We examined the intrinsic and reflex contributions to ankle @entity2693 in @entity1 with @entity64 and healthy subjects using the parallel system identification technique. Modulation of intrinsic and reflex @entity2693 was characterized by applying pseudorandom binary sequence (PRBS) perturbations to the ankle at different initial ankle joint over the entire range of motion (ROM). The experiments were performed for both paretic ( @entity64 ) and contralateral (control) side. Healthy (normal) subjects were used a secondary control. Reflex @entity2693 gain significantly increased in @entity64 than in control side at most positions. @entity1017 also increased significantly at dorsiflexing positions. These changes were position dependent. Thus, the @entity1017 increased continuously from middle plantarflexion to full dorsiflexion while the major increase in reflex @entity2693 happened at the middle ROM. No significant changes were found in other @entity4329 . As compared to the normal ankle, the reflex @entity2693 gain of the control side was significantly larger, indicating that the control side is not normal. These findings demonstrate that both intrinsic and reflex @entity2693 contribute significantly to the @entity854 associated with @entity508 ankle in @entity64 subjects. The results also suggest that the contralateral limb may not be used as a suitable control.

Mechanical abnormalities of the XXXX ankle in @entity64 subjects.

@entity1 :: ('9606', 'Species') :: ['people'] @entity64 :: ('MESH:D020521', 'Disease') :: ['stroke', 'chronic stroke', 'hemiplegic stroke'] @entity508 :: ('MESH:D009128', 'Disease') :: ['spastic'] @entity854 :: ('MESH:D018376', 'Disease') :: ['mechanical abnormality'] @entity1017 :: ('MESH:D015430', 'Disease') :: ['abnormalities in intrinsic stiffness gain', 'Intrinsic stiffness gain'] @entity4329 :: ('MESH:C563242', 'Disease') :: ['intrinsic and reflex stiffness parameters'] @entity2693 :: ('MESH:C566112', 'Disease') :: ['stiffness']
@entity508 :: (MESH:D009128,Disease) :: ['spastic']
