Journal of Osteoporosis [20] A. A. Welch, A. Mulligan, S. A. Bingham, and K.-T. Khaw, “Urine pH is an indicator of dietary acid-base load, fruit and vegetables and meat intakes: results from the European Prospective Investigation into Cancer and Nutrition (EPIC)- Norfolk population study,” British Journal of Nutrition, vol. 99, no. 6, pp. 1335–1343, 2008. [21] E. A. Newsholme and A. R. Leech, Biochemistry for the Medical Sciences, Wiley, Chichester, UK, 1983. [22] A. T. Høstmark, “Dietary ‘acid load’ and self reporrted preva- lence of hip fractures: a population based, cross sectional study,” Norwegian Epidemiology, vol. 13, no. 1, pp. 193–198, 2003. [23] S. Kapicioglu, A. H. Baki, Y. Tekeliog˘lu, and K. Araz, “The eﬀect of cola consumption on oral mucosa in rats,” Diseases of the Esophagus, vol. 13, no. 1, pp. 69–71, 2000. [24] T. Jensdottir, P. Holbrook, B. Nauntofte, C. Buchwald, and A. Bardow, “Immediate erosive potential of cola drinks and orange juices,” Journal of Dental Research, vol. 85, no. 3, pp. 226–230, 2006. [25] E. N. Taylor and G. C. Curhan, “Fructose consumption and the risk of kidney stones,” Kidney International, vol. 73, no. 2, pp. 207–212, 2008. [26] U. S. Barzel and J. Jowsey, “The eﬀects of chronic acid and alkali administration on bone turnover in adult rats,” Clinical science, vol. 36, no. 3, pp. 517–524, 1969. [27] T. Remer and F. Manz, “Potential renal acid load of foods and its inﬂuence on urine pH,” Journal of the American Dietetic Association, vol. 95, no. 7, pp. 791–797, 1995. [28] T. Buclin, M. Cosma, M. Appenzeller et al., “Diet acids and alkalis inﬂuence calcium retention in bone,” Osteoporosis Inter- national, vol. 12, no. 6, pp. 493–499, 2001. [29] J. A. Bell and S. J. Whiting, “Eﬀect of fruit on net acid and urinary calcium excretion in an acute feeding trial of women,” Nutrition, vol. 20, no. 5, pp. 492–493, 2004. [30] H. M. Macdonald, A. J. Black, L. Aucott et al., “Eﬀect of potas- sium citrate supplementation or increased fruit and vegeta- ble intake on bone metabolism in healthy postmenopausal women: a randomized controlled trial,” American Journal of Clinical Nutrition, vol. 88, no. 2, pp. 465–474, 2008. [31] P. Burckhardt, “The eﬀect of the alkali load of mineral water on bone metabolism: interventional studies,” Journal of Nutrition, vol. 138, no. 2, pp. 435S–437S, 2008. [32] K. L. Tucker, K. Morita, N. Qiao, M. T. Hannan, L. A. Cupples, and D. P. Kiel, “Colas, but not other carbonated beverages, are associated with low bone mineral density in older women: the Framingham osteoporosis study,” American Journal of Clinical Nutrition, vol. 84, no. 4, pp. 936–942, 2006. [33] E. Wynn, M. A. Krieg, S. A. Lanham-New, and P. Burckhardt, “Postgraduate symposium: positive inﬂuence of nutritional alkalinity on bone health,” Proceedings of the Nutrition Society, vol. 69, no. 1, pp. 166–173, 2010. [34] A. A. Welch, S. A. Bingham, J. Reeve, and K. T. Khaw, “More acidic dietary acid-base load is associated with reduced cal- caneal broadband ultrasound attenuation in women but not in men: results from the EPIC-Norfolk cohort study,” American Journal of Clinical Nutrition, vol. 85, no. 4, pp. 1134–1141, 2007. [35] C. Pedone, N. Napoli, P. Pozzilli et al., “Quality of diet and potential renal acid load as risk factors for reduced bone density in elderly women,” Bone, vol. 46, no. 4, pp. 1063–1067, 2010. [36] T. R. Fenton, A. W. Lyon, M. Eliasziw, S. C. Tough, and D. A. Hanley, “Phosphate decreases urine calcium and increases cal- cium balance: a meta-analysis of the osteoporosis acid-ash diet hypothesis,” Nutrition Journal, vol. 8, article 41, 2009. 7 [37] T. R. Fenton, M. Eliasziw, S. C. Tough, A. W. Lyon, J. P. Brown, and D. A. Hanley, “Low urine pH and acid excretion do not predict bone fractures or the loss of bone mineral density: a prospective cohort study,” BMC Musculoskeletal Disorders, vol. 11, article 88, 2010. [38] R. P. Heaney and D. K. Layman, “Amount and type of protein inﬂuences bone health,” American Journal of Clinical Nutri- tion, vol. 87, no. 5, pp. 1567S–1570S, 2008. [39] R. F. Farquharson, W. T. Salter, D. M. Tibbetts, and J. C. Aub, “Studies of calcium and phosphorus metabolism. XII: the eﬀect of the ingestion of acid producing substances,” The Journal of Clinical Investigation, vol. 10, no. 2, pp. 221–249, 1931. [40] L. Langsetmo, S. Poliquin, D. A. Hanley et al., “Dietary pat- terns in Canadian men and women ages 25 and older: rela- tionship to demographics, body mass index, and bone mineral density,” BMC Musculoskeletal Disorders, vol. 11, article 20, 2010. [41] The Norwegian Directorate of Health, Development of Norwe- gian Nutrition, 2005. [42] F. Garcia-Contreras, R. Paniagua, M. Avila-D´ıaz et al., “Cola beverage consumption induces bone mineralization reduction in ovariectomized rats,” Archives of Medical Research, vol. 31, no. 4, pp. 360–365, 2000. [43] R. Ogur, B. Uysal, T. Ogur et al., “Evaluation of the eﬀect of cola drinks on bone mineral density and associated factors,” Basic and Clinical Pharmacology and Toxicology, vol. 100, no. 5, pp. 334–338, 2007. [44] N. S. Larson, R. Amin, C. Olsen, and M. A. Poth, “Eﬀect of diet and cola on urine calcium excretion,” in Proceedings of the The Endocrine Society’s 92nd Meeting & Expo, pp. 2–198, 2010. [45] The Norwegian Directorate of Health, “Most people may replace sugar-sweetened soft drinks with sugar-free products,” Oslo, Norway, 2007, http://www.helsedirektoratet.no/portal/ page? pageid=134,67714& dad=portal& schema=PORTAL& piref134 67727 134 67714 67714.artSectionId=252& piref 134 67727 134 67714 67714.articleId=71477.