Following an @entity583 ( @entity583 ) there is immediate deterioration of contractility in the infarcted left ventricular (LV) wall. This can be followed by regional dilation (expansion) as well as global remodeling. We examined 35 consecutive @entity1 --with no history of @entity741 --who were admitted to hospital within 3 hours after initial symptoms and with ST-segment changes on an electrocardiogram consistent with transmural @entity63 . Echocardiography was performed at admission, and at 6 hours, 12 hours, 24 hours, 3 days, and 6 days after onset of the @entity583 . Within 3 hours after onset of symptoms an increase in both end-diastolic volume index (EDVI) and end-systolic volume index (ESVI) was found in both anterior and inferior @entity1188 when compared with healthy controls (mean +/- SD EDVI: 99 +/- 13 ml/m2 [anterior], 69 +/- 17 ml/m2 [inferior], 51 +/- 15 ml/m2 [controls], p < or = 0.00001; ESVI: 62 +/- 12 ml/m2 [anterior], 38 +/- 11 ml/m2 [inferior], 17 +/- 6 ml/m2 [controls], p < or = 0.00001). At all points in time, volumes were larger in anterior @entity1188 than in inferior @entity1188 (p < 0.05). The volumes did not change during the 6 days (p > 0.1). Thus, major @entity5988 is present within 3 hours after onset of symptoms of first @entity583 . The dilation is more pronounced in anterior versus inferior @entity1188 . From 3 hours until day 6 no further changes in LV volumes occurred.

Comparison of degrees of left ventricular dilation within three hours and up to six days after onset of first XXXX .

@entity1 :: ('9606', 'Species') :: ['patients'] @entity5988 :: ('MESH:C565277', 'Disease') :: ['LV dilation'] @entity63 :: ('MESH:D007511', 'Disease') :: ['ischemia'] @entity1188 :: ('MESH:D007238', 'Disease') :: ['infarcts'] @entity741 :: ('MESH:D017202', 'Disease') :: ['myocardial ischemia'] @entity583 :: ('MESH:D009203', 'Disease') :: ['AMI', 'acute myocardial infarction']
@entity583 :: (MESH:D009203,Disease) :: ['acute myocardial infarction']
