31768 Federal Register / Vol. 63, No. 111 /Wednesday, June 10, 1998 / Notices

ENVIRONMENTAL PROTECTION

AGENCY

[FRL–6109–9]

Board of Scientific Counselors; Notice

of Charter Renewal

AGENCY: Environmental Protection

Agency (EPA).

ACTION: Notice of charter renewal.

SUMMARY: The Charter for the

Environmental Protection Agency’s

(EPA) Board of Scientific Counselors

(BOSC) will be renewed for an

additional two-year period. The BOSC is

deemed a necessary committee which is

in the public interest, and is in

accordance with the provisions of the

Federal Advisory Committee Act

(FACA), 5 U.S.C. appl. 2 section 9(c).

The purpose of BOSC is to counsel the

Assistant Administrator for Research

and Development (AA/ORD), on the

operation of ORD’s research program. It

is determined that BOSC is in the public

interest in connection with the

performance of duties imposed on the

Agency by law.

Inquiries may be directed to Ms.

Shirley Hamilton, Designated Federal

Officer, BOSC, U.S. EPA, Office of

Research and Development (mail code

8701R), 401 M Street, S.W., Washington,

DC 20460.

Dated: June 3, 1998.

Henry L. Longest II,

Acting Assistant Administrator for Research

and Development.

[FR Doc. 98–15445 Filed 6–9–98; 8:45 am]

BILLING CODE 6560–50–M

ENVIRONMENTAL PROTECTION

AGENCY

[OPP–00541; FRL–5796–7]

EPA-USDA Tolerance Reassessment

Advisory Committee; Notice of Public

Meetings

AGENCY: Environmental Protection

Agency (EPA).

ACTION: Notice.

SUMMARY: The EPA-USDA Tolerance

Reassessment Advisory Committee

(TRAC) has been established as a

subcommittee under the auspices of the

EPA National Advisory Council for

Environmental Policy and Technology

(NACEPT). The TRAC is in response to

Vice President Gore’s request for EPA

and the U.S. Department of Agriculture

(USDA) to work together to ensure the

smooth implementation of the Food

Quality Protection Act (FQPA).

DATES: The second set of TRAC

meetings will be held on Monday, June

22, 1998, from 1 p.m. to 5 p.m. and

Tuesday, June 23, 1998, from 9 a.m. to

5 p.m. The third set of TRAC meetings

will be held on Monday, July 13, 1998,

from 1 p.m. to 5 p.m. and Tuesday, July

14, 1998, from 9 a.m. to 5 p.m. The

dates of the final set of TRAC meetings

are July 27 and 28, 1998.

ADDRESSES: The second and third TRAC

meetings will be held at the

International Trade Center—Conference

Center, 1300 Pennsylvania Ave., NW.,

Washington, DC; telephone: (202) 312–

1300 and fax: (202) 312–1310. Specific

times and location of the final meeting

will be announced in the Federal

Register prior to that meeting. The

permanent record is available for

inspection during normal business

hours, Monday through Friday,

excluding legal holidays at the

Environmental Protection Agency,

Crystal Mall 2, Rm. 101, 1921 Jefferson

Davis Hwy., Arlington, VA, telephone:

(703) 305–5805.

FOR FURTHER INFORMATION CONTACT: By

mail: Margie Fehrenbach or Linda

Murray, Office of Pesticide Programs

(7501C), Environmental Protection

Agency, 401 M St., SW., Washington,

DC 20460. Office location, telephone

number, and e-mail address: Crystal

Mall 2, Rm. 1119, 1921 Jefferson Davis

Hwy., Arlington, VA; telephone: (703)

305–7090; e-mail:

fehrenbach.margie@epamail.epa.gov or

murray.linda@epamail.epa.gov.

SUPPLEMENTARY INFORMATION: FQPA,

Pub. L.104–170, was passed in 1996,

this new law strengthens the nation’s

system for regulating pesticides on food.

The TRAC will be asked to provide

policy guidance on sound science, ways

to increase transparency in

decisionmaking, strategies for a

reasonable transition for agriculture,

and ways to enhance consultations with

stakeholders, as pesticide tolerances are

reassessed, including those for

organophosphates.

The TRAC is co-chaired by EPA

Deputy Administrator Fred Hansen and

USDA Deputy Secretary Richard

Rominger. The TRAC is composed of

experts that include farmers,

environmentalists, public health

officials, pediatric experts, pesticide

companies, food processors and

distributors, public interest groups,

academicians, and tribal, State, and

local governments.

The TRAC meetings are open to the

public under section 10(a)(2) of the

Federal Advisory Committee Act, Pub.

L. 92–463. Outside statements by

observers are welcome. Oral statements

will be limited to 2–3 minutes, and it is

preferred that only one person per

organization present the statement. Any

person who wishes to file a written

statement may do so before or after a

TRAC meeting. These statements will

become part of the permanent record

and will be provided to the TRAC

members. The permanent record will be

available for public inspection at the

address in ‘‘Addresses’’ at the beginning

of this document.

Agendas and other background

information specific to these meetings,

as well as information from the first

meeting, will be available on the EPA

TRAC World Wide Web site (http://

www.epa.gov/pesticides/trac) 1 week

before each meeting or can be obtained

by calling (703) 305–7090.

List of Subjects

Environmental protection,

Agriculture, Chemicals, Foods,

Pesticides and pests.

Dated: June 4, 1998.

Marcia E. Mulkey,

Acting Director, Office of Pesticide Programs.

[FR Doc. 98–15444 Filed 6–9–98; 8:45 am]

BILLING CODE 6560–50––F

ENVIRONMENTAL PROTECTION

AGENCY

[PF–810; FRL–5793–1]

FMC Corporation; Pesticide Tolerance

Petition Filing

AGENCY: Environmental Protection

Agency (EPA).

ACTION: Notice.

SUMMARY: This notice announces the

initial filing of a pesticide petition

proposing the establishment of

regulations for residues of a certain

pesticide chemical in or on various food

commodities.

DATES: Comments, identified by the

docket control number PF–810, must be

received on or before July 10, 1998.

ADDRESSES: By mail submit written

comments to: Information and Records

Integrity Branch, Public Information and

Services Divison (7502C), Office of

Pesticides Programs, Environmental

Protection Agency, 401 M St., SW.,

Washington, DC 20460. In person bring

comments to: Rm. 119, CM #2, 1921

Jefferson Davis Highway, Arlington, VA.

Comments and data may also be

submitted electronically by following

the instructions under

‘‘SUPPLEMENTARY INFORMATION.’’

No confidential business information

should be submitted through e-mail.

Information submitted as a comment

concerning this document may be

claimed confidential by marking any
Federal Register / Vol. 63, No. 111 /Wednesday, June 10, 1998 / Notices 31769

part or all of that information as

‘‘Confidential Business Information’’

(CBI). CBI should not be submitted

through e-mail. Information marked as

CBI will not be disclosed except in

accordance with procedures set forth in

40 CFR part 2. A copy of the comment

that does not contain CBI must be

submitted for inclusion in the public

record. Information not marked

confidential may be disclosed publicly

by EPA without prior notice. All written

comments will be available for public

inspection in Rm. 1132 at the address

given above, from 8:30 a.m. to 4 p.m.,

Monday through Friday, excluding legal

holidays.

FOR FURTHER INFORMATION CONTACT:

Joanne Miller, Registration Support

Branch, Registration Division (7505C),

Office of Pesticide Programs,

Environmental Protection Agency, 401

M St., SW, Washington, DC 20460.

Office location, telephone number, and

e-mail address: Rm. 237, Crystal Mall

CM #2, 1900 Jefferson Davis Highway,

Arlington, VA 22202, (703) 305–6224; email: miller.joanne@epamail.epa.gov.

SUPPLEMENTARY INFORMATION: EPA has

received a pesticide petition as follows

proposing the establishment and/or

amendment of regulations for residues

of certain pesticide chemical in or on

various food commodities under section

408 of the Federal Food, Drug, and

Comestic Act (FFDCA), 21 U.S.C. 346a.

EPA has determined that this petition

contains data or information regarding

the elements set forth in section

408(d)(2); however, EPA has not fully

evaluated the sufficiency of the

submitted data at this time or whether

the data supports granting of the

petition. Additional data may be needed

before EPA rules on the petition.

The official record for this notice of

filing, as well as the public version, has

been established for this notice of filing

under docket control number [PF–810]

(including comments and data

submitted electronically as described

below). A public version of this record,

including printed, paper versions of

electronic comments, which does not

include any information claimed as CBI,

is available for inspection from 8:30

a.m. to 4 p.m., Monday through Friday,

excluding legal holidays. The official

record is located at the address in

‘‘ADDRESSES’’ at the beginning of this

document.

Electronic comments can be sent

directly to EPA at:

opp-docket@epamail.epa.gov

Electronic comments must be

submitted as an ASCII file avoiding the

use of special characters and any form

of encryption. Comment and data will

also be accepted on disks in

Wordperfect 5.1 file format or ASCII file

format. All comments and data in

electronic form must be identified by

the docket number FRL-5793-1 and

appropriate petition number. Electronic

comments on this proposed rule may be

filed online at many Federal Depository

Libraries.

List of Subjects

Environmental protection,

Agricultural commodities, Food

additives, Feed additives, Pesticides and

pests, Reporting and recordkeeping

requirements.

Dated: May 26, 1998.

James Jones,

Director, Registration Division, Office of

Pesticide Programs.

Summaries of Petitions

Petitioner summaries of the pesticide

petitions are printed below as required

by section 408(d)(3) of the FFDCA. The

summaries of the petitions were

prepared by the petitioners and

represent the views of the petitioners.

EPA is publishing the petition

summaries verbatim without editing

them in any way. The petition summary

announces the availability of a

description of the analytical methods

available to EPA for the detection and

measurement of the pesticide chemical

residues or an explanation of why no

such method is needed.

1. FMC Corporation

PP 6G4615

EPA has received a pesticide petition

(PP 6G4615) from FMC Corporation,

1735 Market Street, Philadelphia, PA

19103, proposing pursuant to section

408(d) of the Federal Food, Drug, and

Cosmetic Act, 21 U.S.C. 346a(d), to

amend 40 CFR part 180 by extending a

temporary tolerance for the combined

residue of the herbicide carfentrazoneethyl (ethyl-alpha-2-dichloro-5-[4-

(difluoromethyl)-4,5-dihydro-3-methyl5-oxo-1H-1,2,4-triazol-1-yl]-4-

fluorobenzene-propanoate) and its major

wheat metabolites: carfentrazone-ethyl

chloropropionic acid (alpha, 2-dichloro5-[4-difluoromethyl)-4,5-dihydro-3-

methyl-5-oxo-1H-1,2,4-triazol-1-yl]-4-

fluorobenzenepropanoic acid), 3-

hydroxymethyl-F8426-chloropropionic

acid (alpha,2-dichloro-5-[4-

difluoromethyl)-4,5-dihydro-3-

hydroxymethyl-5-oxo-1H-1,2,4-triazol-lyl]-4-fluorobenzenepropanoic acid), and

3-desmethyl-F8426 chloropropionic

acid (alpha, 2-dichloro-5-[4-

difluoromethyl)-4,5-dihydro-5-oxo-1H1,2,4-triazol-l-yl]-4-

fluorobenzenepropanoic acid) in or on

wheat raw agricultural commodities: 0.2

ppm in or on wheat hay, 0.2 ppm in or

on wheat straw, 0.2 ppm in or on wheat

grain; and extending tolerance for

combined residue of the herbicide

carfentrazone-ethyl (ethyl-alpha-2-

dichloro-5-[4-(difluoromethyl)-4,5-

dihydro-3-methyl-5-oxo-1H-1,2,4-

triazol-1-yl]-4-fluorobenzenepropanoate) and its major corn

metabolites: carfentrazone-ethyl

chloropropionic acid (alpha, 2-dichloro5-[4-difluoromethyl)-4,5-dihydro-3-

methyl-5-oxo-1H-1,2,4-triazol-l-yl]-4-

fluorobenzenepropanoic acid),and 3-

desmethyl-F8426 chloropropionic acid

(alpha, 2-dichloro-5-[4-difluoromethyl)-

4,5-dihydro-5-oxo-1H-1,2,4-triazol-1-yl]-

4-fluorobenzenepropanoic acid) in or on

corn raw agricultural commodities: 0.15

ppm in or on corn forage, 0.15 ppm in

or on corn fodder, 0.15 ppm in or on

corn grain.

EPA has determined that the petition

contains data or information regarding

the elements set forth in section

408(d)(2) of the FFDCA; however, EPA

has not fully evaluated the sufficiency

of the submitted data at this time or

whether the data supports granting of

the petition. Additional data may be

needed before EPA rules on the petition.

A. Residue Chemistry

1. Plant metabolism. The metabolism

of carfentrazone-ethyl in plants is

adequately understood. Corn and wheat

metabolism studies with carfentrazoneethyl have shown uptake of material

into plant tissue with no significant

movement into grain or seeds. All three

plants extensively metabolized

carfentrazone-ethyl and exhibited a

similar metabolic pathway. The residues

of concern are the combined residues of

carfentrazone-ethyl and carfentrazoneethyl- chloropropionic acid.

2. Analytical method There is a

practical analytical method for detecting

and measuring levels of carfentrazone

and its metabolites in or on food with

a limit of quantitation that allows

monitoring of food with residues at or

above the levels set in the tolerances.

The analytical method for

carfentrazone-ethyl involves separate

analyses for parent and its metabolites.

The parent is analyzed by GC/ECD. The

metabolites are derivatized with boron

trifluoride and acetic anhydride for

analysis by GC/MSD using selective ion

monitoring.

3. Magnitude of residues.

Carfentrazone-ethyl 50DF was applied

postemergent to 28 wheat trials, and 24

corn trials in the appropriate EPA

regions. The RAC’s were harvested at
31770 Federal Register / Vol. 63, No. 111 /Wednesday, June 10, 1998 / Notices

the appropriate growth stages and

subsequent analyses determined that the

residues of carfentrazone-ethyl and its

metabolites will not exceed the

proposed tolerances of 1.0, 0.3, 0.2 and

0.1 ppm for wheat forage, hay, straw

and grain, respectively; 0.1 ppm each

for corn forage, fodder, and grain.

Residue data from a cow feeding study

demonstrated that no accumulation of

carfentrazone-ethyl or its metabolites

occurred in milk or tissues.

B. Toxicological Profile

1. Acute toxicity. Carfentrazone-ethyl

demonstrates low oral, dermal and

inhalation toxicity. The acute oral LD50

value in the rat was greater than 5,000

milligram/Kilograms (mg/kg), the acute

dermal LD50 value in the rat was greater

than 4,000 mg/kg and the acute

inhalation LC50 value in the rat was

greater than 5.09 mg/L/4h.

Carfentrazone-ethyl is non-irritating to

rabbit skin and minimally irritating to

rabbit eyes. It did not cause skin

sensitization in guinea pigs. An acute

neurotoxicity study in the rat had a

systemic no-observed-adverse-effect

level (NOAEL) of 500 mg/kg based on

clinical signs and decreased motor

activity levels; the NOAEL for

neurotoxicity was greater than 2,000

mg/kg highest dose tested (HDT) based

on the lack of neurotoxic clinical signs

or effects on neuropathology.

2. Genotoxicty. Carfentrazone-ethyl

did not cause mutations in the Ames

assay with or without metabolic

activation. There was a positive

response in the Chromosome Aberration

assay without activation but a negative

response with activation. The Mouse

Micronucleus assay (an in vivo test

which also measures chromosome

damage), the CHO/HGPRT forward

mutation assay and the Unscheduled

DNA Synthesis assay were negative. The

overwhelming weight of the evidence

supports the conclusion that

Carfentrazone-ethyl is not genotoxic.

3. Reproductive and developmental

toxicity. Carfentrazone-ethyl is not

considered to be a reproductive or a

developmental toxin. In the 2-

generation reproduction study, the

NOEL for reproductive toxicity was

greater than 4,000 ppm (greater than 323

to greater than 409 mg/kg/day). In the

developmental toxicity studies, the rat

and rabbit maternal NOELs were 100

mg/kg/day and 150 mg/kg/day,

respectively. The developmental NOEL

for the rabbit was greater than 300 mg/

kg/day which was the HDT and for the

rat the NOEL was 600 mg/kg/day based

on increased litter incidences of

thickened and wavy ribs at 1,250 mg/kg/

day. These two findings (thickened and

wavy ribs) are not considered adverse

effects of treatment but related delays in

rib development which are generally

believed to be reversible.

4. Subchronic toxicity 90-day feeding

studies were conducted in mice, rats

and dogs with carfentrazone-ethyl. The

NOEL for the mouse study was 4,000

ppm (571 mg/kg/day), for the rat study

was 1,000 ppm (57.9 mg/kg/day for

males; 72.4 mg/kg/day for females) and

for dogs was 150 mg/kg/day. A 90-day

subchronic neurotoxicity study in the

rat had a systemic NOEL of 1,000 ppm

(59.0 mg/kg/day for males; 70.7 mg/kg/

day for females) based on decreases in

body weights, body weight gains and

food consumption at 10,000 ppm; the

neurotoxicity NOEL was greater than

20,000 ppm (1178.3 mg/kg/day for

males; 1433.5 mg/kg/day for females)

which was the HDT.

5. Chronic toxicity. Carfentrazoneethyl is not carcinogenic to rats or mice.

A 2-year combined chronic toxicity/

oncogenicity study in the rat was

negative for carcinogenicity and had a

chronic toxicity NOEL of 200 ppm (9

mg/kg/day) for males and 50 ppm (3

mg/kg/day) for females based on red

fluorescent granules consistent with

porphyrin deposits in the liver at the

500 and 200 ppm levels, respectively.

An 18-month oncogenicity study in the

mouse had a carcinogenic NOEL that

was greater than 7,000 ppm (>1090 mg/

kg/day for males; >1296 mg/kg/day for

females) based on no evidence of

carcinogenicity at the HDT. A 1-year

oral toxicity study in the dog had a

NOEL of 50 mg/kg/day based on

isolated increases in urine porphyrins in

the 150 mg/kg/day group (this finding

was not considered adverse).

Using the Guidelines for Carcinogen

Risk Assessment, carfentrazone-ethyl

should be classified as Group ‘‘E’’ for

carcinogenicity -- no evidence of

carcinogenicity -- based on the results of

carcinogenicity studies in two species.

There was no evidence of

carcinogenicity in an 18-month feeding

study in mice and a 2-year feeding study

in rats at the dosage levels tested (DLT).

The doses tested are adequate for

identifying a cancer risk. Thus, a cancer

risk assessment is not necessary.

6. Animal metabolism. The

metabolism of carfentrazone-ethyl in

animals is adequately understood.

Carfentrazone-ethyl was extensively

metabolized and readily eliminated

following oral administration to rats,

goats, and poultry via excreta. All three

animals exhibited a similar metabolic

pathway. As in plants, the parent

chemical was metabolized by hydrolytic

mechanisms to predominantly form

carfentrazone-ethyl-chloropropionic

acid which was readily excreted.

7. Endocrine disruption. An

evaluation of the potential effects on the

endocrine systems of mammals has not

been determined; however, no evidence

of such effects were reported in the

chronic or reproductive toxicology

studies described above. There was no

observed pathology of the endocrine

organs in these studies. There is no

evidence at this time that carfentrazoneethyl causes endocrine effects.

C. Aggregate Exposure

Dietary exposure—i. Acute dietary.

The Agency has determine that there is

no concern for an acute dietary risk

assessment since the available data do

not indicate any evidence of significant

toxicity from a 1-day or single event

exposure by the oral route Federal

Register of September 30, 1997 (62, FR

189). Thus an acute dietary risk

assessment is not necessary.

ii. Food. Dietary exposure from the

proposed uses would account for 1.3%

or less of the RfD in subpopulations

(including infants and children).

iii. Drinking water. Studies have

indicated that carfentrazone-ethyl will

not move into groundwater, therefore

water has not been included in the

dietary risk assessment.

iv. Non-dietary exposure. No specific

worker exposure tests have been

conducted with carfentrazone-ethyl.

The potential for non-occupational

exposure to the general population has

not been fully assessed. No specific

worker exposure tests have been

conducted with carfentrazone-ethyl.

D. Cumulative Effects

EPA is also required to consider the

potential for cumulative effects of

carfentrazone-ethyl and other

substances that have a common

mechanism of toxicity. EPA

consideration of a common mechanism

of toxicity is not appropriate at this time

since EPA does not have information to

indicate that toxic effects produced by

carfentrazone-ethyl would be

cumulative with those of any other

chemical compounds; thus only the

potential risks of carfentrazone-ethyl are

considered in this exposure assessment.

E. Safety Determination

1. U.S. population. Using the

conservative exposure assumptions

described and based on the

completeness and reliability of the

toxicity data, the aggregate exposure to

carfentrazone-ethyl will utilize 0.61% of

the RfD for the US population. EPA

generally has no concern for exposures

below 100% of the RfD. Therefore,
Federal Register / Vol. 63, No. 111 /Wednesday, June 10, 1998 / Notices 31771

based on the completeness and

reliability of the toxicity data and the

conservative exposure assessment, there

is a reasonable certainty that no harm

will result from aggregate exposure to

residues of carfentrazone-ethyl,

including all anticipated dietary

exposure and all other non-occupational

exposures. 2. Infants and children. In assessing

the potential for additional sensitivity of

infants and children to residues of

carfentrazone-ethyl, EPA considers data

from developmental toxicity studies in

the rat and rabbit and the 2-generation

reproduction study in the rat. The

developmental toxicity studies are

designed to evaluate adverse effects on

the developing organism resulting from

pesticide exposure during prenatal

development. Reproduction studies

provide information relating to effects

on the reproductive capacity of males

and females exposed to the pesticide.

Developmental toxicity was not

observed in developmental toxicity

studies using rats and rabbits. In these

studies, the rat and rabbit maternal

NOELs were 100 mg/kg/day and 150

mg/kg/day, respectively. The

developmental NOEL for the rabbit was

greater than 300 mg/kg/day which was

the HDT and for the rat was 600 mg/kg/

day based on increased litter incidences

of thickened and wavy ribs. These two

findings are not considered adverse

effects of treatment but related delays in

rib development which are generally

believed to be reversible.

In a 2-generation reproduction study

in rats, no reproductive toxicity was

observed under the conditions of the

study at 4,000 ppm which was the HDT. Section 408 of the FFDCA provides

that EPA may apply an additional safety

factor for infants and children in the

case of threshold effects to account for

pre- and post-natal toxicity and the

completeness of the database. Based on

the current toxicological data

requirements, the database relative to

pre- and post-natal effects for children

is complete and an additional

uncertainty factor is not warranted.

Therefore at this time, the provisional

RfD of 0.06 mg/kg/day is appropriate for

assessing aggregate risk to infants and

children.

F. Reference Dose

Using the conservative exposure

assumptions described above, the

percent of the RfD that will be utilized

by aggregate exposure to residues of

carfentrazone-ethyl for non-nursing

infants (<1-year old) would be 0.28%

and for children 1-6 years of age would

be 1.37% (the most highly exposed

group). Based on the completeness and

reliability of the toxicity data and the

conservative exposure assessment, there

is a reasonable certainty that no harm

will result to infants and children from

aggregate exposure to the residues of

carfentrazone-ethyl including all

anticipated dietary exposure.

G. International Tolerances

There are no Codex Alimentarius

Commission (Codex) Maximum Residue

Levels (MRLs) for carfentrazone-ethyl

on any crops at this time. However,

MRLs for small grains in Europe have

been proposed which consist of

carfentrazone-ethyl and carfentrazoneethyl-chloropropionic acid.

[FR Doc. 98–15177 Filed 6–9–98; 8:45 am]

BILLING CODE 6560–50–F

ENVIRONMENTAL PROTECTION

AGENCY

[PF–812; FRL–5793–4]

Notice of Filing of a Pesticide Petition

AGENCY: Environmental Protection

Agency (EPA).

ACTION: Notice.

SUMMARY: This notice announces the

amendment of pesticide petition (PP

5F4483), proposing the establishment of

regulations for residues of certain

pesticide chemicals in or on various

food commodities.

DATES: Comments, identified by the

docket control number PF–812, must be

received on or before July 10, 1998.

ADDRESSES: By mail submit written

comments to: Public Information and

Records Integrity Branch (7502C),

Information Resources and Services

Division, Office of Pesticides Programs,

Environmental Protection Agency, 401

M St., SW., Washington, DC 20460. In

person bring comments to: Rm. 119, CM

#2, 1921 Jefferson Davis Highway,

Arlington, VA.

Comments and data may also be

submitted electronically to: oppdocket@epamail.epa.gov. Follow the

instructions under ‘‘SUPPLEMENTARY

INFORMATION.’’ No confidential

business information should be

submitted through e-mail.

Information submitted as a comment

concerning this document may be

claimed confidential by marking any

part or all of that information as

‘‘Confidential Business Information’’

(CBI). CBI should not be submitted

through e-mail. Information marked as

CBI will not be disclosed except in

accordance with procedures set forth in

40 CFR part 2. A copy of the comment

that does not contain CBI must be

submitted for inclusion in the public

record. Information not marked

confidential may be disclosed publicly

by EPA without prior notice. All written

comments will be available for public

inspection in Rm. 119 at the address

given above, from 8:30 a.m. to 4 p.m.,

Monday through Friday, excluding legal

holidays.

FOR FURTHER INFORMATION CONTACT: By

mail: Shanaz Bacchus, Biopesticides

and Pollution Prevention Division

(7511W), Office of Pesticide Programs,

Environmental Protection Agency, 401

M St., SW., Washington, DC 20460.

Office location and telephone number:

Rm. CS51B6, Westfield Building North

Tower, 2800 Crystal Drive, Arlington,

VA 22202, (703) 308–8097; e-mail:

bacchus.shanaz@epamail.epa.gov.

SUPPLEMENTARY INFORMATION: EPA has

received a pesticide petition as follows

proposing the establishment and/or

amendment of regulations for residues

of certain pesticide chemicals in or on

various food commodities under section

408 of the Federal Food, Drug, and

Comestic Act (FFDCA), 21 U.S.C. 346a.

EPA has determined that this petition

contains data or information regarding

the elements set forth in section

408(d)(2); however, EPA has not fully

evaluated the sufficiency of the

submitted data at this time or whether

the data supports granting of the

petition. Additional data may be needed

before EPA rules on the petition.

The official record for this notice of

filing, as well as the public version, has

been established for this notice of filing

under docket control number [PF–812]

(including comments and data

submitted electronically as described

below). A public version of this record,

including printed, paper versions of

electronic comments, which does not

include any information claimed as CBI,

is available for inspection from 8:30

a.m. to 4 p.m., Monday through Friday,

excluding legal holidays. The official

record is located at the address in

‘‘ADDRESSES’’ at the beginning of this

document.

Electronic comments can be sent

directly to EPA at:

opp-docket@epamail.epa.gov

Electronic comments must be

submitted as an ASCII file avoiding the

use of special characters and any form

of encryption. Comment and data will

also be accepted on disks in

Wordperfect 5.1/6.1 or ASCII file

format. All comments and data in

electronic form must be identified by

the docket control number [PF–812] and

appropriate petition number. Electronic

comments on this notice may be filed

online at many Federal Depository

Libraries.