cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.15121/2301597
title: Utah FORGE: Microseismic Surface Network Catalogs for Wells 16A(78)-32 and 16B(78)-32 Stimulation 2022 and Circulation 2023

description: This dataset includes microseismic surface network catalogs for Utah FORGE. Data were recorded during the stimulation of well 16A(78)-32 in 2022 and the circulation tests between wells 16A(78)-32 and 16B(78)-32 in 2023. Near-surface seismic monitoring during circulation experiments in wells 16A(78)-32 and 16B(78)-32 revealed fracture growth after shut-in. The catalog provided here offers a comprehensive list of microseismic events, including details on their spatial and temporal distribution, magnitude, and clustering behavior.
DOI: 10.7799/2301601
title: University of Louisiana at Lafayette

description: A partnership with the University of Louisiana and U.S. Department of Energy's National Renewable Energy Laboratory (NREL) to collect solar data to support future solar power generation in the United States. The measurement station monitors global, direct, and diffuse irradiance to define the amount of solar energy that hits this particular location. The solar measurement instrumentation is also accompanied by meteorological monitoring equipment to provide scientists with a complete picture of the solar power possibilities.
DOI: 10.7799/2301607
title: University of Florida

description: A partnership with the University of Florida and U.S. Department of Energy's National Renewable Energy Laboratory (NREL) to collect solar data to support future solar power generation in the United States. The measurement station monitors global, direct, and diffuse irradiance to define the amount of solar energy that hits this particular location. The solar measurement instrumentation is also accompanied by meteorological monitoring equipment to provide scientists with a complete picture of the solar power possibilities.
DOI: 10.21227/2z1k-bn65
title: Laboratory-generated noisy voltage signal with a dc component under nonideal sampling

description: The Excel file contains samples of a laboratory-generated noisy voltage signal with a dc component under nonideal sampling.This test signal is generated in a laboratory for assessing power frequency estimation algorithms.The first column represents the sample time.The second column represents the voltage signal samples.The reference fundamental frequency is 46 Hz.The nominal voltage amplitude is 10 V.The actual sampling rate varies in the range of 9.99834~10.01027 kHz.
DOI: 10.15151/esrf-es-1491588397
title: Assessing TET aminopeptidase dynamics by room-temperature crystallography

description: The present proposal aims at getting access to the dynamic of TET amino peptidases by determinating the structure at various temperatures of one hyperthermophilic TET peptidase, called PhTET3, isolated from the archaeon P. horikoshii.
DOI: 10.15151/esrf-es-1511541654
title: scanning diffraction test on guanine crystals

description: scanning diffraction test on guanine crystals
DOI: 10.6084/m9.figshare.25149093
title: Gasque et al 2024_S3_Dpi_CNS_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149093.v1
title: Gasque et al 2024_S3_Dpi_CNS_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149096.v1
title: Metadata for Gasque et al 2024_S2_For_survival_analysis from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149096
title: Metadata for Gasque et al 2024_S2_For_survival_analysis from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149099
title: Gasque et al 2024_S1_Whole_caterpillar_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149102
title: Metadata for Gasque et al 2024_S1_Whole_caterpillar_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.15151/esrf-es-1435450781
title: Operando determination of the kinetics and structure of the copper-sites in copper-exchanged offretite using copper K-edge XANES spectroscop

description: The methane-to-methanol conversion is seen as one of the "holy grail" reactions of catalysis. One extensively examined approach is the chemical looping approach using copper exchanged zeolites. In-house studies have determined that copper-exchanged zeolite offretite outperforms all other zeolites in multiple metrics, not least yield. 

Using quick XAS scans, we will investigate the kinetics of the different copper-species, as well as using XRD to examine the location of the copper-sites during each stage of the chemical looping cycle. On-line mass spectrometry will be used to observe the kinetics of product desorption and to determine the yield. Using the timeframes and yield will allow us to dermine the productivity of the process, a metric paramount to any other for determining if a process merits industrial valorisation. 

As quick XAS scans and XRD may both be performed at BM31, it is the most suited beamline for such an experiment.
DOI: 10.6084/m9.figshare.25149099.v1
title: Gasque et al 2024_S1_Whole_caterpillar_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149102.v1
title: Metadata for Gasque et al 2024_S1_Whole_caterpillar_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149105
title: Metadata for Gasque et al 2024_S3_Dpi_CNS_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.
DOI: 10.6084/m9.figshare.25149105.v1
title: Metadata for Gasque et al 2024_S3_Dpi_CNS_infections from Baculovirus entry into the central nervous system of <i>Spodoptera exigua</i> caterpillars is independent of the viral protein tyrosine phosphatase

description: Neuroparasitism concerns the hostile take-over of a host's nervous system by a foreign invader, in order to alter the behaviour of the host in favour of the parasite. One of the most remarkable cases of parasite-induced host behavioural manipulation comprises the changes baculoviruses induce in their caterpillar hosts. Baculoviruses may manipulate caterpillar behaviour in two ways: hyperactivity (increased movement in the horizontal plane) and/or tree-top disease (movement to elevated levels in the vertical plane). Those behavioural changes are followed by liquefaction and death of the caterpillar. In <i>Autographa californica</i> multiple nucleopolyhedrovirus (AcMNPV)-infected <i>Spodoptera exigua</i> caterpillars, an enzymatic active form of the virally encoded protein tyrosine phosphatase (PTP) is needed for the expression of hyperactivity from 3 days post infection (dpi). Using eGFP-expressing recombinant AcMNPV strains, we show that infection of the caterpillar's central nervous system (CNS) can be observed primarily from 3 dpi onwards. In addition, we demonstrate that the structural and enzymatic function of PTP does not play a role in infection of the CNS. Instead we show that the virus entered the CNS via the trachea, progressing caudally to frontally through the CNS and that the infection progressed from the outermost cell layers towards the inner cell layers of the CNS, in a PTP independent manner. These findings help to further understand parasitic manipulation and the mechanisms by which neuroparasites infect the host nervous system to manipulate host behaviour.