cometadata/datacite-titles-descriptions-related-identifiers huggingface dataset:
DOI: 10.5281/zenodo.11047499
title: Supplementary data to `Do science maps from open access literature capture the overall topic structure of an academic field?`

description: The dataset contains the 8,528 academic articles records related to Sustainable Food research sourced with the query `TS=("sustainab*" NEAR/2 "food*")` .

They are the records present in the largest component of the citation network, as specified in the manuscript.  

The dataset was sourced from OpenAlex based on the original data used in the manuscript and it is composed of the following columns:




Column
Description


Id
OpenAlex ID


DOI
Document Object Identifier


display_name
The article title


publication_year
The publication year of the article


open_access
An object with details of the open access status of the article




We choose the `.rdata` format for easy loading in R. Use the function `load()` to add the data frame to the enviroment.
DOI: 10.5281/zenodo.11047498
title: Supplementary data to `Do science maps from open access literature capture the overall topic structure of an academic field?`

description: The dataset contains the 8,528 academic articles records related to Sustainable Food research sourced with the query `TS=("sustainab*" NEAR/2 "food*")` .

They are the records present in the largest component of the citation network, as specified in the manuscript.  

The dataset was sourced from OpenAlex based on the original data used in the manuscript and it is composed of the following columns:




Column
Description


Id
OpenAlex ID


DOI
Document Object Identifier


display_name
The article title


publication_year
The publication year of the article


open_access
An object with details of the open access status of the article




We choose the `.rdata` format for easy loading in R. Use the function `load()` to add the data frame to the enviroment.
DOI: 10.15468/dl.b27zyq
title: Occurrence Download

description: A dataset containing 12 species occurrences available in GBIF matching the query:
{
 "TaxonKey" : [
 "is Aristolochia cortinata Reinecke"
 ]
}

 The dataset includes 12 records from 6 constituent datasets; see https://api.gbif.org/v1/occurrence/download/0049953-230224095556074/datasets/export for details.
 
Data from some individual datasets included in this download may be licensed under less restrictive terms.
DOI: 10.48597/62c5-mrr7
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/6rym-3x6u
title: Measurement of Inorganics in precipitation at Hvasser

description: Measurement of Inorganics in precipitation at Hvasser
DOI: 10.48597/7cxq-u5af
title: Measurement of Inorganics in precipitation at Løken

description: Measurement of Inorganics in precipitation at Løken
DOI: 10.48597/7ewj-u5jd
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/82bk-axnr
title: Measurement of Inorganics in precipitation at Løken

description: Measurement of Inorganics in precipitation at Løken
DOI: 10.48597/88cy-n3f3
title: Measurement of Inorganics in precipitation at Løken

description: Measurement of Inorganics in precipitation at Løken
DOI: 10.48597/9c7j-gbw5
title: Measurement of Inorganics in precipitation at Karpbukt

description: Measurement of Inorganics in precipitation at Karpbukt
DOI: 10.48597/9k6j-g4cj
title: Measurement of Inorganics in precipitation at Karpbukt

description: Measurement of Inorganics in precipitation at Karpbukt
DOI: 10.48597/9m9e-bzq9
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/9zsv-kduu
title: Measurement of Inorganics in precipitation at Vatnedalen

description: Measurement of Inorganics in precipitation at Vatnedalen
DOI: 10.48597/a56w-6w45
title: Measurement of Inorganics in precipitation at Vatnedalen

description: Measurement of Inorganics in precipitation at Vatnedalen
DOI: 10.48597/abyn-4s4a
title: Measurement of Inorganics in precipitation at Grimelid

description: Measurement of Inorganics in precipitation at Grimelid
DOI: 10.48597/acwd-qw9q
title: Measurement of Inorganics in precipitation at Tangen

description: Measurement of Inorganics in precipitation at Tangen
DOI: 10.48597/adpp-wht2
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/akbk-xfmm
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/b7w8-k2xq
title: Measurement of Inorganics in precipitation at Karpbukt

description: Measurement of Inorganics in precipitation at Karpbukt
DOI: 10.48597/babe-k349
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/bg7h-wdem
title: Measurement of Inorganics in precipitation at Skei i Jølster

description: Measurement of Inorganics in precipitation at Skei i Jølster
DOI: 10.48597/cmm8-xfet
title: Measurement of Inorganics in precipitation at Tovdal

description: Measurement of Inorganics in precipitation at Tovdal
DOI: 10.48597/edyj-h3m5
title: Measurement of Inorganics in precipitation at Tågmyra

description: Measurement of Inorganics in precipitation at Tågmyra
DOI: 10.48597/fauk-phg8
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/fbdw-tygb
title: Measurement of Inorganics in precipitation at Vatnedalen

description: Measurement of Inorganics in precipitation at Vatnedalen
DOI: 10.48597/fjm6-33pr
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/fw7c-x9e6
title: Measurement of Inorganics in precipitation at Skei i Jølster

description: Measurement of Inorganics in precipitation at Skei i Jølster
DOI: 10.48597/fwan-mtgh
title: Measurement of Inorganics in precipitation at Løken

description: Measurement of Inorganics in precipitation at Løken
DOI: 10.48597/fwqu-2p35
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/g6rk-waup
title: Measurement of Inorganics in precipitation at Vatnedalen

description: Measurement of Inorganics in precipitation at Vatnedalen
DOI: 10.48597/gasq-2p75
title: Measurement of Inorganics in precipitation at Tovdal

description: Measurement of Inorganics in precipitation at Tovdal
DOI: 10.48597/hm5m-jrg7
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/hs8j-ggep
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/htx7-6f7s
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/hufm-6a32
title: Measurement of Inorganics in precipitation at Løken

description: Measurement of Inorganics in precipitation at Løken
DOI: 10.48597/hznf-2vqg
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/jawy-5pkk
title: Measurement of Inorganics in precipitation at Fitjar

description: Measurement of Inorganics in precipitation at Fitjar
DOI: 10.48597/jbry-52gg
title: Measurement of Inorganics in precipitation at Tovdal

description: Measurement of Inorganics in precipitation at Tovdal
DOI: 10.48597/jdvx-yhn6
title: Measurement of Inorganics in precipitation at Karpbukt

description: Measurement of Inorganics in precipitation at Karpbukt
DOI: 10.48597/jjbj-3gu5
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/jmte-fzat
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/jrn6-5fxx
title: Measurement of Inorganics in precipitation at Hvasser

description: Measurement of Inorganics in precipitation at Hvasser
DOI: 10.48597/k25d-eusa
title: Measurement of Inorganics in precipitation at Skei i Jølster

description: Measurement of Inorganics in precipitation at Skei i Jølster
DOI: 10.48597/k4ew-8sgn
title: Measurement of Inorganics in precipitation at Gjerstad

description: Measurement of Inorganics in precipitation at Gjerstad
DOI: 10.48597/k65e-e7tv
title: Measurement of Inorganics in precipitation at Førde

description: Measurement of Inorganics in precipitation at Førde
DOI: 10.48597/mdzh-93cr
title: Measurement of Inorganics in precipitation at Treungen

description: Measurement of Inorganics in precipitation at Treungen
DOI: 10.48597/nc99-sbad
title: Measurement of Inorganics in precipitation at Fitjar

description: Measurement of Inorganics in precipitation at Fitjar
DOI: 10.48597/nsp6-7t4f
title: Measurement of Inorganics in precipitation at Tovdal

description: Measurement of Inorganics in precipitation at Tovdal
DOI: 10.48597/p6rk-kfhq
title: Measurement of Inorganics in precipitation at Hvasser

description: Measurement of Inorganics in precipitation at Hvasser
DOI: 10.48597/pass-h354
title: Measurement of Inorganics in precipitation at Løken

description: Measurement of Inorganics in precipitation at Løken
DOI: 10.48597/pgf7-7uvc
title: Measurement of Inorganics in precipitation at Karpbukt

description: Measurement of Inorganics in precipitation at Karpbukt
DOI: 10.48597/qmkv-2xdk
title: Measurement of Inorganics in precipitation at Bislingen

description: Measurement of Inorganics in precipitation at Bislingen
DOI: 10.48597/qxmf-s9rd
title: Measurement of Inorganics in precipitation at Løken

description: Measurement of Inorganics in precipitation at Løken
DOI: 10.48597/qzxv-rnwf
title: Measurement of Inorganics in precipitation at Karpbukt

description: Measurement of Inorganics in precipitation at Karpbukt
DOI: 10.6084/m9.figshare.17798865
title: Additional file 10 of N4-acetyldeoxycytosine DNA modification marks euchromatin regions in Arabidopsis thaliana

description: Additional file 10: Table S9. DMRs identified in met1 and rdd mutants compared to Col-0 wild-type.
DOI: 10.6084/m9.figshare.17798889.v1
title: Additional file 4 of N4-acetyldeoxycytosine DNA modification marks euchromatin regions in Arabidopsis thaliana

description: Additional file 4: Table S3. GO analysis of 4acC-containing expressed genes.
DOI: 10.6084/m9.figshare.16750609.v1
title: Autophagy deficiency activates rDNA transcription

description: Macroautophagy/autophagy, a highly conserved lysosome-dependent degradation pathway, has been intensively studied in regulating cell metabolism by degradation of intracellular components. In this study, we link autophagy to RNA metabolism by uncovering a regulatory role of autophagy in ribosomal RNA (rRNA) synthesis. Autophagy-deficient cells exhibit much higher <i>47S</i> precursor rRNA level, which is caused by the accumulation of SQSTM1/p62 (sequestosome 1) but not other autophagy receptors. Mechanistically, SQSTM1 accumulation potentiates the activation of MTOR (mechanistic target of rapamycin kinase) complex 1 (MTORC1) signaling and promotes the assembly of RNA polymerase I pre-initiation complex at ribosomal DNA (rDNA) promoters, which leads to an increase of <i>47S</i> rRNA transcribed from rDNA. Functionally, autophagy deficiency promotes protein synthesis, cell growth and cell proliferation, both of which are dependent on SQSTM1 accumulation. Taken together, our findings suggest that autophagy deficiency is involved in RNA metabolism by activating rDNA transcription and provide novel mechanisms for the reprogramming of cell metabolism in autophagy-related diseases including multiple types of cancers. <b>Abbreviations:</b> 5-FUrd: 5-fluorouridine; AMPK: AMP-activated protein kinase; ATG: autophagy related; CALCOCO2/NDP52: calcium binding and coiled-coil domain 2; ChIP: chromatin immunoprecipitation; MAP1LC3/LC3: microtubule associated protein 1 light chain 3; MAPK/ERK: mitogen-activated protein kinase; MTOR: mechanistic target of rapamycin kinase; NBR1: NBR1 autophagy cargo receptor; NFKB/NF-κB: nuclear factor kappa B; NFE2L2/NRF2: nuclear factor, erythroid 2 like 2; OPTN: optineurin; PIC: pre-initiation complex; POLR1: RNA polymerase I; POLR1A/RPA194: RNA polymerase I subunit A; POLR2A: RNA polymerase II subunit A; rDNA: ribosomal DNA; RPS6KB1/S6K1: ribosomal protein S6 kinase B1; rRNA: ribosomal RNA; RUBCN/Rubicon: rubicon autophagy regulator; SQSTM1/p62: sequestosome 1; STX17: syntaxin 17; SUnSET: surface sensing of translation; TAX1BP1: Tax1 binding protein 1; UBTF/UBF1: upstream binding transcription factor; WIPI2: WD repeat domain, phosphoinositide interacting 2; WT: wild-type.
DOI: 10.6084/m9.figshare.16750615
title: Bioinformatics-based identification of SPNS3 (Spinster homolog 3) as a prognostic biomarker of apoptosis resistance in acute myeloid leukemia

description: Spinster homolog 3 (SPNS3) belongs to the Spinster (SPNS) family which participates in sphingolipid transportation through the cell membrane. However, the functions of SPNS3 in acute myeloid leukemia (AML) are unknown. This study obtained SPNS3 from a gene set that was related to AML relapse and evaluate whether high SPNS3 expression induced apoptosis resistance in an AML cell line, which is consistent with the role of SPNS3 as a marker of poor prognosis in the clinic. Moreover, internal tandem duplication of FMS-like tyrosine kinase 3 (FLT3-ITD) mutation and the AC127521.1/ MIR-139/SPNS3 competing endogenous RNA axis were found to regulate SPNS3 expression. In addition, we noted that SPNS3 may play an important role in the Sphingosine-1-phosphate signal pathway that is involved in the maintenance of the AML microenvironment. These results highlight the anti-apoptosis effect of SPNS3 in AML, and the potential mechanism mediating this effect was explored through bioinformatics. <b>Abbreviations:</b> AML: acute myeloid leukemia; FLT3-ITD: internal tandem duplication of FMS-like tyrosine kinase 3; SPNS3: spinster homolog 3; SPNS1: spinster homolog 1; SPNS2: spinster homolog 2; GO: gene ontology; S1P: sphingosine-1-phosphate; ceRNA: competing endogenous RNA; dAML: acute myeloid leukemia at diagnosis; iAML: acute myeloid leukemia after induction chemotherapy; rAML: acute myeloid leukemia at relapse; DEGs: differentially expressed genes; BP: biological processes; CC: cellular components; MF: molecular functions; MRD: minimal residual disease; EFS: event-free survival; OS: overall survival; KEGG: Kyoto Encyclopedia of Genes and Genomes; SPHK: Sphingosine kinase.
DOI: 10.6084/m9.figshare.16750615.v2
title: Bioinformatics-based identification of SPNS3 (Spinster homolog 3) as a prognostic biomarker of apoptosis resistance in acute myeloid leukemia

description: Spinster homolog 3 (SPNS3) belongs to the Spinster (SPNS) family which participates in sphingolipid transportation through the cell membrane. However, the functions of SPNS3 in acute myeloid leukemia (AML) are unknown. This study obtained SPNS3 from a gene set that was related to AML relapse and evaluate whether high SPNS3 expression induced apoptosis resistance in an AML cell line, which is consistent with the role of SPNS3 as a marker of poor prognosis in the clinic. Moreover, internal tandem duplication of FMS-like tyrosine kinase 3 (FLT3-ITD) mutation and the AC127521.1/ MIR-139/SPNS3 competing endogenous RNA axis were found to regulate SPNS3 expression. In addition, we noted that SPNS3 may play an important role in the Sphingosine-1-phosphate signal pathway that is involved in the maintenance of the AML microenvironment. These results highlight the anti-apoptosis effect of SPNS3 in AML, and the potential mechanism mediating this effect was explored through bioinformatics. <b>Abbreviations:</b> AML: acute myeloid leukemia; FLT3-ITD: internal tandem duplication of FMS-like tyrosine kinase 3; SPNS3: spinster homolog 3; SPNS1: spinster homolog 1; SPNS2: spinster homolog 2; GO: gene ontology; S1P: sphingosine-1-phosphate; ceRNA: competing endogenous RNA; dAML: acute myeloid leukemia at diagnosis; iAML: acute myeloid leukemia after induction chemotherapy; rAML: acute myeloid leukemia at relapse; DEGs: differentially expressed genes; BP: biological processes; CC: cellular components; MF: molecular functions; MRD: minimal residual disease; EFS: event-free survival; OS: overall survival; KEGG: Kyoto Encyclopedia of Genes and Genomes; SPHK: Sphingosine kinase.
DOI: 10.6084/m9.figshare.16750615.v1
title: Bioinformatics-based identification of SPNS3 (Spinster homolog 3) as a prognostic biomarker of apoptosis resistance in acute myeloid leukemia

description: Spinster homolog 3 (SPNS3) belongs to the Spinster (SPNS) family which participates in sphingolipid transportation through the cell membrane. However, the functions of SPNS3 in acute myeloid leukemia (AML) are unknown. This study obtained SPNS3 from a gene set that was related to AML relapse and evaluate whether high SPNS3 expression induced apoptosis resistance in an AML cell line, which is consistent with the role of SPNS3 as a marker of poor prognosis in the clinic. Moreover, internal tandem duplication of FMS-like tyrosine kinase 3 (FLT3-ITD) mutation and the AC127521.1/ MIR-139/SPNS3 competing endogenous RNA axis were found to regulate SPNS3 expression. In addition, we noted that SPNS3 may play an important role in the Sphingosine-1-phosphate signal pathway that is involved in the maintenance of the AML microenvironment. These results highlight the anti-apoptosis effect of SPNS3 in AML, and the potential mechanism mediating this effect was explored through bioinformatics. <b>Abbreviations:</b> AML: acute myeloid leukemia; FLT3-ITD: internal tandem duplication of FMS-like tyrosine kinase 3; SPNS3: spinster homolog 3; SPNS1: spinster homolog 1; SPNS2: spinster homolog 2; GO: gene ontology; S1P: sphingosine-1-phosphate; ceRNA: competing endogenous RNA; dAML: acute myeloid leukemia at diagnosis; iAML: acute myeloid leukemia after induction chemotherapy; rAML: acute myeloid leukemia at relapse; DEGs: differentially expressed genes; BP: biological processes; CC: cellular components; MF: molecular functions; MRD: minimal residual disease; EFS: event-free survival; OS: overall survival; KEGG: Kyoto Encyclopedia of Genes and Genomes; SPHK: Sphingosine kinase.
DOI: 10.6084/m9.figshare.14129177
title: Additional file 5 of Identification, characterization and functional analysis of grape (Vitis vinifera L.) mitochondrial transcription termination factor (mTERF) genes in responding to biotic stress and exogenous phytohormone

description: Additional file 5: Table S1. Primer sequences used for semi-quantitative RT-PCR and quantitative real-time PCR of the 25 grape mTERF genes. Primer 5.0 software were used to designed specific primers.
DOI: 10.6084/m9.figshare.14129177.v1
title: Additional file 5 of Identification, characterization and functional analysis of grape (Vitis vinifera L.) mitochondrial transcription termination factor (mTERF) genes in responding to biotic stress and exogenous phytohormone

description: Additional file 5: Table S1. Primer sequences used for semi-quantitative RT-PCR and quantitative real-time PCR of the 25 grape mTERF genes. Primer 5.0 software were used to designed specific primers.
DOI: 10.6084/m9.figshare.16750618
title: UNC5B-AS1 promotes the proliferation, migration and EMT of hepatocellular carcinoma cells via regulating miR-4306/KDM2A axis

description: Being one of the most prevalent malignancies, hepatocellular carcinoma (HCC) threatens the health of population all over the world. Numerous researches have confirmed that long noncoding RNAs (lncRNAs) play an important role in tumor progression. Nonetheless, the mechanisms of unc-5 netrin receptor B antisense RNA 1 (UNC5B-AS1) in HCC remain obscure. Thus, this study aims to investigate the regulatory role and mechanism of UNC5B-AS1 in HCC cells. In our research, UNC5B-AS1 was subjected to gene expression analysis by RT-qPCR. Biological functions of UNC5B-AS1 in HCC cells were measured by MTT, colony formation, EdU and transwell assays. The combination between UNC5B-AS1, lysine demethylase 2A (KDM2A) and miR-4306 was validated by mechanism assays. Result showed UNC5B-AS1 was upregulated in HCC tissues and cells, contributing to the development of cancer staging and survival rate of HCC patients. Moreover, UNC5B-AS1 deficiency inhibited the proliferation, migration and epithelial–mesenchymal transition (EMT) of HCC cells. Furthermore, UNC5B-AS1 could interact with miR-4306 in HCC cells. Similarly, KDM2A was proved as the target gene of miR-4306. Finally, miR-4306 downregulation or KDM2A overexpression reversed the prohibitive role of UNC5B-AS1 knockdown in HCC progression. In short, UNC5B-AS1 accelerates the proliferation, migration and EMT of HCC cells via the regulation of miR-4306/KDM2A axis.
DOI: 10.6084/m9.figshare.16750618.v1
title: UNC5B-AS1 promotes the proliferation, migration and EMT of hepatocellular carcinoma cells via regulating miR-4306/KDM2A axis

description: Being one of the most prevalent malignancies, hepatocellular carcinoma (HCC) threatens the health of population all over the world. Numerous researches have confirmed that long noncoding RNAs (lncRNAs) play an important role in tumor progression. Nonetheless, the mechanisms of unc-5 netrin receptor B antisense RNA 1 (UNC5B-AS1) in HCC remain obscure. Thus, this study aims to investigate the regulatory role and mechanism of UNC5B-AS1 in HCC cells. In our research, UNC5B-AS1 was subjected to gene expression analysis by RT-qPCR. Biological functions of UNC5B-AS1 in HCC cells were measured by MTT, colony formation, EdU and transwell assays. The combination between UNC5B-AS1, lysine demethylase 2A (KDM2A) and miR-4306 was validated by mechanism assays. Result showed UNC5B-AS1 was upregulated in HCC tissues and cells, contributing to the development of cancer staging and survival rate of HCC patients. Moreover, UNC5B-AS1 deficiency inhibited the proliferation, migration and epithelial–mesenchymal transition (EMT) of HCC cells. Furthermore, UNC5B-AS1 could interact with miR-4306 in HCC cells. Similarly, KDM2A was proved as the target gene of miR-4306. Finally, miR-4306 downregulation or KDM2A overexpression reversed the prohibitive role of UNC5B-AS1 knockdown in HCC progression. In short, UNC5B-AS1 accelerates the proliferation, migration and EMT of HCC cells via the regulation of miR-4306/KDM2A axis.