Urine calcium excretion rates were consistent with osteoporosis development ; however calcium balance studies did not demonstrate loss of whole body calcium with higher net acid excretion .
No study revealed a biologic mechanism functioning at physiological pH. Finally , r and omized studies did not provide evidence for an adverse role of phosphate , milk , and grain foods in osteoporosis .
Conclusions A causal association between dietary acid load and osteoporotic bone disease is not supported by evidence and there is no evidence that an alkaline diet is protective of bone health
Background Modern diets have been suggested to increase systemic acid load and net acid excretion .
In response , alkaline diets and products are marketed to avoid or counteract this acid , help the body regulate its pH to prevent and cure disease .
The objective of this systematic review was to evaluate causal relationships between dietary acid load and osteoporosis using Hill 's criteria .
Consumption of a high calcium mineral water lowers biochemical indices of bone remodeling in postmenopausal women with low calcium intake
Many postmenopausal women have a calcium intake far below the recommended amount and , in addition to attempting to improve their diet , need a calcium supplement . The aim of the study was to assess the effects of the consumption of a high calcium mineral water ( HCaMW ) on biochemical indices of bone remodeling in postmenopausal women with low Ca intake . A 6-month r and omized double-blind placebo-controlled trial was design ed to assess the effects of a daily consumption of 1 liter of a HCaMW ( 596 mg Ca/l ) on serum parathyroid hormone ( PTH ) and biochemical markers of bone remodeling in postmenopausal women with a dietary Ca intake lower than 700 mg/day . The placebo group drank 1 liter of a mineral water with a low calcium content ( 10 mg/l ) . One hundred eighty healthy women were recruited ( mean age : 70.1±4.0 years ) ; 152 completed the 6-month trial . The changes from baseline of biochemical indices after 6 months consisted of a significant 14.1 % decrease of serum PTH , osteocalcin ( –8.6 % ) , bone alkaline phosphatase ( –11.5 % ) , serum ( –16.3 % ) and urine ( –13.0 % ) type-1 collagen C-telopeptide in the HCaMW group compared to the placebo group , where all biochemical indices increased after 6 months . The additive effect of a small vitamin D supplement ( 400 iu/day ) was also evaluated . In women receiving vitamin D in addition to HCaMW , the decrease in bone indices was not found to be greater than in women drinking only the HCaMW . A daily supplement of 596 mg of Ca through the consumption of 1 l of HCaMW was able to lower serum PTH and the indices of bone turnover in postmenopausal women with a low Ca intake . This could contribute to the repair of calcium deficiency and to the reduction of age-related bone loss in this population
Urine calcium excretion rates were consistent with osteoporosis development ; however calcium balance studies did not demonstrate loss of whole body calcium with higher net acid excretion .
No study revealed a biologic mechanism functioning at physiological pH. Finally , r and omized studies did not provide evidence for an adverse role of phosphate , milk , and grain foods in osteoporosis .
Conclusions A causal association between dietary acid load and osteoporotic bone disease is not supported by evidence and there is no evidence that an alkaline diet is protective of bone health
Background Modern diets have been suggested to increase systemic acid load and net acid excretion .
In response , alkaline diets and products are marketed to avoid or counteract this acid , help the body regulate its pH to prevent and cure disease .
The objective of this systematic review was to evaluate causal relationships between dietary acid load and osteoporosis using Hill 's criteria .