cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.10184594
title: MOESM2 of RstA, a two-component response regulator, plays important roles in multiple virulence-associated processes in enterohemorrhagic Escherichia coli O157:H7

description: Additional file 2: Excel file S1. Down-regulated genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S2. Up-regulated genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain. Excel file S3. Differently expressed LEE genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S4. Differently expressed acid tolerance genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S5. Differently expressed diguanylate cyclases and phosphodiesterases genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S6. Differently expressed Stxs and Nle genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S7. Differently expressed regulatory genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain.
DOI: 10.6084/m9.figshare.10184594.v1
title: MOESM2 of RstA, a two-component response regulator, plays important roles in multiple virulence-associated processes in enterohemorrhagic Escherichia coli O157:H7

description: Additional file 2: Excel file S1. Down-regulated genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S2. Up-regulated genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain. Excel file S3. Differently expressed LEE genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S4. Differently expressed acid tolerance genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S5. Differently expressed diguanylate cyclases and phosphodiesterases genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S6. Differently expressed Stxs and Nle genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain, Excel file S7. Differently expressed regulatory genes in the ΔrstA mutant compared with EHEC O157:H7 wild type strain.
DOI: 10.6084/m9.figshare.10184597
title: MOESM4 of Transcriptomic and metabolomic profiling provide novel insights into fruit development and flesh coloration in Prunus mira Koehne, a special wild peach species

description: Additional file 4: Table S4. List of the optimized genes based on gene structure optimization analysis in P. persica. (XLSX 301 kb)
DOI: 10.6084/m9.figshare.10184597.v1
title: MOESM4 of Transcriptomic and metabolomic profiling provide novel insights into fruit development and flesh coloration in Prunus mira Koehne, a special wild peach species

description: Additional file 4: Table S4. List of the optimized genes based on gene structure optimization analysis in P. persica. (XLSX 301 kb)
DOI: 10.6084/m9.figshare.7559249
title: Additional file 8: of Pathway crosstalk perturbation network modeling for identification of connectivity changes induced by diabetic neuropathy and pioglitazone

description: Jaccard index matrix of similarity for PXPN nodes in XLSX format. (XLSX 9 kb)
DOI: 10.6084/m9.figshare.10184600
title: MOESM5 of Transcriptomic and metabolomic profiling provide novel insights into fruit development and flesh coloration in Prunus mira Koehne, a special wild peach species

description: Additional file 5: Table S5. Intron-exon structure of the optimized genes in P. persica. (XLSX 3036 kb)
DOI: 10.6084/m9.figshare.10184600.v1
title: MOESM5 of Transcriptomic and metabolomic profiling provide novel insights into fruit development and flesh coloration in Prunus mira Koehne, a special wild peach species

description: Additional file 5: Table S5. Intron-exon structure of the optimized genes in P. persica. (XLSX 3036 kb)
DOI: 10.6084/m9.figshare.10184606
title: MOESM6 of Transcriptomic and metabolomic profiling provide novel insights into fruit development and flesh coloration in Prunus mira Koehne, a special wild peach species

description: Additional file 6: Table S6. List of all the differentially expressed genes identified in this study along with their FPKM data. (XLSX 4901 kb)
DOI: 10.6084/m9.figshare.10184606.v1
title: MOESM6 of Transcriptomic and metabolomic profiling provide novel insights into fruit development and flesh coloration in Prunus mira Koehne, a special wild peach species

description: Additional file 6: Table S6. List of all the differentially expressed genes identified in this study along with their FPKM data. (XLSX 4901 kb)
DOI: 10.6084/m9.figshare.25218065
title: Demo for the 2023 tropical cyclone research

description: Tropical cyclones.
DOI: 10.6084/m9.figshare.25218065.v1
title: Demo for the 2023 tropical cyclone research

description: Tropical cyclones.
DOI: 10.6084/m9.figshare.8087465
title: Additional file 5: of Exploring the adaptive mechanism of Passiflora edulis in karst areas via an integrative analysis of nutrient elements and transcriptional profiles

description: Table S1. The annotation of DEGs listed in Table 4. (XLS 55 kb)
DOI: 10.6084/m9.figshare.8087474.v1
title: Additional file 6: of Exploring the adaptive mechanism of Passiflora edulis in karst areas via an integrative analysis of nutrient elements and transcriptional profiles

description: Table S2. The details of DEGs enriched in KEGG pathways. (XLS 28 kb)
DOI: 10.6084/m9.figshare.12277964
title: Equation_Tumor2.m represents the condition in which the formation rate of GADD45B|MKK7 complex is inhibited after the introduction of MM cells. from Mathematical modelling of the role of GADD45<i>β</i> in the pathogenesis of multiple myeloma

description: Multiple myeloma (MM) is an incurable disease with relatively high morbidity and mortality rates. Great efforts were made to develop nuclear factor-kappa B (NF-κB)-targeted therapies against MM disease. However, these treatments influence MM cells as well as normal cells, inevitably causing serious side effects. Further research showed that NF-κB signalling promotes the survival of MM cells by interacting with JNK signalling through growth arrest and DNA damage-inducible beta (GADD45β), the downstream module of NF-κB signalling. The GADD45β-targeted intervention was suggested to be an effective and MM cell-specific treatment. However, the underlying mechanism through which GADD45β promotes the survival of MM cells is usually ignored in the previous models. A mathematical model of MM is built in this paper to investigate how NF-κB signalling acts along with JNK signalling through GADD45β and MKK7 to promote the survival of MM cells. The model cannot only mimic the variations in bone cells, the bone volume and MM cells with time, but it can also examine how the NF-κB pathway acts with the JNK pathway to promote the development of MM cells. In addition, the model also investigates the efficacies of GADD45β<i>-</i> and NF-κB-targeted treatments, suggesting that GADD45β-targeted therapy is more effective but has no apparent side effects. The simulation results match the experimental observations. It is anticipated that this model could be employed as a useful tool to initially investigate and even explore potential therapies involving the NF-κB and JNK pathways in the future.
DOI: 10.6084/m9.figshare.12277964.v1
title: Equation_Tumor2.m represents the condition in which the formation rate of GADD45B|MKK7 complex is inhibited after the introduction of MM cells. from Mathematical modelling of the role of GADD45<i>β</i> in the pathogenesis of multiple myeloma

description: Multiple myeloma (MM) is an incurable disease with relatively high morbidity and mortality rates. Great efforts were made to develop nuclear factor-kappa B (NF-κB)-targeted therapies against MM disease. However, these treatments influence MM cells as well as normal cells, inevitably causing serious side effects. Further research showed that NF-κB signalling promotes the survival of MM cells by interacting with JNK signalling through growth arrest and DNA damage-inducible beta (GADD45β), the downstream module of NF-κB signalling. The GADD45β-targeted intervention was suggested to be an effective and MM cell-specific treatment. However, the underlying mechanism through which GADD45β promotes the survival of MM cells is usually ignored in the previous models. A mathematical model of MM is built in this paper to investigate how NF-κB signalling acts along with JNK signalling through GADD45β and MKK7 to promote the survival of MM cells. The model cannot only mimic the variations in bone cells, the bone volume and MM cells with time, but it can also examine how the NF-κB pathway acts with the JNK pathway to promote the development of MM cells. In addition, the model also investigates the efficacies of GADD45β<i>-</i> and NF-κB-targeted treatments, suggesting that GADD45β-targeted therapy is more effective but has no apparent side effects. The simulation results match the experimental observations. It is anticipated that this model could be employed as a useful tool to initially investigate and even explore potential therapies involving the NF-κB and JNK pathways in the future.
DOI: 10.6084/m9.figshare.10184645
title: MOESM2 of The luxury effect beyond cities: bats respond to socioeconomic variation across landscapes

description: Additional file 2. Regression model results. Preliminary regression model results on the effects of temperature and year on worksheet 1; interaction model results on income and selected land cover on worksheet 2.
DOI: 10.6084/m9.figshare.10184645.v1
title: MOESM2 of The luxury effect beyond cities: bats respond to socioeconomic variation across landscapes

description: Additional file 2. Regression model results. Preliminary regression model results on the effects of temperature and year on worksheet 1; interaction model results on income and selected land cover on worksheet 2.
DOI: 10.6084/m9.figshare.10184663
title: MOESM1 of Ghd8 controls rice photoperiod sensitivity by forming a complex that interacts with Ghd7

description: Additional file 1. Table S1. Functional alleles of Ghd8, Ghd7 and Hd1 and promoter type of Hd3a in the panel of 196 rice varieties.
DOI: 10.6084/m9.figshare.10184672
title: MOESM2 of Ghd8 controls rice photoperiod sensitivity by forming a complex that interacts with Ghd7

description: Additional file 2. Table S2. PCR primers and EMSA probes used in this study.
DOI: 10.6084/m9.figshare.10184672.v1
title: MOESM2 of Ghd8 controls rice photoperiod sensitivity by forming a complex that interacts with Ghd7

description: Additional file 2. Table S2. PCR primers and EMSA probes used in this study.
DOI: 10.6084/m9.figshare.10184873
title: MOESM1 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 1: Table S1. Number of SSRs and SSR markers identified on different chromosomes. Table S2. Summary of SSR motifs and repeats.
DOI: 10.6084/m9.figshare.10184873.v1
title: MOESM1 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 1: Table S1. Number of SSRs and SSR markers identified on different chromosomes. Table S2. Summary of SSR motifs and repeats.
DOI: 10.6084/m9.figshare.10184885
title: MOESM3 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 3: Table S3. Summary of SSR markers in subgenome A01â A05.
DOI: 10.6084/m9.figshare.10184885.v1
title: MOESM3 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 3: Table S3. Summary of SSR markers in subgenome A01â A05.
DOI: 10.6084/m9.figshare.10184888
title: MOESM4 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 4: Table S4. Summary of SSR markers in subgenome A06â A10.
DOI: 10.6084/m9.figshare.10184888.v1
title: MOESM4 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 4: Table S4. Summary of SSR markers in subgenome A06â A10.
DOI: 10.6084/m9.figshare.12277970.v1
title: tumor_IKK.m is used to reproduce what happens after inhibiting the activation rate of IKK. from Mathematical modelling of the role of GADD45<i>β</i> in the pathogenesis of multiple myeloma

description: Multiple myeloma (MM) is an incurable disease with relatively high morbidity and mortality rates. Great efforts were made to develop nuclear factor-kappa B (NF-κB)-targeted therapies against MM disease. However, these treatments influence MM cells as well as normal cells, inevitably causing serious side effects. Further research showed that NF-κB signalling promotes the survival of MM cells by interacting with JNK signalling through growth arrest and DNA damage-inducible beta (GADD45β), the downstream module of NF-κB signalling. The GADD45β-targeted intervention was suggested to be an effective and MM cell-specific treatment. However, the underlying mechanism through which GADD45β promotes the survival of MM cells is usually ignored in the previous models. A mathematical model of MM is built in this paper to investigate how NF-κB signalling acts along with JNK signalling through GADD45β and MKK7 to promote the survival of MM cells. The model cannot only mimic the variations in bone cells, the bone volume and MM cells with time, but it can also examine how the NF-κB pathway acts with the JNK pathway to promote the development of MM cells. In addition, the model also investigates the efficacies of GADD45β<i>-</i> and NF-κB-targeted treatments, suggesting that GADD45β-targeted therapy is more effective but has no apparent side effects. The simulation results match the experimental observations. It is anticipated that this model could be employed as a useful tool to initially investigate and even explore potential therapies involving the NF-κB and JNK pathways in the future.
DOI: 10.6084/m9.figshare.8087738
title: MOESM5 of Comparative analysis of differentially expressed genes between the ovaries from pregnant and nonpregnant goats using RNA-Seq

description: Additional file 5. Differential gene detected in two libraries.
DOI: 10.6084/m9.figshare.12277979
title: tumor.m is used to simulate what happens after the introduction and removal of MM cells from Mathematical modelling of the role of GADD45<i>β</i> in the pathogenesis of multiple myeloma

description: Multiple myeloma (MM) is an incurable disease with relatively high morbidity and mortality rates. Great efforts were made to develop nuclear factor-kappa B (NF-κB)-targeted therapies against MM disease. However, these treatments influence MM cells as well as normal cells, inevitably causing serious side effects. Further research showed that NF-κB signalling promotes the survival of MM cells by interacting with JNK signalling through growth arrest and DNA damage-inducible beta (GADD45β), the downstream module of NF-κB signalling. The GADD45β-targeted intervention was suggested to be an effective and MM cell-specific treatment. However, the underlying mechanism through which GADD45β promotes the survival of MM cells is usually ignored in the previous models. A mathematical model of MM is built in this paper to investigate how NF-κB signalling acts along with JNK signalling through GADD45β and MKK7 to promote the survival of MM cells. The model cannot only mimic the variations in bone cells, the bone volume and MM cells with time, but it can also examine how the NF-κB pathway acts with the JNK pathway to promote the development of MM cells. In addition, the model also investigates the efficacies of GADD45β<i>-</i> and NF-κB-targeted treatments, suggesting that GADD45β-targeted therapy is more effective but has no apparent side effects. The simulation results match the experimental observations. It is anticipated that this model could be employed as a useful tool to initially investigate and even explore potential therapies involving the NF-κB and JNK pathways in the future.
DOI: 10.18465/faa4572d-623c-431f-b03a-cfe8a5100c53
title: Protocole de calcul des MNB - Noirmoutier 2020

description: Exemple de suites de traitements des données LiDAR de l'approche classique à l'analyse des formes d'ondes. En 2020 le site de l'Herbaudière a été l'un des 1er sites d'entrainement de la combinaison des formes d'ondes du vert et de l'infrarouge pour identifier les algues des berges et améliorer le suivi de la continuité terre-mer. Sous l'eau ces algues sont sombres sans composante infrarouge alors qu'en dehors de l'eau elles sont fortement réfléchissantes dans l'infrarouge. La combinaison avec le vert, sombre pour les algues brunes, neutralise cette effet et facilité l'observation de la continuité des surfaces des la terre à la mer.
DOI: 10.6084/m9.figshare.12277979.v1
title: tumor.m is used to simulate what happens after the introduction and removal of MM cells from Mathematical modelling of the role of GADD45<i>β</i> in the pathogenesis of multiple myeloma

description: Multiple myeloma (MM) is an incurable disease with relatively high morbidity and mortality rates. Great efforts were made to develop nuclear factor-kappa B (NF-κB)-targeted therapies against MM disease. However, these treatments influence MM cells as well as normal cells, inevitably causing serious side effects. Further research showed that NF-κB signalling promotes the survival of MM cells by interacting with JNK signalling through growth arrest and DNA damage-inducible beta (GADD45β), the downstream module of NF-κB signalling. The GADD45β-targeted intervention was suggested to be an effective and MM cell-specific treatment. However, the underlying mechanism through which GADD45β promotes the survival of MM cells is usually ignored in the previous models. A mathematical model of MM is built in this paper to investigate how NF-κB signalling acts along with JNK signalling through GADD45β and MKK7 to promote the survival of MM cells. The model cannot only mimic the variations in bone cells, the bone volume and MM cells with time, but it can also examine how the NF-κB pathway acts with the JNK pathway to promote the development of MM cells. In addition, the model also investigates the efficacies of GADD45β<i>-</i> and NF-κB-targeted treatments, suggesting that GADD45β-targeted therapy is more effective but has no apparent side effects. The simulation results match the experimental observations. It is anticipated that this model could be employed as a useful tool to initially investigate and even explore potential therapies involving the NF-κB and JNK pathways in the future.
DOI: 10.6084/m9.figshare.7559588.v1
title: Additional file 4: of Genome-wide dynamic network analysis reveals a critical transition state of flower development in Arabidopsis

description: Table S1. Functional categories for the DNB members of Arabidopsis flower development. (TXT 7 kb)
DOI: 10.6084/m9.figshare.10184900
title: MOESM5 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 5: Table S5. Summary of SSR markers in subgenome B01â B05.
DOI: 10.6084/m9.figshare.10184900.v1
title: MOESM5 of Genome-wide identification of microsatellite markers from cultivated peanut (Arachis hypogaea L.)

description: Additional file 5: Table S5. Summary of SSR markers in subgenome B01â B05.
DOI: 10.6084/m9.figshare.12277982
title: parameters.m represents the parameters in the NF-kB pathway. from Mathematical modelling of the role of GADD45<i>β</i> in the pathogenesis of multiple myeloma

description: Multiple myeloma (MM) is an incurable disease with relatively high morbidity and mortality rates. Great efforts were made to develop nuclear factor-kappa B (NF-κB)-targeted therapies against MM disease. However, these treatments influence MM cells as well as normal cells, inevitably causing serious side effects. Further research showed that NF-κB signalling promotes the survival of MM cells by interacting with JNK signalling through growth arrest and DNA damage-inducible beta (GADD45β), the downstream module of NF-κB signalling. The GADD45β-targeted intervention was suggested to be an effective and MM cell-specific treatment. However, the underlying mechanism through which GADD45β promotes the survival of MM cells is usually ignored in the previous models. A mathematical model of MM is built in this paper to investigate how NF-κB signalling acts along with JNK signalling through GADD45β and MKK7 to promote the survival of MM cells. The model cannot only mimic the variations in bone cells, the bone volume and MM cells with time, but it can also examine how the NF-κB pathway acts with the JNK pathway to promote the development of MM cells. In addition, the model also investigates the efficacies of GADD45β<i>-</i> and NF-κB-targeted treatments, suggesting that GADD45β-targeted therapy is more effective but has no apparent side effects. The simulation results match the experimental observations. It is anticipated that this model could be employed as a useful tool to initially investigate and even explore potential therapies involving the NF-κB and JNK pathways in the future.
DOI: 10.6084/m9.figshare.7559600
title: Additional file 7: of Genome-wide dynamic network analysis reveals a critical transition state of flower development in Arabidopsis

description: Table S5. Node properties file for the network. (TXT 4 kb)
DOI: 10.6084/m9.figshare.12277973
title: Equation_Tumor1.m represents the condition with the introduction of MM cells. from Mathematical modelling of the role of GADD45<i>β</i> in the pathogenesis of multiple myeloma

description: Multiple myeloma (MM) is an incurable disease with relatively high morbidity and mortality rates. Great efforts were made to develop nuclear factor-kappa B (NF-κB)-targeted therapies against MM disease. However, these treatments influence MM cells as well as normal cells, inevitably causing serious side effects. Further research showed that NF-κB signalling promotes the survival of MM cells by interacting with JNK signalling through growth arrest and DNA damage-inducible beta (GADD45β), the downstream module of NF-κB signalling. The GADD45β-targeted intervention was suggested to be an effective and MM cell-specific treatment. However, the underlying mechanism through which GADD45β promotes the survival of MM cells is usually ignored in the previous models. A mathematical model of MM is built in this paper to investigate how NF-κB signalling acts along with JNK signalling through GADD45β and MKK7 to promote the survival of MM cells. The model cannot only mimic the variations in bone cells, the bone volume and MM cells with time, but it can also examine how the NF-κB pathway acts with the JNK pathway to promote the development of MM cells. In addition, the model also investigates the efficacies of GADD45β<i>-</i> and NF-κB-targeted treatments, suggesting that GADD45β-targeted therapy is more effective but has no apparent side effects. The simulation results match the experimental observations. It is anticipated that this model could be employed as a useful tool to initially investigate and even explore potential therapies involving the NF-κB and JNK pathways in the future.
DOI: 10.6084/m9.figshare.7559603
title: Additional file 8: of Genome-wide dynamic network analysis reveals a critical transition state of flower development in Arabidopsis

description: Table S6. The gene regulatory network Cytoscape file. (TXT 7 kb)