cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.11829531
title: Extensive immunoinformatics study for the prediction of novel peptide-based epitope vaccine with docking confirmation against envelope protein of Chikungunya virus: a computational biology approach

description: Chikungunya virus (CHIKV) instigating Chikungunya fever is a global infective menace resulting in high fever, weakened joint-muscle pain, and brain inflammation. Inaccessibility and unavailability of effective drugs have led us to an uncertain arena when it comes to providing proper medical treatment to the affected people. In this study, authentic encroachment has been made concerning the peptide-based epitope vaccine designing against CHIKV. A Proteome-wide search was performed to locate a conserved portion among the accessible viral outer membrane proteins which showcase a remarkable immune response using specific immunoinformatics and docking simulation tools. Primarily, the most probable immunogenic envelope glycoproteins E1 and E2 were identified from the UniProt database depending on their antigenicity scores. Subsequently, we selected two distinctive sequences “SEDVYANTQLVLQRP” and “IMLLYPDHPTLLSYR” in both E1 and E2 glycoproteins respectively. These two sequences identified as the most potent T and B cell epitope-based peptides as they interacted with 6 and 7 HLA-I and 5 HLA-II molecules with an extremely low IC50 score that was verified by molecular docking. Moreover, the sequences possess no allergenicity and are certainly located outside the transmembrane region. In addition, the sequences exhibited 88.46% and 100.00% Conservancy, covering high population coverage of 89.49% to 94.74% and 60.51% to 88.87% respectively in endemic countries. The identified peptide SEDVYANTQLVLQRP and IMLLYPDHPTLLSYR can be utilized next for the development of peptide-based epitope vaccine contrary to CHIKV, so further documentations and experimentations like Antigen testing, Antigen production, Clinical trials are needed to prove the validity of it. Communicated by Ramaswamy H. Sarma
DOI: 10.6084/m9.figshare.11889303
title: A computational structural biology study to understand the impact of mutation on structure–function relationship of inward-rectifier potassium ion channel Kir6.2 in human

description: Type 2 diabetes (T2D) is clinically characterized via hyperglycemia. Polymorphism rs5219 in the <i>KCNJ11</i> gene is a risk factor for developing T2D in humans. <i>KCNJ11</i> encodes the ‘inward-rectifier potassium ion channel (Kir6.2)’. However, because of the absence of the complete crystal/NMR structures of Kir6.2 proteins, insight into its structure and function and its interaction with diverse ligands remain elusive to date. Therefore, a computational approach was employed for predicting the best plausible ‘three-dimensional’ structure of Kir6.2 as well as for studying the influence of mutation (p. GLU23LYS) on both architectures as well as the function of Kir6.2 employing simulation studies. Results obtained revealed that though, with increased time, ‘Gibbs free energy’ becomes positive, residues in wild type Kir6.2 experiences less random movement as compared to mutant Kir6.2. The less random movement of residues in wild type Kir6.2 represents the standard coupling between open and closing of ‘K<sub>ATP</sub> channel’ and thus the normal secretion of insulin. The more dispersed motion of mutant Kir6.2 residues represents ‘overactivity’ of the ‘K<sub>ATP</sub> channel’ and thus insulin ‘under-secretion’. Further, molecular docking and simulation studies identified two phytochemicals/drugs, namely, A-348441 and chushizisin I, which retains the wild type property of Kir6.2 after binding with mutant protein. Unlike A-348441, this is for the first time, the present study is reporting about the plausible anti-diabetic property of chushizisin I. As these two phytochemicals/drugs, namely, A-348441 and chushizisin I, have passed ADMET test, in the near future, they may be utilized as anti-diabetic drugs after further investigation. Communicated by Ramaswamy H. Sarma
DOI: 10.6084/m9.figshare.11889303.v2
title: A computational structural biology study to understand the impact of mutation on structure–function relationship of inward-rectifier potassium ion channel Kir6.2 in human

description: Type 2 diabetes (T2D) is clinically characterized via hyperglycemia. Polymorphism rs5219 in the <i>KCNJ11</i> gene is a risk factor for developing T2D in humans. <i>KCNJ11</i> encodes the ‘inward-rectifier potassium ion channel (Kir6.2)’. However, because of the absence of the complete crystal/NMR structures of Kir6.2 proteins, insight into its structure and function and its interaction with diverse ligands remain elusive to date. Therefore, a computational approach was employed for predicting the best plausible ‘three-dimensional’ structure of Kir6.2 as well as for studying the influence of mutation (p. GLU23LYS) on both architectures as well as the function of Kir6.2 employing simulation studies. Results obtained revealed that though, with increased time, ‘Gibbs free energy’ becomes positive, residues in wild type Kir6.2 experiences less random movement as compared to mutant Kir6.2. The less random movement of residues in wild type Kir6.2 represents the standard coupling between open and closing of ‘K<sub>ATP</sub> channel’ and thus the normal secretion of insulin. The more dispersed motion of mutant Kir6.2 residues represents ‘overactivity’ of the ‘K<sub>ATP</sub> channel’ and thus insulin ‘under-secretion’. Further, molecular docking and simulation studies identified two phytochemicals/drugs, namely, A-348441 and chushizisin I, which retains the wild type property of Kir6.2 after binding with mutant protein. Unlike A-348441, this is for the first time, the present study is reporting about the plausible anti-diabetic property of chushizisin I. As these two phytochemicals/drugs, namely, A-348441 and chushizisin I, have passed ADMET test, in the near future, they may be utilized as anti-diabetic drugs after further investigation. Communicated by Ramaswamy H. Sarma
DOI: 10.5281/zenodo.4569982
title: The impact of Covid-19 outbreak on sleep behavior and training characteristics of top-level track and field athletes.

description: The Covid-19 outbreak forced many governments to enter a nationwide lockdown and many national and international competitions, such as the Olympic Games in Tokyo, were canceled. The lockdown determined several negative changes in the general population, such as the increase in anxiety, stress, sedentary levels and sleep problems and prevented athletes from training; therefore, the aim of this study was to evaluate, by means of a survey, changes in sleep parameters and PA characteristics of elite athletes of the Italian Track and Field Federation in three periods of the 2020: before the lockdown (T0), during the lockdown (09<sup>th</sup> March – 03<sup>rd</sup> May, T1) and the first month after the lockdown (T2). This study was conducted from May 2020 to June 2020 and data were collected using an offline survey with 89 elite track and field athletes (mean age: 24.7±5.4; mean BMI: 20.7±3.0; n=43 males [48.3%] and n=46 females [51.7%]). The survey was composed by demographic data and questions on PA and sleep behavior at T0, T1 and T2. Primary outcomes of PA were hours of training per week (Th/w), hours of training per day (Th/d) and number of training sessions per week (T/w) whereas sleep parameters included wake-up time (WT), bedtime (BT), sleep latency (SL), total sleep time (TST) and sleep quality (SQ). SQ decreased at T1 compared to T0 and T2 (p&lt;0.0001) and athletes registered delayed BT (00:13±1:05), WT (8:41±1:20) and longer SL (28.1±29.8) at T1 compared to T0 (BT: 23:27±0:45; WT: 7:57±1:05; SL: 18.1±14.4) and T2 (BT: 23:38±0:44; WT: 8:04±0:53; SL: 19.4±15.9) whereas no changes in TST were reported. Similarly, significantly lower values of Th/w, Th/d, and T/w (p&lt;0.0001) were registered at T1 compared to T0 and T2: the weekly training volume decreased from 16.1±5.7 hours at T0 to 10.7±5.7 hours during the lockdown. Conversely, no PA and sleep differences between male and female athletes and between short- and long-term disciplines were detected. In addition, alcohol (p=0.136) and caffeine intake (p=0.990) and the use of electronic devices (p=0.317) were similar pre-, during, and post-lockdown. The lockdown measures determined a significant decrease in training volume and frequency along with a worsening in sleep quality, sleep latency, and a delay in bedtimes and get-up times in top-level track and field athletes. With continuous spikes in Covid-19 cases around the world and associated local/regional lockdowns, these results provides important insights to better manage sleep and training in a population for whom optimal performance is essential.
DOI: 10.5281/zenodo.4569981
title: The impact of Covid-19 outbreak on sleep behavior and training characteristics of top-level track and field athletes.

description: The Covid-19 outbreak forced many governments to enter a nationwide lockdown and many national and international competitions, such as the Olympic Games in Tokyo, were canceled. The lockdown determined several negative changes in the general population, such as the increase in anxiety, stress, sedentary levels and sleep problems and prevented athletes from training; therefore, the aim of this study was to evaluate, by means of a survey, changes in sleep parameters and PA characteristics of elite athletes of the Italian Track and Field Federation in three periods of the 2020: before the lockdown (T0), during the lockdown (09<sup>th</sup> March – 03<sup>rd</sup> May, T1) and the first month after the lockdown (T2). This study was conducted from May 2020 to June 2020 and data were collected using an offline survey with 89 elite track and field athletes (mean age: 24.7±5.4; mean BMI: 20.7±3.0; n=43 males [48.3%] and n=46 females [51.7%]). The survey was composed by demographic data and questions on PA and sleep behavior at T0, T1 and T2. Primary outcomes of PA were hours of training per week (Th/w), hours of training per day (Th/d) and number of training sessions per week (T/w) whereas sleep parameters included wake-up time (WT), bedtime (BT), sleep latency (SL), total sleep time (TST) and sleep quality (SQ). SQ decreased at T1 compared to T0 and T2 (p&lt;0.0001) and athletes registered delayed BT (00:13±1:05), WT (8:41±1:20) and longer SL (28.1±29.8) at T1 compared to T0 (BT: 23:27±0:45; WT: 7:57±1:05; SL: 18.1±14.4) and T2 (BT: 23:38±0:44; WT: 8:04±0:53; SL: 19.4±15.9) whereas no changes in TST were reported. Similarly, significantly lower values of Th/w, Th/d, and T/w (p&lt;0.0001) were registered at T1 compared to T0 and T2: the weekly training volume decreased from 16.1±5.7 hours at T0 to 10.7±5.7 hours during the lockdown. Conversely, no PA and sleep differences between male and female athletes and between short- and long-term disciplines were detected. In addition, alcohol (p=0.136) and caffeine intake (p=0.990) and the use of electronic devices (p=0.317) were similar pre-, during, and post-lockdown. The lockdown measures determined a significant decrease in training volume and frequency along with a worsening in sleep quality, sleep latency, and a delay in bedtimes and get-up times in top-level track and field athletes. With continuous spikes in Covid-19 cases around the world and associated local/regional lockdowns, these results provides important insights to better manage sleep and training in a population for whom optimal performance is essential.
DOI: 10.5281/zenodo.4450345
title: The decarbonisation of Europe powered by lifestyle changes

description: This is the main data set for the publication "The decarbonisation of Europe powered by lifestyle changes".
DOI: 10.5281/zenodo.4450344
title: The decarbonisation of Europe powered by lifestyle changes

description: This is the main data set for the publication "The decarbonisation of Europe powered by lifestyle changes".
DOI: 10.6084/m9.figshare.14135162
title: Job Crafting and Work Engagement: the mediating role of Work Meaning.

description: Job Crafting and Work Engagement: the mediating role of Work Meaning.
DOI: 10.7284/134659
title: CTD (Conductivity, Temperature, Depth) data collected during research cruise AR31-A using a Sea-Bird SBE-911+ instrument system onboard the platform RV Neil Armstrong

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.7284/134674
title: CTD (Conductivity, Temperature, Depth) data collected during research cruise AR31-B using a Sea-Bird SBE-911+ instrument system onboard the platform RV Neil Armstrong

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.5281/zenodo.4569980
title: Impaired dopamine- and adenosine-mediated signaling and plasticity in a novel rodent model for DYT25 dystonia

description: <strong>Raw data of Figure 5 </strong>- (D) Summary plots of Tau values (left) and AMPA/NMDA ratio (right), measured from Gnal+/+ and Gnal+/- EPSCs showing no significant difference between genotypes (Tau: Gnal+/+, 9.47 ± 0.50 ms, n=31; Gnal+/-, 10.05 ± 0.66 ms, n=27; p=0.4832; AMPA/NMDA ratio: Gnal+/+ , 1.27 ± 0.14, n=10; Gnal+/-, 1.24 ± 0.13, n=15; Mann Whitney test p=0.9779). (E) Summary plot of PPR values showing similar facilitation in both genotypes. Each data point represents mean ± SEM (Gnal+/+, n=30, 50 ms: 1.22 ± 0.04; 100 ms: 1.16 ± 0.03; 150 ms: 1.11 ± 0.40; Gnal+/-, n=25, 50 ms: 1.28 ± 0.03; 100 ms: 1.17 ± 0.03; 150 ms: 1.09 ± 0.02; two-way ANOVA and Bonferroni posttest: ISI p&lt;0.0001, genotype p=0.2405).
DOI: 10.5281/zenodo.4569979
title: Impaired dopamine- and adenosine-mediated signaling and plasticity in a novel rodent model for DYT25 dystonia

description: <strong>Raw data of Figure 5 </strong>- (D) Summary plots of Tau values (left) and AMPA/NMDA ratio (right), measured from Gnal+/+ and Gnal+/- EPSCs showing no significant difference between genotypes (Tau: Gnal+/+, 9.47 ± 0.50 ms, n=31; Gnal+/-, 10.05 ± 0.66 ms, n=27; p=0.4832; AMPA/NMDA ratio: Gnal+/+ , 1.27 ± 0.14, n=10; Gnal+/-, 1.24 ± 0.13, n=15; Mann Whitney test p=0.9779). (E) Summary plot of PPR values showing similar facilitation in both genotypes. Each data point represents mean ± SEM (Gnal+/+, n=30, 50 ms: 1.22 ± 0.04; 100 ms: 1.16 ± 0.03; 150 ms: 1.11 ± 0.40; Gnal+/-, n=25, 50 ms: 1.28 ± 0.03; 100 ms: 1.17 ± 0.03; 150 ms: 1.09 ± 0.02; two-way ANOVA and Bonferroni posttest: ISI p&lt;0.0001, genotype p=0.2405).
DOI: 10.7284/134767
title: CTD (Conductivity, Temperature, Depth) data collected during research cruise AR31-C using a Sea-Bird SBE-911+ instrument system onboard the platform RV Neil Armstrong

description: Original (unprocessed) data collected during a Field_expedition
DOI: 10.6084/m9.figshare.14135150
title: Supplementary Material for: Long Noncoding RNA PRNCR1 Reduces Renal Epithelial Cell Apoptosis in Cisplatin-Induced AKI by Regulating miR-182-5p/EZH1

description: <b><i>Background/Aims:</i></b> This study was designed to examine the role of long noncoding RNA PRNCR1 in cisplatin-induced acute kidney injury (AKI) in vitro and in vivo. <b><i>Methods:</i></b> The expression levels of PRNCR1 and miR-182-5p in cisplatin-induced AKI mice were examined. HK-2 cells were treated with cisplatin to induce cell damage. Then, the effects of PRNCR1 and miR-182-5p on cisplatin-stimulated HK-2 cell viability and apoptosis were detected by the CCK-8 and annexin V-FITC/PI method. Target genes of PRNCR1 and miR-182-5p were analyzed by bioinformatics analysis and luciferase. <b><i>Results:</i></b> The expression level of PRNCR1 was significantly reduced in cisplatin-induced AKI mice. In addition, overexpression of PRNCR1 attenuated the damage of cisplatin to HK-2. The expression level of miR-182-5p was significantly raised in cisplatin-induced AKI mice. MiR-182-5p was negatively regulated by PRNCR1 and leaded to an upregulation of EZH1 expression. Overexpression of PRNCR1 attenuated cisplatin-induced apoptosis by downregulating the miR-182-5p/EZH1 axis. <b><i>Conclusion:</i></b> LncPRNCR1 reduced the apoptosis of renal epithelial cells induced by cisplatin by modulating miR-182-5p/EZH1.
DOI: 10.6084/m9.figshare.14135150.v1
title: Supplementary Material for: Long Noncoding RNA PRNCR1 Reduces Renal Epithelial Cell Apoptosis in Cisplatin-Induced AKI by Regulating miR-182-5p/EZH1

description: <b><i>Background/Aims:</i></b> This study was designed to examine the role of long noncoding RNA PRNCR1 in cisplatin-induced acute kidney injury (AKI) in vitro and in vivo. <b><i>Methods:</i></b> The expression levels of PRNCR1 and miR-182-5p in cisplatin-induced AKI mice were examined. HK-2 cells were treated with cisplatin to induce cell damage. Then, the effects of PRNCR1 and miR-182-5p on cisplatin-stimulated HK-2 cell viability and apoptosis were detected by the CCK-8 and annexin V-FITC/PI method. Target genes of PRNCR1 and miR-182-5p were analyzed by bioinformatics analysis and luciferase. <b><i>Results:</i></b> The expression level of PRNCR1 was significantly reduced in cisplatin-induced AKI mice. In addition, overexpression of PRNCR1 attenuated the damage of cisplatin to HK-2. The expression level of miR-182-5p was significantly raised in cisplatin-induced AKI mice. MiR-182-5p was negatively regulated by PRNCR1 and leaded to an upregulation of EZH1 expression. Overexpression of PRNCR1 attenuated cisplatin-induced apoptosis by downregulating the miR-182-5p/EZH1 axis. <b><i>Conclusion:</i></b> LncPRNCR1 reduced the apoptosis of renal epithelial cells induced by cisplatin by modulating miR-182-5p/EZH1.
DOI: 10.15468/388gzv
title: Two new genera and two new species of troglobitic harvestmen of Stygnopsidae (Opiliones, Laniatores, Gonyleptoidea) from Oaxaca, Mexico, with notes on selected morphological characters

description: This dataset contains the digitized treatments in Plazi based on the original journal article Cruz-López, Jesús A. (2021): Two new genera and two new species of troglobitic harvestmen of Stygnopsidae (Opiliones, Laniatores, Gonyleptoidea) from Oaxaca, Mexico, with notes on selected morphological characters. Zoosystema 43 (5): 101-112, DOI: 10.5252/zoosystema2021v43a5
DOI: 10.25505/fiz.icsd.cc2285sb
title: CSD 1914491: Experimental Crystal Structure Determination

description: Related Article: Meriem Goudjil, Enrique Gutiérrez-Puebla, Paola Bonazzi, Eugenio Lunedei, Djillali Mezaoui, Luca Bindi|2019|Z.Krist.Cryst.Mater.|234|569|doi:10.1515/zkri-2019-0018
DOI: 10.25505/fiz.icsd.cc2284zh
title: CSD 1914466: Experimental Crystal Structure Determination

description: Related Article: Meriem Goudjil, Enrique Gutiérrez-Puebla, Paola Bonazzi, Eugenio Lunedei, Djillali Mezaoui, Luca Bindi|2019|Z.Krist.Cryst.Mater.|234|569|doi:10.1515/zkri-2019-0018
DOI: 10.25505/fiz.icsd.cc2286vf
title: CSD 1914524: Experimental Crystal Structure Determination

description: Related Article: Meriem Goudjil, Enrique Gutiérrez-Puebla, Paola Bonazzi, Eugenio Lunedei, Djillali Mezaoui, Luca Bindi|2019|Z.Krist.Cryst.Mater.|234|569|doi:10.1515/zkri-2019-0018
DOI: 10.25505/fiz.icsd.cc2286zk
title: CSD 1914528: Experimental Crystal Structure Determination

description: Related Article: Meriem Goudjil, Enrique Gutiérrez-Puebla, Paola Bonazzi, Eugenio Lunedei, Djillali Mezaoui, Luca Bindi|2019|Z.Krist.Cryst.Mater.|234|569|doi:10.1515/zkri-2019-0018
DOI: 10.25505/fiz.icsd.cc22878w
title: CSD 1914537: Experimental Crystal Structure Determination

description: Related Article: Meriem Goudjil, Enrique Gutiérrez-Puebla, Paola Bonazzi, Eugenio Lunedei, Djillali Mezaoui, Luca Bindi|2019|Z.Krist.Cryst.Mater.|234|569|doi:10.1515/zkri-2019-0018
DOI: 10.25505/fiz.icsd.cc23bmwz
title: CSD 1946641: Experimental Crystal Structure Determination

description: Related Article: Hamdi Ben Yahia, Masahiro Shikano|2019|Z.Naturforsch.,B:Chem.Sci.|74|677|doi:10.1515/znb-2019-0116
DOI: 10.5517/ccdc.csd.cc23nbdj
title: CCDC 1955957: Experimental Crystal Structure Determination

description: Related Article: N. Harsha Attanayake, Zhiming Liang, Yilin Wang, Aman Preet Kaur, Sean R. Parkin, Justin K. Mobley, Randy H. Ewoldt, James Landon, Susan A. Odom|2021|Mat.Advs.|2|1390|doi:10.1039/D0MA00881H
DOI: 10.5517/ccdc.csd.cc23nbfk
title: CCDC 1955958: Experimental Crystal Structure Determination

description: Related Article: N. Harsha Attanayake, Zhiming Liang, Yilin Wang, Aman Preet Kaur, Sean R. Parkin, Justin K. Mobley, Randy H. Ewoldt, James Landon, Susan A. Odom|2021|Mat.Advs.|2|1390|doi:10.1039/D0MA00881H
DOI: 10.25505/fiz.icsd.cc24bz0h
title: CSD 1976746: Experimental Crystal Structure Determination

description: Related Article: Dong Woo Lee, Kang Min Ok|2014|Inorg.Chem.|53|10642|doi:10.1021/ic501739u
DOI: 10.25505/fiz.icsd.cc24bz1j
title: CSD 1976747: Experimental Crystal Structure Determination

description: Related Article: Dong Woo Lee, Kang Min Ok|2014|Inorg.Chem.|53|10642|doi:10.1021/ic501739u
DOI: 10.5517/ccdc.csd.cc2542dt
title: CCDC 1999915: Experimental Crystal Structure Determination

description: Related Article: Li-Zhen Cai, Pei-Yu Guo, Ming-Sheng Wang, Guo-Cong Guo|2021|J.Mater.Chem.C|9|2231|doi:10.1039/D0TC05490A
DOI: 10.5517/ccdc.csd.cc2580s8
title: CCDC 2003709: Experimental Crystal Structure Determination

description: Related Article: Adrian-Alexandru Someşan, Cristian Silvestru, Richard A. Varga|2021|New J.Chem.|45|3817|doi:10.1039/D0NJ06016J
DOI: 10.5517/ccdc.csd.cc2580t9
title: CCDC 2003710: Experimental Crystal Structure Determination

description: Related Article: Adrian-Alexandru Someşan, Cristian Silvestru, Richard A. Varga|2021|New J.Chem.|45|3817|doi:10.1039/D0NJ06016J
DOI: 10.5517/ccdc.csd.cc25vnf4
title: CCDC 2021616: Experimental Crystal Structure Determination

description: Related Article: Jibiao Jin, Wuji Wang, Peiran Xue, Qingqing Yang, He Jiang, Ye Tao, Chao Zheng, Guohua Xie, Wei Huang, Runfeng Chen|2021|J.Mater.Chem.C|9|2291|doi:10.1039/D0TC05624C
DOI: 10.5517/ccdc.csd.cc25w5dn
title: CCDC 2022111: Experimental Crystal Structure Determination

description: Related Article: Yahui Zhang, Xiangyu Zhao, Ce Bi, Wenqi Lu, Mengyuan Song, Dongdong Wang, Guangyan Qing|2021|Green Chemistry|23|1691|doi:10.1039/D0GC03890C
DOI: 10.5517/ccdc.csd.cc25vnb1
title: CCDC 2021613: Experimental Crystal Structure Determination

description: Related Article: Qian-Feng Qiu, Cheng-Xia Chen, Zheng Zeng, Zhang-Wen Wei, Neng-Xiu Zhu, Chen-Chen Cao, Wei Wang, Dawei Wang, Hai-Ping Wang, Ji-Jun Jiang|2020|Inorg.Chem.|59|14856|doi:10.1021/acs.inorgchem.0c02123
description: Related Article: Qian-feng Qiu, Cheng-Xia Chen, Zhang-Wen Wei, Chen-Chen Cao, Neng-Xiu Zhu, Hai-Ping Wang, Dawei Wang, Ji-Jun Jiang, Cheng-Yong Su|2019|Inorg.Chem.|58|61|doi:10.1021/acs.inorgchem.8b02993
DOI: 10.5517/ccdc.csd.cc269gcc
title: CCDC 2034882: Experimental Crystal Structure Determination

description: Related Article: Alexander S. Phearman, Jewelianna M. Moore, Dayanni D. Bhagwandin, Jonathan M. Goldberg, D. Michael Heinekey, Karen I. Goldberg|2021|Green Chemistry|23|1609|doi:10.1039/D0GC03809A
DOI: 10.5517/ccdc.csd.cc1lxdmj
title: CCDC 1487058: Experimental Crystal Structure Determination

description: Related Article: Letícia B. Silva, Felipe S. Stefanello, Sarah C. Feitosa, Clarissa P. Frizzo, Marcos A. P. Martins, Nilo Zanatta, Bernardo A. Iglesias, Helio G. Bonacorso|2021|New J.Chem.|45|4061|doi:10.1039/D0NJ05740A
DOI: 10.5517/ccdc.csd.cc26g5c7
title: CCDC 2039408: Experimental Crystal Structure Determination

description: Related Article: Daniel Tchoń, David Bowskill, Isaac Sugden, Piotr Piotrowski, Anna Makal|2021|J.Mater.Chem.C|9|2491|doi:10.1039/D0TC05522K
DOI: 10.5517/ccdc.csd.cc26g5d8
title: CCDC 2039409: Experimental Crystal Structure Determination

description: Related Article: Daniel Tchoń, David Bowskill, Isaac Sugden, Piotr Piotrowski, Anna Makal|2021|J.Mater.Chem.C|9|2491|doi:10.1039/D0TC05522K
DOI: 10.5517/ccdc.csd.cc26g5f9
title: CCDC 2039410: Experimental Crystal Structure Determination

description: Related Article: Daniel Tchoń, David Bowskill, Isaac Sugden, Piotr Piotrowski, Anna Makal|2021|J.Mater.Chem.C|9|2491|doi:10.1039/D0TC05522K