cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.1136017.v38
title: Low-frequency sound affects active micromechanics in the human inner ear

description: Noise-induced hearing loss is one of the most common auditory pathologies, resulting from overstimulation of the human cochlea, an exquisitely sensitive micromechanical device. At very low frequencies (&lt;250 Hz), however, the sensitivity of human hearing, and therefore the perceived loudness is poor. The perceived loudness is mediated by the inner hair cells of the cochlea, which are driven very inadequately at low frequencies. To assess the impact of low-frequency sound, we exploited a by-product of the active amplification of sound outer hair cells perform, so-called spontaneous otoacoustic emissions. These are faint sounds produced by the inner ear that can be used to detect changes of cochlear physiology. We show that a short exposure to perceptually unobtrusive, low-frequency sounds significantly affects outer hair cells: A 90 s, 80 dB(A) low-frequency sound induced slow, concordant and positively correlated frequency- and level oscillations of spontaneous otoacoustic emissions which lasted for about two minutes after low-frequency sound offset. Low-frequency sounds, contrary to their unobtrusive perception, strongly stimulate the human cochlea and affect amplification processes in the most sensitive and important frequency range of human hearing.
DOI: 10.6084/m9.figshare.1136017.v39
title: Low-frequency sound affects active micromechanics in the human inner ear

description: Noise-induced hearing loss is one of the most common auditory pathologies, resulting from overstimulation of the human cochlea, an exquisitely sensitive micromechanical device. At very low frequencies (&lt;250 Hz), however, the sensitivity of human hearing, and therefore the perceived loudness is poor. The perceived loudness is mediated by the inner hair cells of the cochlea, which are driven very inadequately at low frequencies. To assess the impact of low-frequency sound, we exploited a by-product of the active amplification of sound outer hair cells perform, so-called spontaneous otoacoustic emissions. These are faint sounds produced by the inner ear that can be used to detect changes of cochlear physiology. We show that a short exposure to perceptually unobtrusive, low-frequency sounds significantly affects outer hair cells: A 90 s, 80 dB(A) low-frequency sound induced slow, concordant and positively correlated frequency- and level oscillations of spontaneous otoacoustic emissions which lasted for about two minutes after low-frequency sound offset. Low-frequency sounds, contrary to their unobtrusive perception, strongly stimulate the human cochlea and affect amplification processes in the most sensitive and important frequency range of human hearing.
DOI: 10.6084/m9.figshare.1136017.v40
title: Low-frequency sound affects active micromechanics in the human inner ear

description: Noise-induced hearing loss is one of the most common auditory pathologies, resulting from overstimulation of the human cochlea, an exquisitely sensitive micromechanical device. At very low frequencies (&lt;250 Hz), however, the sensitivity of human hearing, and therefore the perceived loudness is poor. The perceived loudness is mediated by the inner hair cells of the cochlea, which are driven very inadequately at low frequencies. To assess the impact of low-frequency sound, we exploited a by-product of the active amplification of sound outer hair cells perform, so-called spontaneous otoacoustic emissions. These are faint sounds produced by the inner ear that can be used to detect changes of cochlear physiology. We show that a short exposure to perceptually unobtrusive, low-frequency sounds significantly affects outer hair cells: A 90 s, 80 dB(A) low-frequency sound induced slow, concordant and positively correlated frequency- and level oscillations of spontaneous otoacoustic emissions which lasted for about two minutes after low-frequency sound offset. Low-frequency sounds, contrary to their unobtrusive perception, strongly stimulate the human cochlea and affect amplification processes in the most sensitive and important frequency range of human hearing.
DOI: 10.6084/m9.figshare.1136017.v41
title: Low-frequency sound affects active micromechanics in the human inner ear

description: Noise-induced hearing loss is one of the most common auditory pathologies, resulting from overstimulation of the human cochlea, an exquisitely sensitive micromechanical device. At very low frequencies (&lt;250 Hz), however, the sensitivity of human hearing, and therefore the perceived loudness is poor. The perceived loudness is mediated by the inner hair cells of the cochlea, which are driven very inadequately at low frequencies. To assess the impact of low-frequency sound, we exploited a by-product of the active amplification of sound outer hair cells perform, so-called spontaneous otoacoustic emissions. These are faint sounds produced by the inner ear that can be used to detect changes of cochlear physiology. We show that a short exposure to perceptually unobtrusive, low-frequency sounds significantly affects outer hair cells: A 90 s, 80 dB(A) low-frequency sound induced slow, concordant and positively correlated frequency- and level oscillations of spontaneous otoacoustic emissions which lasted for about two minutes after low-frequency sound offset. Low-frequency sounds, contrary to their unobtrusive perception, strongly stimulate the human cochlea and affect amplification processes in the most sensitive and important frequency range of human hearing.
DOI: 10.6084/m9.figshare.1134597
title: Genotype calls from Rare Variant Sanger Sequencing in 1,998 Individuals

description: Available genotype calls from rare variant sanger sequencing performed on 1,998 individuals. From published paper: <em>The Empirical Power of Rare Variant Association Methods: Results from Sanger Sequencing in 1,998 Individuals</em>. Martin Ladouceur, Zari Dastani, Yurii S. Aulchenko, Celia M. T. Greenwood, J. Brent Richards. Published online February 2nd 2012. DOI: 10.1371/journal.pgen.1002496
DOI: 10.6084/m9.figshare.1136017.v42
title: Low-frequency sound affects active micromechanics in the human inner ear

description: Noise-induced hearing loss is one of the most common auditory pathologies, resulting from overstimulation of the human cochlea, an exquisitely sensitive micromechanical device. At very low frequencies (&lt;250 Hz), however, the sensitivity of human hearing, and therefore the perceived loudness is poor. The perceived loudness is mediated by the inner hair cells of the cochlea, which are driven very inadequately at low frequencies. To assess the impact of low-frequency sound, we exploited a by-product of the active amplification of sound outer hair cells perform, so-called spontaneous otoacoustic emissions. These are faint sounds produced by the inner ear that can be used to detect changes of cochlear physiology. We show that a short exposure to perceptually unobtrusive, low-frequency sounds significantly affects outer hair cells: A 90 s, 80 dB(A) low-frequency sound induced slow, concordant and positively correlated frequency- and level oscillations of spontaneous otoacoustic emissions which lasted for about two minutes after low-frequency sound offset. Low-frequency sounds, contrary to their unobtrusive perception, strongly stimulate the human cochlea and affect amplification processes in the most sensitive and important frequency range of human hearing.
DOI: 10.6084/m9.figshare.1134597.v1
title: Genotype calls from Rare Variant Sanger Sequencing in 1,998 Individuals

description: Available genotype calls from rare variant sanger sequencing performed on 1,998 individuals. From published paper: <em>The Empirical Power of Rare Variant Association Methods: Results from Sanger Sequencing in 1,998 Individuals</em>. Martin Ladouceur, Zari Dastani, Yurii S. Aulchenko, Celia M. T. Greenwood, J. Brent Richards. Published online February 2nd 2012. DOI: 10.1371/journal.pgen.1002496
DOI: 10.6084/m9.figshare.1134559.v1
title: Gaussian Job Archive for C10H13(1+)

description: Archive of work produced using the resources of the Imperial College High Performance Computing Service.
DOI: 10.6084/m9.figshare.1136017.v43
title: Low-frequency sound affects active micromechanics in the human inner ear

description: Noise-induced hearing loss is one of the most common auditory pathologies, resulting from overstimulation of the human cochlea, an exquisitely sensitive micromechanical device. At very low frequencies (&lt;250 Hz), however, the sensitivity of human hearing, and therefore the perceived loudness is poor. The perceived loudness is mediated by the inner hair cells of the cochlea, which are driven very inadequately at low frequencies. To assess the impact of low-frequency sound, we exploited a by-product of the active amplification of sound outer hair cells perform, so-called spontaneous otoacoustic emissions. These are faint sounds produced by the inner ear that can be used to detect changes of cochlear physiology. We show that a short exposure to perceptually unobtrusive, low-frequency sounds significantly affects outer hair cells: A 90 s, 80 dB(A) low-frequency sound induced slow, concordant and positively correlated frequency- and level oscillations of spontaneous otoacoustic emissions which lasted for about two minutes after low-frequency sound offset. Low-frequency sounds, contrary to their unobtrusive perception, strongly stimulate the human cochlea and affect amplification processes in the most sensitive and important frequency range of human hearing.
DOI: 10.6084/m9.figshare.1136017.v44
title: Low-frequency sound affects active micromechanics in the human inner ear

description: Noise-induced hearing loss is one of the most common auditory pathologies, resulting from overstimulation of the human cochlea, an exquisitely sensitive micromechanical device. At very low frequencies (&lt;250 Hz), however, the sensitivity of human hearing, and therefore the perceived loudness is poor. The perceived loudness is mediated by the inner hair cells of the cochlea, which are driven very inadequately at low frequencies. To assess the impact of low-frequency sound, we exploited a by-product of the active amplification of sound outer hair cells perform, so-called spontaneous otoacoustic emissions. These are faint sounds produced by the inner ear that can be used to detect changes of cochlear physiology. We show that a short exposure to perceptually unobtrusive, low-frequency sounds significantly affects outer hair cells: A 90 s, 80 dB(A) low-frequency sound induced slow, concordant and positively correlated frequency- and level oscillations of spontaneous otoacoustic emissions which lasted for about two minutes after low-frequency sound offset. Low-frequency sounds, contrary to their unobtrusive perception, strongly stimulate the human cochlea and affect amplification processes in the most sensitive and important frequency range of human hearing.
DOI: 10.6084/m9.figshare.1134556.v1
title: Gaussian Job Archive for C10H13(1+)

description: Archive of work produced using the resources of the Imperial College High Performance Computing Service.
DOI: 10.6084/m9.figshare.1134543
title: Properties of anti-RhD antibodies from alternative cell lines

description: Supplementary figures, data and full datasets from the manuscript entitled "Clearance of human IgG1-sensitised red blood cells i<em>n vivo</em> in humans relates to the <em>in vitro</em> properties of antibodies from alternative cell lines."
DOI: 10.6084/m9.figshare.1134540
title: The Physicality of Monetary Agency

description: 10 brief theses for monetary reform
DOI: 10.6084/m9.figshare.1134540.v1
title: The Physicality of Monetary Agency

description: 10 brief theses for monetary reform
DOI: 10.6084/m9.figshare.1134543.v2
title: Properties of anti-RhD antibodies from alternative cell lines

description: Supplementary figures, data and full datasets from the manuscript entitled "Clearance of human IgG1-sensitised red blood cells i<em>n vivo</em> in humans relates to the <em>in vitro</em> properties of antibodies from alternative cell lines."
DOI: 10.6084/m9.figshare.1134536
title: Figure 4 full dataset

description: Full dataset for Figure 4 from "Clearance of human IgG1-sensitised red blood cells <em>in vivo</em> in humans relates to the <em>in vitro</em> properties of antibodies from alternative cell lines."
DOI: 10.6084/m9.figshare.1134535
title: Figure 3 full dataset

description: Full dataset for Figure 3 from "Clearance of human IgG1-sensitised red blood cells <em>in vivo</em> in humans relates to the<em> in vitro</em> properties of antibodies from alternative cell lines." - See more at: http://figshare.com/preview/_preview/1134536#sthash.NWlsQhPA.dpuf
DOI: 10.6084/m9.figshare.1134543.v1
title: Properties of anti-D antibodies from alternative cell lines

description: Supplementary figures, data and full datasets from the manuscript entitled "Clearance of human IgG1-sensitised red blood cells i<em>n vivo</em> in humans relates to the <em>in vitro</em> properties of antibodies from alternative cell lines."
DOI: 10.6084/m9.figshare.1134534
title: Figure 2 full dataset

description: Full dataset for Figure 2 from "Clearance of human IgG1-sensitised red blood cells i<em>n vivo</em> in humans relates to the <em>in vitro</em> properties of antibodies from alternative cell lines."
DOI: 10.6084/m9.figshare.1134535.v1
title: Figure 3 full dataset

description: Full dataset for Figure 3 from "Clearance of human IgG1-sensitised red blood cells <em>in vivo</em> in humans relates to the<em> in vitro</em> properties of antibodies from alternative cell lines." - See more at: http://figshare.com/preview/_preview/1134536#sthash.NWlsQhPA.dpuf
DOI: 10.6084/m9.figshare.1134536.v1
title: Figure 4 full dataset

description: Full dataset for Figure 4 from "Clearance of human IgG1-sensitised red blood cells <em>in vivo</em> in humans relates to the <em>in vitro</em> properties of antibodies from alternative cell lines."
DOI: 10.6084/m9.figshare.1134533
title: Figure 1 full dataset

description: Full dataset for Figure 1 from "Clearance of human IgG1-sensitised red blood cells in vivo in humans relates to the in vitro properties of antibodies from alternative cell lines."
DOI: 10.6084/m9.figshare.1134510
title: Geochemical constraints on the tectonic setting of basaltic host rocks to the Windy Craggy Cu-Co-Au massive sulphide deposit, northwestern British Columbia

description: Windy Craggy is an approximately 300 Mt Cu-Co-Au volcanogenic massive sulphide (VMS) deposit in northwestern British Columbia, Canada. The Windy Craggy deposit is hosted by the Middle Tats Volcanics (MTV), a Late Triassic volcano-sedimentary sequence of intercalated mafic pillowed to massive volcanic flows and sills and calcareous argillite that are part of the Alexander terrane. The host footwall and hangingwall flows and sills are predominantly alkalic basalts (Nb/Y &gt; 0.70). MTV alkali basalts at Windy Craggy are enriched in light rare earth elements (LREEs) &gt;100X chondrite compared to chondrite, have steep REE patterns [(La/Yb)<sub>cn</sub> = 7.1–25.4], and generally lack the Ta and Nb depletions relative to primitive mantle (e.g. [Nb/Th]<sub>pm</sub> = 0.68–1.94) characteristic of arc environments, although most have [Nb/La]<sub>pm</sub> cn = 0.41–10.6], and show well-developed Ta and Nb depletions (arc signatures; [Nb/Th]<sub>pm</sub> = 0.20–0.79), consistent with formation in an oceanic arc environment. The co-occurrence of tholeiitic/calc-alkaline arc rocks with alkalic rocks indicates that the LTV (former) and MTV (latter) formed from melts that were influenced to varying degrees by subducted oceanic crust, and likely formed within a back-arc basin setting formed on a rifted oceanic arc. There is no geochemical or isotopic evidence for major involvement of continental crust. The LTV basalts likely were produced by progressive depletion in the source by partial melting of mantle overlying the subducting oceanic crust. The presence of the MTV alkalic Windy Craggy rocks overlying the LTV is consistent with the presence of a slab-window, perhaps related to subduction of a spreading centre, which allowed more enriched magmas to reach the surface with only minimal interaction with subduction-modified mantle. The presence of this slab-window might have provided the mechanism for the generation of anomalously high heat flow close to the seafloor, which initiated and sustained vigorous, long-lived hydrothermal activity necessary for the precipitation of large accumulations of massive sulphide. To our knowledge, this is the first example of a large VMS deposit associated with a slab-window.
DOI: 10.6084/m9.figshare.1134506
title: Table 11. Keywords centrality measures

description: Table 11. Keywords centrality measures
DOI: 10.6084/m9.figshare.1134506.v1
title: Table 11. Keywords centrality measures

description: Table 11. Keywords centrality measures
DOI: 10.6084/m9.figshare.1134501
title: Table 9. Articles' methodological approach

description: Table 9. Articles' methodological approach
DOI: 10.6084/m9.figshare.1134510.v1
title: Geochemical constraints on the tectonic setting of basaltic host rocks to the Windy Craggy Cu-Co-Au massive sulphide deposit, northwestern British Columbia

description: Windy Craggy is an approximately 300 Mt Cu-Co-Au volcanogenic massive sulphide (VMS) deposit in northwestern British Columbia, Canada. The Windy Craggy deposit is hosted by the Middle Tats Volcanics (MTV), a Late Triassic volcano-sedimentary sequence of intercalated mafic pillowed to massive volcanic flows and sills and calcareous argillite that are part of the Alexander terrane. The host footwall and hangingwall flows and sills are predominantly alkalic basalts (Nb/Y &gt; 0.70). MTV alkali basalts at Windy Craggy are enriched in light rare earth elements (LREEs) &gt;100X chondrite compared to chondrite, have steep REE patterns [(La/Yb)<sub>cn</sub> = 7.1–25.4], and generally lack the Ta and Nb depletions relative to primitive mantle (e.g. [Nb/Th]<sub>pm</sub> = 0.68–1.94) characteristic of arc environments, although most have [Nb/La]<sub>pm</sub> cn = 0.41–10.6], and show well-developed Ta and Nb depletions (arc signatures; [Nb/Th]<sub>pm</sub> = 0.20–0.79), consistent with formation in an oceanic arc environment. The co-occurrence of tholeiitic/calc-alkaline arc rocks with alkalic rocks indicates that the LTV (former) and MTV (latter) formed from melts that were influenced to varying degrees by subducted oceanic crust, and likely formed within a back-arc basin setting formed on a rifted oceanic arc. There is no geochemical or isotopic evidence for major involvement of continental crust. The LTV basalts likely were produced by progressive depletion in the source by partial melting of mantle overlying the subducting oceanic crust. The presence of the MTV alkalic Windy Craggy rocks overlying the LTV is consistent with the presence of a slab-window, perhaps related to subduction of a spreading centre, which allowed more enriched magmas to reach the surface with only minimal interaction with subduction-modified mantle. The presence of this slab-window might have provided the mechanism for the generation of anomalously high heat flow close to the seafloor, which initiated and sustained vigorous, long-lived hydrothermal activity necessary for the precipitation of large accumulations of massive sulphide. To our knowledge, this is the first example of a large VMS deposit associated with a slab-window.
DOI: 10.6084/m9.figshare.1134500
title: Table 8. Universities

description: Table 8. Universities
DOI: 10.6084/m9.figshare.1134499
title: Table 7. Countries citations

description: Table 7. Countries citations
DOI: 10.6084/m9.figshare.1134500.v1
title: Table 8. Universities

description: Table 8. Universities
DOI: 10.6084/m9.figshare.1134510.v3
title: Geochemical constraints on the tectonic setting of basaltic host rocks to the Windy Craggy Cu-Co-Au massive sulphide deposit, northwestern British Columbia

description: Windy Craggy is an approximately 300 Mt Cu-Co-Au volcanogenic massive sulphide (VMS) deposit in northwestern British Columbia, Canada. The Windy Craggy deposit is hosted by the Middle Tats Volcanics (MTV), a Late Triassic volcano-sedimentary sequence of intercalated mafic pillowed to massive volcanic flows and sills and calcareous argillite that are part of the Alexander terrane. The host footwall and hangingwall flows and sills are predominantly alkalic basalts (Nb/Y &gt; 0.70). MTV alkali basalts at Windy Craggy are enriched in light rare earth elements (LREEs) &gt;100X chondrite compared to chondrite, have steep REE patterns [(La/Yb)<sub>cn</sub> = 7.1–25.4], and generally lack the Ta and Nb depletions relative to primitive mantle (e.g. [Nb/Th]<sub>pm</sub> = 0.68–1.94) characteristic of arc environments, although most have [Nb/La]<sub>pm</sub> cn = 0.41–10.6], and show well-developed Ta and Nb depletions (arc signatures; [Nb/Th]<sub>pm</sub> = 0.20–0.79), consistent with formation in an oceanic arc environment. The co-occurrence of tholeiitic/calc-alkaline arc rocks with alkalic rocks indicates that the LTV (former) and MTV (latter) formed from melts that were influenced to varying degrees by subducted oceanic crust, and likely formed within a back-arc basin setting formed on a rifted oceanic arc. There is no geochemical or isotopic evidence for major involvement of continental crust. The LTV basalts likely were produced by progressive depletion in the source by partial melting of mantle overlying the subducting oceanic crust. The presence of the MTV alkalic Windy Craggy rocks overlying the LTV is consistent with the presence of a slab-window, perhaps related to subduction of a spreading centre, which allowed more enriched magmas to reach the surface with only minimal interaction with subduction-modified mantle. The presence of this slab-window might have provided the mechanism for the generation of anomalously high heat flow close to the seafloor, which initiated and sustained vigorous, long-lived hydrothermal activity necessary for the precipitation of large accumulations of massive sulphide. To our knowledge, this is the first example of a large VMS deposit associated with a slab-window.
DOI: 10.6084/m9.figshare.1134498
title: Table 5. MOOC average number of citations per year

description: Table 5. MOOC average number of citations per year
DOI: 10.6084/m9.figshare.1134499.v1
title: Table 7. Countries citations

description: Table 7. Countries citations
DOI: 10.6084/m9.figshare.1134496
title: Table 4. MOOC evolution of citations

description: Table 4. MOOC evolution of citations