cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.5281/zenodo.3712750
title: Decision problem for renewable energy planning in Turkey (Dataset). June 2019

description: This dataset compromises the decision problem for renewable energy planning in Turkey, adopted from the literature (Kahraman and Kaya 2010; Erdogan and Kaya 2015; Mousavi et al. 2017). The decision problem consists in evaluating and selecting the most appropriate renewable energy alternative for Turkey, and includes: Five alternatives (<strong>Hydro, Wind, Solar, Biomass, and Geothermal</strong>). Five dimensions fo criteria (<strong>Technological, Technical, Economical, Environmental, and Socio-politic</strong>), with 31 sub-criteria. Weights of criteria. Direction of criteria: "max" if the sub-criterion needs to be maximized ("bigger is better”) and "min" if it needs to be minimized (”smaller is better”). The dataset is used in the following paper: <em>Ezbakhe, F. and Pérez-Foguet, A. (2019) Decision analysis for sustainable development: the case of renewable energy planning under uncertainty. European Journal of Operational Research. </em>https://doi.org/10.1016/j.ejor.2020.02.037
DOI: 10.5281/zenodo.3712751
title: Decision problem for renewable energy planning in Turkey (Dataset). June 2019

description: This dataset compromises the decision problem for renewable energy planning in Turkey, adopted from the literature (Kahraman and Kaya 2010; Erdogan and Kaya 2015; Mousavi et al. 2017). The decision problem consists in evaluating and selecting the most appropriate renewable energy alternative for Turkey, and includes: Five alternatives (<strong>Hydro, Wind, Solar, Biomass, and Geothermal</strong>). Five dimensions fo criteria (<strong>Technological, Technical, Economical, Environmental, and Socio-politic</strong>), with 31 sub-criteria. Weights of criteria. Direction of criteria: "max" if the sub-criterion needs to be maximized ("bigger is better”) and "min" if it needs to be minimized (”smaller is better”). The dataset is used in the following paper: <em>Ezbakhe, F. and Pérez-Foguet, A. (2019) Decision analysis for sustainable development: the case of renewable energy planning under uncertainty. European Journal of Operational Research. </em>https://doi.org/10.1016/j.ejor.2020.02.037
DOI: 10.5281/zenodo.3559715
title: Demonstration of Two-Color X-ray Free-Electron Laser Pulses with a Sextupole Magnet

description: In compliance with guidelines from SNSF (Swiss National Science Foundation), we upload the raw data that belongs to a publication we submitted to <em>Physical Review Accelerators and Beams</em>.<br> The dataset also contains the analysis scripts used to produce most of the plots we show in the publication.
DOI: 10.5281/zenodo.3559714
title: Demonstration of Two-Color X-ray Free-Electron Laser Pulses with a Sextupole Magnet

description: In compliance with guidelines from SNSF (Swiss National Science Foundation), we upload the raw data that belongs to a publication we submitted to <em>Physical Review Accelerators and Beams</em>.<br> The dataset also contains the analysis scripts used to produce most of the plots we show in the publication.
DOI: 10.5257/iea/et/2019
title: Energy Technology Research Development (2019 Edition)

description: Energy Technology Research Development includes data on budgets in national currencies (in nominal and real prices), in USD (at latest year prices and exchange rates), in USD (at latest year prices and PPP) and in Euros (at latest year prices and exchange rates). Also, the database shows RD budgets and calculating indicators. The government energy technology RD budgets are submitted on an annual questionnaire every year to the IEA Secretariat by appropriate Administrations in national currencies. The exchange rates are published in Main Economic Indicators (OECD) and the GDP and GDP deflators come from the National Accounts (OECD).
DOI: 10.5281/zenodo.3712753
title: Dataset related to article "The Atypical Receptor CCRL2 Is Essential for Lung Cancer Immune Surveillance."

description: CCRL2 is a nonsignaling seven-transmembrane domain receptor. CCRL2 binds chemerin, a protein that promotes chemotaxis of leukocytes, including macrophages and natural killer (NK) cells. In addition, CCRL2 controls the inflammatory response in different pathologic settings, such as hypersensitivity, inflammatory arthritis, and experimental autoimmune encephalitis. Here, we investigated the role of CCRL2 in the regulation of lung cancer-related inflammation. The genetic deletion of <em>Ccrl2</em> promoted tumor progression in urethane-induced and in <em>Kras</em> <sup>G12D/+</sup>/<em>p53</em> <sup>LoxP</sup> lung tumor mouse models. Similarly, a <em>Kras</em>-mutant lung tumor displayed enhanced growth in <em>Ccrl2</em>-deficient mice. This phenotype was associated with a reduced inflammatory infiltrate characterized by the impaired recruitment of several leukocyte populations including NK cells. Bone marrow chimeras showed that CCRL2 expression by the nonhematopoietic cell compartment was responsible for the increased tumor formation observed in <em>Kras</em>-mutant <em>Ccrl2</em>-deficient mice. In human and mouse lungs, CCRL2 was expressed by a fraction of CD31<sup>+</sup> endothelial cells, where it could control NK infiltration. Elevated CCRL2 expression in biopsies from human lung adenocarcinoma positively correlated with clinical outcome. These results provide evidence for a crucial role of CCRL2 in shaping an anti-lung tumor immune response.
DOI: 10.5281/zenodo.3712752
title: Dataset related to article "The Atypical Receptor CCRL2 Is Essential for Lung Cancer Immune Surveillance."

description: CCRL2 is a nonsignaling seven-transmembrane domain receptor. CCRL2 binds chemerin, a protein that promotes chemotaxis of leukocytes, including macrophages and natural killer (NK) cells. In addition, CCRL2 controls the inflammatory response in different pathologic settings, such as hypersensitivity, inflammatory arthritis, and experimental autoimmune encephalitis. Here, we investigated the role of CCRL2 in the regulation of lung cancer-related inflammation. The genetic deletion of <em>Ccrl2</em> promoted tumor progression in urethane-induced and in <em>Kras</em> <sup>G12D/+</sup>/<em>p53</em> <sup>LoxP</sup> lung tumor mouse models. Similarly, a <em>Kras</em>-mutant lung tumor displayed enhanced growth in <em>Ccrl2</em>-deficient mice. This phenotype was associated with a reduced inflammatory infiltrate characterized by the impaired recruitment of several leukocyte populations including NK cells. Bone marrow chimeras showed that CCRL2 expression by the nonhematopoietic cell compartment was responsible for the increased tumor formation observed in <em>Kras</em>-mutant <em>Ccrl2</em>-deficient mice. In human and mouse lungs, CCRL2 was expressed by a fraction of CD31<sup>+</sup> endothelial cells, where it could control NK infiltration. Elevated CCRL2 expression in biopsies from human lung adenocarcinoma positively correlated with clinical outcome. These results provide evidence for a crucial role of CCRL2 in shaping an anti-lung tumor immune response.
DOI: 10.5285/bca0d930-a10d-4ae8-9afc-ad1bf73de35a
title: Hidden Crisis Project, Survey 1 dataset: detailed functionality assessments of hand pump boreholes in Ethiopia, Uganda and Malawi

description: This dataset provides the field data collected in 2016 to assess: the functionality and performance of 600 handpumped borehole supplies (HPBs) across the three countries, Ethiopia, Malawi and Uganda, and also the capacity of the community water management arrangements. The individual HPBs surveyed in the dataset were selected by a two or three stage randomised sampling approach. The UPGro Hidden Crisis project (2015-20) was an international research project aimed at developing a robust evidence base and understanding of the complex and multi-faceted causes which underlie the current high failure rates of many new groundwater supplies in Africa. The project was jointly funded by: DFID, NERC and ESRC as part of the UPGro programme (Unlocking the Potential of Groundwater for the Poor). The project carried out the research work in three countries - Ethiopia, Malawi and Uganda – to examine functionality and performance of groundwater supplies in a range of hydrogeological, climatic and social, institutional and governance environments in East Africa. The work focused specifically on examining the functionality of HPBs – these being the main form of improved community water supply across rural Africa. The Survey 1 dataset, is major dataset developed by the project.
DOI: 10.6073/pasta/2328111146107185f1edd8fc76c8f0f3
title: North Temperate Lakes LTER: High Frequency Water Temperature Data - Sparkling Lake Raft 1989 - current

description: The instrumented raft on Sparkling Lake is equipped with a thermistor chain that measures water temperature from depths ranging from the surface to 18m at intervals from 0.5 to 3m throughout the water column. The surface temperature sensors are attached to floats so that they are as close to the surface as feasible. The raft on Sparkling Lake is also equipped with a dissolved oxygen sensor and meteorological sensors that provide fundamental information on lake thermal structure, weather conditions, evaporation rates, and lake metabolism. Estimating the flux of solutes to and from lakes requires accurate water budgets. Evaporation rates are a critical component of the water budget of lakes. Data from the instrumented raft on Sparkling Lake includes micrometeorological parameters from which evaporation can be calculated. Raft measurements of relative humidity and air temperature (2 m height), wind velocity (1, 2, and 3 m heights), and water temperatures are combined with measurements of total long-wave and short-wave radiation data from a nearby shore station to determine evaporation by the energy budget technique. Comparable evaporation estimates from mass transfer techniques are calibrated against energy budget estimates to produce a lake-specific mass transfer coefficient for use in estimating evaporation rates Sampling Frequency: one minute; averaged to hourly and daily values as well as higher resolution values such as 2 min and 10 min. Number of sites: 1
DOI: 10.5519/qd.mal1j3fw
title: Query on the Natural History Museum Data Portal (data.nhm.ac.uk) (788 records)
DOI: 10.5281/zenodo.3712775
title: Dataset related to article "Re-education of Tumor-Associated Macrophages by CXCR2 Blockade Drives Senescence and Tumor Inhibition in Advanced Prostate Cancer."

description: Tumor-associated macrophages (TAMs) represent a major component of the tumor microenvironment supporting tumorigenesis. TAMs re-education has been proposed as a strategy to promote tumor inhibition. However, whether this approach may work in prostate cancer is unknown. Here we find that Pten-null prostate tumors are strongly infiltrated by TAMs expressing C-X-C chemokine receptor type 2 (CXCR2), and activation of this receptor through CXCL2 polarizes macrophages toward an anti-inflammatory phenotype. Notably, pharmacological blockade of CXCR2 receptor by a selective antagonist promoted the re-education of TAMs toward a pro-inflammatory phenotype. Strikingly, CXCR2 knockout monocytes infused in Pten<sup>pc-/-</sup>; Trp53<sup>pc-/-</sup> mice differentiated in tumor necrosis factor alpha (TNF-α)-releasing pro-inflammatory macrophages, leading to senescence and tumor inhibition. Mechanistically, PTEN-deficient tumor cells are vulnerable to TNF-α-induced senescence, because of an increase of TNFR1. Our results identify TAMs as targets in prostate cancer and describe a therapeutic strategy based on CXCR2 blockade to harness anti-tumorigenic potential of macrophages against this disease. This dataset contain some prism documents, in order to help we attach a pdf information file about it
DOI: 10.5281/zenodo.3712776
title: Dataset related to article "Re-education of Tumor-Associated Macrophages by CXCR2 Blockade Drives Senescence and Tumor Inhibition in Advanced Prostate Cancer."

description: Tumor-associated macrophages (TAMs) represent a major component of the tumor microenvironment supporting tumorigenesis. TAMs re-education has been proposed as a strategy to promote tumor inhibition. However, whether this approach may work in prostate cancer is unknown. Here we find that Pten-null prostate tumors are strongly infiltrated by TAMs expressing C-X-C chemokine receptor type 2 (CXCR2), and activation of this receptor through CXCL2 polarizes macrophages toward an anti-inflammatory phenotype. Notably, pharmacological blockade of CXCR2 receptor by a selective antagonist promoted the re-education of TAMs toward a pro-inflammatory phenotype. Strikingly, CXCR2 knockout monocytes infused in Pten<sup>pc-/-</sup>; Trp53<sup>pc-/-</sup> mice differentiated in tumor necrosis factor alpha (TNF-α)-releasing pro-inflammatory macrophages, leading to senescence and tumor inhibition. Mechanistically, PTEN-deficient tumor cells are vulnerable to TNF-α-induced senescence, because of an increase of TNFR1. Our results identify TAMs as targets in prostate cancer and describe a therapeutic strategy based on CXCR2 blockade to harness anti-tumorigenic potential of macrophages against this disease. This dataset contain some prism documents, in order to help we attach a pdf information file about it
DOI: 10.5281/zenodo.3712806
title: lianneharrisonBAS/LCIS_paper: First release for paper review

description: This is the first release of data used for analysis of GRL paper for review.
DOI: 10.5281/zenodo.3712805
title: lianneharrisonBAS/LCIS_paper: First release for paper review

description: This is the first release of data used for analysis of GRL paper for review.
DOI: 10.5281/zenodo.3712793
title: Wikidata Dump species

description: RDF dump of wikidata produced with wdumps. species<br> View on wdumper <b>entity count</b>: 2722650, <b>statement count</b>: 24913063, <b>triple count</b>: 239639564
DOI: 10.5281/zenodo.3712792
title: Wikidata Dump species

description: RDF dump of wikidata produced with wdumps. species<br> View on wdumper <b>entity count</b>: 2722650, <b>statement count</b>: 24913063, <b>triple count</b>: 239639564
DOI: 10.5281/zenodo.3712856
title: Top PC Games based on the metascore (metacritic.com)

description: The dataset contains 6 columns and 199 rows of pc games with the highest metascore available on metacritic. The title and publisher column contains the title of the game and the name of the company that published the game. The rating column contains the rating of the game with "-" which means there is no rating. The user_score column contains the average rating given by the user to the game. The metascore column contains the results of the assessment conducted by the metacritics of the game.
DOI: 10.5281/zenodo.3712869
title: Dataset related to article "The macrophage tetraspan MS4A4A enhances dectin-1-dependent NK cell-mediated resistance to metastasis."

description: The plasma membrane tetraspan molecule MS4A4A is selectively expressed by macrophage-lineage cells, but its function is unknown. Here we report that MS4A4A was restricted to murine and human mononuclear phagocytes and was induced during monocyte-to-macrophage differentiation in the presence of interleukin 4 or dexamethasone. Human MS4A4A was co-expressed with M2/M2-like molecules in subsets of normal tissue-resident macrophages, infiltrating macrophages from inflamed synovium and tumor-associated macrophages. MS4A4A interacted and colocalized with the β-glucan receptor dectin-1 in lipid rafts. In response to dectin-1 ligands, Ms4a4a-deficient macrophages showed defective signaling and defective production of effector molecules. In experimental models of tumor progression and metastasis, Ms4a4a deficiency in macrophages had no impact on primary tumor growth, but was essential for dectin-1-mediated activation of macrophages and natural killer (NK) cell-mediated metastasis control. Thus, MS4A4A is a tetraspan molecule selectively expressed in macrophages during differentiation and polarization, essential for dectin-1-dependent activation of NK cell-mediated resistance to metastasis.
DOI: 10.5281/zenodo.3712870
title: Dataset related to article "The macrophage tetraspan MS4A4A enhances dectin-1-dependent NK cell-mediated resistance to metastasis."

description: The plasma membrane tetraspan molecule MS4A4A is selectively expressed by macrophage-lineage cells, but its function is unknown. Here we report that MS4A4A was restricted to murine and human mononuclear phagocytes and was induced during monocyte-to-macrophage differentiation in the presence of interleukin 4 or dexamethasone. Human MS4A4A was co-expressed with M2/M2-like molecules in subsets of normal tissue-resident macrophages, infiltrating macrophages from inflamed synovium and tumor-associated macrophages. MS4A4A interacted and colocalized with the β-glucan receptor dectin-1 in lipid rafts. In response to dectin-1 ligands, Ms4a4a-deficient macrophages showed defective signaling and defective production of effector molecules. In experimental models of tumor progression and metastasis, Ms4a4a deficiency in macrophages had no impact on primary tumor growth, but was essential for dectin-1-mediated activation of macrophages and natural killer (NK) cell-mediated metastasis control. Thus, MS4A4A is a tetraspan molecule selectively expressed in macrophages during differentiation and polarization, essential for dectin-1-dependent activation of NK cell-mediated resistance to metastasis.
DOI: 10.21335/nmdc-1664980441
title: Physical oceanography data from moorings in the Lofoten Basin, Norwegian Sea: June 2016 – September 2017

description: As a part of the "Water mass transformation processes and vortex dynamics in the Lofoten Basin of the Norwegian Sea" (PROVOLO) project, a set of 4 moorings was deployed in the eastern Lofoten Basin. The observations cover a 14-month period from June 2016 to September 2017. Three moorings were located on the shelf break and slope of the Lofoten Escarpment and one deeper in the basin. Mooring name convention is Mooring North (MN), South (MS), West (MW) and Basin (MB). MB was located in an energetic location but away from the slope to address the mesoscale variability in the basin. One netCDF file per mooring is provided: PROVOLO_Mooring_MS.nc PROVOLO_Mooring_MN.nc PROVOLO_Mooring_MW.nc PROVOLO_Mooring_MB.nc Time series of temperature salinity and horizontal currents are averaged in hourly intervals and vertically gridded at 10 m levels. The mooring motion is accounted for before gridding. PROVOLO was supported by the Research Council of Norway (project number 250784).
DOI: 10.5281/zenodo.3712912
title: Link to dataset related to article "FOXA2 controls the cis-regulatory networks of pancreatic cancer cells in a differentiation grade-specific manner."

description: Differentiation of normal and tumor cells is controlled by regulatory networks enforced by lineage-determining transcription factors (TFs). Among them, TFs such as FOXA1/2 bind naïve chromatin and induce its accessibility, thus establishing new gene regulatory networks. Pancreatic ductal adenocarcinoma (PDAC) is characterized by the coexistence of well- and poorly differentiated cells at all stages of disease. How the transcriptional networks determining such massive cellular heterogeneity are established remains to be determined. We found that FOXA2, a TF controlling pancreas specification, broadly contributed to the cis-regulatory networks of PDACs. Despite being expressed in both well- and poorly differentiated PDAC cells, FOXA2 displayed extensively different genomic distributions and controlled distinct gene expression programs. Grade-specific functions of FOXA2 depended on its partnership with TFs whose expression varied depending on the differentiation grade. These data suggest that FOXA2 contributes to the regulatory networks of heterogeneous PDAC cells via interactions with alternative partner TFs.
DOI: 10.14469/hpc/6991
title: 2-isopropyl-5-methylphenyl 2-(p-tolyl)acetate

description: Ester: clear, colourless liquid at standard conditions. Facile purification with aqueous organic workup. NMR in deuterated trichloromethane Impurities: Hexane, trichloromethane
DOI: 10.5281/zenodo.3712911
title: Link to dataset related to article "FOXA2 controls the cis-regulatory networks of pancreatic cancer cells in a differentiation grade-specific manner."

description: Differentiation of normal and tumor cells is controlled by regulatory networks enforced by lineage-determining transcription factors (TFs). Among them, TFs such as FOXA1/2 bind naïve chromatin and induce its accessibility, thus establishing new gene regulatory networks. Pancreatic ductal adenocarcinoma (PDAC) is characterized by the coexistence of well- and poorly differentiated cells at all stages of disease. How the transcriptional networks determining such massive cellular heterogeneity are established remains to be determined. We found that FOXA2, a TF controlling pancreas specification, broadly contributed to the cis-regulatory networks of PDACs. Despite being expressed in both well- and poorly differentiated PDAC cells, FOXA2 displayed extensively different genomic distributions and controlled distinct gene expression programs. Grade-specific functions of FOXA2 depended on its partnership with TFs whose expression varied depending on the differentiation grade. These data suggest that FOXA2 contributes to the regulatory networks of heterogeneous PDAC cells via interactions with alternative partner TFs.
DOI: 10.5281/zenodo.3712904
title: Link to dataset related to article "Dissection of acute stimulus-inducible nucleosome remodeling in mammalian cells"

description: Accessibility of the genomic regulatory information is largely controlled by the nucleosome-organizing activity of transcription factors (TFs). While stimulus-induced TFs bind to genomic regions that are maintained accessible by lineage-determining TFs, they also increase accessibility of thousands of <em>cis</em>-regulatory elements. Nucleosome remodeling events underlying such changes and their interplay with basal positioning are unknown. Here, we devised a novel quantitative framework discriminating different types of nucleosome remodeling events in micrococcal nuclease ChIP-seq (chromatin immunoprecipitation [ChIP] combined with high-throughput sequencing) data sets and used it to analyze nucleosome dynamics at stimulus-regulated <em>cis</em>-regulatory elements. At enhancers, remodeling preferentially affected poorly positioned nucleosomes while sparing well-positioned nucleosomes flanking the enhancer core, indicating that inducible TFs do not suffice to overrule basal nucleosomal organization maintained by lineage-determining TFs. Remodeling events appeared to be combinatorially driven by multiple TFs, with distinct TFs showing, however, different remodeling efficiencies. Overall, these data provide a systematic view of the impact of stimulation on nucleosome organization and genome accessibility in mammalian cells.