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Title:   Author  item
                                            REYNA, M - TEXAS A&M-KINGSVILLEitem
                                            MARTINEZ, S - TEXAS A&M-KINGSVILLEitem
                                            WELSH, T - TAMU&TAESitem
                                            Carroll, Jeffery

                                                         - *Jeff Carroll*item
                                            LAURENZ, J - TEXAS A&M-KINGSVILLE
  Submitted to: Meeting Abstract
                                    Publication Type: Proceedings
                                    Publication Acceptance Date: 11/4/2005
                                    Publication  Date: 11/5/2005
                                    Citation: Reyna, Maribel, Martinez, Susan, Welsh, Jr., Thomas H., Carroll, J.A., Laurenz, J.C. 2005. Maternal reprogramming of the stress response in pigs. Abstract No. 13. Available: http://www.tamus.edu/pathways/documents/Symposium/abstracts/2005/agriculture.doc.Interpretive Summary:
                                Technical Abstract: Early life stress (pre and early postnatal) can predispose individuals to a variety of diseases later in life. The mechanistic basis of this relationship remains to be determined. However, evidence suggests that early life stress permanently alters the "programming" of the central nervous, neuroendocrine, and immune systems. This study used a pig model to identify components of the stress system that are altered by maternal stress and the relationship between these alterations, growth, and immune function. Results from this study confirm that maternal stress (restraint stress) during late gestation alters the progeny's hypothalamic-pituitary-adrenal (cortisol secretion) and sympatho-medullary (epinephrine and norepinephrine) responses to acute restraint stress. Furthermore, the altered stress response of the progeny is reflected by changes in the in vitro assessments of immune function.


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