OBJECTIVES: Our study was designed to clarify the role of the thalamus in the generation of the electrically elicited long-latency reflexes (LLR) in voluntarily activated hand muscles. MATERIALS AND METHODS: EMG responses of the thenar muscles were evoked by electrical stimulation of the median nerve at the wrist at motor threshold intensity in 10 @entity1 with acute pure @entity64 due to @entity1188 . Concomitant recording of somatosensory evoked potentials (SEPs) was performed. The subjects were asked to steadily abduct the thumb at 20-30% of maximal force against a force transducer. Rectified and averaged EMG activities were recorded. RESULTS: The LLR II was missing completely or significantly attenuated in the majority of the @entity1 (9 of 10), of whom 3 also had delayed latency. Abnormal SEPs were documented in 7 @entity1 (7 of 10). In the follow-up, 5 @entity1 had partial reversal of LLR II. LLR II was still pathological in 1 fully recovered @entity1 . CONCLUSION: Our results further confirm the transcortical generation of LLR II and imply that a thalamic relay is present in the afferent limb of the LLR.

Long latency responses in pure @entity64 due to XXXX .

@entity1 :: ('9606', 'Species') :: ['patients', 'patient'] @entity64 :: ('MESH:D020521', 'Disease') :: ['sensory stroke'] @entity1188 :: ('MESH:D007238', 'Disease') :: ['thalamic infarction']
@entity1188 :: (MESH:D007238,Disease) :: ['thalamic infarction']
