BACKGROUND: Reflection pressure may influence the clinical course of @entity740 ( @entity740 ). The relationship between the augmentation index (AI) and progression of @entity2316 was characterized. METHODS: Ninety-nine @entity1 were enrolled into the study. Pulse wave form analysis was performed to determine AI that assesses arterial stiffness. RESULTS: In a cross-sectional study, a multiple regression analysis found that AI correlated positively to age and weight, and negatively to height and heart rate (R(2) = 0.50). Furthermore, echocardiography was performed in 51 @entity1 who gave their consent. In male @entity1 under angiotensin inhibition, left ventricular mass index increased as AI was elevated (r = 0.33, slope = 0.85 +/- 0.30 g/m(2)/%, p < 0.05, n = 23). A prospective study was performed in 41 @entity1 who consented to having their @entity1011 clearance measured repeatedly. In the @entity1 with angiotensin inhibition a higher basal AI resulted in a greater annual decrease in @entity1011 clearance (r = -0.52, slope = -0.43 +/- 0.14 ml/min/year/%, p < 0.01, n = 27). CONCLUSION: The present data indicate that AI as well as angiotensin contribute to the development of @entity4346 . Furthermore, our results suggest that in addition to angiotensin, AI is a risk factor of progression of @entity2316 .

Qualification of arterial stiffness as a risk factor to the progression of XXXX .

@entity1 :: ('9606', 'Species') :: ['patients'] @entity2316 :: ('MESH:D003928', 'Disease') :: ['non-diabetic CKDs'] @entity740 :: ('MESH:D051436', 'Disease') :: ['CKDs', 'chronic kidney diseases'] @entity4346 :: ('MESH:D017379', 'Disease') :: ['left ventricular hypertrophy'] @entity1011 :: ('MESH:D003404', 'Chemical') :: ['creatinine']
@entity740 :: (MESH:D051436,Disease) :: ['chronic kidney diseases']
