cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.25452/figshare.plus.29998021.v1
title: Supporting data for: <b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during </b><b><i>Toxoplasma gondii</i></b><b> </b><b>infection</b> (Fas LPR data)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.<br>This item contains raw data that pertains to Fas-/- (LPR) mice. Data includes various time points post T. gondii infection and include flow cytometry based cell population quantifications, RT-PCR quantifications, and parasite burden quantifications. Information regarding date collected, timepoint, sex, and data type can be found in the file title.
DOI: 10.25346/s6eecgix/bhqyqd
title: msXL_322v_31_23.jpg
DOI: 10.25346/s6eecgix/0qmhqf
title: msXL_322v_31_22.jpg
DOI: 10.25346/s6eecgix/1vudzf
title: msXL_322v_31_21.jpg
DOI: 10.25346/s6eecgix/moewts
title: msXL_322v_31_20.jpg
DOI: 10.25346/s6eecgix/rnzaxc
title: msXL_322v_31_19.jpg
DOI: 10.25346/s6eecgix/s3ckqa
title: msXL_322v_31_18.jpg
DOI: 10.25346/s6eecgix/lbqgs1
title: msXL_322v_31_17.jpg
DOI: 10.25452/figshare.plus.30005716
title: Supporting data for: <b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during </b><b><i>Toxoplasma gondii</i></b><b> </b><b>infection</b> (Casp8Ripk3 KO data)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.This item contains raw data that pertains to Casp8-/-Ripk3-/- mice. Data ranges from naive to various time points post T. gondii infection and include flow cytometry based cell population quantifications, RT-PCR quantifications, ELISA cytokine measurements, and parasite burden quantifications. Information regarding date collected, timepoint, sex, and data type can be found in the file title.
DOI: 10.25452/figshare.plus.30005716.v1
title: Supporting data for: <b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during </b><b><i>Toxoplasma gondii</i></b><b> </b><b>infection</b> (Casp8Ripk3 KO data)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.This item contains raw data that pertains to Casp8-/-Ripk3-/- mice. Data ranges from naive to various time points post T. gondii infection and include flow cytometry based cell population quantifications, RT-PCR quantifications, ELISA cytokine measurements, and parasite burden quantifications. Information regarding date collected, timepoint, sex, and data type can be found in the file title.
DOI: 10.25346/s6eecgix/o3xb63
title: msXL_322v_31_16.jpg
DOI: 10.25346/s6eecgix/gmcnsu
title: msXL_322v_31_15.jpg
DOI: 10.25346/s6eecgix/qyyl3v
title: msXL_322v_31_14.jpg
DOI: 10.25346/s6eecgix/vvvvb8
title: msXL_322v_31_13.jpg
DOI: 10.25346/s6eecgix/1zgryt
title: msXL_322v_15_12.jpg
DOI: 10.25346/s6eecgix/r1tdsq
title: msXL_322v_15_11.jpg
DOI: 10.25346/s6eecgix/nu44ey
title: msXL_322v_15_10.jpg
DOI: 10.25452/figshare.plus.30076663
title: Supporting data for: <b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during </b><b><i>Toxoplasma gondii</i></b><b> </b><b>infection</b> (MERFISH naive)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.<br>This item contains data for MERFISH (Multiplexed Error-Robust Fluorescence In Situ Hybridization), a single-molecule imaging technique that allows for spatially mapping of single nuclear transcripts. This item contains data for a naive brain including raw data, cell clustering script, cell by gene identification, and detected transcripts.
DOI: 10.25452/figshare.plus.30076663.v1
title: Supporting data for: <b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during </b><b><i>Toxoplasma gondii</i></b><b> </b><b>infection</b> (MERFISH naive)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.<br>This item contains data for MERFISH (Multiplexed Error-Robust Fluorescence In Situ Hybridization), a single-molecule imaging technique that allows for spatially mapping of single nuclear transcripts. This item contains data for a naive brain including raw data, cell clustering script, cell by gene identification, and detected transcripts.
DOI: 10.25346/s6eecgix/zhdny4
title: msXL_322v_15_09.jpg
DOI: 10.25346/s6eecgix/02sarw
title: msXL_322v_15_08.jpg
DOI: 10.25346/s6eecgix/qmfmcq
title: msXL_322v_15_07.jpg
DOI: 10.25346/s6eecgix/kcjtn4
title: msXL_322v_15_06.jpg
DOI: 10.25346/s6eecgix/7syiiw
title: msXL_322v_15_05.jpg
DOI: 10.25346/s6eecgix/y2ozfq
title: msXL_322v_15_04.jpg
DOI: 10.25346/s6eecgix/acyp7m
title: msXL_322v_15_03.jpg
DOI: 10.25346/s6eecgix/qv9trq
title: msXL_322v_15_02.jpg
DOI: 10.15468/dl.gz5r6y
title: Occurrence Download

description: A dataset containing 5334 species occurrences available in GBIF matching the query:
{
 "and" : [
 "Country is Costa Rica",
 "Geometry POLYGON((-83.7455 9.50226,-83.67471 9.50226,-83.67471 9.54203,-83.7455 9.54203,-83.7455 9.50226))",
 "OccurrenceStatus is Present",
 "Year 2015-2025"
 ]
}

 The dataset includes 5334 records from 6 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0016411-250914085247600/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.25346/s6eecgix/kenkk5
title: msXL_322v_15_01.jpg
DOI: 10.25346/s6eecgix/bwl65z
title: msXL_322v_0_ring.jpg
DOI: 10.25346/s6eecgix/1tocas
title: msXL_322v.jpg
DOI: 10.25346/s6eecgix/uw9uvl
title: LEGGIMI_IT.txt
DOI: 10.25346/s6eecgix/vqwoyf
title: msXL_334r_31_15.tif
DOI: 10.25346/s6eecgix/oaubm3
title: msXL_334r_31_14.tif
DOI: 10.25346/s6eecgix/38rcrk
title: msXL_334r_31_13.tif
DOI: 10.25452/figshare.plus.30080899
title: Supporting data for: <b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during </b><b><i>Toxoplasma gondii </i></b><b>infection </b>(MERFISH infected)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.This item contains data for MERFISH (Multiplexed Error-Robust Fluorescence In Situ Hybridization), a single-molecule imaging technique that allows for spatially mapping of single nuclear transcripts. This item contains data for a T. gondii infected brain at 28 dpi including raw data, cell clustering script, cell by gene identification, and detected transcripts.
DOI: 10.25452/figshare.plus.30080899.v1
title: Supporting data for: <b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during </b><b><i>Toxoplasma gondii </i></b><b>infection </b>(MERFISH infected)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.This item contains data for MERFISH (Multiplexed Error-Robust Fluorescence In Situ Hybridization), a single-molecule imaging technique that allows for spatially mapping of single nuclear transcripts. This item contains data for a T. gondii infected brain at 28 dpi including raw data, cell clustering script, cell by gene identification, and detected transcripts.
DOI: 10.25346/s6eecgix/4cu50z
title: msXL_334r_15_12.tif
DOI: 10.25346/s6eecgix/ldtjyf
title: msXL_334r_15_11.tif
DOI: 10.25346/s6eecgix/ihkkue
title: msXL_334r_15_09.tif
DOI: 10.25346/s6eecgix/x0aoyx
title: msXL_334r_15_08.tif
DOI: 10.25346/s6eecgix/setlfz
title: msXL_334r_15_07.tif
DOI: 10.25452/figshare.plus.30151057
title: Supporting data for:<b> </b><b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during</b><b><i> </i></b><b><i>Toxoplasma gondii</i></b><b> </b><b>infection</b> (Casp8Ripk3 KO Images)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.<br>This item contains immunohistochemistry images that pertain to the Casp8-/-Ripk3-/- experiments. The images are of brain sagittal sections. Information regarding genotype, antibody, and magnification can all be found in the title of the file.
DOI: 10.25452/figshare.plus.30151057.v1
title: Supporting data for:<b> </b><b>Caspase-8 expression in CD8</b><sup><strong>+ </strong></sup><b>T cells promotes pathogen restriction in the brain during</b><b><i> </i></b><b><i>Toxoplasma gondii</i></b><b> </b><b>infection</b> (Casp8Ripk3 KO Images)

description: Cell death is an integral restriction mechanism against intracellular pathogens. We have previously reported extensive cell death in the brain during infection with the intracellular parasite, <i>Toxoplasma gondii.</i> Here we focus on the role of caspase-8, a regulator of extrinsic apoptosis, during <i>T. gondii </i>infection. We find that <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice have increased brain parasite burden in comparison to controls and succumb to infection, despite the generation of robust immune responses. We observed that neurons, astrocytes, and CD8<sup>+</sup> T cells had high rates of parasite interactions in <i>Casp8</i><sup><em>-/-</em></sup><i>Ripk3</i><sup><em>-/-</em></sup> mice compared to WT mice. While <i>Casp8 </i>deficiency in neurons and astrocytes did not impact control of infection, deletion of <i>Casp8</i> in CD8<sup>+</sup> T cells led to impaired survival, increased parasite burden and direct infection of CD8<sup>+</sup> T cells in the brain. We conclude that in addition to well-characterized effector functions, CD8<sup>+</sup> T cells utilize caspase-8 to control <i>T. gondii</i> in the brain.<br>This item contains immunohistochemistry images that pertain to the Casp8-/-Ripk3-/- experiments. The images are of brain sagittal sections. Information regarding genotype, antibody, and magnification can all be found in the title of the file.
DOI: 10.25346/s6eecgix/lfts1p
title: msXL_334r_15_06.tif
DOI: 10.25346/s6eecgix/th73bi
title: msXL_334r_15_05.tif
DOI: 10.25346/s6eecgix/qr6er5
title: msXL_334r_15_04.tif
DOI: 10.25346/s6eecgix/dqtwit
title: msXL_334r_15_03.tif
DOI: 10.25346/s6eecgix/8yf9qe
title: msXL_334r_15_02.tif
DOI: 10.25346/s6eecgix/yslt1z
title: msXL_334r_15_01.tif
DOI: 10.15468/dl.yhaqcu
title: Occurrence Download

description: A dataset containing 2 species occurrences available in GBIF matching the query:
{
 "and" : [
 "TaxonKey is Delphinium cultorum",
 "Country is Austria",
 "HasCoordinate is true"
 ]
}

 The dataset includes 2 records from 2 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0016412-250914085247600/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.25346/s6eecgix/05exrw
title: msXL_334r_0_ring.tif
DOI: 10.25346/s6eecgix/vzlcv9
title: msXL_334r.tif
DOI: 10.25346/s6eecgix/nolnqo
title: plane_8.jpg
DOI: 10.25346/s6eecgix/mtakbo
title: plane_7.jpg
DOI: 10.25346/s6eecgix/rtivcq
title: plane_6.jpg
DOI: 10.25346/s6eecgix/zinb57
title: plane_5.jpg
DOI: 10.25346/s6eecgix/hfaslq
title: plane_4.jpg
DOI: 10.25346/s6eecgix/twvfua
title: plane_3.jpg
DOI: 10.25346/s6eecgix/yxyrri
title: plane_2.jpg
DOI: 10.25346/s6eecgix/98nb8d
title: plane_1.jpg
DOI: 10.25346/s6eecgix/gbvcx8
title: plane_0.jpg
DOI: 10.25346/s6eecgix/q5tz7l
title: info.json
DOI: 10.25346/s6eecgix/tnhedw
title: msXL_334r.relight
DOI: 10.25346/s6eecgix/azlw8a
title: msXL_334r_65_49.jpg
DOI: 10.25346/s6eecgix/m6prnd
title: msXL_334r_65_48.jpg
DOI: 10.25346/s6eecgix/dwalmo
title: msXL_334r_65_47.jpg
DOI: 10.25346/s6eecgix/kei0dl
title: msXL_334r_65_46.jpg
DOI: 10.25346/s6eecgix/d43uk7
title: msXL_334r_65_45.jpg
DOI: 10.25346/s6eecgix/1hia9l
title: msXL_334r_65_44.jpg
DOI: 10.25346/s6eecgix/q7knuk
title: msXL_334r_65_43.jpg
DOI: 10.25346/s6eecgix/ajouyh
title: msXL_334r_65_42.jpg
DOI: 10.25346/s6eecgix/xvkjls
title: msXL_334r_65_41.jpg
DOI: 10.25346/s6eecgix/nglhwt
title: msXL_334r_65_40.jpg
DOI: 10.25346/s6eecgix/t5skq5
title: msXL_334r_65_39.jpg
DOI: 10.25346/s6eecgix/5vmx3o
title: msXL_334r_65_38.jpg
DOI: 10.25346/s6eecgix/wwuvgj
title: msXL_334r_46_37.jpg
DOI: 10.25346/s6eecgix/gajvyk
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DOI: 10.25346/s6eecgix/y5wpmz
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DOI: 10.25346/s6eecgix/cdjm1v
title: msXL_334r_46_34.jpg
DOI: 10.25346/s6eecgix/awmzll
title: msXL_334r_46_33.jpg
DOI: 10.25346/s6eecgix/tlrzdu
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DOI: 10.25346/s6eecgix/korq8e
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DOI: 10.25346/s6eecgix/tggwvr
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DOI: 10.25346/s6eecgix/5auiuc
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DOI: 10.25346/s6eecgix/bjcwpa
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DOI: 10.25346/s6eecgix/4jiq1e
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DOI: 10.25346/s6eecgix/onkc48
title: msXL_334r_46_26.jpg
DOI: 10.25346/s6eecgix/mhwjug
title: msXL_334r_46_25.jpg
DOI: 10.25346/s6eecgix/hybgco
title: msXL_334r_31_24.jpg
DOI: 10.25346/s6eecgix/tunyh8
title: msXL_334r_31_23.jpg
DOI: 10.25346/s6eecgix/oijkhg
title: msXL_334r_31_22.jpg
DOI: 10.25346/s6eecgix/2qdnmn
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DOI: 10.25346/s6eecgix/qoatjg
title: msXL_334r_31_20.jpg
DOI: 10.25346/s6eecgix/1uupo5
title: msXL_334r_31_19.jpg
DOI: 10.25346/s6eecgix/gtc8u1
title: msXL_334r_31_18.jpg
DOI: 10.25346/s6eecgix/0ri8xi
title: msXL_334r_31_17.jpg
DOI: 10.25346/s6eecgix/wfhlxy
title: msXL_334r_31_16.jpg
DOI: 10.25346/s6eecgix/1hdtin
title: msXL_334r_31_15.jpg
DOI: 10.25346/s6eecgix/xuiobk
title: msXL_334r_31_14.jpg
DOI: 10.25346/s6eecgix/3dh7ic
title: msXL_334r_31_13.jpg
DOI: 10.25346/s6eecgix/1i39c6
title: msXL_334r_15_12.jpg
DOI: 10.25346/s6eecgix/yigthb
title: msXL_334r_15_11.jpg
DOI: 10.25346/s6eecgix/lbw7s4
title: msXL_334r_15_10.jpg
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