Urine calcium excretion rates were consistent with osteoporosis development ; however calcium balance studies did not demonstrate loss of whole body calcium with higher net acid excretion .
No study revealed a biologic mechanism functioning at physiological pH. Finally , r and omized studies did not provide evidence for an adverse role of phosphate , milk , and grain foods in osteoporosis .
Conclusions A causal association between dietary acid load and osteoporotic bone disease is not supported by evidence and there is no evidence that an alkaline diet is protective of bone health
Background Modern diets have been suggested to increase systemic acid load and net acid excretion .
In response , alkaline diets and products are marketed to avoid or counteract this acid , help the body regulate its pH to prevent and cure disease .
The objective of this systematic review was to evaluate causal relationships between dietary acid load and osteoporosis using Hill 's criteria .
Higher calcium urinary loss induced by a calcium sulphate-rich mineral water intake than by milk in young women.
It is well known that the intestinal availability of Ca from Ca-rich mineral waters is equivalent to that of milk Ca . However , the effect of associated anions on Ca urinary loss needs to be addressed . The aim of the current study was to compare , under ordinary conditions of consumption , milk and a SO4-rich mineral water as the Ca provider in a large number of subjects consuming the same quantity of Ca from the two sources in a crossover study lasting for an extended period . Thirty-seven healthy women completed a 12-week protocol , divided into four periods of 3 weeks ( W ) . In the first ( W1 - 3 ) and third ( W6 - 9 ) periods , dietary Ca intake was restricted to 600 mg/d . In the second ( W4 - 6 ) and final ( W10 - 12 ) periods , either 400 ml/d medium-fat milk or 1 litre of a Ca- and SO4-rich mineral water , each providing about 480 mg Ca/d , was added to the diet in a r and om manner . Dietary evaluation , blood and urinary measures were performed during the last week ( W6 and W12 ) of each Ca supplementation period . The urinary excretion of Ca was higher ( 0.5 mmol/d more ) with water than with milk ( P<0.001 ) . An examination of all the dietary factors known to influence calciuria suggested that the acidogenic action of SO4 was responsible for this increased calciuria . Thus , despite an equal Ca intake and assuming an unchanged intestinal absorption , these results suggest that Ca balance is better with milk consumption than with CaSO4-rich water
Urine calcium excretion rates were consistent with osteoporosis development ; however calcium balance studies did not demonstrate loss of whole body calcium with higher net acid excretion .
No study revealed a biologic mechanism functioning at physiological pH. Finally , r and omized studies did not provide evidence for an adverse role of phosphate , milk , and grain foods in osteoporosis .
Conclusions A causal association between dietary acid load and osteoporotic bone disease is not supported by evidence and there is no evidence that an alkaline diet is protective of bone health
Background Modern diets have been suggested to increase systemic acid load and net acid excretion .
In response , alkaline diets and products are marketed to avoid or counteract this acid , help the body regulate its pH to prevent and cure disease .
The objective of this systematic review was to evaluate causal relationships between dietary acid load and osteoporosis using Hill 's criteria .