UNASSIGNED: We herein investigated the effects of passive heat stress on @entity1 somatosensory processing recorded by somatosensory-evoked potentials (SEPs). Fifteen healthy subjects received a median nerve stimulation at the left wrist under two thermal conditions: Heat stress and normothermic Time Control. The latencies and amplitudes of @entity7817 , N20, @entity4667 , N35, @entity19360 , and N60 at C4' and @entity7817 , N18, @entity9822 , and N30 at Fz were evaluated. Under the @entity8291 , SEPs were recorded at normothermic baseline (1st), early in heat stress (2nd), when esophageal temperature had increased by ~1.0   C (3rd) and ~2.0   C (4th), and after heat stress (5th). In the Time control condition, SEPs were measured at the same time intervals as those in the @entity8291 . The peak latencies and amplitudes of SEPs did not change early in heat stress. However, the latencies of @entity7817 , N20, and N60 at C4' and @entity7817 , N18, and @entity9822 at Fz were significantly shorter in the 4th session than in the 1st session. Furthermore, the peak amplitudes of @entity4667 and N60 at C4', and @entity9822 and N30 at Fz decreased with increases in body temperature. On the other hand, under the Time control condition, no significant differences were observed in the amplitudes or latencies of any component of SEPs. These results suggested that the conduction velocity of the ascending somatosensory input was accelerated by increases in body temperature, and @entity221 impaired the neural activity of cortical somatosensory processing.

Effects of passive heat stress on XXXX somatosensory processing.

@entity1 :: ('9606', 'Species') :: ['human'] @entity19360 :: ('4778', 'Gene') :: ['P45'] @entity7817 :: ('11102', 'Gene') :: ['P14'] @entity9822 :: ('11261', 'Gene') :: ['P22'] @entity8291 :: ('MESH:D018882', 'Disease') :: ['Heat stress condition'] @entity221 :: ('MESH:D005334', 'Disease') :: ['hyperthermia'] @entity4667 :: ('11076', 'Gene') :: ['P25']
@entity1 :: (9606,Species) :: ['human']
