Age: 1.42
Sex: M
Vaccine: MMR, DTP, HIBV, IPV, MMR
Symptoms: Sinusitis, Skin laceration, Somatosensory evoked potentials, Speech disorder developmental, Tympanometry normal
Clinical narrative: Information has been received regarding a case in litigation concerning a minor male pt who was exposed to high levels of mercury from thimerosal through exposure to unspecified products. In additional, the pt was exposed to mercury from continually inhaling and ingesting industry mercury emissions from fossil fuel driven power plants. The mercury included in thimerosal laden products and power plant emissions together caused the pt to suffer from serious and permanent neurological injuries. The pt was exposed to airborne mercury vapor and mercury through food chain. The exposure to airborne mercury in the environment through inhalation, the food chain, and other sources contributed to the cumulative mercury toxicity and was a substantial factor in causing his neurological damage. it was noted that the pt had a heightened vulnerability to suffer neurological injuries from industrial mercury as a direct result of thimerosal exposure. As a direct and proximate cause of the toxic mercury exposure to  the pt, minor male was caused to suffer serious and permanent neurological damage which included past and future mental, intellectual, development and neurological incapacity and associated learning disabilities, disorders and impairments. Follow up information has been received from medical records concerning a 20 month old male whose mother toxaemia during pregnancy and who has a history chickenpox, colic, URI, gastroenteritis, GERD, hospitalisation for right inguinal hernia, slight obesity, otitis serious, allergic rhinitis, and a family history of diabetes, cancer, cardiovascular disorder, kidney failure, and epilepsy who vaccinated with a first and second dose of MMR II on 5/22/1992 and 8/22/1995. Concomitant vaccination on 8/22/1995 included fifth dose of DPT, a fourth dose of Polio virus vaccine and a third dose of PPD. Illness at the time of vaccination included tinea capitis. On 4/28/1994 the pt was seen for a psychological evaluation and was diagnosed with hyperactivity, oppositional defiant behavior, language delay and separation anxiety. On 4/24/1996 the pt was considered to be a child who had a non progressive encephalopathy due to some type of organic brain problem. On 5/10/1996 the pot underwent an MRI of the brain which was normal. On 11/24/1999 the pt was diagnosed with Autism and was treated with Prozac 1.5 cc every morning and Ritalin 1/2 tablet in the morning for one week than 1 tablet every morning and one in the afternoon. On 11/1/1999 it was noted the the pt had been diagnosed with pervasive developmental delays and ADHD. It was noted that he had a history of seizures starting in 1995. On 11/01/99 the pt was seen for a central auditory test. The results were consistent with a significant auditory processing disorder. On 4/3/01 it was noted that the pt was treated with Paxil 10 mg daily. On 2/4/02 it was noted that the pt was currently on therapy with Zoloft. On 3/27/02 the pt was seen in the neurology clinic. It was reported that the pt could not be clearly diagnosed as having wither Asperger syndrome or high functioning autism. The central auditory features were typical of Asperger syndrome. The language delay was more constant with high functioning autism. It was noted that he may fall in the gray zone between the two diagnoses. It was noted that he was on therapy with Zoloft 50 mg QHS and Melatonin 3 mg (which discontinued after this visit). On 5/17/04 the pt was seen in follow up at the neurology clinic for autism, apparently high functioning. At the time of the report the pts outcome was not recovered with the exception of seizure which was unknown. Follow up information received on 9/12/06 contained the following adverse experiences, diarrhea 8/20/92, vomiting 8/27/92, sinusitis 9/14/95, fall resulting in scalp laceration 1/13/96, significant conductive hearing loss, particularly on right side approx 6/18/96, adenoid hypertrophy hospitalized 7/15/96, cough 8/20/99, pharyngitis 8/23/99, slight obese 1/31/02, poor sleep 11/25/03, frequent urination 11/25/03 and foul breath onset unknown. Upon internal review cumulative mercury toxicity, language delay, non progressive encephalopathy, significant auditory processing disorder, pervasive developmental delays, ADHD, autism and seizures were considered to be other important medical events (OMIC). The pts learning disability was considered to be disabling. No further information is available. All available medical records will be provided upon request.