A clinicopathological study of @entity191 witch occurred after @entity64 , is done by the authors. That is a retrospective study concerning 2 anatomic cases from 21 vascular @entity304 . All the @entity1 were admitted in the clinique neurologique de Fann from 1970 to 1990. They were 15 @entity1 and 6 @entity1 . Their age ranged between 45 to 75 years (mean 59.66). The 2 autopsied cases had lesions located in the thalamus as the superior part and a discreet @entity194 in the one case, and in the other case, we can see similar lesions in the right external capsule and in the inferior part of the putamen. Another lesion of haemorrhage @entity1188 was located in the left temporal lobe. The locus @entity3456 was macroscopically normal in the 2 cases. Anatomical lesions observed have been localized in the putamen and in the external capsule in the associatical cerebral cortex in the second case and in the first one, lesions are located in the thalamus and in the cortex. These lesions were not well correlated with neurological signs. The different clinical aspects are discussed. From this study, it results that arterial damages associated with @entity101 and atheromatosis might touch dopaminergic structures responsible of @entity304 with sometime good sensitivity to levodopa.

[Vascular XXXX in 21 case studies, two of which were anatomic].

@entity1 :: ('9606', 'Species') :: ['women', 'patients', 'men'] @entity64 :: ('MESH:D020521', 'Disease') :: ['stroke'] @entity101 :: ('MESH:D006973', 'Disease') :: ['hypertension'] @entity304 :: ('MESH:D010302', 'Disease') :: ['parkinsonism'] @entity191 :: ('MESH:D010300', 'Disease') :: ['parkinsonian syndrome'] @entity1188 :: ('MESH:D007238', 'Disease') :: ['infarct'] @entity194 :: ('MESH:D001284', 'Disease') :: ['cortical atrophy'] @entity3456 :: ('4230', 'Species') :: ['niger']
@entity191 :: (MESH:D010300,Disease) :: ['parkinsonian syndrome']
