cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.3474032
title: PROCESSING_OF_SENTENCES_TEMPORAL_CONNECTIVES_DATASET.csv

description: Column 1= subject (participant number). Column 2= months (age of participant in months). Column 3 = memory (Participant memory scores). Column 4 = vocab (Participant vocabulary scores). Column 5= item (item number: 1 to 64). Column 6 = rt (DV: reaction time). Column 7 = correct (DV: correct or incorrect). Column 8 = sentence (experimental sentence). Column 9 = connective (IV: sentence with before or after). Column 10= position (does the sentence use the connective at the start or middle of the sentence?) Column 11= order (is the order of mention of events in the sentence chronological or reverse-chronological?). Column 12= age (Is the participant 3to4, 4to5, 5to6, or 6to7 years old?)
DOI: 10.6084/m9.figshare.3474029
title: Evaluation material and outcomes of the analysis of RDF annotations in the RASH papers

description: This archive contains all the source materials and the results of the analysis of RDF annotations in the RASH papers performed, which is the main contribution of the paper<br><br>Osborne, F., Peroni, S. (2016). How Do Researchers Annotate Their Work? A Study on Semantic Annotations in RASH Papers. https://rawgit.com/essepuntato/rash/master/papers/rash-poster-iswc2016.html<br><br>submitted to the ISWC 2016 Poster and Demo Track.
DOI: 10.6084/m9.figshare.3474029.v1
title: Evaluation material and outcomes of the analysis of RDF annotations in the RASH papers

description: This archive contains all the source materials and the results of the analysis of RDF annotations in the RASH papers performed, which is the main contribution of the paper<br><br>Osborne, F., Peroni, S. (2016). How Do Researchers Annotate Their Work? A Study on Semantic Annotations in RASH Papers. https://rawgit.com/essepuntato/rash/master/papers/rash-poster-iswc2016.html<br><br>submitted to the ISWC 2016 Poster and Demo Track.
DOI: 10.6084/m9.figshare.3473963
title: Polymorphism analysis of prion protein gene in 11 Pakistani goat breeds

description: The association between caprine PrP gene polymorphisms and its susceptibility to scrapie has been investigated in current years. As the ORF of the PrP gene is extremely erratic in different breeds of goats, we studied the PrP gene polymorphisms in 80 goats which belong to 11 Pakistani indigenous goat breeds from all provinces of Pakistan. A total of 6 distinct polymorphic sites (one novel) with amino acid substitutions were identified in the PrP gene which includes 126 (A -&gt; G), 304 (G -&gt; T), 379 (A -&gt; G), 414 (C -&gt; T), 428 (A -&gt; G) and 718 (C -&gt; T). The locus c.428 was found highly polymorphic in all breeds as compare to other loci. On the basis of these PrP variants NJ phylogenetic tree was constructed through MEGA6.1 which showed that all goat breeds along with domestic sheep and Mauflon sheep appeared as in one clade and sharing its most recent common ancestors (MRCA) with deer species while Protein analysis has shown that these polymorphisms can lead to varied primary, secondary and tertiary structure of protein. Based on these polymorphic variants, genetic distance, multidimensional scaling plot and principal component analyses revealed the clear picture regarding greater number of substitutions in cattle PrP regions as compared to the small ruminant species. In particular these findings may pinpoint the fundamental control over the scrapie in <i>Capra hircus</i> on genetic basis.
DOI: 10.6084/m9.figshare.3473963.v2
title: Polymorphism analysis of prion protein gene in 11 Pakistani goat breeds

description: The association between caprine PrP gene polymorphisms and its susceptibility to scrapie has been investigated in current years. As the ORF of the PrP gene is extremely erratic in different breeds of goats, we studied the PrP gene polymorphisms in 80 goats which belong to 11 Pakistani indigenous goat breeds from all provinces of Pakistan. A total of 6 distinct polymorphic sites (one novel) with amino acid substitutions were identified in the PrP gene which includes 126 (A -&gt; G), 304 (G -&gt; T), 379 (A -&gt; G), 414 (C -&gt; T), 428 (A -&gt; G) and 718 (C -&gt; T). The locus c.428 was found highly polymorphic in all breeds as compare to other loci. On the basis of these PrP variants NJ phylogenetic tree was constructed through MEGA6.1 which showed that all goat breeds along with domestic sheep and Mauflon sheep appeared as in one clade and sharing its most recent common ancestors (MRCA) with deer species while Protein analysis has shown that these polymorphisms can lead to varied primary, secondary and tertiary structure of protein. Based on these polymorphic variants, genetic distance, multidimensional scaling plot and principal component analyses revealed the clear picture regarding greater number of substitutions in cattle PrP regions as compared to the small ruminant species. In particular these findings may pinpoint the fundamental control over the scrapie in <i>Capra hircus</i> on genetic basis.
DOI: 10.6084/m9.figshare.3473963.v1
title: Polymorphism analysis of prion protein gene in eleven Pakistani goat breeds

description: The association between caprine PrP gene polymorphisms and its susceptibility to scrapie has been investigated in current years. As the ORF of the PrP gene is extremely erratic in different breeds of goats, we studied the PrP gene polymorphisms in 80 goats which belong to 11 Pakistani indigenous goat breeds from all provinces of Pakistan. A total of six distinct polymorphic sites (one novel) with amino acid substitutions were identified in the PrP gene which includes 126 (A −&gt; G), 304 (G −&gt; T), 379 (A −&gt; G), 414 (C −&gt; T), 428 (A −&gt; G) and 718 (C −&gt; T). The locus c.428 was found highly polymorphic in all breeds as compare to other loci. On the basis of these PrP variants NJ phylogenetic tree was constructed through MEGA6.1 which showed that all goat breeds along with domestic sheep and Mauflon sheep appeared as in one clade and sharing its most recent common ancestors (MRCA) with deer species while Protein analysis has shown that these polymorphisms can lead to varied primary, secondary and tertiary structure of protein. Based on these polymorphic variants, genetic distance, multidimensional scaling plot and principal component analyses revealed the clear picture regarding greater number of substitutions in cattle PrP regions as compared to the small ruminant species. In particular these findings may pinpoint the fundamental control over the scrapie in <i>Capra hircus</i> on genetic basis.
DOI: 10.6084/m9.figshare.3473801
title: FokPLoSOne2016_Fig7

description: These are the Excel and Matlab files for Figure 7 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements".<br><br>Solid curves are produced by cubic splines as described in the paper above. Data was extracted from <br><br> Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br><br>and<br><br>Inaba et al. "Compensatory Enlargement of the Left Main Coronary Artery: Insights from the PROSPECT Study," Coronary Artery Disease (2014)<br><br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published in these papers.<br><br>
DOI: 10.6084/m9.figshare.3473801.v2
title: FokPLoSOne2016_Fig7

description: These are the Excel and Matlab files for Figure 7 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements".<br><br>Solid curves are produced by cubic splines as described in the paper above. Data was extracted from <br><br> Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br><br>and<br><br>Inaba et al. "Compensatory Enlargement of the Left Main Coronary Artery: Insights from the PROSPECT Study," Coronary Artery Disease (2014)<br><br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published in these papers.<br><br>
DOI: 10.6084/m9.figshare.3473798
title: FokPLoSOne2016_Fig5

description: These are the Excel and Matlab files for Figure 5 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>Solid curves are produced by the model as described in the paper above. Data was extracted from<br><br>Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br><br>and<br><br>Inaba et al. "Compensatory Enlargement of the Left Main Coronary Artery: Insights from the PROSPECT Study," Coronary Artery Disease (2014)<br><br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so these Figures are not exactly the same as the ones published in these papers.<br>
DOI: 10.6084/m9.figshare.3473798.v5
title: FokPLoSOne2016_Fig5

description: These are the Excel and Matlab files for Figure 5 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>Solid curves are produced by the model as described in the paper above. Data was extracted from<br><br>Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br><br>and<br><br>Inaba et al. "Compensatory Enlargement of the Left Main Coronary Artery: Insights from the PROSPECT Study," Coronary Artery Disease (2014)<br><br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so these Figures are not exactly the same as the ones published in these papers.<br>
DOI: 10.6084/m9.figshare.3473798.v4
title: FokPLoSOne2016_Fig5.fig

description: These are the Excel and Matlab files for Figure 5 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>Solid curves are produced by the model as described in the paper above. Data was extracted from<br><br>Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br><br>and<br><br>Inaba et al. "Compensatory Enlargement of the Left Main Coronary Artery: Insights from the PROSPECT Study," Coronary Artery Disease (2014)<br><br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so these Figures are not exactly the same as the ones published in these papers.<br>
DOI: 10.6084/m9.figshare.3473795
title: FokPLoSOne2016_Fig6

description: These are the Excel and Matlab files for Figure 6 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>The solid curves are produced by the model described in the paper above.<br><br>
DOI: 10.6084/m9.figshare.3473795.v3
title: FokPLoSOne2016_Fig6

description: These are the Excel and Matlab files for Figure 6 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>The solid curves are produced by the model described in the paper above.<br><br>
DOI: 10.6084/m9.figshare.3473795.v2
title: FokPLoSOne2016_Fig6.fig

description: These are the Excel and Matlab files for Figure 6 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>The solid curves are produced by the model described in the paper above.<br><br>
DOI: 10.6084/m9.figshare.3473786
title: FokPLoSOne2016_Fig3

description: These are the Excel and Matlab files for Figure 3 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements".<br><br>Solid curves are produced by the model described in the paper above. Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br><br><br><br>
DOI: 10.6084/m9.figshare.3473786.v3
title: FokPLoSOne2016_Fig3.fig

description: This is the Matlab file for Figure 3 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements".<br><br>Solid curves are produced by the model described in the paper above. Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br><br><br><br>
DOI: 10.6084/m9.figshare.3473786.v5
title: FokPLoSOne2016_Fig3

description: These are the Excel and Matlab files for Figure 3 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements".<br><br>Solid curves are produced by the model described in the paper above. Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br><br><br><br>
DOI: 10.6084/m9.figshare.3473786.v4
title: FokPLoSOne2016_Fig3.fig

description: These are the Excel and Matlab files for Figure 3 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements".<br><br>Solid curves are produced by the model described in the paper above. Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987)<br>using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br><br><br><br>
DOI: 10.6084/m9.figshare.3473777
title: New species and phylogenetic relationships of the spider genus Coptoprepes using morphological and sequence data (Araneae: Anyphaenidae)

description: <br>To assist with the matching of sexes in somatically similar species of <i>Coptoprepes</i> and in the absence of sequence data, we performed a dual cladistic analysis as proposed by Platnick & Shadab (1978), scoring the male and female as separate terminals in the morphological phylogenetic dataset (named <i>split</i> matrix). If they were the same species, the expectation is that they are grouped together or nearby in the tree. Once the sexes were matched, the final matrix was composed merging the scorings of male and female (named <i>merge </i>matrix).
DOI: 10.6084/m9.figshare.3473777.v1
title: New species and phylogenetic relationships of the spider genus Coptoprepes using morphological and sequence data (Araneae: Anyphaenidae)

description: <br>To assist with the matching of sexes in somatically similar species of <i>Coptoprepes</i> and in the absence of sequence data, we performed a dual cladistic analysis as proposed by Platnick & Shadab (1978), scoring the male and female as separate terminals in the morphological phylogenetic dataset (named <i>split</i> matrix). If they were the same species, the expectation is that they are grouped together or nearby in the tree. Once the sexes were matched, the final matrix was composed merging the scorings of male and female (named <i>merge </i>matrix).
DOI: 10.6084/m9.figshare.3473762
title: FokPLoSOne2016_Fig1

description: These are the Matlab and Excel files for Figure 1 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>The piece-wise linear fit was done "by eye". Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987) using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br>
DOI: 10.6084/m9.figshare.3473762.v3
title: FokPLoSOne2016_Fig1.fig

description: These are the Matlab and Excel files for Figure 1 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>The piece-wise linear fit was done "by eye". Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987) using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br>
DOI: 10.6084/m9.figshare.3473762.v5
title: FokPLoSOne2016_Fig1

description: These are the Matlab and Excel files for Figure 1 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>The piece-wise linear fit was done "by eye". Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987) using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br>
DOI: 10.6084/m9.figshare.3473762.v4
title: FokPLoSOne2016_Fig1.fig

description: These are the Matlab and Excel files for Figure 1 of the paper by Fok, PW "Multi-layer Mechanical Model of Glagov Remodeling in Coronary Arteries: Differences between In-vivo and Ex-vivo Measurements"<br><br>The piece-wise linear fit was done "by eye". Data was extracted from Glagov et al. "Compensatory Enlargement of Human Atherosclerotic Coronary Arteries", New England Journal of Medicine (1987) using WebPlotDigitizer:arohatgi.info/WebPlotDigitizer. Errors **are** made when using this tool, so this Figure is not exactly the same as the one published by Glagov. <br>
DOI: 10.6084/m9.figshare.3473690.v1
title: iSALE-Dellen manual

description: Manual for the Dellen release of the iSALE shock physics code: A multi-material, multi-rheology shock physics code for simulating impact phenomena in two and three dimensions.
DOI: 10.6084/m9.figshare.3473687
title: network FTQC

description: code for generating data in<br>Hierarchical surface code for network quantum computing with modules of arbitrary sizeYing Li and Simon C. BenjaminarXiv:1509.07796
DOI: 10.6084/m9.figshare.3473687.v1
title: network FTQC

description: code for generating data in<br>Hierarchical surface code for network quantum computing with modules of arbitrary sizeYing Li and Simon C. BenjaminarXiv:1509.07796
DOI: 10.6084/m9.figshare.3473654
title: Using_protoarray_data_for_diagnostics_of_Alzheimers_disease.zip

description: Protoarray Data from http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE29676
DOI: 10.6084/m9.figshare.3473654.v1
title: DATA_RAW.zip

description: Protoarray Data from http://www.ncbi.nlm.nih.gov/geo/query/acc.cgi?acc=GSE29676
DOI: 10.6084/m9.figshare.3473642
title: An activating <i>gucy2c</i> mutation causes impaired contractility and fluid stagnation in the small bowel

description: <b>Objective:</b> Familial GUCY2C diarrhoea syndrome (FGDS) is caused by an activating mutation in the GUCY2C gene encoding the receptor guanylate cyclase C in enterocytes. Activation leads to increased secretion of fluid into the intestinal lumen. Twenty percent of the patients have increased risk of Crohn’s disease and intestinal obstruction (CD, 20%) and the condition resembles irritable bowel syndrome with diarrhoea. We aimed to describe fluid content, contractility, peristaltic activity and bowel wall thickness in the intestine in fasting FGDS patients, using ultrasound, with healthy volunteers serving as controls. <b>Methods:</b> Twenty-three patients with FGDS and 22 healthy controls (HC) were examined with a Logiq E9 scanner in a fasting state. Bowel wall thickness was measured and fluid-filled small bowel loops were counted using three-dimensional (3D) magnetic positioning navigation. The HC ingested 500 ml PEG solution, an electrolyte balanced, non-absorbable solution, in order to investigate the contractions of the small bowel. <b>Results:</b> The fasting 23 FGDS patients had significantly higher number of fluid-filled small bowel segments compared to 22 fasting HC, <i>p</i> p <b>Conclusions:</b> FGDS is characterised by multiple, fluid-filled small bowel loops with incomplete contractions and fluid stagnation in fasting state. These findings may play a role in the increased risk of bowel obstruction as well as IBS-like symptoms observed in these patients.
DOI: 10.6084/m9.figshare.3473642.v1
title: An activating <i>gucy2c</i> mutation causes impaired contractility and fluid stagnation in the small bowel

description: <b>Objective:</b> Familial GUCY2C diarrhoea syndrome (FGDS) is caused by an activating mutation in the GUCY2C gene encoding the receptor guanylate cyclase C in enterocytes. Activation leads to increased secretion of fluid into the intestinal lumen. Twenty percent of the patients have increased risk of Crohn’s disease and intestinal obstruction (CD, 20%) and the condition resembles irritable bowel syndrome with diarrhoea. We aimed to describe fluid content, contractility, peristaltic activity and bowel wall thickness in the intestine in fasting FGDS patients, using ultrasound, with healthy volunteers serving as controls. <b>Methods:</b> Twenty-three patients with FGDS and 22 healthy controls (HC) were examined with a Logiq E9 scanner in a fasting state. Bowel wall thickness was measured and fluid-filled small bowel loops were counted using three-dimensional (3D) magnetic positioning navigation. The HC ingested 500 ml PEG solution, an electrolyte balanced, non-absorbable solution, in order to investigate the contractions of the small bowel. <b>Results:</b> The fasting 23 FGDS patients had significantly higher number of fluid-filled small bowel segments compared to 22 fasting HC, <i>p</i> p <b>Conclusions:</b> FGDS is characterised by multiple, fluid-filled small bowel loops with incomplete contractions and fluid stagnation in fasting state. These findings may play a role in the increased risk of bowel obstruction as well as IBS-like symptoms observed in these patients.