cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.13310764.v1
title: Additional file 8 of Deep sampling and pooled amplicon sequencing reveals hidden genic variation in heterogeneous rye accessions

description: Additional file 8: Table S4. Sanger sequencing validation of variants in single plants from an accession.
DOI: 10.6084/m9.figshare.13310719.v1
title: Additional file 1 of Deep sampling and pooled amplicon sequencing reveals hidden genic variation in heterogeneous rye accessions

description: Additional file 1: Table S1. Sequencing coverage for each nucleotide position in the experiment.
DOI: 10.6084/m9.figshare.13310758
title: Additional file 7 of Deep sampling and pooled amplicon sequencing reveals hidden genic variation in heterogeneous rye accessions

description: Additional file 7: Table S3. Number of polymorphic positions detected in common by three algorithms per gene.
DOI: 10.6084/m9.figshare.13310758.v1
title: Additional file 7 of Deep sampling and pooled amplicon sequencing reveals hidden genic variation in heterogeneous rye accessions

description: Additional file 7: Table S3. Number of polymorphic positions detected in common by three algorithms per gene.
DOI: 10.6084/m9.figshare.13310764
title: Additional file 8 of Deep sampling and pooled amplicon sequencing reveals hidden genic variation in heterogeneous rye accessions

description: Additional file 8: Table S4. Sanger sequencing validation of variants in single plants from an accession.
DOI: 10.6084/m9.figshare.13310740.v1
title: Additional file 4 of Deep sampling and pooled amplicon sequencing reveals hidden genic variation in heterogeneous rye accessions

description: Additional file 4: Table S2. Allele frequencies and number of accessions harboring alleles of predicted deleterious variants common to GATK, SNVer and CRISP.
DOI: 10.6084/m9.figshare.13310794
title: Additional file 1 of A novel method for locating the source of sustained oscillation in power system using synchrophasors data

description: Additional file 1.
DOI: 10.6084/m9.figshare.13310794.v1
title: Additional file 1 of A novel method for locating the source of sustained oscillation in power system using synchrophasors data

description: Additional file 1.
DOI: 10.6084/m9.figshare.13310824.v1
title: Additional file 1 of Spatio-temporal mapping of breast and prostate cancers in South Iran from 2014 to 2017

description: Additional file 1.
DOI: 10.6084/m9.figshare.13310827
title: Additional file 2 of Spatio-temporal mapping of breast and prostate cancers in South Iran from 2014 to 2017

description: Additional file 2.
DOI: 10.6084/m9.figshare.13310824
title: Additional file 1 of Spatio-temporal mapping of breast and prostate cancers in South Iran from 2014 to 2017

description: Additional file 1.
DOI: 10.6084/m9.figshare.13310827.v1
title: Additional file 2 of Spatio-temporal mapping of breast and prostate cancers in South Iran from 2014 to 2017

description: Additional file 2.
DOI: 10.6084/m9.figshare.13310905.v1
title: Additional file 3 of The fibronectin type-III (FNIII) domain of ATF7IP contributes to efficient transcriptional silencing mediated by the SETDB1 complex

description: Additional file 3: Table S1. Related to Fig. 3. List of FNIII domain-binding proteins. Proteins highlighted in light blue, light orange, light yellow, and light green are the validated proteins, the LCH (LSD1-CoREST-HDAC1) complex, the bait (ATF7IP) and the CPSF (cleavage and polyadenylation specificity factor) complex.
DOI: 10.6084/m9.figshare.13310887
title: Additional file 12 of The fibronectin type-III (FNIII) domain of ATF7IP contributes to efficient transcriptional silencing mediated by the SETDB1 complex

description: Additional file 12: Table S3. Primers and plasmids.
DOI: 10.6084/m9.figshare.13310887.v1
title: Additional file 12 of The fibronectin type-III (FNIII) domain of ATF7IP contributes to efficient transcriptional silencing mediated by the SETDB1 complex

description: Additional file 12: Table S3. Primers and plasmids.
DOI: 10.6084/m9.figshare.13310932
title: Additional file 8 of The fibronectin type-III (FNIII) domain of ATF7IP contributes to efficient transcriptional silencing mediated by the SETDB1 complex

description: Additional file 9: Table S2. Related to Fig. 5. List of DE genes and repeats in Atf7ip KO, Atf7ip KO rescued with WT or the FNIII domain mutant of ATF7IP and Zmym2 KO ESCs.
DOI: 10.6084/m9.figshare.13310932.v1
title: Additional file 8 of The fibronectin type-III (FNIII) domain of ATF7IP contributes to efficient transcriptional silencing mediated by the SETDB1 complex

description: Additional file 9: Table S2. Related to Fig. 5. List of DE genes and repeats in Atf7ip KO, Atf7ip KO rescued with WT or the FNIII domain mutant of ATF7IP and Zmym2 KO ESCs.
DOI: 10.6084/m9.figshare.13310905
title: Additional file 3 of The fibronectin type-III (FNIII) domain of ATF7IP contributes to efficient transcriptional silencing mediated by the SETDB1 complex

description: Additional file 3: Table S1. Related to Fig. 3. List of FNIII domain-binding proteins. Proteins highlighted in light blue, light orange, light yellow, and light green are the validated proteins, the LCH (LSD1-CoREST-HDAC1) complex, the bait (ATF7IP) and the CPSF (cleavage and polyadenylation specificity factor) complex.
DOI: 10.6084/m9.figshare.13311028
title: Additional file 2 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 2. Maximum likelihood phylogenetic tree.
DOI: 10.6084/m9.figshare.13311025
title: Additional file 1 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 1. Consensus phylogenetic tree.
DOI: 10.6084/m9.figshare.13311025.v1
title: Additional file 1 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 1. Consensus phylogenetic tree.
DOI: 10.6084/m9.figshare.13311034.v1
title: Additional file 4 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 4. WGS Variant Analysis.
DOI: 10.6084/m9.figshare.13311037.v1
title: Additional file 5 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 5. Gene Ontology (GO) Data.
DOI: 10.6084/m9.figshare.13311034
title: Additional file 4 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 4. WGS Variant Analysis.
DOI: 10.6084/m9.figshare.13311037
title: Additional file 5 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 5. Gene Ontology (GO) Data.
DOI: 10.6084/m9.figshare.13311028.v1
title: Additional file 2 of Biochemical and genetic analysis of Ecm14, a conserved fungal pseudopeptidase

description: Additional file 2. Maximum likelihood phylogenetic tree.
DOI: 10.6084/m9.figshare.13311061.v1
title: Additional file 1 of Elderly patients with atrial fibrillation in routine clinical practice—peri-procedural management of edoxaban oral anticoagulation therapy is associated with a low risk of bleeding and thromboembolic complications: a subset analysis of the prospective, observational, multinational EMIT-AF study

description: Additional file 1. EMIT study Ethics Committees.
DOI: 10.6084/m9.figshare.13311061
title: Additional file 1 of Elderly patients with atrial fibrillation in routine clinical practice—peri-procedural management of edoxaban oral anticoagulation therapy is associated with a low risk of bleeding and thromboembolic complications: a subset analysis of the prospective, observational, multinational EMIT-AF study

description: Additional file 1. EMIT study Ethics Committees.
DOI: 10.6084/m9.figshare.13311073
title: Additional file 3 of Platelet proteome changes in dogs with congestive heart failure

description: Additional file 3. Protein identifications and analytic details. Protein identifications with peptide sequences, mass errors and modifications.
DOI: 10.25919/16r7-5260
title: Parkes observations for project P456 semester 2020APRS_17

description: The Parkes Pulsar Timing Array (PPTA) project has three primary goals: (a) detection of gravitational waves from astronomical sources, (b) establishment of a pulsar timescale, and (c) improvement of our understanding of Solar-system dynamics. The PPTA is the oldest of three international pulsar timing groups. We have the smallest telescope and the smallest group, but the best measurements and the best results. Our bound on the strength of the incoherent gravitational wave background is the only bound which significantly constrains theoretical models. We have maintained our leading position because: we have the southern sky; we have a small but well-focussed group; we have the best-calibrated receivers; and we have been able to observe with an almost regular cadence of 15 to 20 days over a wide bandwidth. With this proposal we aim to maintain our pre-eminent position in the field. Unlike most observing proposals, this is a continuing proposal for which the observations will continue to improve bounds on ultra-low-frequency gravitational waves until they are finally detected. Continued Parkes observations will remain valuable at least until the first stage of the SKA is able to improve on our sensitivity and observing cadence. Even after the gravitational wave background is detected we will want to continue observations in support of the nascent field of gravitational wave astronomy!
DOI: 10.25919/6nk3-eg68
title: Parkes observations for project P1014 semester 2020APRS_06

description: The spatial distribution of neutral gas is important for understanding the interstellar medium (ISM), its evolution, and star formation. However, the distribution of Tiny Scale Atomic Structures (TSAS), especially the extremely small scale structures with spatial scales ranging from a few to hundreds of AU is largely unexplored. The formation and evolution of TSAS are still unknown. Multi-epoch observations of HI absorption against pulsars are a tested, practical observational method to probe the TSAS. Based on our observations in the 2019 APRS semester, we successfully processed baseband data and detected the HI absorption variation at over 4-months towards B1557-50. PSR B1451-68 is another ideal source to trace the TSAS because of the intervening cold gas. We thus propose one-epoch observation towards B1557-50 which should be as early in the semester as possible, and two-epoch observations towards B1451-68 to be separated by about 5 months, for monitoring the HI absorption variations and probing structures on a range of scales. This will better constrain the characteristics of variations in HI absorption, thus contribute significantly to understanding the origin of TSAS.
DOI: 10.6084/m9.figshare.13311073.v1
title: Additional file 3 of Platelet proteome changes in dogs with congestive heart failure

description: Additional file 3. Protein identifications and analytic details. Protein identifications with peptide sequences, mass errors and modifications.
DOI: 10.25919/phth-6k75
title: Parkes observations for project PX600 semester 2020APRS_25

description: The Galactic Center (GC) is a key target for the radio component of Breakthrough Listen (BL) program. Offering the largest amount of stars in any given direction in the sky, the GC is widely cited as a location believed to harbour advanced civilizations, and it is also the most energetic region in the Milky Way. We aim to conduct BL-GC Survey with Parkes radio telescope for around 350 hours. We will cover the frequency range from 700 MHz to 4 GHz, utilizing the newly installed ultra-wideband receiver. Our team has already leveraged both standard and bespoke tools to construct a flexible software stack to search data for signals of interest. We will look for signals from extraterrestrial intelligence (ETI) by searching for both simple narrow-band signals and complex modulated signals. Along with that, as an ancillary science, we will also search for accelerated pulsars ? likely orbiting a supermassive black hole at the centre of the Milky Way Galaxy. Data products produced during this program will also be publically available to engage larger pulsar community
DOI: 10.25919/pbjw-eq02
title: Parkes observations for project P895 semester 2020APRS_02

description: Since the year 2005, the Parkes Pulsar Timing Array (PPTA) project has been placing ever more stringent constraints on the amplitude of a gravitational wave background signal. Such upper bounds on the amplitude of the gravitational wave signal are currently dominated by a single pulsar: PSR J1909-3744. The upper bound with this pulsar alone already cuts into the range of tenable theoretical models. Further reducing the amplitude bound (by continued regular observations of this pulsar) will significantly narrow that range. The first evidence of gravitational waves in the pulsar data are likely to be observed in the timing for PSR J1909-3744, but with only a single pulsar we will not be able to make an unambiguous detection. However, knowledge of the likely signal strength (derived from this pulsar) will help to optimise the observing strategy for the detection of the gravitational wave signals with existing or future pulsar timing arrays.
DOI: 10.6084/m9.figshare.13311154
title: Additional file 10 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S10. Effects of core promoter-located GWAS-traits on gene expression by eQTL analysis
DOI: 10.6084/m9.figshare.13311154.v1
title: Additional file 10 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S10. Effects of core promoter-located GWAS-traits on gene expression by eQTL analysis
DOI: 10.6084/m9.figshare.13311166
title: Additional file 2 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S2. Variants in each population
DOI: 10.6084/m9.figshare.13311157.v1
title: Additional file 11 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S11. Novel variants identified from the study
DOI: 10.6084/m9.figshare.13311172
title: Additional file 4 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S4. Population-common and population-specific variants
DOI: 10.6084/m9.figshare.13311169
title: Additional file 3 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S3. Variant distribution between whole genome and core promoter
DOI: 10.6084/m9.figshare.13311163
title: Additional file 1 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S1. Exome data used in the study
DOI: 10.6084/m9.figshare.13311166.v1
title: Additional file 2 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S2. Variants in each population
DOI: 10.6084/m9.figshare.13311175
title: Additional file 5 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S5. Simple repeats identified in core promoter region
DOI: 10.6084/m9.figshare.13311175.v1
title: Additional file 5 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S5. Simple repeats identified in core promoter region
DOI: 10.6084/m9.figshare.13311178.v1
title: Additional file 6 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S6. Functional classification of genes with high-frequent core promoter variation
DOI: 10.6084/m9.figshare.13311187.v1
title: Additional file 9 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S9. Disease-associated core promoter variants
DOI: 10.6084/m9.figshare.13311178
title: Additional file 6 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S6. Functional classification of genes with high-frequent core promoter variation
DOI: 10.6084/m9.figshare.13311157
title: Additional file 11 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S11. Novel variants identified from the study
DOI: 10.6084/m9.figshare.13311163.v1
title: Additional file 1 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S1. Exome data used in the study
DOI: 10.6084/m9.figshare.13311172.v1
title: Additional file 4 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S4. Population-common and population-specific variants
DOI: 10.6084/m9.figshare.13311184.v1
title: Additional file 8 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S8. Core promoter variation in DNA damage repair genes
DOI: 10.6084/m9.figshare.13311181
title: Additional file 7 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S7. Effects of core promoter variants on gene expression by eQTL analysis
DOI: 10.6084/m9.figshare.13311181.v1
title: Additional file 7 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S7. Effects of core promoter variants on gene expression by eQTL analysis
DOI: 10.6084/m9.figshare.13311184
title: Additional file 8 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S8. Core promoter variation in DNA damage repair genes
DOI: 10.6084/m9.figshare.13311187
title: Additional file 9 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S9. Disease-associated core promoter variants
DOI: 10.6084/m9.figshare.13311169.v1
title: Additional file 3 of Highly diversified core promoters in the human genome and their effects on gene expression and disease predisposition

description: S3. Variant distribution between whole genome and core promoter
DOI: 10.6084/m9.figshare.13311222
title: Additional file 1 of Sequencing and analysis of gerbera daisy leaf transcriptomes reveal disease resistance and susceptibility genes differentially expressed and associated with powdery mildew resistance

description: Additional file 1: Table S1. Description of read data, quality control and GC content of UFGE 4033 and 06–245-03 gerbera breeding lines. Table S2. Functional annotation of gerbera transcripts performed using Blast2GO. Table S3. Mapping statistics of UFGE 4033 and 06–245-03 samples to de novo transcriptome assembly using Samtools and Qualimap. Samples R1, R2 and R3 represent three biological replicates of UFGE 4033 and S1, S2 and S3 represent three biological replicates of 06–245-03. Table S4. List of gerbera transcripts from powdery mildew resistant (UFGE 4033) and susceptible (06–245-03) breeding lines from the differential expression analysis of gerbera transcriptome. Table S5. List of gerbera transcripts from powdery mildew resistant (UFGE 4033) and susceptible (06–245-03) breeding lines functionally annotated as disease resistance in gerbera transcriptome. Table S6. List of Mildew locus O (MLO)-like transcripts present in the gerbera transcriptome constructed using powdery mildew resistant (UFGE 4033) and susceptible (06–245-03) breeding lines. Table S7. Single nucleotide polymorphisms identified between powdery mildew resistant (UFGE 4033) and susceptible (06–245-03) gerbera breeding lines using RNA-seq and SAMtools, FreeBayes and GATK pipelines. Table S8. Statistics on simple sequence repeats identified in gerbera transcriptome using powdery mildew resistant (UFGE 4033) and susceptible (06–245-03) breeding lines and MISA tools. Table S9. Primers for RT-qPCR analysis of differentially up-regulated R-genes in gerbera for powdery mildew resistance.
DOI: 10.6084/m9.figshare.13311258.v1
title: Additional file 2 of Retracted articles in rehabilitation: just the tip of the iceberg? A bibliometric analysis

description: Additional file 2. Journals that were found to have issued retraction notices.
DOI: 10.6084/m9.figshare.13311262.v1
title: Additional file 2 of Recommendations for the use of long-term experience sampling in bipolar disorder care: a qualitative study of patient and clinician experiences

description: Additional file 2. R code for the feedback report.
DOI: 10.6084/m9.figshare.13311262
title: Additional file 2 of Recommendations for the use of long-term experience sampling in bipolar disorder care: a qualitative study of patient and clinician experiences

description: Additional file 2. R code for the feedback report.
DOI: 10.6084/m9.figshare.13311258
title: Additional file 2 of Retracted articles in rehabilitation: just the tip of the iceberg? A bibliometric analysis

description: Additional file 2. Journals that were found to have issued retraction notices.
DOI: 10.6084/m9.figshare.13311280.v1
title: Additional file 3 of Eltrombopag inhibits the proliferation of Ewing sarcoma cells via iron chelation and impaired DNA replication

description: Additional file 3: Supplementary Table 1. Ewing sarcoma cell lines included in the PRISM drug screen.
DOI: 10.6084/m9.figshare.13311283
title: Additional file 4 of Eltrombopag inhibits the proliferation of Ewing sarcoma cells via iron chelation and impaired DNA replication

description: Additional file 4: Supplementary Table 2. Drug dependencies enriched in the Ewing sarcoma cell lines.
DOI: 10.6084/m9.figshare.13311280
title: Additional file 3 of Eltrombopag inhibits the proliferation of Ewing sarcoma cells via iron chelation and impaired DNA replication

description: Additional file 3: Supplementary Table 1. Ewing sarcoma cell lines included in the PRISM drug screen.
DOI: 10.6084/m9.figshare.13311283.v1
title: Additional file 4 of Eltrombopag inhibits the proliferation of Ewing sarcoma cells via iron chelation and impaired DNA replication

description: Additional file 4: Supplementary Table 2. Drug dependencies enriched in the Ewing sarcoma cell lines.
DOI: 10.6084/m9.figshare.13311306
title: Additional file 2 of Differential gene expression analysis reveals pathways important in early post-traumatic osteoarthritis in an equine model

description: Additional file 2. Genes differentially expressed with a fold-change (FC) cut-off &gt; |1.1|. Genes upregulated in OA samples are represented by a positive FC; genes downregulated in OA samples are represented by a negative FC.
DOI: 10.6084/m9.figshare.13311318
title: Additional file 6 of Differential gene expression analysis reveals pathways important in early post-traumatic osteoarthritis in an equine model

description: Additional file 6. DE genes assigned to clusters by STRING using a MCL algorithm. Functional annotation of clusters with 10 or more genes was performed based on GO-Biological Process ontology in PANTHER.
DOI: 10.6084/m9.figshare.13311315.v1
title: Additional file 5 of Differential gene expression analysis reveals pathways important in early post-traumatic osteoarthritis in an equine model

description: Additional file 5. Genes falling within pathways identified as enriched by Reactome analysis.