cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.3394639.v10
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v11
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v12
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v13
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v14
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v15
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v16
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v17
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v18
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times.
DOI: 10.6084/m9.figshare.3394639.v19
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times. In fact, expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v20
title: How an ABO-incomcatible graft may survive by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may show extremely long survival times. In fact, expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v21
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v22
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v23
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v24
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v25
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v26
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v27
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v28
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v29
title: How an ABO-incompatible graft may improve its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v30
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v31
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v32
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may, taking into account the effect of all therapeutic measures, show extreme long survival times. <br> <br>
DOI: 10.6084/m9.figshare.3394639.v33
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v34
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v35
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v36
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v37
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v38
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v39
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v40
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v41
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v42
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>
DOI: 10.6084/m9.figshare.3394639.v43
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO-incompatible red cells, which from a HLA-compatible donor are transfused to a blood group O(H) recipient and undergo immediate destruction, a nucleated, metabolic active tissue that from the same donor is transplanted to the same recipient may,<i> </i>taking into account the effect of all therapeutic measures, occasionally show extreme long survival times. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO-incompatible, metabolically active tissue potentially uses its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient. <br>