The pathogenesis of small intestinal @entity16 in @entity326 was explored in experiments on 15 @entity468 given live @entity526 i.v. Villous (absorptive site) blood flow was studied by the @entity1223 uptake technique using isolated small intestinal segments. In eight of the @entity468 , segments were perfused intraluminally with oxygenated or nitrogenated saline. The main part of the small intestine was unperfused and served as control. Nine @entity468 (60%) developed @entity16 . They had significantly lower arterial blood pressure at the end of @entity295 (56 +/- 6) than @entity468 without @entity16 (76 +/- 3 mmHg). Total intestinal blood flow was similar before or during @entity295 . Villous blood flow before @entity295 was 3.7 +/- 0.5 ml/min X 100 g intestine and 5.1 +/- 1.0 (n.s.) in the two groups, respectively, and remained unchanged. Intraluminal perfusion with oxygenated but not with nitrogenated saline prevented the development of @entity16 . It was concluded that the small intestinal @entity16 is due to @entity519 in spite of unchanged villous blood supply.

Small intestinal mucosal lesions in feline XXXX : a study on the pathogenesis.

@entity295 :: ('MESH:D018805', 'Disease') :: ['septicemia'] @entity1223 :: ('MESH:D002248', 'Chemical') :: ['carbon monoxide'] @entity326 :: ('MESH:D012772', 'Disease') :: ['septic shock'] @entity16 :: ('MESH:D009422', 'Disease') :: ['mucosal damage'] @entity519 :: ('MESH:D000860', 'Disease') :: ['hypoxia'] @entity468 :: ('9685', 'Species') :: ['cats'] @entity526 :: ('562', 'Species') :: ['E coli']
@entity326 :: (MESH:D012772,Disease) :: ['septic shock']
