cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.17188/1698684
title: Materials Data on LiGaO10 by Materials Project

description: LiGaO8O2 crystallizes in the monoclinic Cc space group. The structure is two-dimensional and consists of eight hydrogen peroxide molecules and two LiGaO8 sheets oriented in the (0, 0, 1) direction. In each LiGaO8 sheet, there are two inequivalent Li sites. In the first Li site, Li is bonded in a 5-coordinate geometry to five O atoms. There are a spread of Li–O bond distances ranging from 2.03–2.55 Å. In the second Li site, Li is bonded in a distorted T-shaped geometry to three O atoms. There are a spread of Li–O bond distances ranging from 1.99–2.06 Å. There are two inequivalent Ga sites. In the first Ga site, Ga is bonded in a tetrahedral geometry to four O atoms. There are a spread of Ga–O bond distances ranging from 1.90–1.97 Å. In the second Ga site, Ga is bonded in a 4-coordinate geometry to four O atoms. There are a spread of Ga–O bond distances ranging from 1.71–2.10 Å. There are sixteen inequivalent O sites. In the first O site, O is bonded in a water-like geometry to two O atoms. There is one shorter (1.40 Å) and one longer (1.53 Å) O–O bond length. In the second O site, O is bonded in a distorted bent 120 degrees geometry to one Li and one O atom. The O–O bond length is 1.30 Å. In the third O site, O is bonded in a 1-coordinate geometry to one Li and one O atom. The O–O bond length is 1.31 Å. In the fourth O site, O is bonded in a distorted trigonal planar geometry to one Li, one Ga, and one O atom. The O–O bond length is 1.48 Å. In the fifth O site, O is bonded in a bent 120 degrees geometry to two O atoms. There is one shorter (1.28 Å) and one longer (1.53 Å) O–O bond length. In the sixth O site, O is bonded in a distorted trigonal non-coplanar geometry to one Li, one Ga, and one O atom. In the seventh O site, O is bonded in a distorted bent 150 degrees geometry to one Li and one O atom. In the eighth O site, O is bonded in a bent 120 degrees geometry to one Li and one O atom. The O–O bond length is 1.32 Å. In the ninth O site, O is bonded in a water-like geometry to two O atoms. In the tenth O site, O is bonded in a water-like geometry to one Ga and one O atom. In the eleventh O site, O is bonded in a distorted bent 150 degrees geometry to one Ga and one O atom. The O–O bond length is 1.27 Å. In the twelfth O site, O is bonded in a single-bond geometry to one Ga atom. In the thirteenth O site, O is bonded in a bent 150 degrees geometry to one Li and one O atom. In the fourteenth O site, O is bonded in a distorted water-like geometry to one Li, one Ga, and one O atom. In the fifteenth O site, O is bonded in a distorted bent 120 degrees geometry to one Ga and one O atom. In the sixteenth O site, O is bonded in a water-like geometry to one Ga and one O atom.
DOI: 10.17188/1698685
title: Materials Data on InAg2Au by Materials Project

description: AuAg2In crystallizes in the orthorhombic Immm space group. The structure is one-dimensional and consists of two AuAg2In ribbons oriented in the (1, 0, 0) direction. Au is bonded in a linear geometry to two equivalent Ag atoms. Both Au–Ag bond lengths are 2.66 Å. Ag is bonded in a linear geometry to one Au and one In atom. The Ag–In bond length is 2.64 Å. In is bonded in a linear geometry to two equivalent Ag atoms.
DOI: 10.17188/1698686
title: Materials Data on Ba2LaPuO6 by Materials Project

description: Ba2PuLaO6 is (Cubic) Perovskite-derived structured and crystallizes in the cubic Fm-3m space group. The structure is three-dimensional. Ba2+ is bonded to twelve equivalent O2- atoms to form BaO12 cuboctahedra that share corners with twelve equivalent BaO12 cuboctahedra, faces with six equivalent BaO12 cuboctahedra, faces with four equivalent PuO6 octahedra, and faces with four equivalent LaO6 octahedra. All Ba–O bond lengths are 3.18 Å. Pu5+ is bonded to six equivalent O2- atoms to form PuO6 octahedra that share corners with six equivalent LaO6 octahedra and faces with eight equivalent BaO12 cuboctahedra. The corner-sharing octahedral tilt angles are 0°. All Pu–O bond lengths are 2.13 Å. La3+ is bonded to six equivalent O2- atoms to form LaO6 octahedra that share corners with six equivalent PuO6 octahedra and faces with eight equivalent BaO12 cuboctahedra. The corner-sharing octahedral tilt angles are 0°. All La–O bond lengths are 2.36 Å. O2- is bonded in a distorted linear geometry to four equivalent Ba2+, one Pu5+, and one La3+ atom.
DOI: 10.17188/1698687
title: Materials Data on BaH6C2SNO5 by Materials Project

description: BaCH6O5CNS crystallizes in the triclinic P-1 space group. The structure is two-dimensional and consists of two thiocyanic acid molecules and one BaCH6O5 sheet oriented in the (0, 0, 1) direction. In the BaCH6O5 sheet, Ba2+ is bonded in a 10-coordinate geometry to ten O2- atoms. There are a spread of Ba–O bond distances ranging from 2.75–3.09 Å. C+3.50+ is bonded in a bent 120 degrees geometry to two O2- atoms. Both C–O bond lengths are 1.27 Å. There are six inequivalent H1+ sites. In the first H1+ site, H1+ is bonded in a single-bond geometry to one O2- atom. The H–O bond length is 0.99 Å. In the second H1+ site, H1+ is bonded in a single-bond geometry to one O2- atom. The H–O bond length is 0.98 Å. In the third H1+ site, H1+ is bonded in a single-bond geometry to one O2- atom. The H–O bond length is 0.98 Å. In the fourth H1+ site, H1+ is bonded in a single-bond geometry to one O2- atom. The H–O bond length is 0.98 Å. In the fifth H1+ site, H1+ is bonded in a single-bond geometry to one O2- atom. The H–O bond length is 0.99 Å. In the sixth H1+ site, H1+ is bonded in a single-bond geometry to one O2- atom. The H–O bond length is 0.98 Å. There are five inequivalent O2- sites. In the first O2- site, O2- is bonded in a distorted single-bond geometry to two equivalent Ba2+ and one C+3.50+ atom. In the second O2- site, O2- is bonded in a distorted single-bond geometry to two equivalent Ba2+ and one C+3.50+ atom. In the third O2- site, O2- is bonded in a water-like geometry to two equivalent Ba2+ and two H1+ atoms. In the fourth O2- site, O2- is bonded in a water-like geometry to two equivalent Ba2+ and two H1+ atoms. In the fifth O2- site, O2- is bonded in a water-like geometry to two equivalent Ba2+ and two H1+ atoms.
DOI: 10.15468/dl.tpn5fm
title: Occurrence Download

description: A dataset containing 1475 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Hippocrepis emerus emeroides"
 ]
}

 The dataset includes 1475 records from 92 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0014824-250827131500795/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.vanegn
title: Occurrence Download

description: A dataset containing 1073 species occurrences available in GBIF matching the query:
{
 "and" : [
 "Country is China",
 "TaxonKey is Grus monacha Temminck, 1835"
 ]
}

 The dataset includes 1073 records from 13 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0107170-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.xkz454
title: Occurrence Download

description: A dataset containing 58638 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Paspalum dilatatum Poir."
 ]
}

 The dataset includes 58638 records from 997 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0107171-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.34740/kaggle/dsv/9270905
title: ICC Men's T-20 World Cup 2024
DOI: 10.15468/dl.p4wmfb
title: Occurrence Download

description: A dataset containing 4256 species occurrences available in GBIF matching the query:
{
 "and" : [
 "TaxonKey is Bonasa bonasia (Linnaeus, 1758)",
 "HasCoordinate is true",
 "Year is greater than or equal to 2011",
 "Year is less than or equal to 2024"
 ]
}

 The dataset includes 4256 records from 63 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0107173-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.vbmvke
title: Occurrence Download

description: A dataset containing 14284 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Paspalum conjugatum P.J.Bergius"
 ]
}

 The dataset includes 14284 records from 489 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0107174-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.b25f7n
title: Occurrence Download

description: A dataset containing 1756231 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Accipiter striatus Vieillot, 1808"
 ]
}

 The dataset includes 1756231 records from 281 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0107172-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.6084/m9.figshare.23908879
title: Additional file 5 of Systematic analysis of histone acetylation regulators across human cancers

description: Supplementary Material 5
DOI: 10.6084/m9.figshare.23908879.v1
title: Additional file 5 of Systematic analysis of histone acetylation regulators across human cancers

description: Supplementary Material 5
DOI: 10.6084/m9.figshare.23908882
title: Additional file 6 of Systematic analysis of histone acetylation regulators across human cancers

description: Supplementary Material 6
DOI: 10.6084/m9.figshare.23908882.v1
title: Additional file 6 of Systematic analysis of histone acetylation regulators across human cancers

description: Supplementary Material 6
DOI: 10.6084/m9.figshare.23908886
title: Additional file 1 of Identifying cellular senescence associated genes involved in the progression of end-stage renal disease as new biomarkers

description: Additional file 1: Table S1. Levels of eight immune cells and two stromal cells in the samples.
DOI: 10.6084/m9.figshare.23908886.v1
title: Additional file 1 of Identifying cellular senescence associated genes involved in the progression of end-stage renal disease as new biomarkers

description: Additional file 1: Table S1. Levels of eight immune cells and two stromal cells in the samples.
DOI: 10.6084/m9.figshare.7131743
title: electroacupuncture upregulates α7nAChR and suppresses JAK2STAT3 signaling pathway in dorsal root ganglion(DRG)20180820.xlsx

description: Methods: The SNI-induced neuropathic pain rat
model was used in this study. After application of 2Hz EA treatment to SNI rats
on day 3, 7, 14, and 21 post-surgery, the α7nAChR, JAK2/STAT3, pro-inflammatory
cytokines IL-1β, IL-6 and anti-inflammatory cytokine IL-10 expression in DRG
were determined by qRT-PCR and western blot analysis.

The related study consists of two sections: 

The 1st section (see tab fig1) was designed
to examine the effect of 2Hz EA on the expression level of α7nAChR in DRG of SNI rat. 48 animals in this
part were randomly divided into 3 groups: sham，SNI，SNI+EA 3 day (D), sham，SNI，SNI+EA
7D, sham，SNI，SNI+EA
14D, sham，SNI，SNI+EA
21D，with 4 rats in each group.

The 2st section (see tab fig2) was designed
to examine western blot. Animals in this part were randomly divided into 3
groups: sham，SNI，SNI+EA
3 day (D), sham，SNI，SNI+EA
7D, sham，SNI，SNI+EA
14D, sham，SNI，SNI+EA
21D，with 4 rats in each group.

<b></b><i></i><sub></sub><sup></sup><br>
DOI: 10.6084/m9.figshare.10277660
title: Testing for Trend in Benefit-Risk Analysis with Prioritized Multiple Outcomes

description: Benefit-risk analysis using prioritized multiple outcomes has been proposed for use in randomized controlled trials with two treatment groups. This research extends the two-group comparison to testing for a trend over multiple treatment groups, for example, multiple doses, in terms of a composite outcome that is derived from pairwise comparisons of subjects between groups or by ranking subjects of pooled groups, according to their prioritized outcomes. Tukey’s trend test, Wilcoxon rank-sum test and Jonkheere-Terpstra (JT) test and their permutation-based trend tests are investigated for detection of an increasing trend over doses with respect to the composite benefit-risk endpoint. Simulation studies show that the permutation-based Tukey’s and JT tests outperform the others in terms of Type I error control and power under various simulation settings. For illustrative purpose, the six approaches are applied to a migraine example data to determine whether an increasing trend exists among four dose groups in terms of a composite benefit-risk endpoint that are measured by four prioritized outcomes.
DOI: 10.6084/m9.figshare.10277660.v2
title: Testing for Trend in Benefit-Risk Analysis with Prioritized Multiple Outcomes

description: Benefit-risk analysis using prioritized multiple outcomes has been proposed for use in randomized controlled trials with two treatment groups. This research extends the two-group comparison to testing for a trend over multiple treatment groups, for example, multiple doses, in terms of a composite outcome that is derived from pairwise comparisons of subjects between groups or by ranking subjects of pooled groups, according to their prioritized outcomes. Tukey’s trend test, Wilcoxon rank-sum test and Jonkheere-Terpstra (JT) test and their permutation-based trend tests are investigated for detection of an increasing trend over doses with respect to the composite benefit-risk endpoint. Simulation studies show that the permutation-based Tukey’s and JT tests outperform the others in terms of Type I error control and power under various simulation settings. For illustrative purpose, the six approaches are applied to a migraine example data to determine whether an increasing trend exists among four dose groups in terms of a composite benefit-risk endpoint that are measured by four prioritized outcomes.
DOI: 10.6084/m9.figshare.14994396.v1
title: MicroRNA expression profile and identification of novel microRNA biomarkers for metabolic syndrome

description: The lack of efficient biomarkers is the main reason for the inaccurate early diagnosis and poor treatment outcomes of patients with metabolic syndrome (MetS). The current study aimed to identify several novel microRNA (miRNA) biomarkers for metabolic syndrome via high-throughput sequencing and comprehensive bioinformatics analysis. Through high-throughput sequencing and differentially expressed miRNA (DEM) analysis, we first identified two upregulated and 36 downregulated DEMs in the plasma samples of patients with MetS compared to the healthy volunteers. Additionally, we also predicted 379 potential target genes and subsequently carried out enrichment analysis and protein–protein interaction network analysis to investigate the signaling pathways and functions of the identified DEMs as well as the interactions between their target genes. Furthermore, we selected two upregulated and top 10 downregulated DEMs with the highest |log2FC| values as the key microRNAs, which may serve as potential biomarkers for MetS. RT-qPCR was performed to validated these result. Finally, hsa-miR-526b-5p, hsa-miR-6516-5p was identified as the novel biomarkers for MetS.
DOI: 10.6084/m9.figshare.8180648
title: MiR-150-5p retards the progression of myocardial fibrosis by targeting <i>EGR1</i>

description: To investigate the differential expression of microRNA-150-5p (miR-150-5p) and early growth response 1 (<i>EGR1)</i> in myocardial fibrosis (MF) cells, and determine the effect between miR-150-5p and <i>EGR1</i> on MF. Human MF cells were generated via <i>Trypanosoma cruzi</i> (<i>T. cruzi)</i> infection, a mouse model of MF was generated via angiotensin II. The expression levels of miR-150-5p and <i>EGR1</i> were detected by quantitative real-time polymerase chain reaction (qRT-PCR) and western blot assay. The correlation between miR-150-5p and <i>EGR1</i> was confirmed by a luciferase reporter assay. The viability, proliferation, and apoptotic rate were detected by cell counting kit-8 (CCK-8), colony-formation and flow cytometry assays. Hematoxylin-eosin (HE) staining and Masson staining visualized the degree of MF. Echocardiography was performed to obtain the levels of left ventricle fractional shortening (LVFS) and left ventricle ejection fraction (LVEF), computer algorithms and a videographics program were used to obtain the levels of left ventricular systolic pressure (LVSP), left ventricular end diastolic pressure (LVEDP) and ±left ventricular dp/dt maximum (LV dp/dtmax). We found that the expression of miR-150-5p in MF cells was lower than normal cardiomyocytes, while the expression level of EGR1 in MF cells were higher than normal cardiomyocytes. Cell experiments demonstrated that <i>EGR1</i> and miR-150-5p could influence the development of MF, and the expression of <i>EGR1</i> in cardiomyocytes was regulated by miR-150-5p directly. Lastly, we confirmed that sh-<i>Egr1</i> would decrease the severity of MF, while miR-150-5p antagomir could aggravate MF. Our results illustrate the mechanism of MF development, and provide a potential target for MF treatment.
DOI: 10.6084/m9.figshare.8180648.v1
title: MiR-150-5p retards the progression of myocardial fibrosis by targeting <i>EGR1</i>

description: To investigate the differential expression of microRNA-150-5p (miR-150-5p) and early growth response 1 (<i>EGR1)</i> in myocardial fibrosis (MF) cells, and determine the effect between miR-150-5p and <i>EGR1</i> on MF. Human MF cells were generated via <i>Trypanosoma cruzi</i> (<i>T. cruzi)</i> infection, a mouse model of MF was generated via angiotensin II. The expression levels of miR-150-5p and <i>EGR1</i> were detected by quantitative real-time polymerase chain reaction (qRT-PCR) and western blot assay. The correlation between miR-150-5p and <i>EGR1</i> was confirmed by a luciferase reporter assay. The viability, proliferation, and apoptotic rate were detected by cell counting kit-8 (CCK-8), colony-formation and flow cytometry assays. Hematoxylin-eosin (HE) staining and Masson staining visualized the degree of MF. Echocardiography was performed to obtain the levels of left ventricle fractional shortening (LVFS) and left ventricle ejection fraction (LVEF), computer algorithms and a videographics program were used to obtain the levels of left ventricular systolic pressure (LVSP), left ventricular end diastolic pressure (LVEDP) and ±left ventricular dp/dt maximum (LV dp/dtmax). We found that the expression of miR-150-5p in MF cells was lower than normal cardiomyocytes, while the expression level of EGR1 in MF cells were higher than normal cardiomyocytes. Cell experiments demonstrated that <i>EGR1</i> and miR-150-5p could influence the development of MF, and the expression of <i>EGR1</i> in cardiomyocytes was regulated by miR-150-5p directly. Lastly, we confirmed that sh-<i>Egr1</i> would decrease the severity of MF, while miR-150-5p antagomir could aggravate MF. Our results illustrate the mechanism of MF development, and provide a potential target for MF treatment.
DOI: 10.6084/m9.figshare.8180684
title: RETRACTED ARTICLE: Long noncoding RNA MAGI1-IT1 promoted invasion and metastasis of epithelial ovarian cancer via the miR-200a/ZEB axis

description: We, the Editors and Publisher of the journal <i>Cell Cycle</i>, have retracted the following article: Hao Gao, Xiaofeng Li, Guangxi Zhan, Yong Zhu, Jing Yu, Jiapo Wang, Li Li, Weimin Wu, Na Liu and Xiaoqing Guo. Long noncoding RNA MAGI1-IT1 promoted invasion and metastasis of epithelial ovarian cancer via the miR-200a/ZEB axis. Cell Cycle. 2019;18(12):1393-1406. doi: 10.1080/15384101.2019.1618121 Since publication, concerns have been raised about the integrity of the data in the article. When approached for an explanation, the authors have been unable to fully address the concerns raised and have not been able to provide their original data in the appropriate format. As verifying the validity of published work is core to the integrity of the scholarly record, we are therefore retracting the article. The corresponding author listed in this publication has been informed. The authors do not agree with the retraction. We have been informed in our decision-making by our policy on publishing ethics and integrity and the COPE guidelines on retractions. The retracted article will remain online to maintain the scholarly record, but it will be digitally watermarked on each page as ‘Retracted’.
DOI: 10.6084/m9.figshare.21285735
title: Additional file 2 of Müller cell degeneration and microglial dysfunction in the Alzheimer’s retina

description: Additional file 2. All p-values.
DOI: 10.6084/m9.figshare.8180684.v1
title: Long noncoding RNA MAGI1-IT1 Promoted Invasion and Metastasis of Epithelial Ovarian Cancer via the miR-200a/ZEB axis

description: Epithelial ovarian cancer (EOC) is the most lethal gynecologic malignancy, and its vulnerability to metastasis contributes to the poor outcomes of EOC patients. Long noncoding RNAs (lncRNAs) were verified to play a pivotal role in EOC metastasis. However, the potential role of lncRNA membrane-associated guanylate kinase inverted 1 (MAGI1) intronic transcript (MAGI1-IT1) in EOC is largely unknown. In this study, the function and mechanisms of MAGI1-IT1 in EOC metastasis were explored profoundly. First, MAGI1-IT1 expression was found to be significantly decreased in overexpressing miR-200a EOC cells. Second, MAGI1-IT1 expression was remarkably increased in metastatic EOC tissues, and high MAGI1-IT1 was dramatically associated with EOC FIGO III-IV stage; in addition, MAGI1-IT1 might be related to EOC dissemination via epithelial-mesenchymal transition (EMT). Next, a series of gain- and loss-of-function assays verified that, although MAGI1-IT1 has no significant role in EOC proliferation and subcutaneous xenograft growth, the upregulation of MAGI1-IT1 can remarkably facilitate EOC EMT phenotype, cells migration and invasion ability and intraperitoneal metastasis in nude mice, while downregulation of MAGI1-IT1 led to the opposite effect <i>in vitro</i>. Moreover, MAGI1-IT1 was validated to promote EOC metastasis through upregulation of ZEB1 and ZEB2 by competitively binding miR-200a, and the restrictive effects of MAGI1-IT1 depletion on EOC metastasis could be reversed by inhibition of miR-200a and upregulation of ZEB1 and ZEB2. Collectively, these results suggest that MAGI1-IT1 may work as a ceRNA in promoting EOC metastasis through miR-200a and ZEB1/2 and may be a potential therapeutic target for EOC.