cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.8341760
title: Supplementary Material for: Analysis of the Risk Factors for Postherpetic Neuralgia

description: <b><i>Background:</i></b> Postherpetic neuralgia is the most common complication of herpes zoster, affecting 30% of patients. It seriously affects the quality of life of patients and the curative effect of treatment is limited. So far, researchers do not fully understand the risk factors for postherpetic neuralgia and more research is needed. <b><i>Objective:</i></b> The aim of this paper was to investigate the risk factors for postherpetic neuralgia and provide reference for clinical diagnosis and treatment. <b><i>Methods:</i></b> A total of 202 inpatients with herpes zoster in the General Hospital of Tianjin Medical University were recruited as study subjects. According to the occurrence of postherpetic neuralgia, the patients were divided into the postherpetic neuralgia group and the nonpostherpetic neuralgia group. Data on age, gender, initial symptoms, clinical classification, involved nerves, pain grading, antiviral therapy, glucocorticoid use, and other clinical data of patients in the two groups were collected and statistically analyzed. Univariate and multivariate analysis methods were used to analyze the differences between the two groups and determine the influencing factors of postherpetic neuralgia. <b><i>Results:</i></b> The univariate statistical analysis of the factors influencing postherpetic neuralgia showed that the contribution of gender, initial symptoms, general clinical classification, use of glucocorticoid, and the interval from onset to antiviral therapy were not statistically significant, while the differences in age, specific clinical classification, involved nerves, severity of pain during the acute stage, and body side of skin lesion distribution were statistically significant. Multivariate logistic regression analysis showed that gender, use of glucocorticoid, interval from onset to antiviral therapy, involved nerves, and specific clinical classification showed no statistical significance. However, there were significant differences in age, body side of skin lesion distribution, general clinical classification, and degree of pain during the acute stage of the disease. <b><i>Conclusion:</i></b> Pain during the acute stage of herpes zoster, age greater than 70 years, and serious type of skin lesion are risk factors for postherpetic neuralgia (<i>p</i> &lt; 0.05, OR &gt;1).
DOI: 10.6084/m9.figshare.8341370
title: Institution information integration dataset of open data

description: integration data of institution informaiton based on grid , orgref, Open ISNI, VIAF and wikidata.<br>
DOI: 10.6084/m9.figshare.8341760.v1
title: Supplementary Material for: Analysis of the Risk Factors for Postherpetic Neuralgia

description: <b><i>Background:</i></b> Postherpetic neuralgia is the most common complication of herpes zoster, affecting 30% of patients. It seriously affects the quality of life of patients and the curative effect of treatment is limited. So far, researchers do not fully understand the risk factors for postherpetic neuralgia and more research is needed. <b><i>Objective:</i></b> The aim of this paper was to investigate the risk factors for postherpetic neuralgia and provide reference for clinical diagnosis and treatment. <b><i>Methods:</i></b> A total of 202 inpatients with herpes zoster in the General Hospital of Tianjin Medical University were recruited as study subjects. According to the occurrence of postherpetic neuralgia, the patients were divided into the postherpetic neuralgia group and the nonpostherpetic neuralgia group. Data on age, gender, initial symptoms, clinical classification, involved nerves, pain grading, antiviral therapy, glucocorticoid use, and other clinical data of patients in the two groups were collected and statistically analyzed. Univariate and multivariate analysis methods were used to analyze the differences between the two groups and determine the influencing factors of postherpetic neuralgia. <b><i>Results:</i></b> The univariate statistical analysis of the factors influencing postherpetic neuralgia showed that the contribution of gender, initial symptoms, general clinical classification, use of glucocorticoid, and the interval from onset to antiviral therapy were not statistically significant, while the differences in age, specific clinical classification, involved nerves, severity of pain during the acute stage, and body side of skin lesion distribution were statistically significant. Multivariate logistic regression analysis showed that gender, use of glucocorticoid, interval from onset to antiviral therapy, involved nerves, and specific clinical classification showed no statistical significance. However, there were significant differences in age, body side of skin lesion distribution, general clinical classification, and degree of pain during the acute stage of the disease. <b><i>Conclusion:</i></b> Pain during the acute stage of herpes zoster, age greater than 70 years, and serious type of skin lesion are risk factors for postherpetic neuralgia (<i>p</i> &lt; 0.05, OR &gt;1).
DOI: 10.6084/m9.figshare.8341370.v1
title: Institution information integration dataset of open data

description: integration data of institution informaiton based on grid , orgref, Open ISNI, VIAF and wikidata.<br>
DOI: 10.6084/m9.figshare.8341370.v2
title: Institution information integration dataset of open data

description: integration data of institution informaiton based on grid , orgref, Open ISNI, VIAF and wikidata.<br>
DOI: 10.6084/m9.figshare.8341370.v3
title: Institution information integration dataset of open data

description: integration data of institution informaiton based on grid , orgref, Open ISNI, VIAF and wikidata.<br>
DOI: 10.6084/m9.figshare.8341187.v1
title: Molecular impact of Deformed wing virus type A in the honey bee brain.

description: Crop pollination by the western honey bee <i>Apis mellifera</i> is vital to agriculture but threatened by alarmingly high levels of colony mortality, especially in Europe and the US. Colony loss is due, in part, to the high viral loads of Deformed wing virus (DWV), transmitted by the ectoparasitic mite <i>Varroa destructor, </i>especially throughout the overwintering period of a honey bee colony. Covert DWV infection is commonplace and has been causally linked to precocious foraging, which itself has been linked to colony loss. Taking advantage of four brain transcriptome studies that unexpectedly revealed evidence of covert DWV-A infection, we set out to explore whether this effect is due to DWV-A mimicking naturally occurring changes in brain gene expression that are associated with behavioral maturation. Consistent with this hypothesis, we found that brain gene expression profiles of DWV-A infected bees resembled those of foragers, even in samples that were much younger than typical foragers. In addition, transcriptional regulatory network analysis revealed a positive association between DWV-A infection and transcription factors previously associated with honey bee foraging behavior. Surprisingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively few glial cells in the insect brain and they are rarely associated with behavioral plasticity. Covert DWV-A infection also has been linked to impaired learning, which together with precocious foraging can lead to increased occurrence of bees from one colony mistakenly joining another, especially under crowded modern apiary conditions. Together, these findings support a mechanistic link by which DWV-A may manipulate host behavior to facilitate horizontal transmission.
DOI: 10.6084/m9.figshare.8341370.v4
title: Institution information integration dataset of open data

description: integration data of institution informaiton based on grid , orgref, Open ISNI, VIAF and wikidata.<br>
DOI: 10.6084/m9.figshare.8341187.v2
title: Molecular impact of Deformed wing virus type A in the honey bee brain.

description: Crop pollination by the western honey bee <i>Apis mellifera</i> is vital to agriculture but threatened by alarmingly high levels of colony mortality, especially in Europe and the US. Colony loss is due, in part, to the high viral loads of Deformed wing virus (DWV), transmitted by the ectoparasitic mite <i>Varroa destructor, </i>especially throughout the overwintering period of a honey bee colony. Covert DWV infection is commonplace and has been causally linked to precocious foraging, which itself has been linked to colony loss. Taking advantage of four brain transcriptome studies that unexpectedly revealed evidence of covert DWV-A infection, we set out to explore whether this effect is due to DWV-A mimicking naturally occurring changes in brain gene expression that are associated with behavioral maturation. Consistent with this hypothesis, we found that brain gene expression profiles of DWV-A infected bees resembled those of foragers, even in samples that were much younger than typical foragers. In addition, transcriptional regulatory network analysis revealed a positive association between DWV-A infection and transcription factors previously associated with honey bee foraging behavior. Surprisingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively few glial cells in the insect brain and they are rarely associated with behavioral plasticity. Covert DWV-A infection also has been linked to impaired learning, which together with precocious foraging can lead to increased occurrence of bees from one colony mistakenly joining another, especially under crowded modern apiary conditions. Together, these findings support a mechanistic link by which DWV-A may manipulate host behavior to facilitate horizontal transmission.
DOI: 10.6084/m9.figshare.8341187.v3
title: Molecular impact of Deformed wing virus type A in the honey bee brain.

description: Crop pollination by the western honey bee <i>Apis mellifera</i> is vital to agriculture but threatened by alarmingly high levels of colony mortality, especially in Europe and the US. Colony loss is due, in part, to the high viral loads of Deformed wing virus (DWV), transmitted by the ectoparasitic mite <i>Varroa destructor, </i>especially throughout the overwintering period of a honey bee colony. Covert DWV infection is commonplace and has been causally linked to precocious foraging, which itself has been linked to colony loss. Taking advantage of four brain transcriptome studies that unexpectedly revealed evidence of covert DWV-A infection, we set out to explore whether this effect is due to DWV-A mimicking naturally occurring changes in brain gene expression that are associated with behavioral maturation. Consistent with this hypothesis, we found that brain gene expression profiles of DWV-A infected bees resembled those of foragers, even in samples that were much younger than typical foragers. In addition, transcriptional regulatory network analysis revealed a positive association between DWV-A infection and transcription factors previously associated with honey bee foraging behavior. Surprisingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively few glial cells in the insect brain and they are rarely associated with behavioral plasticity. Covert DWV-A infection also has been linked to impaired learning, which together with precocious foraging can lead to increased occurrence of bees from one colony mistakenly joining another, especially under crowded modern apiary conditions. Together, these findings support a mechanistic link by which DWV-A may manipulate host behavior to facilitate horizontal transmission.
DOI: 10.6084/m9.figshare.8341370.v5
title: Institution information integration dataset of open data

description: integration data of institution informaiton based on grid , orgref, Open ISNI, VIAF and wikidata.<br>
DOI: 10.6084/m9.figshare.8341187.v4
title: Molecular impact of Deformed wing virus type A in the honey bee brain.

description: Crop pollination by the western honey bee <i>Apis mellifera</i> is vital to agriculture but threatened by alarmingly high levels of colony mortality, especially in Europe and the US. Colony loss is due, in part, to the high viral loads of Deformed wing virus (DWV), transmitted by the ectoparasitic mite <i>Varroa destructor, </i>especially throughout the overwintering period of a honey bee colony. Covert DWV infection is commonplace and has been causally linked to precocious foraging, which itself has been linked to colony loss. Taking advantage of four brain transcriptome studies that unexpectedly revealed evidence of covert DWV-A infection, we set out to explore whether this effect is due to DWV-A mimicking naturally occurring changes in brain gene expression that are associated with behavioral maturation. Consistent with this hypothesis, we found that brain gene expression profiles of DWV-A infected bees resembled those of foragers, even in samples that were much younger than typical foragers. In addition, transcriptional regulatory network analysis revealed a positive association between DWV-A infection and transcription factors previously associated with honey bee foraging behavior. Surprisingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively few glial cells in the insect brain and they are rarely associated with behavioral plasticity. Covert DWV-A infection also has been linked to impaired learning, which together with precocious foraging can lead to increased occurrence of bees from one colony mistakenly joining another, especially under crowded modern apiary conditions. Together, these findings support a mechanistic link by which DWV-A may manipulate host behavior to facilitate horizontal transmission.
DOI: 10.6084/m9.figshare.8341187.v5
title: Molecular impact of Deformed wing virus type A in the honey bee brain.

description: Crop pollination by the western honey bee <i>Apis mellifera</i> is vital to agriculture but threatened by alarmingly high levels of colony mortality, especially in Europe and the US. Colony loss is due, in part, to the high viral loads of Deformed wing virus (DWV), transmitted by the ectoparasitic mite <i>Varroa destructor, </i>especially throughout the overwintering period of a honey bee colony. Covert DWV infection is commonplace and has been causally linked to precocious foraging, which itself has been linked to colony loss. Taking advantage of four brain transcriptome studies that unexpectedly revealed evidence of covert DWV-A infection, we set out to explore whether this effect is due to DWV-A mimicking naturally occurring changes in brain gene expression that are associated with behavioral maturation. Consistent with this hypothesis, we found that brain gene expression profiles of DWV-A infected bees resembled those of foragers, even in samples that were much younger than typical foragers. In addition, transcriptional regulatory network analysis revealed a positive association between DWV-A infection and transcription factors previously associated with honey bee foraging behavior. Surprisingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively few glial cells in the insect brain and they are rarely associated with behavioral plasticity. Covert DWV-A infection also has been linked to impaired learning, which together with precocious foraging can lead to increased occurrence of bees from one colony mistakenly joining another, especially under crowded modern apiary conditions. Together, these findings support a mechanistic link by which DWV-A may manipulate host behavior to facilitate horizontal transmission.
DOI: 10.6084/m9.figshare.8341187.v6
title: Molecular impact of Deformed wing virus type A in the honey bee brain.

description: Crop pollination by the western honey bee <i>Apis mellifera</i> is vital to agriculture but threatened by alarmingly high levels of colony mortality, especially in Europe and the US. Colony loss is due, in part, to the high viral loads of Deformed wing virus (DWV), transmitted by the ectoparasitic mite <i>Varroa destructor, </i>especially throughout the overwintering period of a honey bee colony. Covert DWV infection is commonplace and has been causally linked to precocious foraging, which itself has been linked to colony loss. Taking advantage of four brain transcriptome studies that unexpectedly revealed evidence of covert DWV-A infection, we set out to explore whether this effect is due to DWV-A mimicking naturally occurring changes in brain gene expression that are associated with behavioral maturation. Consistent with this hypothesis, we found that brain gene expression profiles of DWV-A infected bees resembled those of foragers, even in samples that were much younger than typical foragers. In addition, transcriptional regulatory network analysis revealed a positive association between DWV-A infection and transcription factors previously associated with honey bee foraging behavior. Surprisingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively few glial cells in the insect brain and they are rarely associated with behavioral plasticity. Covert DWV-A infection also has been linked to impaired learning, which together with precocious foraging can lead to increased occurrence of bees from one colony mistakenly joining another, especially under crowded modern apiary conditions. Together, these findings support a mechanistic link by which DWV-A may manipulate host behavior to facilitate horizontal transmission.
DOI: 10.7483/opendata.cms.nbje.c4yj
title: /BTag/Run2011B-12Oct2013-v1/AOD
DOI: 10.6084/m9.figshare.8341187.v7
title: Molecular impact of Deformed wing virus type A in the honey bee brain.

description: Crop pollination by the western honey bee <i>Apis mellifera</i> is vital to agriculture but threatened by alarmingly high levels of colony mortality, especially in Europe and the US. Colony loss is due, in part, to the high viral loads of Deformed wing virus (DWV), transmitted by the ectoparasitic mite <i>Varroa destructor, </i>especially throughout the overwintering period of a honey bee colony. Covert DWV infection is commonplace and has been causally linked to precocious foraging, which itself has been linked to colony loss. Taking advantage of four brain transcriptome studies that unexpectedly revealed evidence of covert DWV-A infection, we set out to explore whether this effect is due to DWV-A mimicking naturally occurring changes in brain gene expression that are associated with behavioral maturation. Consistent with this hypothesis, we found that brain gene expression profiles of DWV-A infected bees resembled those of foragers, even in samples that were much younger than typical foragers. In addition, transcriptional regulatory network analysis revealed a positive association between DWV-A infection and transcription factors previously associated with honey bee foraging behavior. Surprisingly, single-cell RNA-Sequencing implicated glia, not neurons, in this effect; there are relatively few glial cells in the insect brain and they are rarely associated with behavioral plasticity. Covert DWV-A infection also has been linked to impaired learning, which together with precocious foraging can lead to increased occurrence of bees from one colony mistakenly joining another, especially under crowded modern apiary conditions. Together, these findings support a mechanistic link by which DWV-A may manipulate host behavior to facilitate horizontal transmission.
DOI: 10.6084/m9.figshare.8341037
title: Critical current characterisation of AMSC Amperium® Type 8502-350 coil formulation 2G HTS superconducting wire

description: Detailed measurements of the critical current of a commercial 2G HTS REBCO wire as a function of temperature, magnetic field, and field angle, performed at the Robinson Research Institute of Victoria University of Wellington.
DOI: 10.7483/opendata.cms.mcvj.1en6
title: /DoubleElectron/Run2011B-12Oct2013-v1/AOD
DOI: 10.6084/m9.figshare.8340920
title: Datasets -Pruning by Acacia Ants

description: Data for the paper entitled: "Plant killing by Neotropical acacia ants: ecology, decision-making and head morphology". It includes four excel files: 1. Info of the plant parts where three different Neotropical acacia ants pruned herbaceous and woody saplings; 2. The measurements of thickness of different plant parts of the grass nodes; 3. The thickness of different plant parts of the woody saplings; 4. the measurements of head length, head width, head broadness(lenght to width) for seven species of <i>Pseudomyrmex</i> ants with different types of associations with plants, and different demands on mandible force.
DOI: 10.7483/opendata.cms.hr3i.ortl
title: /DoubleMu/Run2011B-12Oct2013-v1/AOD
DOI: 10.6084/m9.figshare.8340920.v1
title: Datasets -Pruning by Acacia Ants

description: Data for the paper entitled: "Plant killing by Neotropical acacia ants: ecology, decision-making and head morphology". It includes four excel files: 1. Info of the plant parts where three different Neotropical acacia ants pruned herbaceous and woody saplings; 2. The measurements of thickness of different plant parts of the grass nodes; 3. The thickness of different plant parts of the woody saplings; 4. the measurements of head length, head width, head broadness(lenght to width) for seven species of <i>Pseudomyrmex</i> ants with different types of associations with plants, and different demands on mandible force.
DOI: 10.6084/m9.figshare.8340899
title: A Dataset for Estimating Trust between Open Source Software Developers

description: This dataset consists of data from 179 Python projects on GitHub used for estimating trust between open source software developers. <br>The data includes (1) pull request data for each project, (2) manual annotations for a subset of pull requests, and (3) computed trust values between pull request generator and members of the corresponding project for a set of accepted and rejected pull requests.
DOI: 10.6084/m9.figshare.8340860
title: A study of knowledge, experience and beliefs about Hepatitis B virus infection in south western Uganda

description: The files here represent the metadata underpinning a study to investigate knowledge, experience and beliefs surrounding hepatitis b virus (HBV) infection in south-west Uganda.<br><b>S1 Table: </b>Data extraction summarising responses to discussion about knowledge, experience and beliefs about Hepatitis B virus infection from 131 study participants in Uganda. Table summarises feedback from individuals and group discussions. <br><b>S2 Tables:</b> Common themes obtained from analysing responses obtained from study participants participating in discussion about knowledge, experience and beliefs about Hepatitis B virus infection in Uganda. These represent responses from all individuals represented in the study. Note there is not a defined denominator, as there were differing approaches to approach discussion with study participants. <br>The study was approved by the Science and Ethics Committee of the Uganda Virus Research Institute (ref: GC/127/18/05/645 and the Oxford Tropical Research Ethics Committee (ref: 516-18).
DOI: 10.6084/m9.figshare.8340842
title: Sequencing Data

description: Raw sequencing data in bam format. No modification have been done on the files except that they are split to the IonXpress index in the forward primer.<br>