The a  ai fruit (Euterpe oleracea Martius), which is native to the Brazilian Amazon region, was shown to have high @entity6398 and @entity8981 contents. In this study, we aimed to assess the effects of a  ai consumption on plasma lipids, apolipoproteins, the transfer of lipids to HDL (which is a relevant HDL function), and some biomarkers of redox metabolism. Forty healthy volunteer @entity1 aged 24      3 years consumed 200  g of a  ai pulp/day for 4 weeks; their clinical variables and blood sample were obtained before and after this period. A  ai pulp consumption did not alter anthropometric parameters, systemic arterial pressure, @entity413 , insulin and total, LDL and HDL @entity165 , @entity1674 and @entity755 , but it did increase the concentration of @entity1675 . A  ai consumption decreased the ROS, ox-LDL and @entity1234 while increasing the activity of antioxidative @entity533 . Overall, the total antioxidant capacity (TAC) was increased. Regarding the transfer of plasma lipids to HDL, a  ai consumption increased the transfer of @entity8557 (p  =  0.0043) to HDL. Unesterified @entity165 , phospholipids and @entity412 transfers were unaffected. The increase in @entity1675 and the @entity34601 transfer to HDL after the a  ai intake period suggests that an improvement in the metabolism of this lipoprotein occurred, and it is well known that HDL is protective against @entity731 . Another important finding was the general improvement of the anti-oxidant defences elicited by a  ai consumption. Our data indicate that a  ai has favourable actions on plasma HDL metabolism and anti-oxidant defence; therefore a  ai could have a beneficial overall role against @entity731 , and it is a consistently good candidate to consider as a functional food.

A  ai (Euterpe oleracea Mart.) dietary intake affects plasma lipids, apolipoproteins, XXXX transfer to high-density lipoprotein and redox metabolism: A prospective study in @entity1 .

@entity1 :: ('9606', 'Species') :: ['women'] @entity533 :: ('5444', 'Gene') :: ['paraoxonase 1'] @entity731 :: ('MESH:D050197', 'Disease') :: ['atherosclerosis'] @entity1674 :: ('MESH:D014280', 'Chemical') :: ['triglycerides'] @entity755 :: ('338', 'Gene') :: ['apolipoprotein (apo) B'] @entity1234 :: ('MESH:D008315', 'Chemical') :: ['malondialdehyde'] @entity412 :: ('CHEBI:17855', 'Chemical') :: ['triglyceride'] @entity34601 :: ('D002788', 'Chemical') :: ['cholesteryl ester'] @entity1675 :: ('335', 'Gene') :: ['apo A-I'] @entity8981 :: ('CHEBI:25413', 'Chemical') :: ['MUFA'] @entity6398 :: ('MESH:D059808', 'Chemical') :: ['polyphenols'] @entity8557 :: ('MESH:D002788', 'Chemical') :: ['cholesteryl esters'] @entity413 :: ('MESH:D005947', 'Chemical') :: ['glucose'] @entity165 :: ('MESH:D002784', 'Chemical') :: ['cholesterol']
@entity34601 :: (D002788,Chemical) :: ['cholesteryl ester']
