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          About NIAMS
          Fatemeh (Elham) Navid, Ph.D.





Fatemeh (Elham) Navid, Ph.D.


Staff Scientist
    Pediatric Translational Research Branch Image
              Headshot of a woman with tied back hair. 
            Contact Information

                                                  301-451-1207
                
                                                9000 Rockville Pike
                  
            10 Center Drive
      
                  
            Building 10, Room 12S253
      
                                   Bethesda MD 20892
                
                                                navidf2@mail.nih.gov 
                                          Media Resources
                                High Resolution Image
                headshot-elham-navid.jpg 
                Summary
          Dr. Fatemeh Navid received her doctoral degree at the University of Hamburg in Germany. She has received numerous awards, including the Phora Photo Dermatology Award (2009), the European Society of Dermatological Research Travel Grant (2010), and the Spartan Fellow Award (2015).
        
                  Research Statement
          
            We are studying endoplasmic reticulum (ER) quality control pathways that regulate the degradation of misfolded forms of HLA-B27, including autophagy and ERAD, and the role of ER stress in the pathogenesis of experimental spondyloarthritis. 

            Scientific Publications
              Causes and consequences of endoplasmic reticulum stress in rheumatic disease.

    Navid F, Colbert RA
        

                    Nat Rev Rheumatol.
        
                    2017 Jan;
        
                    13(1).
        
                    doi: 10.1038/nrrheum.2016.192
                            PMID: 27904144
  Generation and differentiation of induced pluripotent stem cells reveal ankylosing spondylitis risk gene expression in bone progenitors.

    Layh-Schmitt G, Lu S, Navid F, Brooks SR, Lazowick E, Davis KM, Montagna C, Gadina M, Colbert RA
        

                    Clin Rheumatol.
        
                    2017 Jan;
        
                    36(1).
        
                    doi: 10.1007/s10067-016-3469-5
                            PMID: 27864696
  The Aryl hydrocarbon receptor is involved in UVR-induced immunosuppression.

    Navid F, Bruhs A, Schuller W, Fritsche E, Krutmann J, Schwarz T, Schwarz A
        

                    J Invest Dermatol.
        
                    2013 Dec;
        
                    133(12).
        
                    doi: 10.1038/jid.2013.221
                            PMID: 23652795
  Induction of regulatory T cells by a murine β-defensin.

    Navid F, Boniotto M, Walker C, Ahrens K, Proksch E, Sparwasser T, Müller W, Schwarz T, Schwarz A
        

                    J Immunol.
        
                    2012 Jan 15;
        
                    188(2).
        
                    doi: 10.4049/jimmunol.1100452
                            PMID: 22174455
  In vivo reprogramming of UV radiation-induced regulatory T-cell migration to inhibit the elicitation of contact hypersensitivity.

    Schwarz A, Navid F, Sparwasser T, Clausen BE, Schwarz T
        

                    J Allergy Clin Immunol.
        
                    2011 Oct;
        
                    128(4).
        
                    doi: 10.1016/j.jaci.2011.06.005
                            PMID: 21762977
  UV-B radiation induces the expression of antimicrobial peptides in human keratinocytes in vitro and in vivo.

    Gläser R, Navid F, Schuller W, Jantschitsch C, Harder J, Schröder JM, Schwarz A, Schwarz T
        

                    J Allergy Clin Immunol.
        
                    2009 May;
        
                    123(5).
        
                    doi: 10.1016/j.jaci.2009.01.043
                            PMID: 19342087 
          Education
              
            **University of Hamburg/Germany**Ph.D.


Last Updated: October 2023


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