BMC Cancer 2009, 9:398 http://www.biomedcentral.com/1471-2407/9/398 bPFleiogaorusdroecne'5sllsc otrreraetlaetdiofnoarl 2anhalaynsdis PbHetAw-esteinmtuhlaetepderbcleonotdocfeDllsNtAre-AatPedsitfoesr a2s4ehviwdeitnhcedtobpyoEsixdoe III and ALS index in non-stimulated Pearson's correlational analysis between the percent of DNA-AP sites as evidenced by Exo III and ALS index in non-stimulated blood cells treated for 2 h and PHA-stimulated blood cells treated for 24 h with etoposide. FDiNguAreda6mage indhiubciteidonbypreotodupcoesdideby(0A, A2.0in7,n2o0n.-7stoimru2l0a7teμdMb)loiondnocenl-lsttimreualtaetdedfobrlo2ohd wceitllhs etrtoeaptoesdidfeor(s2o-lhid(obparesn) bars) and DNA damage induced by etoposide (0, 2.07, 20.7 or 207 μM) in non-stimulated blood cells treated for 2-h (open bars) and DNA damage inhibition produced by AA in non-stimulated blood cells treated for 2 h with etoposide (solid bars). Every bar represents the mean value of three independent experiments. Data were analyzed using the Mann- Whitney U test. (*= p < 0.05; **= p < 0.005 vs. control; ***= p < 0.001 vs. corresponding no AA condition). Page 7 of 9 (page number not for citation purposes)