bluuebunny/crossref_metadata_2025_split huggingface dataset:
Analisis Pengendalian Kualitas Produk Tiang Pancang di PT. X dengan Menggunakan Metode Failure Mode and Effect Analysis (FMEA)
Authors: Alda Tania , Sukma Ompusunggu Andika , Shalihin Ahmad , Elsanna Revadi Chindy , Ramadhani Putri Nasution Fadylla , Cevania Purba Naomi 
Published: February 2024

This research was conducted at a company engaged in the precast concrete industry. The company produces and distributes piling products both domestically and abroad. The quality of pile products made by the company is one of the considerations of consumers. For this reason, the company must provide the best quality by utilizing all production factors as efficiently as possible. This is done to maintain consumer confidence and reduce the number of products that must meet company standards. This research aims to find the leading cause of defects in pile products. The method used is failure mode and effect analysis (FMEA). The FMEA method will look at the factors that cause defects based on the highest risk priority number (RPN). From the research results obtained, four factors cause defects in pile products, namely human factors, machine factors, material factors and method factors. Based on calculating the priority risk value in the FMEA method, the RPN value results are Machine Factors of 192, Material Factors of 180, Human Factors of 120, and Method Factors of 75. The factor causing the highest pile product defects is the machine factor, with an RPN value of 192. Based on this, it is known that the spinning machine used is too old and many machine components need to be replaced, so the proposed improvements given are to carry out regular and scheduled machine checks and maintenance and immediately repair or replace damaged machine components.
Inmigración, racismo y sueñitos en En un barrio de Nueva York (In the Heights, 2021). Una aproximación semiótico-culturalista al número musical Paciencia y fe
Authors: Palomares Navarro Óscar 
Published: February 2024

Como cualquier manifestación cultural, el cine musical clásico estadounidense se desarrolló en unas coordenadas sociales e históricas concretas. Pero, históricamente, este género ha excluido la representación de realidades como las desigualdades socioeconómicas relacionadas con el paradigma racista hegemónico desde el que se piensa la diversidad cultural. Las nuevas fórmulas del cine musical introducen dichas coordenadas sociales, históricas y culturales. El presente artículo se centra en cómo En un barrio de Nueva York (In the Heights, 2021), y particularmente el personaje de la Abuela Claudia y su número musical Paciencia y fe, plantea dinámicas de apropiación de las fórmulas genéricas del musical, mostrando discursos de resistencia asociados a temas como la migración o el racismo
Deep Learning Approach for Classification of Tweets in Detecting Cyber Truculent
Authors: Olaitan Olawale Lukman , David Adeniji Oluwashola , Michael Odejayi Adeniyi 
Published: February 2024

Identification and Classification of truculent tweets is a serious problem. Studies have shown how truculent tweets on Twitter have caused anxiety and in extreme cases death of victims. Various researches have applied Machine Learning approaches while others Deep Learning in classifying toxic sentences. Impressive results have been obtained using Deep Learning approaches, the focus of the research is to develop an extended model for deep learning classification using cyber tweets. The Model developed in this research work used labelled dataset (twitter_parsed_dataset.csv), Maximum Entropy was used to extract keywords and entities. One-Dimensional Convolutional Neural Network (1d-CNN) was used to detect truculent in tweets. The experimental result from the developed model consider four preprocess datasets for classification, the Unigram, Bigram, Trigram and N-gram. The result that was obtained for Accuracy 96.1%, Precision 93.6%, Recall 73.7% and F1-Score 83.8% for different test during classification. The result obtained from the developed model for accuracy, 96.1% was compared with related work Banerjee’s accuracy of 93.97%. The developed model can be used for auto detection of truculent messages in cyberspace.
Hemothorax Due to Transdiaphragmat-ic Liver Penetrating Injury Caused by Broken Knife
Authors: Emre Tunca I. , Çağlar Şahin , Emrah Cengiz , Ali Nehir , Sinem Bayrakçı , Semih Uğuz , Tuna Çelik , Uğur Şener , Onur Bayrakçı 
Published: February 2024

Thoracic trauma is one of the most fatal cases of patients admitted to emergency departments. Thoracic trauma occurs in 20–25% of all trauma patients worldwide and is the third cause of trauma-related deaths. Approximately 5% of trauma admissions are liver injuries. In some blunt or penetrating injuries, both intrathoracic and intra-abdominal organs may be injured. It is aimed to present the case of a broken knife entering from the right paravertebral location, causing hemothorax as a result of diaphragm and liver injury without injuring the lungs, due to the importance of the multidisciplinary approach among surgical departments in the study.
A 44-year-old male patient was admitted to the emergency department due to penetrating injury. An oblique injury of approximately 5 cm was seen in the posterior part of the right hemithorax. Radiologically, It was detected that the knife, which entered through the 10th intercostal space, was stuck inside and the knife handle was broken. It was observed that the broken blade entered from the paravertebral region, passed into the intrathoracic area, causing hemothorax, and passed from the edge of the inferior vena cava and embedded itself in the liver. Multidisciplinary operation planning was made among the departments. All department surgeons were present in the operating room at the same time. The paravertebral space was assessed and the embedded blade was slowly withdrawn by the neurosurgeon. Explorative laparotomy was done by the general surgeon and gastroenterology surgeon. Transdiaphragmatic injury was evaluated by the thoracic surgeon and major vascular structures were evaluated by the cardiovascular surgeon. In the postoperative period, the patient was discharged on the 5th postoperative day with full recovery without any complications.
Penetrating trauma is one of the most fatal cases of patients admitted to emergency departments. In some cases, intrathoracic and intra-abdominal penetrating injury may occur together, rarely from a single entrance location. The timing of critical intervention varies depending on whether the patient's vitals are stable or unstable. If the hemodynamics are stable in penetrating injuries, all affected anatomical structures should be evaluated with radiological examinations. For successful, uncomplicated surgical treatments, controlled techniques, interdepartmental consensus and, when necessary, simultaneous presence of surgeons in the surgery are essential
Electrosynthesis of NbTi and Nb3Sn Superconductors from Oxide Precursors in CaCl2‐Based Melts
Authors: Yan X. Y. , Fray D. J. 
Published: October 2005

AbstractNbTi and Nb3Sn superconductors have been synthesized directly from the oxides by electro‐deoxidation, where the cathode is made by placing an intimate mixture of the sintered oxide in a molten bath of calcium chloride and sodium chloride. The favored cathodic reaction is the removal of the oxygen from the oxides to form the superconducting intermetallic alloys and not the deposition of calcium. The superconducting properties of the intermetallic compounds are confirmed.
Ultrarapid Engineering of Biomimetic Materials and Tissues: Fabrication of Nano‐ and Microstructures by Plastic Compression
Authors: Brown R. A. , Wiseman M. , Chuo C.‐B. , Cheema U. , Nazhat S. N. 
Published: October 2005

AbstractCurrently, the concept of engineered tissues depends on the ability of cultured cells to fabricate new tissue around a scaffold. This is inherently slow and expensive and has had limited success so far. We report here a new process for the cell‐independent, controlled engineering of biomimetic scaffolds by rapid removal of fluid from hyperhydrated collagen gel (or other) constructs, using plastic compression (PC). PC fabrication produces dense, cellular, mechanically strong native collagen structures with controllable nano‐ and microscale biomimetic structures. The huge‐scale shrinkage (> 100‐fold) provides the ability to introduce controllable mechanical properties, microlayering, and embossed interface topography without cell participation, but with high cell viability. Critically, this takes minutes rather than the conventional days and weeks. The rapidity and biomimetic potential of the PC fabrication process at the mesoscale opens a new route for the production of biomaterials and patient‐customized tissues. It also represents a new concept in ‘engineering’ tissues.
Zinc Oxide Nanoparticles with Defects
Authors: Ischenko V. , Polarz S. , Grote D. , Stavarache V. , Fink K. , Driess M. 
Published: October 2005

AbstractZinc oxide in the form of nanoscale materials can be regarded as one of the most important semiconductor oxides at present. However, the question of how chemical defects influence the properties of nanoscale zinc oxide materials has seldom been addressed. In this paper, we report on the introduction of defects into nanoscale ZnO, their comprehensive analysis using a combination of techniques (powder X‐ray diffraction (PXRD), X‐ray absorption spectroscopy/extended X‐ray absorption fine structure (XAS/EXAFS), electron paramagnetic resonance (EPR), magic‐angle spinning nuclear magnetic resonance (MAS‐NMR), Fourier‐transform infrared (FTIR), UV‐vis, and photoluminescence (PL) spectroscopies coupled with ab‐initio calculations), and the investigation of correlations between the different types of defects. It is seen that defect‐rich zinc oxide can be obtained under kinetically controlled conditions of ZnO formation. This is realized by the thermolysis of molecular, organometallic precursors in which ZnO is pre‐organized on a molecular scale. It is seen that these precursors form ZnO at low temperatures far from thermodynamic equilibrium. The resulting nanocrystalline ZnO is rich in defects. Depending on conditions, ZnO of high microstructural strain, high content of oxygen vacancies, and particular content of heteroatom impurities can be obtained. It is shown how the mentioned defects influence the electronic properties of the semiconductor nanoparticles.
Ferrocene‐Containing Optically Active Liquid‐Crystalline Side‐Chain Polysiloxanes with Planar Chirality
Authors: Brettar J. , Bürgi T. , Donnio B. , Guillon D. , Klappert R. , Scharf T. , Deschenaux R. 
Published: October 2005

AbstractOptically active liquid‐crystalline side‐chain polysiloxanes have been prepared by grafting planar chiral ferrocene‐based vinyl monomers onto commercially available polyhydrosiloxane. Two ferrocene monomers have been synthesized: a linear‐type monomer, which displays a monotropic chiral smectic C (SC*) phase and enantiotropic smectic A (SA) and chiral N (N*) phases, and a laterally branched monomer, which shows an enantiotropic N* phase. X‐ray diffraction analysis indicates a monomolecular organization of the monomeric units within the smectic layers. The polymers retain the liquid‐crystalline phases of their corresponding monomers. The UV‐vis and circular dichroism (CD) spectra are in agreement with the structure of the monomers and polymers. The molar absorption coefficient (ϵ) and molar circular dichroic absorption coefficient (Δϵ) values of the polymers are proportional to the number of monomeric units grafted onto them. The absolute configuration of the ferrocene carboxylic acid intermediate, used to synthesize the monomers, has been determined on the basis of CD spectra. The helical twisting power (HTP) of the nematogenic monomer and polymer have been determined in E7, and indicate that such materials could be used as chiral dopants. Finally, this study demonstrates that the nature of chiral phases can be controlled by structural engineering of the organic groups only, with ferrocene acting as the source of chirality.
Automatic reaction network generation using RMG for steam cracking of n‐hexane
Authors: Van Geem Kevin M. , Reyniers Marie‐Francoise , Marin Guy B. , Song Jing , Green William H. , Matheu David M. 
Published: October 2005

AbstractA new reaction mechanism generator RMG is used to automatically construct a pressure dependent kinetic model for the steam cracking of n‐hexane. Comparison between simulated and pilot plant data shows that RMG is able to generate detailed reaction networks that accurately predict the conversion and the yields of the major products although none of the kinetic parameters are fit to the experiments. RMG generates reaction networks based on minimal assumptions, making it possible to test commonly used assumptions such as the μ‐hypothesis and the quasi steady‐state approximation (QSSA) for μ‐radicals, traditionally used in steam cracking,1, 2 as well as in pyrolysis.3 The RMG‐reaction network for n‐hexane confirms that no bimolecular reactions of heavy radical species are important at the examined conditions (COT: 953 K ‐ 1090 K; COP: 0.20 MPa ‐0.24 MPa; <80% conversion), and that the QSSA for the group of μ‐radicals leads to negligible errors. RMG also offers the possibility to estimate the error introduced by neglecting the pressure dependence of most of the reactions. In the case studied, this frequently made (but seldom tested) approximation appears to be justified. © 2005 American Institute of Chemical Engineers AIChE J, 2006
Cleidocranial dysplasia: Molecular genetic analysis and phenotypic‐based description of a Middle European patient group
Authors: Baumert Uwe , Golan Ilan , Redlich Meir , Aknin Jean‐Jacques , Muessig Dieter 
Published: October 2005

AbstractCleidocranial dysplasia (CCD) (OMIM 119600) is a rare dysplasia of osseous and dental tissue. Characteristic features are typical facial and dental appearance plus morphologic anomalies. RUNX2 (OMIM 600211), the responsible gene for CCD, is considered to be a master gene for bone development and bone homeostasis. This study describes the genotype–phenotype correlation based on craniofacial features involving an interdisciplinary approach. Our patient cohort consisted of 31 CCD patients from 20 families; five patients from two families were unavailable for clinical examination. Since CCD mostly affects the craniofacial region, phenotypic characterization of each individual focused on craniofacial and dental aspects. After recording patient medical and family history, the phenotypic data was analyzed using homogeneity analysis (HOMALS), a statistical procedure for data reduction in categorical data analysis. The coding sequence of the RUNX2 gene was analyzed using PCR, direct sequencing, and restriction endonuclease digestion. Eight unpublished and four known heterozygous mutations in a total of 14/20 index patients (70%) were identified. In total, we detected 7 missense mutations, 5 frameshift mutations, and 2 nonsense mutations in 14 index patients (35%, 25%, 10%, respectively). The overall CCD phenotype varied from mild to fullblown expression. Using HOMALS, we were able to discriminate four groups of patients showing significant differences in phenotypic expressivity, thereby simplifying the grouping of our large patient cohort into clear distinguishable entities. Analysis of the mutation patterns revealed that mutational frequency and types of mutations found can be attributed to the gene's structure and function. © 2005 Wiley‐Liss, Inc.
Sensorineural hearing loss in children and adults with Williams syndrome
Authors: Marler Jeffrey A. , Elfenbein Jill L. , Ryals Brenda M. , Urban Zsolt , Netzloff Michael L. 
Published: October 2005

AbstractWilliams syndrome (WS) is a genetic neurodevelopmental disorder, most often accompanied by mild‐to‐moderate mental retardation. Individuals with WS show unique communication strengths and impairments that are challenging to treat in community, educational, and vocational settings. Many issues regarding characteristics of auditory sensitivity in WS remain to be resolved. Our purpose was to obtain behavioral (screening and pure‐tone audiometry) and objective (distortion product otoacoustic emission—DPOAE) measures of auditory system function from a group of 27 individuals with WS, 6–48 years of age. These measures were gathered both at an international professional conference (n = 19) and in a clinic setting (n = 8). In the behavioral screening conditions, 16/19 (84%) of the individuals failed the hearing screening; and in the behavioral diagnostic hearing condition, 6/8 (75%) demonstrated sensorineural hearing loss (SNHL) and 1/8 demonstrated a hearing loss of undetermined type. In the objective DPOAE testing, 19/25 (76%) had DPOAE absolute amplitudes below the 5th percentile for ears with normal hearing [Gorga et al. (1997); Ear Hear 18(6):440–455]. We report SNHL in 14/18 (78%) of school‐age children with WS. Post hoc analyses revealed a significant effect for age, suggesting a pattern of progressive hearing loss. An effect size analysis indicated a clinically meaningful difference in the hearing sensitivity between school‐aged children and adults in the high frequencies (4,000 and 8,000 Hz). Similar hearing loss phenotype was observed in patients with familial nonsyndromic supravalvular aortic stenosis (SVAS), suggesting that molecular defects in the elastin gene in the pathogenesis of SNHL in WS. This study highlights the importance of early and regular hearing testing for WS patients and suggests that elastin may have a previously unappreciated function in maintaining hearing sensitivity. © 2005 Wiley‐Liss, Inc.
Familial gigantism caused by an NSD1 mutation
Authors: van Haelst Mieke M. , Hoogeboom Jeannette J.M. , Baujat Genevieve , Brüggenwirth Hennie T. , Van de Laar Ingrid , Coleman Kim , Rahman Nazneen , Niermeijer Martinus F. , Drop Sten L.S. , Scambler Peter J. 
Published: October 2005

AbstractA three‐generation family with autosomal dominant segregation of a novel NSD1 mutation (6605G → A, resulting in Cys2202Tyr) is reported. Haploinsufficiency of NSD1 has been identified as the major cause of Sotos syndrome. The overgrowth condition (MIM 117550) is characterized by facial anomalies, macrocephaly, advanced bone age, and learning disabilities. Manifestations in the present family include dramatically increased height, weight, and head circumference together with a long face, large mandible, and large ears, but mental deficiency was absent. © 2005 Wiley‐Liss, Inc.
Medial temporal lobe dysgenesis in hypochondroplasia
Authors: Kannu Peter , Hayes Ian M , Mandelstam Simone , Donnan Leo , Savarirayan Ravi 
Published: October 2005

AbstractWe describe two patients who have hypochondroplasia with medial temporal lobe dysgenesis. This association has only been reported once before. Both patients had an FGFR3 mutation: 1620C → A, resulting in Asn540Lys. FGFR3 is expressed in the brain during development and plays a role in hippocampal formation. We suggest FGFR3 mutations might cause cerebral malformations in hypochondroplasia as well as in thanatophoric dysplasia. Further neuroimaging studies of patients with hypochondroplasia and epilepsy or developmental delay may clarify the proportion of patients with hypochondroplasia who have this pattern of central nervous system abnormalities. © 2005 Wiley‐Liss, Inc.
Homozygosity for a gross partial gene deletion of the C‐terminal end of ATP7B in a Wilson patient with hepatic and no neurological manifestations
Authors: Møller Lisbeth Birk , Ott Peter , Lund Connie , Horn Nina 
Published: October 2005

AbstractWe identified a partial gene deletion of ATP7B in a patient with Wilson disease with hepatic onset. The deletion covered exon 20 including major parts of the flanking introns. The breakpoints were identified and the size of the deletion determined to be 2144 bp. The deletion is predicted to lead to a mutated protein product containing 45 aberrant amino acids after transmembrane domain 7, and lacking the transmembrane domain 8 as well as the entire C‐terminal cytoplasmic tail. This is the first time a partial gene deletion has been demonstrated in ATP7B. The patient presented at age 10 with hepatic manifestations, including severe jaundice, hepato‐splenomegaly, ascites, and spider naevi. The liver biopsy showed fibrosis and early signs of cirrhosis. There was a Kayser–Fleischer ring but no neurological manifestations. All symptoms disappeared with penicillamine therapy. This suggests that the C‐terminal cytoplasmatic tail of ATP7B, is not essential for its neurological function. Large deletions in ATP7B may be an overlooked cause of Wilson disease. Patients that are homozygotes for deletions may be valuable for the understanding of the function of various regions of the ATP7B protein. © 2005 Wiley‐Liss, Inc.
Histopathology and fibrillin‐1 distribution in severe early onset Marfan syndrome
Authors: Summers K.M. , Nataatmadja M. , Xu D. , West M.J. , McGill J.J. , Whight C. , Colley A. , Adès L.C. 
Published: October 2005

AbstractMarfan syndrome (MFS) is an autosomal dominant condition which may involve the cardiovascular, ocular, skeletal, and other systems. Mutations causing MFS are found in the FBN1 gene, encoding fibrillin‐1, an extracellular matrix protein involved in microfibril formation. In the most severe cases, mutations are generally found in exons 24–32, and children with these mutations usually die in the first years of life, of cardiopulmonary failure. We present clinical, molecular and histopathological studies on a patient with severe early onset MFS. He has a mutation in exon 25 of FBN1, a G>A transition at nucleotide position 3131 that converts the codon TGC, coding for cysteine at position 1044, to TAC, coding for tyrosine (C1044Y). This has resulted in abnormalities of the extracellular matrix and a severe clinical phenotype, although he has survived to the age of 14 years. © 2005 Wiley‐Liss, Inc.
New approach for the refinement of the location of the X‐chromosome breakpoint in a previously described female patient with choroideremia carrying a X;4 translocation
Authors: García‐Hoyos M. , Sanz R. , Diego‐Álvarez D. , Lorda‐Sánchez I. , Trujillo‐Tiebas M.J. , Cantalapiedra D. , Ramos C. , Ayuso C. 
Published: October 2005

AbstractChoroideremia (CHM) is an X‐linked recessive ophthalmic disease characterized by a progressive degeneration of the choroid and the pigmented epithelium of the retina. We present the genetic characterization of a female patient affected with CHM who has been previously studied cytogenetically and showed a balanced translocation between chromosomes X and 4 [46,X,t(X;4)(q21;p16)]. The breakpoint in the X chromosome lies in the locus of CHM gene and for this reason, we have elucidated whether or not CHM was disrupted in the X chromosome involved in the translocation using different techniques. FISH showed that the 3′UTR and the last exons of the CHM were on the der(X) chromosome, and the 5′UTR and first exons of this gene were on the der(4) chromosome. Expression level analysis revealed that the breakpoint in the der(X) was located between exons 8 and 9 of the CHM gene because the expression level decreased from this point onwards. Based on this result the expression level analysis proved to be a valid method to pinpoint the location of breakpoints when the gene being expressed in peripheral blood is disrupted. Our results confirmed that the CHM gene was indeed disrupted in the X chromosome involved in the translocation. Besides, the nonrandom inactivation of the normal X chromosome observed using a methylation‐specific polymerase chain reaction (M‐PCR) technique explained the CHM in the female patient. © 2005 Wiley‐Liss, Inc.