Urine calcium excretion rates were consistent with osteoporosis development ; however calcium balance studies did not demonstrate loss of whole body calcium with higher net acid excretion .
No study revealed a biologic mechanism functioning at physiological pH. Finally , r and omized studies did not provide evidence for an adverse role of phosphate , milk , and grain foods in osteoporosis .
Conclusions A causal association between dietary acid load and osteoporotic bone disease is not supported by evidence and there is no evidence that an alkaline diet is protective of bone health
Background Modern diets have been suggested to increase systemic acid load and net acid excretion .
In response , alkaline diets and products are marketed to avoid or counteract this acid , help the body regulate its pH to prevent and cure disease .
The objective of this systematic review was to evaluate causal relationships between dietary acid load and osteoporosis using Hill 's criteria .
Elevated secretion and action of serum parathyroid hormone in young adults consuming high phosphorus, low calcium diets assembled from common foods.
We sought to determine if the high phosphorus , moderately low calcium intake typical of U.S. teenagers and young adults alters parathyroid function as it does in experimental animals . Because those animals ultimately developed osteopenia , it has been suggested that low dietary calcium to phosphorus ratios may reduce peak bone mass and increase susceptibility to osteoporotic fracture later in life . However , it is not known whether PTH secretion or action increases in response to commonly consumed phosphorus-rich , calcium-poor foods . We studied the 24-h mineral and hormonal responses of eight men and eight women , aged 18 - 25 yr , after 8 days of ingesting a control diet that had calcium ( 820 mg ) and phosphorus ( 930 mg ) contents near the recommended daily intakes , and a test diet with calcium and phosphorus contents ( 1660 mg phosphorus , 420 mg calcium ) typical of current intakes . Both diets were made from common grocery store foods . The 24-h mean serum immunoreactive PTH levels increased in men ( 11 % ; P less than 0.006 ) and women ( 22 % ; P less than 0.003 ) during the test diet . In both sexes , the test diet significantly increased serum phosphorus , plasma 1,25-dihydroxyvitamin D , and urinary hydroxyproline and cAMP excretion ; in women only it decreased serum ionized and total calcium levels . Thus , short term ingestion of a diet typifying current levels of calcium and phosphorus intake result ed in elevated serum iPTH levels and indexes of PTH action in young adults
Urine calcium excretion rates were consistent with osteoporosis development ; however calcium balance studies did not demonstrate loss of whole body calcium with higher net acid excretion .
No study revealed a biologic mechanism functioning at physiological pH. Finally , r and omized studies did not provide evidence for an adverse role of phosphate , milk , and grain foods in osteoporosis .
Conclusions A causal association between dietary acid load and osteoporotic bone disease is not supported by evidence and there is no evidence that an alkaline diet is protective of bone health
Background Modern diets have been suggested to increase systemic acid load and net acid excretion .
In response , alkaline diets and products are marketed to avoid or counteract this acid , help the body regulate its pH to prevent and cure disease .
The objective of this systematic review was to evaluate causal relationships between dietary acid load and osteoporosis using Hill 's criteria .