chrisvoncsefalvay/vaers-narrative-generation huggingface dataset:
Age: 57.0
Sex: F
Vaccine: COVID19
Symptoms: Abnormal behaviour, Blood test, Bradykinesia, Brain injury, Carbon monoxide poisoning
Clinical narrative: At the beginning of the year my mother, patient, a 57-year-old lung transplant recipient came down with Covid. She received monoclonal antibodies and was better within 4 to 5 days. On May 12 she received her first Covid vaccine. Within a few days, she was feeling sick, my sister had to bring her food and help take care of her cat. By May 19 my sister noticed my mom was experiencing leg pain, walking slower, disengaged with people around her, and not responding in her normal ways. On May 22, around 12 pm, my mom left her apartment under unusual circumstances. She wears false teeth but didn?t put them in, left her cell phone behind, and left her unlocked. She got into her car, ran a red light, and caused a three-car collision. The officer that responded to the scene did not call an ambulance. He issued her a citation and let her go. At this point, we didn?t even know she had been in a wreck. She was later found almost 14 hours after the car wreck at 2:43 AM, 5 miles away at a closed gas station. The officer that found her stated that she was confused, she thought the date was January 1962. She said she was getting gas to put in her truck because she had been in a wreck. She had no vehicle or gas can. The officer placed her under the emergency Act and took her to hospital. She was at the hospital for approximately a week where she was diagnosed with encephalopathy and altered mental status. Because she is a lung transplant patient, she was then transferred to the different hospital. She was at that hospital for another week. They diagnosed her with toxic brain damage consistent with carbon monoxide poisoning. We had her apartment checked, no detectable levels of carbon monoxide. She also does not have a garage or other source of carbon monoxide. She did not have any carbon monoxide in her system either. The doctors hypothesized that she may have been showing signs of dementia when she got into her car and then was exposed to five minutes or less of carbon monoxide during the car wreck which would have wreaked havoc on her faster than a typical person due to her lung transplant underlying health. My sister and I told the doctors about the vaccine and how she had developed the symptoms shortly after. We asked about the possibility of the vaccine causing toxic brain damage. They agreed that it wasn?t impossible but didn?t have research to back it as the cause. On Tuesday, June 7, she was transferred back to hospital with the recommendation to enter hospice care and given a 7 to 10 days life expectancy due to irreversible, extensive brain damage. The damage is attributed directly to toxicity in her brain, not the car wreck.
Age: 57.0
Sex: F
Vaccine: COVID19
Symptoms: Computerised tomogram, Confusional state, Electrocardiogram, Encephalopathy, Gait disturbance
Clinical narrative: At the beginning of the year my mother, patient, a 57-year-old lung transplant recipient came down with Covid. She received monoclonal antibodies and was better within 4 to 5 days. On May 12 she received her first Covid vaccine. Within a few days, she was feeling sick, my sister had to bring her food and help take care of her cat. By May 19 my sister noticed my mom was experiencing leg pain, walking slower, disengaged with people around her, and not responding in her normal ways. On May 22, around 12 pm, my mom left her apartment under unusual circumstances. She wears false teeth but didn?t put them in, left her cell phone behind, and left her unlocked. She got into her car, ran a red light, and caused a three-car collision. The officer that responded to the scene did not call an ambulance. He issued her a citation and let her go. At this point, we didn?t even know she had been in a wreck. She was later found almost 14 hours after the car wreck at 2:43 AM, 5 miles away at a closed gas station. The officer that found her stated that she was confused, she thought the date was January 1962. She said she was getting gas to put in her truck because she had been in a wreck. She had no vehicle or gas can. The officer placed her under the emergency Act and took her to hospital. She was at the hospital for approximately a week where she was diagnosed with encephalopathy and altered mental status. Because she is a lung transplant patient, she was then transferred to the different hospital. She was at that hospital for another week. They diagnosed her with toxic brain damage consistent with carbon monoxide poisoning. We had her apartment checked, no detectable levels of carbon monoxide. She also does not have a garage or other source of carbon monoxide. She did not have any carbon monoxide in her system either. The doctors hypothesized that she may have been showing signs of dementia when she got into her car and then was exposed to five minutes or less of carbon monoxide during the car wreck which would have wreaked havoc on her faster than a typical person due to her lung transplant underlying health. My sister and I told the doctors about the vaccine and how she had developed the symptoms shortly after. We asked about the possibility of the vaccine causing toxic brain damage. They agreed that it wasn?t impossible but didn?t have research to back it as the cause. On Tuesday, June 7, she was transferred back to hospital with the recommendation to enter hospice care and given a 7 to 10 days life expectancy due to irreversible, extensive brain damage. The damage is attributed directly to toxicity in her brain, not the car wreck.
Age: 57.0
Sex: F
Vaccine: COVID19
Symptoms: Hyporesponsive to stimuli, Impaired driving ability, Indifference, Loss of personal independence in daily activities, Lumbar puncture
Clinical narrative: At the beginning of the year my mother, patient, a 57-year-old lung transplant recipient came down with Covid. She received monoclonal antibodies and was better within 4 to 5 days. On May 12 she received her first Covid vaccine. Within a few days, she was feeling sick, my sister had to bring her food and help take care of her cat. By May 19 my sister noticed my mom was experiencing leg pain, walking slower, disengaged with people around her, and not responding in her normal ways. On May 22, around 12 pm, my mom left her apartment under unusual circumstances. She wears false teeth but didn?t put them in, left her cell phone behind, and left her unlocked. She got into her car, ran a red light, and caused a three-car collision. The officer that responded to the scene did not call an ambulance. He issued her a citation and let her go. At this point, we didn?t even know she had been in a wreck. She was later found almost 14 hours after the car wreck at 2:43 AM, 5 miles away at a closed gas station. The officer that found her stated that she was confused, she thought the date was January 1962. She said she was getting gas to put in her truck because she had been in a wreck. She had no vehicle or gas can. The officer placed her under the emergency Act and took her to hospital. She was at the hospital for approximately a week where she was diagnosed with encephalopathy and altered mental status. Because she is a lung transplant patient, she was then transferred to the different hospital. She was at that hospital for another week. They diagnosed her with toxic brain damage consistent with carbon monoxide poisoning. We had her apartment checked, no detectable levels of carbon monoxide. She also does not have a garage or other source of carbon monoxide. She did not have any carbon monoxide in her system either. The doctors hypothesized that she may have been showing signs of dementia when she got into her car and then was exposed to five minutes or less of carbon monoxide during the car wreck which would have wreaked havoc on her faster than a typical person due to her lung transplant underlying health. My sister and I told the doctors about the vaccine and how she had developed the symptoms shortly after. We asked about the possibility of the vaccine causing toxic brain damage. They agreed that it wasn?t impossible but didn?t have research to back it as the cause. On Tuesday, June 7, she was transferred back to hospital with the recommendation to enter hospice care and given a 7 to 10 days life expectancy due to irreversible, extensive brain damage. The damage is attributed directly to toxicity in her brain, not the car wreck.
Age: 57.0
Sex: F
Vaccine: COVID19
Symptoms: Magnetic resonance imaging, Malaise, Mental status changes, Pain in extremity, Road traffic accident
Clinical narrative: At the beginning of the year my mother, patient, a 57-year-old lung transplant recipient came down with Covid. She received monoclonal antibodies and was better within 4 to 5 days. On May 12 she received her first Covid vaccine. Within a few days, she was feeling sick, my sister had to bring her food and help take care of her cat. By May 19 my sister noticed my mom was experiencing leg pain, walking slower, disengaged with people around her, and not responding in her normal ways. On May 22, around 12 pm, my mom left her apartment under unusual circumstances. She wears false teeth but didn?t put them in, left her cell phone behind, and left her unlocked. She got into her car, ran a red light, and caused a three-car collision. The officer that responded to the scene did not call an ambulance. He issued her a citation and let her go. At this point, we didn?t even know she had been in a wreck. She was later found almost 14 hours after the car wreck at 2:43 AM, 5 miles away at a closed gas station. The officer that found her stated that she was confused, she thought the date was January 1962. She said she was getting gas to put in her truck because she had been in a wreck. She had no vehicle or gas can. The officer placed her under the emergency Act and took her to hospital. She was at the hospital for approximately a week where she was diagnosed with encephalopathy and altered mental status. Because she is a lung transplant patient, she was then transferred to the different hospital. She was at that hospital for another week. They diagnosed her with toxic brain damage consistent with carbon monoxide poisoning. We had her apartment checked, no detectable levels of carbon monoxide. She also does not have a garage or other source of carbon monoxide. She did not have any carbon monoxide in her system either. The doctors hypothesized that she may have been showing signs of dementia when she got into her car and then was exposed to five minutes or less of carbon monoxide during the car wreck which would have wreaked havoc on her faster than a typical person due to her lung transplant underlying health. My sister and I told the doctors about the vaccine and how she had developed the symptoms shortly after. We asked about the possibility of the vaccine causing toxic brain damage. They agreed that it wasn?t impossible but didn?t have research to back it as the cause. On Tuesday, June 7, she was transferred back to hospital with the recommendation to enter hospice care and given a 7 to 10 days life expectancy due to irreversible, extensive brain damage. The damage is attributed directly to toxicity in her brain, not the car wreck.
Age: 57.0
Sex: F
Vaccine: COVID19
Symptoms: Scan with contrast
Clinical narrative: At the beginning of the year my mother, patient, a 57-year-old lung transplant recipient came down with Covid. She received monoclonal antibodies and was better within 4 to 5 days. On May 12 she received her first Covid vaccine. Within a few days, she was feeling sick, my sister had to bring her food and help take care of her cat. By May 19 my sister noticed my mom was experiencing leg pain, walking slower, disengaged with people around her, and not responding in her normal ways. On May 22, around 12 pm, my mom left her apartment under unusual circumstances. She wears false teeth but didn?t put them in, left her cell phone behind, and left her unlocked. She got into her car, ran a red light, and caused a three-car collision. The officer that responded to the scene did not call an ambulance. He issued her a citation and let her go. At this point, we didn?t even know she had been in a wreck. She was later found almost 14 hours after the car wreck at 2:43 AM, 5 miles away at a closed gas station. The officer that found her stated that she was confused, she thought the date was January 1962. She said she was getting gas to put in her truck because she had been in a wreck. She had no vehicle or gas can. The officer placed her under the emergency Act and took her to hospital. She was at the hospital for approximately a week where she was diagnosed with encephalopathy and altered mental status. Because she is a lung transplant patient, she was then transferred to the different hospital. She was at that hospital for another week. They diagnosed her with toxic brain damage consistent with carbon monoxide poisoning. We had her apartment checked, no detectable levels of carbon monoxide. She also does not have a garage or other source of carbon monoxide. She did not have any carbon monoxide in her system either. The doctors hypothesized that she may have been showing signs of dementia when she got into her car and then was exposed to five minutes or less of carbon monoxide during the car wreck which would have wreaked havoc on her faster than a typical person due to her lung transplant underlying health. My sister and I told the doctors about the vaccine and how she had developed the symptoms shortly after. We asked about the possibility of the vaccine causing toxic brain damage. They agreed that it wasn?t impossible but didn?t have research to back it as the cause. On Tuesday, June 7, she was transferred back to hospital with the recommendation to enter hospice care and given a 7 to 10 days life expectancy due to irreversible, extensive brain damage. The damage is attributed directly to toxicity in her brain, not the car wreck.
Age: 54.0
Sex: M
Vaccine: COVID19
Symptoms: Blood pressure increased, Condition aggravated, Congenital cystic kidney disease, Glomerular filtration rate decreased
Clinical narrative: Controlled BP jumped to 180/110 causing GFR to go from 35 down to 19 in a couple of weeks, leveling out at GFR of 13.
Age: 76.0
Sex: F
Vaccine: COVID19
Symptoms: Atrial fibrillation, Blood test, Echocardiogram, Electrocardiogram, Heart rate irregular
Clinical narrative: I got the second booster at my pharmacy at 10:00am. That night around 1:00am I went into rapid afib. I was taken to hospital by ambulance. I was hospitalized for several day before my heart rate went into normal rhythm under medication. I had never had afib before.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Acute kidney injury, Acute respiratory failure, Areflexia, Blood creatinine increased, Blood lactic acid
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Blood potassium decreased, Bradycardia, Brain injury, Breath sounds absent, COVID-19
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: COVID-19 pneumonia, Cardiac arrest, Cerebral ischaemia, Chest X-ray abnormal, Coma scale
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Computerised tomogram head normal, Corneal reflex decreased, Cough, Death, Electroencephalogram abnormal
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Endotracheal intubation, Haemoglobin decreased, Hyporesponsive to stimuli, Intensive care, Laboratory test
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Lung opacity, Myoclonus, Neurological symptom, Oxygen saturation decreased, Platelet count normal
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Positive airway pressure therapy, Post-anoxic myoclonus, Procalcitonin normal, Pulseless electrical activity, Pupil fixed
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Respiratory arrest, Resuscitation, SARS-CoV-2 test positive, Seizure, Status epilepticus
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Syncope, Toxicologic test normal, Troponin normal, Unresponsive to stimuli, Urine analysis normal
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 61.0
Sex: M
Vaccine: COVID19
Symptoms: Vomiting
Clinical narrative: Pt was admitted on 12/12 after presenting to the ECC after a witnessed cardiac arrest.  The patient lived in an group home and was noted to stand up from the dinner table and suddenly collapsed.  CPR was started by staff. Arrival of EMS noted a PEA arrest.  Two rounds of CPR and 2 mg of epi were administered prior to ROSC.  Upon arrival to the ECC, he was noted to have minimal neurologic response with no gag reflex, no corneal reflex, and pupils fixed and dilated.  He also had a witnessed seizure of 15-30 seconds in the ECC and received Ativan with termination of the seizure activity.  CT of the head was performed which resulted as negative.  Lab work showed no leukocytosis, hemoglobin 12.5, platelets normal, potassium 3.3, creatinine 1.4, troponin negative, lactic acid 6.3, negative procalcitonin, UA negative, urine tox screen negative, COVID-19 positive.  Cardiology was consulted.  Patient was admitted to the ICU.   12/13-12/14 patient remained intubated and sedated on propofol.  Receiving Jevity 30. Noted to have myoclonic jerks and intermittent episodes of severe hacking and coughing.  Neurology consulted.  EEG reported to be severely abnormal consistent with post-anoxic mild clonus status epilepticus after cardiac arrest/severe diffuse anoxic ischemic brain injury and with an extremely poor prognosis for survival. 12/16:  Remained intubated, off infusions, unresponsive,  myoclonic jerks and seizure-like activity was noted.  Episodes of emesis. 12/17:  Patient was extubated to BiPAP.  Remained unresponsive.  GCS 3. Continued myoclonic jerks.  Neurology resumed home medications of Klonopin.  Depakote added. 12/18:  Early a.m., patient was noted to have episodes of desaturation of 83-87% on 6 L O2.  He was transitioned back to BiPAP.  Patient remained in significant distress in spite of multiple doses of morphine. Patient was continued on BiPAP/Ativan/morphine for comfort. The evening of 12/18, patient was found to have marginal blood pressures.  He continued on BiPAP with high settings.  Guardian was contacted again-requested no escalation of care.    12/19:  Patient remained on supportive care .  He developed significant bradycardia with decreasing SpO2.  Clergy was called in for bedside prayer.  The patient developed asystole with no breath sounds or respirations noted at 0220 hr with a five-minute confirmation.   Discharge Diagnoses:  PEA cardiac arrest, acute hypoxic respiratory failure, anoxic brain injury, COVID-19 pneumonia, AKI, death.
Age: 73.0
Sex: F
Vaccine: COVID19
Symptoms: COVID-19, Cough, Dyspnoea, Pyrexia, SARS-CoV-2 test positive
Clinical narrative: Patient came into the emergency department due to shortness of breath, she was diagnosed with COVID 19 on November 22nd, started on dexamethasone November 24th, she reported progressively worsening shortness of breath, no pain, some cough and fever as well.  She was admitted for further care.   Hospital Course:  Patient was treated with dexamethasone, seen by infectious disease and started on Remdesivir which has been completed, was also given vitamin-C, zinc, vitamin-D, She was discharged when she no longer was requiring supplemental oxygen, other home medications were continued.
Age: 72.0
Sex: M
Vaccine: COVID19
Symptoms: COVID-19, SARS-CoV-2 test positive, Syncope, Urinary tract infection
Clinical narrative: This is a 72 y male with PMH significant for multiple medical problems including CAD status post CABG, HTN, and obesity who was admitted on 12/21/2021 because of syncopal episode and was found to be COVID-19 positive .  In regard to COVID-19 infection initially patient was started on Remdesivir. However given the fact that he was not hypoxic Remdesivir was discontinued and patient does not qualify for steroids either.  Patient was treated symptomatically with antitussives.  Patient was a candidate for monoclonal antibody which was ordered for the patient.  However patient declined that.  Patient was found to have UTI and was started on antibiotic therapy initially with IV ceftriaxone but at the time of discharge he was switch to oral Keflex.  Patient was discharged home
Age: 66.0
Sex: F
Vaccine: UNK
Symptoms: Fatigue, Pain, Pyrexia
Clinical narrative: fatigue the next morning lasting 48 hrs, fever 24 hrs after vaccine lasting 16 hrs, bodyaches. 24 hrs after vaccine lasting 28 hrs.
Age: 94.0
Sex: F
Vaccine: COVID19
Symptoms: Atelectasis, Atrial fibrillation, COVID-19, Chest X-ray abnormal, Cough
Clinical narrative: because of worsening shortness of breath for last 24 hours.  The patient had recent diagnosis of atrial fibrillation with RVR.  She was tested positive for COVID- 19 in January 2022.  The patient reported poor appetite lately, intermittent dry cough, but no fever, no chills.  In the ER, her O2 saturation was 100% on 2 L nasal cannula.  Chest x-ray showed worsening left-sided pleural effusion. The patient was admitted for further evaluation. atelectasis, small pleural effusion.   She has recent COVID-19 infection 1/19/22 Repeated covid test  2/5 is still positive   Procalcitonin 0.1. She was discharged on 02/07/22 in stable condition.
Age: 94.0
Sex: F
Vaccine: COVID19
Symptoms: Decreased appetite, Dyspnoea, Pleural effusion, Procalcitonin, SARS-CoV-2 test positive
Clinical narrative: because of worsening shortness of breath for last 24 hours.  The patient had recent diagnosis of atrial fibrillation with RVR.  She was tested positive for COVID- 19 in January 2022.  The patient reported poor appetite lately, intermittent dry cough, but no fever, no chills.  In the ER, her O2 saturation was 100% on 2 L nasal cannula.  Chest x-ray showed worsening left-sided pleural effusion. The patient was admitted for further evaluation. atelectasis, small pleural effusion.   She has recent COVID-19 infection 1/19/22 Repeated covid test  2/5 is still positive   Procalcitonin 0.1. She was discharged on 02/07/22 in stable condition.
Age: 57.0
Sex: F
Vaccine: COVID19
Symptoms: Heart rate increased, Insomnia, Tinnitus
Clinical narrative: The night I received my booster dose of Moderna Covid -19 vaccine, I couldn?t sleep due to loud and rapid heartbeat (greater than 100 beats per minute). Since the vaccine, I have pulsatile tinnitus, especially in my right ear, sounding like a swooshing with every heartbeat when I am in a quiet environment.
Age: 57.0
Sex: F
Vaccine: COVID19
Symptoms: Asthenia, Dyspnoea, Feeling abnormal, Malaise, Pain in extremity
Clinical narrative: Following the shot I felt a bit ill and some arm soreness, but understood that was expected. The arm pain has persisted and is considreable, intermittent pain with occassional loss of strength. It is so sudden and can take my breath away. It is sometimes accompanied by a popping feeling and sound. Prior to the shot my arm was 100% in good shape and I have had no injuries or exceptional use of it.
Age: 46.0
Sex: F
Vaccine: COVID19
Symptoms: Gingival swelling, Lip swelling, Mouth injury, Stomatitis, Tongue injury
Clinical narrative: Gingivostomatitis. Swollen lips, swollen gums, lacerations on gums and tongue x two weeks and continuing
Age: 28.0
Sex: M
Vaccine: UNK
Symptoms: Fatigue, Headache, Pyrexia
Clinical narrative: Low grade fever Headache Tiredness
Age: 54.0
Sex: F
Vaccine: COVID19
Symptoms: Body temperature increased, Breast swelling, COVID-19, Chills, Fatigue
Clinical narrative: 1.) POSITIVE for COVID with home antigen test on June 6, 2022.  Began with a sore throat about 5 days prior, then worse chills, low fever, fatigue.  I had a home test and a molecular test on June 3 and was negative at the time.  2.) swollen and painful lymph nodes under axilla of same ar that received vaccine shot (Left).  Had a mammogram a few days after vaccine and very swollen in axilla and breast region.  Radiologist said NO signs of breast cancer but I have to follow up in 6 to 12 weeks with another diagnostic mammogram to make sure the swelling went down.  The radiologist said she sees this often after vaccination.
Age: 54.0
Sex: F
Vaccine: COVID19
Symptoms: Lymph node pain, Lymphadenopathy, Mammogram abnormal, Oropharyngeal pain, Peripheral swelling
Clinical narrative: 1.) POSITIVE for COVID with home antigen test on June 6, 2022.  Began with a sore throat about 5 days prior, then worse chills, low fever, fatigue.  I had a home test and a molecular test on June 3 and was negative at the time.  2.) swollen and painful lymph nodes under axilla of same ar that received vaccine shot (Left).  Had a mammogram a few days after vaccine and very swollen in axilla and breast region.  Radiologist said NO signs of breast cancer but I have to follow up in 6 to 12 weeks with another diagnostic mammogram to make sure the swelling went down.  The radiologist said she sees this often after vaccination.
Age: 54.0
Sex: F
Vaccine: COVID19
Symptoms: SARS-CoV-2 test positive
Clinical narrative: 1.) POSITIVE for COVID with home antigen test on June 6, 2022.  Began with a sore throat about 5 days prior, then worse chills, low fever, fatigue.  I had a home test and a molecular test on June 3 and was negative at the time.  2.) swollen and painful lymph nodes under axilla of same ar that received vaccine shot (Left).  Had a mammogram a few days after vaccine and very swollen in axilla and breast region.  Radiologist said NO signs of breast cancer but I have to follow up in 6 to 12 weeks with another diagnostic mammogram to make sure the swelling went down.  The radiologist said she sees this often after vaccination.