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passages.text: CTD binding and allosteric activation of the mammalian guanylyltransferase Mce1 was studied by introducing alanine substitutions for Tyr1, Pro3, Thr4, or Pro6 of each heptad of the 28-mer synthetic CTD Ser5-PO4 peptide, or by eliminating each of the phosphates at Ser5 (Pei et al., 2001a). 
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