Federal Register / Vol. 66, No. 31 / Wednesday, February 14, 2001 / Notices 10289

data to further refine the risk

assessment, such as percent crop treated

information or submission of residue

data from food processing studies, or

could address the Agency’s risk

assessment methodologies and

assumptions as applied to this specific

chemical. Comments should be limited

to issues raised within the preliminary

risk assessment and associated

documents. EPA will provide other

opportunities for public comment on

other science issues associated with the

pesticide tolerance reassessment

program. Failure to comment on any

such issues as part of this opportunity

will in no way prejudice or limit a

commenter’s opportunity to participate

fully in later notice and comment

processes. All comments should be

submitted by April 16, 2001 using the

methods in Unit I. of the

SUPPLEMENTARY INFORMATION. Comments

will become part of the Agency record

for atrazine.

List of Subjects

Environmental protection, Chemicals,

Pesticides and pests.

Dated: February 8, 2001.

Jack E. Housenger,

Acting Director, Special Review and

Reregistration Division, Office of Pesticide

Programs.

[FR Doc. 01–3844 Filed 2–12–01; 2:53 pm]

BILLING CODE 6560–50–S

ENVIRONMENTAL PROTECTION

AGENCY

[PF–998; FRL–6768–7]

Notice of Filing a Pesticide Petition to

Establish a Tolerance for a Certain

Pesticide Chemical in or on Food

AGENCY: Environmental Protection

Agency (EPA).

ACTION: Notice.

SUMMARY: This notice announces the

initial filing of a pesticide petition

proposing the establishment of

regulations for residues of a certain

pesticide chemical in or on various food

commodities.

DATES: Comments, identified by docket

control number PF–998, must be

received on or before March 16, 2001.

ADDRESSES: Comments may be

submitted by mail, electronically, or in

person. Please follow the detailed

instructions for each method as

provided in Unit I.C. of the

SUPPLEMENTARY INFORMATION. To ensure

proper receipt by EPA, it is imperative

that you identify docket control number

PF–998 in the subject line on the first

page of your response.

FOR FURTHER INFORMATION CONTACT: By

mail: George T. LaRocca, Registration

Division (7505C), Office of Pesticide

Programs, Environmental Protection

Agency, 1200 Pennsylvania Ave., NW.,

Washington, DC 20460; telephone

number: (703) 305–6100; e-mail address:

larocca.george@epa.gov.

SUPPLEMENTARY INFORMATION:

I. General Information

A. Does this Action Apply to Me?

You may be affected by this action if

you are an agricultural producer, food

manufacturer or pesticide manufacturer.

Potentially affected categories and

entities may include, but are not limited

to:

Categories NAICS

codes

Examples of potentially affected

entities

Industry 111 Crop production

112 Animal production

311 Food manufacturing

32532 Pesticide manufacturing

This listing is not intended to be

exhaustive, but rather provides a guide

for readers regarding entities likely to be

affected by this action. Other types of

entities not listed in the table could also

be affected. The North American

Industrial Classification System

(NAICS) codes have been provided to

assist you and others in determining

whether or not this action might apply

to certain entities. If you have questions

regarding the applicability of this action

to a particular entity, consult the person

listed under FOR FURTHER INFORMATION

CONTACT.

B. How Can I Get Additional

Information, Including Copies of this

Document and Other Related

Documents?

1. Electronically. You may obtain

electronic copies of this document, and

certain other related documents that

might be available electronically, from

the EPA Internet Home Page at http://

www.epa.gov/. To access this

document, on the Home Page select

‘‘Laws and Regulations’’ and then look

up the entry for this document under

the ‘‘Federal Register—Environmental

Documents.’’ You can also go directly to

the Federal Register listings at http://

www.epa.gov/fedrgstr/.

2. In person. The Agency has

established an official record for this

action under docket control number PF–

998. The official record consists of the

documents specifically referenced in

this action, any public comments

received during an applicable comment

period, and other information related to

this action, including any information

claimed as confidential business

information (CBI). This official record

includes the documents that are

physically located in the docket, as well

as the documents that are referenced in

those documents. The public version of

the official record does not include any

information claimed as CBI. The public

version of the official record, which

includes printed, paper versions of any

electronic comments submitted during

an applicable comment period, is

available for inspection in the Public

Information and Records Integrity

Branch (PIRIB), Rm. 119, Crystal Mall

#2, 1921 Jefferson Davis Highway,

Arlington, VA, from 8:30 a.m. to 4 p.m.,

Monday through Friday, excluding legal

holidays. The PIRIB telephone number

is (703) 305–5805.

C. How and to Whom Do I Submit

Comments?

You may submit comments through

the mail, in person, or electronically. To

ensure proper receipt by EPA, it is

imperative that you identify docket

control number PF–998 in the subject

line on the first page of your response.

1. By mail. Submit your comments to:

Public Information and Records

Integrity Branch (PIRIB), Information

Resources and Services Division

(7502C), Office of Pesticide Programs

(OPP), Environmental Protection

Agency, 1200 Pennsylvania Ave., NW.,

Washington, DC 20460.

2. In person or by courier. Deliver

your comments to: Public Information

and Records Integrity Branch (PIRIB),

Information Resources and Services

Division (7502C), Office of Pesticide

Programs (OPP), Environmental

Protection Agency, Rm. 119, Crystal

Mall #2, 1921 Jefferson Davis Highway,

Arlington, VA. The PIRIB is open from

8:30 a.m. to 4 p.m., Monday through

Friday, excluding legal holidays. The

PIRIB telephone number is (703) 305–

5805.

3. Electronically. You may submit

your comments electronically by e-mail

to: opp-docket@epa.gov, or you can

submit a computer disk as described

above. Do not submit any information

electronically that you consider to be

CBI. Avoid the use of special characters

and any form of encryption. Electronic

submissions will be accepted in

Wordperfect 6.1/8.0 or ASCII file

format. All comments in electronic form

must be identified by docket control

number PF–998. Electronic comments

10290 Federal Register / Vol. 66, No. 31 / Wednesday, February 14, 2001 / Notices

may also be filed online at many Federal

Depository Libraries.

D. How Should I Handle CBI That I

Want to Submit to the Agency?

Do not submit any information

electronically that you consider to be

CBI. You may claim information that

you submit to EPA in response to this

document as CBI by marking any part or

all of that information as CBI.

Information so marked will not be

disclosed except in accordance with

procedures set forth in 40 CFR part 2.

In addition to one complete version of

the comment that includes any

information claimed as CBI, a copy of

the comment that does not contain the

information claimed as CBI must be

submitted for inclusion in the public

version of the official record.

Information not marked confidential

will be included in the public version

of the official record without prior

notice. If you have any questions about

CBI or the procedures for claiming CBI,

please consult the person identified

under FOR FURTHER INFORMATION

CONTACT.

E. What Should I Consider as I Prepare

My Comments for EPA?

You may find the following

suggestions helpful for preparing your

comments:

1. Explain your views as clearly as

possible.

2. Describe any assumptions that you

used.

3. Provide copies of any technical

information and/or data you used that

support your views.

4. If you estimate potential burden or

costs, explain how you arrived at the

estimate that you provide.

5. Provide specific examples to

illustrate your concerns.

6. Make sure to submit your

comments by the deadline in this

notice.

7. To ensure proper receipt by EPA,

be sure to identify the docket control

number assigned to this action in the

subject line on the first page of your

response. You may also provide the

name, date, and Federal Register

citation.

II. What Action is the Agency Taking?

EPA has received a pesticide petition

as follows proposing the establishment

and/or amendment of regulations for

residues of a certain pesticide chemical

in or on various food commodities

under section 408 of the Federal Food,

Drug, and Cosmetic Act (FFDCA), 21

U.S.C. 346a. EPA has determined that

this petition contains data or

information regarding the elements set

forth in section 408(d)(2); however, EPA

has not fully evaluated the sufficiency

of the submitted data at this time or

whether the data support granting of the

petition. Additional data may be needed

before EPA rules on the petition.

List of Subjects

Environmental protection,

Agricultural commodities, Feed

additives, Food additives, Pesticides

and pests, Reporting and recordkeeping

requirements.

Dated: January 31, 2001.

James Jones,

Director, Registration Division, Office of

Pesticide Programs.

Summary of Petition

The petitioner summary of the

pesticide petition is printed below as

required by section 408(d)(3) of the

FFDCA. The summary of the petition

was prepared by the petitioner and

represents the view of the petitioner.

EPA is publishing the petition summary

verbatim without editing it in any way.

The petition summary announces the

availability of a description of the

analytical methods available to EPA for

the detection and measurement of the

pesticide chemical residues or an

explanation of why no such method is

needed.

FMC Corporation

0E6216

EPA has received a pesticide petition

(0E6216) from FMC Corporation, 1735

Market Street, Philadelphia, PA

proposing, pursuant to section 408(d) of

the (FFDCA), 21 U.S.C. 346a(d), to

amend 40 CFR part 180 by establishing

a tolerance for residues of bifenthrin ((2-

methyl [1,1’-biphenyl]-3-yl) methyl-3-(2-

chloro-3,3,3,-trifluoro-1-propenyl)-2,2-

dimethylcyclopropanecarboxylate) in or

on the raw agricultural commodity

(RAC) bananas at 0.1 parts per million

(ppm). EPA has determined that the

petition contains data or information

regarding the elements set forth in

section 408(d)(2) of the FFDCA;

however, EPA has not fully evaluated

the sufficiency of the submitted data at

this time or whether the data support

granting of the petition. Additional data

may be needed before EPA rules on the

petition.

A. Residue Chemistry

1. Plant metabolism. The metabolism

of bifenthrin in plants is adequately

understood. Studies have been

conducted to delineate the metabolism

of radiolabelled bifenthrin in various

crops all showing similar results. The

residue of concern is the parent

compound only.

2. Analytical method. There is a

practical analytical method for detecting

and measuring levels of bifenthrin in or

on food with a limit of detection that

allows monitoring of food with residues

at or above the levels set in these

tolerances gas chromatography with

electron capture detection (GC/ECD)

analytical method P–2132M, (PP)

0E3921, MRID 41658601.

3. Magnitude of residues. Field

residue trials meeting EPA study

requirements have been conducted at

the maximum label rate for the crop

bananas. Results from these trials

demonstrate that the highest bifenthrin

residues found will not exceed 0.1

(ppm) when the product is applied

following the proposed use directions.

B. Toxicological Profile

1. Acute toxicity. For the purposes of

assessing acute dietary risk, FMC

Corporation has used the maternal no

observed adverse effect level (NOAEL)

of 1.0 milligram/kilogram (mg/kg)/day

from the oral developmental toxicity

study in rats. The maternal lowest effect

level (LEL) of this study of 2.0 mg/kg/

day was based on tremors from day 7–

17 of dosing. This acute dietary

endpoint is used to determine acute

dietary risks to all population

subgroups.

2. Genotoxicity. The following

genotoxicity tests were all negative.

Gene mutation in Salmonella (Ames);

chromosomal aberrations in Chinese

hamster ovary and rat bone marrow

cells; hypoxanthine guanine

phophoribosyl transferase (HGPRT)

locus mutation in mouse lymphoma

cells; and unscheduled DNA synthesis

in rat hepatocytes.

3. Reproductive and developmental

toxicity. i. In the rat reproduction study,

parental toxicity occurred as decreased

body weight (bwt) at 5.0 mg/kg/day with

a NOAEL of 3.0 mg/kg/day. There were

no developmental (pup) or reproductive

effects up to 5.0 mg/kg/day (highest

dose tested (HDT)).

ii. Based on the absence of pup

toxicity up to dose levels which

produced toxicity in the parental

animals, there is no evidence of special

postnatal sensitivity to infants and

children in the rat reproduction study.

4. Subchronic toxicity—Short- and

intermediate-term toxicity. The maternal

NOAEL of 1.0 mg/kg/day from the oral

developmental toxicity study in rats is

also used for short- and intermediateterm margin of exposure (MOE)

calculations (as well as acute, discussed

in (1) above). The maternal LEL of this

Federal Register / Vol. 66, No. 31 / Wednesday, February 14, 2001 / Notices 10291

study of 2.0 mg/kg/day was based on

tremors from day 7–17 of dosing.

5. Chronic toxicity. i. The reference

dose (RfD) has been established at 0.015

mg/kg/day. This RfD is based on a 1–

year oral feeding study in dogs with a

NOAEL of 1.5 mg/kg/day, based on

intermittent tremors observed at the

LOAEL of 3.0 mg/kg/day; an uncertainty

factor (UF) of 100 is used.

ii. Bifenthrin is classified as a Group

C chemical (possible human carcinogen)

based upon urinary bladder tumors in

mice; assignment of a Q* has not been

recommended.

6. Animal metabolism. The

metabolism of bifenthrin in animals is

adequately understood. Metabolism

studies in rats with single doses

demonstrated that about 90% of the

parent compound and its hydroxylated

metabolites are excreted.

7. Metabolite toxicology. The Agency

has previously determined that the

metabolites of bifenthrin are not of

toxicological concern and need not be

included in the tolerance expression.

8. Endocrine disruption. No special

studies investigating potential

estrogenic or other endocrine effects of

bifenthrin have been conducted.

However, no evidence of such effects

was reported in the standard battery of

required toxicology studies, which have

been completed and found acceptable.

Based on these studies, there is no

evidence to suggest that bifenthrin has

an adverse effect on the endocrine

system.

C. Aggregate Exposure

1. Dietary exposure—i. Food.

Tolerances have been established for the

residues of bifenthrin, in or on a variety

of RACs. Tolerances, in support of

registrations, currently exist for residues

of bifenthrin on hops, strawberries, corn

grain, forage, and fodder, sweet corn,

cottonseed, artichokes, the crop group

cucurbit vegetables, the crop group

legume vegetables - subgroup ediblepodded legume vegetables, and

subgroup succulent shelled pea,

eggplant, the subgroup head and stem

brassica, and livestock commodities of

cattle, goats, hogs, horses, sheep,

poultry, eggs, and milk. Pending

tolerances for citrus, bananas, grapes,

peanuts, pears, potatoes, caneberries,

peppers (bell and non-bell), lettuce

(head), and herbs also exist. For the

purposes of assessing the potential

dietary exposure for these existing and

pending tolerances FMC Corporation

has utilized available information on

anticipated residues, monitoring data

and percent crop treated as follows:

ii. Acute exposure and risk. Acute

dietary exposure risk assessments are

performed for a food-use pesticide if a

toxicological study has indicated the

possibility of an effect of concern

occurring as a result of a one day or

single exposure. For the purposes of

assessing acute dietary risk for

bifenthrin, the maternal NOAEL of 1.0

mg/kg/day from the oral developmental

toxicity study in rats was used. The

maternal LEL of this study of 2.0 mg/kg/

day was based on tremors from day 7–

17 of dosing. This acute dietary

endpoint was used to determine acute

dietary risks to all population

subgroups. Available information on

anticipated residues, monitoring data

and percent crop treated was

incorporated into a Tier 3 analysis;

using Monte Carlo modeling for

commodities that may be consumed in

a single serving. These assessments

show that the MOEs are greater than the

EPA standard of 100 for all

subpopulations. The 99.9th percentile of

exposure for the overall U. S.

population was estimated to be

0.005506 mg/kg/day (MOE of 181). The

99.9th percentile of exposure for all

infants <1–year old was estimated to be

0.005825 mg/kg/day (MOE of 171). The

99.9th percentile of exposure for nursing

infants <1–year old was estimated to be

0.004056 mg/kg/day (MOE of 246). The

99.9th percentile of exposure for nonnursing infants <1–year old was

estimated to be 0.005910 mg/kg/day

(MOE of 169). The 99.9th percentile of

exposure for children 1 to 6 years old

(the most highly exposed population

subgroup) was estimated to be 0.009741

mg/kg/day (MOE of 102). Therefore,

FMC Corporation concludes that the

acute dietary risk of bifenthrin, as

estimated by the dietary risk

assessment, does not appear to be of

concern.

iii. Chronic exposure and risk. The

acceptable reference dose (RfD) is based

on a NOAEL of 1.5 mg/kg/day from the

chronic dog study and an UF of 100 is

0.015 mg/kg/day. The endpoint effect of

concern was tremors in both sexes of

dogs at the LEL of 3.0 mg/kg/day. A

chronic dietary exposure/risk

assessment has been performed for

bifenthrin using the above RfD. The

chronic exposures are estimated to be

0.000186 mg/kg bwt/day and utilize

1.2% of the RfD for the overall U. S.

population; children 7-12 years old and

children 1-6 years old (subgroups most

highly exposed) are estimated to be

0.000229 mg/kg bwt/day and 0.000371

mg/kg bwt/day and utilizes 1.5% and

2.5% of the RfD, respectively. Generally

speaking, the EPA has no cause for

concern if the total dietary exposure

from residues for uses for which there

are published and proposed tolerances

is less than 100% of the RfD. Therefore,

FMC Corporation concludes that the

chronic dietary risk of bifenthrin, as

estimated by the dietary risk

assessment, does not appear to be of

concern.

iv. Drinking water. Laboratory and

field data have demonstrated that

bifenthrin is immobile in soil and will

not leach into groundwater. Other data

show that bifenthrin is virtually

insoluble in water and extremely

lipophilic. As a result, FMC Corporation

concludes that residues reaching surface

waters from field runoff will quickly

adsorb to sediment particles and be

partitioned from the water column.

Further, a screening evaluation of

leaching potential of a typical

pyrethroid was conducted using EPA’s

pesticide root zone model (PRZM3).

Based on this screening assessment, the

potential concentrations of a pyrethroid

in groundwater at depths of 1 and 2

meters are essentially zero (<<0.001

parts per billion (ppb)). Surface water

concentrations for pyrethroids were

estimated using PRZM3 and exposure

analysis modeling system (EXAMS)

using standard EPA cotton runoff and

Mississippi pond scenarios. The

maximum concentration predicted in

the simulated pond was 0.052 ppb.

Concentrations in actual drinking water

would be much lower than the levels

predicted in the hypothetical, small,

stagnant farm pond model since

drinking water derived from surface

water would normally be treated before

consumption. Based on these analysis,

the contribution of water to the dietary

risk estimate is negligible. Therefore,

FMC Corporation concludes that

together these data indicate that

residues are not expected to occur in

drinking water.

2. Non-dietary exposure. Analysis

were conducted which included an

evaluation of potential non-dietary

(residential) applicator, post-application

and chronic dietary aggregate exposures

associated with bifenthrin products

used for residential flea infestation

control and agricultural/commercial

applications. The aggregate analysis

conservatively assumes that a person is

concurrently exposed to the same active

ingredient via the use of consumer or

professional flea infestation control

products and to chronic level residues

in the diet.

In the case of potential non-dietary

health risks, conservative point

estimates of non-dietary exposures,

expressed as total systemic absorbed

dose (summed across inhalation and

incidental ingestion routes) for each

relevant product use category (i.e., lawn

10292 Federal Register / Vol. 66, No. 31 / Wednesday, February 14, 2001 / Notices

care) and receptor subpopulation (i.e.,

adults, children 1-6 years and infants

<1–year) are compared to the systemic

absorbed dose NOAEL for bifenthrin to

provide estimates of the MOEs. Based

on the toxicity endpoints selected by

EPA for bifenthrin, inhalation and

incidental oral ingestion absorbed doses

were combined and compared to the

relevant systemic NOAEL for estimating

MOEs.

In the case of potential aggregate

health risks, the above mentioned

conservative point estimates of

inhalation and incidental ingestion nondietary exposure (expressed as systemic

absorbed dose) are combined with

estimates (arithmetic mean values) of

chronic average dietary (oral) absorbed

doses. These aggregate absorbed dose

estimates are also provided for adults,

children 1-6 years and infants <1–year.

The combined or aggregated absorbed

dose estimates (summed across nondietary and chronic dietary) are then

compared with the systemic absorbed

dose NOAEL to provide estimates of

aggregate MOEs.

The non-dietary and aggregate (nondietary + chronic dietary) MOEs for

bifenthrin indicate a substantial degree

of safety. The total non-dietary

(inhalation + incidental ingestion)

MOEs for post-application exposure for

the lawn care product evaluated was

estimated to be 194,000 for adults,

52,400 for children 1-6 years old and

56,700 for infants <1–year. The

aggregate MOE (inhalation + incidental

oral + chronic dietary, summed across

all product use categories) was

estimated to be 4,878 for adults, 1,117

for children 1-6 years old and 1,361 for

infants (<1–year). It can be concluded

that the potential non-dietary and

aggregate (non-dietary + chronic dietary)

exposures for bifenthrin are associated

with substantial margins of safety.

D. Cumulative Effects

In consideration of potential

cumulative effects of bifenthrin and

other substances that may have a

common mechanism of toxicity, to our

knowledge there are currently no

available data or other reliable

information indicating that any toxic

effects produced by bifenthrin would be

cumulative with those of other chemical

compounds; thus only the potential

risks of bifenthrin have been considered

in this assessment of its aggregate

exposure. FMC Corporation intends to

submit information for the EPA to

consider concerning potential

cumulative effects of bifenthrin

consistent with the schedule established

by EPA in the Federal Register at 62 FR

42020 (August 4, 1997), FRL–5734–6

and other EPA publications pursuant to

the Food Quality Protection Act.

E. Safety Determination

1. U.S. population. For the overall

U.S. population, the calculated MOE at

the 95th percentile was estimated to be

650, 359 at the 99th percentile; and 181

at the 99.9th percentile. For all infants

<1–year old, the calculated MOE at the

95th percentile was estimated to be 540;

241 at the 99th percentile; and 171 at the

99.9th percentile. For nursing infants

<1–year old, the calculated MOE at the

95th percentile was estimated to be

1,311; 451 at the 99th percentile; and 246

at the 99.9th percentile. For non-nursing

infants <1–year old, the calculated

margins of exposure MOE at the 95th

percentile was estimated to be 476, 197

at the 99th percentile; and 169 at the

99.9th percentile. For the most highly

exposed population subgroup, children

1-6 years old, the calculated MOE at the

95th percentile was estimated to be 330,

214 at the 99th percentile; and 102 at the

99.9th percentile. Therefore, FMC

Corporation concludes that there is

reasonable certainty that no harm will

result from acute exposure to bifenthrin.

2. Infants and children—a. General. In

assessing the potential for additional

sensitivity of infants and children to

residues of bifenthrin, FMC Corporation

considered data from developmental

toxicity studies in the rat and rabbit,

and a 2–generation reproductive study

in the rat. The developmental toxicity

studies are designed to evaluate adverse

effects on the developing organism

resulting from pesticide exposure

during prenatal development to one or

both parents. Reproduction studies

provide information relating to effects

from exposure to the pesticide on the

reproductive capability of mating

animals and data on systemic toxicity.

FFDCA section 408 provides that EPA

may apply an additional margin of

safety for infants and children in the

case of threshold effects to account for

prenatal and postnatal toxicity and the

completeness of the data base.

b. Developmental toxicity studies. In

the rabbit developmental study, there

were no developmental effects observed

in the fetuses exposed to bifenthrin. The

maternal NOAEL was 2.67 mg/kg/day

based on head and forelimb twitching at

the LOAEL of 4 mg/kg/day. In the rat

developmental study, the maternal

NOAEL was 1 mg/kg/day, based on

tremors at the LOAEL of 2 mg/kg/day.

The developmental (pup) NOAEL was

also 1 mg/kg/day, based upon increased

incidence of hydroureter at the LOAEL

2 mg/kg/day. There was 5/23 (22%)

litters affected (5/141 fetuses since each

litter only had one affected fetus) in the

2 mg/kg/day group, compared with zero

in the control, 1, and 0.5 mg/kg/day

groups. According to recent historical

data (1992–1994) for this strain of rat,

incidence of distended ureter averaged

11% with a maximum incidence of

90%.

c. Reproductive toxicity study. In the

rat reproduction study, parental toxicity

occurred as decreased bwt at 5.0 mg/kg/

day with a NOAEL of 3.0 mg/kg/day.

There were no developmental (pup) or

reproductive effects up to 5.0 mg/kg/day

HDT.

d. Prenatal and postnatal sensitivity—

i. Prenatal. Since there was not a doserelated finding of hydroureter in the rat

developmental study and in the

presence of similar incidences in the

recent historical control data, the

marginal finding of hydroureter in rat

fetuses at 2 mg/kg/day (in the presence

of maternal toxicity) is not considered a

significant developmental finding. Nor

does it provide sufficient evidence of a

special dietary risk (either acute or

chronic) for infants and children which

would require an additional safety

factor. Based on the absence of pup

toxicity up to dose levels, which

produced toxicity in the parental

animals, there is no evidence of special

postnatal sensitivity to infants and

children in the rat reproduction study.

e. Conclusion. Based on the above,

FMC Corporation concludes that

reliable data support use of the standard

100–fold UF, and that an additional UF

is not needed to protect the safety of

infants and children. As stated above,

aggregate exposure assessments utilized

less than 10% of the RfD for either the

entire U. S. population or any of the 26

population subgroups including infants

and children. Therefore, it may be

concluded that there is reasonable

certainty that no harm will result to

infants and children from aggregate

exposure to bifenthrin residues.

F. International Tolerances

There are no Codex, Canadian, or

Mexican residue limits for residues of

bifenthrin in or on bananas.

[FR Doc. 01–3621 Filed 2–13–01; 8:45 am]

BILLING CODE 6560–50–S

ENVIRONMENTAL PROTECTION

AGENCY

[PF–996; FRL–6765–8]

Notice of Filing a Pesticide Petition to

Establish a Tolerance for a Certain

Pesticide Chemical in or on Food

AGENCY: Environmental Protection

Agency (EPA).