cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.6084/m9.figshare.24558565.v1
title: Anti-tumour Reactivity of immune cells in oral and salivary gland cancer microenvironment

description: Inhibition of Anti-tumour Reactivity of immune cells in the Salivary Gland Cancer: A Proteomic Approach<br>
DOI: 10.6084/m9.figshare.24558565
title: Anti-tumour Reactivity of immune cells in oral and salivary gland cancer microenvironment

description: Inhibition of Anti-tumour Reactivity of immune cells in the Salivary Gland Cancer: A Proteomic Approach<br>
DOI: 10.5281/zenodo.10122141
title: Data for Project 'Diagnostic Accuracy, Reliability, and Construct Validity of the German Quick Mild Cognitive Impairment Screen'

description: Data for Project 'Diagnostic Accuracy, Reliability, and Construct Validity of the German Quick Mild Cognitive Impairment Screen' consisting of (1) the complete data set of all data analyzed for the project 'Diagnostic Accuracy, Reliability, and Construct Validity of the German Quick Mild Cognitive Impairment Screen' ('Data_Brain-IT-Validation-Qmci_for-publication.xlsx'; and (2) a corresponding README file including (a) general information, (b) data and file overview, (c) sharing and access information, (d) methodological information, and (e) data-specific information.
DOI: 10.5281/zenodo.10122140
title: Data for Project 'Diagnostic Accuracy, Reliability, and Construct Validity of the German Quick Mild Cognitive Impairment Screen'

description: Data for Project 'Diagnostic Accuracy, Reliability, and Construct Validity of the German Quick Mild Cognitive Impairment Screen' consisting of (1) the complete data set of all data analyzed for the project 'Diagnostic Accuracy, Reliability, and Construct Validity of the German Quick Mild Cognitive Impairment Screen' ('Data_Brain-IT-Validation-Qmci_for-publication.xlsx'; and (2) a corresponding README file including (a) general information, (b) data and file overview, (c) sharing and access information, (d) methodological information, and (e) data-specific information.
DOI: 10.6084/m9.figshare.24558595.v1
title: Varanus salavator morphology sex prediction (19 Features).csv

description: Dataset consists of morphological measurements made from <i>V. salvator</i> from a skin trade in Malaysia.
DOI: 10.6084/m9.figshare.24558613
title: Source data

description: <b>Crop rotation and native microbiome inoculation restore soil capacity to suppress a root disease</b>
DOI: 10.6084/m9.figshare.24558613.v1
title: Source data

description: <b>Crop rotation and native microbiome inoculation restore soil capacity to suppress a root disease</b>
DOI: 10.6084/m9.figshare.24558673
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.6084/m9.figshare.24558673.v1
title: Supplementary Material for: QTc interval prolongation independently associates with FGF23 and predicts mortality in predialysis CKD.

description: Introduction: QTc interval prolongation is increasingly frequent as CKD advances and predicts death in dialysis. However, predictors and mortality-risk in predialysis CKD are understudied. FGF23 induces left ventricular hypertrophy (LVH) which is associated with QTc interval prolongation and death, suggesting a possible pathway from FGF23 to death that entails LVH and QTc prolongation. We looked for links between FGF23 and prolonged QTc intervals mediated by LVH, and for deaths associated with QTc prolongation in a prospective observational cohort of patients with predialysis CKD. Methods: Participants underwent protocolized baseline and semi-annual FGF23 testing, baseline and study end echocardiograms, and baseline and annual electrocardiograms over three years. Results: 2,254 participants (34.1% female; mean age 68.7 years; mean glomerular filtration rate 41.4 ml/min/m2) enrolled. Baseline LVH (left ventricular mass index &gt;131 g/m2 (&gt;100 g/m2 if female)) was present in 10.8% and prolonged QTc intervals (&gt;=500 ms) in 1.5%. One hundred thirty-eight (6.1%) participants died during the study. In generalized mixed effects regression, each unit increase in the natural log of FGF23 - but not LVH - predicted an odds ratio of 1.76 (1.15, 2.70, p=0.009) for prolonged QTc intervals independently of 15 other covariates. Mediation analysis showed that only 13% of FGF23's total effect on prolonged QTc intervals was mediated by LVH. Patients with prolonged QTc intervals had higher unadjusted (log rank p&lt;0.001) and adjusted (hazard ratio 3.15 (1.38, 7.16, p=0.006)) mortality rates than those with QTc intervals &lt;500 ms. Conclusions: QTc interval prolongation ≥500 ms was prospectively associated with FGF23 independently of LVH, and with a tripling of mortality-risk in patients with predialysis CKD.
DOI: 10.6084/m9.figshare.24558628.v1
title: Csi_v1_peps.fasta

description: The protein sequences of <i>Chrysosplenium</i> sinicum genome
DOI: 10.6084/m9.figshare.24558628
title: Csi_v1_peps.fasta

description: The protein sequences of <i>Chrysosplenium</i> sinicum genome
DOI: 10.5281/zenodo.10122132
title: Hard Point Cloud Localization Dataset

description: This is a dataset to evaluate the robustness of point-cloud-based localization algorithms in extremely severe situations including aggressive sensor motion, point cloud degeneration, and data interruptions.
The dataset contains five indoor sequences recorded with a Microsoft Azure Kinect and three outdoor sequences recorded with a Livox MID360. Each outdoor sequence is split into two consecutive rosbags.
indoor_easy_01 & 02    : "Easy" sequences without aggressive sensor motion and data interruptions.indoor_hard_01             : "Hard" sequence involving quick sensor motion and point cloud degeneration.indoor_kidnap_01 & 02 : Sequences that involve kidnapping situations (the sensor was moved to another room while its view was completely occluded several times)outdoor_hard_01 & 02  : "Hard" outdoor sequence involving quick sensor motion and point cloud degeneration.outdoor_kidnap             : Outdoor sequence that involve kidnapping situations.Each rosbag corresponds to an environmental map file as follows:indoor_easy       : map_indoor_easy.plyindoor_kidnap    : map_indoor_easy.plyindoor_hard        : map_indoor_hard.plyoutdoor_kidnap  : map_outdoor_kidnap.plyoutdoor_hard     : map_outdoor_hard.plyEach indoor sequence contains the following ROS2 messages:- /points2/decompressed : sensor_msgs/msg/PointCloud2 : Point cloud data- /imu                                : sensor_msgs/msg/Imu              : Imu dataEach outdoor sequence contains the following ROS2 messages:- /livox/points           : sensor_msgs/msg/PointCloud2       : Point cloud data- /livox/lidar              : livox_ros_driver2/msg/CustomMsg : Point cloud data in the Livox format- /livox/imu               : sensor_msgs/msg/Imu                    : Imu dataWe used the follwoing LiDAR-IMU transformation parameters (T_lidar_imu : [tx, ty, tz, qx, qy, qz, qw]) for the indoor and outdoor sequences:- indoor  : [0.003, 0.004, -0.051, -0.476, 0.474, 0.524, 0.525]- outdoor: [0.000, 0.000, 0.000, 0.000, 0.000, 0.000, 1.000]Groundtruth trajectories were obtained through batch optimization of point cloud registration residuals and IMU motion residuals. Each line in a GT trajectory file represents the LiDAR pose in the map frame [time, tx, ty, tz, qx, qy, qz, qw] (TUM format). We recommend evo toolkit (https://github.com/MichaelGrupp/evo) for quantitative evaluation.
DOI: 10.5281/zenodo.10122133
title: Hard Point Cloud Localization Dataset

description: This is a dataset to evaluate the robustness of point-cloud-based localization algorithms in extremely severe situations including aggressive sensor motion, point cloud degeneration, and data interruptions.
The dataset contains five indoor sequences recorded with a Microsoft Azure Kinect and three outdoor sequences recorded with a Livox MID360. Each outdoor sequence is split into two consecutive rosbags.
indoor_easy_01 & 02    : "Easy" sequences without aggressive sensor motion and data interruptions.indoor_hard_01             : "Hard" sequence involving quick sensor motion and point cloud degeneration.indoor_kidnap_01 & 02 : Sequences that involve kidnapping situations (the sensor was moved to another room while its view was completely occluded several times)outdoor_hard_01 & 02  : "Hard" outdoor sequence involving quick sensor motion and point cloud degeneration.outdoor_kidnap             : Outdoor sequence that involve kidnapping situations.Each rosbag corresponds to an environmental map file as follows:indoor_easy       : map_indoor_easy.plyindoor_kidnap    : map_indoor_easy.plyindoor_hard        : map_indoor_hard.plyoutdoor_kidnap  : map_outdoor_kidnap.plyoutdoor_hard     : map_outdoor_hard.plyEach indoor sequence contains the following ROS2 messages:- /points2/decompressed : sensor_msgs/msg/PointCloud2 : Point cloud data- /imu                                : sensor_msgs/msg/Imu              : Imu dataEach outdoor sequence contains the following ROS2 messages:- /livox/points           : sensor_msgs/msg/PointCloud2       : Point cloud data- /livox/lidar              : livox_ros_driver2/msg/CustomMsg : Point cloud data in the Livox format- /livox/imu               : sensor_msgs/msg/Imu                    : Imu dataWe used the follwoing LiDAR-IMU transformation parameters (T_lidar_imu : [tx, ty, tz, qx, qy, qz, qw]) for the indoor and outdoor sequences:- indoor  : [0.003, 0.004, -0.051, -0.476, 0.474, 0.524, 0.525]- outdoor: [0.000, 0.000, 0.000, 0.000, 0.000, 0.000, 1.000]Groundtruth trajectories were obtained through batch optimization of point cloud registration residuals and IMU motion residuals. Each line in a GT trajectory file represents the LiDAR pose in the map frame [time, tx, ty, tz, qx, qy, qz, qw] (TUM format). We recommend evo toolkit (https://github.com/MichaelGrupp/evo) for quantitative evaluation.
DOI: 10.5281/zenodo.10122307
title: Dataset related to article "Denosumab improves trabecular bone score in relationship with decrease in fracture risk of women exposed to aromatase inhibitors "

description: This record contains raw data related to article "Denosumab improves trabecular bone score in relationship with decrease in fracture risk of women exposed to aromatase inhibitors"
Abstract
Purpose: Trabecular bone score (TBS) is a gray-level textural metric that has shown to correlate with risk of fractures in several forms of osteoporosis. The value of TBS in predicting fractures and the effects of bone-active drugs on TBS in aromatase inhibitors (AIs)-induced osteoporosis are still largely unknown. The primary objective of this retrospective study was to assess the effects of denosumab and bisphosphonates (BPs) on TBS and vertebral fractures (VFs) in women exposed to AIs.
Methods: 241 consecutive women (median age 58 years) with early breast cancer undergoing treatment with AIs were evaluated for TBS, bone mineral density (BMD) and morphometric VFs at baseline and after 18-24 months of follow-up. During the study period, 139 women (57.7%) received denosumab 60 mg every 6 months, 53 (22.0%) BPs, whereas 49 women (20.3%) were not treated with bone-active drugs.
Results: Denosumab significantly increased TBS values (from 1.270 to 1.323; P < 0.001) accompanied by a significant decrease in risk of VFs (odds ratio 0.282; P = 0.021). During treatment with BPs, TBS did not significantly change (P = 0.849) and incidence of VFs was not significantly different from women untreated with bone-active drugs (P = 0.427). In the whole population, women with incident VFs showed higher decrease in TBS vs. non-fractured women (P = 0.003), without significant differences in changes of BMD at any skeletal site.
Conclusions: TBS variation predicts fracture risk in AIs treated women. Denosumab is effective to induce early increase of TBS and reduction in risk of VFs
DOI: 10.5281/zenodo.10122306
title: Dataset related to article "Denosumab improves trabecular bone score in relationship with decrease in fracture risk of women exposed to aromatase inhibitors "

description: This record contains raw data related to article "Denosumab improves trabecular bone score in relationship with decrease in fracture risk of women exposed to aromatase inhibitors"
Abstract
Purpose: Trabecular bone score (TBS) is a gray-level textural metric that has shown to correlate with risk of fractures in several forms of osteoporosis. The value of TBS in predicting fractures and the effects of bone-active drugs on TBS in aromatase inhibitors (AIs)-induced osteoporosis are still largely unknown. The primary objective of this retrospective study was to assess the effects of denosumab and bisphosphonates (BPs) on TBS and vertebral fractures (VFs) in women exposed to AIs.
Methods: 241 consecutive women (median age 58 years) with early breast cancer undergoing treatment with AIs were evaluated for TBS, bone mineral density (BMD) and morphometric VFs at baseline and after 18-24 months of follow-up. During the study period, 139 women (57.7%) received denosumab 60 mg every 6 months, 53 (22.0%) BPs, whereas 49 women (20.3%) were not treated with bone-active drugs.
Results: Denosumab significantly increased TBS values (from 1.270 to 1.323; P < 0.001) accompanied by a significant decrease in risk of VFs (odds ratio 0.282; P = 0.021). During treatment with BPs, TBS did not significantly change (P = 0.849) and incidence of VFs was not significantly different from women untreated with bone-active drugs (P = 0.427). In the whole population, women with incident VFs showed higher decrease in TBS vs. non-fractured women (P = 0.003), without significant differences in changes of BMD at any skeletal site.
Conclusions: TBS variation predicts fracture risk in AIs treated women. Denosumab is effective to induce early increase of TBS and reduction in risk of VFs
DOI: 10.6084/m9.figshare.24558772
title: Csi_v1_cds.fasta

description: The coding sequences file of <i>Chrysosplenium sinicum</i> genome
DOI: 10.6084/m9.figshare.24558772.v1
title: Csi_v1_cds.fasta

description: The coding sequences file of <i>Chrysosplenium sinicum</i> genome
DOI: 10.6084/m9.figshare.24558778
title: Csi_v1_genome.fa.gz

description: The chromosome-level genome assembly of <i>Chrysosplenium sinicum</i> genome using PacBio Technologies and Hi-C technology.
DOI: 10.34740/kaggle/dsv/6964385
title: AutoXGB
DOI: 10.34740/kaggle/dsv/6964386
title: OnlineFree
DOI: 10.6084/m9.figshare.24558787
title: Csi_mitochondrial_genome_and_annotation

description: <i>Chrysosplenium sinicum</i> mitochondria genome sequence and genome annotation.
DOI: 10.6084/m9.figshare.24558787.v2
title: Csi_mitochondrial_genome_and_annotation

description: <i>Chrysosplenium sinicum</i> mitochondria genome sequence and genome annotation.
DOI: 10.6084/m9.figshare.24558787.v1
title: Mitochondrial_genome_and_annotation

description: <i>Chrysosplenium sinicum</i> mitochondria genome sequence and genome annotation.
DOI: 10.6084/m9.figshare.23997939
title: Márton-et-al-Cecropin-A-Investigation-of-a-Host-Defense-Peptide-with-Multifaceted-immunomodulatory-Activity-in-a-Chicken-Hepatic-Cell-Culture-dataset

description: Data were obtained from measurements of supernatant samples taken after treatment of primary hepatic cell cultures of chicken origin.
DOI: 10.6084/m9.figshare.23997939.v1
title: Márton-et-al-Cecropin-A-Investigation-of-a-Host-Defense-Peptide-with-Multifaceted-immunomodulatory-Activity-in-a-Chicken-Hepatic-Cell-Culture-dataset

description: Data were obtained from measurements of supernatant samples taken after treatment of primary hepatic cell cultures of chicken origin.
DOI: 10.34740/kaggle/dsv/6964376
title: LUFlow Network Intrusion Detection Data Set
DOI: 10.5281/zenodo.8348434
title: Code and tables for main figures of Synthesis of the land carbon fluxes of the Amazon region between 2010 and 2020

description: Code and tables to reproduce the main figures (2a,2b,3a,3b,4a,5a) of Synthesis of the land carbon fluxes of the Amazon region between 2010 and 2020.
DOI: 10.5281/zenodo.8348435
title: Code and tables for main figures of Synthesis of the land carbon fluxes of the Amazon region between 2010 and 2020

description: Code and tables to reproduce the main figures (2a,2b,3a,3b,4a,5a) of Synthesis of the land carbon fluxes of the Amazon region between 2010 and 2020.
DOI: 10.17632/ydfkrv849v.1
title: Partial correction of immunodeficiency by lentiviral vector gene therapy in mouse models carrying Rag1 hypomorphic mutations

description: Recombination activating genes (RAG) 1 and 2 defects are the most frequent form of severe combined immunodeficiency (SCID). Patients with residual RAG activity have a spectrum of clinical manifestations ranging from Omenn syndrome to delayed-onset combined immunodeficiency, often associated with granulomas and/or autoimmunity (CID-G/AI). Lentiviral vector (LV) gene therapy (GT) has been proposed as an alternative treatment to the standard hematopoietic stem cell transplant and a clinical trial for RAG1 SCID patients recently started. However, GT in patients with hypomorphic RAG mutations poses additional risks, because of the residual endogenous RAG1 expression and the general state of immune dysregulation and associated inflammation. In this study, we assessed the efficacy of GT in 2 hypomorphic Rag1 murine models (Rag1F971L/F971L and Rag1R972Q/R972Q), exploiting the same LV used in the clinical trial encoding RAG1 under control of the MND promoter. Starting 6 weeks after transplant, GT-treated mice showed a decrease in proportion of myeloid cells and a concomitant increase of B, T and total white blood cells. However, counts remained lower than in mice transplanted with WT Lin- cells. At euthanasia, we observed a general redistribution of immune subsets in tissues, with the appearance of mature recirculating B cells in the bone marrow. In the thymus, we demonstrated correction of the block at double negative stage, with a modest improvement in the cortical/medullary ratio. Analysis of antigen-specific IgM and IgG serum levels after in vivo challenge showed an amelioration of antibody responses, suggesting that the partial immune correction could confer a clinical benefit. Notably, no overt signs of autoimmunity were detected, with B-cell activating factor decreasing to normal levels and autoantibodies remaining stable after GT. On the other hand, thymic enlargement was frequently observed, although not due to vector integration and insertional mutagenesis. In conclusion, our work shows that GT could partially alleviate the combined immunodeficiency of hypomorphic RAG1 patients and that extensive efficacy and safety studies with alternative models are required before commencing RAG gene therapy in these highly complex patients.
DOI: 10.11588/data/avff2e
title: Simulation-based Inference for Model Parameterization on Analog Neuromorphic Hardware [data]

description: This data is presented in the paper "Simulation-based Inference for Model Parameterization on Analog Neuromorphic Hardware".
DOI: 10.17632/86p5rjdtvc.3
title: DATA OF IMPACT OF MERGER ON THE FINANCIAL PERFORMANCE OF SBI

description: The present exploratory study is based on secondary data collected from the finncial reports of State Bank of India (SBI). The collected data was examined by applyimg ratio analysis using CAMEL framework. This ratio analayis was performed in pre and post merger period of SBI separately. The pre-merger period in the current study includes a period of 4 years (financial year 2013-14 to 2016-17) succedding year 2017 in which the associate banks were merged with SBI.While the post-merger is also of four years (financial year 2018-19 to 2021-22). The compaartive analysis of the financial peformance of SBI in Pre-Merger Period and Post-Merger Period, was performed with the help of Paired T Test on the ratios representing different indicators of the CAMEL model. 

Table-1 CAMEL Framework
CAMEL Framework
Indicators of Financial Performance with Abbreviations


Capital Adequacy Measures
1.	Capital Adequacy Ratio (CAR)
2.	Total Equity Ratio (TER)
Asset Quality Measures
3.	Gross NPA Ratio (GNPAR)
4.	Net NPA to Net Advance Ratio (NNPP TO NAR)
5.	Provision Coverage Ratio (PCR)
Management Capability measures 
6.	Slippage Ratio (SR)
7.	Cost to Income Ratio (CIR)
8.	Cost to Asset Ratio (CtoAR)
9.	Operational Profit to Income Ratio (OPIR)
10.	Asset to Turnover Ratio (ATR)
Earnings Measures
11.	CASA Ratio
12.	Return on Asset Ratio (RAR)
13.	Return on Equity Ratio (RER)
14.	Gross Profit Margin Ratio (GPMR)
15.	Net Profit Margin Ratio (NPMR)
16.	Net Interest Margin Ratio (NIMR)
Liquidity Measures
17.	Liquid assets to deposit-borrowing ratio (LADST)
18.	Net Loans to total asset ratio (NLTA)
19.	Net loans to deposit and borrowing (NLDST)