atepeq/tico19 huggingface dataset:
Other human coronaviruses have since been identified, including SARS-CoV in 2003, HCoV NL63 in 2004, HKU1 in 2005, MERS-CoV in 2012, and SARS-CoV-2 in 2019.
Koronaviriis neɗɗo feere mandotto diga nden, bana SARS-CoV ha 2003, HCoV NL63 ha 2004, HKU1 ha 2005, MERS-CoV ha 2012, be SARS-CoV-2 ha 2019.
Most of these have involved serious respiratory tract infections.
Ɓurna fu ɗon memma nyauji lawol pofɗe.
The name was first used in 1968 by an informal group of virologists in the journal Nature to designate the new family of viruses.
Inde man wurti aranool man ha 1968 gal geeɓe jaanginooɓe viriisji je aandakke ha nder batakkewol Nature ngam ewnugo caalaje viriisji keese.
The name refers to the characteristic appearance of virions (the infective form of the virus) by electron microscopy, which have a fringe of large, bulbous surface projections creating an image reminiscent of a crown or of a solar corona.
Inde ndu hoccitti do wondugo ɓiɓe viriisji ndu (viriis man je raɓɓatta) gal electron microskopy, je ɗon mari bana gi'e je fuɗɗi do ɓaandu maɗa je ɓiɓe lekki, je wangi bana naange je ɗon jalɓitta.
This morphology is created by the viral spike peplomers, which are proteins on the surface of the virus.
Ndu woni bana ni ngam gi'e peplomerji viriis, je lati proteinji do ɓaandu viriis ndu.
Coronaviruses are large pleomorphic spherical particles with bulbous surface projections.
Koronaviriisji woni bana ɓiɓe lekki bana gi'e ɗon fuɗɗi do ɓaandu maɓɓe je wurti gal laaral ɓaandu.
The envelope of the virus in electron micrographs appears as a distinct pair of electron dense shells.The viral envelope consists of a lipid bilayer where the membrane (M), envelope (E) and spike (S) structural proteins are anchored.
Laaral viriis ha electron micrographji ɗon lara bana bawo electronji je woni ɗiɗi. Laaral viriis ɗon ɗiɗi e tardi ba ɓeelere je ɗon sigga proteinji laaral (M), Laaral (E) be hoore (S).
The lipid bilayer envelope, membrane proteins, and nucleocapsid protect the virus when it is outside the host cell.
Laaral ɓaandu je ɗon ɗiɗi bana ɓeelere, proteinji laaral, be nucleocapsid ɗon fadda viriis to o wurti ɓaandu suudu siggorde maɗa..
Coronaviruses contain a positive-sense, single-stranded RNA genome.
Koronaviriisji ɗon mari genome je positive, be kosngal RNA gooto.
The genome size for coronaviruses ranges from 26.4 to 31.7 kilobases.
Maunugo genome koronaviriis ɗon fuɗɗa diga kilobaseji 26.4 ha 31.7.
The genome size is one of the largest among RNA viruses.
Maunugo genome woni bana je ɓuri maunugo ha kunde viriisji RNA.
The open reading frames 1a and 1b, which occupy the first two-thirds of the genome, encode the replicase/transcriptase polyprotein.
Jaangugo frameji 1a be 1b je ɗon maɓɓitti, je ɗon hoosa genomeji ɗiɗi do tati, lamba polyprotein replicase/transcriptase.
The replicase/transcriptase polyprotein self cleaves to form nonstructural proteins.The later reading frames encode the four major structural proteins: spike, envelope, membrane, and nucleocapsid.
Polyprotein replicase/transcriptase senda ɓaandu maɓɓe ngam lata proteinji je wala tagle. Kuje jaangirde je warango lamban ɓurna proteinji tagle ngel: hoore, laaral, laaral, be nucleocappsid.
Interspersed between these reading frames are the reading frames for the accessory proteins.
Ta'irde ha kunde jaangirde ndu lati jaangirdeji proteinji feere.
The number of accessory proteins and their function is unique depending on the specific coronavirus.
Limtol proteinji feere be kugal maɓɓe lati feerejo be irin koronaviriis.
Infection begins when the viral spike (S) glycoprotein attaches to its complementary host cell receptor.
Raɓɓende ɗo fuɗɗa to glycoprotein hooer (S) hawti be jaɓɓowo cell maɗa je feere ndu.
After attachment, a protease of the host cell cleaves and activates the receptor-attached spike protein.
Ha ɓawo hawtirol, protease cell jaɓɓowo ɗon ta'a e ummina protein hoore je ɗon woni.
Depending on the host cell protease available, cleavage and activation allows the virus to enter the host cell by endocytosis or direct fusion of the viral envelop with the host membrane.On entry into the host cell, the virus particle is uncoated, and its genome enters the cell cytoplasm.
Do protease cell jaɓɓowo je ɗon woni, ta'irde be umminki ɗo waɗa viriis nasta cell jaɓɓowo gal endocytosis mala hawtirde laaral viriis be kuusel ɓaandu jaɓɓowo. Ha wakati nastirde ha nder cell jaɓɓowo, viriis ɓooran, genome maɗa ɗon nasta ha cytoplasm cell.
The host ribosome translates the initial overlapping open reading frame of the virus genome and forms a long polyprotein.
Ko mbiyotto host ribosome ɗon lornita arandere ko goɗɗo footi jaanga ha do gilɗi ndu e ɗon latina ɗum bana poliprotein je juttungel.
The polyprotein has its own proteases which cleave the polyprotein into multiple nonstructural proteins.
Polyprotein ɗon mari proteasji maɗa je ɗon ta'a polyprotein do proteinji je maray ɓaandu.
A number of the nonstructural proteins coalesce to form a multi-protein replicase-transcriptase complex (RTC).
Limgal nonstructural proteinji ɗon hawta ngam wurtinugo multi-protein replicase-transcriptase complex (RTC).
The main replicase-transcriptase protein is the RNA-dependent RNA polymerase (RdRp).
Aranool protein replicase-transcriptase lati RNA-dependent RNA polymerase (RdRp).
It is directly involved in the replication and transcription of RNA from an RNA strand.
Larni ha nder nuftol e jaangirde RNA ha kosɗe RNA.
The other nonstructural proteins in the complex assist in the replication and transcription process.
Proteinji feere je maray ɓaandu ha nder kuje wallan nder nuftol e laahal jaangirde.
The exoribonuclease nonstructural protein, for instance, provides extra fidelity to replication by providing a proofreading function which the RNA-dependent RNA polymerase lacks.One of the main functions of the complex is to replicate the viral genome.
Protein exoribonuclease nonstructural, bana sappinol, ɗon hokka amana ha nuftol gal hokkugo dabare jaangirde je RNA polymerase RNA woda. Gooto ha cakka kuɗe maɓɓe arandere woni nuftugo genome viriis.
RdRp directly mediates the synthesis of negative-sense genomic RNA from the positive-sense genomic RNA.
RdRp sendan lartol negative-sense genomic RNA diga positive-sense genomic RNA.
This is followed by the replication of positive-sense genomic RNA from the negative-sense genomic RNA.
Ndu ɗon tokkitiri be nuftol positive-sense genomic RNA ha negative-sense genomic RNA.
The other important function of the complex is to transcribe the viral genome.
Kugal kanduɗum kuje ndu woni jaangugo genome viriis.
RdRp directly mediates the synthesis of negative-sense subgenomic RNA molecules from the positive-sense genomic RNA.
RdRp ɗon hawta lartol negative-sense subgenomic RNA moleculeji ha positive-sense genomic RNA.
This is followed by the transcription of these negative-sense subgenomic RNA molecules to their corresponding positive-sense mRNAs.
Ndu ɗon tokkiti be irin negative-sense subgenomic RNA moleculeji ha positive-sense mRNAs je ɗon naandi.
The replicated positive-sense genomic RNA becomes the genome of the progeny viruses.
Positive-sense genomic RNA je nuftama latan genome viriis je waratta.
The mRNAs are gene transcripts of the last third of the virus genome after the initial overlapping reading frame.
mRNAs lati jaangirde gene gooto nder tati ragare genome viriis ha ɓawo aranol jaangirde ndu.
These mRNAs are translated by the host's ribosomes into the structural proteins and a number of accessory proteins.
mRNAs ndu lornama gal jaɓɓowo ribosomeji ha nder structural proteinji be proteinji feere.
RNA translation occurs inside the endoplasmic reticulum.
lornugo RNA ɗon salla nder retikulum endoplasmic.
The viral structural proteins S, E, and M move along the secretory pathway into the Golgi intermediate compartment.
Viral structural proteins S, E, be M ɗon yahda gal lawol tuttol ha nder suudu Golgi je cakkare.
There, the M proteins direct most protein-protein interactions required for assembly of viruses following its binding to the nucleocapsid.
Ha ton, M proteinji M ɗon nelda kuɗe protein-protein mari haaje mofturde viriisji gal tokkitol hawtirde maɓɓe be nucleocapsid.
Progeny viruses are then released from the host cell by exocytosis through secretory vesicles.
Viriisji asngol yoffotto ha host cell gal exocytosis gal lawol wurtol tuttol.
The interaction of the coronavirus spike protein with its complement host cell receptor is central in determining the tissue tropism, infectivity, and species range of the virus.
Kuɗe koronaviriis spike protein be host cell receptor kanduɗum nder fammugo tissue tropism, raɓɓ, asngol e caalage viriis.
The SARS coronavirus, for example, infects human cells by attaching to the angiotensin-converting enzyme 2 (ACE2) receptor.
Koronaviriis SARS, bana sappinol, ɗon memma cellji gal hawtugo be angiotensin-converting enzyme 2 (ACE2) receptor.
The scientific name for coronavirus is Orthocoronavirinae or Coronavirinae.
Inde jaangirde koronaviriis woni Orthocoronavirinae mala Coronavirinae.
Coronaviruses belong to the family of Coronaviridae, order Nidovirales, and realm Riboviria.
Koronaviriis ɗon nasta nder geeɓe Coronaviridae, tokkitol Nidovirales, be asli Riboviria.
Genus Betacoronavirus; type species: Murine coronavirus
Inde Betacoronavirus; irin geeɓe: Murine coronavirus
Species: Betacoronavirus 1 (Bovine Coronavirus, Human coronavirus OC43), Human coronavirus HKU1, Murine coronavirus, Pipistrellus bat coronavirus HKU5, Rousettus bat coronavirus HKU9, Severe acute respiratory syndrome-related coronavirus (SARS-CoV, SARS-CoV-2), Tylonycteris bat coronavirus HKU4, Middle East respiratory syndrome-related coronavirus, Hedgehog coronavirus 1 (EriCoV)
Asli: Betacoronavirus 1 (Koronaviriis Na'i, Koronaviriis Neɗɗo OC43), Neɗɗo HKU1, Murine koronaviriis, Pipistrellus koronaviriis bilwili HKU5, Rousettus koronaviriis bilwili HKU9, koronaviriis je ɗon hokka nyau pofɗe nyaɗum (SARS-CoV, SARS-CoV-2), Tylonycteris koronaviriis bilwili HKU4, koronaviriis je ɗon hokka nyau je saɗirma nder pofɗe Cakkare Funaange, koronaviriis 1 camnagel (EriCoV)
Species: Beluga whale coronavirus SW1, Infectious bronchitis virus
Asli: Beluga whale koronaviriiss SW1, viriis Infectious bronchitis
The most recent common ancestor (MRCA) of all coronaviruses is estimated to have existed as recently as 8000 BCE, although some models place the common ancestor as far back as 55 million years or more, implying long term coevolution with bat and avian species.
Maama asngol koronaviriis je wangi neeɓay (MRCA) waɗi bana yahan gal 8000 BCE, ko to irin feere ɗon yara maama je ɓurna fu gal duɓɓi milionji 55 mala ɓuri, je yiɗi sappinugo yahidottol juttungel be bilwili be cooli.
The most recent common ancestor of the alphacoronavirus line has been placed at about 2400 BCE, the betacoronavirus line at 3300 BCE, the gammacoronavirus line at 2800 BCE, and the deltacoronavirus line at about 3000 BCE.
Maama asngol alphakoronaviriis je wangi neeɓay ndu wonan gal 2400 BCE, asngol betakoronaviriis ha 3300 BCE, asngol gammakoronaviriis ha 2800 BCE, be asngol ddeltakoronaviriis gal 3000 BCE.
The large number of host bat and avian species, and their global range, has enabled extensive evolution and dissemination of coronaviruses.Many human coronavirus have their origin in bats.
Limtol geeɓeji bilwili siggorde be cooli, be hawtirde maɓɓe, sappini yahde be sankittol koronaviriisji je juttungel. Koronaviriis neɗɗo ɗuɗɗum ummi ha bilwili.
More recently, alpaca coronavirus and human coronavirus 229E diverged sometime before 1960.
Neeɓay ɗo, koronaviriis alpaka be koronaviriis neɗɗo 229E waylitti ha wakati feere hiddeko 1960.
MERS-CoV emerged in humans from bats through the intermediate host of camels.
MERS-CoV wangi ha neɗɗo diga bilwili be suudu cakkare mako ngeeloba.
MERS-CoV, although related to several bat coronavirus species, appears to have diverged from these several centuries ago.
MERS-CoV, ko to larni asli koronaviriis bilwili jur ma, bana waylitti ha maɓɓe diga ujineje ɗuɗum.
The most closely related bat coronavirus and SARS-CoV diverged in 1986.
Koronaviriis bilwili je ɓuri ɓadugo be SARS-CoV waylitti ha 1986.
A possible path of evolution, of SARS coronavirus and keen bat coronaviruses, suggests that SARS related coronaviruses coevolved in bats for a long time.
Lawol yahde ndu koronaviriis SARS be koronaviriisji bilwili, ɗon sappina yo'o koronaviriisji SARS yahidi no ha nder bilwili diga ɓoyma.
In the 1790s, equine coronavirus diverged from the bovine coronavirus after a cross-species jump.
Ha 1790, koronaviriis pucci waylitti diga koronaviriis na'i ha ɓawo hawtirde maɓɓe.
Later in the 1890s, human coronavirus OC43 diverged from bovine coronavirus after another cross-species spillover event.
Ha ɓawo man gal 1890, koronaviriis neɗɗo OC43 waylitti diga koronaviriis na'i ha ɓawo hawtirde maɓɓe.
It is speculated that the flu pandemic of 1890 may have been caused by this spillover event, and not by the influenza virus, because of the related timing, neurological symptoms, and unknown causative agent of the pandemic.
Yimɓe tammi yo'o nyau ndamba je 1890 je memmei duniyaru fu footi wangi gal kuje feere, amma na gal viriis ndamba, ngam wakati man, mandolji neuronji, be gilɗi je footi wangini nyau duniyaru ndu je aandakke.
Human coronavirus OC43 besides causing respiratory infections is also suspected of playing a role in neurological diseases.
Koronaviriis neɗɗo OC43 do hokkugo nyauji pofɗe woni bana ɗon fiija nder sallinugo nyauji neuronji.
In the 1950s, the human coronavirus OC43 began to diverge into its present genotypes.
Gal 1950, koronaviriis OC43 fuɗɗi waylittugo ha yotta genotypeji ndu.
Phylogentically, mouse hepatitis virus (Murine coronavirus), which infects the mouse's liver and the central nervous system, is related to human coronavirus OC43 and bovine coronavirus.
Bana lartol wangirde maɗa, viriis hengre doombi (koronaviriis Murine), je ɗon memma hengre doomru be kugal ngandi, ɗon hawti be koronaviriis neɗɗo OC43 be koronaviriis na'i.
Human coronavirus HKU1, like the aforementioned viruses, also has its origins in rodents.
Koronaviriis neɗɗo HKU1, bana viriisji je en limti ha do ndu, wangi gal yaamooɓe.
Coronaviruses vary significantly in risk factor.
Koronaviriis ɗon saanja ɗuɗɗum do ko ɗon wangina ɗum.
Some can kill more than 30% of those infected, such as MERS-CoV, and some are relatively harmless, such as the common cold.
Feere footi mbara ɓuran 30 nder 100 ha cakka nyauɓe, bana MERS-CoV, e feere lati bana nyaɗay, bana ndamba tan.
Coronaviruses can cause colds with major symptoms, such as fever, and a sore throat from swollen adenoids.
Koronaviriisji footi hokka ndamba be ɓurna mandolji bana wullol baandu, be daande be huudure ngam adenoidji ɗon uppi.
Coronaviruses can cause pneumonia (either direct viral pneumonia or secondary bacterial pneumonia) and bronchitis (either direct viral bronchitis or secondary bacterial bronchitis).
Koronaviriisji footi hokka pneumonia (pneumonia viriis aranool mala pneumonia bakteriyaji ɗiɗaɓool) be bronchitis (bronchitis viriis aranool mala bronchitis bakteriyaji ɗiɗiɓool).
The human coronavirus discovered in 2003, SARS-CoV, which causes severe acute respiratory syndrome (SARS), has a unique pathogenesis because it causes both upper and lower respiratory tract infections.Six species of human coronaviruses are known, with one species subdivided into two different strains, making seven strains of human coronaviruses altogether.
Koronaviriis neɗɗo je tawama ha 2003, SARS-CoV, je ɗon hokka nyau pofɗe je nyaɗum (SARS), ɗon mari mandol nyau feerejo ngam ɗon raɓɓa lawol pofɗe je do be je les. Asli koronaviriis neɗɗo joowey e go'o ɗon aandin, be asngol gootoo je ɗon sendi nde caalaje ɗiɗi, ko wartiri ndu koronaviriisji neɗɗo joowey e ɗiɗi.
Four of these coronaviruses continually circulate in the human population and produce the generally mild symptoms of the common cold in adults and children worldwide: -OC43, -HKU1, HCoV-229E, -NL63.
Nay ha cakka koronaviriisji ndu ɗon tokkita doggugo ha cakka ummatore e ɓurna fu ɗon wangina mandolji ndamba je nyaɗay ha mauɓe be ɓikkon ha nder duniyaru fu: -OC43, -HKU1, HCoV-229E, -NL63.
Coronaviruses cause about 15% of commons colds.
Koronaviriisji ɗon hokka 15% ndamɗa meere.
The majority of colds are caused by rhinoviruses.
Ndambaji ɓurna fu wangi gal rhinoviriisji.
The four mild coronaviruses have a seasonal incidence occurring in the winter months in temperate climates.
Koronaviriisji nay je nyaɗay sam ɗon mari yahde be wakati e ɗon wanga ha yakke peewol ha wakkereji feere.
There is no preference towards a particular season in tropical climates.
Wala saanjol be wakatiji nde gal tropikji.
Four human coronaviruses produce symptoms that are generally mild:
Koronaviriisji neɗɗo nay ɗon sappina mandolji je ɓurna fu ɗon:
The virus was officially named the SARS coronavirus (SARS-CoV).
Viriis hoosi inde SARS coronaviriis (SARS-CoV).
More than 8,000 people were infected, about ten percent of whom died.
Yimɓe ɓuran 8000 raɓɓi nyau ndu, je sappo nder temere ha cakka maɓɓe mayi.
In September 2012, a new type of coronavirus was identified, initially called Novel Coronavirus 2012, and now officially named Middle East respiratory syndrome coronavirus (MERS-CoV).
Ha septambre 2012, wodi koronaviriis keese wangi, aranool man inde man Koronaviriis 2012 Keese, amma jonta hoosi inde koronaviriis Cakkare Funaange je ɗon hokka nyau pofɗe (MERS-CoV)
The World Health Organization issued a global alert soon after.
Geeɓe Duniyaru do Jam Ɓaanɗu daasi hakkilo duniyarru fu ha ɓawo man.
The WHO update on 28 September 2012 said the virus did not seem to pass easily from person to person.
Matinolji WHO je ɗon larti ha 28 Septambre 2012 mbi'i viriis ndu bana woway sallugo diga neɗɗo ha neɗɗo.
However, on 12 May 2013, a case of human-to-human transmission in France was confirmed by the French Ministry of Social Affairs and Health.
Amma, ha 12 Me 2013, wodi nyau man je salli diga neɗɗo ha neɗɗo wangi ha Faraansa e Ardudo ko Larni Kujeji Ummatore be Jam Ɓaandudu lesdi Faraansa on matini.
In addition, cases of human-to-human transmission were reported by the Ministry of Health in Tunisia.
Ha ɓesdugo, nyauɓe je sallinol neɗɗo ha neɗɗo matinama gal Aduɗo jam ɓaandu ha Tunisiya.
Two confirmed cases involved people who seemed to have caught the disease from their late father, who became ill after a visit to Qatar and Saudi Arabia.
Nyauɓe ɗiɗi je ɗon aandi mari nyau larni yimɓe je nangi nyau ha baabirawo maɓɓe je maynama, je nyawi ha ɓawo ngilli ha Qatar be Saudi Arabiya.
Despite this, it appears the virus had trouble spreading from human to human, as most individuals who are infected do not transmit the virus.
Ngam ndu, lati bana viriis man mari larula sallugo diga neɗɗo ha neɗɗo, ngam ɓurna yimɓe je mari nyau sallinatta viriis.
The final name for the virus is Middle East respiratory syndrome coronavirus (MERS-CoV).
Ragare inde viriis nyau koronaviriis Cakkare Funaange je ɗon hokka saɗirma nder pofɗe (MERS-CoV).
The only U.S. cases (both survived) were recorded in May 2014.In May 2015, an outbreak of MERS-CoV occurred in the Republic of Korea, when a man who had traveled to the Middle East, visited four hospitals in the Seoul area to treat his illness.
Nyauɓe Lesɗi America (ɗiɗi fu nyamɓitti) wangi ha Me 2014. Ha Me 2015, wodi nyau feere wangi ha Republic of Korea, to goɗɗo feere je waɗi jaahangal he Cakkare Funaange, ngilli cuuɗi lekki nay ha wakkere Seoul ngam hurgugo nyau mako.
This caused one of the largest outbreaks of MERS-CoV outside the Middle East.
Ndu wartiri gooto cakka nyau MERS-CoV je ɓuri yajjugo ha yaasi Cakkare Funaange
As of December 2019, 2,468 cases of MERS-CoV infection had been confirmed by laboratory tests, 851 of which were fatal, a mortality rate of approximately 34.5%.
Ha Desambre 2019, nyauɓe MERS-CoV 2 468 ɗon aandi bana ni gal lartolji suudu lartol, 851 ha kunde maɓɓe maynama, limtol mayde ɓadi 34.5%.
In December 2019, a pneumonia outbreak was reported in Wuhan, China.
Ha Desambre 2019, wangol pneumonia matinama ha Wuhan, Cayna.
On 31 December 2019, the outbreak was traced to a novel strain of coronavirus, which was given the interim name 2019-nCoV by the World Health Organization (WHO), later renamed SARS-CoV-2 by the International Committee on Taxonomy of Viruses.
Ha 31 Desambre 2019, nyau wangi bana asngol koronaviriis keese, je ɓe hokki mo inde 2019-n-CoV tawon gal Geeɓe Duniyaru do Jam Ɓaandu.
Some researchers have suggested the Huanan Seafood Wholesale Market may not be the original source of viral transmission to humans.As of 17 April 2020, there have been at least 153,822 confirmed deaths and more than 2,240,191 confirmed cases in the coronavirus pneumonia pandemic.
Teffoɓe feere tammi yo'o Huanan Seafood Market footi na kanjuum on woni ɓundu sallinol viriis ndu ha neɗɗo. Ha 17 Aviiril 2020, yimɓe pettum man 153 822 maynama be 2 240 191 nangi nyau pneumonia koronaviriis je ɗon aandi.
The Wuhan strain has been identified as a new strain of Betacoronavirus from group 2B with approximately 70% genetic similarity to the SARS-CoV.
Asngol Wuhan ɗon maandi no bana Betakoronaviriis ha nder geeɓe 2B be ɓadi 70% naandu gene SARS-CoV.
The virus has a 96% similarity to a bat coronavirus, so it is widely suspected to originate from bats as well.
Viriis ndu ɗon naandi be 96% be koronaviriis bilwili feere, nden kadi yimɓe jur ɗon tammi wangi gal bilwili.
The pandemic has resulted in travel restrictions and nationwide lockdowns in several countries.
Nyau duniyaru ndu wangini haɗɗolji jaahangal be je wurtugo ha nder lesɗe fu.
Coronaviruses have been recognized as causing pathological conditions in veterinary medicine since the 1930s.
koronaviriis maandotto bana wurtinugo maandolji ha nder jam ɓaandu daabaji diga 1930.
Except for avian infectious bronchitis, the major related diseases have mainly an intestinal location.
Sna je bronchitis cooli je ɗon raɓɓa, ɓurna nyauji man ɗon wondi ha nder wakkere teeteki.
Coronaviruses primarily infect the upper respiratory and gastrointestinal tract of mammals and birds.
Koronaviriisji ɗon raɓɓa lawol pofɗe je do be gastrointestinal daabaji be cooli.
They also cause a range of diseases in farm animals and domesticated pets, some of which can be serious and are a threat to the farming industry.
Fahin bo'o ɓe ɗon wangina nyauji feere ha daabaji je daabaji saare, feere'en bo'o footi wona nyaɗum e lati sababu ha wakkere durugo.
In chickens, the infectious bronchitis virus (IBV), a coronavirus, targets not only the respiratory tract but also the urogenital tract.
Ha goortoɗe, viriis bronchitis je ɗɓon raɓɓa (IBV), wodi koronaviriis feere, larnay lawol pofɗe tan amma lawol siile.
The virus can spread to different organs throughout the chicken.
Viriis ndu footi sankitta ha nder kujeji ɓaandu feere fu ha goortoɗe.
Economically significant coronaviruses of farm animals include porcine coronavirus (transmissible gastroenteritis coronavirus, TGE) and bovine coronavirus, which both result in diarrhea in young animals.
Koronaviriis je ɓurna fu larni daabaji saare bana koronaviriis gaduji (koronaviriis gastroenteritis je ɗon raɓɓe, TGE) be koronaviriis na'i, be sappinol fu be caarol ha ɓikkon daabaji.
Feline coronavirus: two forms, feline enteric coronavirus is a pathogen of minor clinical significance, but spontaneous mutation of this virus can result in feline infectious peritonitis (FIP), a disease associated with high mortality.
Koronaviriis paturu: irin ɗiɗi, koronaviriis paturu ɗon raɓɓa goɗɗo amma nyaɗay masin, amma sanjol laugel viriis ndu footi lata nyau paturu peritonitis (FIP), nyau je ɗon mbara yimbe jur.
Similarly, there are two types of coronavirus that infect ferrets: Ferret enteric coronavirus causes a gastrointestinal syndrome known as epizootic catarrhal enteritis (ECE), and a more lethal systemic version of the virus (like FIP in cats) known as ferret systemic coronavirus (FSC).
Fahin bo'o, wodi irin koronaviriisji ɗiɗi je ɗon raɓɓa, jiire: koronaviriis enteric jiire ɗon hokka nyau reedu je ɗon aandi bana ndamba daabaji enteritis (ECE), be wangol viriis man keese je ɓuri nyaɗugo (bana FIP ha paatuji je ɗon aandi bana koronnaviriis systemic ferret (FSC).
There are two types of canine coronavirus (CCoV), one that causes mild gastrointestinal disease and one that has been found to cause respiratory disease.
Wodi irin koronaviriis paatuji ɗiɗi (CCoV), gooto je ɗon hokka nyau reedu e je ɗon tawi bana je hokkata nyau pofɗe.
Mouse hepatitis virus (MHV) is a coronavirus that causes an epidemic murine illness with high mortality, especially among colonies of laboratory mice.
Viriis henre Doomdu (MHV) woni koronaviriis je ɗon hokka nyau murine be limtol mayde, ɓurna fu ha cakka doombi suddu lartol
Sialodacryoadenitis virus (SDAV) is highly infectious coronavirus of laboratory rats, which can be transmitted between individuals by direct contact and indirectly by aerosol.
Viriis Sialodacryoadenitis (SDAV) woni coronaviris je ɗon nyaɗi masin ha doomruweela suudu lartol je footi yimɓe sallina ha kunde maɓɓe gal ngidol mala gal hendu.
Some strains of MHV cause a progressive demyelinating encephalitis in mice which has been used as a murine model for multiple sclerosis.
Asli MHV feere ɗon hokka demyelinating encephalitis je ɗon yahida ha doombi je hoccama bana irin murine ha sclerosis jur.
Significant research efforts have been focused on elucidating the viral pathogenesis of these animal coronaviruses, especially by virologists interested in veterinary and zoonotic diseases.
Dabare teffol kanduɗum wondi do laɓɓinugo fuɗɗam pathogenesis viriis koronaviriis irin daabaji ndu, ɓurna fu gal jaanginooɓe viriisji je yardi ekkitol nyauji dabaji.
Porcine coronavirus (transmissible gastroenteritis coronavirus of pigs, TGEV).
Koronaviisus gaduji (koronaviriis reedu gaduji, TGEV).
Bovine coronavirus (BCV), responsible for severe profuse enteritis in of young calves.
Koronaviriis na'i (BCV), je ɗon hokka nyau teeteki ha ɓikkon na'i.
Feline coronavirus (FCoV) causes mild enteritis in cats as well as severe Feline infectious peritonitis (other variants of the same virus).
Koronaviriis paaturu (FCoV) ɗon hokka nyau teeteki seeɗa ha paatuji be nyau reedu Paatuji je ɗon nyaɗi (irin viriis man je feere).
the two types of canine coronavirus (CCoV) (one causing enteritis, the other found in respiratory diseases).
irin koronviriis paatuji je ɗon ɗiɗi ndu (CCoV) (gooto ɗon raɓɓa teeteki, feere tawama ha nder nyau pofɗe).
Turkey coronavirus (TCV) causes enteritis in turkeys.
Koronaviriis paula (TCV) ɗon raɓɓa teeteki paulaji.
Ferret enteric coronavirus causes epizootic catarrhal enteritis in ferrets.
Koronaviriis Jiire ɗon raɓɓa jirre be ndamba nder teeteki.
Ferret systemic coronavirus causes FIP-like systemic syndrome in ferrets.
Koronaviriis systemic Jiire ɗon hokka irin nyau systemic ha jiire.
Rabbit enteric coronavirus causes acute gastrointestinal disease and diarrhea in young European rabbits.
Koronaviriis systemic boojel ɗon memma reedu be teeteki be caarol ha ɓikkon boojelji Erop.
Porcine epidemic diarrhea virus (PED or PEDV), has emerged around the world.
Viriis nyau caarol gaduji (PED mala PEDV), waangi ha nder duniyaru.
b'Coronavirus disease 2019 (COVID-19) is an infectious disease caused by severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2).
Nyau koronaviriis b' 2019 (COVID-19) lati nyau je ɗon raɓɓa be saɗirma nder pofɗe je wangi gal koronaviiris 2 (SARS-CoV-2).
Common symptoms include fever, cough, and shortness of breath.
Mandolji je ɓurna larni wullol ɓaandu, sondaru, be pofɗe bana daran.
Other symptoms may include fatigue, muscle pain, diarrhea, sore throat, loss of smell, and abdominal pain.
Mandolji feere footi larni tampere, nawne ɓaandu, caarol, daande be huudure, majjinol haccenga, nawne reedu.
The time from exposure to onset of symptoms is typically around five days but may range from two to fourteen days.
Wakati diga raɓɓol ha fuɗɗam mandolji ɗon gal balɗe joowey amma footi yaha diga balɗe ɗiɗi ha sappo e nay.
While the majority of cases result in mild symptoms, some progress to viral pneumonia and multi-organ failure.
Nyauɓe ɓurna fu ɗon mari mandolji je nyaɗay, amma feere footi yotta ha pneumonia be nyau kujeji reedu feere-feere.
As of 17 April 2020, more than 2.24 million cases have been reported across 210 countries and territories, resulting in more than 153,000 deaths.
Ha 17 Aviiril 2020, ɓuri nyauɓe milionji 2.24 matinama bana mari nyau ha nder lesɗe be wakkereji 210, be ɓuri yimɓe 153 000 maynama.
More than 568,000 people have recovered.The virus is primarily spread between people during close contact, often via small droplets produced by coughing, sneezing, or talking.
Yimɓe ɓuri 568 000 nyamɓitti. Viriis sankitti ha kunde ummatore ha aranool man gal wondidugo, ɓurna fu gal tuɗe je ɗon toɓa ha yakke sondaru, ɓislol, mala ngewte.
While these droplets are produced when breathing out, they usually fall to the ground or onto surfaces rather than being infectious over long distances.
Ha yakke je tuɗe ɗon toɓa ha wakati fuffugo, ɗon do'a ha lesdi mala ha do daaldal na hoorama ngam raɓɓugo yimɓe gal hendu.
People may also become infected by touching a contaminated surface and then touching their eyes, nose, or mouth.
Yimɓe footi nanga nyau gal memmugo baabe je ɗon raɓɓi e ha ɓawo man memma gite gite, kine mala hunduko maɓɓe.
The virus can survive on surfaces up to 72 hours.
Viriis ndu footi yeeɗa do daldal ha njamdi 72.
It is most contagious during the first three days after the onset of symptoms, although spread may be possible before symptoms appear and in later stages of the disease.The standard method of diagnosis is by real-time reverse transcription polymerase chain reaction (rRT-PCR) from a nasopharyngeal swab.
Nyau ndu ɓuri raɓɓugo ha arandere balɗe tati diga fuɗɗorde nyau ndu, ko to sankittol footi wona hiddeko wangol mandolji be gal ragare nyau ndu. Dabare lartol je yimɓe ɓuri aandugo woni jaangirde polymerase chain reaction (rRT-PCR) gal ɓawo gal illam kine mala daande.
The use of masks is recommended for those who suspect they have the virus and their caregivers.
Wondugo be leepolji suddugo yeeso ɗon feeli ha yimɓe je tammi marugo viriis be ɓe'en je suklata do maɓɓe.
Recommendations for mask use by the general public vary, with some authorities recommending against their use, some recommending their use, and others requiring their use.
Wondugo be leepol suddugo yeeso gal ummatore ɗon saanja, be arduɓe feere je yarday yimɓe ɓorna ɗum, feere'en bo'o ɗon feela ɓornugo ɗum, e feere bo'o ɗon dula ɓornugo ndu.
Currently, there is no vaccine or specific antiviral treatment for COVID-19.
Ha jonta, walla baatal mala lekki hurgol viriis je ɗon aandi ngam COVID-19.
Local transmission of the disease has been recorded in most countries across all six WHO regions.
Sallinol nyau ha cakka ummatore ɗon matinama he ɓurna lesɗe fu nder wakkereji WHO.
Those infected with the virus may be asymptomatic or develop flu-like symptoms such as fever, cough, fatigue, and shortness of breath.
Yimɓe je viriis raɓɓi ɓe footi marata mandolji nyau mala sappina mandolji bana je ndamba bana wullol baandu, sondaru, tampere, be pofɗe bana daran.
Emergency symptoms include difficulty breathing, persistent chest pain or pressure, confusion, difficulty waking, and bluish face or lips; immediate medical attention is advised if these symptoms are present.
Mandolji fuɗɗam larni saɗirma pofɗe, nawol mala harlol ɓernde je ɗon neeɓa, harbol, saɗirma be finnugo, saanjol yeeso be hunduko; to mandolji ndu wangi sey a ɓadita yimɓe jam ɓaandu.
Less commonly, upper respiratory symptoms such as sneezing, runny nose or sore throat may be seen.
Ha yakke feere, mandolji pofɗe do bana ɓislugo, illol kine mala daande be huudure footi wona.
Gastrointestinal symptoms such as nausea, vomiting and diarrhoea have been observed in varying percentages.
Mandolji reedu mala teeteki bana tutta, tuttol be caarol woni be limtol je ɗon saanja tum.
Some cases in China initially presented only with chest tightness and palpitations.
Nyauɓe feere ha Cayna sappini ha arandere harlol ɓernde be duwinol ɓernde.
In some, the disease may progress to pneumonia, multi-organ failure, and death.
Ha feere'en, nyau man footi waylitta do pneumonia, saɗirma kujeji reedu jur be wade.
This is called the incubation period.
Ndu ewnotto wakati nuftol.
The incubation period for COVID-19 is typically five to six days but may range from two to 14 days.
Wakati nuftol COVID-19 woni balɗe joowey ha joowey go'o amma footi wona ha kunde balɗe ɗiɗi ha 14.
97.5% of people who develop symptoms will do so within 11.5 days of infection.Reports indicate that not all who are infected develop symptoms.
97.5% yimɓe je sappini mandolji waɗan bana ni ha nder balɗe 11.5 nyau ndu. Batakkeji hubaruji ɗon sappina yo'o na kala moy fu je mari nyau on sappinatta mandolji ndu.
The role of these asymptomatic carriers in transmission is not yet fully known; however, preliminary evidence suggests that they may contribute to the spread of the disease.
Kugal irin roondooɓe ha nder sallinol aandake boɗɗum tawon; amma, arandere sappinolji ɗon holla yo'o footi ɓe naffa nder sankittugo nyau ndu.
The proportion of infected people who do not display symptoms is currently unknown and being studied, with the Korea Centers for Disease Control and Prevention (KCDC) reporting that 20% of all confirmed cases remained asymptomatic during their hospital stay.
Ta'irde yimɓe je ɗon mari gilɗi ndu amma sappinata mandolji nyau ngu aandake e lati lartotto haande, ngam Ɗowirdeji Aynol be Fadol Nyau (KCDC) wi'i yo'o 20 nder 100 sappinay mandolji ha wakati jonde maɓɓe ha suudu lekki.
China's National Health Commission began including asymptomatic cases in its daily cases on 1 April; of the 166 infections on that day, 130 (78%) were asymptomatic at the time of testing.
China's National Health Commission fuɗɗi nastinugo nyauɓe je sappinata mandolji nyau ha nder nyauɓe balɗe ha 1 Aviiril; ha cakka nyauɓe 166 ha nder balɗe nde, 130 (78%) sappinatta no mandolji nyau ha wakati lartol.
Both sputum and saliva can carry large viral loads.
Didi fu tuutol be tuuɗe footi ronda gildi duɗɗum.
Loud talking releases more droplets than normal talking.
Wolwugo be sembe ɗon toɓina tuɗe jur do wolwugo be leesare.
A study in Singapore found that an uncovered cough can lead to droplets travelling up to 4.5 meters (15 feet).
Ekkitol feere ha Singapore tawi yo'o sondaru je sudday footi waɗa tuɗe do'a e footi yaha metaaji 4.5 (kosɗe).
Though the virus is not generally airborne, the National Academy of Science has suggested that bioaerosol transmission may be possible and air collectors positioned in the hallway outside of people's rooms yielded samples positive for viral RNA.
Ko to viriis hortakke gal hendu, National Academy of Science holli yo'o sallinol gal hendu footi waɗa, e hoccooɓe hendu je ɗon jo'ini ha dammuɗe jawleruji ɗon no roondi lekkuji je ɗon mari viris RNA.
Some medical procedures such as intubation and cardiopulmonary resuscitation (CPR) may cause respiratory secretions to be aerosolised and thus result in airborne spread.
Laahalji jam ɓaandu feere bana nastinugo kuje gal daande be ummitinugo ɓernde be fuffu (CPR) footi wangina illam pofɗe je footi wurta be pofɗe e bana ni hendu sankitta ndu.
While there are concerns it may spread via feces, this risk is believed to be low.The virus is most contagious when people are symptomatic; while spread may be possible before symptoms emerge, the risk is low.
Ha yakke je wodi larulaji ngam fammugo to footi salla gal buuwe, sababu ndu tammi lessi; ha yakke je sankittol footi salla hiddeko mandolji wanga, sababu ndu lessi.
The European Centre for Disease Prevention and Control (ECDC) says while it is not entirely clear how easily the disease spreads, one person generally infects two to three others.The virus survives for hours to days on surfaces.
Ɗowirɗe Aynol be Lartol Nyau (ECDC) wi'i ko to wanay laɓɗe fammugo noy nyau man footi sankitta be kooyɗum, goɗɗo gooto footi raɓɓa yimɓe ɗiɗi mala tati. Viriis footi nyeeɗa do daaldalji nder njamɗe ha balɗe.
Specifically, the virus was found to be detectable for one day on cardboard, for up to three days on plastic (polypropylene) and stainless steel (AISI 304), and for up to four hours on 99% copper.
Viriis ndu tawama bana footi maanda ɗum nder balɗe woore ha do takkarde, ngam ha balɗe tati do leeda (polypylene) be nyama larta (AISI 304), e ha njamɗe do 99% cardi.
This, however, varies depending on the humidity and temperature.
Amma, ndu ɗon saanja be peewol be gulɗam.
Soap and detergent are also effective if correctly used; soap products degrade the virus' fatty protective layer, deactivating it, as well as freeing them from skin and other surfaces.
Sabulu be omo ɗon naffa bo'o to hocci be lawol man; kujeji sabulu ɗon ɓoora ɓeelere je ɗon tardi gilɗi ndu, itta ɗum, e willa ɓe do ɓaandu be daaldalji feere.
Other solutions, such as benzalkonium chloride and chlorhexidine gluconate (a surgical disinfectant), are less effective.In a Hong Kong study, saliva samples were taken a median of two days after the start of hospitalization.
Dabareji feere bana benzalkonium chloride be chlorhexidine gluconate (kuje laɓɓinugo seekkowo), maray sembe jur. Ekkitol feere ha Hong Kong, leeku tuɗe ɗon no hoosa ɓurna balɗe ɗiɗi ha ɓawo fuɗɗam wondugo ha suudu lekki.
In five of six patients, the first sample showed the highest viral load, and the sixth patient showed the highest viral load on the second day tested.
Ha nder cakka nyauɓe nay do joowey, aranool leeku sappini limtol viriis je ɓuri fu, be joowey e woore nyauɗo sappini limtol viriisji je ɓuri ha ɗiɗaɓre balɗe lartol.
Severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) is a novel severe acute respiratory syndrome coronavirus, first isolated from three people with pneumonia connected to the cluster of acute respiratory illness cases in Wuhan.
Koronaviriis 2 (SARS-CoV-2) je ɗon hokka nyau saɗirma pofɗe woni koronaviriis keese be je saɗirma pofɗe, tawama arandere ha yimɓe tati be pneumonia je hawti be sammere nyau pofɗe ha Wuhan.
All features of the novel SARS-CoV-2 virus occur in related coronaviruses in nature.
Mandolji viriis SARS-CoV keese ɗon salla bana je koronaviriis.
Outside the human body, the virus is killed by household soap, which bursts its protective bubble.SARS-CoV-2 is closely related to the original SARS-CoV.
Ha yaasi ɓaandu neɗɗo, sabulu saare ɗon mbarda viriis man, je ɗon feetina nguffu je ɗon fadi mo. SARS-CoV-2 ɗon naandi be arandere SARS-CoV.
The lungs are the organs most affected by COVID-19 because the virus accesses host cells via the enzyme angiotensin-converting enzyme 2 (ACE2), which is most abundant in the type II alveolar cells of the lungs.
Fuffu lati ko je COVID-19 ɗon memma jur ngam viriis ɗon nasta cellji jaɓɓoowo gal neebam angiotensin-converting enzyme 2 (ACE2), je ɓuri wondugo ha type II alveolar cellji fuffu.
The virus uses a special surface glycoprotein called a "spike" (peplomer) to connect to ACE2 and enter the host cell.
Viriis ndu ɗon hoosa daaldal glycoprotein feerejo ewnotto "hoore" (peplomer) ngam hawtugo be ACE2 e nastugo nder cell jaɓɓowo.
Acute cardiac injury was found in 12% of infected people admitted in hospital in Wuhan, China, and is more frequent in severe disease.
Huudure seeɗa do ɓernde tawama ha 12% yimɓe je mari nyau e je wondi ha suudu lekki ha Wuhan, Cayna, e ɗon woni ɓurna fu ha nder nyau ndu.
Rates of cardiovascular symptoms is high, owing to the systemic inflammatory response and immune system disorders during disease progression, but acute myocardial injury may also be related to ACE2 receptors in the heart.
Limtolji mandolji nyau ɓernde ɗon wa'i, ngam jabbuye do dabare hurgugo huudure be fadol ha wakati yahde nyau, amma huudure myokard footi bo'o larna jaɓɓoowo ACE2 ha nder ɓernde.
ACE2 receptors are highly expressed in the heart and are involved in heart function.
Jaɓɓoowo ACE2 ɗon sappini ha nder ɓernde e ɗon naffa nder kugal ɓernde.
A high incidence of thrombosis (31%) and venous thromboembolism (25%) have been found in ICU patients with COVID-19 infections and may be related to poor prognosis.Autopsies of people who died of COVID-19 have found diffuse alveolar damage (DAD), and lymphocyte-containing inflammatory infiltrates within the lung.
Be tawi yo'o wodi kuje feere ɗon fuɗa ha nder iyam maɓɓe (31%) e ko mbiyotto thromboembolism (25%) tawama ha nyauɓe ICU je ɗon mari nyau COVID-19 e footi en yecca yahay. Lartol ɓaandu nyauɓe COVID-19 je maynama sappini nawnol fuffu (DAD), e ndiyam iyam je ɗon mari ko nyauwata ndu ɗon no illa nder fuffu.
Although SARS-COV-2 has a tropism for ACE2-expressing epithelial cells of the respiratory tract, patients with severe COVID-19 have symptoms of systemic hyperinflammation.
Ko to SARS-COV-2 ɗon mari tropism ngam ACE2-expressing epithelial cellji lawol pofɗe, nyauɓe be COVID-19 je nyaɗi ɗon mari mandolji yajjugo huudure ndu.
In particular, pathogenic GM-CSF-secreting T-cells were shown to correlate with the recruitment of inflammatory IL-6-secreting monocytes and severe lung pathology in COVID-19 patients.
Ba wi'ugo, pathogenic GM-CSF-je ɗon ruffa T-cellji bana ɗon yahida be nastirde inflammatory IL-6-je ɗon fuffa monocyteji be mandolji fuffu je nyaɗum ha nyauɓe COVID-19.
Lymphocytic infiltrates have also been reported at autopsy.
Illol lymphocyt ha toon bo'o matinama ha nder mayde.
The WHO has published several testing protocols for the disease.
WHO wangini dabareji lartol nyau ndu.
The standard method of testing is real-time reverse transcription polymerase chain reaction (rRT-PCR).
Dabare lartol je ɗon aandi woni real-time reverse transcription polymerase chain reaction (rRT-PCR).
The test is typically done on respiratory samples obtained by a nasopharyngeal swab; however, a nasal swab or sputum sample may also be used.
Lartol ndu ngaɗama ɓurna fu be leeku pofɗe je ɓe ɗon hoosa gal illam kine mala je daande; amma, footi bo'o hoosa leeku ndamba mala illam kine.
Results are generally available within a few hours to two days.
Sappinolji ɓurna fu ɗon wanga ha nder njamɗe seeɗa mala balɗe ɗiɗi.
Blood tests can be used, but these require two blood samples taken two weeks apart and the results have little immediate value.
Lartolji iyam footi naffa, amma kanjuum yiɗi sey hoosa leekuji ɗiɗi je hoccama asaweji ɗiɗi be sappinolji ndu je wangata law.
Chinese scientists were able to isolate a strain of the coronavirus and publish the genetic sequence so laboratories across the world could independently develop polymerase chain reaction (PCR) tests to detect infection by the virus.
Teffooɓe Cayna footi no wurtinugo asli koronaviriis feere e sappini ta'irde gene heɓɓa cuuɗi lartol ha nder duniyaru footi wangina lartolji polymerase chain reaction (PCR) ngam maandugo nyau viriis ndu.
As of 4 April 2020, antibody tests (which may detect active infections and whether a person had been infected in the past) were in development, but not yet widely used.
Ha Aviiril 2020, lartolji fadooɓe ɓaandu (je footi maanda nyauji e to goɗɗo nangi no nyau ara) ɗon no do yahde, amma je na yimɓe fu on huwatta be man tawon.
The Chinese experience with testing has shown the accuracy is only 60 to 70%.
Ekkitol yimɓe Cayna be lartol sappini yo'o goonakku maɗa woni 60 ha 70% tan.
The FDA in the United States approved the first point-of-care test on 21 March 2020 for use at the end of that month.Diagnostic guidelines released by Zhongnan Hospital of Wuhan University suggested methods for detecting infections based upon clinical features and epidemiological risk.
FDA ha Lesɗe America yardi aranol lartol ha wakkere jam ɓaandu ha 21 Maars 2020 ngam huwugo be man ha ragare lewru. Dabare lartol je waangi gal Zhongnan Hospital Wuhan University feeli laahalji maandugo nyauji gal mandolji nyau be sababu nyau.
Bilateral multilobar ground-glass opacities with a peripheral, asymmetric and posterior distribution are common in early infection.
Bilateral multilobar ground-glass opacities be sallinol yaasi, je yahiday be ɓawo tawama ɓurna fu ha nder aranool nyau.
Subpleural dominance, crazy paving (lobular septal thickening with variable alveolar filling), and consolidation may appear as the disease progresses.
Subpleural dominance, crazy paving (uppugo lobular septal be illol je ɗon saanja ha nder alveolji), be sattugo footi wona to bana nyau ndu ɗon yahda.
Few data are available about microscopic lesions and the pathophysiology of COVID-19.
Wala matinolji ɗuɗɗum do huudure petpetel be yahde COVID-19 ɓaandu.
The main pathological findings at autopsy are:
Sappinolji mandolji aranool ha lartol mayde woni:
Macroscopy: pleurisy, pericarditis, lung consolidation and pulmonary oedema
Makroscopy: pleurisy, pericarditis, sattugo fuffu be uppugo fuffu
Four types of severity of viral pneumonia can be observed:
Pneumonia viriis fotti sappina saɗirmaji nay:
mild pneumonia: pulmonary oedema, pneumocyte hyperplasia, large atypical pneumocytes, interstitial inflammation with lymphocytic infiltration and multinucleated giant cell formation
pneumonia je deydey: uppugo fuffu, pneumocyte hyperplasia, maunugo pneumocyteji, huudure interstitial be illol lymphocyte be wondugo cell je ɗon mauni be mari ɓiɓe jur
severe pneumonia: diffuse alveolar damage (DAD) with diffuse alveolar exudates.
pneumonia nyaɗum: diffuse alveolar damage (DAD) be diffuse alveolar exudateji.
DAD is the cause of acute respiratory distress syndrome (ARDS) and severe hypoxemia.
DAD is the cause of acute respiratory distress syndrome (ARDS) and severe hypoxemia.
healing pneumonia: organisation of exudates in alveolar cavities and pulmonary interstitial fibrosis
pneumonia je hurgotto: kawtirde exudateji ha nder alveolar cavities be pulmonary interstitial fibrosis
Blood: disseminated intravascular coagulation (DIC); leukoerythroblastic reaction
Iyam : ɗacce intravascular sankitti (DIC); leukoerythroblastic reaction
Preventive measures to reduce the chances of infection include staying at home, avoiding crowded places, washing hands with soap and water often and for at least 20 seconds, practising good respiratory hygiene and avoiding touching the eyes, nose or mouth with unwashed hands.
Dabareji aynol ngam ustugo sa'iji raɓɓugo nyau larni wondugo ha saare, luttugo kawtirde ummatore, lallugo juɗe be sabulu be ndiyam ha wakati fu ha nder sekonji 20 pettum man, laɓɓinugo lawol pofɗe be aynol tato on memma gite, kine mala hunduko be juɗe je lallay.
The CDC recommends covering the mouth and nose with a tissue when coughing or sneezing and recommends using the inside of the elbow if no tissue is available.
CDC ɗon feela suddugo hunduko be kine to a ɗon sonda mala ɓislugo e ɗon feela bo'o nder jokke jungo moɗon to wala leepol.
Proper hand hygiene after any cough or sneeze is encouraged.
Lallugo juɗe ha ɓawo sondaru mala ɓislol feelama.
The CDC has recommended the use of cloth face coverings in public settings, in part to limit transmission by asymptomatic individuals.Social distancing strategies aim to reduce contact of infected persons with large groups by closing schools and workplaces, restricting travel and cancelling large public gatherings.
CDC feeli ɓornugo limse suddugo yeeso ha wakkere ummatore, ngam ustugo raɓɓol gal yimɓe je sappinata mandolji tawon. Dabareji dayol be ummatore yiɗi ustugo ngidol be yimɓe je ɗon mari nyau ha nder wakkareji je yimɓe ɗuɗɗi ha ton gal maɓɓugo cuuɗi jaangirde be baabe kugal, ustugo jaahangalji be haɗɗugo mofturdeji ummatore.
Distancing guidelines also include that people stay at least 6 feet (1.8 m) apart.
Dabareji dayol larni bo'o wondugo ha pettum man kosɗe 6 (1.8 m) be goɗɗo.
There is no medication known to be effective at preventing COVID-19.As a vaccine is not expected until 2021 at the earliest, a key part of managing COVID-19 is trying to decrease the epidemic peak, known as "flattening the curve".
Wala lekki je ɗon aandi be fakatre ngam aynol nyau COVID-19. Ngam baatal footay wanga hiddeko 2021 ha pettum man, dabare aynugo do COVID-19 lati ustugo yahde nyau, je ɗon aandi bana "jippinugo yahde".
The CDC also recommends that individuals wash hands often with soap and water for at least 20 seconds, especially after going to the toilet or when hands are visibly dirty, before eating and after blowing one's nose, coughing or sneezing.
CDC feeli bo'o yimɓe lalla juɗe maɓɓe ha wakati fu be sabulu be ndiyam nder sekonji 20 pettum man, ɓurna bo'o to a yeehi ha gaɗɗa mala to bana juɗe ɗon mari cuddi, hiddeko nyammugo e ha ɓawo fuffugo kine, sondugo mala ɓislugo.