cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.9178550.v1
title: Additional file 4: of Identification of adjuvants for clinical trials performed with Plasmodium falciparum AMA1 in rabbits

description: Comparison between treatment groups for day 70/140 titre ratios for 4 AMA1 alleles. (XLSX 9 kb)
DOI: 10.6084/m9.figshare.9178541
title: Additional file 3: of Identification of adjuvants for clinical trials performed with Plasmodium falciparum AMA1 in rabbits

description: Ratio of total IgG in day 140/day 70 sera to the homologous FVO AMA1 antigen and 3 heterologous variants (N = 4) in rabbits. (XLSX 13 kb)
DOI: 10.6084/m9.figshare.9178541.v1
title: Additional file 3: of Identification of adjuvants for clinical trials performed with Plasmodium falciparum AMA1 in rabbits

description: Ratio of total IgG in day 140/day 70 sera to the homologous FVO AMA1 antigen and 3 heterologous variants (N = 4) in rabbits. (XLSX 13 kb)
DOI: 10.6084/m9.figshare.9178532
title: Additional file 2: of Identification of adjuvants for clinical trials performed with Plasmodium falciparum AMA1 in rabbits

description: Comparison between treatment groups for total IgG in day 140 sera for 4 AMA1 alleles in rabbits. Between group differences are expressed as ratios with 95% confidence intervals. P values are adjusted for multiple comparisons using Tukeyâ s honest significant difference test (Tukeyâ s HSD test). (XLSX 12 kb)
DOI: 10.6084/m9.figshare.9178520
title: Additional file 1: of Identification of adjuvants for clinical trials performed with Plasmodium falciparum AMA1 in rabbits

description: Comparison between treatment groups for total IgG in day 70 sera for 4 AMA1 alleles in rabbits. Between group differences are expressed as ratios with 95% confidence intervals. P values are adjusted for multiple comparisons using Tukeyâ s honest significant difference test (Tukeyâ s HSD test). (XLSX 15 kb)
DOI: 10.6084/m9.figshare.9178532.v1
title: Additional file 2: of Identification of adjuvants for clinical trials performed with Plasmodium falciparum AMA1 in rabbits

description: Comparison between treatment groups for total IgG in day 140 sera for 4 AMA1 alleles in rabbits. Between group differences are expressed as ratios with 95% confidence intervals. P values are adjusted for multiple comparisons using Tukeyâ s honest significant difference test (Tukeyâ s HSD test). (XLSX 12 kb)
DOI: 10.6084/m9.figshare.9178520.v1
title: Additional file 1: of Identification of adjuvants for clinical trials performed with Plasmodium falciparum AMA1 in rabbits

description: Comparison between treatment groups for total IgG in day 70 sera for 4 AMA1 alleles in rabbits. Between group differences are expressed as ratios with 95% confidence intervals. P values are adjusted for multiple comparisons using Tukeyâ s honest significant difference test (Tukeyâ s HSD test). (XLSX 15 kb)
DOI: 10.6084/m9.figshare.9178481
title: Additional file 1: of Knowledge, attitudes and practices on tuberculosis transmission and prevention among auxiliary healthcare professionals in three Brazilian high-burden cities: a cross-sectional survey

description: Raw de-identified dataset analysed in this study, in MicrosoftÂŽ format. (XLSX 152 kb)
DOI: 10.6084/m9.figshare.9178481.v1
title: Additional file 1: of Knowledge, attitudes and practices on tuberculosis transmission and prevention among auxiliary healthcare professionals in three Brazilian high-burden cities: a cross-sectional survey

description: Raw de-identified dataset analysed in this study, in MicrosoftÂŽ format. (XLSX 152 kb)
DOI: 10.6084/m9.figshare.9178460
title: Additional file 1: of Spatial chromatin architecture alteration by structural variations in human genomes at the population scale

description: Supplemental tables. (XLSX 12146 kb)
DOI: 10.6084/m9.figshare.9178460.v1
title: Additional file 1: of Spatial chromatin architecture alteration by structural variations in human genomes at the population scale

description: Supplemental tables. (XLSX 12146 kb)
DOI: 10.6084/m9.figshare.9178451
title: Additional file 1: of Correlates of exclusive breastfeeding practices in rural and urban Niger: a community-based cross-sectional study

description: Questionnaire on correlates of exclusive breastfeeding practices in Niger. (XLSX 20 kb)
DOI: 10.6084/m9.figshare.9178451.v1
title: Additional file 1: of Correlates of exclusive breastfeeding practices in rural and urban Niger: a community-based cross-sectional study

description: Questionnaire on correlates of exclusive breastfeeding practices in Niger. (XLSX 20 kb)
DOI: 10.6084/m9.figshare.9178388
title: Additional file 6: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: KEGG pathways significantly upregulated in the 8 W pyramidotomy group compared to the 8 W sham group. KEGG pathways significantly (adjusted P
DOI: 10.6084/m9.figshare.9178391
title: Additional file 7: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: List of primers used for qRT-PCR. Forward and reverse primers used for qRT-PCR are listed. (XLSX 10 kb)
DOI: 10.6084/m9.figshare.9178385
title: Additional file 5: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: KEGG pathways significantly upregulated in the P7 pyramidotomy group compared to the P7 sham group. KEGG pathways significantly (adjusted Pâ
DOI: 10.6084/m9.figshare.9178388.v1
title: Additional file 6: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: KEGG pathways significantly upregulated in the 8 W pyramidotomy group compared to the 8 W sham group. KEGG pathways significantly (adjusted P
DOI: 10.6084/m9.figshare.9178391.v1
title: Additional file 7: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: List of primers used for qRT-PCR. Forward and reverse primers used for qRT-PCR are listed. (XLSX 10 kb)
DOI: 10.6084/m9.figshare.9178385.v1
title: Additional file 5: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: KEGG pathways significantly upregulated in the P7 pyramidotomy group compared to the P7 sham group. KEGG pathways significantly (adjusted Pâ
DOI: 10.6084/m9.figshare.9178373
title: Additional file 4: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: KEGG pathways significantly upregulated in the P7 sham group compared to the 8 W sham group. KEGG pathways significantly (adjusted P
DOI: 10.6084/m9.figshare.9178373.v1
title: Additional file 4: of Comparison of gene expression profile of the spinal cord of sprouting-capable neonatal and sprouting-incapable adult mice

description: KEGG pathways significantly upregulated in the P7 sham group compared to the 8 W sham group. KEGG pathways significantly (adjusted P
DOI: 10.6084/m9.figshare.9178343
title: Additional file 3: of Mechanistic interpretation of non-coding variants for discovering transcriptional regulators of drug response

description: Transcription factor binding motifs. We obtained 239 PWMs from three different data sources: (1) factor book motifs processed by ENCODE UCSC; (2) motifs downloaded from Factorbook; (3) HOCOMOCO Human (v10) motifs. STAP models are trained separately for each motif. We excluded a motif if the maximum prediction of the corresponding STAP model is too small (
DOI: 10.6084/m9.figshare.9178343.v1
title: Additional file 3: of Mechanistic interpretation of non-coding variants for discovering transcriptional regulators of drug response

description: Transcription factor binding motifs. We obtained 239 PWMs from three different data sources: (1) factor book motifs processed by ENCODE UCSC; (2) motifs downloaded from Factorbook; (3) HOCOMOCO Human (v10) motifs. STAP models are trained separately for each motif. We excluded a motif if the maximum prediction of the corresponding STAP model is too small (
DOI: 10.6084/m9.figshare.9178334
title: Additional file 2: of Mechanistic interpretation of non-coding variants for discovering transcriptional regulators of drug response

description: The file includes seven supplementary tables. All supplementary tables are included as sheets in this Excel file. Legends for these tables are provided here. (1) Table S1. The number of motifs for each TF. (2) Table S2. Accuracy values (correlation coefficient) of binding strength predictions for each TF. (3) Table S3. The AUROCs and AUPRCs of TFs in the ASB enrichment using four different TF binding predictors. (4) Table S4. TFs discovered regulating drug response using Delta-MOP alone to predict binding-change SNPs. (5) Table S5. TFs discovered regulating drug response using Delta-gkm-SVM alone to predict binding-change SNPs. (6) Table S6. The results of drug associated enrichment tests for all 888 TF-drug pairs. (7) Table S7. Mediator genes in ELF1-doxorubicin Analysis. SNPs in the intersection of TF-drug hypergeometric tests are: (1) eQTLs associated to drug response genes; (2) â binding-change SNPsâ of the TF. DRGs associated with those SNPs are potentially mediators regulated by the TF. Here we list all mediator genes in ELF1-Docorubicin analysis. (XLS 149 kb)
DOI: 10.6084/m9.figshare.9178328
title: Additional file 9 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S19. List of mouse genes displaying significant association with refractive error in CREAM cohort and whose expression correlates with baseline refractive error in mice (Blue identifies PBonferroni
DOI: 10.6084/m9.figshare.9178334.v1
title: Additional file 2: of Mechanistic interpretation of non-coding variants for discovering transcriptional regulators of drug response

description: The file includes seven supplementary tables. All supplementary tables are included as sheets in this Excel file. Legends for these tables are provided here. (1) Table S1. The number of motifs for each TF. (2) Table S2. Accuracy values (correlation coefficient) of binding strength predictions for each TF. (3) Table S3. The AUROCs and AUPRCs of TFs in the ASB enrichment using four different TF binding predictors. (4) Table S4. TFs discovered regulating drug response using Delta-MOP alone to predict binding-change SNPs. (5) Table S5. TFs discovered regulating drug response using Delta-gkm-SVM alone to predict binding-change SNPs. (6) Table S6. The results of drug associated enrichment tests for all 888 TF-drug pairs. (7) Table S7. Mediator genes in ELF1-doxorubicin Analysis. SNPs in the intersection of TF-drug hypergeometric tests are: (1) eQTLs associated to drug response genes; (2) â binding-change SNPsâ of the TF. DRGs associated with those SNPs are potentially mediators regulated by the TF. Here we list all mediator genes in ELF1-Docorubicin analysis. (XLS 149 kb)
DOI: 10.6084/m9.figshare.12867254.v1
title: R0191_20170922a_videos

description: Rat 0191, skilled reaching data collected on 9/22/2017 in Leventhal laboratory by Alexandra Bova
DOI: 10.6084/m9.figshare.9178325
title: Additional file 8 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S17. Gene ontology categories significantly associated with genes localized within human myopia QTLs and whose expression correlates with baseline refractive error in mice (BP, biological process; CC, cellular component; MF, molecular function). Table S18. Gene ontology categories significantly associated with genes localized within human myopia QTLs and whose expression correlates with susceptibility to myopia in mice (BP, biological process; CC, cellular component; MF, molecular function). (XLSX 15 kb)
DOI: 10.6084/m9.figshare.9178319
title: Additional file 7 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S16. List of human myopia QTLs showing overlap with genes exhibiting correlation with either refractive error or susceptibility to myopia in mice. (XLSX 16 kb)
DOI: 10.6084/m9.figshare.9178325.v1
title: Additional file 8 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S17. Gene ontology categories significantly associated with genes localized within human myopia QTLs and whose expression correlates with baseline refractive error in mice (BP, biological process; CC, cellular component; MF, molecular function). Table S18. Gene ontology categories significantly associated with genes localized within human myopia QTLs and whose expression correlates with susceptibility to myopia in mice (BP, biological process; CC, cellular component; MF, molecular function). (XLSX 15 kb)
DOI: 10.6084/m9.figshare.9178328.v1
title: Additional file 9 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S19. List of mouse genes displaying significant association with refractive error in CREAM cohort and whose expression correlates with baseline refractive error in mice (Blue identifies PBonferroni
DOI: 10.6084/m9.figshare.9178319.v1
title: Additional file 7 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S16. List of human myopia QTLs showing overlap with genes exhibiting correlation with either refractive error or susceptibility to myopia in mice. (XLSX 16 kb)
DOI: 10.6084/m9.figshare.9178310
title: Additional file 5 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S12. List of human myopia QTLs and candidate genes located within 200â kb of the lead SNP (or within critical region). (XLSX 28 kb)
DOI: 10.6084/m9.figshare.9178316
title: Additional file 6 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S13. List of genes localized within human myopia QTLs and whose expression correlates with baseline refractive error in mice. Table S14. List of genes localized within human myopia QTLs and whose expression correlates with susceptibility to myopia in mice. Table S15. List of genes localized within human myopia QTLs and whose expression correlates with both baseline refractive error and susceptibility to myopia in mice. (XLSX 28 kb)
DOI: 10.6084/m9.figshare.9178307
title: Additional file 4 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S9. List of genes whose expression correlates with both baseline refractive error and susceptibility to myopia in Collaborative Cross mice. Table S10. Gene ontology categories significantly associated with genes whose expression correlates with both baseline refractive error and susceptibility to myopia in Collaborative Cross mice (BP, biological process; CC, cellular component; MF, molecular function). Table S11. Canonical signaling pathways affected by genes whose expression correlates with both baseline refractive error and susceptibility to myopia in Collaborative Cross mice. (XLSX 110 kb)
DOI: 10.6084/m9.figshare.9178310.v1
title: Additional file 5 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S12. List of human myopia QTLs and candidate genes located within 200â kb of the lead SNP (or within critical region). (XLSX 28 kb)
DOI: 10.6084/m9.figshare.9178316.v1
title: Additional file 6 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S13. List of genes localized within human myopia QTLs and whose expression correlates with baseline refractive error in mice. Table S14. List of genes localized within human myopia QTLs and whose expression correlates with susceptibility to myopia in mice. Table S15. List of genes localized within human myopia QTLs and whose expression correlates with both baseline refractive error and susceptibility to myopia in mice. (XLSX 28 kb)
DOI: 10.6084/m9.figshare.9178307.v1
title: Additional file 4 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S9. List of genes whose expression correlates with both baseline refractive error and susceptibility to myopia in Collaborative Cross mice. Table S10. Gene ontology categories significantly associated with genes whose expression correlates with both baseline refractive error and susceptibility to myopia in Collaborative Cross mice (BP, biological process; CC, cellular component; MF, molecular function). Table S11. Canonical signaling pathways affected by genes whose expression correlates with both baseline refractive error and susceptibility to myopia in Collaborative Cross mice. (XLSX 110 kb)
DOI: 10.6084/m9.figshare.9178298
title: Additional file 3 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S6. List of genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice. Table S7. Gene ontology categories significantly associated with genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice (BP, biological process; CC, cellular component; MF, molecular function). Table S8. Canonical signaling pathways affected by genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice. (XLSX 158 kb)
DOI: 10.6084/m9.figshare.9178292
title: Additional file 2 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S3. List of genes whose expression correlates with baseline refractive error in Collaborative Cross mice. Table S4. Gene ontology categories significantly associated with genes whose expression correlates with baseline refractive error in Collaborative Cross mice (BP, biological process; CC, cellular component; MF, molecular function). Table S5. Canonical signaling pathways affected by genes whose expression correlates with baseline refractive error in Collaborative Cross mice. (XLSX 192 kb)
DOI: 10.6084/m9.figshare.9178298.v1
title: Additional file 3 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S6. List of genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice. Table S7. Gene ontology categories significantly associated with genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice (BP, biological process; CC, cellular component; MF, molecular function). Table S8. Canonical signaling pathways affected by genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice. (XLSX 158 kb)
DOI: 10.6084/m9.figshare.9178286
title: Additional file 1 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S1. Baseline refractive errors in Collaborative Cross mice measured at P40 (diopters). Table S2. Form-deprivation myopia in Collaborative Cross mice (deprived eye versus control eye, diopters). (XLSX 14 kb)
DOI: 10.6084/m9.figshare.9178292.v1
title: Additional file 2 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S3. List of genes whose expression correlates with baseline refractive error in Collaborative Cross mice. Table S4. Gene ontology categories significantly associated with genes whose expression correlates with baseline refractive error in Collaborative Cross mice (BP, biological process; CC, cellular component; MF, molecular function). Table S5. Canonical signaling pathways affected by genes whose expression correlates with baseline refractive error in Collaborative Cross mice. (XLSX 192 kb)
DOI: 10.6084/m9.figshare.9178286.v1
title: Additional file 1 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S1. Baseline refractive errors in Collaborative Cross mice measured at P40 (diopters). Table S2. Form-deprivation myopia in Collaborative Cross mice (deprived eye versus control eye, diopters). (XLSX 14 kb)
DOI: 10.6084/m9.figshare.9178271
title: Additional file 11 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S29. List of genes whose expression correlates with baseline refractive error in Collaborative Cross mice (molecular function). Table S30. List of genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice (molecular function). (XLSX 163 kb)
DOI: 10.6084/m9.figshare.9178271.v1
title: Additional file 11 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S29. List of genes whose expression correlates with baseline refractive error in Collaborative Cross mice (molecular function). Table S30. List of genes whose expression correlates with susceptibility to myopia in Collaborative Cross mice (molecular function). (XLSX 163 kb)
DOI: 10.6084/m9.figshare.9178265
title: Additional file 10 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S23. List of mouse genes displaying significant association with refractive error in both UK Biobank and CREAM cohorts, and whose expression correlates with baseline refractive error in mice (Blue identifies PBonferroni
DOI: 10.6084/m9.figshare.9178265.v1
title: Additional file 10 of Analysis of genetic networks regulating refractive eye development in collaborative cross progenitor strain mice reveals new genes and pathways underlying human myopia

description: Table S23. List of mouse genes displaying significant association with refractive error in both UK Biobank and CREAM cohorts, and whose expression correlates with baseline refractive error in mice (Blue identifies PBonferroni
DOI: 10.6084/m9.figshare.9178238
title: MOESM4 of Molecular characterization and functional analysis of the Schistosoma mekongi Ca2+-dependent cysteine protease (calpain)

description: Additional file 4: Table S3. Identification of rSmeCalp1 using liquid chromatography-tandem mass spectrometry (LC-MS/MS).