cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.4315049
title: Toxicity Assays Sargassum Data

description: These Excel files are the raw data output from the toxicity assays for the brown alga <i>Sargassum mangarevense. </i>Each Excel file includes the data from four blank wells (A1, A6, B1, B6), two positive control wells (A2, A3), two negative control wells (B2, B3), and two toxin assay wells (A4, A5). Analysis happens through an R script uploaded elsewhere.
DOI: 10.6084/m9.figshare.4315049.v1
title: Toxicity Assays Sargassum Data

description: These Excel files are the raw data output from the toxicity assays for the brown alga <i>Sargassum mangarevense. </i>Each Excel file includes the data from four blank wells (A1, A6, B1, B6), two positive control wells (A2, A3), two negative control wells (B2, B3), and two toxin assay wells (A4, A5). Analysis happens through an R script uploaded elsewhere.
DOI: 10.6084/m9.figshare.4315046
title: Toxicity Assays Boodlea Data

description: These Excel files are the raw data output from the toxicity assays for the green alga <i>Boodlea kaeneana</i><i>. </i>Each Excel file includes the data from four blank wells (A1, A6, B1, B6), two positive control wells (A2, A3), two negative control wells (B2, B3), and two toxin assay wells (A4, A5). Analysis happens through an R script uploaded elsewhere.
DOI: 10.4225/03/58507ddae3022
title: Duffy_Chown_MEPS12011_model_virtualspecies

description: R function and example data to perform simple 2.5D-SDM as discussed in Duffy & Chown, 'Explicitly integrating a third dimension in marine species distribution modelling', Marine Ecology Progress Series, doi: 10.3354/meps12011.
DOI: 10.6084/m9.figshare.4315046.v1
title: Toxicity Assays Boodlea Data

description: These Excel files are the raw data output from the toxicity assays for the green alga <i>Boodlea kaeneana</i><i>. </i>Each Excel file includes the data from four blank wells (A1, A6, B1, B6), two positive control wells (A2, A3), two negative control wells (B2, B3), and two toxin assay wells (A4, A5). Analysis happens through an R script uploaded elsewhere.
DOI: 10.6084/m9.figshare.4315040
title: Paired Sargassum Data

description: This is the raw data as collected from the Presens respirometer. Each day has its own Excel file, and each Excel file has two blank wells (A1 and A6), two control wells (A2 and A3) and two treatment wells (A4 and A5). R code for data collection is uploaded elsewhere.
DOI: 10.6084/m9.figshare.4315043
title: R code used for Pairing Experiment

description: All collected was processed in R. The scripts here collect data from various sources and collate them into useful Excel files using the R package xlsx
DOI: 10.6084/m9.figshare.4315040.v1
title: Paired Sargassum Data

description: This is the raw data as collected from the Presens respirometer. Each day has its own Excel file, and each Excel file has two blank wells (A1 and A6), two control wells (A2 and A3) and two treatment wells (A4 and A5). R code for data collection is uploaded elsewhere.
DOI: 10.6084/m9.figshare.4315043.v1
title: R code used for Pairing Experiment

description: All collected was processed in R. The scripts here collect data from various sources and collate them into useful Excel files using the R package xlsx
DOI: 10.6084/m9.figshare.4315028
title: Paired Boodlea Data

description: This is the raw data as collected from the Presens respirometer. Each day has its own Excel file, and each Excel file has two blank wells (A1 and A6), two control wells (A2 and A3) and two treatment wells (A4 and A5). R code for data collection is uploaded elsewhere.
DOI: 10.6084/m9.figshare.4315028.v1
title: Paired Boodlea Data

description: This is the raw data as collected from the Presens respirometer. Each day has its own Excel file, and each Excel file has two blank wells (A1 and A6), two control wells (A2 and A3) and two treatment wells (A4 and A5). R code for data collection is uploaded elsewhere.
DOI: 10.6084/m9.figshare.4312604
title: Sour Rot Paper Wasp Microbial Community

description: Unrarefied OTU table w/Tax ITS: otu_table_wTax_noUnFunP.....biom<br>Rarefied OTU table w/Tax ITS: otu_table_wTax_noUnFunP...even....biom<br>Metadata file for OTU table ITS: ITS_MappingFile....txt<br>OTU representative sequences ITS: rep_set_numbered_ITS.fa<br>Unrarefied OTU table w/Tax 16S: otu_table_wTax_noUnChloroMito...biom<br>Rarefied OTU table w/Tax 16S: otu_table_wTax_noUnChloroMito...even...biom<br>Metadata file for OTU table 16S: 16SMapping....txt<br>OTU representative sequences 16S: rep_set_numbered_16S.fa<br><br>
DOI: 10.6084/m9.figshare.4312604.v1
title: Sour Rot Paper Wasp Microbial Community

description: Unrarefied OTU table w/Tax ITS: otu_table_wTax_noUnFunP.....biom<br>Rarefied OTU table w/Tax ITS: otu_table_wTax_noUnFunP...even....biom<br>Metadata file for OTU table ITS: ITS_MappingFile....txt<br>OTU representative sequences ITS: rep_set_numbered_ITS.fa<br>Unrarefied OTU table w/Tax 16S: otu_table_wTax_noUnChloroMito...biom<br>Rarefied OTU table w/Tax 16S: otu_table_wTax_noUnChloroMito...even...biom<br>Metadata file for OTU table 16S: 16SMapping....txt<br>OTU representative sequences 16S: rep_set_numbered_16S.fa<br><br>
DOI: 10.1285/i24212113v6i2-1p128
title: Towards a community clinical psychology? Insights from a systematic review of peer-reviewed literature

description: The present systematic review provides a narrative synthesis of the peer-reviewed literature concerning the synergy between community and clinical psychology, with the specific aim of detecting the theoretical, research, and practical basis for the development of a well-integrated community clinical psychology perspective. The authors screened 216 records on the topic, found in major citation databases (PsycArticles, PsycINFO, Scopus, and Web of Science) without time or language restrictions. Six articles addressing the review question were identified and examined through seven conceptual criteria referred to contextual premises, definitions, addressed problems/issues, application areas, reference theoretical models, examined constructs, and required professional competences. The results do not show evidence for a well-integrated community clinical psychological perspective, despite the many synergies between such disciplines on a theoretical and applied level. Community and clinical psychology could thus be further integrated in the future, potentially giving rise to a new and independent field of knowledge to get more comprehensive understanding of the relationship between individuals and social contexts.
DOI: 10.6084/m9.figshare.4312469
title: ICASSP2017 publication - Experimental results for DNN approach with speaker channels

description: Results in RTTM format for the experiments performed in the ICASSP 2017 paper titled 'DNN approach to speaker diarisation using speaker channels'<br>
DOI: 10.6084/m9.figshare.4312469.v1
title: ICASSP2017 publication - Experimental results for DNN approach with speaker channels

description: Results in RTTM format for the experiments performed in the ICASSP 2017 paper titled 'DNN approach to speaker diarisation using speaker channels'<br>
DOI: 10.6084/m9.figshare.4312595
title: Gulf SAFE data.xlsx

description: Sample data of sex differences in Atrial fibrillation patients in Gulf region
DOI: 10.6084/m9.figshare.4312595.v1
title: Gulf SAFE data.xlsx

description: Sample data of sex differences in Atrial fibrillation patients in Gulf region
DOI: 10.6084/m9.figshare.4312382
title: PowerPoint Slides for: Pulmonary Hypertension Is Associated with Mortality and Cardiovascular Events in Chronic Kidney Disease Patients

description: <b><i>Background:</i></b> We evaluated the prevalence, determinants and prognosis value of pulmonary hypertension (PH) in non-dialysis chronic kidney disease (CKD) patients. <b><i>Methods:</i></b> This is a prospective study with stages 3-5 non-dialysis-dependent CKD patients. PH was estimated by Doppler echocardiography and defined as a pulmonary artery systolic pressure above 35 mm Hg. <b><i>Results:</i></b> Three hundred fifty-three patients were recruited, of whom 94 (26.6%) had PH. Prevalence of PH increased with the decline of renal function: 21.6, 24.1, and 31.7% in stages 3, 4, and 5, respectively. Independent predictors of PH were age, estimated glomerular filtration rate (eGFR), history of cardiovascular (CV) events, the presence of an arteriovenous fistulae (AVF), and left ventricular (systolic and diastolic) dysfunction. Over a median follow-up of 22 months, 71 patients died (20%). After multivariate adjustment for age, gender, previous CV disease, diastolic and systolic dysfunction, PH remained as an independent predictor of all-cause mortality (hazards ratio [HR] 1.84, 95% CI 1.06-3.18, <i>p</i> = 0.02). One hundred patients (28%) had a new onset CV event. After adjustment for age, gender, previous CV disease, systolic and diastolic dysfunction, PH maintains its independent association with CV events (HR 2.77, 95% CI 2.00-3.25, <i>p</i> &lt; 0.001). <b><i>Conclusions:</i></b> PH prevalence rises as kidney function declines. Main determinants of PH are age, eGFR, previous CV disease, the presence of an AVF and left ventricular systolic or diastolic dysfunction. PH is an independent predictor of all-cause mortality and CV events.
DOI: 10.6084/m9.figshare.4312382.v1
title: PowerPoint Slides for: Pulmonary Hypertension Is Associated with Mortality and Cardiovascular Events in Chronic Kidney Disease Patients

description: <b><i>Background:</i></b> We evaluated the prevalence, determinants and prognosis value of pulmonary hypertension (PH) in non-dialysis chronic kidney disease (CKD) patients. <b><i>Methods:</i></b> This is a prospective study with stages 3-5 non-dialysis-dependent CKD patients. PH was estimated by Doppler echocardiography and defined as a pulmonary artery systolic pressure above 35 mm Hg. <b><i>Results:</i></b> Three hundred fifty-three patients were recruited, of whom 94 (26.6%) had PH. Prevalence of PH increased with the decline of renal function: 21.6, 24.1, and 31.7% in stages 3, 4, and 5, respectively. Independent predictors of PH were age, estimated glomerular filtration rate (eGFR), history of cardiovascular (CV) events, the presence of an arteriovenous fistulae (AVF), and left ventricular (systolic and diastolic) dysfunction. Over a median follow-up of 22 months, 71 patients died (20%). After multivariate adjustment for age, gender, previous CV disease, diastolic and systolic dysfunction, PH remained as an independent predictor of all-cause mortality (hazards ratio [HR] 1.84, 95% CI 1.06-3.18, <i>p</i> = 0.02). One hundred patients (28%) had a new onset CV event. After adjustment for age, gender, previous CV disease, systolic and diastolic dysfunction, PH maintains its independent association with CV events (HR 2.77, 95% CI 2.00-3.25, <i>p</i> &lt; 0.001). <b><i>Conclusions:</i></b> PH prevalence rises as kidney function declines. Main determinants of PH are age, eGFR, previous CV disease, the presence of an AVF and left ventricular systolic or diastolic dysfunction. PH is an independent predictor of all-cause mortality and CV events.
DOI: 10.6084/m9.figshare.4312361
title: PowerPoint Slides for: Serum Bicarbonate Is Associated with Heart Failure in the Multi-Ethnic Study of Atherosclerosis

description: <b><i>Background:</i></b> Low serum bicarbonate concentrations are associated with mortality and kidney disease progression. Data regarding associations between bicarbonate and cardiovascular disease (CVD) are scarce. <b><i>Methods:</i></b> We performed a cohort study of 6,229 adult participants from the Multi-Ethnic Study of Atherosclerosis, a community-based cohort free of CVD at baseline. Serum bicarbonate was measured at baseline. Cardiovascular outcomes were defined as: (1) subclinical CVD (left ventricular mass [LVM] and aortic pulse pressure [PP] measured at baseline), (2) incident atherosclerotic cardiovascular events (CVE; composite of myocardial infarction, resuscitated cardiac arrest, stroke, coronary heart disease death, and stroke death), and (3) incident heart failure. <b><i>Results:</i></b> During a median (interquartile range) follow-up of 8.5 (7.7-8.6) years, 331 (5.3%) participants had an incident CVE and 174 (2.8%) developed incident heart failure. We stratified analyses by use of diuretics because we observed a significant interaction between diuretic use and bicarbonate with study outcomes. Among diuretic nonusers, with adjustment, bicarbonate ≥25 mEq/L was associated with an estimated 3.0 g greater LVM (95% CI 0.5-5.0) and 1.0 mm Hg higher aortic PP (95% CI 0.4-2.0) compared to bicarbonate 23-24 mEq/L. Each 1 mEq/L of bicarbonate increase was associated with a 13% higher risk of incident heart failure (hazards ratio 1.13, 95% CI 1.01-2.11). Among diuretic users, higher bicarbonate was not associated with CVD. Bicarbonate was not associated with incident atherosclerotic CVE irrespective of diuretic use. <b><i>Conclusion:</i></b> Among nonusers of diuretics in a large community-based study, higher serum bicarbonate concentrations are associated with subclinical CVD and new heart failure.
DOI: 10.6084/m9.figshare.4312379
title: time preferences and fisheries data --data for paper.dta

description: Data on individual time preferences and their relationship to natural resource extraction.
DOI: 10.6084/m9.figshare.4312379.v1
title: time preferences and fisheries data --data for paper.dta

description: Data on individual time preferences and their relationship to natural resource extraction.
DOI: 10.6084/m9.figshare.4312334
title: Supplementary Material for: The Crucial Role of C18-Cer in Fat-Induced Skeletal Muscle Insulin Resistance

description: <b><i>Background/Aims: </i></b>Muscle bioactive lipids accumulation leads to several disorder states. The most common are insulin resistance (IR) and type 2 diabetes. There is an ongoing debate which of the lipid species plays the major role in induction of muscle IR. Our aim was to elucidate the role of particular lipid group in induction of muscle IR. <b><i>Methods:</i></b> The analyses were performed on muscle from the following groups of rats: 1. Control, fed standard diet, 2 HFD, fed high fat diet, 3. HFD/Myr, fed HFD and treated with myriocin (Myr), an inhibitor of ceramide de novo synthesis. We utilized [U<sup>13</sup>C] palmitate isotope tracer infusion and mass spectrometry to measure content and synthesis rate of muscle long-chain acyl-CoA (LCACoA), diacylglycerols (DAG) and ceramide (Cer). <b><i>Results:</i></b> HFD led to intramuscular accumulation of LCACoA, DAG and Cer and skeletal muscle IR. Myr-treatment caused decrease in Cer (most noticeable for stearoyl-Cer and oleoyl-Cer) and accumulation of DAG, possibly due to re-channeling of excess of intramuscular LCACoA towards DAG synthesis. An improvement in insulin sensitivity at both systemic and muscular level coincided with decrease in ceramide, despite elevated intramuscular DAG. <b><i>Conclusion:</i></b> The improved insulin sensitivity was associated with decreased muscle stearoyl- and oleoyl-ceramide content. The results indicate that accumulation of those ceramide species has the greatest impact on skeletal muscle insulin sensitivity in rats.
DOI: 10.6084/m9.figshare.4312361.v1
title: PowerPoint Slides for: Serum Bicarbonate Is Associated with Heart Failure in the Multi-Ethnic Study of Atherosclerosis

description: <b><i>Background:</i></b> Low serum bicarbonate concentrations are associated with mortality and kidney disease progression. Data regarding associations between bicarbonate and cardiovascular disease (CVD) are scarce. <b><i>Methods:</i></b> We performed a cohort study of 6,229 adult participants from the Multi-Ethnic Study of Atherosclerosis, a community-based cohort free of CVD at baseline. Serum bicarbonate was measured at baseline. Cardiovascular outcomes were defined as: (1) subclinical CVD (left ventricular mass [LVM] and aortic pulse pressure [PP] measured at baseline), (2) incident atherosclerotic cardiovascular events (CVE; composite of myocardial infarction, resuscitated cardiac arrest, stroke, coronary heart disease death, and stroke death), and (3) incident heart failure. <b><i>Results:</i></b> During a median (interquartile range) follow-up of 8.5 (7.7-8.6) years, 331 (5.3%) participants had an incident CVE and 174 (2.8%) developed incident heart failure. We stratified analyses by use of diuretics because we observed a significant interaction between diuretic use and bicarbonate with study outcomes. Among diuretic nonusers, with adjustment, bicarbonate ≥25 mEq/L was associated with an estimated 3.0 g greater LVM (95% CI 0.5-5.0) and 1.0 mm Hg higher aortic PP (95% CI 0.4-2.0) compared to bicarbonate 23-24 mEq/L. Each 1 mEq/L of bicarbonate increase was associated with a 13% higher risk of incident heart failure (hazards ratio 1.13, 95% CI 1.01-2.11). Among diuretic users, higher bicarbonate was not associated with CVD. Bicarbonate was not associated with incident atherosclerotic CVE irrespective of diuretic use. <b><i>Conclusion:</i></b> Among nonusers of diuretics in a large community-based study, higher serum bicarbonate concentrations are associated with subclinical CVD and new heart failure.
DOI: 10.6084/m9.figshare.4312313
title: Supplementary Material for: DcR3, TFF3, and Midkine Are Novel Serum Biomarkers in Small Intestinal Neuroendocrine Tumors

description: Small intestinal neuroendocrine tumors (SI-NETs) are amine- and peptide-producing neoplasms. Most patients display metastases at the time of diagnosis; they have an unpredictable individual disease course and the tumors are often therapy resistant. Chromogranin A and 5-hydroxyindoleacetic acid are the biomarkers clinically used most often today, but there is a great need for novel diagnostic and prognostic biomarkers and new therapeutic targets. Sixty-nine biomarkers were screened in serum from 23 SI-NET patients and 23 healthy controls using the multiplex proximity ligation assay (PLA). A refined method, the proximity extension assay (PEA), was used to analyze 76 additional biomarkers. Statistical testing and multivariate classification were performed. Immunohistochemistry and ELISA were performed in an extended cohort. Using PLA, 19 biomarkers showed a significant difference in serum concentrations between patients and controls, and PEA revealed a difference in the concentrations of 17 proteins. Multivariate classification analysis revealed decoy receptor 3 (DcR3), trefoil factor 3 (TFF3), and midkine to be good biomarkers for the disease, which was confirmed by ELISA analysis. All 3 biomarkers were expressed in tumor tissue. DcR3 concentrations were elevated in patients with stage IV disease. High concentrations of DcR3 and TFF3 were correlated to poor survival. DcR3, TFF3, and midkine exhibited elevated serum concentrations in SI-NET patients compared to healthy controls, and DcR3 and TFF3 were associated with poor survival. DcR3 seems to be a marker for liver metastases, while TFF3 and midkine may be new diagnostic biomarkers for SI-NETs.
DOI: 10.6084/m9.figshare.4312334.v1
title: Supplementary Material for: The Crucial Role of C18-Cer in Fat-Induced Skeletal Muscle Insulin Resistance

description: <b><i>Background/Aims: </i></b>Muscle bioactive lipids accumulation leads to several disorder states. The most common are insulin resistance (IR) and type 2 diabetes. There is an ongoing debate which of the lipid species plays the major role in induction of muscle IR. Our aim was to elucidate the role of particular lipid group in induction of muscle IR. <b><i>Methods:</i></b> The analyses were performed on muscle from the following groups of rats: 1. Control, fed standard diet, 2 HFD, fed high fat diet, 3. HFD/Myr, fed HFD and treated with myriocin (Myr), an inhibitor of ceramide de novo synthesis. We utilized [U<sup>13</sup>C] palmitate isotope tracer infusion and mass spectrometry to measure content and synthesis rate of muscle long-chain acyl-CoA (LCACoA), diacylglycerols (DAG) and ceramide (Cer). <b><i>Results:</i></b> HFD led to intramuscular accumulation of LCACoA, DAG and Cer and skeletal muscle IR. Myr-treatment caused decrease in Cer (most noticeable for stearoyl-Cer and oleoyl-Cer) and accumulation of DAG, possibly due to re-channeling of excess of intramuscular LCACoA towards DAG synthesis. An improvement in insulin sensitivity at both systemic and muscular level coincided with decrease in ceramide, despite elevated intramuscular DAG. <b><i>Conclusion:</i></b> The improved insulin sensitivity was associated with decreased muscle stearoyl- and oleoyl-ceramide content. The results indicate that accumulation of those ceramide species has the greatest impact on skeletal muscle insulin sensitivity in rats.
DOI: 10.6084/m9.figshare.4312313.v1
title: Supplementary Material for: DcR3, TFF3, and Midkine Are Novel Serum Biomarkers in Small Intestinal Neuroendocrine Tumors

description: Small intestinal neuroendocrine tumors (SI-NETs) are amine- and peptide-producing neoplasms. Most patients display metastases at the time of diagnosis; they have an unpredictable individual disease course and the tumors are often therapy resistant. Chromogranin A and 5-hydroxyindoleacetic acid are the biomarkers clinically used most often today, but there is a great need for novel diagnostic and prognostic biomarkers and new therapeutic targets. Sixty-nine biomarkers were screened in serum from 23 SI-NET patients and 23 healthy controls using the multiplex proximity ligation assay (PLA). A refined method, the proximity extension assay (PEA), was used to analyze 76 additional biomarkers. Statistical testing and multivariate classification were performed. Immunohistochemistry and ELISA were performed in an extended cohort. Using PLA, 19 biomarkers showed a significant difference in serum concentrations between patients and controls, and PEA revealed a difference in the concentrations of 17 proteins. Multivariate classification analysis revealed decoy receptor 3 (DcR3), trefoil factor 3 (TFF3), and midkine to be good biomarkers for the disease, which was confirmed by ELISA analysis. All 3 biomarkers were expressed in tumor tissue. DcR3 concentrations were elevated in patients with stage IV disease. High concentrations of DcR3 and TFF3 were correlated to poor survival. DcR3, TFF3, and midkine exhibited elevated serum concentrations in SI-NET patients compared to healthy controls, and DcR3 and TFF3 were associated with poor survival. DcR3 seems to be a marker for liver metastases, while TFF3 and midkine may be new diagnostic biomarkers for SI-NETs.