Sturge-Weber Syndrome Fig. 2. Skin biopsy from one of the angiomatous nodules showed a cavernous hemangioma pattern. Dilated and ectatic thin-walled vessels in the superficial dermis were observed (panel A). Immunohistochemistry results (immunoperoxidase method) showed CD34(＋), CD31(＋), and Ki67(−) (panels B, C, and D). this syndrome and high awareness of this entity may lead to earlier diagnosis and more timely and appropriate med- ical interventions. REFERENCES 1. Comi AM. Update on Sturge-Weber syndrome: diagnosis, treatment, quantitative measures, and controversies. Lym- phat Res Biol 2007;5:257-264. 2. Hennedige AA, Quaba AA, Al-Nakib K. Sturge-Weber syn- drome and dermatomal facial port-wine stains: incidence, association with glaucoma, and pulsed tunable dye laser treatment effectiveness. Plast Reconstr Surg 2008;121:1173- 1180. 3. Baselga E. Sturge-Weber syndrome. Semin Cutan Med Surg 2004;23:87-98. 4. Pascual-Castroviejo I, Pascual-Pascual SI, Velazquez-Fragua R, Viaño J. Sturge-Weber syndrome: study of 55 patients. Can J Neurol Sci 2008;35:301-307. 5. Rodofile C, Grees SA, Valle LE, Martino G. Sturge-Weber syndrome. Report of a case with poor dermatological mani- festations. Arch Argent Pediatr 2011;109:e42-e45. 6. Bourgeois M, Crimmins DW, de Oliveira RS, Arzimanoglou A, Garnett M, Roujeau T, et al. Surgical treatment of epilepsy in Sturge-Weber syndrome in children. J Neurosurg 2007;106(1 Suppl):20-28. 7. Parsa CF. Sturge-weber syndrome: a unified pathophy- siologic mechanism. Curr Treat Options Neurol 2008;10: 47-54. Vol. 23, No. 4, 2011 553