Judul: Pendugaan Sebaran Zooplankton Dengan Metode Hidroakuistik Di Perairan Pesisir Barat Sumatra
Abstrak: Jurnlah pulau di Indonesia sebanyak 17.508 pulau, panjang garis pantai 8 1.000 km dan terbentang landas kontinen yang luas, secara keseluruhan menghasilkan ekosistem yang baik dan berarti untuk daerah penangkapan ikan yang menjanjikan (Pasanbu, 1999). Masa depan perikanan Indonesia sendin (.le.~ avsa ntai dan samudera'l akan lebih berorientasi ke Lautan Hindia, karena di perairan ini persaingan terhadap kapal-kapal asing relatif kecil. Selain itu, daerah penangkapan ikan lainnya adalah Sumudera Pasifik di utara Irian Jaya, perairan utara ~ u l a i e sdi in ~ a uCti na ~elaianT. etapi, pemanfaatan yang ditujukan kepada bidang perikanan ini masih belum dilaksanakan secara optimal. Hal ini dikarenakan informasi mengenai sumber daya perikanan, terutama perikanan laut masih belum mencukupi. Perairan Pesisir Barat Sumatera merupakan salah satu wilayah laut Indonesia yang kaya akan sumber daya hayati. Salah satu cara untuk menduga kekayaan sumber hayati di perairan tersebut adalah dengan mengetahui jumlah kepadatan dan kelimpahan plankton. Menduga ukuran, jumlah kepadatan dan kelimpahan plankton serta sebaran plankton dapat menggunakan pendugaan rata-rata /urge/ s~reng~dlza n buck scullering volun~ep lankton yaitu dengan menggunakan sistem hidroakustik.
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Judul: Pendugaan Sebaran Makrozooplankton Menggunakna Metode Hidroakuistik di Samudera Hindia Perairan Barat Sumatera
Abstrak: Wilayah perairan Indonesia memiliki potcnsi sumbadaya alam baik hayati maupun non hayati yang sangat menjanjikan. Sumberdaya alam hayati seperti ikan menjadi salah satu ujung tombak ekspor hasillaut Indonesia. Salah satu pammeter yang cukup menentukan kelimpahan ikan di suatu perairan adalah kelimpahan plank-ton di perairan tersebut khususnya zooplankton yang merupakan sumber makanan bagi jenis ikan komersial penting yang hidup di lautan. Pendugaan sebaran dan kelimpahan zooplankton tersebut dapat dilakukan dengan metode hidroakustik.
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Judul: Uji kinerja promoter gen LEAFY dan AGAMOUS tanaman kakao (Theobroma cacao L.) pada tanaman tembakau (Nicotiana tabacum L.)
Abstrak: Performance of cocoa’s (Theobroma cacao L.) gene promoters, TcLFY and TcAG, in a tobacco plant (Nicotiana tabacum L.) used as a plant model has been tested. The two promoters were introduced into the tobacco plant cells using a modified leaf discs method employing Agrobacterium tumifaciens AGL0. The transformed tobacco plant cells were then regenerated and their morphology were observed periodically. The tobacco plants harbouring the gene promoter TcLFY, during regeneration, showed a leaf morphology similar to that of the positive control. However, the growth rate of tobacco plants inserted with promoter TcLFY was faster compared to that of the positive control. Tobacco plants harbouring gene promoter TcAG, during regeneration, showed imperfect leaf morphology. The growth rate of these tobacco plants was slower than that of the positive control and of the tobacco plants inserted harbouring gene promoter TcLFY. These results indicated that the introduced promoters affect the morphology of the tobacco plants. In order to further confirm the presence of the GFP gene and activation of TcLFY: GFP and TcAG: GFP, a molecular verification test was carried out. Variation of sucrose concentration in the media was made to examine the effect of sucrose on the activity promoters TcAG and TcLFY in tobacco plants. The sucrose concentration tested were 0%, 0,5% and 4% followed by observation on the 3rd and 7th day of cultivation. At 0% sucrose concentration the tobacco plants harbouring promoter TcLFY showed yellowish leaf, while those cultivated in the media with sucrose concentration of 0.5% and 4% grew well and normal. This indicated that sucrose affects the activity of the promoter TcLFY in tobacco plants. Tobacco plants inserted with promoter TcAG did not show significant morphological changes when grown in a medium of 0%, 0,5% or 4% sucrose concentration. This indicated that addition of sucrose does not significantly affect the induction of expression of the AG gene and the activation of the promoter TcAG.
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