cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.1455478.v4
title: Seattle Street Clock Database

description: This spreadsheet contains three tables: Clocks, Owners, and Evidence - Clocks tab's name column and Owners tab's corporation column keys in Evidence. - Evidence is a list of every unique fact I have via newspaper, photograph, document, or otherwise about when a clock was in a particular location, and who owned it at that time. v1 has 1033 pieces of evidence. Please contact me if you have additions or find errors in my data. I will be refreshing this periodically. I constantly find new resources. Along with my friend Paul Middents we have proven the connection between many owners, and I know that there will be more discoveries to show which clocks were later owned by another jeweler.<br><br>A few notes on fields: <br>- In Evidence, "Var_Year" indicates a +/- value on the Source_Year. Empty means exact. 0 means it was the first year that the clock was at that location. - If the Source_Type is Photograph, Source_ID has a value, and there is no URL, then I have the photo on my harddrive but can't post it. - In Clocks, if the Unique field is Yes, then I'm sure that this clock cannot be matched with any other clocks in the list. There are 114 clocks in the list which is excessive. It should be down closer to 60. - There are a few large sign clocks in the database. They are all in downtown. The O. B. McClintocks definitely belong in the database, and the few other signs are described that way in the Notes field of both Evidence and Clocks. On the linked page I will maintain a list of open questions, and areas I know I need to work on.
DOI: 10.6084/m9.figshare.1454535
title: Replication data for No Place to Call Home NEJM 2015

description: NHIS 2011 data via IHIS system to replicate graph in Friedman, Saloner, Hsia. "No Place to Call Home" NEJM 2015. Includes packrat and other changes to facilitate replication.
DOI: 10.6084/m9.figshare.1455990.v9
title: A Functional Approach to Violent Radicalization. Building a Systemic Model Based on a Real Case

description: A paradox in the study of violent radicalization is that while each of the empirical findings can be explained from multiple theories, very few theories can explain a relevant number of these findings simultaneously. In this paper functional behavior assessment is applied to violent radical behavior, trying to find out the factors responsible for its initial learning and subsequent maintenance. Specifically, a radicalization model is proposed, which can explain a wide range of observed phenomena, accommodate apparent exceptions, and obtain testable consequences. It also allows challenging some firmly rooted ideas, as the alleged existence of aggressive influence practices, or brainwashing. Finally, the model is also able to provide valuable predictions for subsequent investigation, such as those related to the reversibility of the process of radicalization.
DOI: 10.6084/m9.figshare.1454522
title: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish

description: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish
DOI: 10.6084/m9.figshare.1454535.v1
title: Replication data for No Place to Call Home NEJM 2015

description: NHIS 2011 data via IHIS system to replicate graph in Friedman, Saloner, Hsia. "No Place to Call Home" NEJM 2015. Includes packrat and other changes to facilitate replication.
DOI: 10.6084/m9.figshare.1455478.v5
title: Seattle Street Clock Database

description: This spreadsheet contains three tables: Clocks, Owners, and Evidence - Clocks tab's name column and Owners tab's corporation column keys in Evidence. - Evidence is a list of every unique fact I have via newspaper, photograph, document, or otherwise about when a clock was in a particular location, and who owned it at that time. v1 has 1033 pieces of evidence. Please contact me if you have additions or find errors in my data. I will be refreshing this periodically. I constantly find new resources. Along with my friend Paul Middents we have proven the connection between many owners, and I know that there will be more discoveries to show which clocks were later owned by another jeweler.<br><br>A few notes on fields: <br>- In Evidence, "Var_Year" indicates a +/- value on the Source_Year. Empty means exact. 0 means it was the first year that the clock was at that location. - If the Source_Type is Photograph, Source_ID has a value, and there is no URL, then I have the photo on my harddrive but can't post it. - In Clocks, if the Unique field is Yes, then I'm sure that this clock cannot be matched with any other clocks in the list. There are 114 clocks in the list which is excessive. It should be down closer to 60. - There are a few large sign clocks in the database. They are all in downtown. The O. B. McClintocks definitely belong in the database, and the few other signs are described that way in the Notes field of both Evidence and Clocks. On the linked page I will maintain a list of open questions, and areas I know I need to work on.
DOI: 10.6084/m9.figshare.1454521
title: Nobel Prize winners in Medicine or Physiology

description: These data support an article submitted to PLoS ONE: Do Nobel Laureates Create Prize-Winning Networks? An Analysis of Collaborative Research in Physiology or Medicine C.S. Wagner et al. 2015
DOI: 10.6084/m9.figshare.1454522.v1
title: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish

description: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish
DOI: 10.6084/m9.figshare.1455478.v7
title: Seattle Street Clock Database

description: This spreadsheet contains three tables: Clocks, Owners, and Evidence - Clocks tab's name column and Owners tab's corporation column keys in Evidence. - Evidence is a list of every unique fact I have via newspaper, photograph, document, or otherwise about when a clock was in a particular location, and who owned it at that time. v1 has 1033 pieces of evidence. Please contact me if you have additions or find errors in my data. I will be refreshing this periodically. I constantly find new resources. Along with my friend Paul Middents we have proven the connection between many owners, and I know that there will be more discoveries to show which clocks were later owned by another jeweler.<br><br>A few notes on fields: <br>- In Evidence, "Var_Year" indicates a +/- value on the Source_Year. Empty means exact. 0 means it was the first year that the clock was at that location. - If the Source_Type is Photograph, Source_ID has a value, and there is no URL, then I have the photo on my harddrive but can't post it. - In Clocks, if the Unique field is Yes, then I'm sure that this clock cannot be matched with any other clocks in the list. There are 114 clocks in the list which is excessive. It should be down closer to 60. - There are a few large sign clocks in the database. They are all in downtown. The O. B. McClintocks definitely belong in the database, and the few other signs are described that way in the Notes field of both Evidence and Clocks. On the linked page I will maintain a list of open questions, and areas I know I need to work on.
DOI: 10.6084/m9.figshare.1454521.v1
title: Nobel Prize winners in Medicine or Physiology

description: These data support an article submitted to PLoS ONE: Do Nobel Laureates Create Prize-Winning Networks? An Analysis of Collaborative Research in Physiology or Medicine C.S. Wagner et al. 2015
DOI: 10.6084/m9.figshare.1454520.v1
title: Exosomes derived from human mesenchymal stem cells confer drug resistance in gastric cancer

description: Mesenchymal stem cells (MSCs) play an important role in chemoresistance. Exosomes have been reported to modify cellular phenotype and function by mediating cell-cell communication. In this study, we aimed to investigate whether exosomes derived from MSCs (MSC-exosomes) are involved in mediating the resistance to chemotherapy in gastric cancer and to explore the underlying molecular mechanism. We found that MSC-exosomes significantly induced the resistance of gastric cancer cells to 5-fluorouracil both in vivo and ex vivo. MSC-exosomes antagonized 5-fluorouracil-induced apoptosis and enhanced the expression of multi-drug resistance associated proteins, including MDR, MRP and LRP. Mechanistically, MSC-exosomes triggered the activation of calcium/calmodulin-dependent protein kinases (CaM-Ks) and Raf/MEK/ERK kinase cascade in gastric cancer cells. Blocking the CaM-Ks/Raf/MEK/ERK pathway inhibited the promoting role of MSC-exosomes in chemoresistance. Collectively, MSC-exosomes could induce drug resistance in gastric cancer cells by activating CaM-Ks/Raf/MEK/ERK pathway. Our findings suggest that MSC-exosomes have profound effects on modifying gastric cancer cells in the development of drug resistance. Targeting the interaction between MSC-exosomes and cancer cells may help improve the efficacy of chemotherapy in gastric cancer.
DOI: 10.6084/m9.figshare.1454522.v2
title: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish

description: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish
DOI: 10.6084/m9.figshare.1454521.v2
title: Nobel Prize winners in Medicine or Physiology

description: These data support an article submitted to PLoS ONE: Do Nobel Laureates Create Prize-Winning Networks? An Analysis of Collaborative Research in Physiology or Medicine C.S. Wagner et al. 2015
DOI: 10.6084/m9.figshare.1454522.v4
title: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish

description: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish
DOI: 10.6084/m9.figshare.1455478.v8
title: Seattle Street Clock Database

description: This spreadsheet contains three tables: Clocks, Owners, and Evidence - Clocks tab's name column and Owners tab's corporation column keys in Evidence. - Evidence is a list of every unique fact I have via newspaper, photograph, document, or otherwise about when a clock was in a particular location, and who owned it at that time. v1 has 1033 pieces of evidence. Please contact me if you have additions or find errors in my data. I will be refreshing this periodically. I constantly find new resources. Along with my friend Paul Middents we have proven the connection between many owners, and I know that there will be more discoveries to show which clocks were later owned by another jeweler.<br><br>A few notes on fields: <br>- In Evidence, "Var_Year" indicates a +/- value on the Source_Year. Empty means exact. 0 means it was the first year that the clock was at that location. - If the Source_Type is Photograph, Source_ID has a value, and there is no URL, then I have the photo on my harddrive but can't post it. - In Clocks, if the Unique field is Yes, then I'm sure that this clock cannot be matched with any other clocks in the list. There are 114 clocks in the list which is excessive. It should be down closer to 60. - There are a few large sign clocks in the database. They are all in downtown. The O. B. McClintocks definitely belong in the database, and the few other signs are described that way in the Notes field of both Evidence and Clocks. On the linked page I will maintain a list of open questions, and areas I know I need to work on.
DOI: 10.6084/m9.figshare.1454520.v2
title: Exosomes derived from human mesenchymal stem cells confer drug resistance in gastric cancer

description: Mesenchymal stem cells (MSCs) play an important role in chemoresistance. Exosomes have been reported to modify cellular phenotype and function by mediating cell-cell communication. In this study, we aimed to investigate whether exosomes derived from MSCs (MSC-exosomes) are involved in mediating the resistance to chemotherapy in gastric cancer and to explore the underlying molecular mechanism. We found that MSC-exosomes significantly induced the resistance of gastric cancer cells to 5-fluorouracil both in vivo and ex vivo. MSC-exosomes antagonized 5-fluorouracil-induced apoptosis and enhanced the expression of multi-drug resistance associated proteins, including MDR, MRP and LRP. Mechanistically, MSC-exosomes triggered the activation of calcium/calmodulin-dependent protein kinases (CaM-Ks) and Raf/MEK/ERK kinase cascade in gastric cancer cells. Blocking the CaM-Ks/Raf/MEK/ERK pathway inhibited the promoting role of MSC-exosomes in chemoresistance. Collectively, MSC-exosomes could induce drug resistance in gastric cancer cells by activating CaM-Ks/Raf/MEK/ERK pathway. Our findings suggest that MSC-exosomes have profound effects on modifying gastric cancer cells in the development of drug resistance. Targeting the interaction between MSC-exosomes and cancer cells may help improve the efficacy of chemotherapy in gastric cancer.
DOI: 10.6084/m9.figshare.1454522.v3
title: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish

description: Introduction to a Special Section: Hatcheries and Management of Aquatic Resources (HaMAR)—Considerations for Use of Hatcheries and Hatchery-Origin Fish
DOI: 10.6084/m9.figshare.1454519.v1
title: DNA Methylation Provides Insight into Intergenerational Risk for Preterm Birth in African Americans

description: African Americans are at increased risk for spontaneous preterm birth (PTB). Though PTB is heritable, genetic studies have not identified variants that account for its intergenerational risk, prompting the hypothesis that epigenetic factors may also contribute. The objective of this study was to evaluate DNA methylation from maternal leukocytes to identify patterns specific to PTB and its intergenerational risk. DNA from peripheral leukocytes from African American women that delivered preterm (24–34 weeks; N=16) or at term (39–41 weeks; N=24) was assessed for DNA methylation using the HumanMethylation450 BeadChip. In maternal samples, 17,829 CpG sites associated with PTB, but no CpG site remained associated after correction for multiple comparisons. Examination of paired maternal-fetal samples identified 5,171 CpG sites in which methylation of maternal samples correlated with methylation of her respective fetus (FDR&lt;0.05). These correlated sites were enriched for association with PTB in maternal leukocytes. The majority of correlated CpG sites could be attributed to one or more genetic variants. They were also significantly more likely to be in genes involved in metabolic, cardiovascular, and immune pathways, suggesting a role for genetic and environmental contributions to PTB risk and chronic disease. The results of this study may provide insight into the factors underlying intergenerational risk for PTB and its consequences.
DOI: 10.6084/m9.figshare.1454520.v3
title: Exosomes derived from human mesenchymal stem cells confer drug resistance in gastric cancer

description: Mesenchymal stem cells (MSCs) play an important role in chemoresistance. Exosomes have been reported to modify cellular phenotype and function by mediating cell-cell communication. In this study, we aimed to investigate whether exosomes derived from MSCs (MSC-exosomes) are involved in mediating the resistance to chemotherapy in gastric cancer and to explore the underlying molecular mechanism. We found that MSC-exosomes significantly induced the resistance of gastric cancer cells to 5-fluorouracil both in vivo and ex vivo. MSC-exosomes antagonized 5-fluorouracil-induced apoptosis and enhanced the expression of multi-drug resistance associated proteins, including MDR, MRP and LRP. Mechanistically, MSC-exosomes triggered the activation of calcium/calmodulin-dependent protein kinases (CaM-Ks) and Raf/MEK/ERK kinase cascade in gastric cancer cells. Blocking the CaM-Ks/Raf/MEK/ERK pathway inhibited the promoting role of MSC-exosomes in chemoresistance. Collectively, MSC-exosomes could induce drug resistance in gastric cancer cells by activating CaM-Ks/Raf/MEK/ERK pathway. Our findings suggest that MSC-exosomes have profound effects on modifying gastric cancer cells in the development of drug resistance. Targeting the interaction between MSC-exosomes and cancer cells may help improve the efficacy of chemotherapy in gastric cancer.
DOI: 10.6084/m9.figshare.1455478.v9
title: Seattle Street Clock Database

description: This spreadsheet contains three tables: Clocks, Owners, and Evidence - Clocks tab's name column and Owners tab's corporation column keys in Evidence. - Evidence is a list of every unique fact I have via newspaper, photograph, document, or otherwise about when a clock was in a particular location, and who owned it at that time. v1 has 1033 pieces of evidence. Please contact me if you have additions or find errors in my data. I will be refreshing this periodically. I constantly find new resources. Along with my friend Paul Middents we have proven the connection between many owners, and I know that there will be more discoveries to show which clocks were later owned by another jeweler.<br><br>A few notes on fields: <br>- In Evidence, "Var_Year" indicates a +/- value on the Source_Year. Empty means exact. 0 means it was the first year that the clock was at that location. - If the Source_Type is Photograph, Source_ID has a value, and there is no URL, then I have the photo on my harddrive but can't post it. - In Clocks, if the Unique field is Yes, then I'm sure that this clock cannot be matched with any other clocks in the list. There are 114 clocks in the list which is excessive. It should be down closer to 60. - There are a few large sign clocks in the database. They are all in downtown. The O. B. McClintocks definitely belong in the database, and the few other signs are described that way in the Notes field of both Evidence and Clocks. On the linked page I will maintain a list of open questions, and areas I know I need to work on.
DOI: 10.6084/m9.figshare.1455478.v10
title: Seattle Street Clock Database

description: This spreadsheet contains three tables: Clocks, Owners, and Evidence - Clocks tab's name column and Owners tab's corporation column keys in Evidence. - Evidence is a list of every unique fact I have via newspaper, photograph, document, or otherwise about when a clock was in a particular location, and who owned it at that time. v1 has 1033 pieces of evidence. Please contact me if you have additions or find errors in my data. I will be refreshing this periodically. I constantly find new resources. Along with my friend Paul Middents we have proven the connection between many owners, and I know that there will be more discoveries to show which clocks were later owned by another jeweler.<br><br>A few notes on fields: <br>- In Evidence, "Var_Year" indicates a +/- value on the Source_Year. Empty means exact. 0 means it was the first year that the clock was at that location. - If the Source_Type is Photograph, Source_ID has a value, and there is no URL, then I have the photo on my harddrive but can't post it. - In Clocks, if the Unique field is Yes, then I'm sure that this clock cannot be matched with any other clocks in the list. There are 114 clocks in the list which is excessive. It should be down closer to 60. - There are a few large sign clocks in the database. They are all in downtown. The O. B. McClintocks definitely belong in the database, and the few other signs are described that way in the Notes field of both Evidence and Clocks. On the linked page I will maintain a list of open questions, and areas I know I need to work on.
DOI: 10.6084/m9.figshare.1454520.v4
title: Exosomes derived from human mesenchymal stem cells confer drug resistance in gastric cancer

description: Mesenchymal stem cells (MSCs) play an important role in chemoresistance. Exosomes have been reported to modify cellular phenotype and function by mediating cell-cell communication. In this study, we aimed to investigate whether exosomes derived from MSCs (MSC-exosomes) are involved in mediating the resistance to chemotherapy in gastric cancer and to explore the underlying molecular mechanism. We found that MSC-exosomes significantly induced the resistance of gastric cancer cells to 5-fluorouracil both in vivo and ex vivo. MSC-exosomes antagonized 5-fluorouracil-induced apoptosis and enhanced the expression of multi-drug resistance associated proteins, including MDR, MRP and LRP. Mechanistically, MSC-exosomes triggered the activation of calcium/calmodulin-dependent protein kinases (CaM-Ks) and Raf/MEK/ERK kinase cascade in gastric cancer cells. Blocking the CaM-Ks/Raf/MEK/ERK pathway inhibited the promoting role of MSC-exosomes in chemoresistance. Collectively, MSC-exosomes could induce drug resistance in gastric cancer cells by activating CaM-Ks/Raf/MEK/ERK pathway. Our findings suggest that MSC-exosomes have profound effects on modifying gastric cancer cells in the development of drug resistance. Targeting the interaction between MSC-exosomes and cancer cells may help improve the efficacy of chemotherapy in gastric cancer.
DOI: 10.6084/m9.figshare.1454519.v2
title: DNA methylation provides insight into intergenerational risk for preterm birth in African Americans

description: African Americans are at increased risk for spontaneous preterm birth (PTB). Though PTB is heritable, genetic studies have not identified variants that account for its intergenerational risk, prompting the hypothesis that epigenetic factors may also contribute. The objective of this study was to evaluate DNA methylation from maternal leukocytes to identify patterns specific to PTB and its intergenerational risk. DNA from peripheral leukocytes from African American women that delivered preterm (24–34 weeks; N = 16) or at term (39–41 weeks; N = 24) was assessed for DNA methylation using the HumanMethylation450 BeadChip. In maternal samples, 17,829 CpG sites associated with PTB, but no CpG site remained associated after correction for multiple comparisons. Examination of paired maternal-fetal samples identified 5,171 CpG sites in which methylation of maternal samples correlated with methylation of her respective fetus (FDR &lt; 0.05). These correlated sites were enriched for association with PTB in maternal leukocytes. The majority of correlated CpG sites could be attributed to one or more genetic variants. They were also significantly more likely to be in genes involved in metabolic, cardiovascular, and immune pathways, suggesting a role for genetic and environmental contributions to PTB risk and chronic disease. The results of this study may provide insight into the factors underlying intergenerational risk for PTB and its consequences.