cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.5281/zenodo.7305615
title: Carrefour's Product Set

description: <strong>El dataset representa els productes de carrefour.es que fan referència a menjar. Consta de 10746 productes dels quals se’n representen 31 atributs. El camp id fa referència a l’identificador únic de cada producte.</strong> <strong>Les dades han estat obtingudes de la web carrefour.es entre els dies 03/11/2022 a 08/11/2022.</strong>
DOI: 10.21942/uva.21493275
title: Dataset Endogenous Group Formation and Responsibility Diffusion

description: The dataset includes all necessary observations and variables to replicate the results for this project. The codebook includes explanations of variables for the dataset decisionsbeliefs.dta.
DOI: 10.21942/uva.21493275.v1
title: Dataset Endogenous Group Formation and Responsibility Diffusion

description: The dataset includes all necessary observations and variables to replicate the results for this project. The codebook includes explanations of variables for the dataset decisionsbeliefs.dta.
DOI: 10.34740/kaggle/dsv/4472446
title: Eurovision Winners 1956 - 2022
DOI: 10.34740/kaggle/dsv/4472452
title: Eurovision Winners 1956 - 2022
DOI: 10.15472/3sfior
title: Línea base general de ecotoxicología para el valle medio del Magdalena - VMM

description: Bajo los Convenios No. 21-095 y 21-450 suscritos con la Agencia Nacional de Hidrocarburos - ANH, se llevó a cabo el levantamiento de la línea base general de la biodiversidad y ecosistemas para el área de interés del Valle Medio del Magdalena. Para la línea base de contaminantes se priorizaron 23 compuestos que suelen estar asociados a procesos de explotación de petróleo y gas a nivel global. Estos se encuentran clasificados por un análisis Toxpi. Los compuestos se tomaron de muestras en las matrices de agua, agua subterránea, sedimentos y suelos. Como compuestos priorizados se encontraban los plaguicidas organoclorados (OCP) delta-Lindano y Endosulfan I, dentro de los plaguicidas organofosforados (OPP) el Tributilfosfato, dentro de los hidrocarburos Poliaromáticos (PAH) el Naftaleno, Benz[a]antraceno, Benzo[b]fluoranteno , Benzo[a]pireno y Dibenzo[a,h]antraceno; dentro de los trihalometanos (THM) se analizaron el Tetracloroetileno, 1,1,2,2-Tetracloroetano, Nitrobenceno y 1,2,3-Triclorobenceno; para los BTEX se analizó el Benceno y Tolueno y adicionalmente el 1,4 diclorobenceno y dentro de la fracción volatil se analizaron el Decano, 1,4-Dioxano, Ácido acético, 2 etil-1-Hexanol, Ácido isovalérico, 1-Decanol, Fenol y Ácido dodecanoico. Adicionalmente de los diferentes tipos de compuestos mencionados, (OCP, OPP, BTEX, THM, y PHAS) se obtuvieron resultados de otros compuestos que se incluían entre los análisis de estos. El presente conjunto de datos cuenta con 87 muestras analizadas.
DOI: 10.6084/m9.figshare.13352827.v1
title: Crocin inhibits the activation of mouse hepatic stellate cells via the lnc-LFAR1/MTF-1/GDNF pathway

description: Crocin is the main monomer of saffron, which is a momentous component of traditional Chinese medicine Lang Qing A Ta. Here, we tried to probe into the role of crocin in liver fibrosis. Hematoxylin-eosin staining and Sirius Red staining were used to observe the pathological changes of liver tissues. After hepatic stellate cells (HSCs) were isolated from liver tissues, lnc-LFAR1, MTF-1, GDNF, and α-SMA expressions were detected by qRT-PCR and western blot. Immunohistochemistry and immunofluorescence were used to detect α-SMA expression. Chromatin immunoprecipitation was used to analyze the binding of MTF-1 to the GDNF promoter. Moreover, the dual-luciferase reporter gene, RNA pull-down, and RNA immunoprecipitation were used to clarify the interaction between MTF-1 and GDNF, lnc-LFAR1 and MTF-1. The degree of liver fibrosis was more severe in the mice from the liver fibrosis model, while the liver fibrosis was alleviated by the injection of crocin. lnc-LFAR1, GDNF, and α-SMA were up-regulated, and MTF-1 was down-regulated in liver fibrosis tissues and cells, while these trends were reversed after the injection of crocin. Besides, lnc-LFAR1 negatively regulated MTF-1 expression, and positively regulated GDNF and α-SMA expressions, and MTF-1 was enriched in the promoter region of GDNF. Furthermore, the cellular direct interactions between MTF-1 and GDNF, lnc-LFAR1 and MTF-1 were verified. <i>In vivo</i> experiments confirmed the relief of crocin on liver fibrosis. Our research expounded that crocin restrained the activation of HSCs through the lnc-LFAR1/MTF-1/GDNF axis, thereby ameliorating liver fibrosis.
DOI: 10.6084/m9.figshare.13359578.v1
title: DCAF13 promotes triple-negative breast cancer metastasis by mediating DTX3 mRNA degradation

description: DCAF13 is firstly identified as a substrate receptor of CUL4-DDB1 E3 ligase complex. This study disclosed that DCAF13 acted as a novel RNA binding protein (RBP) that contributed to triple-negative breast cancer (TNBC) metastasis. Clinical data obtained from TCGA and our collection showed that DCAF13 was closely correlated with poor clinicopathological characteristics and overall survival, which indicated DCAF13 may serve as a diagnostic marker for TNBC metastasis. Functionally, DCAF13 overexpression or suppression was sufficient to enhance or decrease breast cancer cell migration and invasion. Mechanistically, DCAF13 functioned as an RBP by binding with the AU-rich element (ARE) of DTX3 mRNA 3ʹUTR to accelerate its degradation. Moreover, we identified that DTX3 promoted the ubiquitination and degradation of NOTCH4. Finally, increased DCAF13 expression led to post-transcriptional decay of DTX3 mRNA and consequently activated of NOTCH4 signaling pathway in TNBC. In conclusion, these results identified that DCAF13 as a diagnostic marker and therapeutic target for TNBC treatment. <b>Abbreviation:</b> DCAF13: DDB1 and CUL4-associated factor 13; DDB1: DNA-binding protein 1; CUL4: Cullin 4; CRL4, Cullin-ring finger ligase 4; RBP: RNA binding protein; TNBC: triple-negative breast cancer; ARE: AU-rich element; DTX3: Deltex E3 ubiquitin ligase 3; HER2: human epidermal growth factor receptor 2; ER: estrogen receptor; PR: progesterone receptor; PTEN: phosphatase and tensin homolog deleted on chromosome 10; EMT: epithelial-mesenchymal transition.
DOI: 10.6084/m9.figshare.13352827
title: Crocin inhibits the activation of mouse hepatic stellate cells via the lnc-LFAR1/MTF-1/GDNF pathway

description: Crocin is the main monomer of saffron, which is a momentous component of traditional Chinese medicine Lang Qing A Ta. Here, we tried to probe into the role of crocin in liver fibrosis. Hematoxylin-eosin staining and Sirius Red staining were used to observe the pathological changes of liver tissues. After hepatic stellate cells (HSCs) were isolated from liver tissues, lnc-LFAR1, MTF-1, GDNF, and α-SMA expressions were detected by qRT-PCR and western blot. Immunohistochemistry and immunofluorescence were used to detect α-SMA expression. Chromatin immunoprecipitation was used to analyze the binding of MTF-1 to the GDNF promoter. Moreover, the dual-luciferase reporter gene, RNA pull-down, and RNA immunoprecipitation were used to clarify the interaction between MTF-1 and GDNF, lnc-LFAR1 and MTF-1. The degree of liver fibrosis was more severe in the mice from the liver fibrosis model, while the liver fibrosis was alleviated by the injection of crocin. lnc-LFAR1, GDNF, and α-SMA were up-regulated, and MTF-1 was down-regulated in liver fibrosis tissues and cells, while these trends were reversed after the injection of crocin. Besides, lnc-LFAR1 negatively regulated MTF-1 expression, and positively regulated GDNF and α-SMA expressions, and MTF-1 was enriched in the promoter region of GDNF. Furthermore, the cellular direct interactions between MTF-1 and GDNF, lnc-LFAR1 and MTF-1 were verified. <i>In vivo</i> experiments confirmed the relief of crocin on liver fibrosis. Our research expounded that crocin restrained the activation of HSCs through the lnc-LFAR1/MTF-1/GDNF axis, thereby ameliorating liver fibrosis.
DOI: 10.6084/m9.figshare.13359578
title: DCAF13 promotes triple-negative breast cancer metastasis by mediating DTX3 mRNA degradation

description: DCAF13 is firstly identified as a substrate receptor of CUL4-DDB1 E3 ligase complex. This study disclosed that DCAF13 acted as a novel RNA binding protein (RBP) that contributed to triple-negative breast cancer (TNBC) metastasis. Clinical data obtained from TCGA and our collection showed that DCAF13 was closely correlated with poor clinicopathological characteristics and overall survival, which indicated DCAF13 may serve as a diagnostic marker for TNBC metastasis. Functionally, DCAF13 overexpression or suppression was sufficient to enhance or decrease breast cancer cell migration and invasion. Mechanistically, DCAF13 functioned as an RBP by binding with the AU-rich element (ARE) of DTX3 mRNA 3ʹUTR to accelerate its degradation. Moreover, we identified that DTX3 promoted the ubiquitination and degradation of NOTCH4. Finally, increased DCAF13 expression led to post-transcriptional decay of DTX3 mRNA and consequently activated of NOTCH4 signaling pathway in TNBC. In conclusion, these results identified that DCAF13 as a diagnostic marker and therapeutic target for TNBC treatment. <b>Abbreviation:</b> DCAF13: DDB1 and CUL4-associated factor 13; DDB1: DNA-binding protein 1; CUL4: Cullin 4; CRL4, Cullin-ring finger ligase 4; RBP: RNA binding protein; TNBC: triple-negative breast cancer; ARE: AU-rich element; DTX3: Deltex E3 ubiquitin ligase 3; HER2: human epidermal growth factor receptor 2; ER: estrogen receptor; PR: progesterone receptor; PTEN: phosphatase and tensin homolog deleted on chromosome 10; EMT: epithelial-mesenchymal transition.
DOI: 10.6084/m9.figshare.13385126
title: Autophosphorylation-induced self-assembly and STIL-dependent reinforcement underlie Plk4’s ring-to-dot localization conversion around a human centriole

description: Polo-like kinase 4 (Plk4) is a key regulator of centriole biogenesis. Studies have shown that Plk4 undergoes dynamic relocalization from a ring-like pattern around a centriole to a dot-like morphology at the procentriole assembly site and this event is central for inducing centriole biogenesis. However, the detailed mechanisms underlying Plk4’s capacity to drive its symmetry-breaking ring-to-dot relocalization remain largely unknown. Here, we showed that Plk4 self-initiates this process in an autophosphorylation–dependent manner and that STIL, its downstream target, is not required for this event. Time-dependent analyses with mEOS-fused photoconvertible Plk4 revealed that a portion of ring-state Plk4 acquires a capacity, presumably through autophosphorylation, to linger around a centriole, ultimately generating a dot-state morphology. Interestingly, Plk4 WT, but not its catalytically inactive mutant, showed the ability to form a nanoscale spherical assembly in the cytosol of human cells or heterologous <i>E. coli</i>, demonstrating its autophosphorylation-dependent self-organizing capacity. At the biochemical level, Plk4 – unlike its N-terminal βTrCP degron motif – robustly autophosphorylated the PC3 SSTT motif within its C-terminal cryptic polo-box, an event critical for inducing its physical clustering. Additional <i>in vivo</i> experiments showed that although STIL was not required for Plk4’s initial ring-to-dot conversion, coexpressed STIL greatly enhanced Plk4’s ability to generate a spherical condensate and recruit Sas6, a major component of the centriolar cartwheel structure. We propose that Plk4’s autophosphorylation-induced clustering is sufficient to induce its ring-to-dot localization conversion and that subsequently recruited STIL potentiates this process to generate a procentriole assembly body critical for Plk4-dependent centriole biogenesis.
DOI: 10.6084/m9.figshare.13385126.v1
title: Autophosphorylation-induced self-assembly and STIL-dependent reinforcement underlie Plk4’s ring-to-dot localization conversion around a human centriole

description: Polo-like kinase 4 (Plk4) is a key regulator of centriole biogenesis. Studies have shown that Plk4 undergoes dynamic relocalization from a ring-like pattern around a centriole to a dot-like morphology at the procentriole assembly site and this event is central for inducing centriole biogenesis. However, the detailed mechanisms underlying Plk4’s capacity to drive its symmetry-breaking ring-to-dot relocalization remain largely unknown. Here, we showed that Plk4 self-initiates this process in an autophosphorylation–dependent manner and that STIL, its downstream target, is not required for this event. Time-dependent analyses with mEOS-fused photoconvertible Plk4 revealed that a portion of ring-state Plk4 acquires a capacity, presumably through autophosphorylation, to linger around a centriole, ultimately generating a dot-state morphology. Interestingly, Plk4 WT, but not its catalytically inactive mutant, showed the ability to form a nanoscale spherical assembly in the cytosol of human cells or heterologous <i>E. coli</i>, demonstrating its autophosphorylation-dependent self-organizing capacity. At the biochemical level, Plk4 – unlike its N-terminal βTrCP degron motif – robustly autophosphorylated the PC3 SSTT motif within its C-terminal cryptic polo-box, an event critical for inducing its physical clustering. Additional <i>in vivo</i> experiments showed that although STIL was not required for Plk4’s initial ring-to-dot conversion, coexpressed STIL greatly enhanced Plk4’s ability to generate a spherical condensate and recruit Sas6, a major component of the centriolar cartwheel structure. We propose that Plk4’s autophosphorylation-induced clustering is sufficient to induce its ring-to-dot localization conversion and that subsequently recruited STIL potentiates this process to generate a procentriole assembly body critical for Plk4-dependent centriole biogenesis.
DOI: 10.6084/m9.figshare.21263191
title: Asynchronous and Distributed Data Augmentation for Massive Data Settings

description: Data augmentation (DA) algorithms are slow in massive data settings due to multiple passes through the entire data. We address this problem by developing a DA extension that exploits asynchronous and distributed computing. The extended DA algorithm is called Asynchronous and Distributed (AD) DA with the original DA as its parent. Any ADDA is indexed by a parameter r∈(0,1) and starts by dividing the entire data into <i>k</i> disjoint subsets and storing them on <i>k</i> processes. Every iteration of ADDA augments only an <i>r</i>-fraction of the <i>k</i> data subsets with some positive probability and leaves the remaining (1−r)-fraction of the augmented data unchanged. The parameter draws are obtained using the <i>r</i>-fraction of new and (1−r)-fraction of old augmented data. We show that the ADDA Markov chain is Harris ergodic with the desired stationary distribution under mild conditions on the parent DA algorithm. We demonstrate that ADDA is significantly faster than its parent for many (<i>k</i>, <i>r</i>) choices in three representative models. We also establish the geometric ergodicity of the ADDA Markov chain for all the three models, which yields asymptotically valid standard errors for estimates of desired posterior quantities. Supplementary materials for this article are available online.
DOI: 10.6084/m9.figshare.21263191.v1
title: Asynchronous and Distributed Data Augmentation for Massive Data Settings

description: Data augmentation (DA) algorithms are slow in massive data settings due to multiple passes through the entire data. We address this problem by developing a DA extension that exploits asynchronous and distributed computing. The extended DA algorithm is called Asynchronous and Distributed (AD) DA with the original DA as its parent. Any ADDA is indexed by a parameter r∈(0,1) and starts by dividing the entire data into <i>k</i> disjoint subsets and storing them on <i>k</i> processes. Every iteration of ADDA augments only an <i>r</i>-fraction of the <i>k</i> data subsets with some positive probability and leaves the remaining (1−r)-fraction of the augmented data unchanged. The parameter draws are obtained using the <i>r</i>-fraction of new and (1−r)-fraction of old augmented data. We show that the ADDA Markov chain is Harris ergodic with the desired stationary distribution under mild conditions on the parent DA algorithm. We demonstrate that ADDA is significantly faster than its parent for many (<i>k</i>, <i>r</i>) choices in three representative models. We also establish the geometric ergodicity of the ADDA Markov chain for all the three models, which yields asymptotically valid standard errors for estimates of desired posterior quantities. Supplementary materials for this article are available online.
DOI: 10.26093/cds/vizier.36420153
title: SAGAN. I. New sample & multi-wavelength studies

description: VizieR online Data Catalogue associated with article published in journal Astronomy & Astrophysics with title 'Search and analysis of giant radio galaxies with associated nuclei (SAGAN). I. New sample and multi-wavelength studies.' (bibcode: 2020A&A...642A.153D)
DOI: 10.26093/cds/vizier.18830156
title: Nobeyama 45m Cygnus-X CO. II. C180 clumps

description: VizieR online Data Catalogue associated with article published in journal Astronomical Journal (AAS) with title 'Nobeyama 45m Cygnus-X CO survey. II. Physical properties of C^18^O clumps.' (bibcode: 2019ApJ...883..156T)
DOI: 10.26093/cds/vizier.18850053
title: Spectroscopy & HST photometry of galaxy Leo V

description: VizieR online Data Catalogue associated with article published in journal Astronomical Journal (AAS) with title 'Signatures of Tidal Disruption in Ultra-faint Dwarf Galaxies: a Combined HST, Gaia, and MMT/Hectochelle study of Leo V.' (bibcode: 2019ApJ...885...53M)
DOI: 10.26093/cds/vizier.18880078
title: The ACS-AGN Catalog: AGN luminosity vs SFR

description: VizieR online Data Catalogue associated with article published in journal Astronomical Journal (AAS) with title 'A catalog of AGN host galaxies observed with HST/ACS: correlations between star formation and AGN activity.' (bibcode: 2020ApJ...888...78S)
DOI: 10.26093/cds/vizier.22060010
title: CANDELS multiwavelength catalog

description: VizieR online Data Catalogue associated with article published in journal Astronomical Journal (AAS) with title 'CANDELS multiwavelength catalogs: source identification and photometry in the CANDELS UKIDSS ultra-deep survey field.' (bibcode: 2013ApJS..206...10G)
DOI: 10.23641/asha.21498711
title: Sensory impairments and subjective cognition (Smith et al., 2022)

description: <strong>Purpose:</strong> Subjective cognitive complaints (SCCs) are associated with poor quality of life, important for clinical care planning and management, and may predict dementia diagnosis. Dual sensory impairment (DSI) is a risk factor for dementia, but whether DSI is associated with SCCs is unknown. We evaluated whether self-reported DSI is associated with SCCs. <strong>Method: </strong>We performed a cross-sectional analysis of 9,899 community-dwelling respondents aged 60+ years without dementia or depression in the 2019 National Health Interview Survey. Participants self-reported difficulty remembering or concentrating, seeing even when wearing corrective lenses, and hearing even when using a hearing aid. We defined SCCs and sensory impairment for each mode as reporting at least some difficulty. We categorized sensory impairment into no sensory impairment, vision impairment only, hearing impairment only, and DSI. We then estimated weighted prevalence ratios (PRs) of SCCs by impairment category. <strong>Results:</strong> After weighting (9,899 participants representing a weighted <em>n</em> = 59,261,749), 12% of participants reported vision impairment only, 19% reported hearing impairment only, and 7% reported DSI. Relative to no impairment, after adjustment for potential confounders, vision impairment (PR = 2.07; 95% confidence interval [CI] [1.79, 2.39]), hearing impairment (PR = 2.26; 95% CI [2.00, 2.55]), and DSI (PR = 3.21; 95% CI [2.83, 3.63]) were associated with an increased prevalence of SCCs. <strong>Conclusions: </strong>In this nationally representative survey of older Americans, DSI was associated with a threefold increased prevalence of SCCs. Although cross-sectional, these data underscore the importance of assessing multiple impairments as exposures when studying subjective cognition in older adults. <strong>Supplemental Material S1.</strong> Description of variables derived from questions in the 2019 NHIS. <strong>Supplemental Material S2.</strong> Interaction between impairment status and age category on subjective cognitive complaints among eligible participants in the 2019 NHIS. <strong>Supplemental Material S3. </strong>Prevalence ratio of subjective cognitive complaints by impairment status after restricting sample to participants aged 60-85 years in the 2019 NHIS. Smith, J. R., Betz, J. F., Garcia, E. E., Jiang, K., Swenor, B. K., Reed, N. S., & Deal, J. A. (2022). Self-reported dual sensory impairment and subjective cognitive complaints among older adults in the 2019 National Health Interview Survey. <em>American Journal of Audiology</em>. Advance online publication. https://doi.org/10.1044/2022_AJA-22-00087
DOI: 10.6084/m9.figshare.21520923
title: Respiratory and diarrhoea pathogens in Malawian children presenting woth diarrhoea and association with short term growth dataset

description: This is a dataset of participants enrolled and followed in the CryptoResp sub-study looking at the association between respiratory and diarrhoea pathogens and growth in children 2-24 months.
DOI: 10.6084/m9.figshare.21520923.v1
title: Respiratory and diarrhoea pathogens in Malawian children presenting woth diarrhoea and association with short term growth dataset

description: This is a dataset of participants enrolled and followed in the CryptoResp sub-study looking at the association between respiratory and diarrhoea pathogens and growth in children 2-24 months.
DOI: 10.3886/icpsr38234.v1
title: Federal Justice Statistics Program: Offenders Admitted to Prison, 2018
title: Version 1

description: The data contain records of sentenced offenders committed to the custody of the Bureau of Prisons (BOP) during fiscal year 2018. The data include commitments of United States District Court, violators of conditions of release (e.g., parole, probation, or supervised release violators), offenders convicted in other courts (e.g., military or District of Columbia courts), and persons admitted to prison as material witnesses or for purposes of treatment, examination, or transfer to another authority. These data include variables that describe the offender, such as age, race, citizenship, as well as variables that describe the sentences and expected prison terms. The data file contains original variables from the Bureau of Prisons' SENTRY database as well as additional analysis variables. Variables containing identifying information (e.g., name, Social Security Number) were either removed, coarsened, or blanked in order to protect the identities of individuals. These data are part of a series designed by the Urban Institute (Washington, D.C.) and the Bureau of Justice Statistics. Data and documentation were prepared by the Urban Institute through 2012. Data from 2013 and on were prepared by Abt Associates.
description: Datasets: DS1: Federal Justice Statistics Program: Offenders Admitted to Prison, 2018
description: Offenders in the custody of the United States Bureau of Prisons during fiscal year 2018.
DOI: 10.7299/x7dn45c2
title: Arctos Project Record: Differential diversification rates of ratsnakes in Asia and Europe