code Tabulation Areas, United States, 2008-2017

description: This dataset contains measures of socioeconomic and demographic characteristics by US ZIP code tabulation area (ZCTA) for the years 2008-2017. Example measures include population density; population distribution by race, ethnicity, age, and income; and proportion of population living below the poverty level, receiving public assistance, and female-headed families. The dataset also contains a set of index variables to represent neighborhood disadvantage and affluence.
description: ZCTAs in the United States. Smallest Geographic Unit: ZIP code tabulation area
description: A ZIP code to ZCTA crosswalk must be used to combine this dataset with ZIP code geocoded data. Such a crosswalk is available on the UDS Mapper website at https://udsmapper.org/zip-code-to-zcta-crosswalk/. Sample code for merging the UDS Mapper crosswalk with NaNDA datasets is available on the NaNDA repository at https://www.openicpsr.org/openicpsr/project/120088/.
DOI: 10.6084/m9.figshare.20126504
title: Pnacreatic Endocrine Development scRNAseq AnnData

description: Data from `Bastidas-Ponce et al. (2019) https://doi.org/10.1242/dev.173849. Pancreatic epithelial and Ngn3-Venus fusion (NVF) cells during secondary transition with transcriptome profiles sampled from embryonic day 15.5. Endocrine cells are derived from endocrine progenitors located in the pancreatic epithelium. Endocrine commitment terminates in four major fates: glucagon- producing α-cells, insulin-producing β-cells, somatostatin-producing δ-cells and ghrelin-producing ε-cells.
DOI: 10.6084/m9.figshare.20126504.v1
title: Pnacreatic Endocrine Development scRNAseq AnnData

description: Data from `Bastidas-Ponce et al. (2019) https://doi.org/10.1242/dev.173849. Pancreatic epithelial and Ngn3-Venus fusion (NVF) cells during secondary transition with transcriptome profiles sampled from embryonic day 15.5. Endocrine cells are derived from endocrine progenitors located in the pancreatic epithelium. Endocrine commitment terminates in four major fates: glucagon- producing α-cells, insulin-producing β-cells, somatostatin-producing δ-cells and ghrelin-producing ε-cells.
DOI: 10.5281/zenodo.6687403
title: Graphium evemon decontaminated gx

description: .
DOI: 10.5281/zenodo.6687402
title: Graphium evemon decontaminated gx

description: .
DOI: 10.5281/zenodo.6687503
title: Research Organization Registry (ROR) Data in Sqlite Database

description: Version 1.1 of the Research Organization Registry data translated into a SQLite database using ROR2DB (https://github.com/Metadata-Game-Changers/ROR2DB). This version adds country, country_code, and status to the ror table.
DOI: 10.5281/zenodo.6672174
title: Research Organization Registry (ROR) Data in Sqlite Database

description: Version 1.1 of the Research Organization Registry data translated into a SQLite database using ROR2DB (https://github.com/Metadata-Game-Changers/ROR2DB). This version adds country, country_code, and status to the ror table.
DOI: 10.5281/zenodo.6687342
title: Future Food Security in Africa under Climate Change

description: This excel file contains data tables (S2-S3) also found in the supplementary materials of the publication titled "Future Food Security in Africa under Climate Change". The tables included here include a regional breakdown of African countries (Table S2), available calories for direct or indirect human consumption under diverse food loss and waste pathways (Table S3), and data on national caloric deficits under different scenarios (Table S4).
DOI: 10.5281/zenodo.6687341
title: Future Food Security in Africa under Climate Change

description: This excel file contains data tables (S2-S3) also found in the supplementary materials of the publication titled "Future Food Security in Africa under Climate Change". The tables included here include a regional breakdown of African countries (Table S2), available calories for direct or indirect human consumption under diverse food loss and waste pathways (Table S3), and data on national caloric deficits under different scenarios (Table S4).
DOI: 10.6084/m9.figshare.20122967
title: supp_data_pigc_OLpaper.zip

description: Expanded simulation and measurement data.
DOI: 10.6084/m9.figshare.20122967.v1
title: supp_data_pigc_OLpaper.zip

description: Expanded simulation and measurement data.