Federal Register / Vol. 62, No. 58 / Wednesday, March 26, 1997 / Notices 29

The following Table 2 includes the names and addresses of record for all registrants of the products in Table

1, in sequence by EPA company number.

TABLE 2. — REGISTRANTS REQUESTING AMENDMENTS TO DELETE USES IN CERTAIN PESTICIDE REGISTRATIONS

Company No. Company Name and Address

000769 SURECO, Inc., 10012 Dale Mabry, Suite 221, Tampa, FL 33618.

005481 AMVAC Chemical Corp., 4100 East Washington Blvd., Los Angeles, CA 90023.

008660 Pursell Industries, Inc., P.O. Box 540, Sylacauga, AL 35150.

034704 Platte Chemical Co., 419 18th Street, P.O. Box 667, Greeley, CO 80632.

III. Existing Stocks Provisions

The Agency has authorized registrants

to sell or distribute product under the

previously approved labeling for a

period of 18 months after approval of

the revision, unless other restrictions

have been imposed, as in special review

actions.

List of Subjects

Environmental protection, Pesticides

and pests, Product registrations.

Dated: March 13, 1997.

Linda A. Travers,

Director, Program Management Support

Division, Office of Pesticide Programs.

[FR Doc. 97–7063 Filed 3–25–97; 8:45 am]

BILLING CODE 6560–50–F

[PF–724; FRL–5594–7]

American Cyanamid Company;

Pesticide Tolerance Petition Filing

AGENCY: Environmental Protection

Agency (EPA).

ACTION: Notice.

SUMMARY: This notice announces the

filing of a pesticide petition proposing

the establishment of a tolerance for

residues of dimethomorph [(E,Z)4-[3-(4-

chlorophenyl)-3-(3,4-dimethoxyphenyl)-

1-oxo-2-propenyl]-morpholine] in or on

the raw agricultural commodity potatoes

and grape commodities. This notice

contains a summary of the petition that

was prepared by the petitioner,

American Cyanamid Company.

DATES: Comments, identified by the

docket control number [PF–724], must

be received on or before April 25, 1997.

ADDRESSES: By mail, submit written

comments to: Public Response and

Program Resources Branch, Field

Operations Division (7506C), Office of

Pesticide Programs, Environmental

Protection Agency, 401 M St., SW.,

Washington, DC 20460. In person, bring

comments to Rm. 1132, CM #2, 1921

Jefferson Davis Highway, Arlington, VA

22202.

Comments and data may also be

submitted electronically be sending

electronic mail (e-mail) to: oppdocket@epamail.epa.gov. Electronic

comments must be submitted as an

ASCII file avoiding the use of special

characters and any form of encryption.

Comments and data will also be

accepted on disks in WordPerfect 5.1

file format or in ASCII file format. All

comments and data in electronic form

must be identified by docket control

number [PF–724]. Electronic comments

on this notice may be filed online at

many Federal Depository Libraries.

Additional information on electronic

submissions can be found in Unit II. of

this document.

Information submitted as a comments

concerning this document may be

claimed confidential by marking any

part or all of that information as

‘‘Confidential Business Information’’

(CBI). CBI should not be submitted

through e-mail. Information marked as

CBI will not be disclosed except in

accordance with procedures set forth in

40 CFR part 2. A copy of the comment

that does not contain CBI must be

submitted for inclusion in the public

record. Information not marked

confidential may be disclosed publicly

by EPA without prior notice. All written

comments will be available for public

inspection in Rm. 1132 at the address

given above, from 8:30 a.m. to 4 p.m.,

Monday through Friday, excluding legal

holidays.

FOR FURTHER INFORMATION CONTACT: By

mail: Connie B. Welch, Product

Manager (PM) 21, Registration Division

(7505W), Office of Pesticide Programs,

Environmental Protection Agency, 401

M St., SW., Washington, DC 20460.

Office location, telephone number, and

e-mail address: Rm. 227, CM #2, 1921

Jefferson Davis Highway, Arlington, VA,

(703) 305–6226; e-mail:

welch.connie@epamail.epa.gov.

SUPPLEMENTARY INFORMATION: EPA has

received a pesticide petition (PP

7F4816) from American Cyanamid

Company, Agricultural Products

Research Division, P.O. Box 400

Princeton, NJ 08543-0400 proposing

pursuant to section 408(d) of the Federal

Food, Drug, and Cosmetic Act, 21 U.S.C.

section 346a, to amend 40 CFR part 180

by establishing tolerances for residues of

the fungicide, dimethomorph [(E,Z)4-[3-

(4-chlorophenyl)-3-(3,4-

dimethoxyphenyl)-1-oxo-2-propenyl]-

morpholine] in or on the raw

agricultural commodity potato at 0.05

parts per million (ppm) and a timelimited tolerance for residues of

dimethomorph in or on the raw

agricultural commodity grape at 2.0

ppm. The proposed analytical method

for determining residues is a High

Performance Liquid Chromatography

(HPLC) method (FAMS 002-04). A

confirmatory method (FAMS 022-03)

also is available which provides for

analysis by either Gas Chromatography/

Nitrogen-Phosphorus Detection or by

HPLC/UV Detection. EPA has

determined that the petition contains

data or information regarding the

elements set forth in section 408(d)(2);

however, EPA has not fully evaluated

the sufficiency of the submitted data at

this time or whether the data supports

granting of the petition. Additional data

may be needed before EPA rules on the

petition.

As required by section 408(d) of the

FFDCA, as recently amended by the

Food Quality Protection Act, (Pub. L.

104-170), American Cyanamid included

in the petition a summary of the petition

and authorization for the summary to be

published in the Federal Register in a

notice of receipt of the petition. The

summary represents the views of

American Cyanamid; EPA is in the

process of evaluating the petition. As

required by section 408(d)(3), EPA is

including the summary as a part of this

notice of filing. EPA has made minor

edits to the summary for the purpose of

clarity.
30 Federal Register / Vol. 62, No. 58 / Wednesday, March 26, 1997 / Notices

I. Petition Summary Prepared by

American Cyanamid Company

A. Residue Chemistry

1. Plant metabolism. American

Cyanamid believes that the results of the

potato metabolism study show only

negligible residues in tubers, 0.01-0.02

ppm total radioactive residues (TRR).

This is in contrast to the aerial portions

of the plant which were found to have

up to 23.5 parts per million (ppm) TRR,

thus demonstrating that translocation of

dimethomorph within the plant was not

significant. Almost all of the radioactive

residue (97.8%) was extractable from

the plant at harvest. In the aerial portion

of the plant, approximately 70% of the

TRR was identified as dimethomorph.

No metabolites were identified that

require regulation. There was no

concentration of residue in the peel or

tuber. The latter point indicates that

during processing dimethomorph is not

expected to concentrate to a level

greater than that of the proposed

tolerance for the raw agricultural

commodity, potato tubers.

The results of the grape metabolism

study showed that the TRR in/on grapes

harvested 35 days following the last of

four applications (0.8 lb active

ingredient/application (ai/A) for 4

consecutive weeks) for a total rate of 3.2

lb ai/A (3x the proposed maximum

seasonal rate) was 14.6 ppm.

Unmetabolized dimethomorph

accounted for 87.3% of the TRR (12.7

ppm). No metabolites were identified

that require regulation.

2. Analytical method. A reliable

method for the determination of

dimethomorph residues in potatoes and

grapes exists. This method (FAMS 002-

04) is appropriate for enforcement

purposes. FAMS 002-04 is a HPLC

method. A confirmatory method (FAMS

022-03) also is available which provides

for analysis by either Gas

Chromatography with NitrogenPhosphorus Detection or by HPLC with

UV Detection.

3. Magnitude of residues. The residue

data for potato submitted to support this

tolerance petition were collected from

studies conducted in several European

countries; these countries are

representative of potato growing regions

of the U.S. Dimethomorph residues

observed in these field residue studies

ranged from <0.01 ppm (the Limit of

Quantitation of the method) to 0.04

ppm; however, most residues were

<0.01 ppm. These trials were conducted

using multiple applications (5-12) with

a maximum seasonal rate of up to 2.56

lb ai/A. The proposed U.S. use pattern

is five applications at a maximum

treatment rate of 0.203 lb ai/A and a

maximum seasonal use rate of 1.015 lb

ai/A. Residue levels in domestic

potatoes would be expected to be

similar or lower (< 0.01 ppm) than that

observed in the European trials.

Therefore, a tolerance of 0.05 ppm is

appropriate.

The residue data for grape submitted

to support this tolerance petition were

collected from studies conducted in

various regions of France; these sites are

representative of grape growing regions

of Europe. Dimethomorph residues

observed in these field residue studies

ranged from <0.01 ppm (the Limit of

Quantitation of the method) to 1.81

ppm. These trials were conducted using

multiple applications (3-11) with a

maximum seasonal rate of up to 2.94 lb

ai/A. In six studies conducted on the

magnitude of residue in grape processed

commodities, residues of dimethomorph

did not concentrate in grape juice or

wine. Therefore, a time-limited

tolerance of 2.0 ppm in/on grape

commodities is appropriate.

B. Toxicological Profile

American Cyanamid believes that the

toxicity of dimethomorph has been

studied extensively and there is a

complete data base to address the acute

and chronic effects, effects on genetic

material, the potential for

carcinogenicity or teratogenicity, and

effects on reproductive performance or

growth of offspring.

The toxicological data submitted to

support the petition for a tolerance for

dimethomorph on potatoes and for a

time-limited tolerance on grape include:

1. Acute toxicity. i. An acute oral

toxicity study in the Sprague-Dawley rat

for dimethomorph technical with a LD50

of 4,300 milligrams/kilograms (mg/kg)

body weight (bwt) for males and 3,500

mg/kg bwt for females. Based upon EPA

toxicity criteria, the acute oral toxicity

category for dimethomorph technical is

Category III or slightly toxic.

ii. Oral LD50 studies were conducted

on the two isomers (E and Z) alone:

a. An acute oral toxicity study in the

Wistar rat for the E-isomer with a LD50

greater than 5,000 mg/kg bwt for males

and approximately 5,000 mg/kg bwt for

females.

b. An acute oral toxicity study in the

Wistar rat for the Z-isomer with a LD50

greater than 5,000 mg/kg bwt for both

males and females.

iii. An acute dermal toxicity study in

the Wistar rat for dimethomorph

technical with a dermal LD50 greater

than 5,000 mg/kg bwt for both males

and females. Based on the EPA toxicity

category criteria, the acute dermal

toxicity category for dimethomorph is

Category IV or relatively non-toxic.

iv. A 4-hour inhalation study in

Wistar rats for dimethomorph technical

with a LC50 greater than 4.2 mg/L for

both males and females. Based on the

EPA toxicity category criteria, the acute

inhalation toxicity category for

dimethomorph technical is Category IV

or relatively non-toxic.

2. Genotoxicity. i. Salmonella reverse

gene mutation assays (2 studies) were

negative up to a limit dose of 5,000 ©g/

plate. Chinese hamster lung cells were

negative in V79 cells up to toxic doses

in 2 studies.

ii. Two Chinese hamster lung

structural chromosomal studies were

reportedly positive for chromosomal

aberrations at the highest dose tested

(HDT) (160 ©g/ml/-S9; 170 ©g/ml/+S9).

Dimethomorph induced only a weak

response in increasing chromosome

aberrations in this test system. These

results were not confirmed in two

micronucleus tests under in vivo

conditions.

iii. Structural Chromosomal

Aberration studies were weakly

positive, in human lymphocyte cultures,

but only in S9 activated cultures treated

at the HDT (422 ©g/ml) which was

strongly cytotoxic. Dimethomorph was

negative in the absence of activation at

all doses. Furthermore, the positive

clastogenic response observed under the

in vitro conditions was not confirmed in

two in vivo micronucleus assays.

iv. Micronucleus assay (2 studies)

indicated that dimethomorph was

negative for inducing micronuclei in

bone marrow cells of mice following i.p.

administration of doses up to 200 mg/

kg or oral doses up to the limit dose of

5,000 mg/kg. Thus, dimethomorph was

found to be negative in these studies for

causing cytogenic damage in vivo.

v. Dimethomorph was negative for

inducing unscheduled DNA synthesis,

in cultured rat liver cells, at doses up to

250 ©g/ml, a weakly cytotoxic level.

vi. Dimethomorph was negative for

transformation in Syrian hamster

embryo cells treated, in the presence

and absence of activation, up to

cytotoxic concentrations (265 ©g/ml/

+S9; 50 ©g/ml/-S9).

3. Reproductive and developmental

toxicity. i. A rat developmental toxicity

study with a maternal toxicity LowestObserved-Effect Level (LOEL) of 160

mg/kg/day and a maternal toxicity NoObserved-Effect Level (NOEL) of 60 mg/

kg/day. The NOEL for developmental

toxicity is 60 mg/kg/day. Dimethomorph

is not teratogenic in the Sprague-Dawley

rat.

ii. A rabbit development toxicity

study with maternal toxicity LOEL of

650 mg/kg/day and a NOEL of 300 mg/

kg/day. The NOEL for developmental
Federal Register / Vol. 62, No. 58 / Wednesday, March 26, 1997 / Notices 31

toxicity is 650 mg/kg/day, the highest

dose tested. Dimethomorph is not

teratogenic in the New Zealand white

rabbit.

iii. A multi-generational rat

reproduction study with parental LOEL

for systemic toxicity of 80 mg/kg/day

and a NOEL of 24 mg/kg/day. The NOEL

for fertility and reproductive function

was 80 mg/kg/day, the highest dose

tested.

4. Subchronic toxicity. i. A 90–day

dietary study in Sprague-Dawley rats

with a NOEL of greater than or equal to

73 mg/kg/day in males and 82 mg/kg/

day in females, the highest doses tested.

ii. A 90–day dog dietary study with a

NOEL 15 mg/kg/day and a LOEL 43 mg/

kg/day.

5. Chronic toxicity. i. A 2–year

chronic toxicity study in SpragueDawley rats with a NOEL 9 mg/kg/day

for males and 12 mg/kg/day for females.

The LOEL for systemic toxicity is 36

mg/kg/day for males and 58 mg/kg/day

for females.

ii. A 1–year chronic toxicity study in

dogs with a NOEL of 14.7 mg/kg/day

and a LOEL of 44.6 mg/kg/day.

iii. A 2–year oncogenicity study in

Sprague-Dawley rats with a NOEL for

systemic toxicity of 9 mg/kg/day for

males and 11 mg/kg/day for females.

The LOEL for systemic toxicity was 34

mg/kg/day for males and 46 mg/kg/day

for females. There was no evidence of

increased incidence of neoplastic

lesions in treated animals. The NOEL

for oncogenicity is 95 mg/kg/day for

males and 132 mg/kg/day for females,

the highest dose tested.

iv. A 2–year oncogenicity study in

mice with a NOEL for systemic toxicity

of 100 mg/kg/day and a LOEL of 1,000

mg/kg/day. There was no evidence of

increased incidence of neoplastic

lesions in treated animals. The NOEL

for oncogenicity is 1,000 mg/kg/day, the

highest dose tested.

6. Animal metabolism. Results from

the livestock and rat metabolism studies

show that orally administered

dimethomorph was rapidly excreted by

the animals. The principal route of

elimination is the feces.

7. Metabolite toxicology. There were

no metabolites identified in potatoes or

animal commodities which require

regulation.

8. Endocrine effects. There is no

evidence of effects of dimethomorph on

the endocrine system. There were no

changes noted in organ weights for the

pituitary, thyroid, ovaries or testes.

There was no increased incidence of

mammary tumors observed. No effects

on fertility or reproduction were noted

and there was no evidence of related

histopathological changes in

reproductive or endocrine system

organs.

C. Aggregate Exposure

1. Dietary (food) exposure. Dietary

exposure should be based solely upon

the Theoretical Maximum Residue

Concentration (TMRC) from the

tolerance of 0.05 ppm dimethomorph in

or on potato and the time-limited

tolerance of 2.0 ppm dimethomorph in

or on grape. The goat metabolism study

demonstrates that there is no reasonable

expectation of transfer of residues of

dimethomorph to meat or milk from

potatoes or from grapes. There are no

potato or grape feed commodities fed to

poultry. Therefore, no consumption data

associated with meat, milk, poultry or

eggs should be included in the

calculation of the TMRC. There are no

other established U.S. tolerances for

dimethomorph, and there are no

registered uses for dimethomorph on

food or feed crops in the United States.

2. Dietary (drinking water) exposure.

There is no available information about

dimethomorph exposure via drinking

water. However, exposure to

dimethomorph from drinking water is

not likely to occur as a result of use on

potatoes. Dimethomorph dissipated

fairly rapidly under field conditions

with half lives ranging from 14 to 57

days. Laboratory and field studies

demonstrate that dimethomorph is not

mobile in soil. No movement below the

top 4 inches was observed in the field

studies. Laboratory leaching studies

result in the classification of

dimethomorph as having medium to

high adsorption onto soil.

3. Non-dietary exposure. There are no

other registered uses of dimethomorph

in the U.S. Thus, there is no potential

for non-dietary exposure.

D. Cumulative Effects

There is no information to indicate

that any toxic effects produced by

dimethomorph would be cumulative

with those of any other chemical. The

fungicidal mode of action of

dimethomorph is unique;

dimethomorph inhibits cell wall

formation only in Oomycete fungi. The

result is lysis of the cell wall which kills

growing cells and inhibits spore

formation in mature hyphae. This

unique mode of action and limited pest

spectrum suggest that there is little or

no potential for cumulative toxic effects

in mammals. In addition, the toxicity

studies submitted to support this

petition do not indicate that

dimethomorph is a particularly toxic

compound. No toxic end-points of

potential concern were identified.

E. Safety Determination

1. U.S. population. The proposed

reference dose (RfD) is 0.1 mg/kg bwt/

day, based on a NOEL of 10 mg/kg bwt/

day from a 2–year dietary toxicity study

in rats that demonstrated decreased

body weight and liver foci in females.

The proposed RfD is also based on an

uncertainty factor of 100. For potatoes,

the TMRC from this proposed action is

estimated at 0.000057 mg/kg bwt/day.

This represents an aggregate exposure to

the general population of the United

States of 0.063 percent of the RfD. The

TMRC for the most highly exposed

group, children ages 1 to 6 is estimated

at 0.000113 mg/kg bwt/day. This

represents 0.125 percent of the RfD.

Establishment of a tolerance for residues

in/on grape commodities is not

expected to significantly change the

exposure estimate to the most highly

exposed group since the commodity

which is most extensively imported is

wine. Since EPA generally has no

concern for exposures below 100

percent of the RfD, EPA should

conclude that there is a reasonable

certainty that no harm will result from

aggregate exposure to dimethomorph

residues in or on potato and grape

commodities.

2. Infants and children. American

Cyanamid believes that the results of the

studies submitted to support this

package provide no evidence that

dimethomorph caused reproductive,

developmental or fetotoxic effects. No

such effects were noted at dose levels

which were not maternally toxic. The

NOELs observed in the developmental

and reproductive studies were 6 to 65

times higher than the NOEL used to

establish the proposed RfD (10 mg/kg

bw/day). There is no evidence to

indicate that children or infants would

be more sensitive than adults to toxic

effects caused by exposure to

dimethomorph.

F. International Issues

No Codex maximum residue levels

(MRLs) have been established for

dimethomorph to date.

II. Public Record

A record has been established for this

notice under docket control number

[PF–724] (including comments and data

submitted electronically as described

below). A public version of the record,

including printed, paper versions of

electronic comments, which does not

include any information claimed as CBI,

is available for inspection from 8:30

a.m. to 4 p.m., Monday through Friday,

excluding legal holidays. The public

record is located in Room 1132 of the
32 Federal Register / Vol. 62, No. 58 / Wednesday, March 26, 1997 / Notices

Public Response and Resources Branch,

Field Operations Division (7506C),

Office of Pesticide Programs,

Environmental Protection Agency,

Crystal Mall #2, 1921 Jefferson Davis

Highway, Arlington, VA.

Electronic comments can be sent

directly to EPA at:

opp-docket@epamail.epa.gov

Electronic comments must be

submitted as an ASCII file avoiding the

use of special characters and any form

of encryption.

The official record for this

rulemaking, as well as the public

version, as described above will be kept

in paper form. Accordingly, EPA will

transfer all comments received

electronically into printed, paper form

as they are received and will place the

paper copies in the official rulemaking

record which will also include all

comments submitted directly in writing.

The official rulemaking record is the

paper record maintained at the address

in ‘‘ADDRESSES’’ at the beginning of

this document.

List of Subjects

Environmental protection,

Administrative practice and procedure,

Agricultural commodities, Pesticides

and pests, Reporting and recordkeeping.

Dated: March 18, 1997.

Stephen L. Johnson,

Director, Registration Division, Office of

Pesticide Programs.

[FR Doc. 97–7494 Filed 3–25–97; 8:45 am]

BILLING CODE 6560–50–F

[PF–722; FRL–5592–8]

DowElanco; Pesticide Tolerance

Petition Filing

AGENCY: Environmental Protection

Agency (EPA).

ACTION: Notice of Filing.

SUMMARY: This notice is a summary

announces the filing of a pesticide

petition proposing the establishment of

a regulation for residues of cloransulammethyl in or on soybeans. This notice

contains a summary was prepared by

the petitioner, DowElanco.

DATES: Comments, identified by the

docket number [PF–722], must be

received on or before April 25, 1997.

ADDRESSES: By mail, submit written

comments to Public Response and

Program Resources Branch, Field

Operations Division (7506C), Office of

Pesticide Programs, Environmental

Protection Agency, 401 M St. SW.,

Washington, DC 20460. In person, bring

comments to Rm. 1132, CM #2. 1921

Jefferson Davis Highway, Arlington, VA

22202. Comments and data may also be

submitted electronically be sending

electronic mail (e-mail) to: oppdocket@epamail.epa.gov. Electronic

comments must be submitted as an

ASCII file avoiding the use of special

characters and any form of encryption.

Comments and data will also be

accepted on disks in WordPerfect 5.1

file format or in ASCII file format. All

comments and data in electronic form

must be identified by docket control

number [PF–722]. Electronic comments

on this notice may be filed online at

many Federal Depository Libraries.

Additional information on electronic

submissions can be found below this

document.

Information submitted as a comments

concerning this document may be

claimed confidential by marking any

part or all of that information as

‘‘Confidential Business Information’’

(CBI). CBI should not be submitted

through e-mail. Information marked as

CBI will not be disclosed except in

accordance with procedures set forth in

40 CFR part 2. A copy of the comment

that does not contain CBI must be

submitted for inclusion in the public

record. Information not marked

confidential may be disclosed publicly

by EPA without prior notice. All written

comments will be available for public

inspection in Rm. 1132 at the address

given above, from 8:30 a.m. to 4 p.m.,

Monday through Friday, excluding legal

holidays.

FOR FURTHER INFORMATION CONTACT:

Philip Errico, Product Manager (PM) 25,

Registration Division, (7505C), Office of

Pesticide Programs, Environmental

Protection Agency, 401 M. St., SW.,

Washington, D.C. Office location,

telephone number and e-mail address:

Rm. 237, CM#2, 1921 Jefferson Davis

Highway, Arlington, VA 703–305–6027.

e-mail: errico.phillip@epamail.epa.gov.

SUPPLEMENTARY INFORMATION: EPA has

received a pesticide petition (PP 5F4560

from DowElanco, 9330 Zionsville Road,

Indianapolis, IN 46268-1054 proposing

pursuant to section 408(d) of the Federal

Food, Drug and Cosmetic Act, 21 U.S.C.

section 346a(d), to amend 40 CFR part

180 by establishing a tolerance for

residues of the herbicide N-(2-

carboxymethyl-6-chlorophenyl)-5-

ethoxy-7-fluoro[1,2,4]

triazolo[1,5c]pyrimidine-2-sulfonamide,

(cloransulam-methyl) in or on the raw

agricultural commodity soybeans at 0.02

ppm, soybean forage at 0.1 part per

million (ppm) and soybean hay at 0.2

ppm. The proposed analytical method is

gas chromatography coupled with a

mass selective detector (GC-MSD).

EPA has determined that the petition

contains data or information regarding

the elements set forth in section

408(d)(2); however, EPA has not fully

evaluated the sufficieny of the

submitted data at this time or whether

the data supports granting of the

petition. Additional data may be needed

before EPA rules on the petition.

Availability of the analytical method:

The proposal analytical method of

enforcement which measures residues

of cloransulam-methyl in soybeans, and

soybean forage and hay discussed below

has not been validated by the Agency.

Public versions of the analytical method

can be obtained from the Pesticide

Docket, U.S. Environmental Protection

Agency, Office of Pesticide Programs,

401 M. St., SW., Washington, D.C.

20460, (703) 305–5805.

As required by section 408(d) of the

FFDCA, as recently amended by the

Food Quality Protection Act,

DowElanco included in the petition a

summary of the petition and

authorization for the summary to be

published in the Federal Register in a

notice of receipt of the petition. The

summary represents the views of

DowElanco; EPA, as mentioned above,

is in the process of evaluating the

petition. As required by section

408(d)(3) EPA is including the summary

as a part of this notice of filing. EPA

may have made minor edits to the

summary for the purpose of clarity.

I. Petition Summary

A. Residue Chemistry

1. Plant metabolism. Nature of residue

studies demonstrated that residues of

cloransulam-methyl and its metabolites

would not be expected to accumulate to

significant levels in soybeans treated

either pre-plant or post-emergence, and

that it was appropriate to base the

magnitude of total terminal residues and

proposed tolerances only on residues of

the parent compound, cloransulammethyl. A rotational crop study showed

no significant level of cloransulammethyl or any structurally-related

metabolite in any crop, or crop

fractions, grown in rotation 120 days

after soil treatment.

2. Analytical method. Residue

analytical methods were validated based

upon gas chromatography coupled with

a mass selective detector (GC- MSD).

The limit of detection of the methods is

0.005 ppm and a level of quantitation is

0.01 ppm.

3. Magnitude of residues. No

detectable residues of cloransulammethyl resulted in soybeans from either

preplant incorporated or post emergence

applications, in soybean forage or hay