Table 2: Second admission inpatient hospital course. Day 0 Fever/rash/malaise 14 8.9 350 115 1.4 Day 1 Fever 108 3.3 Albumin 2.5 Bld cx-neg∗2 Bld cx-neg ASA and Treatment ASA∗1 Prednisone/ASA Inﬂiximab/ASA ASA None prednisone tapering ∗1 aspirin/∗2blood culture negative/∗3discharge/∗4asymptomatic/∗5followup after 10 days from discharge/∗6followup after 10 months from discharge/∗7right coronary artery/∗8left main coronary artery. Table 3: Classic Kawasaki disease clinical diagnostic criteria. What did the case patient have? No (patient presented with fever for ≥5 with only 3 of the following) Case Reports in Medicine Symptoms/signs WBC count, ×103 cells/µL Hemoglobin, g/dL Platelet count, ×103 platelets/µL ESR, mm/h CRP, mg/dL Mycoplasma IgM Other Echo Clinical criteria Fever for ≥5 days plus 4 of the following must be present to make a deﬁnitive diagnosis: Polymorphous rash Bilateral conjunctival injection At least one of the following: (i) Erythema or ﬁssuring of the lips (ii) Strawberry tongue (iii) Diﬀuse injection of oral and pharyngeal mucosa Acute, nonpurulent cervical lymphadenopathy (at least one node ≥1.5 cm) At least one of the following: (i) Erythema of palms and soles (ii) Indurative edema of hands and feet (iii) Membranous desquamation from ﬁngertips classic KD [6]. MP has been reported to cause cutaneous vasculitis by immune-complex-mediated mechanisms [7]. Another species of the Mycoplasma genome, Mycoplasma ar- thritidis, has been shown to produce a superantigen sug- gesting the possibility that other Mycoplasma species may do likewise [8]. The patient had serologic evidence of acute MP infection. To the best knowledge of the writers, this is the ﬁrst case report of MP infection, or at least evidence of acute infection, followed by incomplete KD. Diagnosing MP is a diﬃcult task, especially in the face of Kawasaki disease. It is hampered by lack of standardized, rapid, and speciﬁc methods. Laboratory tests for MP include culture, serology, and PCR. There is a high ratio of asymp- tomatic respiratory carriers, and because of continued car- riage of the organism after infection and treatment, PCR and cultures from respiratory tract specimens carry the risk of de- Day 3 Fever Day 4 Afebrile/DC∗3 124 1.7 Yes Yes Yes No No F/U∗5 Asx∗4 5.2 12.5 253 31 <0.5 Improved RCA∗7 diameter, but more dilated LMCA∗8 3 F/U∗6 Asx Negative Normal tecting healthy carriers. In recent years, the diagnosis of MP has relied mostly on serological methods. Given the simplicity and benign treatment for MP infec- tion, many clinicians use single-point Mycoplasma IgM se- rology testing to determine treatment instead of the “gold standard” paired sera conversion test. IgM antibodies are usually produced acutely, and raised IgM levels indicate acute infection. However, it was observed that elevated levels of Mycoplasma IgM might persist for several years raising con- cern for false-positive results [9]. When only one is positive, antibiotics are generally given as treatment, however, a sec- ond sample is ideally required to conﬁrm. In this patient, repeat anti-Mycoplasma level IgM test ten months after in- itial diagnosis was negative favoring a true-positive initial test. MP infection can present similarly to KD with prolonged fever, rash, conjunctivitis, and lymphadenopathy, and certain