Changes in FOX did not reach the level of statistical significance .
Finding showed that EGCG intake moderately accelerates EE and reduces RQ .
The analyses revealed that the EGCG result ed in difference in RQ and EE but the effect on the other measures of energy metabolism was relatively mild .
Possibly , EGCG alone has the potential to increase metabolic rate at 300 mg dose .
Collectively , the outcome supports the findings that EGCG has an effect on metabolic parameters .
Green tea catechins ( GTCs ) are known to improve fat oxidation ( FOX ) during fasted , rested and exercise conditions wherein epigallocatechin-3-gallate ( EGCG ) is thought to be the most pharmacologically active and has been studied extensively .
From the available data of r and omized controlled trials ( RCTs ) on EGCG , we carried out a systematic review and meta- analysis to eluci date whether EGCG consumption indeed increase energy expenditure ( EE ) and promote FOX .
Efficacy of a green tea extract rich in catechin polyphenols and caffeine in increasing 24-h energy expenditure and fat oxidation in humans.
BACKGROUND Current interest in the role of functional foods in weight control has focused on plant ingredients capable of interfering with the sympathoadrenal system . OBJECTIVE We investigated whether a green tea extract , by virtue of its high content of caffeine and catechin polyphenols , could increase 24-h energy expenditure ( EE ) and fat oxidation in humans . DESIGN Twenty-four-hour EE , the respiratory quotient ( RQ ) , and the urinary excretion of nitrogen and catecholamines were measured in a respiratory chamber in 10 healthy men . On 3 separate occasions , subjects were r and omly assigned among 3 treatments : green tea extract ( 50 mg caffeine and 90 mg epigallocatechin gallate ) , caffeine ( 50 mg ) , and placebo , which they ingested at breakfast , lunch , and dinner . RESULTS Relative to placebo , treatment with the green tea extract result ed in a significant increase in 24-h EE ( 4 % ; P < 0.01 ) and a significant decrease in 24-h RQ ( from 0.88 to 0.85 ; P < 0.001 ) without any change in urinary nitrogen . Twenty-four-hour urinary norepinephrine excretion was higher during treatment with the green tea extract than with the placebo ( 40 % , P < 0.05 ) . Treatment with caffeine in amounts equivalent to those found in the green tea extract had no effect on EE and RQ nor on urinary nitrogen or catecholamines . CONCLUSIONS Green tea has thermogenic properties and promotes fat oxidation beyond that explained by its caffeine content per se . The green tea extract may play a role in the control of body composition via sympathetic activation of thermogenesis , fat oxidation , or both
Changes in FOX did not reach the level of statistical significance .
Finding showed that EGCG intake moderately accelerates EE and reduces RQ .
The analyses revealed that the EGCG result ed in difference in RQ and EE but the effect on the other measures of energy metabolism was relatively mild .
Possibly , EGCG alone has the potential to increase metabolic rate at 300 mg dose .
Collectively , the outcome supports the findings that EGCG has an effect on metabolic parameters .
Green tea catechins ( GTCs ) are known to improve fat oxidation ( FOX ) during fasted , rested and exercise conditions wherein epigallocatechin-3-gallate ( EGCG ) is thought to be the most pharmacologically active and has been studied extensively .
From the available data of r and omized controlled trials ( RCTs ) on EGCG , we carried out a systematic review and meta- analysis to eluci date whether EGCG consumption indeed increase energy expenditure ( EE ) and promote FOX .