There are two forms of @entity6 , @entity1723 ( @entity1723 ) and @entity2175 ( @entity2175 ). There is evidence to suggest that reactive @entity26 is involved in the pathogenicity and complications arising from @entity1723 , but there is little to suggest a role of oxidative stress in the pathogenesis of @entity2175 . In order to investigate this hypothesis further, peripheral blood samples were taken from control individuals and @entity1723 and @entity2175 @entity1 and examined for antioxidant capacity and in the Comet assay for DNA strand breakage. The individuals answered a questionnaire to provide information relating to lifestyle factors in case such factors might have a confounding effect. There were 20 controls, 22 @entity1723 @entity1 and 23 @entity2175 @entity1 . No differences could be detected in control and @entity6 @entity1 groups in terms of @entity1011 levels and antioxidant capacity. DNA damage in the Comet assay was at a lower level than in the control in the @entity1723 @entity1 and a slightly lower level in the @entity2175 @entity1 , which might indicate that these cells are handling more oxidative damage on a regular basis. As expected, there were differences in glycosylated haemoglobin (HbA(1C)) levels. The confounding factors (smoking, drinking and vitamin intakes, etc.) appeared to have no effect.

An examination of DNA strand breakage in the comet assay and antioxidant capacity in XXXX @entity1 .

@entity2175 :: ('MESH:D003924', 'Disease') :: ['NIDDM', 'non-insulin dependent Diabetes mellitus'] @entity1 :: ('9606', 'Species') :: ['patients', 'patient'] @entity6 :: ('MESH:D003920', 'Disease') :: ['diabetic', 'diabetes'] @entity26 :: ('MESH:D010100', 'Chemical') :: ['oxygen'] @entity1723 :: ('MESH:D003922', 'Disease') :: ['IDDM', 'insulin-dependent Diabetes mellitus'] @entity1011 :: ('MESH:D003404', 'Chemical') :: ['creatinine']
@entity6 :: (MESH:D003920,Disease) :: ['diabetic']
