AIMS: We studied the relationship of @entity1386 ( @entity1386 ) measured at 15 specified body sites with @entity1243 before and after a weight loss program for three weeks. SUBJECTS AND METHODS: In 70 @entity28 @entity1 , @entity1386 were measured by means of the optical device, lipometer. Fat mass (FM) was estimated by means of bioelectrical impedance. RESULTS: At the beginning of the study, all estimates of @entity1386 , insulin, and @entity1386 from the upper body (from 1-neck to 6-lateral chest) were correlated to @entity1243 at a P-value of<0.0001. Percentage FM together with @entity615 and insulin explained 75% of the variation in @entity1243 (P<0.0001). After three weeks, estimates of @entity1386 and @entity1243 were reduced (all P<0.0001). Most @entity1386 were reduced, but @entity1386 8-lower abdomen and 9-lower back were significantly increased. Changes in @entity1243 were best explained by initial @entity1243 , but percentage change (Delta) in insulin, Delta SAT-layer 1-neck, and Delta SAT-layer 3-biceps contributed to the Delta @entity1243 (adj. r(2)=0.47, P<0.0001). In the weight-reduced state, circulating @entity1243 was best explained by three @entity1386 and insulin (adj. r(2)=0.67, P<0.0001). DISCUSSION: The results suggest that Delta changes in @entity1243 are attributable to changes in the endocrine state and subcutaneous fat, and @entity1386 may serve as a stable correlate of @entity1243 in the weight-reduced state.

Subcutaneous adipose tissue layers as a stable correlate of @entity1243 in response to short term energy restriction in XXXX @entity1 .

@entity1 :: ('9606', 'Species') :: ['girls'] @entity28 :: ('MESH:D009765', 'Disease') :: ['obese'] @entity1243 :: ('3952', 'Gene') :: ['leptin'] @entity615 :: ('MESH:D001416', 'Disease') :: ['SAT-layer 4-upper back'] @entity1386 :: ('MESH:D018205', 'Disease') :: ['SAT-layers', 'subcutaneous adipose tissue layers', 'adiposity']
@entity28 :: (MESH:D009765,Disease) :: ['obese']
