cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.15468/dl.g46d5z
title: Occurrence Download

description: A dataset containing 827 species occurrences available in GBIF matching the query:
{
 "and" : [
 "BasisOfRecord is one of (Observation, Machine Observation, Human Observation, Material sample, Material citation, Specimen, Living Specimen, Occurrence evidence)",
 "TaxonKey is one of (Peltogastridae, Peltogasterellidae)"
 ]
}

 The dataset includes 827 records from 67 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0002939-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.w2397s
title: Occurrence Download

description: A dataset containing 889 species occurrences available in GBIF matching the query:
{
 "and" : [
 "TaxonKey is one of (Mimosa dysocarpa, Mimosa dysocarpa)",
 "HasCoordinate is true",
 "HasGeospatialIssue is false"
 ]
}

 The dataset includes 889 records from 54 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0013064-251009101135966/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.3535/27d-wpb-7ys
title: †Fusulinidae Möller, 1878

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/ypa-kaz-rnl
title: †Fusulinidae Möller, 1878

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/3jq-ghl-dza
title: Equus (Equus) caballus Linnaeus, 1758

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/rzl-939-zxh
title: Ostracoda Latreille, 1802

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/sxw-m00-hky
title: †Lepidocyclina Gümbel, 1870

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/15k-ly3-8yk
title: Ostracoda Latreille, 1802

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/s7t-60f-mah
title: †Miliola subgen. Quinqueloculina Egger, 1857

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/ct4-mgp-jj7
title: Trilobatus trilobus (Reuss, 1850)

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/dy5-54q-0pe
title: Ostracoda Latreille, 1802

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/b5f-grp-yl7
title: †Thalmanninella reicheli (Mornod, 1950)

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/l6l-7e8-cvf
title: Ostracoda Latreille, 1802

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/hc3-zhw-5nj
title: Foraminifera d'Orbigny, 1826

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/byb-v5a-30l
title: Karreriella bradyi (Cushman, 1911)

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/zba-s1j-t9j
title: †Hedbergella Brönnimann & Brown, 1958

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/k6q-5x4-zy3
title: Foraminifera d'Orbigny, 1826

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/wse-f9x-dvd
title: †Daxia Cuvillier & Szakall, 1949

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/f2k-2pc-z9k
title: Animalia

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/nf0-apx-bzx
title: Foraminifera d'Orbigny, 1826

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/v75-kxn-6c5
title: †Nummulites globulus Leymerie, 1846

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/ytg-264-2aa
title: Canis lupus familiaris Linnaeus, 1758

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.6084/m9.figshare.29930405
title: The Society for Clinical Neuropsychology’s 2023 strategic plan: Group differences in strategic planning survey results

description: To examine differences between demographic and cultural identity group responses on the Society for Clinical Neuropsychology’s (SCN) Strategic Planning Survey. Respondents were grouped into self-identified demographic and cultural identity groups. Descriptive statistics were used to summarize responses to demographic and survey data. We used Chi-square and Fisher’s Exact tests to compare group differences. Adjusted residuals were calculated for post-hoc testing, when appropriate. Membership satisfaction was high across groups. Respondents who identified as persons of color, women, with a disability, or students/trainees were more likely to indicate that the SCN could better represent them by increasing diversity and inclusion efforts. Persons of color and other historically marginalized/minoritized groups viewed the SCN’s equity, diversity, and inclusion (EDI) efforts as less successful than comparative groups, but they expressed more optimism that initiatives could increase the engagement of persons of color in the SCN. Respondents earlier in their career and from marginalized/minoritized groups more frequently viewed the lack of diversity in clinical neuropsychology as an important issue facing the specialty and the desire for the SCN to prioritize improving EDI within clinical neuropsychology. Women and earlier career respondents were more likely to indicate that the SCN should focus efforts on increasing clinical neuropsychology’s value to society. Respondents earlier in their careers and from marginalized/minoritized groups were more likely to identify EDI related issues as significant to the SCN and specialty, and a desire for the SCN to prioritize efforts to improve EDI.
DOI: 10.6084/m9.figshare.29930405.v1
title: The Society for Clinical Neuropsychology’s 2023 strategic plan: Group differences in strategic planning survey results

description: To examine differences between demographic and cultural identity group responses on the Society for Clinical Neuropsychology’s (SCN) Strategic Planning Survey. Respondents were grouped into self-identified demographic and cultural identity groups. Descriptive statistics were used to summarize responses to demographic and survey data. We used Chi-square and Fisher’s Exact tests to compare group differences. Adjusted residuals were calculated for post-hoc testing, when appropriate. Membership satisfaction was high across groups. Respondents who identified as persons of color, women, with a disability, or students/trainees were more likely to indicate that the SCN could better represent them by increasing diversity and inclusion efforts. Persons of color and other historically marginalized/minoritized groups viewed the SCN’s equity, diversity, and inclusion (EDI) efforts as less successful than comparative groups, but they expressed more optimism that initiatives could increase the engagement of persons of color in the SCN. Respondents earlier in their career and from marginalized/minoritized groups more frequently viewed the lack of diversity in clinical neuropsychology as an important issue facing the specialty and the desire for the SCN to prioritize improving EDI within clinical neuropsychology. Women and earlier career respondents were more likely to indicate that the SCN should focus efforts on increasing clinical neuropsychology’s value to society. Respondents earlier in their careers and from marginalized/minoritized groups were more likely to identify EDI related issues as significant to the SCN and specialty, and a desire for the SCN to prioritize efforts to improve EDI.
DOI: 10.3535/522-p2l-rzj
title: Equus (Equus) caballus Linnaeus, 1758

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/9fq-hqm-37f
title: Amphistegina d'Orbigny, 1826

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/l3x-fd3-wkt
title: Foraminifera d'Orbigny, 1826

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/8b6-vdj-gs1
title: Animalia

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/djl-1sw-a1p
title: Globobulimina affinis (d'Orbigny, 1839)

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/nx0-9xk-0bt
title: Foraminifera d'Orbigny, 1826

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/f8v-vkc-m88
title: †Cibicides minimalis Schijfsma, 1946

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/s5y-crb-1hj
title: †Arnaudiella grossouvrei Douvillé, 1907

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/atj-8zh-xqz
title: †Miogypsina Sacco, 1893

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/lmw-gtl-wa7
title: †Lepidorbitoides Silvestri, 1907

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/q9f-3w2-g0g
title: Chromista

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.3535/sft-8qt-jt5
title: †Hedbergella Brönnimann & Brown, 1958

description: Digital Specimen for the physical specimen hosted at Naturalis Biodiversity Center.
DOI: 10.6084/m9.figshare.29581853
title: Supplementary Material for: The impact of endoscopic closure treatment on outcomes in patients with esophageal cancer-associated fistula: A retrospective analysis

description: Introduction: Esophageal cancer-associated fistula is strongly linked to elevated mortality. This study aims to investigate the impact of endoscopic closure on outcomes in patients with esophageal cancer-related fistula.
Methods: We retrospectively analyzed the clinical data of patients with esophageal cancer-related fistula. These patients were categorized into endoscopic closure group and conservative treatment group. The Clinical Pulmonary Infection Score (CPIS) gap, duration of hospitalization, ICU admission rates, in-hospital mortality rates, and hospitalization costs were compared between endoscopic group and conservative group. Additionally, factors associated with post-fistula survival and healing were assessed.
Results: Univariate and multivariate COX regression analyses revealed that endoscopic closure could significantly improve short-term pulmonary infections based on
CPIS gap (3 versus 2; P=0.004) but did not influence survival or fistula healing outcomes, and the hospitalization costs were elevated ($6653 versus $3350; P=0.005). Subgroup analysis focusing on esophagotracheal fistulas was also consistent with these results. Protective factors associated with improved survival prognosis included higher albumin levels (HR=0.928, 95%CI: 0.875-0.984, P=0.012), absence of bloodstream infections (positive blood culture (HR=23.055, 95%CI: 5.193-102.357, P&lt;0.001)), non-T4 stage (T4 stage (HR=1.792, 95%CI: 1.052-3.052, P=0.032)) and no distant metastasis (distant metastasis (HR=2.122, 95%CI: 1.127-3.996, P=0.020)). Cervical esophageal fistula (upper (HR=0.154, 95% CI: 0.041-0.570, P=0.005); middle (HR=0.128, 95% CI: 0.027-0.609, P=0.010); lower (HR=0.218, 95% CI: 0.052-0.902, P=0.036)) was significantly associated with improved fistula healing outcomes, while a history of radiotherapy (HR=0.265, 95% CI: 0.089-0.788, P=0.017) was a risk factor for esophageal fistula healing.
Conclusion: Our study indicates that multiple factors are significantly associated with the prognosis of patients with esophageal fistula. Endoscopic closure treatment effectively manages short-term infections but it associates with higher hospitalization costs and doesn't significantly enhance long-term healing or survival prognosis.
DOI: 10.6084/m9.figshare.29581853.v1
title: Supplementary Material for: The impact of endoscopic closure treatment on outcomes in patients with esophageal cancer-associated fistula: A retrospective analysis

description: Introduction: Esophageal cancer-associated fistula is strongly linked to elevated mortality. This study aims to investigate the impact of endoscopic closure on outcomes in patients with esophageal cancer-related fistula.
Methods: We retrospectively analyzed the clinical data of patients with esophageal cancer-related fistula. These patients were categorized into endoscopic closure group and conservative treatment group. The Clinical Pulmonary Infection Score (CPIS) gap, duration of hospitalization, ICU admission rates, in-hospital mortality rates, and hospitalization costs were compared between endoscopic group and conservative group. Additionally, factors associated with post-fistula survival and healing were assessed.
Results: Univariate and multivariate COX regression analyses revealed that endoscopic closure could significantly improve short-term pulmonary infections based on
CPIS gap (3 versus 2; P=0.004) but did not influence survival or fistula healing outcomes, and the hospitalization costs were elevated ($6653 versus $3350; P=0.005). Subgroup analysis focusing on esophagotracheal fistulas was also consistent with these results. Protective factors associated with improved survival prognosis included higher albumin levels (HR=0.928, 95%CI: 0.875-0.984, P=0.012), absence of bloodstream infections (positive blood culture (HR=23.055, 95%CI: 5.193-102.357, P&lt;0.001)), non-T4 stage (T4 stage (HR=1.792, 95%CI: 1.052-3.052, P=0.032)) and no distant metastasis (distant metastasis (HR=2.122, 95%CI: 1.127-3.996, P=0.020)). Cervical esophageal fistula (upper (HR=0.154, 95% CI: 0.041-0.570, P=0.005); middle (HR=0.128, 95% CI: 0.027-0.609, P=0.010); lower (HR=0.218, 95% CI: 0.052-0.902, P=0.036)) was significantly associated with improved fistula healing outcomes, while a history of radiotherapy (HR=0.265, 95% CI: 0.089-0.788, P=0.017) was a risk factor for esophageal fistula healing.
Conclusion: Our study indicates that multiple factors are significantly associated with the prognosis of patients with esophageal fistula. Endoscopic closure treatment effectively manages short-term infections but it associates with higher hospitalization costs and doesn't significantly enhance long-term healing or survival prognosis.
DOI: 10.6084/m9.figshare.29581856
title: Supplementary Material for: Immune and histopathological biomarkers for prognosis and neoadjuvant immunotherapy response in intrahepatic cholangiocarcinoma

description: Introduction: 
The tumor microenvironment (TME) plays a critical role in determining the clinical outcomes in patients with intrahepatic cholangiocarcinoma (iCCA). This study investigates the prognostic significance of intratumoural CD8+ T cells and regulatory T cells (Tregs), their relationship to histopathological features and their potential as predictive biomarkers for neoadjuvant immunotherapy (NAT) response.

Methods: 
Immunofluorescence analysis was conducted on a cohort of 34 resected iCCA cases to assess CD8+ T cell and Treg densities. Statistical correlations with survival and histopathological features were examined. An independent cohort of 95 iCCA pre-neoadjuvant therapy (NAT) biopsy specimens was analyzed to evaluate the potential of these biomarkers in predicting therapeutic response. 
Results: 
In the cohort of resected iCCA cases, immunofluorescence revealed that increased CD8+ T cell infiltration is associated with improved survival (P = 0.018), underscoring their role in anti-tumor immunity. Conversely, higher density of Tregs is linked to poorer survival (P = 0.038), suggesting its tumor-promoting, immunosuppressive effects. Intriguingly, CD8+ T cells and Tregs are associated with distinct histopathological features, with CD8+ T cells correlating with dense tumor infiltrating lymphocytes (TIL, Pearson’s R = 0.498; P = 0.004) and Tregs being more prevalent in regions of tumor budding (TB, Pearson’s R = 0.382; P = 0.029). These observations were validated in an independent pre-NAT cohort with (n = 95), whereby higher TIL density, particularly increased CD8+ T cells and reduced Treg, and lower TB predict favorable response to NAT, as shown by improved overall and disease-free survival. 
Conclusion: 
These findings provide critical insights for stratifying patients and highlight the potential for optimizing immune-targeted therapies in patients with iCCA.
DOI: 10.6084/m9.figshare.29581856.v1
title: Supplementary Material for: Immune and histopathological biomarkers for prognosis and neoadjuvant immunotherapy response in intrahepatic cholangiocarcinoma

description: Introduction: 
The tumor microenvironment (TME) plays a critical role in determining the clinical outcomes in patients with intrahepatic cholangiocarcinoma (iCCA). This study investigates the prognostic significance of intratumoural CD8+ T cells and regulatory T cells (Tregs), their relationship to histopathological features and their potential as predictive biomarkers for neoadjuvant immunotherapy (NAT) response.

Methods: 
Immunofluorescence analysis was conducted on a cohort of 34 resected iCCA cases to assess CD8+ T cell and Treg densities. Statistical correlations with survival and histopathological features were examined. An independent cohort of 95 iCCA pre-neoadjuvant therapy (NAT) biopsy specimens was analyzed to evaluate the potential of these biomarkers in predicting therapeutic response. 
Results: 
In the cohort of resected iCCA cases, immunofluorescence revealed that increased CD8+ T cell infiltration is associated with improved survival (P = 0.018), underscoring their role in anti-tumor immunity. Conversely, higher density of Tregs is linked to poorer survival (P = 0.038), suggesting its tumor-promoting, immunosuppressive effects. Intriguingly, CD8+ T cells and Tregs are associated with distinct histopathological features, with CD8+ T cells correlating with dense tumor infiltrating lymphocytes (TIL, Pearson’s R = 0.498; P = 0.004) and Tregs being more prevalent in regions of tumor budding (TB, Pearson’s R = 0.382; P = 0.029). These observations were validated in an independent pre-NAT cohort with (n = 95), whereby higher TIL density, particularly increased CD8+ T cells and reduced Treg, and lower TB predict favorable response to NAT, as shown by improved overall and disease-free survival. 
Conclusion: 
These findings provide critical insights for stratifying patients and highlight the potential for optimizing immune-targeted therapies in patients with iCCA.
DOI: 10.15468/dl.um8zyz
title: Occurrence Download

description: A dataset containing 468 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Draba hispida"
 ]
}

 The dataset includes 468 records from 47 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0023457-250711103210423/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.6084/m9.figshare.29581859
title: Supplementary Material for: AKAP12 variant 1 knockout enhances vascular endothelial cell motility

description: This study investigates the role of AKAP12 in endothelial cell motility with a specific focus on AKAP12 variants, AKAP12v1 and AKAP12v2. Previous work has shown that AKAP12, a multivalent A-kinase anchoring protein that binds to PKA and several other proteins regulating protein phosphorylation, is expressed at low levels in most endothelia in vivo but is expressed at higher levels in cells in vitro. Here, we found that AKAP12 expression in endothelial cell (HUVEC) cultures was cell density-dependent, with the expression being highest in subconfluent cultures and lowest in confluent cultures. AKAP12 expression was also elevated in cells at the wound edge of wounded endothelial cell monolayers. Knockdown of variants 1 and 2 inhibited cell migration. However, CRISPR/Cas9 knockout of AKAP12v1 enhanced migration, indicating that the absence of this variant and the presence of AKAP12v2 likely alters the signaling events controlling cell motility. Further analysis using bulk RNA sequencing revealed that the loss of AKAP12v1 affected genes associated with cell migration and intercellular junctions. We propose that AKAP12v1 and AKAP12v2 play distinct yet complementary roles in endothelial cell migration and likely work together in controlling the signaling events associated with vascular repair and development.
DOI: 10.6084/m9.figshare.29581859.v1
title: Supplementary Material for: AKAP12 variant 1 knockout enhances vascular endothelial cell motility

description: This study investigates the role of AKAP12 in endothelial cell motility with a specific focus on AKAP12 variants, AKAP12v1 and AKAP12v2. Previous work has shown that AKAP12, a multivalent A-kinase anchoring protein that binds to PKA and several other proteins regulating protein phosphorylation, is expressed at low levels in most endothelia in vivo but is expressed at higher levels in cells in vitro. Here, we found that AKAP12 expression in endothelial cell (HUVEC) cultures was cell density-dependent, with the expression being highest in subconfluent cultures and lowest in confluent cultures. AKAP12 expression was also elevated in cells at the wound edge of wounded endothelial cell monolayers. Knockdown of variants 1 and 2 inhibited cell migration. However, CRISPR/Cas9 knockout of AKAP12v1 enhanced migration, indicating that the absence of this variant and the presence of AKAP12v2 likely alters the signaling events controlling cell motility. Further analysis using bulk RNA sequencing revealed that the loss of AKAP12v1 affected genes associated with cell migration and intercellular junctions. We propose that AKAP12v1 and AKAP12v2 play distinct yet complementary roles in endothelial cell migration and likely work together in controlling the signaling events associated with vascular repair and development.