cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.15468/dl.j8w8wk
title: Occurrence Download

description: A dataset containing 0 species occurrences available in GBIF matching the query:
{
 "and" : [
 "TaxonKey is one of (Caledoconcha carnosa Haase & Bouchet, 1998)",
 "OccurrenceStatus is Present",
 "Year is greater than or equal to 2000",
 "BasisOfRecord is one of (Human Observation, Observation, Machine Observation)",
 "HasCoordinate is true",
 "HasGeospatialIssue is false",
 {
 "or" : [
 "CoordinateUncertaintyInMeters is less than 100",
 "CoordinateUncertaintyInMeters is null"
 ]
 }
 ]
}

 The dataset includes 0 records from 0 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0093580-240626123714530/datasets/export for details.
DOI: 10.15468/dl.fh6kd8
title: Occurrence Download

description: A dataset containing 9235741 species occurrences available in GBIF matching the query:
{
 "and" : [
 "Country is China",
 "HasCoordinate is true",
 "HasGeospatialIssue is false",
 "OccurrenceStatus is Present"
 ]
}

 The dataset includes 9235741 records from 2997 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0093558-240626123714530/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.15468/dl.44y78h
title: Occurrence Download

description: A dataset containing 6 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Memecylon isaloense Jacq.-Fél."
 ]
}

 The dataset includes 6 records from 2 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0056114-240321170329656/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.5281/zenodo.10894921
title: What are the Leadership and Management Positions in Medical Education? A Scoping Review

description: Dataset Info

1) Supplementary Material

# The first row of the dataset identifies the columns.# The first column represents the article IDs.# The second column represents the titles of the articles.# The third column represents the authors of the articles.# The fourth column represents the years the articles were published.# The fifth column represents the country of the corresponding author.# The sixth column represents  the type of study conducted.# The seventh column represents the positions identified from that article.# The eighth column represents the discipline researched in that article.# The ninth column represents the context of the article.
DOI: 10.5281/zenodo.10894922
title: What are the Leadership and Management Positions in Medical Education? A Scoping Review

description: Dataset Info

1) Supplementary Material

# The first row of the dataset identifies the columns.# The first column represents the article IDs.# The second column represents the titles of the articles.# The third column represents the authors of the articles.# The fourth column represents the years the articles were published.# The fifth column represents the country of the corresponding author.# The sixth column represents  the type of study conducted.# The seventh column represents the positions identified from that article.# The eighth column represents the discipline researched in that article.# The ninth column represents the context of the article.
DOI: 10.15468/dl.hp44pw
title: Occurrence Download

description: A dataset containing 1352559 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Dactylis glomerata L."
 ]
}

 The dataset includes 1352559 records from 4769 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0056055-240321170329656/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.6084/m9.figshare.3394639.v68
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v69
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v70
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v71
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v72
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v73
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v74
title: How an ABO-incompatible graft may contribute to its survival by phenotype-specific glycosylation of the host. A hypothesis.

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v75
title: How an ABO-incompatible graft may be protected by phenotypespecific glycosylation of the host. A hypothesis

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v76
title: How an ABO-incompatible graft may be protected by phenotypespecific glycosylation of the host. A hypothesis

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v77
title: How an ABO-incompatible graft may be protected by phenotype-specific glycosylation of the host. A hypothesis

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after a HLA-incompatible liver transplantation without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v78
title: How an ABO-incompatible graft may be protected by phenotype-specific glycosylation of the host. A hypothesis

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after an incompatible liver transplantation, without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon likely represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.
DOI: 10.6084/m9.figshare.3394639.v79
title: How an ABO-incompatible graft may be protected by phenotype-specific glycosylation of the host. A hypothesis

description: In contrast to non-nucleated ABO(H)-incompatible red cells, which when transfused to an HLA-compatible blood group O(H) recipient undergo destruction within minutes, such hyperacute, humoral rejection occurs relatively rare in transplantations of highly nucleated, metabolically active solid organs, and it is extremely rare in liver transplantations (Adams 1991; Della-Guardia et al. 2008;). Moreover, a case of selective disappearance of preexisting donor-specific HLA-antibodies after an incompatible liver transplantation, without any rejection episodes, has been reported by Bastiani (2006), and according to Taner et al. (2014), such decrease of donor-specific HLA-antibodies is not uncommon after liver transplantation. While this phenomenon likely represents a metabolic achievement of the graft in overcoming one of the mechanism of rejection, respective observations on anti-A/B-isoagglutinins are missing, because they are always removed before transplantation. It appears to be established that tissue transplants always maintain their original, phenotype-specific metabolic properties, and expanding the concept of “glycosidic exclusion”, a transplanted ABO(H)-incompatible, metabolically active tissue may use its phenotype-specific enzymatic equipment to contribute to a compatible environment by consistent glycosylation of complementary sites of the plasma proteins and the differentiating B-cell surfaces of a HLA-compatible recipient.