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passages.text: On binding of LPS to TLRs, specifically TLR2 and TLR4, via CD14, a number of molecules are recruited to the receptor to mediate NF-κB activation, which include the adaptor molecule MyD88, the protein kinase IRAK, and the adaptor molecule TRAF-6, thus forming the receptor complex [29], resulting in interaction of downstream molecules and the subsequent activation of NF-κB.
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