cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.5945935.v1
title: Additional file 1: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S1. The MtN3/saliva domain sequences of A. thaliana. (XLSX 27Â kb)
DOI: 10.6084/m9.figshare.5945947
title: Additional file 2: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S2. Primer sequences for qRT-PCR analysis. (XLSX 11Â kb)
DOI: 10.6084/m9.figshare.5945947.v1
title: Additional file 2: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S2. Primer sequences for qRT-PCR analysis. (XLSX 11Â kb)
DOI: 10.6084/m9.figshare.5945980
title: Additional file 5: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S3. Putative cis-elements in promoter region of the 34 MtN3/saliva/SWEETs genes in B. rapa. (XLSX 14Â kb)
DOI: 10.6084/m9.figshare.5945980.v1
title: Additional file 5: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S3. Putative cis-elements in promoter region of the 34 MtN3/saliva/SWEETs genes in B. rapa. (XLSX 14Â kb)
DOI: 10.6084/m9.figshare.5945992
title: Additional file 6: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S4. Secondary structural prediction of theMtN3/saliva/SWEETs proteins of B. rapa. (XLSX 52Â kb)
DOI: 10.6084/m9.figshare.5945992.v1
title: Additional file 6: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S4. Secondary structural prediction of theMtN3/saliva/SWEETs proteins of B. rapa. (XLSX 52Â kb)
DOI: 10.6084/m9.figshare.5946013
title: Additional file 7: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S5. Hydrophobic character prediction based on the Kyteâ Doolittle scale for the deduction of MtN3/saliva/SWEETs proteins in B. rapa. (XLSX 59Â kb)
DOI: 10.6084/m9.figshare.5946013.v1
title: Additional file 7: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S5. Hydrophobic character prediction based on the Kyteâ Doolittle scale for the deduction of MtN3/saliva/SWEETs proteins in B. rapa. (XLSX 59Â kb)
DOI: 10.6084/m9.figshare.5946025
title: Additional file 8: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S6. Protein subcellular localization prediction of MtN3/saliva/SWEETs proteins in B. rapa. (XLSX 11Â kb)
DOI: 10.6084/m9.figshare.5946025.v1
title: Additional file 8: of Genome-wide identification, phylogeny, evolution, and expression patterns of MtN3/saliva/SWEET genes and functional analysis of BcNS in Brassica rapa

description: Table S6. Protein subcellular localization prediction of MtN3/saliva/SWEETs proteins in B. rapa. (XLSX 11Â kb)
DOI: 10.6084/m9.figshare.5946115
title: Additional file 1: of Virulence gene profiles: alpha-hemolysin and clonal diversity in Staphylococcus aureus isolates from bovine clinical mastitis in China

description: Figure S1. Original picture of PCR result-1: hla; 2: hlb; 3: hlc; 4: icaD; 5: sec; 6: sei; 7: seg; 8: icaA; 9: tsst; 10: coa; 11: nuc; 12: clfA; 13: clfB; 14: fnbA; 15: fnbB. (ZIP 31 kb)
DOI: 10.6084/m9.figshare.5946115.v1
title: Additional file 1: of Virulence gene profiles: alpha-hemolysin and clonal diversity in Staphylococcus aureus isolates from bovine clinical mastitis in China

description: Figure S1. Original picture of PCR result-1: hla; 2: hlb; 3: hlc; 4: icaD; 5: sec; 6: sei; 7: seg; 8: icaA; 9: tsst; 10: coa; 11: nuc; 12: clfA; 13: clfB; 14: fnbA; 15: fnbB. (ZIP 31 kb)
DOI: 10.6084/m9.figshare.5946223
title: MOESM2 of Glycan analysis of colorectal cancer samples reveals stage-dependent changes in CEA glycosylation patterns

description: Additional file 2. 31 lectins bind different concentrations of CEA.
DOI: 10.6084/m9.figshare.5946223.v1
title: MOESM2 of Glycan analysis of colorectal cancer samples reveals stage-dependent changes in CEA glycosylation patterns

description: Additional file 2. 31 lectins bind different concentrations of CEA.
DOI: 10.6084/m9.figshare.5946238
title: MOESM3 of Glycan analysis of colorectal cancer samples reveals stage-dependent changes in CEA glycosylation patterns

description: Additional file 3. The correlation between other diseases and glycans on CEA.
DOI: 10.6084/m9.figshare.5946238.v1
title: MOESM3 of Glycan analysis of colorectal cancer samples reveals stage-dependent changes in CEA glycosylation patterns

description: Additional file 3. The correlation between other diseases and glycans on CEA.
DOI: 10.6084/m9.figshare.5946535
title: Distance-locations

description: Distances between locations
DOI: 10.6084/m9.figshare.5946535.v1
title: Distance-locations

description: Distances between locations
DOI: 10.6084/m9.figshare.5946547
title: Day-#-mobility

description: origin, destination, weight
DOI: 10.6084/m9.figshare.5946547.v1
title: Day-#-social

description: origin, destination, weight
DOI: 10.6084/m9.figshare.5946547.v2
title: Day-#-social

description: origin, destination, weight
DOI: 10.6084/m9.figshare.5946547.v3
title: Day-#-mobility

description: origin, destination, weight
DOI: 10.6084/m9.figshare.5946805
title: ecological risk assessment.xlsx

description: To examine the residues and distributions of
tetracycline antibiotics in Weihe basin, SPE-UPLC (solid phase extraction-ultra performance liquid
chromatography) was employed to analyze the Oxytetracycline (OTC),
Chlortetracycline (CTC) and Minocycline (MC) of 41 surface water and 35
sediment samples collected from main streams, tributaries and main sewage outlets. The Results showed that (1) The order of
residual levels of tetracycline antibiotics in water and sediment from high to
low was following: OTC&gt;CTC&gt;MC. The concentrations of OTC in water and
sediment were in the range of 1.56-87.89 ng/L and 6.13-45.38 ng/g (mean value
of 16.13 ng/L and 20.60 ng/g), respectively; while CTC was 1.07-26.78 ng/L and
6.17-32.29 ng/g (mean value of 4.96 ng/L and 14.48 ng/g), respectively; and MC
was 0.28-12.35 ng/L and 4.80-29.74 ng/g (mean value of 1.70 ng/L and 12.96
ng/g), respectively. There were maximum concentrations in all sewage outlets.
Compared with other areas in China, tetracyclines residues in Weihe basin was
at a medium level; (2) In spatial
distribution, the levels of tetracyclines in water and sediment from the middle
and upper reaches were higher than the lower reaches. Meanwhile, the sewage
outfalls and livestock farm waste water discharge were main sources of
tetracycline antibiotics in Weihe basin; (3) Ecological risk assessment
revealed that in main streams and tributaries, OTC and CTC may be at low
ecological risk level; while in sewage outfalls, they may be at medium ecological risk level.
DOI: 10.6084/m9.figshare.5946949
title: Datafile's for: Both parasite infection and social stress contribute to density-dependent immunity in populations of a small rodent species

description: Data for enclosure and lab experiment of Brandt's voles
DOI: 10.6084/m9.figshare.5946949.v1
title: Datafile's for: Both parasite infection and social stress contribute to density-dependent immunity in populations of a small rodent species

description: Data for enclosure and lab experiment of Brandt's voles
DOI: 10.6084/m9.figshare.5947435
title: 418filtering.hmp.txt

description: The SNPs which detected by 418 peach accession
DOI: 10.6084/m9.figshare.5947435.v1
title: 418filtering.hmp.txt

description: The SNPs which detected by 418 peach accession
DOI: 10.6084/m9.figshare.5951116
title: Additional file 2: of Serum lipids as an indicator for the alteration of liver function in patients with hepatitis B

description: Lipid profile and serum fatty acid composition data of hepatitis B patients and controls. (XLSX 57 kb)
DOI: 10.6084/m9.figshare.5951116.v1
title: Additional file 2: of Serum lipids as an indicator for the alteration of liver function in patients with hepatitis B

description: Lipid profile and serum fatty acid composition data of hepatitis B patients and controls. (XLSX 57 kb)
DOI: 10.6084/m9.figshare.5951203
title: Additional file 1: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Experimental design of microarray hybridizations. A Microsoft Excel file containing a single work sheet “Array Hybridization Design” which describes the hybridization scheme for 32 Arizona 20.7 K chicken oligo arrays used for the riboflavin deficient chick embryo study. (XLSX 15 kb)
DOI: 10.6084/m9.figshare.5951203.v1
title: Additional file 1: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Experimental design of microarray hybridizations. A Microsoft Excel file containing a single work sheet “Array Hybridization Design” which describes the hybridization scheme for 32 Arizona 20.7 K chicken oligo arrays used for the riboflavin deficient chick embryo study. (XLSX 15 kb)
DOI: 10.6084/m9.figshare.5951224
title: Additional file 2: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Primers used for qRT-PCR analysis. A Microsoft Excel file containing a single work sheet “qRT-PCR Primer Information”. This table provides the chicken gene symbol, Roslin Institute Gallus gallus (RIGG) oligo ID number, 5′-3′ sequence for forward and reverse primers, and amplicon size (bp) for each gene used for qRT-PCR analysis. (XLSX 16 kb)
DOI: 10.6084/m9.figshare.5951224.v1
title: Additional file 2: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Primers used for qRT-PCR analysis. A Microsoft Excel file containing a single work sheet “qRT-PCR Primer Information”. This table provides the chicken gene symbol, Roslin Institute Gallus gallus (RIGG) oligo ID number, 5′-3′ sequence for forward and reverse primers, and amplicon size (bp) for each gene used for qRT-PCR analysis. (XLSX 16 kb)
DOI: 10.6084/m9.figshare.5951233
title: Additional file 3: of TNFα induces Ca2+ influx to accelerate extrinsic apoptosis in hepatocellular carcinoma cells

description: Figure S2. Decreased cytosolic Ca2+ level attenuates TNFα-induced apoptosis of HCC cells. (a) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in SNU739 and HLF cells with treatment as indicated. BAPTA-AM: 10 μM. (b) Cell apoptosis analysis by flow cytometry 24 h after treatment as indicated before TNFα (100 ng/mL) stimulation. BAPTA-AM: 10 μM. (c) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in HCC cells with treatment as indicated. EV: cells transfected with the empty vector; PV-OE: cells stably forced expressing Parvalbumin protein. (d) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. (e) Western Blot analysis for Parvalbumin expression in SNU739 and HLF cells with treatment as indicated. (f) Cell apoptosis analysis by flow cytometry 24 h after treatment as indicated. CAI: 10 μM; SKF96365: 100 μM. (g) Cell apoptosis analysis by flow cytometry 24 h after treatment as indicated. siTRPM7: siRNA against TRPM7; si Ctrl: negative control siRNA. Data were shown as mean ± SD. All experiments were performed at least three times. * P &lt; 0.05; ** P &lt; 0.01. (ZIP 1986 kb)
DOI: 10.6084/m9.figshare.5951233.v1
title: Additional file 3: of TNFα induces Ca2+ influx to accelerate extrinsic apoptosis in hepatocellular carcinoma cells

description: Figure S2. Decreased cytosolic Ca2+ level attenuates TNFα-induced apoptosis of HCC cells. (a) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in SNU739 and HLF cells with treatment as indicated. BAPTA-AM: 10 μM. (b) Cell apoptosis analysis by flow cytometry 24 h after treatment as indicated before TNFα (100 ng/mL) stimulation. BAPTA-AM: 10 μM. (c) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in HCC cells with treatment as indicated. EV: cells transfected with the empty vector; PV-OE: cells stably forced expressing Parvalbumin protein. (d) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. (e) Western Blot analysis for Parvalbumin expression in SNU739 and HLF cells with treatment as indicated. (f) Cell apoptosis analysis by flow cytometry 24 h after treatment as indicated. CAI: 10 μM; SKF96365: 100 μM. (g) Cell apoptosis analysis by flow cytometry 24 h after treatment as indicated. siTRPM7: siRNA against TRPM7; si Ctrl: negative control siRNA. Data were shown as mean ± SD. All experiments were performed at least three times. * P &lt; 0.05; ** P &lt; 0.01. (ZIP 1986 kb)
DOI: 10.6084/m9.figshare.5951248
title: Additional file 3: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Differentially-expressed (Adjusted P ≤ 0.05; FDR ≤ 0.05) genes identified in liver of e13 and e15 embryos. A Microsoft Excel file containing two worksheets. Work sheets “Riboflavin e13_396 DE genes” and “Riboflavin e15_1467 DE genes” list information about differentially expressed genes determined by microarray analysis. Each list provides the Roslin Institute Gallus gallus (RIGG) oligo ID, gene symbol, gene description, log2 fold change (Rf+/Rf-), average expression (Ave/Expr), t-statistic, P-value, P-value adjusted for multiple testing (adj.P ≤ 0.05), B-statistic from Limma software, and the Ref-Seq peptide ID for each gene (oligo). (XLSX 328 kb)
DOI: 10.6084/m9.figshare.5951248.v1
title: Additional file 3: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Differentially-expressed (Adjusted P ≤ 0.05; FDR ≤ 0.05) genes identified in liver of e13 and e15 embryos. A Microsoft Excel file containing two worksheets. Work sheets “Riboflavin e13_396 DE genes” and “Riboflavin e15_1467 DE genes” list information about differentially expressed genes determined by microarray analysis. Each list provides the Roslin Institute Gallus gallus (RIGG) oligo ID, gene symbol, gene description, log2 fold change (Rf+/Rf-), average expression (Ave/Expr), t-statistic, P-value, P-value adjusted for multiple testing (adj.P ≤ 0.05), B-statistic from Limma software, and the Ref-Seq peptide ID for each gene (oligo). (XLSX 328 kb)
DOI: 10.6084/m9.figshare.5951251
title: Additional file 4: of TNFα induces Ca2+ influx to accelerate extrinsic apoptosis in hepatocellular carcinoma cells

description: Figure S3. Cytosolic Ca2+ sensitized HCC cells to TNFα-induced apoptosis. (a) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. (b) and (c) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in HLF and QSG-7701 cells with treatment as indicated. TNFα (50 ng/mL) + Iono: 50 ng/mL TNFα combined with 1 μM ionomycin, TNFα (100 ng/mL) + Iono: 100 ng/mL TNFα combined with 1 μM ionomycin, TNFα (200 ng/mL) + Iono: 200 ng/mL TNFα combined with 1 μM ionomycin. TNFα (50 ng/mL) + IP3: 50 ng/mL TNFα combined with 10 μM IP3; TNFα (100 ng/mL) + IP3: 100 ng/mL TNFα combined with 10 μM IP3; TNFα (200 ng/mL) + IP3: 200 ng/mL TNFα combined with 10 μM IP3; (d)-(f) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. Data were shown as mean ± SD. All experiments were performed at least three times. * P &lt; 0.05; ** P &lt; 0.01. (ZIP 2448 kb)
DOI: 10.6084/m9.figshare.5951251.v1
title: Additional file 4: of TNFα induces Ca2+ influx to accelerate extrinsic apoptosis in hepatocellular carcinoma cells

description: Figure S3. Cytosolic Ca2+ sensitized HCC cells to TNFα-induced apoptosis. (a) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. (b) and (c) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in HLF and QSG-7701 cells with treatment as indicated. TNFα (50 ng/mL) + Iono: 50 ng/mL TNFα combined with 1 μM ionomycin, TNFα (100 ng/mL) + Iono: 100 ng/mL TNFα combined with 1 μM ionomycin, TNFα (200 ng/mL) + Iono: 200 ng/mL TNFα combined with 1 μM ionomycin. TNFα (50 ng/mL) + IP3: 50 ng/mL TNFα combined with 10 μM IP3; TNFα (100 ng/mL) + IP3: 100 ng/mL TNFα combined with 10 μM IP3; TNFα (200 ng/mL) + IP3: 200 ng/mL TNFα combined with 10 μM IP3; (d)-(f) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. Data were shown as mean ± SD. All experiments were performed at least three times. * P &lt; 0.05; ** P &lt; 0.01. (ZIP 2448 kb)
DOI: 10.6084/m9.figshare.5951263
title: Additional file 4: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Lists of “Analysis Ready” DE genes (AR-DE genes) assigned by IPA to “Canonical Pathways”. A Microsoft Excel file containing a five worksheets of the top “Canonical Pathways” identified by IPA in riboflavin-rescued and riboflavin deficient chick embryos on embryonic day 15 (e15). Five major canonical pathways identified by IPA among DE genes on embryonic day 15 (e15) were: “EIF2 Signaling, Acute-phase Signaling, LXR-RXR Activation, FXR-RXR Activation, Intrinsic Prothrombin Activation”, and “Blood Clotting” in the “Biological Function Category” of IPA. Each worksheet provides the gene symbol, Entrez gene name, gene expression as log2 ratio (Rf+/Rf-), and Ref-Seq protein ID or Entrez gene ID. (XLSX 27 kb)
DOI: 10.6084/m9.figshare.5951263.v1
title: Additional file 4: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Lists of “Analysis Ready” DE genes (AR-DE genes) assigned by IPA to “Canonical Pathways”. A Microsoft Excel file containing a five worksheets of the top “Canonical Pathways” identified by IPA in riboflavin-rescued and riboflavin deficient chick embryos on embryonic day 15 (e15). Five major canonical pathways identified by IPA among DE genes on embryonic day 15 (e15) were: “EIF2 Signaling, Acute-phase Signaling, LXR-RXR Activation, FXR-RXR Activation, Intrinsic Prothrombin Activation”, and “Blood Clotting” in the “Biological Function Category” of IPA. Each worksheet provides the gene symbol, Entrez gene name, gene expression as log2 ratio (Rf+/Rf-), and Ref-Seq protein ID or Entrez gene ID. (XLSX 27 kb)
DOI: 10.6084/m9.figshare.5951269
title: Additional file 5: of TNFα induces Ca2+ influx to accelerate extrinsic apoptosis in hepatocellular carcinoma cells

description: Figure S4. The level of extracellular calcium influx is positively correlated with TNFα-mediated apoptosis. (a) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in 10 kinds of HCC cells with treatment as indicated. (b) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. All experiments were performed at least three times. (ZIP 2184 kb)
DOI: 10.6084/m9.figshare.5951269.v1
title: Additional file 5: of TNFα induces Ca2+ influx to accelerate extrinsic apoptosis in hepatocellular carcinoma cells

description: Figure S4. The level of extracellular calcium influx is positively correlated with TNFα-mediated apoptosis. (a) Confocal microscope analysis of [Ca2+]c level using fluorescent probe Fura-2/AM in 10 kinds of HCC cells with treatment as indicated. (b) Apoptosis analysis by flow cytometry 24 h after treatment as indicated. All experiments were performed at least three times. (ZIP 2184 kb)
DOI: 10.6084/m9.figshare.5951275
title: Additional file 5: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: IPA “Molecular and Cellular Functions” of AR-DE genes in riboflavin-rescued and riboflavin deficient chick embryos on embryonic day 15 (e15). A Microsoft Excel file containing nine worksheets of over-populated “Molecular and Cellular Functions” identified by IPA: “Fatty Acid Metabolism, Oxidation of Lipid, Carbohydrate (CHO) Metabolism, Catabolism of Protein, Metabolism of Amino Acids (AA), Protein Ubiquitination, Metabolism of Vitamins” and 29 AR-DE genes found in the chicken “Flavoproteome”. Each worksheet provides protein ID, gene symbol and gene expression as log2 ratio (Rf+/Rf-). (XLSX 35 kb)
DOI: 10.6084/m9.figshare.5951275.v1
title: Additional file 5: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: IPA “Molecular and Cellular Functions” of AR-DE genes in riboflavin-rescued and riboflavin deficient chick embryos on embryonic day 15 (e15). A Microsoft Excel file containing nine worksheets of over-populated “Molecular and Cellular Functions” identified by IPA: “Fatty Acid Metabolism, Oxidation of Lipid, Carbohydrate (CHO) Metabolism, Catabolism of Protein, Metabolism of Amino Acids (AA), Protein Ubiquitination, Metabolism of Vitamins” and 29 AR-DE genes found in the chicken “Flavoproteome”. Each worksheet provides protein ID, gene symbol and gene expression as log2 ratio (Rf+/Rf-). (XLSX 35 kb)
DOI: 10.6084/m9.figshare.5951290
title: Additional file 6: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Pearson’s correlation analysis of gene expression levels of 21 DE “candidate” genes determined by 20.7 K microarray analysis and by qRT-PCR analysis. A Microsoft Excel file containing normalized expression levels (log2 ratio of Rf+/Rf-) of 21 DE candidate genes averaged across e13 and e15 as determined by both microarray analysis and qRT-PCR analysis. The Pearson’s correlation coefficient (r = 0.834; 19 degrees of freedom) indicates a highly significant (P ≤ 0.01) correlation between gene expression levels obtained from both microarray and qRT-PCR analyses. (XLSX 13 kb)
DOI: 10.6084/m9.figshare.5951290.v1
title: Additional file 6: of Transcriptional profiling of liver in riboflavin-deficient chicken embryos explains impaired lipid utilization, energy depletion, massive hemorrhaging, and delayed feathering

description: Pearson’s correlation analysis of gene expression levels of 21 DE “candidate” genes determined by 20.7 K microarray analysis and by qRT-PCR analysis. A Microsoft Excel file containing normalized expression levels (log2 ratio of Rf+/Rf-) of 21 DE candidate genes averaged across e13 and e15 as determined by both microarray analysis and qRT-PCR analysis. The Pearson’s correlation coefficient (r = 0.834; 19 degrees of freedom) indicates a highly significant (P ≤ 0.01) correlation between gene expression levels obtained from both microarray and qRT-PCR analyses. (XLSX 13 kb)