bluuebunny/crossref_metadata_2025_split huggingface dataset:
IMMUNOGLOBULIN ALLOTYPES IN EUROPEAN POPULATIONS. IV. Gm, Am AND Km ALLOTYPIC MARKERS IN VALENCIA, SPAIN
Authors: Schanfield* M. S. , Baylerian* R. , Maiquez J. , Carbonell F. 
Published: April 2007

SUMMARYThe distribution of Glm(f, z, a and x), G3m(b0, b1, b3, b5, c3, c5, g, s and t), A2m (1 and 2) and Km(1) allotypic determinants in a series of Spanish blood donors. The following results were obtained: Gmz,a;gAm1= 0.199; Gmz,a;gAm2= 0.019; Gmz,a;bAm1= 0.081; Gmz,a,x;gAm2= 0.005; Gmf;bAm1= 0.677; Gmz,a;bAm1= 0.019 and Km1= 0.096. The results are similar to those reported for adjacent Basque and Portuguese populations. The origin of the Gmz,a;bAm1 haplotype is unknown.
GENETIC AND SEROLOGIC RE‐EVALUATION OF LyM‐1 ANTIGEN SPECIFIED BY A GENE CLOSELY LINKED TO Mls: ESTABLISHMENT OF LyM‐1 ANTIGEN OF THE MOUSE
Authors: Sato H. , And S. Kimura , Itakura* K. 
Published: April 2007

SUMMARYAn alloantiserum prepared against LyM‐1 antigen of the mouse has been analysed in detail. This antiserum, C3H/HeJ anti‐CBA/J, has turned out to define two antigens which are specified by independently segregating loci. One of these is Lyb‐2.1 or similar antigen specified by a gene located on chromosome 4. The other is identified as LyM‐1.2 based on the result that a gene determining alloantigenicity in the Lyb‐2‐ strain is closely linked to Mls with recombination frequency of 0.066 ± 0.028. Since both antigens are expressed preferentially on B lymphocytes and their strain distribution is significantly overlapped, there has been much confusion about LyM‐1 antigen. This is overcome by re‐evaluating anti‐LyM‐1.2 serum under the condition in which Lyb‐2.1 is not involved in analysis. The strain distribution of LyM‐l antigen, its preferential expression on B lymphocytes and close genetic linkage of LyM‐l to Mls have been established.
CHARACTERIZATION OF THE RT2 ANTIGENS OF THE RAT ERYTHROCYTE MEMBRANE
Authors: Misra D. N. , Gill T. J. , Kunz H. W. 
Published: April 2007

SUMMARYHaemagglutination inhibition studies using isolated membrane components from rat red blood cells showed that the activity of the major antigenic system RT2 was contained exclusively in the glycoprotein fraction and confirmed immunochemically the existence of two alleles, RT2a and RT2b. The SDS‐PAGE analysis of the membrane showed two interconvertible glycoproteins of apparent molecular weight 72,000 and 36,000, and the higher molecular weight component appeared to be a dimer of the lower molecular weight one. Analysis of the various membrane preparations after 125I‐labelling showed that the glycoproteins existed in at least two molecular species on the surface of the red cells and that one species was more readily radio‐iodinated and dimerized than the other. The RT2 antigenic specificities were preferentially associated with this component.
DIFFERENTIAL SUSCEPTIBILITY OF SPLEEN CELLS FROM C3H/HeN (H‐2k) AND C3HfB/HeN (H‐2kv1) MOUSE STRAINS TO CYTOTOXICITY BY MONOCLONAL ANTI‐Kk ANTIBODY (CLONE 11‐4)
Authors: Martin W. J. , Imamura M. 
Published: April 2007

SUMMARYSpleen cells from C3HfB/HeN mice (H‐2kv1) were markedly less susceptible to lysis by monoclonal anti‐Kk antibody (Clone 11‐4) than spleen cells from C3H/HeN mice (H‐2k). Less than 50% of C3HfB/HeN cells were lysed even at the highest antibody concentration tested compared to 70% of C3H/HeN cells. The difference in end point titre of the antibody tested on cells from the two strains was 5‐10‐fold. Similar differences were not observed using the conventional H‐2Kk‐typing antiserum (D‐23).
UNUSUAL HEAVY CHAINS OF HUMAN IgG IMMUNOGLOBULINS: REARRANGEMENTS OF THE CH DOMAIN EXONS
Authors: Lefranc G. , Lefranc Marie Paule , Helal A. N. , Boukef K. , Chaabani H. , Gandoura M. Sfar , Loghem Erna van 
Published: April 2007

SummaryUnusual combinations—unexpected sets, excess or lack—of antigenic determinants, or Gm allotypes, on the constant regions of the heavy chains of the human IgG1 and IgG3 immunoglobulins are accounted for in terms of genetic events (exchanges, duplications and deletions) involving the DNA sequences, or exons, coding for the three CH1‐, CH2‐ and CH3 domains of the γ1 and γ3 chains. Equal and unequal cross‐overs at the level of the introns without damage to the CH exons are postulated.
ANALYSIS OF UNEXPECTED INHIBITIONS OF T LYMPHOCYTE PROLIFERATION TO SOLUBLE ANTIGEN, ALLOANTIGEN AND MITOGEN BY UNFRAGMENTED ANTI‐I‐Ak OR ANTI‐I‐E/Ck MONOCLONAL ANTIBODIES
Authors: Dubreuil P. C. , Caillol Danielle H. , Lemonnier F. A. 
Published: April 2007

SummaryWe investigated the capacity of anti‐I‐Ak and anti‐I‐E/Ck monoclonal antibodies (m.Ab.) to inhibit T lymphocyte proliferative responses to soluble antigen (Keyhole limpet hemocyanin), alloantigens (H‐2 or non‐H‐2 related) and a mitogen (Concanavalin A). Surprisingly, specific inhibition was observed in all circumstances, and with both anti‐I‐Ak and anti‐I‐E/Ck m.Ab., whether the responses tested were I restricted in cell mixing experiments or not.The significance of the inhibition by anti‐Ia m.Ab. of non‐Ia‐restricted responses is still not completely understood. These results, however, strongly suggest that in vitro inhibition by anti‐Ia antibodies of T cell proliferative responses does not necessarily indicate I restriction of the presentation to T lymphocytes of the corresponding antigen.
QUANTITATIVE INHERITANCE OF HETEROPHILE RHEUMATOID FACTOR RESPONSIVENESS IN MICE
Authors: Dresser D. W. 
Published: April 2007

SummaryA heterophile rheumatoid factor (RF) response to LPS was measured in mice by means of a plaque assay using target cells coated with bovine IgG. CBA mice were high responders and A, and DBA/2 were low responders, while strains C57B1, and LP/J showed low to intermediate responsiveness. Use of appropriate congenic strains confirmed that responsiveness is not closely associated with either H‐2 or Igh. Failure to respond is not likely to be due to a V‐region deficiency, nor can it be readily circumvented, by heterosis (complementation) or the use of high doses of LPS or an extra virulent form of LPS. Breeding experiments show a quantitative trait with maternal responder status having a measurably positive effect.
IMMUNOGLOBULIN ALLOTYPES IN PATIENTS WITH GIANT CELL ARTERITIS AND POLYMYALGIA RHEUMATICA
Authors: Pandey J. P. , Hunder G. G. , Fudenberg H. H. , Loadholt C. B. 
Published: April 2007

SummarySeveral Gm, A2m, and Km allotypes were examined in Caucasian patients with giant cell arteritis and polymyalgia rheumatica. No significant associations were found for any of the allotypes tested.
ATYPICAL TRANSMISSION OF ABO BLOOD GROUPS IN A FRENCH FAMILY
Authors: Badet J. , Lopez M. , Habibi B. , Cartron J. P. , Noel A. , Salmon D. , Seger J. , Salmon C. 
Published: April 2007

SummaryA group AB mother (Mrs P.D.) gave birth to a group O female baby (C.D.). Extensive study of the blood group genetic markers in both the parents and the child, carried out on several occasions, showed nothing unusual outside the ABO system. Mrs P.D. then, gave birth to a second female baby who was also group O. Mrs P.D. had normal amounts of A, B, H and Lewis antigens in her saliva. The H, A and B agglutinability of her red cells was in the range of normal A2B group. This A2B blood group was characterized by very low A gene‐specified glycosyltransferase activity in serum. Moreover this activity was undettable in red blood cell membranes. These results are discussed in the light of various hypotheses in order to explain this unusual transmission of ABO blood group.
DETECTION OF A t‐COMPLEX ANTIGEN BY SECONDARY CELL‐MEDIATED LYMPHOCYTOTOXICITY
Authors: Hammerberg C. 
Published: April 2007

SummaryBecause of the inconsistency in published results concerning the serological detection of cell surface antigens coded for by the t‐complex, a cell‐mediated lymphocytotoxicity (CML) assay, secondary CML, was used in a search for t‐antigens. By sensitizing C3H. Ttf (C3H. Brachyury, tufted) with the congenic strain C3H. Ttf/tw18 splenic cells, a response against lipopolysaccharide (LPS) stimulated splenic cells from C3H. Ttf/tw18 mice is obtained. The locus coding for the antigen detected by this reaction lies to the left of tf on the murine seventeenth chromosome. The secondary CML response to this antigen is H‐2 restricted and detects an antigen on all t‐haplotypes tested: tw18, tw18tf, t12, t6, th2tf, and tw5.
INHIBITION OF SECONDARY IgG RESPONSES BY MONOSACCHARIDES: EVIDENCE FOR I‐REGION CONTROL
Authors: Tomaska Luba D. , Parish Ch. R. 
Published: April 2007

SUMMARYCertain monosaccharides selectively inhibit secondary IgG responses in vitro. Genetic analyses described in this report revealed that the inhibitory sugars differed between mouse strains and these differences mapped to the I‐J and I‐C subregions of the murine MHC. These results imply that interaction between T and B lymphocytes can involve the recognition of I‐region controlled carbohydrate structures.
GENETIC CONTROL OF THE IMMUNE RESPONSE TO HAEMOGLOBIN: IV. Ly‐1+ T‐CELLS AND APPROPRIATE NON‐H‐2 GENES ARE REQUIRED FOR IN VITRO RESPONSES TO α ‐ AND β‐SUBUNITS OF HUMAN ADULT HAEMOGLOBIN
Authors: Krco C. J. , Kazim A. L. , Atassi M. Z. , David C. S. 
Published: April 2007

SUMMARYExperiments were conducted to determine if non‐H‐2 gene effects could be demonstrated in mice which had been primed to either the α‐subunit or β‐subunit of human haemoglobin. It was found that C3H.SW (H‐2b) and Balb/c (H‐2d) mice are low responder mice to α‐chain of a haemoglobin when compared to H‐2 identical B10 (H‐2b) and B10.D2(H‐2d) mice. B120.S and A.SW (both H‐2s) are responsive to β‐chain challenge while Balb/c mice are low responders in contrast to high responder B10.D2 mice. Ly‐1+ cells were demonstrated to be required (by cell depletion experiments) for an in vitro T‐cell proliferative response to either subunit. In these experiments, Ly‐2+ cells were not of crucial importance.
ALLOTYPE AND CHARGE‐RELATED DIFFERENCES IN THE IMMUNE RESPONSE
Authors: Notenboom, R. H. , Dubiski, S. , And B. Cinader , Good P. W. 
Published: April 2007

SUMMARYRabbits homozygous at the Ab kappa chain allotypic locus (Ab4/Ab4 and Ab9/Ab9) were immunized with negatively and positively charged antigens. The negatively charged antigens were bovine serum albumin (BSA; pI = 4.9), ovalbumin (OV; pI = 4.9) and two synthetic polypeptides: copolymer L‐glutamic acid L‐tyrosine (abbreviated TG; pI = 4.8) and multichain poly (L‐tyrosine: L‐glutamic acid) poly D‐alanine: poly L‐lysine (poly L‐lysine backbone) (abbreviated (TG) ‐ AL; pI = 4.8). The positively charged antigen was hen egg lysozyme (LYS; pI = 10.2).Homozygous Ab9 rabbits responding to negatively charged antigens made less antibody than did Ab4 homozygotes. In contrast, both groups of animals responded equally well to positively charged lysozyme. The relative electric charges of the antibodies were assessed by Sephadex A‐50 chromatography. The charge distribution was found to depend on the charge of the eliciting antigen. The positively charged antibodies of Ab9/Ab9 rabbits were not nearly as positively charged as those raised in Ab4/Ab4 rabbits.The difference in average electric charges and in the range of these charges between Ig Ab4 and Ig Ab9 explain quantitative differences in antibody formation in response to negatively charged antigens and may be a contributing factor to the ‘pecking order’, i.e. unequal phenotypic expression of ligh chain genes in Ab4/Ab9 heterozygotes.
EXPRESSION OF HLA ANTIGENS ON LEUKAEMIA CELLS
Authors: Mayer Simone , Tongio M.‐M. , Falkenrodt A. , Pfeiffer B. , Bergerat J. P. 
Published: April 2007

SUMMARYVirus infected cells can carry HLA antigens not demonstrated in uninfected cells. In a wider context, it is known that tumour cells in the mouse can exhibit H2‐like antigens of foreign haplotypes which they resemble. Our aim was to investigate, using an absorption method, the HLA antigens of ten leukaemic cells and to compare these results with those of normal lymphocytes. The following sera was used for absorption: anti‐B5 serum, anti‐B7 serum, anti‐B12 serum. These sera were absorbed on the ten leukaemic cells and fifteen healthy lymphocytes. An anti‐A1 + B8 serum and an anti‐A2 serum were absorbed on the ten leukaemic cells, but there was not enough serum to carry out the similar absorption with the healthy lymphocytes. Two points emerged from these results. (1) Leukaemia cells absorbed anti‐HLA antibodies as effectively as healthy lymphocytes when the cells carried the antigen corresponding to the serum specificity. The curves obtained with leukaemia cells were comparable to those of healthy cells. (2) When the cells were not carriers of the antigens corresponding to the antibody's specificity, leukaemia cells were also capable of absorbing antibodies, unlike healthy lymphocytes which had no or poor absorption.
EQUIVALENT DECREASE OF H‐2Kk AND H‐2Dk EXPRESSION AFTER VACCINIA VIRUS INFECTION
Authors: Lakdhar M. , Senik Anna 
Published: April 2007

SUMMARYExpression of H‐2Kk and H‐2Dk molecules was studied by indirect immunofluorescence on vaccinia virus‐infected Ls cells (derived from the L929 cell line), using D‐23b and ASA‐21 monospecific alloantisera, respectively directed against the H‐2.23 and H‐2.32 private specificities of the H‐2k haplotype. Our study demonstrates that the increase in vaccinia virus‐induced antigens on the membrane of infected cells (as a function of the multiplicity of infection) is associated with a concomitant decrease in the expression of both H‐2Kk and H‐2Dk serologically defined private specificities. Absorption experiments of D‐23b and ASA‐21 sera, using infected or uninfected Ls cells, also indicate that after virus infection, H‐2Kk and H‐2Dk private specificities are equally altered in their serological expression. We finally show that this alteration consists in a mere quantitative decrease of H‐2 molecules, since the cytolytic capacity of a rabbit anti‐H‐2 serum, specifically reacting against the constant part of the heavy chains of H‐2 molecules, was significantly more absorbed by uninfected cells than by infected cells. We conclude that no preferential decrease in the expression of H‐2 Kk and H‐2 Dk molecules is induced during cell infection by vaccinia virus.
POLYMORPHISM OF SERUM ACID PHOSPHATASE IN THE RAT
Authors: Stolc, V. , And H. W. Kunz , Gill T. J. 
Published: April 2007

SUMMARYPolymorphism of serum acid phosphatase was detected by electrofocusing in acrylamide gel. The locus has two alleles Apsa and Apsb that segregated in the backcross progeny (A x B) Fl x B in a 1:1 ratio. The phenotype APS‐A is dominant in the F1 generation. The gene for serum acid phosphatase segregated independently of the major histocompatibility complex and of the sex of the rats.
SURFACE MARKERS OF PRIMATE B AND T LYMPHOID CELL LINES IDENTIFIED BY ANTIBODIES TO HUMAN AND SIMIAN LYMPHOCYTE ANTIGENS
Authors: Neubauer R. H. , Marchalonis J. J. , Strnad B. C. , Rabin H. 
Published: April 2007

SUMMARYSimian B and T lymphoid cell lines were shown to maintain surface markers found on mature lymphocytes in vivo. The T lymphoid cell lines expressed Ia‐like antigens on their surfaces, further suggesting that they represent mature, activated T cells. These Ia antigens show a structural similarity to Ia on human cells although some diversity exists. The Ia antigen expressed on T lymphoid cell lines was shown to be very similar to those on B lymphoid cell lines. Owl monkey and marmoset T lymphoid cell lines were also shown to express a VH immunoglobulin‐related determinant, a marker which is thought to be associated with T cell antigen receptor. Owl monkey and marmoset T cell lines express a surface antigen which identifies the sheep erythrocyte receptor on human T cells and some of these lines express an antigen found on human helper T cells. It is noteworthy that substantial conservation of surface components has occurred within primate evolution such that monoclonal antibodies to human Ia, OKT‐11a and Leu 3a markers can be used to type lymphocytes of lower primates.
CLEFT PALATE SUSCEPTIBILITY MAPS IN TWO H‐2 SUBREGIONS, H‐2K TO I‐B AND G TO H‐2D
Authors: Bonner J. J. , Tyan M. L. 
Published: April 2007

SUMMARYData presented here defines the map of H‐2 associated genes which affect the glucocorticoid‐induced cleft palate frequency to the regions H‐2K to I‐B and G to H‐2D. This was done by observing in four congenic strains the frequency of cleft palate induced by 160 mg/kg of dexamethasone administered to pregnant females on day 12 of gestation. The strains used were B10.A/SgSn, C57BL/10Sn, B10.A(5R)/SgSn and B10.A(18R)/Sg. Additionally the cleft palate frequency was observed in a large number of litters from saline‐treated pregnant females. The cleft palate frequency in this control was very low and the rank of strains was the same as for the glucocorticoid‐induced cleft palate frequency. This observation suggests that the induced cleft palate frequency reflects the spontaneous occurrence of isolated cleft palate in these strains.
REGULATION OF EXPERIMENTAL AUTOIMMUNE THYROIDITIS: INFLUENCE OF NON‐H‐2 GENES
Authors: Beisel K. W. , Kong Y. M. , Babu K. S. J. , David C. S. , Rose N. R. 
Published: April 2007

SUMMARYExperiments were conducted to investigate the non‐H‐2 genetic effects on experimental autoimmune thyroiditis (EAT). Strains having C3H or BALB backgroud in general produced higher autoimmune responses to mouse thyroglobulin (MTg) than the B10 or A strains. Comparisons of C3H and B10 congenic strains carrying similar H‐2 haplotypes demonstrated that the C3H congenics had significantly higher MTg antibody titres and more severe thyroid damage, even when the strains carry the low responder H‐2 haplotypes. These observations show that non‐H‐2 gene(s) influences EAT, in addition to genes in the MHC.
BIOCHEMICAL ANALYSIS OF A PUBLIC H‐2 SPECIFICITY REVEALED BY AN ANOMALOUS REACTION OF AN ALLOANTISERUM WITH A CHEMICALLY INDUCED C57BL/10 FIBROSARCOMA
Authors: Ballinari D. , Pierotti M. A. , Grazioli Laura , Cattaneo Mariella , Rogers M. J. , Parmiani G. 
Published: April 2007

SUMMARYD‐25 is a H‐2 alloantiserum produced in (B10.D2 x C3H.NB) (H‐2d x H‐2p)F1 mice after immunization with B10.RIII(H‐2r) cells, and which is known to recognize the H‐2.25 public antigen on H‐2k haplotypes. The ‘anomalous’ reaction of D‐25 with a partially purified deoxycholate‐solubilized glycoprotein of B10‐1 (H‐2b) fibrosarcoma was studied with various biochemical techniques. The 125I‐labelled precipitates were analysed both in one‐ and two‐dimensional SDS‐PAGE and by a partial proteolysis peptide mapping (Cleveland's mapping). The results indicate first that the D‐25‐related antigen was a genuine H‐2 antigen normally associated with the B2‐microglobulin, second that this antigen was borne by the Kb but not Db gene products, and finally that D‐25 was able to precipitate the same antigen on normal H‐2b spleen cells. We conclude that this serum recognized a normal H‐2 specificity shared by H‐2b and H‐2r haplotypes and tentatively identified as the public antigen H‐2.54 which for the first time could be assigned to the K but not to the D region of the H‐2b haplotype.
MURINE IN VIVO CARRIER ‐SPECIFIC IgG MEMORY TO DNP‐FICOLL: VARIATION AMONG INBRED STRAINS
Authors: Schott C. F. , Merchant B. 
Published: April 2007

SUMMARYBALB/cAnN and C3H/HeN mice were primed and challenged with DNP‐Ficoll, a thymic independent (TI‐2) antigen. They developed significant carrier‐specific IgG memory responses: DNP‐pneumococcal polysaccharide or DNP‐hemocyanin challange did not elicit memory responses. NZB/B1NJ mice failed to develop this memory response. Genetically controlled events apparently regulate the phenotypic expression of this effect.
ORIENTATION OF LOCI IN THE MAJOR HISTOCOMPATIBILITY COMPLEX OF THE RAT AND ITS COMPARISON TO MAN AND THE MOUSE
Authors: Gill T. J. , Kunz H. W. , Schaid D. J. , VandeBerg J. L. , Stolc V. 
Published: April 2007

SUMMARYAmong 290 F2 progeny of an r10 x ACP cross were two recombinants which allowed the loci for glyoxalase‐1 and neuraminidase‐1 to be mapped relative to the RT1.A and dw‐3 loci in the major histocompatibility complex (MHC) of the rat. In 673 progeny of the same cross there was a recombinant between ft and dw‐3, and in 403 progeny of the backcross BY1 x (BY1 x BDIX)F1 there was another recombinant between ft and dw‐3. These data, combined with those from previous studies, provide the information for constructing a detailed map of the rat major histocompatibility complex: the gene order and size in the rat are very similar to those in the mouse and different from those in man and in the other species that have been studied. Comparison of the structures of the MHC in the various species leads to a hypothesis about the evolution of the MHC which involves sequential duplications of the genes coding for class I and class II loci and an inversion in the prototypic muridae which placed the class II loci between the class I loci.
MOUSE MONOCLONAL ANTI‐N: I. PRODUCTION AND SEROLOGICAL CHARACTERIZATION
Authors: Fraser R. H. , Munro A. C. , Williamson A. R. , Barrie E. K. , Hamilton E. A. , Mitchell R. 
Published: April 2007

SUMMARYTwo examples of mouse monoclonal anti‐N are described. The antibodies were derived from mice immunized with sialoglycoprotein extracts of group O MM ss erythrocyte membranes and the probable stimulus for immunization was glycophorin B associated N‐antigen. Both antibodies reacted as direct agglutinins but appeared to recognize different epitopes with one having a greater dependence on sialic acid. The antibodies could prove to be valuable alternatives to those reagents used currently for N‐blood grouping.
MOUSE MONOCLONAL ANTI‐N: II. PHYSICOCHEMICAL CHARACTERIZATION AND ASSESSMENT FOR ROUTINE BLOOD GROUPING
Authors: Fraser R. H. , Munro A. C. , Williamson A. R. , Barrie E. K. , Hamilton E. A. , Mitchell R. 
Published: April 2007

SUMMARYThe precise physicochemical conditions under which two examples of mouse monoclonal anti‐N can be used as blood grouping reagents have been defined. The antibodies were shown to belong to the IgG1 and IgG2b subclasses respectively and both reacted within discrete ranges of temperature and pH with optimal reactions occurring at 20d̀C or less and pH 8.5. Under these conditions and at concentrations of 2‐3 μg/ml, the antibodies were used, in parallel with conventional polyclonal antisera, to type a series of random blood donors. The results obtained with one of the monoclonal antibodies and the polyclonal reagents were in perfect agreement and the monoclonal antibody was judged to have considerable potential as an N‐grouping reagent.
EQUINE LEUCOCYTE ANTIGEN SYSTEM: III. NON‐MHC LINKED ALLOANTIGENIC SYSTEM IN HORSES
Authors: Lazary, S. , Gerber, H. , Weck A. L. de , Arnold P. 
Published: April 2007

SUMMARYA new, non‐MHC linked alloantigenic membrane antigen on the equine lymphocytes is described. This antigen was characterized with alloantisera in the two‐stage microcytotoxicity test and designated as ELy‐1 antigen.The frequency of ELy‐1 antigen positive animals in various populations is close to 50%. ELy‐1 shows an autosomal, dominant inheritance. Since an allelic antigen (s) could not be demonstrated in family studies, it is assumed that only two alleles ELy‐I+ and ELy‐I‐ exist. The ELy‐1 antigen in positive animals is expressed on both T and B lymphocytes but it is not present on erythrocytes and thrombocytes.The incidence of ELy‐1 antigen positive animals is significantly higher in horse groups suffering from chronic bronchitis or from laminitis, compared to clinically healthy controls.
A NEW FAST VARIANT (BF*F08) OF THE BF POLYMORPHISM
Authors: Davrinche C. , Rivat C. , Rivat‐Peran L. , Cazes M. H. , Chaventre A. 
Published: April 2007

SUMMARYA new variant in the BF polymorphism was found in one individual from an isolate (Dogons) of Mali. It might correspond to the expression of a new allele BF*F08 with haemolytic activity.