cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.17632/vnkbnw8h74.1
title: Research on the Contagion Effect of Bank Systemic Risk Embedded with the Fintech Public Opinion Index

description: 1. Sample Selection: Sixteen systemically important banks listed on China's A-share market were selected as research subjects, covering the period from January 2011 to December 2024. Unlisted and late-listed institutions were excluded. These include state-owned banks, joint-stock banks, and city commercial banks.
2. Core Data Sources:
(1) Fintech public opinion data: Obtained from Infobank’s "China Economic News Database", news texts containing "bank name + fintech keywords + sentiment words" were crawled through Python web scraping.
(2) Bank stock return data: Retrieved from the Wind Database, with weekly logarithmic returns calculated using average closing prices.
(3) Macro - micro variables: Seven macroeconomic and four bank - specific variables were sourced from the Wind Database; quarterly characteristic variables were converted to weekly frequency via cubic spline interpolation.
(4) Fintech keyword lexicon: Independently constructed with 359 terms (three dimensions).
(5) Financial sentiment dictionary: Integrated Jiang et al. (2019) and other Chinese financial sentiment dictionaries, including 11,487 positive and 18,014 negative words.
3. Data Processing Methods:
(1) Quantification of public opinion index: The frequency of positive and negative sentiment words in news texts was counted. The daily public opinion index was calculated through indexing formulas, and weekly average smoothing was applied to alleviate data gaps and random fluctuations.
(2) Rolling window analysis: A 52 - week rolling window was adopted, combined with CoVaR at the 1% quantile and the TENET model to construct a dynamic risk spillover network.
(3) Threshold filtering: Edge connections below the median in network analysis were removed to avoid information redundancy, and the Fruchterman - Reingold algorithm was used to present node clustering characteristics.
DOI: 10.17632/mtvms3fpdk
title: Timelapse data of microtubule behaviours in plant cotyledon cells

description: Microtubule movies showing microtubule dynamics including the discovery of crossover and severing as shown as supplemental data in the following paper:
Wightman, R. and Turner, S.R. (2007), Severing at sites of microtubule crossover contributes to microtubule alignment in cortical arrays. The Plant Journal, 52: 742-751. https://doi.org/10.1111/j.1365-313X.2007.03271.x

All the video files are of YFP-labelled microtubules in Arabidopsis thaliana cotyledons. 

The original movies no longer have native support for playback on modern operating systems. They have been converted to H.264 MPEG-4 and deposited here. There are 6 movies in total:

Movie 1. Dynamics of a net-like array. The video clip is taken from a pavement cell (YFP-MBD). Bar = 2 μm. 

Movie 2. Microtubule (MT) bundles resist lateral movement. An MT leading end (left, red arrow) is forced against an obstructing MT running along the edge of the cell. Initial bending only occurs up to an apparent crossover (right, red arrow). Further bending results in an S–shaped configuration but no bending occurs further back from the crosslink where the MT is part of a bundle (green arrow). Bar = 2 μm. 

Movie 3. Microtubule (MT) collision results in a lower polymerization rate. This movie is taken from a pavement cell. An MT is identified by a red arrow and is seen to pause whilst it negotiates an obstruction. Bar = 2 μm. 

Movie 4. Microtubule (MT) severing during MT alignment into an ordered array. MTs were observed in a petiole cell undergoing alignment. MTs growing in an arc are severed at sites marked by white arrows. The posterior portions of two MTs undergo a catastrophe (green arrows) leaving the posterior portions intact (yellow arrows). The upper anterior MT then undergoes a catastrophe whilst the lower MT appears to migrate by hybrid treadmilling. Bar = 2 μm. The Fire look-up table of ImageJ was used to aid visualization of the MTs. 

Movie 5. Microtubule (MT) severing resulting in a new leading end. A bundled leading end (green arrow) undergoes a severing event further back along the MT (white arrow). The posterior portion of the severed MT then undergoes a catastrophe followed by rescue. Growth at the leading end then occurs along with a change in trajectory. Bar = 5 μm. The Fire look-up table of ImageJ was used to aid visualization of the MTs. 

Movie 6. Nucleation-associated severing. Two clips where, in each case, a microtubule (MT) is seen to emerge from an existing MT (arrowheads). After a period of growth the lagging end retreats from its site of nucleation. Bar = 2 μm.
DOI: 10.17632/mtvms3fpdk.1
title: Timelapse data of microtubule behaviours in plant cotyledon cells

description: Microtubule movies showing microtubule dynamics including the discovery of crossover and severing as shown as supplemental data in the following paper:
Wightman, R. and Turner, S.R. (2007), Severing at sites of microtubule crossover contributes to microtubule alignment in cortical arrays. The Plant Journal, 52: 742-751. https://doi.org/10.1111/j.1365-313X.2007.03271.x

All the video files are of YFP-labelled microtubules in Arabidopsis thaliana cotyledons. 

The original movies no longer have native support for playback on modern operating systems. They have been converted to H.264 MPEG-4 and deposited here. There are 6 movies in total:

Movie 1. Dynamics of a net-like array. The video clip is taken from a pavement cell (YFP-MBD). Bar = 2 μm. 

Movie 2. Microtubule (MT) bundles resist lateral movement. An MT leading end (left, red arrow) is forced against an obstructing MT running along the edge of the cell. Initial bending only occurs up to an apparent crossover (right, red arrow). Further bending results in an S–shaped configuration but no bending occurs further back from the crosslink where the MT is part of a bundle (green arrow). Bar = 2 μm. 

Movie 3. Microtubule (MT) collision results in a lower polymerization rate. This movie is taken from a pavement cell. An MT is identified by a red arrow and is seen to pause whilst it negotiates an obstruction. Bar = 2 μm. 

Movie 4. Microtubule (MT) severing during MT alignment into an ordered array. MTs were observed in a petiole cell undergoing alignment. MTs growing in an arc are severed at sites marked by white arrows. The posterior portions of two MTs undergo a catastrophe (green arrows) leaving the posterior portions intact (yellow arrows). The upper anterior MT then undergoes a catastrophe whilst the lower MT appears to migrate by hybrid treadmilling. Bar = 2 μm. The Fire look-up table of ImageJ was used to aid visualization of the MTs. 

Movie 5. Microtubule (MT) severing resulting in a new leading end. A bundled leading end (green arrow) undergoes a severing event further back along the MT (white arrow). The posterior portion of the severed MT then undergoes a catastrophe followed by rescue. Growth at the leading end then occurs along with a change in trajectory. Bar = 5 μm. The Fire look-up table of ImageJ was used to aid visualization of the MTs. 

Movie 6. Nucleation-associated severing. Two clips where, in each case, a microtubule (MT) is seen to emerge from an existing MT (arrowheads). After a period of growth the lagging end retreats from its site of nucleation. Bar = 2 μm.
DOI: 10.17632/gg2bf2xz9g
title: radiologic_exams_and_labels_dataset

description: This dataset contains filtered data from the PadChest Dataset, which provides information about thousands of radiologic exams conduced in Europe. We added custom fields such as SimpleID and Patient Age and removed unused columns in order to make it easier to organize and utilize data.

The original dataset can be found at: https://www.sciencedirect.com/science/article/abs/pii/S1361841520301614
DOI: 10.17632/gg2bf2xz9g.1
title: radiologic_exams_and_labels_dataset

description: This dataset contains filtered data from the PadChest Dataset, which provides information about thousands of radiologic exams conduced in Europe. We added custom fields such as SimpleID and Patient Age and removed unused columns in order to make it easier to organize and utilize data.

The original dataset can be found at: https://www.sciencedirect.com/science/article/abs/pii/S1361841520301614
DOI: 10.15468/dl.9w2crs
title: Occurrence Download

description: A dataset containing 3 species occurrences available in GBIF matching the query:
{
 "and" : [
 "TaxonKey is Brada ochotensis",
 "HasCoordinate is true",
 "HasGeospatialIssue is false"
 ]
}

 The dataset includes 3 records from 3 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0046083-251120083545085/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.17632/mvycmbybts
title: Quercetin modulates appetite regulating hormones, redox parameters and inflammation in aged male Wistar rats

description: Quercetin (3,5,7,3′,4′-pentahydroxy flavone) is a flavanol that is comprehensively apportioned 
in fruits and vegetables. Quercetin is widely reported to ease inflammation and improve redox balance, with many experimental studies showing lowered cytokine levels and better oxidative status. Additionally, recent research has shown that quercetin is linked to appetite modulation. This research examines the effectiveness of quercetin supplementation in addressing age-related metabolic issues in a rat model of accelerated aging induced by D-galactose treatment. Four groups of Wistar rats (Rattus norvegicus) were taken, Group I (fed on standard diet), Group II (Quercetin treated with oral dose of 100 mg/kg b.w.) Group III (accelerated aging model with D-galactose, 300 mg/kg b.w, subcutaneous), group IV (Quercetin administration + D-gal) for 28 days. We assessed biomarkers of oxidative stress, antioxidant parameters and hormonal balance in erythrocyte, plasma and serum. We observed that aged rats showed poor appetite regulation, marked by increased leptin levels and decreased ghrelin levels, along with signs of insulin resistance, lipid abnormalities, and liver dysfunction. Quercetin supplementation helped to correct these hormonal discrepancies, improving insulin sensitivity, and restored lipid and liver enzyme profiles to healthier ranges. Quercetin supplementation regulated hormonal equilibrium, potentially enhancing antioxidant defences, and relieving inflammation associated with aging, thereby underscoring its potential as both an anti-aging agent and an appetite modulator. This study could improve the understanding of how natural substances like quercetin might play a role in maintaining metabolic health and reduce age-related issues through appetite regulation.
DOI: 10.17632/mvycmbybts.1
title: Quercetin modulates appetite regulating hormones, redox parameters and inflammation in aged male Wistar rats

description: Quercetin (3,5,7,3′,4′-pentahydroxy flavone) is a flavanol that is comprehensively apportioned 
in fruits and vegetables. Quercetin is widely reported to ease inflammation and improve redox balance, with many experimental studies showing lowered cytokine levels and better oxidative status. Additionally, recent research has shown that quercetin is linked to appetite modulation. This research examines the effectiveness of quercetin supplementation in addressing age-related metabolic issues in a rat model of accelerated aging induced by D-galactose treatment. Four groups of Wistar rats (Rattus norvegicus) were taken, Group I (fed on standard diet), Group II (Quercetin treated with oral dose of 100 mg/kg b.w.) Group III (accelerated aging model with D-galactose, 300 mg/kg b.w, subcutaneous), group IV (Quercetin administration + D-gal) for 28 days. We assessed biomarkers of oxidative stress, antioxidant parameters and hormonal balance in erythrocyte, plasma and serum. We observed that aged rats showed poor appetite regulation, marked by increased leptin levels and decreased ghrelin levels, along with signs of insulin resistance, lipid abnormalities, and liver dysfunction. Quercetin supplementation helped to correct these hormonal discrepancies, improving insulin sensitivity, and restored lipid and liver enzyme profiles to healthier ranges. Quercetin supplementation regulated hormonal equilibrium, potentially enhancing antioxidant defences, and relieving inflammation associated with aging, thereby underscoring its potential as both an anti-aging agent and an appetite modulator. This study could improve the understanding of how natural substances like quercetin might play a role in maintaining metabolic health and reduce age-related issues through appetite regulation.
DOI: 10.17632/6wfwfk3n2t
title: Effect of changes in social group composition on the behaviour of female gorillas (Primates: Gorilla gorilla gorilla) under human care

description: Data used in the evaluation of behavioural changes in a group of captive gorillas due to chamges in group composition.
DOI: 10.17632/6wfwfk3n2t.2
title: Effect of changes in social group composition on the behaviour of female gorillas (Primates: Gorilla gorilla gorilla) under human care

description: Data used in the evaluation of behavioural changes in a group of captive gorillas due to chamges in group composition.
DOI: 10.17632/fxx76637vt
title: Calycosin inhibits doxorubicin-induced mitochondrial oxidative stress and ferroptosis by activating the NRF2 pathway

description: fig.1 Calycosin significantly ameliorated cardiac dysfunction in DIC mice. (A) Echocardiographic assessment (n = 6). (B) Quantitative analysis of cardiac function parameters (n = 6). (C) Serum biomarkers of myocardial injury (n = 4). (D) Representative histopathological images (H&E staining; scale bar=100 μm). fig.2 Calycosin alleviated DOX-mediated mitochondrial dysfunction in vivo. (A) TEM analysis showing calycosin treatment preserved mitochondrial architecture. (B-C) Cardiac oxidative stress parameters (lipid peroxidation marker MDA and antioxidant enzyme SOD; n = 3). (D) Representative immunofluorescence images of DNA damage evaluation via 8-OHdG (scale bar=20 μm). fig.3 Calycosin stimulated the NRF2/NRF1-TFAM signaling pathway in mice model. (A) The expression of NRF2 visualized by immunofluorescence microscopy (scale bar=20 μm). (B-C) Cardiac expression levels of NRF2, NRF1, TFAM, NDUFB8, COX1 and GPX4 (n = 6). fig.4 Calycosin rescued mitochondrial respiration chain in NRF2-dependent way. (A) Dose-response of calycosin on H9c2 viability (n = 6). (B) Rescue of DOX-impaired cell viability by calycosin cotreatment (n = 6). (C) NRF2 nuclear localization (n = 3). (D-E) Western blot analysis of NRF2, NRF1, TFAM, COX1, NDUFB8, SDHB and ATP5A (n = 3). fig.5 Calycosin alleviated ferroptosis by triggering the NRF2 signaling pathway in H9c2 cells. (A-B) Intracellular Fe²⁺ levels visualized by FerroOrange staining in H9c2 cardiomyocytes (scale bar = 20 μm). (C) Western blot analysis of iron regulatory proteins FPN and FTMT . fig.6 Calycosin protected mitochondrial respiration chain in NRF1-dependent way. (A-B) The expression of NRF2, NRF1 after interference with NRF2 siRNA, NRF1 siRNA. (C-D) Western blot of NRF1, TFAM, COX1, NDUFB8 after interference with NRF1 siRNA (n = 3 per group). fig.7 Calycosin alleviated mitochondrial DNA damage, oxidative stress and membrane potential in H9c2 cells through activation of the NRF2–NRF1 pathway. (A) Immunofluorescence detection of oxidative DNA damage (8-OHdG). (B) Mitochondria (MitoTracker Green, green) and mitochondrial ROS (MitoSOX Red, red) in live cells (n = 3). (C) Variations in mitochondrial membrane potential following treatment (Scale bar = 50 μm). (D) Quantification of the ratio of mitochondria to mitochondrial ROS in live cells. (E) Quantitative analysis of the ratio of JC-1 monomer to JC-1 aggreates. fig.8 Calycosin protected against DIC by activation of NRF2. (A) Representative M-mode echocardiographic images (n = 4). (B) Quantitative analysis of cardiac function parameters (n = 4). (C) Serum biomarkers of myocardial injury (CK-MB and LDH, n = 4). (D) MDA and SOD levels in cardiac tissues of mice (n = 3). (E) Hematoxylin and eosin staining . fig.9 Calycosin regulated mitochondrial proteins and ferroptosis-associated proteins by activating NRF2. (A-B) Western blot analysis of NRF2, NRF1, TFAM, COX1, and NDUFB8 expression levels. (C-D) FPN and FTMT expression levels (n = 3).
DOI: 10.17632/fxx76637vt.1
title: Calycosin inhibits doxorubicin-induced mitochondrial oxidative stress and ferroptosis by activating the NRF2 pathway

description: fig.1 Calycosin significantly ameliorated cardiac dysfunction in DIC mice. (A) Echocardiographic assessment (n = 6). (B) Quantitative analysis of cardiac function parameters (n = 6). (C) Serum biomarkers of myocardial injury (n = 4). (D) Representative histopathological images (H&E staining; scale bar=100 μm). fig.2 Calycosin alleviated DOX-mediated mitochondrial dysfunction in vivo. (A) TEM analysis showing calycosin treatment preserved mitochondrial architecture. (B-C) Cardiac oxidative stress parameters (lipid peroxidation marker MDA and antioxidant enzyme SOD; n = 3). (D) Representative immunofluorescence images of DNA damage evaluation via 8-OHdG (scale bar=20 μm). fig.3 Calycosin stimulated the NRF2/NRF1-TFAM signaling pathway in mice model. (A) The expression of NRF2 visualized by immunofluorescence microscopy (scale bar=20 μm). (B-C) Cardiac expression levels of NRF2, NRF1, TFAM, NDUFB8, COX1 and GPX4 (n = 6). fig.4 Calycosin rescued mitochondrial respiration chain in NRF2-dependent way. (A) Dose-response of calycosin on H9c2 viability (n = 6). (B) Rescue of DOX-impaired cell viability by calycosin cotreatment (n = 6). (C) NRF2 nuclear localization (n = 3). (D-E) Western blot analysis of NRF2, NRF1, TFAM, COX1, NDUFB8, SDHB and ATP5A (n = 3). fig.5 Calycosin alleviated ferroptosis by triggering the NRF2 signaling pathway in H9c2 cells. (A-B) Intracellular Fe²⁺ levels visualized by FerroOrange staining in H9c2 cardiomyocytes (scale bar = 20 μm). (C) Western blot analysis of iron regulatory proteins FPN and FTMT . fig.6 Calycosin protected mitochondrial respiration chain in NRF1-dependent way. (A-B) The expression of NRF2, NRF1 after interference with NRF2 siRNA, NRF1 siRNA. (C-D) Western blot of NRF1, TFAM, COX1, NDUFB8 after interference with NRF1 siRNA (n = 3 per group). fig.7 Calycosin alleviated mitochondrial DNA damage, oxidative stress and membrane potential in H9c2 cells through activation of the NRF2–NRF1 pathway. (A) Immunofluorescence detection of oxidative DNA damage (8-OHdG). (B) Mitochondria (MitoTracker Green, green) and mitochondrial ROS (MitoSOX Red, red) in live cells (n = 3). (C) Variations in mitochondrial membrane potential following treatment (Scale bar = 50 μm). (D) Quantification of the ratio of mitochondria to mitochondrial ROS in live cells. (E) Quantitative analysis of the ratio of JC-1 monomer to JC-1 aggreates. fig.8 Calycosin protected against DIC by activation of NRF2. (A) Representative M-mode echocardiographic images (n = 4). (B) Quantitative analysis of cardiac function parameters (n = 4). (C) Serum biomarkers of myocardial injury (CK-MB and LDH, n = 4). (D) MDA and SOD levels in cardiac tissues of mice (n = 3). (E) Hematoxylin and eosin staining . fig.9 Calycosin regulated mitochondrial proteins and ferroptosis-associated proteins by activating NRF2. (A-B) Western blot analysis of NRF2, NRF1, TFAM, COX1, and NDUFB8 expression levels. (C-D) FPN and FTMT expression levels (n = 3).
DOI: 10.17632/fdpwmgg2kh.1
title: Paleoenvironmental Evolution Characteristics and Mineral Enrichment Patterns of the Lacustrine System Under Orbital Forcing in the Yin'e Basin During the Early Cretaceous

description: he samples cover the entire first member of the Bayingebi Formation (total thickness: 189 m). Specific grouping: 26 samples were prepared as thin sections for microscopic and scanning electron microscope (SEM) observations; 71 samples were analyzed for mineral composition using X-ray diffraction (XRD); 51 mudstone and marlstone samples were selected for determinations of total organic carbon (TOC), organic carbon isotope (δ¹³C), and nitrogen isotope (δ¹⁵N); while 20 high-organic-content mudstone samples were used for biomarker analysis via gas chromatography-mass spectrometry (GC-MS); and 369 samples were subjected to chromaticity and magnetic susceptibility (MS) measurements.
DOI: 10.17632/yfxmp2jvf5
title: Crossing Borders, Facing Bullying: Immigrant Generational Status and the Moderating Role of Gender in Bullying Victimization across 64 Countries

description: This dataset includes the subset of variables from the PISA 2018 microdata required to reproduce the analyses presented in the article “Crossing Borders, Facing Bullying: Immigrant Generational Status and the Moderating Role of Gender in Bullying Victimization across 64 Countries.” The dataset contains student- and school-level variables used to estimate the regression models described in the paper.
The full PISA 2018 database is publicly available through the OECD website: https://www.oecd.org/pisa/data/2018database/. Due to file size only the relevant variables needed to execute the Stata regression scripts have been included here.
DOI: 10.17632/yfxmp2jvf5.1
title: Crossing Borders, Facing Bullying: Immigrant Generational Status and the Moderating Role of Gender in Bullying Victimization across 64 Countries

description: This dataset includes the subset of variables from the PISA 2018 microdata required to reproduce the analyses presented in the article “Crossing Borders, Facing Bullying: Immigrant Generational Status and the Moderating Role of Gender in Bullying Victimization across 64 Countries.” The dataset contains student- and school-level variables used to estimate the regression models described in the paper.
The full PISA 2018 database is publicly available through the OECD website: https://www.oecd.org/pisa/data/2018database/. Due to file size only the relevant variables needed to execute the Stata regression scripts have been included here.
DOI: 10.17632/kz5czn9vjy
title: Research Data

description: Research Data
DOI: 10.17632/kz5czn9vjy.1
title: Research Data

description: Research Data
DOI: 10.17632/wkpz2vkj4f
title: TRIGGER POINT DRY NEEDLING FOR PAIN, FUNCTION, AND MOUTH OPENING IN ORAL SUBMUCOUS FIBROSIS: A PILOT FEASIBILITY STUDY

description: Oral submucous fibrosis (OSF) is a chronic, progressive oral potentially malignant disorder characterized by inflammation and fibrosis of the oral mucosa, causing trismus, pain, and functional limitation. Conventional treatments offer variable relief, prompting interest in minimally invasive adjuncts. Trigger point dry needling (TrP-DN) uses filiform needles to target myofascial trigger points, reducing pain, normalizing muscle tone, and improving function. Purpose is to evaluate feasibility, safety, and preliminary effects of TrP-DN on pain intensity, maximal mouth opening, and jaw functional limitation in Stage I or II OSF patients.
DOI: 10.17632/wkpz2vkj4f.1
title: TRIGGER POINT DRY NEEDLING FOR PAIN, FUNCTION, AND MOUTH OPENING IN ORAL SUBMUCOUS FIBROSIS: A PILOT FEASIBILITY STUDY

description: Oral submucous fibrosis (OSF) is a chronic, progressive oral potentially malignant disorder characterized by inflammation and fibrosis of the oral mucosa, causing trismus, pain, and functional limitation. Conventional treatments offer variable relief, prompting interest in minimally invasive adjuncts. Trigger point dry needling (TrP-DN) uses filiform needles to target myofascial trigger points, reducing pain, normalizing muscle tone, and improving function. Purpose is to evaluate feasibility, safety, and preliminary effects of TrP-DN on pain intensity, maximal mouth opening, and jaw functional limitation in Stage I or II OSF patients.
DOI: 10.17632/xgpk9y4kgw.1
title: Redescription and Embryonic Developmental Dynamics of Aphelenchoides bicaudatus with Implications for Nematode Taxonomy and Ecology

description: High-resolution microscopy image