Journal of Aging Research [102] M. R. Brown, J. W. Geddes, and P. G. Sullivan, “Brain region- speciﬁc, age-related, alterations in mitochondrial responses to elevated calcium,” Journal of Bioenergetics and Biomem- branes, vol. 36, no. 4, pp. 401–406, 2004. [103] R. Gredilla, L. Weissman, J.-L. Yang, V. A. Bohr, and T. Stevnsner, “Mitochondrial base excision repair in mouse synaptosomes during normal aging and in a model of Alzheimer’s disease,” Neurobiology of Aging. In press. [104] B. Szczesny, A. W. Tann, and S. Mitra, “Age- and tissue- speciﬁc changes in mitochondrial and nuclear DNA base excision repair activity in mice: susceptibility of skeletal muscles to oxidative injury,” Mechanisms of Ageing and Development, vol. 131, no. 5, pp. 330–337, 2010. [105] C. Borghouts, E. Kimpel, and H. D. Osiewacz, “Mitochon- drial DNA rearrangements of Podospora anserina are under the control of the nuclear gene grisea,” Proceedings of the National Academy of Sciences of the United States of America, vol. 94, no. 20, pp. 10768–10773, 1997. 9