DRAF T

VOLUM E I V APPENDIXE S

375798

TAILING S DISPOSAL

AREA REMEDIAL

INVESTIGATION REPORT,

CALIFORNIA GULCH SITE ,

LEADVILLE, COLORADO

Prepared for ASARCO, Inc

1019 Eighth Street, Suite 304 A Golden, CO 80401 February 1993

Distributed for public comment June 1993

Woodward-Clyde

Woodward-Clyde Consultants

4582 South Ulster Street Stanford Place 3, Suite 1000 Denver, Colorado 80237

22909C T2001
VOLUME I I TABLE OF CONTENTS

APPENDI X A APPENDIX B APPENDIX C APPENDIX D

APPENDIX E APPENDI X F

APPENDIX G

MASTER SAMPLE LIST SURFAC E WATER TAILING S GEOCHEMISTR Y DRILLING LOGS AND GEOTECHNICA L TESTING RESULT S GROUND WATER TOXICOLOGICAL PROFILES, INTERPRETATION OF ASSES S OUTPUT , ASSES S OUTPUT SUMMAR Y OF THE CHEMICA L DATA QUALITY ASSURANCE PROCEDURES

22909C-22327/R22-II.TC 01-28-93(9:32pm)/RPT/16 -1-
APPENDI X E

GROUNDWATER

22909C-22327/R22.TS 12-23-92(9:2Spm)/RPT/10
APACHE TAILING S IMPOUNDMEN T
Monitorin g Wel l ID

AP1TMW 1

AP1TMW 2

AP1TMW 3

AP1TMW 7

AP1TMW 8

AP1TMW 9

AP1TMW1 0

AP2TMW 1

Development Date

10/9/91

10/4/91

10/7-8/91

10/16/91

11/21/9 1

11/21-26/91

11/21/9 1

11/21/9 1

Development Method

4" Pump

4" Pump

4" Pump

4" Pump

N / A

Disposable Bailer

N /A

N /A

MONITOF

***^*"*^^"^Bsrr^

g

**^g

Initial Water Level From T op of PVC

\ (ft)

12.38

3.34

2.47

62.96

Dry

19.41

Dry

Dry

IING WELL I

Volume Purged

(gal)

88.8

85.8

151.8

140.2

N /A

16.5

N /A

N /A

DEVELOPME

Casing Volumes Purged

10.4

10.3

11.4

14.8

N /A

10.6

N / A

N/A f

I

E NT DATA S

Purging Time (min)

89.0

292.0

210.0

39.0

N /A

66 min over

4 days

N /A

fj/A

JMMAR Y " •

Estimated Production

(gpm )

1.0

0.3

0.7

3.6

N / A

•= 0.01

N / A

N /A

P H 1

(std units)

5.68

6.72

5.87

7.26

N / A

6.29

N /A

N /A

.• ••• , , ==

Specifi c Conductivity1

(jumhos/cm)

2,926

908

1,520

439

N /A

6,720

N /A

N /A

— — — -

Temperature 1

(°C )

11.6

9.2

10.5

5.9

N /A

6.5

N /A

N /A

1 T h e reported pH, specifi c conductivity and temperature values were measured at the conclusion of well development .
..„ IV =.. '", Monitoring Wel l ID

AP1TMW 1

AP1TMW 2

AP1TMW 3

AP1TMW 7

AP1TMW 8

AP1TMW 9

AP1TMW1 0

AP2TMW 1

MONITORIN G WELL SAMPLIN G DATA SUMMAR Y

Sampl e Date

11/21/9 1

11/21/9 1

11/21/9 1

11/22/9 1

11/21/9 1

11/27/9 1

11/21/9 1

11/21/9 1

Samplin g Method

2" Grundfos

2" Grundfo s

2" Grundfos

2" Grundfo s

N /A

Disposable Bailer

N /A

N /A

Initial Water Level From T op of PVC

(ft )

11.94

2.69

2.56

61.98

Dry

19.05

Dry

Dry

Volume Purged

(gal )

28

27

40

34

N /A

6

N /A

N / A

Casing Volumes Purged

3.0

3.0

2.4

3.4

N /A

1.9

N /A

N / A

—

P H 1

(std units)

5.50

6.49

7.07

7.27

N / A

6.15

N /A

N /A

Specifi c Conductivity 1

(//mhos/cm )

1,720

827

523

441

N / A

6,580

N /A

N /A

Temperature 1

(°C )

8.4

7.0

8.0

4.3

N /A

7.4

N /A

N / A

reported pH, specifi c conductivit y and temperature were measured at the time of sample collection .
Monitoring Wel l ID

CZ1TMW 6

AP1TMW 1

AP1TMW 2

AP1TMW 3

AP1TMW 7

AP1TMW 8

AP1TMW 9

AP1TMW1 0

AP2TMW 1

GROUNDWATE R QUALIT i

Sample ID

L-CZ1TMW06-03-91 1116

L-CZ1TMW06-04-911116

L-AP1TMW01-01-91112 1

L-AP1TMW01-02-911121

L-AP1TMW01-MS-91112 1

L-AP1TMW02-01-911121

L-AP1TMW03-01-91112 1

L-AP1TMW03-04-911121

L- AP 1 TM W07-0 1 -9 1 11 2 2

L-AP1 TMW07-02-91 1 1 22

L-AP1TMW07-MS-91 1 1 22

N /A

L-AP1TMW09-01-91 1 127

L-AP1TMW09-02-91 1127

L-AP1TMW09-MS-91 1 127

L-AP1TMW09-03-91 1 1 27

N /A

N / A

f SAMPLES C

Sample Date

11/16/9 1

11/16/9 1

11/21/9 1

11/21/9 1

11/21/9 1

11/21/9 1

11/21/9 1

11/21/9 1

11/22/91

11/22/91

11/22/91

N /A

1 1/27/91

11/27/91

11/27/91

11/27/91

N /A

N /A

3LLECTED =

Sample Time

1840

1800

1215

1215

1215

1715

1430

1500

1000

1000

1000

N /A

1330

1330

1330

1230

N /A

N / A

^^^T^^^^^^^^^^^^^^^T^^^5SI5

Type of Sample

Field Blank

Equipment Rinsate

Primary

Duplicate

Matrix Spike

Primary

Primary

Equipmen t Rinsate

Primary

Duplicate

Matrix Spike

N /A

Primary

Duplicate

Matrix Spike

Field Blank

N /A

N / A
Monitoring Wel l ID

AP1TMW 1

AP1TMW 2

MONITORIN G

Measurement Date

10/09/91

10/18/91

11/21/9 1

01/17/92

02/18/92

03/1 6/92

04/13/92

05/18/92

06/1 5/92

07/20/92

08/17/92

09/21/92

10/26/92

10/04/91

10/05/91

11/21/9 1

01/15/9 2

02/18/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

WEL ^L WATER LE

 »j Measurement Time

1049

1631

1125

1245

1710

1433

0904

11 30

1457

1 642

1714

, 1555

1236

1205

0855

1630

1600

1725

1403

0857

1140

1430

1624

1739

1542

1229

V EL SUMMAR Y r5grrr?^*^r*Ts*TBr*^s^T5S

Depth to Water

(ft )

12.38

12.40

11.94

12.27

12.05

12.03

11.72

11.56

11.61

11.44

11.93

12.19

12.55

3.34

3.22

2.69

FROZEN

FROZEN

FROZEN

FROZEN

3.21

3.23

2.84

2.89

3.50

3.39

••IMHMI^MMMiMMCBBMMMiMMMBHH^

Groundwater

Elevation

(ft )

10031.78

10031.76

10032.22

10031.89

10032.11

10032.13

10032.44

10032.60

10032.55

10032.72

10032.23

10031.97

10031.61

10037.80

10037.92

10038.45

FROZEN

FROZEN

FROZEN

FROZEN

10037.93

10037.91

10038.30

10038.25

10037.64

10037.75
— Monitoring Well ID

AP1TMW 3

A P1 TMW 7

MONITORIN G TM*^M

"*^*""^^^I*TM***'^TMM"M

Measurement Date

10/07/91

10/18/91

11/21/91

01/15/92

02/1 8/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

10/11/91

10/16/91

11/22/91

01/17/92

01/21/92

02/18/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

WELL WATER LE

SSSSSSS^nmf^^SSSS^^S^Sm

Measurement Time

1425

1625

1330

1615

1730

1408

0849

1135

1420

1630

1727

1540

1233

1740

0915

0915

1358

0908

1615

1428

0909

1055

1559

1550

1646

1615

1700

V EL SUMMARY

Depth to Water

(ft)

2.47

2.67

2.56

2.72

FROZEN

FROZEN

FROZEN

2.82

2.80

2.63

2.72

2.82

2.61

63.20

62.96

61.98

61.83

61.85

62.15

62.68

62.93

63.68

64.12

63.61

61.98

59.41

57.15

Groundwater

Elevation

(ft)

10027.48

10027.28

10027.39

10027.23

FROZEN

FROZEN

FROZEN

10027.13

10027.15

10027.32

10027.23

10027.13

10027.34

10083.42

10083.66

10084.64

10084.79

10084.77

10084.47

10083.94

10083.69

10082.94

10082.50

10083.01

10084.64

10087.21

10089.47
Monitoring Wel l ID

AP1TMW 8

AP1TMW 9

MONITORIN G

Measurement Date

10/23/91

11/21/91

01/17/92

02/18/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

11/21/9 1

11/26/91

11/27/91

01/15/9 2

02/18/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

WEL L WATER LE

Measurement Time

NA

NA

1542

1640

1352

1139

1120

1530

1510

1705

1630

1642

0855

0814

0700

1133

1635

1348

1132

1108

1514

1600

1657

1625

1632

V EL SUMMAR Y ===:====

Depth to Water

(ft)

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

19.41

19.57

19,05

20.04

21.38

20.39

20.72

20.39

19.85

19.45

19.02

18.79

18.66

- —— — --

Groundwater

Elevation

(ft)

DRY

DRY

DRY

DRY

DRY

DRY

D RY

DRY

DRY

D RY

DRY

DRY

10094.76

10094.60

10095.12

10094.13

10092.79

10093.78

10093.45

10093.78

10094.32

10094.72

10095.15

10095.38

10095.51
MONITORIN G WELL WATER LEVEL SUMMAR Y

Monitoring Wel l ID

AP1TMW1 0

AP2TMW 1

Measurement Date

10/23/91

11/21/9 1

01/17/92

02/18/92

03/1 6/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

10/29/91

11/21/9 1

01/15/92

02/18/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

Measurement Time

NA

NA

1535

1645

1344

1128

1112

1521

1605

1701

1630

1636

: :, NA

NA

1121

1 740

1358

NA

1145

1445

1629

1749

1550

1705

L»,— — :r~ ~" .~ •!••• ——— .-g-^i — .

Depth to Water

<ft)

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

D RY

D RY

DRY

DRY

DRY

DRY

DRY

FROZEN

DRY

DRY

DRY

DRY

DRY

DRY

[==^====^===

Ground water

Eevation

(ft)

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

FROZEN

DRY

DRY

DRY

DRY

DRY

DRY

1

 Water levels measured from the north side of the top of PVC casing.
Monitorin g Wel l I D

AP1TMW 1

AP1TMW 1

AP1TMW 2

AP1TMW 3

AP1TMW 3

AP1TMW 7

AP1TMW 7

AP1TMW 7

AP1TMW 7

AP1TMW 8

AP1TMW 9

AP1TMW1 0

AP2TMW 1

1

Date

01/1 7/92

01/17/92

01/15/92

01/15/9 2

01/15/92

01/17/92

01/17/92

01/21/9 2

01/21/9 2

01/17/92

01/15/9 2

01/17/9 2

01/15/9 2

MONITORIN G WEL L

Initial Water Level 1

(ft )

12.27

12.27

2.3 Frozen

2.72

2.72

61.83

61.83

61.85

61.85

Dry

20.04

Dry

Dry

Screened Interval 1

(ft )

15.0- 25.0

15.0- 25.0

11.0- 16.0

15.0 - 25.0

15.0-25.0

64.45 - 74.45

64.45-74.45

64.45 - 74.45

64.45 - 74.45

22.5 - 37.5

11.3-21.3

11.5-21.5

6.0-9.1

SLU G TEST D/>

Type of Slu g Tes t

Fallin g Head

Recovery

N /A

Fallin g Head

Recovery

Fallin g Head

Recovery

Fallin g Head

Recovery

N /A

Recovery

(bai l down)

N / A

N / A

I

i TA SUMMAR Y

mS^—*****immH***£^^SSSSSSSS**»^*^f

Hydraulic Cond. Bouwer & Rice

(Partially 2 ) (cm/sec )

3.5x1 0'

3

3.0x10

3

N /A

1.5x10'

4

1.3x10'

4

3.8x10'

2

4.7x10-

z

8.5x1 0'

2

1.2x10-

N /A

7.2x10'

B

N / A

N / A

— •

Hydraulic Cond. Bouwer & Rice

(Fully 2 ) (cm/sec )

4.5x1 0'

3

3.0x1 0'

3

N /A

2.0x1 O' 4

1.7x10'

4

4.8x10'

2

6.0x1 0

2

1.1x10'

1.5x10"

N /A

N /A

N / A

N / A

1

 : —— ! ——— Hydraulic Cond. Hvorslev

(cm/sec)

7.7x10'

3

7.0x10'

3

N /A

3.0x10'

4

2.8x10"*

9.4x10'

2

1.1x10"

1.7X10' 1

1.7x10

N /A

5.9x1 0'

8

N / A

N / A

1Water levels and screened intervals measured from the top of 2 Partiall y refers to the screen partiall y penetrates the saturated

P VC casing.

zone, while Fully refers to the screen fully penetrates the saturated zone.
~£>w SS& s

=

'^^^^'*KSWSi+

s&S^'Sag^tsa^Ss;'-- :

~^^^"~"~-

^^i^^iga^s^-^^^" ^ • -"rSKE^-rf^E1

OREGON GULCH TAILING S IMPOUNDMEN T

SiSS^^^.'-^S^*3:;?^-^

1

^^^^^^^*^

^P^^^^^^S^L^^eS^^^-e^""^
iS^SSSSSSSSSS^SSSm

Monitoring Wel l ID

OG1TMW 1

OG1TMW 2

OG1TMW 3

OG1TMW 4

OG1TMW 5

OG1TMW6A

OG1TMW 7

OG1TMW 8

"-

Development Date

10/2/91

9/26/91

9/25/9 1

10/1/91

10/2-3/91

10/3/91

10/1/9 1

9/26/91

i

MONITORIN G WEL L DEVELOPMEN T DATA

Development Method

2" Bailer N /A

4" Pump

2" Pump

2" Bailer

4" Pump

N / A

4" Pump

Initial Water Level From T op of PVC

(ft )

: 185.10

Dry

8.89

25.76

22.72

57.67

Dry ;

5.60

Volume Purged

(gal )

29.0

N / A

81,4

92.0

28

87.9

N / A

103.0

Casing Volumes Purged

10.1

N /A

10.4

10.7

10,0

10.3

N /A ,

10.4

Pumpin g Time

(min)

16 days

N /A

517

18 days

2 days

300

N / A

186

SUMMAR Y

•••••••^•JSiSSaaa*

Estimated Productio n

(gpm )

<0.01

N /A

0.16

0.02

0.06

0.3

N / A

0.55

iSStSSSSSSSSSSSSm

P H (etd units)

7.75

N /A

2.25

4.02

4.89

4.72

N / A

3.06

*M M~*^M"*^^^"''**M^

Specifi c Conductivity 1

(pmhos/cm)

324

N /A

1 6,900

2,740

5,389

5,273

N / A

19,800

_____^____— _

Temperature (°C )

8.1

N /A

11.9

8.3

9.0

14.0

N / A

13.7

'The reported pH, temperature, an d specifi c conductivity values were measured at the conclusion of well development .
Monitoring Wel l ID

OG1TMW 1

OG1TMW 3

OG1TMW 4

OG1TMW 5

OG1TMW6 A

OG1TMW 8

GROUNDWATE R QUALIT Y

Sample ID

L-OG1TMW1-03-911030

L-OG 1 TMW 1 -04-9 1 1 030

L-OG1TMW1-01-911031

L-OG1TMW3-01-911031

L-OG 1 TM W4-0 1 -9 1 1 030

L-OG1TMW5-01-913030

L-OG 1 TMW5-02-9 1 3030

L-OG 1 TMW5-MS-9 1 3030

L-OG1TMW6-01-911031

L-OG 1 TM W6-02-9 1 1 03 1

L-OG1TMW6-MS-911031

L-OG 1TMW8-01 -911 031

SAMPLE S COLLECTED

Sample Date

10/30/91

10/30/91

10/31/91

10/31/91

10/30/91

10/30/91

1.0730/91

10/30/91

10/31/91

10/31/91

10/31/91

10/31/91

Sample Time

1200

1220

1350

1530

1630

1530

1530

1530

1130

1130

1130

1945

Type of Sample

Field Blank

Equipment Rinsate

Primary

Primary

Primary

Primary

Duplicate

Matrix Spike

Primary

Duplicate

Matrix Spike

Primary
Monitorin g Wel l ID

OG1TMW 1

OG1TMW 2

OG1TMW 3

OG1TMW 4

OG1TMW 5

OG1TMW6 A

OGITMW 7

OG1TMW 8

Sampl e Date

10/31/91

Dry

10/31/9 1

10/30/91

10/30/91

10/31/91

Dry

10/31/91

t

HI*^^MHII I I •• •!. ——^~*^~m—

Samplin g Method

Disposable Bailer

N /A

Disposable Bailer

Disposable Bailer

Disposable Bailer

Disposable Bailer

N /A

Disposable Bailer

MONITORING V\ ^=

Initial Water Level From T op of PVC

(ft )

182.76

• -" N /A

7.91

27. 05

22.89

58.24

N /A

5.84

/EL L SAMP l ===•=====

Volume Purged (gal )

8.3

N /A

30.8

17.5

10.0

29.1

N /A

32.2

I

.ING DATA ! • " —

Casin g Volume s Purged

1.9

N /A

3.0

2.1

2.5

3.3

N / A

3.2

SUMMAR Y — _

P H 1

(std. units)

7.72

N /A

2.15

4.14

5.22

5.10

N / A

4.06

Specifi c Conductance1

U/mhos/cm )

291

N /A

16,500

24,900

12,450

17,700

N /A

20,700

— —

Temperature 1

(°C )

5.5

N /A

9.9

-0.9

7.0

9.3

N /A

9.2

1 T he reported pH, specifi c conductivit y and temperature were measured at the time of sample collection .
Monitoring Wel l ID

OG1TMW 1

OG1TMW 2

MONITORIN

Measurement Date

10/30/91

01 /1 6/92

02/19/92

03/1 6/92

04/13/92

05/18/92

06/1 5/92

07/20/92

08/17/92

09/21/92

10/26/92

09/12/91

09/20/91

09/26/91

02/19/92

03/1 6/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

1 0/26/92

G WELL WATER

Measurement Time

1324

0950

1350

1334

0837

1408

1600

1311

1440

1525

1218

0830

1332

1415

1500

1305

0814

1317

1500

1215

1226

1512

1656

LEVEL SUMMAR Y ^*^^^^^g<^^^**^^^"^"TM<

^^

Depth to Water1

(ft)

182.76

182.33

182.54

182.89

183.50

183.95

183.79

183.08

181.84

180.29

178.83

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

».^___»_____^»_ a

Ground water

Elevation

(ft )

10101.29

10101.72

10101.51

10101.16

10100.55

10100.10

10100.26

10100.97

10102.21

10103.76

10105.22

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY
MONITORING WELL WATER

Monitoring Wel l ID

OG1TMW 3

OG1TMW 4

Measurement Date

09/25/91

10/31/91

01/17/92

02/19/92

03/1 6/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

10/01/91

10/30/91

01/16/9 2

• • : 02/19/92

03/1 6/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

Measurement Time

0915

1350

0925

1510

1255

0820

1309

1450

1200

1430

1503

1704

0930

1138

1055

1420

1320

0755

1348

1642

1250

1455

1552

1616

LEVEL SUMMAR Y ^^ M^»^M^^^^^»»55

Depth to Water1

(ft)

8.89

7.91

8.70

8.46

6.74

4.64

6.18

8.71

8.88

7.83

7.95

8.65

25.76

27.05

27.97

27.92

28.34

28.27

26.90

26.22

26.32

25.87

24.80

24.63

""

Groundwater Bevation

(ft )

10073.04

10074.02

10073.23

10073.47

10075.19

10077.29

10075.75

10073.22

10073.05

10074.10

10073.98

10073.28

10218.83

10217.54

10216.62

10216.67

10216.25

10216.32

10217.69

10218.37

10218.27

10218.72

10219.79

10219.96
• ! • •• Monitoring Well ID

OG1TMW 5

OG1TMW6A

MONITORIN

Measurement Date

10/02/91

10/30/91

01/17/92

02/19/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

10/03/91

10/31/91

01/16/92

02/19/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

G WELL WATER •••^"•••"•••••^•'"•••^—i

Measurement Time

1040

1209

1023

1435

1315

0802

1355

1633

1246

1458

1543

1610

1055

0925

1 232

1430

1329

0748

1337

1620

1240

1450

1603

1630

LEVEL SUMMAR Y ___ ,^___

Depth to Water1

(ft)

22.72

22.89

23.53

23.96

24.21

24.56

23.74

22.78

22.81

22.72

22.60

22.39

57.67

58.24

58.29

58.38

58.45

58.39

57.94

57.68

57.91

57.93

57.89

57.78

Groundwater

Elevation

(ft)

10223.10

10222.93

10222.29

10221.86

10221.61

10221.26

10222.08

10223.04

10223.01

10223.10

10223.22

10223.43

10195.69

10195.12

10195.07

10194.98

10194.91

10194.97

10195.42

10195.68

10195.45

10195.43

10195.47

10195.58
MONITORIN G WELL WATER

Monitoring Wel l ID

OG1TMW 7

OG1TMW 8

Measurement Date

09/12/91

10/01/91

02/1 9/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

09/26/91

10/31/91

01/16/9 2

02/19/92

03/16/92

04/13/92

05/18/92

06/15/92

07/20/92

08/17/92

09/21/92

10/26/92

Measurement Time

0922

0845

1405

1324

0752

1343

1650

1255

1500

1558

1623

1015

1810

1623

1455

1310

0810

1324

1535

1226

1507

1524

1650

LEVEL SUMMARY

Depth to Water1

(ft)

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

5.60

5.84

5.76

5.63

5.63

5.61

5.44

5.24

5.46

5.32

5.27

5.27

Groundwater

Elevation

(ft)

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

. 10162.08

10161.84

10161.92

10162.05

10162.05

10162.07

10162.24

10162.44

10162.22

10162.36

10162.41

10162.41

'Water levels measured from north side of the top of PVC casing.
vi^*m*m*iHmMHm^SSSSiSSS££m Monitorin g Wel l ID

OG1TMW 1

OG1TMW 2

OG1TMW 3

OG1TMW 3

OG1TMW 4

OG1TMW 5

OG1TMW6A

OG1TMW 7

OG1TMW 8

OG1TMW 8

•^•^••••^••^•••••••••••••J

Date

01/16/9 2

N / A

01/17/92

01/17/9 2

01/16/9 2

01/16/9 2

01/16/9 2

N / A

01/16/9 2

01/16/9 2

MONITORIN G WEL L SLU G TES T DAT A SUMMAR Y

Initial Water Level 1

(ft )

182.33

N / A

8.70

8.70

27.97

23.53

58.29

N / A

5.76

5.76

Screened Interval 1

(ft )

179.0 - 189.0

15.7-25.7

10.7 -20.7

10.7-20.7

16.1 -36.1

64.45 - 74.45

50.5 - 70.5

16.0-36.0

10.7 -20.7

10.7 - 20.7

Typ e of Slu g Tes t

Recovery

N /A

Fallin g Head

Recovery

Recovery

(bai l down)

Recovery

(bail down)

/ Recovery N /A

Fallin g Head

Recovery

Hydrauli c Cond. Bouwer & Rice

(Partially 2 )

(cm/sec )

~

N /A

8.0x1 0'

6

1.5x10-"

--

••-..--

-- N /A

3.5x1 O' 4

3.1x10-"

Hydrauli c Cond. Bouwer & Rice

(Fully 2 )

(cm/sec )

1.3x10-

N / A

1.0x10'

4

1.9x10'

4

4.8x10'

e

5.8x1 0

s

1.2x10

3

N /A

4.5x1 q*

4.0x1 0'

4

Hydrauli c Cond. Hvorslev (cm/sec )

4.3x1 0'

6

N /A

2.1x10'

4

3.7x10'

4

2.2x1 0-"

4.8x10

6

6.7x1 0'

3

N /A

7.6x1 0-

4

7.0x10-*

'Water levels and screened intervals were measured from the top of PVC casing. 2Partiall y refers to the screen partiall y penetratin g the saturated zone, while Fully refers to the screen fully penetratin g the saturated zone.
MALTA TAILING S IMPOUNDMEN T

•*^•~^*£v^-£3£&-^&£Z-:?'-~ "-•
Monitorin g Wel l ID

MA1TMW 1

MA1TMW 2

MA1TMW 3

MA1TMW 4

MA1TMW 7

Developmen t Date

11/19/9 1

10/18/91

10/10/91

10/09/91

10/10/91

=

Developmen t

Method

2" Pump

4" Pump

4" Pump

4" Pump

N /A

MONITORIN G-~

Initial Water Level From T op of PVC

(ft )

] 160.32

66.65

74.92

75.69

Dry

WEL L DEV •**••—•

Volume Purged

(gal )

132.0

179.5

132.1

70.9

N /A

ELOPMEN

,,.—^—

Casin g Volumes Purged

10.0

19.2

10.3

9.8

N /A

I

T DATA SUMMAR Y ——~— —— ••

Pumpin g Tim e

(min.)

200.0

78.0

22.0

89.0

N /A

Estimated Productio n

(gpm )

0.66

2.30

6.00

0.80

N /A

.

p H (etd unite )

6.80

7.00

6.97

7.38

N /A

••••••••••••••••••••••S^Sj

Specifi c Conductivity 1

(^mhos/cm)

793

1067

674

733

N /A

=

Temperature 1

<°C )

6.8

6.5

6.3

6.2

N /A

'The reported pH, temperature, an d specifi c conductivit y values were measured at the conclusion of well development .
Monitoring Wel l ID

MA1TMW 4

MA1TMW 3

MA1TMW 2

MA1TMW 1

GROUNDWATER QUALIT Y S

'^^^i^^^Z^^S^^^m^^HmKi^^ii^^^mi^^Smm

Sample ID

L-MA1 TMW04-01 -91 1 1 20

L-MA1TMW04-02-91 1 1 20

L-MA1TMW04-MS-91 1 1 20

L-MA1TMW03-01 -91 1 1 20

L-MA1TMW02-01-911120

L-MA1TMW01-01-911120

AMPLES COLl

==

Sample Date

11/20/91

11/20/91

11/20/91

11/20/91

11/20/91

1 1 /2.0/91

.ECTED

Sample Time

1030

1030

1030

1300

1430

1630

=

Type of Sample

Primary

Duplicate

Matrix Spike

Primary

Primary

Primary

QA/QC Samples

CZ1TMW 6

AP1TMW 3

L-CZ1TMW06-03-911116

L-AP1TMW03-04-91 1121

11/16/9 1

11/21/9 1

1840

1500

Trip Blank

Rinsate
1 MONITORIN G WELL SAMPLIN G DATA SUMMAR Y

Monitorin g Wel l ID

MA1TMW 1

MA1TMW 2

MA1TMW 3

MA1TMW 4

MA1TMW 7

Sampl e Date

11/20/91

11/20/91

11/20/9 1

11/20/91

Dry

Samplin g Method

2" SS Pump

2" SS Pump

2" SS Pump

2" SS Pump

N /A

Initial Water Level From T op of PVC

(ft )

150.05

66.64

74J91

75.65

Dry

Volum e Purged

(gal )

41.0

28.0

40.0

37.0

N/A /

Casin g Volume s Purged

3.1

3.0

3.1

5.1

N /A

P H 1

(std. units)

7.01

6.77

6.87

7.05

N /A

Specifi c Conductivity 1

(yumhos/cm)

693

951

763

777

N /A

Temperature 1

(°C )

6.6

6.2

6.7

6.6

N /A

1T he reported pH, specific conductivity and temperature were measured at the time of sample collection.
^ —— ——— •

Monitoring Wel l ID

MA1TMW 1

MA1TMW 2

MONITORIN G

Measurement Date

11/19/9 1

11/20/91

01/15/92

02/19/92

03/17/92

04/13/92

05/18/92

06/1 7/92

07/21/92

08/17/92

09/21/92

10/26/92

10/18/91

01/14/92

02/19/92

03/17/92

04/13/92

05/18/92

06/16/92

07/20/92

08/17/92

09/21/92

10/26/92

WELL WATER LE

SSTflTSI^^'^^TffT^^TT^^^^ Measurement Time

1030

1547

1200

1610

1437

1443

1110

1030

843

1735

1450

1330

1145

1530

1715

1429

1500

1224

1415

1735

1705

1440

1350

V EL SUMMAR Y ^"*""TM"*"*""""""p

^"*^"^"""^

Depth to Water

(ft)

150.32

150.05

150.30

150.46

150.75

151.27

151.85

152.23

152.50

152.65

152.85

152.98

66.65

66.70

66.86

67.03

67.29

67.28

67.73

68.14

68.43

68.81

69.13

—

Groundwater

Elevation

(ft )

9734.62

9734.89

9734.64

9734.48

9734.19

9733.67

9733.09

9732.71

9732.44

9732.29

9732.09

9731.96

9717.02

9716.97

9716.81

9716.64

9716.38

9716.39

9715.94

9715.53

9715.24

9714.86

9714.54
— Monitoring Well ID

MA1TMW 3

MA1TMW 4

MONITORIN G •^B*"^^^^^TM1

^^*^TM'**^^"^ Measurement Date

10/10/91

11/20/91

11/26/91

01/15/92

02/19/92

03/1 7/92

04/13/92

05/18/92

06/16/92

07/20/92

08/17/92

09/21/92

10/26/92

10/09/91

11/20/91

11/26/9 1

01/15/92

02/19/92

03/17/92

04/13/92

05/18/92

06/16/92

07/20/92

08/17/92

09/21/92

10/26/92

WELL WATER LE

^^^^^^ _ —^ — Measurement Time

1529

1210

0857

1343

1705

1425

1456

1207

1406

1727

1 700

1433

1347

1714

0930

0833

1420

1700

1421

1452

1204

1355

1721

1655

1430

1345

V EL SUMMAR Y .. • - -ii ii • «r-n I--M. Depth to Water

(ft)

74.92

74.91

74.92

75.14

75.15

75.28

75.58

75.63

75.96

76.28

76.53

76.91

77.13

75.69

75.65

75.59

75.79

75.80

75.92

76.23

76.39

76.74

77.06

77.31

77.63

77.79

sss

Groundwater

Elevation

(ft)

9714.86

9714.87

9714.86

9714.64

9714.63

9714.50

9714.20

9714.15

9713.82

9713.50

9713.25

9712.87

9712.65

9720.32

9720.36

9720.42

9720.22

9720.21

9720.09

9719.78

9719.62

9719.27

9718.95

9718.70

9718.38

9718.22
=

Monitoring Well ID

MA1TMW 7

MONITORIN G

^^^^^STSJT^^^i — i -i Measurement Date

09/20/91

11/26/91

02/19/92

03/1 7/92

04/13/92

05/18/92

06/16/92

07/20/92

08/17/92

09/21/92

10/26/92

i WELL1 WATER LE

Measurement Time

1100

0845

1655

1417

1504

1157

1345

1717

1645

1425

1340

V EL SUMMAR Y

Depth to Water

(ft)

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

=

Grouridwater

Elevation

(ft>

DRY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

D RY

DRY

DRY

'Water levels measured from the north side of the top of PVC casing.
1 MONITORIN G WEL L SLU G TES T DAT A SUMMAR Y — - Monitorin g Wel l ID

MA1TMW 1

MA1TMW 1

MA1TMW 2

MA1TMW 3

MA1TMW 3

MA1TMW 4

._—— I I .

Date

01/15/9 2

01/15/9 2

01/14/9 2

01/15/9 2

01/15/9 2

01/15/9 2

Initial Water Level 1

(ft )

1 50.3

150.3

66.70

75.14

75.15 i

75.79

Screened Interval (ft )

151.9 - 166.9

151.9 - 166.9

61.5- 76.5

81.05- 91.05

81.05 - 91.05

71.32 - 86.32

Type of Slug Tes t

Fallin g Head

Recovery

Recovery

Fallin g Head

Recovery

Recovery

Hydraulic Cond. Bouwer & Rice

(Partially 2 )

(cm/sec )

1.1x10'

2

1.2x10'

2

1.6x10'

2

5.8x1 0'

3

4.9x1 0'

3

4.1x10'

3

Hydraulic Cond. Bouwer & Rice

(Fully 2 )

(cm/sec)

1.3x10'

2

1.4x10'

2

2.0x1 0'*

7.4x1 0'

3

6.4x1 0'

3

5.1x10'

3

Hydraulic Cond. Hvorslev (cm/sec)

2.1 x10

2

2.2x1 0'

2

1.4x10' *

1.1x10'

2

9.8x10'

a

3.6x1 0-

3

1

 Water levels and screened intervals were measured from the top of PVC casing. 2Partiall y refers to the screen partiall y penetratin g the saturated zone, while Fully refers to the screen fully penetratin g the saturated zone.
8Spbs:«v-;

SgS£-<-&.--

S§S5'>^:- -

COLORADO ZIN C LEAD TAILING S IMPOUNDMEN T
1 MONITORIN G WELL DEVELOPMEN T DATA SUMMAR Y

Monitorin g Wel l ID

CZ1TMW 1

C21TMW 4

CZ1TMW 6

CZ1TMW7A

CZ1TMW 8

Developmen t Date

11/19/9 1

10/17/9 1

10/17/9 1

10/24/91

10/23/91

Developmen t

Method

2" Bailer

2" Bailer

4" Pump

4" Pump

4" Pump

Initial Water Level From T op of PVC

(ft )

i 5,51

12.30

3.55

4.90

3.60

Volume Purged

(gal )

104.5

13.0

119.8

103.0

306.2

Casin g Volumes Purged

10.77

10.48

10.17

18.73

39.47

Pumpin g Time

(min)

816.0

312.0

205.0

147.8

474.0

Estimated Productio n

(gpm )

0.13

0.04

0.58

0.70

0.64

P H (std units)

6.70

5.43

5.80

3.01

4.79

Specifi c Conductivity 1

(jumhos/cm)

1238

1236

1095

1801

1636

Temperature 1

(°C )

5.8

10.6

8.4

11.5

9.6

'The reported pH, specifi c conductivity , an d temperature values Were measured at the conclusion of well development .
SSSSSSSSS^SSSSSS^SiiS

Monitorin g Wel l ID

CZ1TMW 1

CZ1TMW 4

CZ1TMW 6

CZ1TMW7 A

CZ1TMW 8

———— — •

Sample Date

11/19/9 1

11/18/9 1

11/16/9 1

11/18/9 1

11/19/9 1

- -••—-- N •MMWWWHM^MMB^PVHMM

Samplin g Method

Disposable Bailer

Disposable Bailer

Disposable Bailer

Disposable Bailer

Disposable Bailer

I

IONITORIN G WELL SAMPLIN G DATA SUMMAR Y

Initial Water Level From T op of PVC

(ft )

5.41

12.21

3.57

4.55

3.51

Volume Purged (gal )

25.8

6.0

36.0

20.0

28.0

Casin g Volume Purged

3.0

4.6

3.1

3.5

3.6

p H 1

(std. units)

6.13

4.85

5.90

2.86

5.17

Specifi c Conductivity 1

(//mhos/cm )

1292

1206

1028

2242

3040

Temperature 1

(°C )

5.9

6.5

5.5

5.0

5.0

1T he reported pH, specific conductivity and temperature were measured at the time of sample collection.
Monitorin g Wel l ID

CZ1TMW 1

CZ1TMW 4

CZ1TMW 6

CZ1TMW7A

CZ1TMW 8

GROUNDWATE R QUALIT YSampl e I D

L-CZ1TM W0 1-0 1-9111 19

L-CZ1TMW04-0 1-911 118

L-CZ1TMW06-01-91111 6

L-CZ1TMW06-02-91 1116

L-CZ1TMW06-MS-9 1 1116

L-CZ1TMW06-04-911116

L-CZ1TMW06-03-911116

L-CZ1TMW7A-01-91111 8

L-CZ1TMW7A-02-91111 8

L-CZ1TMW7A-MS-9 1 1 1 1 8

L-CZ1TMW08-01-91111 9

L-CZ1TMW08-02-91 1 1 19

L-CZ1TMW08-MS-9 1 1 1 1 9

SAMPLES C ^^___-^__ss:

Sample Date

11/19/9 1

11/18/9 1

11/16/9 1

11/16/9 1

11/16/9 1

11/16/9 1

11/16/9 1

11/18/9 1

11/18/9 1

11/18/9 1

11719/91

11/19/9 1

11/19/9 1

OLLECTED

Sampl e Time

1330

1045

1600

1600

1600

1 800

1840

1330

1330

1330

1545

1 545

1545

•MMMmBWMW^BBVWB^^H^M^^MHMVm

Type of Sampl e

Primary

Primary

Primary

Duplicate

Matrix Spike

Equipment Rinsate

Field Blank

Primary

Duplicate

Matrix Spik e

Primary

Duplicate

Matrix Spike
MONITORING WELL WATER LEVEL SUMMAR Y

Monitoring Wel l ID

CZ1TMW 1

CZ1TMW 4

Measurement Date

11/06/91

11/19/91

01/20/92

02/19/92

03/17/92

04/13/92

05/18/92

06/16/92

07/21/92

08/17/92

09/21/92

10/26/92

10/11/9 1

10/17/91

11/17/9 1

01/17/92

01/20/92

02/19/92

03/17/92

04/13/92

05/18/92

06/16/92

07/21/92

08/17/92

09/21/92

10/26/92

Measurement Time

1634

1005

1232

1810

1354

1432

1147

1505

918

1612

1712

1507

1722

0840

1155

0941

1 237

1750

1346

1423

1135

1450

905

1628

1700

1530

Depth to Water1

(ft)

5.51

5.41

5.32

5.51

5.56

4.96

5.43

5.50

5.28

5.17

5.37

5.26

12.38

12.30

12.21

11.94

12.06

12.42

12.32

11.54

12.02

12.14

12.15

12.03

12.21

12.40

Groundwater

Elevation

(ft)

9833.32

9833.42

9833.51

9833.32

9833.27

9833.87

9833.40

9833.33

9833.55

9833.66

9833.46

9833.57

9820.43

9820.51

9820.60

9820.87

9820.75

9820.39

9820.49

9821.27

9820.79

9820.67

9820.66

9820.78

9820.60

9820.41
• =

Monitoring Well ID

CZ1TMW 6

CZ1TMW7 A

MONITORIN G

Measurement Date

10/11/9 1

10/17/91

11/16/91

01/17/92

02/1 9/92

03/17/92

04/13/92

05/1 8/92

06/16/92

07/21/92

08/1 7/92

09/21/92

10/26/92

10/24/91

11/17/9 1

01/17/92

01/20/92

02/19/92

03/17/92

04/13/92

05/18/92

06/1 6/92

07/21/92

08/17/92

09/21/92

10/26/92

WEL L WATER L _— .^____^__^_:

Measurement Time

1726

1450

1047

0941

1755

1349

1426

1138

1457

908

1 634

1705

1540

0955

1500

0937

1558

1740

1341

1417

1120

1430

855

1617

1652

1551

EVEL SUMMAR Y ^^^••^^•••^^^^^•^••••i^^^

Depth to Water1

(ft)

2.50

3.55

3.57

FROZEN

FROZEN

FROZEN

FROZEN

3.67

3.78

3.58

3.41

3.65

3.50

4.90

4.55

4.34

4.58

4.78

4.45

2.69

4.34

4.41

4.71

4.13

4.47

4.61

Ground water

Elevation

(ft )

9823.05

9822.00

9821.98

FROZEN

FROZEN

FROZEN

FROZEN

9821.88

9821.77

9821.97

9822.14

9821.90

9822.05

9812.50

9812.85

9813.06

9812.82

9812.62

9812.95

9814.71

9813.06

9812.99

9812.69

9813.27

9812.93

9812.79
Monitoring Wel l ID

CZ1TMW 8

MONITORIN G _^ ~__«.^__^^ Measurement Date

10/23/91

11/18/9 1

01/17/92

02/19/92

03/1 7/92

04/13/92

05/18/92

06/16/92

07/21/92

08/17/92

09/21/92

10/26/92

WELL WATER L

Measurement Time

0930

1620

0930

1745

1343

1420

1124

1439

900

1 622

1656

1544

EVEL SUMMARY

Depth to Water1

(ft )

3.60

3.46

•» 3.51 Frozen

** 3.40 Frozen

•» 3.35 Frozen

•» 3.25 Frozen

3.52

3.60

3.49

3.70

3.41

3.29

Groundwater

Elevation

(ft)

9817.23

9817.37

Frozen

Frozen

Frozen

Frozen

9817.31

9817.23

9817.34

9817.13

9817.42

9817.54

'Water levels measured from north side of the top of PVC casing.
Monitorin g Wel l ID

CZ1TMW 1

CZ1TMW 1

CZ1TMW 4

CZ1TMW7A

CZ1TMW7 A

Date

01/20/92

01/20/92

01/20/92

01/20/92

01/20/92

M(

**^**>*^^^^^"MI''

i

^^*M^

Initial Water Level1

(ft )

5.32

5.30

12.06

4.58

4.58

DNITORIN1 G WE L

 — ~— Screened Interval 1

(ft )

10.0-20.0

10.0-20.0

6.6 - ir.6

5.2-10.2

5.2-10.2

L SLUG TEST DAT A SUMMARY

Type of Slug Test

Fallin g Head

Recovery

Recovery

Fallin g Head

Recovery

Hydraulic Cond. Bouwer & Rice

(Partially 2) (cm/sec)

N /A

N /A

N / A

N /A

N / A

•SSlS5^MMMiHiM55S««HH«HBHB Hydraulic Cond. Bouwer & Rice

(Fully 2 ) (cm/sec)

1.2x10-*

1.7x10-"

8.6x1 0'

4

2.6x1 0'

3

3.8x10'

3

^"^**^""**TM"M M*

i M^'^***'"TMM

^ Hydraulic Cond. Horslev (cm/sec)

3.5X10"4

2.8x10-*

8.9x1 0'

4

5.0x10-

3

7.9x1 0'

3

1

2 Water level s and Partiall y refers to

screened interval s were measured from the top of the PVC casing.

t he screen partiall y penetratin g the saturated zone, while Fully refers to the screen fully penetratin g the saturated zone.
HAMM' S MIL L TAILING S IMPOUNDMEN T

229OTC-2232t/R22>TS2. 01 -28,93(3-40|)m)/RPT ; ]0
1 MONITORIN G WELL DEVELOPMEN T DATA SUMMAR Y __•__—— ^______ ——— — ^""TM'TM S

Monitorin g Well ID

HM1TMW 4

HM1TMW 5

WMW2

Developmen t Date

12/05/91-

01/06/92

12/05-13/91

11/05/91-

12/11/9 1

Developmen t

Method

2" SS Bailer

& 2" SS Pump

2" SS Bailer

2" SS Bailer

& Disposable Bailer

Initial Water Level From T op of PVC

: (ft )

129.53

106.12

92.53

Volume Removed

(gal)

103

68

28

Casin g Volumes Purged

10.0

10.0

10.0

Pumpin g Time

(min)

1 4 Days

292

9 Days

Estimated Productio n

(gpm )

-0.01

-0.06

-0.07

P H (std units)

6.74

6.40

6.83

Specifi c Conductivity 1

(pmhos/cm)

385

592

1785

Temperature 1

(°C )

5.0

6.2

8.5

'The reportad pH, specifi c conductivity , an d temperature values were measured at the conclusion of well development .
"*^^^^^^^^^"***^*M

**"^TM

Monitorin g Wel l I D

HM1TMW 4

HM1TMW 5

WMW 2

WMW2 2

""•^— •••

Sampl e Date

01/08/92

01/08/92

01/09/92

01/10/92

MONITORIN G WEL L SAMPLIN G DAT A SUMMAR Y

Samplin g Method

2" SS Bailer

2" SS Bailer

2" SS Bailer

2" SS Bailer

Initia l Water Level From T op of PVC

(ft )

128.50

85.28

92.69

93.69

Volume Purged

(gal )

19.2

40.2

6.4

2.2

Casin g Volumes Purged

1.74

2.07

2.39

0.8

P H 1

(std. units)

7.56

6.85

6.61

6.44

Specifi c Conductivity 1

Ot/mhos/cm )

396

559

3050

3185

Temperature 1

(°C )

4.5

5.2

5.1

4.4

1T he reported pH, specific conductivity, and temperature were measured at the time of sample collection. 2T he second samplin g of WMW 2 was performed to collect an EPA split .
•••••^^^••^•••••••^••^^•iMHiH

Monitoring Wel l ID

HM1TMW 4

HM1TMW 5

WMW 2

GROUNDWATE R QUALIT Y

Sample ID

L-HM1TMW4-01-910108

L-HM1TMW4-02-910108

L-HM1TMW4-03-910108

L-HM1TMW4-04-910108

L-HM1TMW4-MS-91010 8

L-HM1TMW5-01-91010 8

L-WMW2-01-920109

L-WMW2-02-9201 09

L-WMW2-MS-920109

SAMPLES C

Sample Date

01/08/92

01/08/92

01/08/92

01/08/92

01/08/92

01/08/92

01/09/92

01/09/92

01 /09/92

OLLECTED

===•====:•

Sample Time

1130

1130

0830

1130

1130

1445

1200

1200

1200

~ " ' • ••i-i" •- i -».

Type of Sample

Primary

Duplicate

Field Blank

Equipment Rinsate

Matrix Spike

Primary

. Primary

Duplicate

Matrix Spike
Monitorin g Wel l ID

HM1TMW 2

-

HM1TMW 3

HM1TMW 4

MONITORING \

Measurement Date

01/06/92

02/1 8/92

03/16/92

04/13/92

05/18/92

06/1 6/92

07/20/92

08/17/92

09/21/92

10/26/92

11/16/9 2

01 /06/92

02/1 8/92

03/16/92

04/13/92

05/18/92

06/16/92

07/20/92

08/17/92

09/21/92

10/26/92

11/17/9 2

12/05/91

01/06/92

01/13/92

01/14/92

02/1 8/92

03/16/92

WEL L WATEI =

Time

1520

1535

1535

0958

1207

0925

1342

1155

1720

1604

945

1511

1550

1523

0957

1638

1534

1335

1 200

1655

1610

935

1030

1124

1330

1240

1555

1527

3 LEVEL SUMMARY

Depth to Water From Top of PVC 1

(ft)

-99.00

-99.00

-99.00

-99.00

-99.00

-99.00

-99.00

-99.00

-99.00

-99.00

-99.00

194.70

194.67

194.73

194.69

194.74

194.70

-99.00

194.67

-99.00

-99.00

-99.00

129.53

128.50

128.67

128.32

128.22

129.21

Ground Water Elevation

(ft )

DRY

D RY

DRY

DRY

DRY

DRY

DRY

DRY

DRY

D RY

DRY

10131.59

10131.62

10131.56

10131.60

10131.55

10131.59

DRY

10131.62

DRY

DRY

DRY

10177.65

10178.68

10178.51

10178.86

10178.96

10177.97
======

Monitorin g Wel l ID

HM1TMW 4

HM1TMW 5

WMW 2

MONITORING \ ""*""""^*"*"*'"P

**^"MTM""^TMP*"

Measurement Date

04/13/92

05/18/92

06/16/92

07/20/92

08/17/92

09/21/92

10/26/92

11/16/9 2

12/05/9 1

01/07/92

01/14/9 2

02/18/92

03/16/92

04/13/92

05/18/92

06/16/92

07/20/92

08/17/92

09/21/92

10/26/92

11/16/9 2

11/25/9 1

01/08/92

01/14/92

02/1 8/92

03/16/92

04/13/92

05/18/92

NELL WATEI

;

Time

0948

1631

1523

1536

1216

1650

1615

1515

1515

0947

1130

1600

1543

0942

1244

1546;

1530

1223

1645

1622

958

1630

1520

0922

1515

1516

1006

1215

=1 LEVEL SUMMARY

^^^^S^^^Sf^^————- '"•^— i^^i-^— . -^^^aa^s^^j —— . ————— —a

Depth to Water From Top of PVC1

<ft)

129.83

130.07

129.94

130.52

130.38

130.68

130.83

130.91

106.12

85.28

88.25

85.54

85.84

86.11

86.09

85.72

85.67

85.39

84.73

84.35

84.10

83.40

92.69

92.90

93.52

94.11

94.75

94.36

=

Ground Water Elevation

(ft)

10177.35

10177.11

10177.24

10176.66

10176.80

10176.50

10176.35

10176.27

10169.18

10190.02

10187.05

10189.76

10189.46

10189.19

10189.21

10189.58

10189.63

10189.91

10190.57

10190.95

10191.20

10434.99

10425.70

10425.49

10424.87

10424.28

10423.64

10424.03
Monitorin g Wel l ID

WMW 2

MONITORING \

•

Measurement Date

06/15/9 2

07/20/92

08/17/92

09/21/92

10/26/92

11/17/9 2

NELL WATE

"*^^**>^^*"^^***'^

Time

1700

1350

1129

1705

1545

1007

=1 LEVEL SUMMARY

Depth to Water From Top of PVC 1

<ft)

95.77

96.38

96.56

-99.00

-99.00

-99.00

===!^==

Ground Water Elevation

(ft)

10422.62

10422.01

10421.83

DRY

DRY

DRY

'Water level s measured in feet , from the north side of the top of PVC casing.
APPENDI X F

TOXICOLOGICA L PROFILES

INTERPRETATION OF ASSESS OUTPUT

ASSES S REPORTS

22909C-22327/R22.F 01-28-93(4:47pm)/RPT'16
APPENDIX F

TABLE OF CONTENT S

Title Page

ANTIMON Y F- l

ARSENIC F-4

BARIUM F-9

BERYLLIUM F

CADMIUM F-

CHROMIUM F

COPPER F-

LEAD F-

MANGANES E , "•, . F-

MERCURY F

ZINC F-

INTERPRETATION OF ASSESS REPORTS F-

ASSESS REPORTS F-

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 -1-
ANTIMON Y

Antimony is often used in association with lead and arsenic in industry. The major uses

of this metal include the manufacture of lead alloys, storage batteries, pewter, rubber,

matches, ceramics, enamels, paints, lacquers, and textiles (Goyer 1986). Antimony forms

a volatile oxide in reaction with steam, nitric acid, or strongly oxidizing salts. When

heated, antimony reacts with halogens (e.g., chlorine or bromine). Oxides, sulfides , and

chlorides of antimony are the most common forms found in industry (Clayton and

Clayton 1981).

Most antimony exposures occur via ingestion or inhalation. Because antimony is usually

found in association with lead and arsenic, it is difficul t to assess the toxicity of antimony

alone, particularly in humans (Sax 1986). Humans exposed to high levels of antimony

dusts may experience gastrointestinal problems and inflammation of the mucous

membranes of the nose and throat. In addition, antimony inhalation may result in

effect s on the nervous system such as irritability, sleeplessness, fatigue, dizziness, and

muscular and neurological pain. Antimony compounds may also be irritating to the skin

upon direct contact (Sax 1986). Animals exposed to the fumes of antimony oxide have

developed pneumonitis, fatty degeneration of the liver, a decreased white blood cell

count, and damage to the heart muscle.

The subchronic and chronic oral RfD for antimony is 4 x 10"4 mg/kg/da y as determined

from a two year study in rats (Schroeder et al. 1970). An inhalation RfC has not yet

been determined.

Studie s suggest that chronic occupational exposure to antimony compounds results in

deleterious nervous system effects . These effect s are very difficul t to attribute to

antimony alone because of the uncertainties noted above. Nonetheless , myocardial

effect s followin g chronic exposure to antimony among factory workers have been

conclusively documented. Individual s exposed to antimony trisulfide at concentrations

ranging from 0.58 to 5.5 mg/m3 exhibited ECG changes and elevated blood pressure

(Carson 1986).

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-l
Stokinger (1981) reported that female workers exposed to antimony experienced a higher

than normal incidence of spontaneous abortions late in the pregnancy, premature births,

a nd depressed infant weight gain. Animal studies failed to reproduce these findings

(Stokinger 1981).

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-2
REFERENCES

Carson, B.L., H.V. Ellis., and J.L. McCann, eds. 1986. Toxicology and Biological

Monitoring of Metals in Humans. Lewis Publishers, Inc. Michigan.

Clayton and Clayton (eds.). 1981. Patty's Industrial Hygien e and Toxicology. Third

Edition (revised). Volume 2B. Wiley-Interscience. New York, New York. Vol.

2A: 1492-2060.

Goyer, R.A. 1986. Toxic Effect s of Metals. In: Casarett and Doull's Toxicology: The

Basic Science of Poisons. 3rd Ed. Edited by C.D. Klaassen , M.O. Amdur, and

J. Doull. MacMillan . New York.

Sax, N.I. 1986. Dangerous Properties of Industrial Materials. Van Nostran d Reinhold. N ew York.

Schroeder, H.A., M. Mitchener and A.P. Nason. 1970. "Zirconium, niobium, antimony

a nd lead in rats: Life-time studies." J. Nutr. 100:56-69.

Stokinger, H.E. 1981. The Metals. In : Patty' s Industrial Hygien e an d Toxicology .

Edited by C.D. Clayton and F.E. Clayton. Wiley-Interscience. New York, New

York. Vol. 2A: 1492-2060.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-3
ARSENI C

Arsenic, like all elements, is an ubiquitous metal found throughout the environment in

relatively low concentrations. There are a number of medicinal, agricultural, and

industrial uses for arsenic compounds. Arsenic has also been used extensively in

medicine (Fowler's Solution ) for the treatment of leukemia, psoriasis, asthma, as a tonic,

and in the formulation of anti-parasitic drugs. Medicinal dosages were frequently as high

as 3 mg/day , and at least one study has reported aplastic anemia among users of

arsenical drugs (Westhof f et al. 1975). In recent years, with the development of less

toxic drugs, the medicinal use of arsenic has declined (shinishi et al. 1986).

Industrial exposure to arsenic compounds is predominantly through inhalation and

ingestion. About four-fifths of absorbed arsenic is widely distributed in the tissues,

includin g the liver, abdominal viscera, bone, skin, and particularly hair and nails. It can

be detected in these tissues in concentrations over and above those resulting from a

normal diet, many months after it has disappeared from the urine and feces. It is

important to note that industrial arsenic exposures primarily involve the trivalent form,

which is considered to be somewhat more toxic than the pentavalent form (Stokinger

1981). The relatively soluble arsenic trioxide has a fatal oral dose of 70 to 180 mg

(Valleee t al. 1960).

The acute effect s associated with both oral and respiratory arsenic exposures in humans

have been investigated. Inhalation of irritant and vesicant arsenicals such as arsenic

trioxide and arsenic trichloride can cause severe damage to the respiratory system as well

as coughing, dyspnea, and chest pains (Ishinishi et al. 1986). Numerous incidences of

acute poisoning (accidental and suicidal) via arsenic ingestion have been reported. One

incident involved approximately 12,000 Japanese infants who were exposed to an infant

formula tainted with pentavalent inorganic arsenic (1.3 to 3.6 mg/day). Among the

exposed population , approximately 1 percent died of acute poisoning, and the majority

of the survivors exhibited one or more of the followin g symptoms: fever, insomnia,

anorexia, liver swelling, melanosis, and disturbed heart functionin g (WH O 1981). Accidental ingestion has also been reported in adults who have been exposed for 2 to

3 weeks to tainted soy sauce. Symptoms included facial edema, anorexia, skin lesions,

a nd liver swellin g (Mizuta et al. 1956). Individual s recovering from poisoning by

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-4
inorganic arsenic exhibited disturbances of the peripheral nervous system with some

axonal degeneration (WHO 1981).

Chronic inhalation and ingestion of arsenic by humans has been reported in a number

of epidemiological studies. Kurtasune (1972) reported that population s neighboring on

an arsenic trioxide refinery exhibited skin lesions and peripheral neuropathy with some

increases in chronic respiratory disease, although a causal relationship with arsenic was

never established. It has been theorized that arsenic tends to accumulate in the skin

because it contains high concentrations of sulfhydry l proteins, which bind arsenic.

Several studies have described the effect s of ingesting water from regions with high

naturally occurring (background) levels of arsenic. Hyperkeratotic skin lesions were seen

in population s in Chile (Borgono et al. 1977) and Taiwan (Tsen g 1977). In addition, a

condition known as blackfoot disease, which is characterized by gangrene of the lower

extremities, was reported in the Taiwanese population. Skin lesions, which occur

primarily on the palm of the hand and the sole of the foot , have been reported followin g

occupational exposures (Hamada arid Horiguchi 1976), from therapeutic administration

of Fowler's Solutio n (Fierz 1965), and from the ingestion of naturally contaminated

drinking water. Other chronic effect s include melanosis of the eyelids, around the

temples, nipples , and folds of the axillae, and the formation of white lines on the

fingernails. Inhalation of arsenic dust caused perforation of the nasal septum (Ishinishi

etal . 1986).

An epidemiological study of female workers at a copper smelter showed a birth defect

rate five times that of the control population , but no conclusion about the role of arsenic

could be drawn because of the simultaneous exposure to other metals and sulfur dioxide

(Nordstrom et al. 1979). Arsenic-induced terata have been noted in hamsters exposed

to high doses of sodium arsenate (6 to 10 mg/kg ) on the eighth day of pregnancy. Defects included anencephaly, renal agenesis, and rib malformations. An increase in

fetal resorption was also noted (Perm 1977). Co-administration of sodium selenite

reportedly impairs arsenic-induced teratogenesis in animals (Holmberg and Perm 1969).

Chromosomal abnormalities have been observed in the lymphocytes of workers and

patients exposed to arsenic (Beckman et al. 1977; Petres et al. 1977). In vitro studies

have shown that only trivalent arsenic compounds induced sister chromatid exchanges

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-5
(SCEs) . The relevance of SCE studies is questionable since lymphocytes from

chronically exposed individuals sufferin g from blackfoot disease showed no difference s

in SCE patterns when compared to control population s (Wen et al. 1981). Mutagenicity

tests for both trivalent and pentavalent arsenic compounds were negative in Salmonella.

E. coli. and Chinese hamster V9 assays, but positive in Bacillus subtitles (Ishinishi et al.

1986).

Skin cancer, in the form of epithelioma, has been seen at the sites of arsenic-induced

keratoses (Borgono et al. 1977; Tsen g et al. 1977). An increased incidence of lung

cancer has been reported among arsenic smelter workers, although it should be noted

that these workers were also exposed to sulfur dioxide and other metals (Ishinishi et al.

1986). No association between arsenic exposure and other forms of cancer has been

established (IARC 1980). The United States Environmental Protection Agency

(USEPA) has classified arsenic as a Clas s A carcinogen based on epidemiological data.

22909C-22327/R22.F 01-28-93{4:26pm)/RPT/16 F-6
REFERENCES

Beckman, G., L. Beckman, and T. Nordenson. 1977. Environmental Health

Perspectives. 19:145-146.

Borgono, J.M. , P. Vincent, H. Venturino, and A. Infante. 1977. Environmental Health

Perspectives. 19:103-105.

Perm, V.H. 1977. Environmental Health Perspectives. 19:215-217.

Fierz, U. 1965. Dermatologica. 131:41-58.

Hamada, T. and S. Horiguchi. 1976. Japanese Journal of Industrial Health. 19:103-115.

Holmberg, R.E. and V.H. Perm. 1969. Archives of Environmental Health. 19:873-877.

IARC Monographs. 1980. Evaluation of the Carcinogenic Risk of Chemicals to

Humans. Some Metals and Metallic Compounds. Vol . 23. International Agency

for Research on Cancer. Lyon.

Ishinishi , N., K. Ksuchiya, M. Vahter, and B.A. Fowler. 1986. (In : Handbook on the

Toxicology of Metals.) 2nd ed. Vol . 2 Edited by L. Fribirg, G.F. Nordberg, and

V.B. Vouk. Elsevier, New York.

Kuratsune, M. 1972. Japanese Public Health Association, Report on Environmental

Health. 13:3-8.

Mizuta, N., M. Mizuta, F. Ito , T. Ito , H. Uchida, H. Yakama, T. Murakami, F. Hayashi , K. Nakamura, T. Yamaguchi, W. Mizuta, S. Oishi, and H. Matsurimura.

1956. Bulletin of Yamaguchi Medical School . 4:131-150.

Nordstrom, S., L. Beckman, and I. Nordenson. 1979. Hereditas . 90:297-302.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-7
Petres, J., D. Baron, and M. Hagedorn. 1977. Environmental Health Perspectives.

19:223-227.

Stokinger, H.E. 1981. The Metals. (In : Patty' s Industrial Hygien e an d Toxicology.)

Edited by C.D. Clayton and F.E. Clayton. Vol . 2A: 1492-2060.

Tseng, W.P. 1977. Environmental Health Perspectives. 19:109-119.

Vallee , B.L., D.D. Uliuer, and W.E.C. Wacker. 1960. American Medical Association

Archives of Industrial Health. 21:132-151.

Wen, W., T. Lieu, H. Chang, S. Wau, M. Yau, and K. Jan. 1981. Human Genetics.

59:201-203.

Westhoff , D.D., R.J. Samaha, and A. Barnes, Jr. 1975. Blood. 45:241-246.

WHO. 1981. Environmental Health Criteria, Arsenic. World Health Organization, Geneva, pp. 1-174.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F~8
BARIUM

Barium, an alkaline earth metal, is widely distributed in the environment. Smal l

amounts may accompany calcium in practically all biological and geological systems.

Absorption of barium compounds depends on the solubility of the compound, but

averages 2 percent for normal dietary barium. It is distributed throughout the body, with

about two-thirds being deposited in the bones. Excretion of barium is slow and occurs

primarily via the feces (Carson et al. 1986).

The acute toxic effect s of barium are primarily seen in muscle. Excitation is followed

by paralysis, probably due to interference with potassium transport. A characteristic

feature of barium toxicity is hypokalemia (insufficien t potassium in the blood). Initial

symptoms are gastrointestinal disturbances (nausea, vomiting, colic, diarrhea). Later

symptoms include tinglin g in the extremities and ECG irregularities leadin g to cardiac

fibrillation , general muscle paralysis, and death from respiratory arrest (Goyer 1986).

T he chronic toxic effect s of barium are not well defined, except for baritosis, an

occupational pneumoconiosis produced by inhalation of barite (an insoluble sulfate salt,

the most commonly occurring barium mineral). The few animal studies conducted have

found only nonspecific effect s (reduced longevity) (Sax 1986; Stokinger 1981).

Exposure to barium may be via inhalation or ingestion. The subchronic and chronic

inhalation RfC is 1 x 10"3 mg/kg/da y and 1 x 10"4 mg/kg/day , respectively. The

subchronic and chronic oral RfD is 5 x 10"2 mg/kg/day . These reference doses were

determined from a study by Tarasenko et al. (1977). There is no evidence for

carcinogenicity or teratogenicity.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-9
REFERENCES

Carson, B.L., H.V. Ellis , and J.L. McCann, eds. 1986. Toxicolog y and Biological

Monitoring of Metal s in Humans. Lewis Publishers, Inc. Michigan.

Goyer, R.A. 1986. Toxic Effect s of Metals. (In: Casarett and Doull's Toxicology : The

Basic Science of Poisons.) 3rd Ed. Edited by C.D. Klaassen, M.O. Amdur, and

J. Doull. MacMillan . New York.

Sax, N.I. 1986. Dangerous Properties of Industrial Materials. Van Nostran d Reinhold. N ew York.

Stokinger, H.E. 1981. The Metals. (In: Patty's Industrial Hygiene and Toxicology.)

Edited by C.D. Clayton and F.E. Clayton. Vol. 2A: 1492-2060.

Tarasenko et al. 1977. "Barium Compounds as Industrial Poisons (An Experimental

Study)." J. Hyg. Epidemiol. Micfobiol . Immunol. 21(4):361-373.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-10
BERYLLIUM

Beryllium is a naturally occurring grayish-white hard metal. Most mined beryllium is

used to make alloys for electronic and structural applications .

The most common route of beryllium exposure is via inhalation. Absorption in the lung

is rather slow and contributes to the long biological half-lif e of beryllium. Gastrointestinal absorption of beryllium is approximately 0.006 percent of that ingested. Beryllium is transported in the plasma, bound to alpha-globulin, transiently deposited

in the liver, kidneys, and lungs, and ultimately stored in bone. Excretion of beryllium

is extremely slow and occurs via the urine (Carson et al. 1986; Goyer 1986).

Toxic effect s associated with beryllium exposure are typically manifested in the skin,

eyes, respiratory system, liver, spleen, and heart. Dermal effects , which generally occur

two weeks followin g exposure, may include contact dermatitis as well as skin

sensitization. Acute systemic effect s followin g inhalation occur primarily in the

respiratory tract. Symptoms include coughing, chest pain, and shortness of breath.

Exposure to high levels may result in severe chemical pneumonitis with pulmonary

edema (Sax 1986; Stokinger 1981).

Intoxication due to chronic inhalation is referred to as chronic beryllium disease or

berylliosis and is characterized by the development of granulomatous lesions in the lungs. Thi s disease progresses in stages ranging from an asymptomatic, non-disabling phase to

a severely disabling phase characterized by significant pulmonary patholog y and heart

failure. Chronic beryllium disease usually has a delayed onset (5 to 10 years) and is of

lon g duration with exacerbations and remissions. Berylliosis can occur followin g

exposure to concentrations in air of 2 /*g/m 3 and above. As of 1983, the U.S. Beryllium

Case Registry had on record only 900 cases of beryllium disease nationwide (ATSDR

1987). Granulomatosis and chemical pneumonitis have also been attributed to beryllium

exposure, but dose-response data were unavailable.

Inhalatio n of a variety of beryllium compounds in experimental animals has been

associated with the development of pulmonary tumors. Rats chronically exposed to the

beryllium ore, beryl, developed lung tumors. Other studies in which rats were exposed

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F'l l
to beryllium compounds for periods ranging from 6 to 18 months generally indicate that

beryllium is a lung carcinogen (ATSDR 1987). Beryllium is classified by the EPA as a

potential human carcinogen (B2). The basis for this classification includes evidence of

carcinogenicity in three animal species by two routes of exposure. The inhalation and

oral slope factors are 8.4E+00 (mg/kg/day)" 1 and 4.3E+00 (mg/kg/day)'1, respectively,

as determined from a study by Wagoner et al. (1980).

The subchronic and chronic oral RfD for beryllium is 5 x 10"3 mg/kg/day , based on a

1975 study by Schroeder and Mitchener. An inhalation RfC is not available at this time.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-12
REFERENCES

Agency for Toxic Substances and Disease Registry (ATSDR). 1987. Toxicological

Profile for Beryllium. U.S. Public Health Service.

Carson, B.L., H.V. Ellis , and J.L. McCann, eds. 1986. Toxicolog y and Biological

Monitoring of Metal s in Humans. Lewis Publishers, Inc. Michigan.

Goyer, R.A. 1986. Toxic Effect s of Metals. (In: Casarett and Doull's Toxicology : The

Basic Science of Poisons.) 3rd Ed. Edited by C.D. Klaassen, M.O. Amdur, and

J. Doull. MacMillan . New York.

Sax, N.I. 1986. Dangerous Properties of Industrial Materials. Van Nostrand Reinhold. N ew York.

Schroeder, H.A. and Mitchener, 1975. "Life-time Studie s in Rats: Effect s of Aluminum, Barium, Beryllium and Tungsten." J. Nutr. 105:421-427.

Stokinger, H.E. 1981. The Metals. (In : Patty' s Industrial Hygien e an d Toxicology.)

Edited by C.D. Clayton and F.E. Clayton. Vol . 2A: 1492-2060.

Wagoner, J.K., P.P. Infante and D.L, Bayliss. 1980. "Beryllium: An Etiologic Agent in

t he Induction of Lung Cancer, Nonneoplastic Respiratory Disease, and Heart

Disease Among Industrially Exposed Workers." Environmental Restoration.

21:15-34.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-13
CADMIUM

Cadmium is a metal commonly used for the production of yellow, orange, and red paint

pigments. Representative pigments include cadmium selenite, cadmium sulfoselenide,

a nd cadmium sulfide. Cadmium also occurs naturally in foods including meat, fish,

fruits, grains and certain organ meats. Shellfish contain the highest amounts of cadmium

and can be a major source in the diet.

Acute inhalation of cadmium fumes (e.g., from welding) causes a chemical pneumonitis

with occasional associated pulmonary edema. Inhalation of concentrations of 5 mg/m3

for over 8 hours have been fatal , and sensitive individuals may show symptoms followin g

exposure to concentrations of 1 mg/m3 for 8 hours. Symptoms include shortness of

breath, general weakness, fever, and in severe cases, respiratory insufficienc y followed

by shock and death (Blinder 1985). Respiratory absorption of cadmium is approximately

15 to 30 percent (Klaassen et al. 1986).

Ingestion of toxic amounts of cadmium can produce nausea, vomiting, abdominal cramps,

headache, diarrhea and, in severe cases, shock. The onset of symptoms usually occurs

within minutes of ingestion. Concentrations found in drinking water of 15 mg/1 are

sufficien t to induce vomiting (Nordberg 1972), while higher concentrations in proteincontaining foods are required to produce the same symptoms (Fribirg et al. 1986).

T he protein metallothioriein offer s a protective effec t against cadmium toxicity in most

organs (with the exception of the kidney) by binding free cadmium. Pretreatment with

cadmium, zinc, or mercury induces metallothionein synthesis, which in turn reduces

overall cadmium toxicity. It has been suggested that cadmium toxicity in the kidney may

be related to early saturation of metallothionein (Fribirg et al. 1986). There is some

evidence that the co-administration of selenium with cadmium minimizes toxicity.

Evidence of toxicity associated with the chronic ingestion of cadmium is sparse.

Ingestio n of food and water from cadmium-contaminated regions of Japa n was related

to a disease known as itai-itai, which was characterized by severe renal tubular damage,

osteomalacia, osteoporosis, and leg and back pain (Nomiyama 1980). It appears that

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-14
dietary deficiencies of calcium and vitamin D may contribute to the disease (Fribirg et

al. 1986).

Most cases of chronic exposure to cadmium occur among industrial workers exposed via

inhalation. Concentrations of cadmium in the workplace have been reported at 4 to

5 mg/m3, although they are typicall y less than 2 mg/m3. Chronic inhalation produces a

number of effects , but the kidney is probably the primary target organ in humans. Kidney damage is characterized by renal tubule damage and associated tubular

proteinuria. In cases of severe exposure, glomerular damage may occur. Physiological

disturbance in the handling of calcium and phosphorus may cause mineral resorption

from the bone, leading to osteomalacia and kidney stone formation. Tubular damage

may persist or even increase after cessation of exposure and is probably related to

cadmium bound to metallothionein in the tubular cells (Blinder 1985). Chronic

cadmium exposure may also produce lung damage leading to emphysema. Reversible

anemia, probably due to hemolysis, has also been reported (Bernard et al. 1979).

Animal studies have indicated that hypertension followin g cadmium exposure may occur

in some species, although epidemiological studies suggest that this does not occur in

humans (Blinder 1985). The liver is another cadmium repository, and some animal

studies report liver damage followin g cadmium exposure. Only slight changes in liver

function have been reported in man, however (Fribirg et al. 1986). Chronic cadmium

inhalation has also been shown to cause increased excretion of calcium and phosphorus

in animals, which results in bone effect s similar to itai-itai. Although epidemiological

studies suggest that osteomalacia may occur among workers (Nicaud et al. 1942), bone

effect s were not seen by Fribirg (1986) in workers with relatively high occupational

exposures.

Teratogenesi s has been induced in rats and hamsters injected with high doses of

cadmium (3 mg/kg or more). Defect s included clef t lips , clef t palates , and limb defect s

(Fribirg et al. 1986). Perm and Hanlon (1983) reported that maternal pretreatment with

calcium minimized the incidence of terata. One epidemiological study among

industrially exposed women did not indicate significant increases in terata (Cvetkova

1970).

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-15
Injectio n of soluble cadmium salts at doses from 1 to 3 mg/kg in animals resulted in

testicular damage (Barlow and Sullivan 1982). However, testicular damage has not been

reported in human populations, probably due to the protective effect s of metallothionein

(Nordberg 1972).

T he epidemiological studies of Elinder (1985) suggest a possible link of cadmium

exposure to prostate and lung cancer. Studie s by Takenaka et al. (1983) have

demonstrated increased lung cancer in rats exposed for 18 months to cadmium chloride

aerosols (12.5 to 50 mg/m3). Cadmium has been classified by the EPA as a Class B2

potential human carcinogen by inhalation only based on animal studies.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-16
REFERENCES

Barlow, S.M. and P.M. Sullivan . 1982. (In: Reproductive Hazards of Industrial

Chemicals.^ Edited by S.M. Barlow, pp. 137-173. Academic Press. London.

Bernard, A., Bucket, J.P., Roels, H., Masson, P., and R. Lauwerys. 1979. Env. J. dim.

Invest. 9:11-22.

Cvetkova, R.P. 1970. Gig. Tr. Prof. Zabol. 14:31-33.

Elinder, C.G. 1985. (In: Cadmium and Health: A Toxicological and Epidemiological

Appraisal Chapter 11.) Edited by L. Friberg, C.G. Elinder, T. Kjellstrom , and

G.F. Nordberg. CRC Press. Boca Raton, Florida.

Ferm, V.H. and D.P. Hanlon. 1983. (In: Reproductive and Developmental Toxicity of Metals.) Edited by T.W. Clarkson, G.F. Nordberg, and P.R. Sager. Plenium

Press. New York. pp. 383-389.

Fribirg, L. 1986. Cadrnium and Health. A Toxicological and Epidemiological

Appraisal . Vol. I, General Aspects; Vol. II, Effect s and Response. CRC Press, Inc., Boca Raton, Fla.

Fribirg, L., G.F. Nordberg, and B.V. Vouk. 1986. Handbook on the Toxicology of Metals. Vol. 2: Specifi c Metals . 2nd ed. Elsevier. New York.

Klaassen, C.D., M.O. Amdur, and J. Doull. 1986. Casarett and Doull's Toxicology : The

Basic Science of Poisons. 3rd ed. MacMillan Publishin g Co. New York.

Nicaud, P., A. Lafitte , and A. Gros. 1942. Arch. Mal, Prof. Med. Trav. Secur. Soc.

4:192-202.

Nomiyama, K. 1980. "Recent Progress and Perspectives in Cadmium Health Effect s

Studies." Sci. Total Environ. 14:199-232.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F'1 7
Nordberg, G.F. 1972. Environ. Phys. Biochem. 2:7-36.

Takenaka et al. 1983. "Carcinogenicity of Cadmium Chloride Aerosol s in Wistar Rats."

J. Natl. Cancer Inst . 70:367-373.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-18
CHROMIUM

Chromium is a relatively common metal and exists in a number of oxidation states,

ranging from Cr2+ to Cr6+. The trivalent and hexavalent forms are of primary

toxicological relevance. A number of chromium compounds are commonly used as paint

pigments, including lead chromate, zinc chromates, and chrome oxides.

Chromium is a cofactor for insulin action, facilitatin g the attachment of insulin to insulin

receptors. It is necessary for glucose tolerance and thus has been classified as an

essential element. Very limited quantities are required for purposes of human health,

however, and no dietary requirements have been established. Evidence for chromium

deficiency in man is sparse (Langard and Norseth 1986).

Hexavalent chromium is carcinogenic by inhalation. It is absorbed more readily than the

trivalent form. Both trivalent and hexavalent chromium appear to be toxic via the

inhalation pathway (EPA 1991). Absorption can occur through the lungs or the

gastrointestinal tract. Studie s in rats suggest that 3 to 6 percent of chromates are

absorbed through the gastrointestinal tract (Mertz et al. 1965). Upon absorption,

hexavalent chromium is rapidl y converted to the trivalent form, which can bind to

intracellular macromolecules. Many of the toxic effect s of chromium have been related

to these macromolecular complexes. Animal studies show that chromium is retained in

the lung, hair, reticuloendothelial system, liver, spleen, and bone marrow (Wisek et al.

1953), as well as the testis and epididymi s (Hopkin s 1965). Tossavainen et al. (1980)

estimated that the elimination half-life for chromium in welders ranged from 15 to

41 hours. Excretion is primarily through the urine.

Acute effect s associated with chromium exposure have been reported for dermal,

respiratory, and oral routes of exposure. Direct contact of broken skin with hexavalent

chromium compounds may cause deep ulcerations of the skin that are slow to heal. In

humans, ingestion can produce local ulceration of the stomach and intestinal mucosa,

and ingestion of very high doses (5 gm) has been reported to cause gastrointestinal

bleeding, fluid loss, and death due to cardiovascular shock (Langard and Norseth 1986). Proximal tubule kidney damage, along with some hepatic necrosis, was seen in humans

followin g ingestion of 1 to 5 gm of chromate (Langard and Norseth 1986). Acute

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-19
inhalation of chrome dust for 4 to 8 hours has reportedly induced bronchial asthma

(Langard 1980).

There is some evidence that chromium can be transported across the placenta.

Increased incidences of terata have also been seen in chicks exposed to chromate (0.002

to 0.05 mg/egg). These effect s include reduced body weight, microphthalmia, short and

twisted limbs, ectopic heart, and inverted viscera (Gilan i and Marano 1979).

In vitro mutagenicity studies have shown that chromium can cause DNA damage, sister

chromatid exchanges, and can induce DNA repair. Chromosomal aberrations have also

been noted in the lymphocytes of chronically exposed workers.

Numerous epidemiological studies in chromate-exposed workers indicate a correlation

between chromium exposures and respiratory cancers (Langard 1983). Animal studies

using certain forms of hexavalent chromium have also provided sufficien t evidence for

carcinogenicity (EPA 1984a). The EPA considers chromium a Class A human

carcinogen by inhalation only. A slope factor exists for the inhalation of chromium,

based on studies by Mancuso (1975) and USEPA (EPA 1984a). RfD values for

chromium have been based pn studies by EPA (1984b), MacKenzie et al. (1958) , and

Lindberg and Hedenstiern a (1983).

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-20
REFERENCES

Gilani, S.H. and M. Marano. 1979. Environmental Restoration. 19:427-431.

Hopkins , Jr. , L.L. 1965. American Journal of Physiology. 209:731-735.

Langard, S. 1980. Int. Arch Occup. Environ. Health. 46:1-9.

Langard, S. 1983. (In: Chromium.) Edited by D< Burrows. CRC Press. Boca Raton, Florida. 13-30.

Langard, S. and T. Norseth. 1986. (In: Handbook on the Toxicology of Metals.) Vol. 2.

2nd ed. Edited by L. Fribirg, G.F. Nordberg, and V.B. Vouk. Elsevier. New

York.

Lindberg, E. and G. Hedenstierna. 1983; Chrome Plating: Symptoms , Findings in the

Upper Airways, and Effect s on Lung Function. Arch. Environ. Health.

38: 367-374.

MacKenzie, R.D., R.U. Byerrum, C.F. Decker et al. 1958. Chronic toxicity studies. II. Hexavalent and Trivalent Chromium Administered in Heavy Drinking Water to

Rats. Am. Med. Assoc. Arch. Ind. Health. 18:232-234.

Mancuso, T.F. 1975. International Conference on Heavy Metal s in the Environment. Toronto, Canada. October 27-31. (Cited in US EPA 1984)

Mertz, W., E.E. Roginski, and R.C. Reba. 1965. American Journal of Physiology.

209:489-494.

Tossavainen, A., Nurminen, P. Mutanen, and S. Tola. 1980. British Journal of Industrial Medicine. 37:285-291.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-21
U.S. Environmental Protection Agency (EPA). 1984a. "Health Effect s Assessment for

Hexavalent Chromium." Prepared by the Offic e of Health and Environmental

Assessment, Environmental Criteria and Assessment Office , Cincinnati, OH for

the Offic e of Emergency and Remedial Response, Washington , DC.

U.S. Environmental Protection Agency (EPA). 1984b. Offic e of Emergency and

Remedial Response. Washington, D.C. EPA/540/1-86-019.

U.S. Environmental Protection Agency (EPA) . 1991. Health Effect s Assessment

Summary Tables (HEAST). Annual FY-1991. OSWER (OS-230).

Wisek, W.J, L.B. Whitney, U.S.G. Kuhn, and C.L. Comar. 1953. Proc. Soc. Exp. Biol. Med. 84:610-615.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-22
COPPER

Copper is a widely distributed element and is an essential component of numerous

enzyme systems within the body. (Carson et al. 1986)

The routes of exposure include inhalation, ingestion, and skin and eye contact. Absorption of copper from the gastrointestinal tract is regulated by body stores of

copper. Once absorbed, copper is initially transported in the plasma, bound to albumin

and later bound to 2-ceruloplasmin (Stokinger 1981). Copper is excreted via the bile

and copper homeostasis is maintained this way. Copper is stored in the liver and bone

marrow and binds to metallotheionein. The most important route of exposure to copper

is through oral intake. The toxicological effect s of copper are exerted in the respiratory

system, gastrointestinal system, liver, hematologic .system, kidneys, and skin (Sax 1986). Acute toxicosis from the ingestion of excessive amounts of copper produces damage to

the liver and gastrointestinal tract. Adverse health effect s associated with acute exposure

include vomiting, gastrointestinal bleeding, red blood cell destruction, jaundice, low

blood pressure, and coma. Chronic ingestion toxicosis is rare and only occurs in

individuals with hepatolenticular degeneration (Wilson's Disease) which results in

abnormally high absorption, retention, and storage of copper by the body. Dermal

exposure results in itching, erythema, and dermatitis. Respiratory symptoms followin g

inhalation include congestion of the nasal and pharyngeal mucous membranes and

possibl y nasal septum ulceration or perforation. Other symptoms followin g inhalation

include nausea, metal fume fever, and discoloration of the skin and hair (Goyer 1986).

T he subchronic and chronic oral RfD for copper is 3.7E-02 (mg/kg-day) . The ingestion

RfC has not yet been determined.

The carcinogenicity of copper is not classified (Group D) by the EPA. This classification

is based on the absence of human data, inadequate animal data, and equivocal

mutagenicity data. A chronic health hazard assessment for noncarcinogenic effect s is not

available at this time (EPA 1991).

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-23
REFERENCES

Carson, B.L., H.V. Ellis., and J.L. McCann, eds. 1986. Toxicology and Biological

Monitoring of Metals in Humans. Lewis Publishers, Inc. Michigan.

Goyer, R.A. 1986. Toxic Effect s of Metals. (In: Casarett and Doull's Toxicology : The

Basic Science of Poisons.) 3rd Ed. Edited by CD. Klaassen , M.O. Amdur, and

J. Doull. MacMillan. New York.

Sax, N.I. 1986. Dangerous Properties of Industrial Materials. Van Nostran d Reinhold. N ew York.

Stokinger, H.E. 1981. The Metals. (In : Patty' s Industrial Hygien e an d Toxicology.')

Edited by C.D. Clayton and F.E. Clayton. Vol. 2A: 1492-2060.

U.S. Environmental Protection Agency (EPA). 1991. Health Effect s Assessment

Summary Tables (HEAST). Annual FY-1991. OSWER (OS-230).

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F~24
LEAD

Lead is a metal with numerous industrial applications. Use of lead in paint pigments

was widespread in the past, but has been curtailed in recent years. Typical lead-based

paint pigments include lead chromate, dibasic lead phosphite, and various lead oxides.

Lead toxicity is generally a function of age-dependent absorption. Gastrointestinal

absorption has been reported from 5 to 15 percent in adults with less than 5 percent

retention, and approximately 42 percent in children with approximately 32 percent

retention. Respiratory absorption is even greater. Approximatel y 90 percent of respired

particles (0.5 jwm or smaller) are absorbed.

The extent of lead absorption in the gastrointestinal tract has been linked to a number

of dietary factors. Absorption is enhanced by milk products , low calcium and vitamin D

levels, fasting, or iron deficiencies. Absorbed lead tends to deposi t into two pool s in the

body, the skeleton and sof t tissue. Lead in the skeleton is released very slowly, with a

biologic half-life of approximately five years. Lead in the sof t tissue has a much shorter

half-life , approximately 3 to 4 weeks. Tsuchiya (1986) reports that 90 percent of ingested

lead is excreted in the feces, while most absorbed lead is excreted in the urine.

T he major targets for lead toxicity are the central nervous system, hematopoietic system,

gastrointestinal tract, and renal system. Gastrointestinal colic is the most common effec t

of acute lead ingestipn. The initial stages of lead intoxication include anorexia,

dyspepsia, and constipation followed by colic, which is characterized by a diffus e

paroxysmal abdominal pain. The skin is usually pale and blood pressure may increase,

reflectin g sporadic contraction of the smooth muscle.

Lead encephalbpathy has also been reported. Although it is rare in adults , numerous

cases have been reported in children exhibiting pica. Pica is characterized by a craving

f or and consumption of unnatural articles of food, e.g., soil, clay, etc. In some cases, the

encephalopathy syndrome in children is characterized by vomiting, apathy, drowsiness,

stupor, ataxia, hyperactivity, and other neurological symptoms. Lead levels in the blood

typicall y range from 80 to 300 ^g/100 ml (Tsuchiy a 1986). Lead encephalopath y may

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-25
be characterized by a sudden onset of seizures and delirium, with commonly associated

papilledema. In severe cases, coma and cardiorespiratory arrest may occur.

Anemia is a common symptom among workers chronically exposed to lead. Leadinduced anemia is probably due to both an inhibition of hemoglobin synthesis and a

shortened lifespa n of the erythrocytes. Decreased hemoglobin synthesis is apparently

due to inhibition of several key enzymes (Wada et al. 1972). Chronic exposure also

affect s the central and peripheral nervous systems, particularly in children. Effect s

include mental deterioration, hyperkinetic or aggressive behavior, sleepin g difficulties ,

and vomiting. Subclinical effect s have also been noted in children with moderately

elevated blood lead levels (40 to 80 ^tg/100 ml). Recent work by Bellinger et al. (1987)

suggests that fetal blood levels as low as 10 /ig/100 ml (compared to background levels

of 6 to 7 /ixg/100 ml) may cause significant deficiencies in learning ability during the firs t

two years of life.

Neural effect s have been reported in chronically exposed workers. Symptoms include

impairment of memory, attention, concentration, arid psychomotor performance (Arnvig

et al. 1980). Peripheral neuropathy is characterized clinically by wrist and foot drop and

subclinically by reduced peripheral nerve conduction. Chronic gastrointestinal effect s

may include loss of appetite, upset stomach, diarrhea, or constipation. Degenerative

changes have been noted in the proximal tubular cells of the kidney and are associated

with swelling of the mitochondria. Long-term exposure produces a characteristic type

of nuclear-inclusion body in the tubular cell s of the kidney. These bodies are composed

of a lead-protein complex and apparently function as a protective mechanism for other

organelles. Long-term exposure is also associated with intense interstitial fibrosis ,

tubular atrophy, and dilation. There is glomerular involvement at the late stages of

chronic exposure (Emmerson 1968). There is little evidence for either hepatic or

cardiovascular effect s associated with chronic lead exposure.

Animal studies suggest that lead may be a teratogen. Lead and cadmium produce a

synergistic teratogenic effec t (Perm 1969), whereas zinc is reported to have an

antagonistic effec t on lead toxicity (Willoughb y et al. 1972).

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-26
Lead has been shown to induce cancer in the kidneys of rodents under conditions of high

exposure (Moore and Meredith 1979). However, there is no evidence of renal

carcinogenicity, nor does lead appear to produce chromosomal anomalies in humans.

Lead has been classified as a Class B2 potential human carcinogen based on animal

studies performed by EPA (EPA 1984, 1986). A slope factor has not been determined

yet for lead.

Infant s and children (ages 0-7) have been identified as the populations most vulnerable

to lead exposure. A child absorbs a greater fraction of an oral dose of lead than does

an adult, and a child's developing nervous system is more sensitive to lead. In addition,

children are known to ingest more soil (e.g., mouthing behavior) than adults , and soil is

a known reservoir of metals in the environment. Furthermore, various physiologic and

anatomic factors result in greater deposition of airborne lead in the respiratory tract of

infant s and children. Finally , transplacental transfer of lead can occur that results in a

lead burden in the fetus , increasing exposure during infancy and early childhood.

Considerable controversy exists regarding the biological significance of some of the

effect s attributed to chronic low lead exposure and the blood lead levels at which they

occur. The EPA has stated that a blood lead level of 10 jug/dl is a level of concern; if

95 percent of children in a given population have levels exceeding this, the EPA

recommends community education programs.

There is a currently no RfD for lead because a threshold for neurotoxic effect s does not

appear to exist. U.S. EPA recommends the use of a model, the Integrated Uptake /

Biokinetic Model (IUBK) (EPA 1991a,b), to estimate blood lead values resulting from

exposure to lead. The IUBK model provides a means for evaluating the relative

contribution of various media to lead exposure. The most recent version available of the

IUBR, version 0.50, will be used in this assessment.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-27
REFERENCES

Arnvig, E., P. Gradjean , and J. Beckmann. 1980. Toxicol . Lett. 5:399-404.

Bellinger, D., A. Leviton, C. Waternaux, H. Needleman , and M. Rabinowitz. 1987. New

England Journal of Medicine. 316:1036-1043.

Emmerson, B.T. 1968. Arthritis Rhemm. 11:623-643.

Perm, V.H. 1969. Experieutia. 25:56-57.

Moore, M.R. and P.A. Meredith. 1979. Arch. Toxicol . 42:87-94.

Tsuchiya, K. 1986. (In: Handbook on the Toxicology of Metals.) Vol . 2. 2nd ed.

Edited by L. Friberg, G.F. Nordberg, and V.B. Vouk. Elsevier. New York.

U.S. Environmental Protection Agency (EPA). 1984. "Health Effect s Assessment for

Lead." Prepared by the Offic e of Health and Environmental Assessment,

Environmental Criteria and Assessment Office , Cincinnati, OH for the Offic e of

Emergency and Remedial Response, Washington , DC.

U.S. Environmental Protection Agency (EPA). 1986. "Air Quality Criteria for Lead."

June, 1986 and Addendum, September, Offic e of Health and Environmental

Assessment, Research Triangle Park, NC. EPA 600/8-83-028F. NTIS PB

87-142378.

U.S. Environmental Protection Agency (EPA) . 1991a. Technical Support Document on

Lead. Offic e of Health and Environmental Assessment.

U.S. Environmental Protection Agency (EPA). 1991b. Users Guide for Lead: A PC

software applicatio n of the Uptake/Biokinetic Model (Version 5.0). Offic e of Health and Environmental Assessment.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-28
Wada, O., Y. Yano, K. Toyokawa, S, Suzuki, and H. Katsunuma. 1972. Industrial

Health. 19:84-92.

Willoughby , R.A., E. MacDonald, B.J. McSherry, and G. Brown. 1972. Vet. Rec.

6:382-383.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-29
MANGANES E

Manganese is used in the manufacture of steel alloys, electrical coils, batteries, weldin g

rods, dyes, ceramics, matches, glass, and fertilizers . It is an essential element and

cofactor for a number of physiologic enzymatic reactions (Sax 1986).

Routes of exposure include inhalation, percutaneous absorption, and ingestion. Gastrointestinal absorption of ingested manganese is estimated at 4 percent. Absorbed

manganese is transported in plasma, bound to beta-globulin. It is widely distributed in

the tissues, with the greatest concentrations occurring in the intestines, kidney, liver,

pancreas, and certain nerve cells of the brain. Manganese is excreted in the feces with

a biological half-life of 37 days (Goyer 1986).

Manganese is not toxic via dermal exposure. Local effect s of inhalation exposure include

irritation of the eyes and respiratory tract. Among Individual s exposed, there is a

marked variation in susceptibility to manganese toxicosis. Inhalation toxicosis is

manifested as two disease entities: (1) manganese pneumonitis, and (2) manganese

psychosis. Manganese pneumonitis is due to acute inhalation exposure and is

characterized by epithelial necrosis and mononuclear proliferatio n (Goyer 1986;

Stokinger 1981). An epidemiologic survey (Roels et al. 1984) indicated an increased

incidence in respiratory disease in occupational exposure situations. Manganese

psychosis is characterized by the onset of a psychiatric disorder with symptoms including

irritability , difficult y walking, impulsive behavior, mental confusion, speech disturbances,

aggressiveness, and hallucinations. With continued exposure, the condition develops into

a Parkinson-like syndrome with similar symptoms and pathological changes. Liver

cirrhosis is also frequently observed with chronic manganese exposure (Goyer 1986;

Stokinger 1981).

Manganese is classified by the EPA as Class D; human carcinogenicity cannot be

determined. This classification is based on the inadequacy of existing studies to assess

t he carcinogenicity of manganese. The reference dose for the assessment of

noncarcinogenic effect s associated with oral manganese exposure has been withdrawn

from IRIS. Medium confidence is placed on this value due to the high confidence in

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-30
available human intake studies and medium confidence in the database from which the

reference dose was derived. The inhalation RfD for manganese is 1.4E-03 mg/kg/day .

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 . F-31
REFERENCES

Goyer, R.A. 1986. Toxic Effect s of Metals . In: Casarett and Doull's Toxicology : The

Basic Science of Poisons. 3rd Ed. Edited by C.D. Klaassen , M.O. Amdur, and

J. Doull. MacMillan . New York.

Roels, H., R. Lauwerys, J-P. Buchet et al. 1984. "Epidemiological survey among

workers exposed to manganese: Effect s on lung, central nervous system, and

some biological indices." Am J. Med. 11:307-327.

Sax, N.I. 1986. Dangerous Properties of Industrial Materials. Van Nostran d Reinhold. N ew York.

Stokinger, H.E. 1981. The Metals. In: Patty's Industrial Hygien e and Toxicology .

Edited by C.D. Clayton and F.E. Clayton. Vol . 2A: 1492-2060.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-32
MERCURY

Mercury is used as a component of batteries, industrial and control instruments,

cathodes, wire and switching devices, fluorescen t lamps, and as a laboratory reagent

and/or catalyst. There are three forms of mercury of toxicological importance:

( 1) elemental (mercury vapor), (2) inorganic (mercuric and mercurous salts) , and (3)

organic (methyl mercury) compounds (Sax 1986).

Routes of exposure for mercury include inhalation, ingestion, skin absorption, and eye

contact. Mercury disposition is influenced by valence state and anionic component. Deposition and retention of mercury in the lungs followin g mercury vapor inhalation is

approximately 80 percent. Elemental mercury has a high affinit y for red blood cell s and

the central nervous system. Gastrointestinal absorption of mercury is .01 percent for

metallic mercury, 7 percent for inorganic mercury, and 90 to 95 percent for organic

mercury. Inorganic mercury concentrates in the kidneys, whereas organic mercury

concentrates in the brain. All forms of mercury have been demonstrated to cross the

placenta, and neonatal exposure to mercury may occur through nursing. Excretion of

mercury is via urine and feces. The biological half-life of mercury is dependen t upon

the form of mercury absorbed and ranges from 35 to 90 days (Carson et al. 1986; Goyer

1986).

Toxicological manifestations of mercury occur in the skin, respiratory system, central

nervous system, kidneys, and eyes. Mercury vapor inhalation may produce an acute,

corrosive bronchitis and interstitial pneumonitis and may be associated with symptoms

affectin g the central nervous system. Symptoms followin g exposure include coughing,

difficult y breathing, chest pains, insomnia, irritability , headaches, fatigue, weakness, and

tremors. Chronic inhalation exposure is associated with degeneration of the central

nervous system which is manifested as neurologic and psychiatric abnormalities.

Ingestion of mercuric mercury produces corrosive ulceration and bleedin g of the

gastrointestinal tract, shock, circulatory collapse, and renal failure (Stokinger 1981;

Goyer 1986).

Mercurous compounds are less toxic than mercuric salts and are associated with a

hypersensitivity response in children referred to as acrodynia or "pink disease." Mercury

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-33
toxicosis due to chronic exposure to inorganic mercury is typically manifested by one or

more of the followin g four signs: (1) steatorrhea, (2) gingivitis, (3) irritability , and (4)

muscular tremors. In terms of toxicity and health effect s from environmental exposures,

methyl mercury is the most important form of mercury. Organic mercury exposure

produces degeneration and death of central and peripheral nervous system tissues, with

resulting neurologic abnormalities. The major lexicological effect s include paresthesia,

ataxia, dysarthria, and deafness. Mercury exposure may also produce a discoloration of

the lens and cornea of the eye. Direct contact may produce a contact dermatiti s with

possible blister formation (Carson et al. 1986; Goyer 1986).

Mercury is not classifiable (Group D) as to human carcinogenicity based on EPA weightof-evidence. Thi s classification is based on the absence of human data and inadequate

animal and supportin g data.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-34
REFERENCES

Carson, B.L., H.V. Ellis., and J.L. McCann, eds. 1986. Toxicolog y and Biological

Monitoring of Metal s in Humans. Lewis Publishers, Inc. Michigan.

Goyer, R.A. 1986. Toxic Effect s of Metals. (In: Casarett and Doull's Toxicology : The

Basic Science of Poisons.) 3rd Ed. Edited by CD. Klaassen, M.O. Amdur, and

J. Doull. MacMillan . New York.

Sax, N.I. 1986. Dangerous Properties of Industrial Materials. Van Nostran d Reinhold. N ew York.

Stokinger, H.E. 1981. The Metals. (In: Patty's Industrial Hygien e and Toxicology.^

Edited by C.D. Clayton and F.E. Clayton. Vol. 2A: 1492-2060.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-35
ZIN C

Zinc is used in wood preservatives, dry battery cells, textiles , soldering flux, oil refining,

oil and gas well operations, and in the manufacture of paper, dyes, activated carbon,

chemicals, deodorants, and disinfectant s (Stokinger 1981). Zinc is a nutritionally

essential element.

Routes of exposure to zinc include inhalation of dust and fumes, ingestion, and skin and

eye exposure. The gastrointestinal absorption of zinc is approximately 20 to 30 percent.

Zinc absorption is thought to occur via a carrier-mediated process and is homeostatically

controlled. Absorbed zinc is bound to albumin and beta-macroglobin. The highest

concentration of zinc is deposited in the prostate, with significant deposition also

occurring in the bone and muscle. Excretion occurs predominantly in the feces. The

biological half-life of zinc is estimated to range from 162 to 500 days.

The effect s of zinc toxicosis are seen in the respiratory system, skin, and eyes (Sax 1986). Toxic effect s associated with dermal exposure include dermatitis and chemical burns due

to the corrosivity of zinc compounds, and skin sensitization characterized by eczematoid

dermatitis. Occupational inhalation of zinc has not been reported. Inhalatio n of zinc

chloride results in severe respiratory irritation and may be accompanied by pulmonary

edema, hemorrhagic alveolitis, and bronchopneumonia (Goyer 1986; Carson 1986).

Zinc has not been evaluated by the EPA for evidence of human carcinogenic potential . T he reference dose for the evaluation of noncarcinogenic health effect s associated with

subchronic and chronic oral zinc exposure is 2.0 E-01 mg/kg/day . A reference dose for

the assessment of chronic inhalation exposure is not available.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-36
REFERENCES

Carson, B.L., H.V. Ellis., and J.L. McCann, eds. 1986. Toxicology and Biological

Monitoring of Metal s in Humans. Lewis Publishers, Inc. Michigan.

Goyer, R.A. 1986. Toxic Effect s of Metals. (In: Casarett and Doull's Toxicology : The

Basic Science of Poisons.) 3rd Ed. Edited by C.D. Klaassen, M.O. Amdur, and

J. Doull. MacMillan . New York.

Sax, N.I. 1986. Dangerous Properties of Industrial Materials. Van Nostran d Reinhold. N ew York.

Stokinger, H.E. 1981. The Metals. (In: Patty's Industrial Hygien e and Toxicology.)

Edited by C.D. Clayton and F.E. Clayton. Vol. 2A: 1492-2060.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-37
INTERPRETATION OF ASSES S OUTPU T

!?: <•:-: -

22909C-22327/R22;ET 01 ;2S-93(4:26pm)/RPT/1 6 F-3 8 ••• •
INTERPRETATION OF ASSESS OUTPUT

ASSESS provides the cancer risk and hazard index calculations for all exposure pathways

evaluated at a site. All information needed to verify calculations is provided. Data are

organized and reported in categories listed below:

• Chemical List

• Receptor/Pathway Summary

• Pathway Summary

• Intake Factors

• Media Concentrations

Numbers in ASSESS are written in scientific notation. The number followin g the E is

a power of 10; so 1.61E-02 is 0.0161.

CHEMICA L LIS T

Thi s sheet list s the EPA toxicity values for each chemical. When available, values are

listed for both oral and inhalation exposure routes for either subchronic or chronic

exposure durations (reference doses or RfDs) or for carcinogenic risk (slope factors or

SF). These values are obtained using the hierarchy recommended by EPA's Risk

Assessment Guidance for Superfun d sites (EPA 1989b, 1991b). The firs t and second

sources in this hierarchy are (1) IRIS (Integrated Risk Information System) , an on-line

computer database of chemical risk information (EPA 1992d), and (2) HEAST (Health

Effect s Assessment Summary Tables ) (EPA 1992e), EPA tables which are periodicall y

up-dated. Toxicity values (RfDs and SFs) do not yet exist for many chemicals. For risk

assessment purposes, when this is true, a chemical may be discussed qualitatively in the

text of the report but will not be reported quantitatively in ASSESS.

RfDs may be derived for either subchronic or chronic exposures and are used to

calculate subchronic or chronic hazard quotients, respectively. A subchronic RfD may

not exist for a particular chemical even though a chronic RfD exists.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-39
RECEPTOR/PATHWA Y SUMMARY

T he Receptor Summary sheet lists each receptor and the potential health risks associated

with chemical exposure from all pathways quantitated. The potential adverse health

risks are divided into two exposure scenarios: average exposure and reasonable

maximum exposure (RME). For each exposure scenario, potential health impacts are

segregated into two categories: cancer risk and a hazard index for both subchronic and

chronic exposures. The values shown on this sheet show the total carcinogenic risks and

noncarcinogenic hazards associated with average and RME exposures for each receptor

listed.

RfDs are used with a level of exposure during a specific time period and expressed as

a ratio or hazard quotient. According to the EPA (EPA 1989b, 1991b), generally, the

greater the value of the hazard quotient (or hazard index), the greater the level of

concern for health impacts. Another area of EPA guidance states that because HI is not

a probability function , an increased value does not indicate a degree of risk. The hazard

index is the sum of a number of hazard quotients for a particular pathway. A hazard

index of one means the estimated chemical intake equals the reference dose. When this

occurs, a more detailed evaluation of the site is indicated. When the HI is less than one,

further evaluation is generally not warranted, and the exposure is expected to be safe if

the HI is equal to or less than one target organ concept.

A chemical' s slope factor is multiplied by the lifetime daily average intake of that

chemical in a certain medium to estimate the incremental probability (or risk) of an

individual developin g cancer during a lifetime. When a receptor potentiall y is exposed

to a variety of chemicals (in a given medium) possessing slope factors , all of the product s

a re summed to yield the total cancer risk for those chemicals for that specific exposure

pathway. This risk number is acceptable if it is less than IE-4 or 10"4 or 1 in 10,000.

Receptor Detail Report

Thi s report is included in the Receptor/Pathway Summary Sheet because it detail s the

carcinogenic risk and the noncarcinogenic hazard for each of the receptor' s exposure

pathway(s) for both the average and RME exposure scenarios. Total carcinogenic risks

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-40
and noncarcinogenic hazards for each receptor are also given. Each receptor may have

a number of exposure pathways. An exposure pathway is a route that a chemical may

take to reach a populatio n or receptor. Each pathway is categorized as one of the

following:

• Inhalation

• Dermal contact with solids

• Dermal contact with water

• Ingestion of solids

• Ingestion of water

PATHWAY SUMMAR Y

Thi s sheet also provides the same basic information as the Receptor Detail Report

described above. The differenc e between the two is that the Receptor Detail Report

includes all receptors while the Pathway Summary Report is receptor-specific, providing

information on only one receptor per page.

Pathway Detail Report

These sheets comprise the majority of the ASSESS report. For both average and RME

exposure scenarios, carcinogenic risk arid noncarcinogenic hazards are listed for each

receptor exposure route and chemical. Total carcinogenic risks and noncarcinogenic

hazards are also provided. Thi s report is especiall y helpful in determining which

chemical(s) is (are) driving the overall risks or hazards for a particular exposure route.

Other values provided in this report include the concentration of each chemical in the

medium, the intake factor for that exposure route, and the daily intake of the chemical

being evaluated. Thi s daily intake of an individual chemical is either multiplied by its

slope factor or divided by its RfD for a given exposure route (oral or inhalation). Unti l

other information is available from EPA, oral RfDs and SFs are used for assessing the

potential health impacts of dermal exposure.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-41
INTAK E FACTOR S

These sheets represent, in equation format , the method of calculating the intake factors

that appear in the pathway detail report explained above. Each of the parameters that

goes into an intake factor equation is defined for both the average and RME scenarios.

These parameters are more full y described in the Exposure Assessment section of this

report.

MEDIA CONCENTRATION S

These sheets describe the chemical concentrations for the average and RME scenarios

for a given medium. In some cases, a higher concentration may be used to evaluate

short-term risks. For example, paniculate concentrations in air may be higher during

a- short construction project than the long-term (25 year) average might be. In most

cases, the same concentrations will be used for subchronic and chronic exposures. These

figures are described in the Data Summary and Selection of Chemicals of Concern

section of this report.

22909C-22327/R22.F 01-28-93(4:26pm)/RPT/16 F-42
ASSES S REPORTS

22909r-22327/R22..S;6f-28-93<4:26pm>/RPT7l6 F-43:
Chemical List: Impounded Tailin g January 28, 1993

=====^=========^==^^=^== 16

Inhalatio n —————————————— — ———— — Non-Inhalatio n Slop e Subchronic Chronic Slop e Subchronic Chronic Facto r RF D RF D Facto r RF D RF D

Chemical (mg/kg/day)-1 mg/kg/da y mg/kg/da y (mg/kg/day)-1 mg/kg/da y mg/kg/da y

Antimon y 1.HE-04 1.14E-04 4.00E-04 4.00E-04

Arseni c 1.50E+01 1.75E+00 3.00E-04 3.00E-04

Berylliu m 8.40E+00 4.30E+00 5.00E-03 5.00E-03

Cadmium (food ) 6.30E+00 1.00E-03

Chromium VI 4.10E+01 5.70E-06 5.70E-06 2.00E-02 5.00E-03

Copper 3.70E-02 3.70E-02

Manganese 1.14E-04 1.14E-04

Zin c 2.00E-01 3.00E-01
Receptor/Pathwa y Summary: Impounded Tailin g January 28, 1993

16:58

Receptor / Pathwa y

Average Exposure

Cancer Subchronic Chronic Risk H.I . H.I .

Adul t Visito r

-- Ingestio n of Tailin g

-- Inhalatio n of Particulate s

Chil d Visito r

-- Ingestio n of Tailin g

Reasonable Maximum Exposure

Cancer Subchroni c Chronic Risk H.I . H.I .

5.05E-07 2.35E-02 2.38E-02 2.86E-05 1.66E-01 1.68E-01

9.98E-11 1.29E-06 1.29E-06 3.95E-09 1.85E-05 1.85E-05

5.05E-07 2.35E-02 2.38E-02 2.86E-05 1.66E-01 1.68E-01

5.68E-06 1.32E-01 1.34E-01 3.89E-05 9.01E-01 9.15E-01

5.68E-06 1.32E-01 1.34E-01 3.89E-05 9.01E-01 9.15E-01

Dir t Biker

-- Ingestio n of Tailin g

-- Inhalatio n of Particulate s

1.89E-06 2.94E-02 2.98E-02 1.70E-05 2.62E-01 2.66E-01

2.66E-10 3.45E-06 3.45E-06 4.19E-09 6.57E-05 6.57E-05

1.89E-06 2.94E-02 2.98E-02 1.70E-05 2.62E-01 2.66E-01
Adul t Visitor Ingestio n of Tailin g Average Exposure

1

| Carcinogenic Risk

i

Chemical Ingestio n Concentratio n Intak e Facto r Chemical

Arseni c Berylliu m

t

| Hazard Index -- Subchronic

i

(mg/kg ) (kg/kg/day )

4.17E+02 6.81E-10

2.96E+00 6.81E-10

Chemical Ingestio n Concentration Intake Factor Chemical

Antimon y Arsenic" Beryl lium Chromium V I

Copper Zin c

1 ————————————————————— —

| Hazard Index -- Chronic

<mg/kg ) (kg/kg/day )

2.39E+U 1.59E-08

4.17E+02 1.59E-08

2.96E+00 1.59E-08

6.41E+00 1.59E-08

4.94E+02 1.59E-08

3.50E+03 1.59E-P8

. • •• 1"

; i

' . : .

Chemical Ingestio n Concentratio n Intak e Facto r Chemical

Antimon y Arseni c

 ; Berylliu m

Cadmium (food ) Chromium V I

Copper Zin c

(mg/kg ) Ckg/kg/day )

2.39E+01 1.59E-08

4.17E+02 1.59E-08

2.96E+UO 1.59E-08

2.51E+01 1.59E-08

6.41E+00 1.59E-08

4.94E+02 1.59E-08

3.50E+03 1.59E-08

Dail y Intak e

(mg/ kg/day )

2.84E-07

2.02E-09

D ai ly Intake

(nig/kg/day )

3.80E-07

6.62E-06

4.70E-08

1.02E-07

7.84E-06

5.57E-05

D ai ly Intak e

(mg/kg/day )

3.80E-07

6.62E-06

4.70E-08

3.99E-07

1.02E-07

7.84E-06

5.57E-05

Slop e Facto r

(mg/kg/day ) -1

1.75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

2.00E-01

Chroni c R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-01

Carcinogeni c Risk

4.96E-07

8.67E-09

Hazard

Quot i ent

9.49E-04

2.21E-02

9.41E-06

5.09E-06

2.12E-04

2.78E-04

Hazard

Quotien t

9.49E-04

2.21E-02

9.41E-06

3.99E-04

2.04E-05

2.12E-04

1.86E-04

Tota l

Risk

5.05E-07

Hazard

Index

2.35E-02

Hazard

Index

2.38E-02
Adul t Visitor

Ingestio n of Tailin g Reasonable Maximum Exposure

1

| Carcinogeni c Risk

i

Chemical

Arseni c Berylliu m

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Arsenic- Berylliu m

Chromium V I

Copper Zin c

i

| Hazard Index -- Chronic

i

Chemical

Antimon y Arsenic Berylliu m

Cadmium (food ) Chromium V I

Copper Zin c

1

1

1

Chemical

Concentratio n

(mg/kg )

6.21E+02

4.69E+00

I

II

Chemical

Concentratio n

(mg/kg )

3.03E+01

6.21E+02

4.69E+00

1.09E+01

7.83E+02

6.28E+03

I

I .•••'" - I : Chemical

Concentratio n

(mg/kg )

3.03E+01

6.21E+02

4.69E+00

4.21E+01

1.09E+01

7.83E+02

6.28E+03

Ingestio n Intake Facto r

(kg/kg/day )

2.58E-08

2.58E-08

I ngest i on Intak e Facto r

(kg/kg/day )

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

Ingestio n Intake Facto r

(kg/kg/day )

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

Dail y Intak e

(ing/kg/day )

1.60E-05

1.21E-07

Dail y Intak e

(mg/kg/day )

2.28E-06

4.67E-05

3.53E-07

8.20E-07

5.89E-05

4^72E-04

D ai ly Intake

(mg/kg/day )

2.28E-06

4.67E-05

3.53E-07

3.17E-06

8.20E-07

5.89E-05

4.72E-04

Slop e Facto r

(mg/kg/day ) -1

1 .75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-01

Carcinogenic Risk

2.80E-05

5.20E-07

Hazard

Quotien t

5.70E-03

1.56E-01

7.06E-05

4.10E-05

1.59E-03

2.36E-03

Hazard

Quot i ent

5.70E-03

1.56E-01

7.06E-05

3.17E-03

1.64E-04

1.59E-03

1.57E-03

Tota l

Risk

2.86E-05

Hazard

Inde x

1.66E-01

Hazard

Index

1.68E-01
Adul t Visitor Inhalatio n of Particulates Average Exposure

1

| Carcinogenic Risk

i

Chemical

Arseni c Berylliu m Cadmium (food ) Chromium V I

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Chromium V I

Manganese

i

| Hazard Index -- Chroni c

i

Chemical

Antimon y Chromium V I

Manganese

1

1

1

Chemical

Concentratio n

(mg/m3 )

9.01E-08

6.39E-10

5.42E-09

1.38E-09

I

II

Chemical

Concentratio n

(mg/m3 )

5.16E-09

1.38E-09

2.42E-07

I

II ... •,.. Chemical

Concentratio n

(mg/m3 )

5,16Er09

1.38E-09

2.42E-07

Inhalatio n Intake Facto r

(m3/kg/day )

6.90E-05

6.90E-05

6.90E-05

6.90E-05

Inhalatio n Intak e Facto r

(m3/kg/day )

5.36E-04

5.36E-04

5.36E-04

Inhalatio n : Intake Facto r

<m3/kg/day )

5.36E-04

5.36E-04

5.36E-04

D ai ly Intak e

(nig/kg/day )

6.21E-12

4.41E-14

3.74E-13

9.52E-14

Dail y Intake Cmg/ kg/day )

•:. 2.77E-12

7.40E-13

1.30E-10

;"" •--::. .

Dail y Intak e

(mg/kg/day )

2.77E-12

7.40E-13

1.30E-10

Slop e Facto r

(mg/ kg/day) - 1

1.50E+01

8.40E+00

6.30E+00

:4.10E+01

Subchronic R F D

(mg/kg/day )

1.14E-04

5.70E-06 .

1.14E-04

Chroni c R F D

(mg/kg/day )

1. HE-04

5.70E-06

1. HE-04

Carcinogenic Risk

9.32E-11

3.70E-13

2.35E-12

3.90E-12

Hazard

Quot i ent

2.43E-08

1.30E-07

1. HE-06

Hazard

Quot i ent

2.43E-08

1.30E-07

1. HE-06

Tota l

Risk

9.98E-11

Hazard

Index

1.29E-06

Hazard

Index

1.29E-06
Adul t Visitor Inhalatio n of Particulates Reasonable Maximum Exposure

1

| Carcinogeni c Risk

i

Chemical

Arsenic Beryl liu n

Cadmium (food ) Chromium V I

i

| Hazard Inde x -- Subchronic

i

Chemical

Antimon y Chromium V I

Manganese

I

| Hazard Index -- Chroni c

i

Chemical

Antimon y Chromium V I

Manganese

1

1

1

Chemical

Concentratio n

(mg/m3 )

1.34E-07

1.01E-09

9.09E-09

2.35E-09

I

II

Chemical

Concentratio n

(mg/m3 )

6.54E-09

2.35E-09

4.45E-07

"I

II

Chemrca I

Concentratio n

(mg/m3 )

6;54E-09

2.35E-09

4.45E-07

Inhalatio n Intak e Facto r

(m3/kg/day )

1.82E-03

1.82E-03

1.82E-03

1.82E-03

Inhalatio n Intak e Facto r

(m3/kg/day )

4.24E-03

4.24E-03

4.24E-03:,.

Inhalatio n Intak e Facto r

(m3/kg/day )

4.24E-03

4.24E-03

4.24E-03

D ai ly Intak e

(mg/kg/day )

2.43E-10

1.84E-12

1.65E-1.1

4.27E-12

D ai l y

intake

(mg/kg/day )

2.77E-11

9.96E-12

1.89E-09

D ai l y Intak e

(mg/kg/day )

2.77E-11

9.96E-12

1.89E-09

Slop e Facto r

(mg/kg/day ) -1

1.50E+01

8.40E+00

6.30E+00

4.10E+01

Subchronic R F D

(mg/kg/day )

1.14E-04

5.70E-06 .

1.14E-04

Chroni c R F D

(mg/kg/day )

1.14E-04

5.70E-06

1.14E-04

Carcinogeni c Risk

3.65E-09

1.54E-11

1.04E-10

1.75E-10

Hazard

Quotien t

2.43E-07

1.75E-06

1.65E-05

Hazard

Quotien t

2.43E-07

1.75E-06

1.65E-05

Tota l

Risk

3.95E-09

Hazard

Index

1.85E-05

Hazard

Index

1.85E-05
Child Visitor

Ingestio n of Tailin g Average Exposure

Carcinogeni c Risk

Chemical

\rsenic

3ery Iliu m

Hazard Index -- Subchronic

Chemical

Antimon y Arsenic" Jerylliu m Chromium V I

Copper

line

Hazard Index -- Chronic

Chemical

Vntimon y Arseni c

.:• Jerylliu m

:admium (food )

:hromium V I

Copper Jin c

1

1

i

Chemical

Concentratio n

(mg/kg )

4.17E+02

2.96E+00

I

II

Chemical

Concentratio n

< mg/kg )

2.39E+01

4.17E+02

2.96E+00

6.41E+00

4.94E+02

3.50E+03

I

| •"•'•'"• • , I

Chemical

Concentratio n

(mg/kg )

2.39E+01

4.17E+02

2.96E+00

2.51E+01

6.41E+00

4.94E+02

3.50E+03

Inges t ion Intake Facto r

(kg/kg/day )

7.66E-09

7.66E-09

I ngest i on Intak e Facto r

(kg/kg/day )

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94ET08

I ngest i on Intak e Facto r

(kg/kg/day )

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

Dail y Intak e

(mg/kg/day )

3.19E-06

2.27E-08

Dail y Intak e

(mg/kg/day )

2.14E-06

3.72E-05

2.65E-07

5.73E-07

4.41E-05

3.13E-04

' ' " "•••- • .

Dail y Intak e

(mg/kg/day )

2.14E-06

3.72E-05

2.65E-07

2.25E-06

5.73E-07

4.41E-05

3.13E-04

Slop e Facto r

(mg/kg/day ) -1

1.75E+00

4.30E+00

: l :

: :

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-01

Carcinogenic Tota l

Risk Risk

5.59E-06

9.75E-08

5.68E-06

Hazard Hazard

Quotien t Index

5.34E-03

1.24E-01

5.29E-05

2.86E-05

1.19E-03

1.57E-03

1.32E-01

Hazard Hazard

Quotien t Index

5.34E-03

1.24E-01

5.29E-05

2.25E-03

1.15E-04

1.19E-03

1.04E-03

1.34E-01
Child Visitor Ingestio n of Tailin g Reasonable Maximum Exposure

Carcinogeni c Risk

Chemical

Vrseni c Jerylliu m

Hazard Index -- Subchronic

Chemical

Vntimon y

\rsenic~

Serylliu m

;hromium VI

:opper

Sin e

Hazard Index -- Chronic

Chemical

\ntimon y

\rsenic

Jerylliu m

:admium (food ) Chromium V I

:opper :. ; Jin c

1

1

1

Chemi ca I

Concentratio n

(mg/kg )

6.21E+02

4.69E+CO

1

1I

Chemical

Concentratio n

(mg/kg )

3.03E+01

6.21E+02

4.69E+00

1.09E+01

7.83E+02

6.28E+03

I

Chemical

Concentratio n

(mg/kg )

3.03E+01

6.21E+02

4.69E+00

4.21E+01

1.09E+01

7.83E+02

6.28E+03

Inges t ion Intak e Facto r

(kg/kg/day )

3.51E-08

3.51E-08

Inges t ion Intak e Facto r

(kg/kg/day )

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

Inges t ion Intak e Factor

(kg/kg/day )

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

D ai l y Intak e

(mg/kg/day )

2.18E-05

1.65E-07

Dail y Intak e

(mg/kg/day )

1.24E-05

2.54E-04

•K92E-06

4.47E-06

3.21E-04

2.57E-03

Dail y Intak e

(mg/kg/day )

1.24E-05

2.54E-04

1.92E-06

1.72E-05

4.47E-06

3.21E-04

2.57E-03

Slop e Facto r

(mg/kg/day)-1

1 .75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

2.00E-01

Chroni c R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-01

Carcinogenic Risk

3.82E-05

7.08E-07

Hazard Quot i ent

3.10E-02

8.48E-01

3.84E-04

2.23E-04

8.67E-03

1.29E-02

Hazard

Quotien t

3.10E-02

8.48E-01

3.84E-04

1.72E-02

8.93E-04

8.67E-03

8.57E-03

Tota l

Risk

3.89E-05

Hazard

Index

9.01E-01

Hazard

Index

9.15E-01
Dirt Biker

Ingestio n of Tailin g Average Exposure

1

| Carcinogenic Risk

i

Chemical

Arseni c Beryl Li urn

i

| Hazard Index -- Subchroni c

i

Chemical

Antimon y Arsenicr Berylliu m Chromium V I

Copper Zin c

I

] Hazard Index -- Chronic

i

Chemical

Antimon y Arseni c Berylliu m : Cadmium (food ) Chromium V I

Copper Zin c

1

1I

Chemical

Concentratio n

(mg/kg )

4.17E+02

2.96E+00

I

i1

Chemical

Concentratio n

(mg/kg )

2.39E+01

4.17E+02

2.96E+00

6.41E+00

4.94E+02

3.50E+03

I

I - "•'• • , I

Chemical

Concentratio n

(mg/kg )

2.39E+01

4.17E+02

2.96E+00

2.51E+01

6.41E+00

4.94E+02

3.50E+03

I ngest i on Intak e Facto r

(kg/kg/day )

2.55E-09

2.55E-09

Ingestio n Intak e Facto r

(kg/kg/day )

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

Ingestio n

Intak e Facto r Ckg/kg/day )

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

Dail y Intak e

(mg/kg/day )

1.06E-06

7.56E-09

Dail y Intak e

(mg/kg/day )

4.75E-07

8.27E-06

5.88E-08

1.27Er07

9.80E-06

6.96E-05

"

: : " "

Dail y Intake .

(mg/kg/day )

4.75E-07

8.27E-06

5.88E-08

4.99E-07

1.27E-07

9.80E-06

6.96E-05

Slop e Facto r

(mg/ kg/day)- !

1.75E+00

4.30E+00

:

: . .

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

2.00E-01

Chroni c R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-01

Carcinogeni c Risk

1.86E-06

3.25E-08

Hazard

Quotien t

1.19E-03

2.76E-02

1.18E-05

6.37E-06

2.65E-04

3.48E-04

Hazard

Quotien t

1.19E-03

2.76E-02

1.18E-05

4.99E-04

2.55E-05

2.65E-04

2.32E-04

Tota l

Risk

1.89E-06

Hazard

Index

2.94E-02

Hazard

Index

2.98E-02
Dirt Biker

Ingestio n of Tailin g Reasonable Maximum Exposure

1

| Carcinogenic Risk

i

Chemical

Arseni c Berylliu m

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Arsenic" Berylliu m Chromium V I

Copper Zin c

i

| Hazard Index -- Chronic

i

Chemical

Antimon y Arseni c Berylliu m

Cadmium (food ) Chromium V I

Copper Z i nc

1

Chemical

Concentratio n

(mg/kg )

6.21E+02

4.69E+00

I

II

Chemical

Concentratio n

(mg/kg )

3.03E+01

6.21E+02

4.69E+OD

1.09E+01

7.83E+02

6.28E+03

I

I

Chemical

Concentratio n

(mg/kg )

3.03E+01

6.21E+02

4.69E+00

4.21E+01

1.09E+01

7.83E+02

6.28E+03

Ingestio n Intak e Facto r

(kg/kg/day )

1.53E-08

1.53E-08

Ingestio n Intake Facto r

(kg/kg/day )

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

Ingestio n Intake Facto r

(kg/kg/day )

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

Dail y Intak e

(mg/kg/day )

9.52E-06

7.19E-08

Dail y Intak e

(rag/ kg/day )

3.61E-06

7.40E-05

5.59E-07

1.30E-06

9.33E-05

7.48E-04

D ai l y Intak e

(mg/kg/day )

3.61E-06

7.40E-05

5.59E-07

5.02E-06

1.30E-06

9.33E-05

7.48E-04

Slop e Facto r

(mg/kg/day ) -1

1.75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-01

Carcinogeni c Risk

1.67E-05

3.09E-07

Hazard

Quotien t

9.03E-03

2.47E-01

1.12E-04

6.50E-05

2.52E-03

3.74E-03

Hazard

Quotient

9.03E-03

2.47E-01

1.12E-04

5.02E-03

2.60E-04

2.52E-03

2.49E-03

Tota l

Risk

1.70E-05

Hazard

Index

2.62E-01

Hazard

Index

2.66E-01
Dirt Biker

Inhalatio n of Particulates Average Exposure

1

| Carcinogeni c Risk

i

Chemical

Arseni c Berylliu m

Cadmium (food ) Chromium V I

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Chromium V I

Manganese

i

| Hazard Index -- Chronic

i

Chemical

Antimon y Chromium V I

Manganese

1

1I

Chemical

Concentratio n

Cmg/m3 )

9.01E-08

6.39E-10

5.42E-09

1.38E-09

I

II

Chemical

Concentratio n

(mg/m3 )

5.16E-09

1.38E-09

2.42E-07

I

II

Chemical

Concentratio n

(mg/m3)

5.16E-09

1.38E-09

2.42E-07

Inhalatio n Intak e Facto r

(m3/kg/day )

1.84E-04

1.84E-04

1.84E-04

1.84E-04

Inhalatio n Intak e Facto r

(m3/kg/day )

1.43E-03

1.43E-03

1.43E-03,

Inhalatio n : : Intak e Facto r

(m3/kg/day )

1.43E-03

1.43E-03

1.43E-03

Dail y Intake

(mg/kg/day )

1.66E-11

1.17E-13

9.97E-13

2.54E-13

D ai ly Intake

(rng/kg/day )

7.38E-12

1.97E-12

3.46E-10 ••... .

D ai ly Intake

(mg/kg/day )

7.38E-12

1.97E-12

3.46E-10

Slop e Facto r

(mg/kg/day ) -1

V.50E+01

8.40E+00

6.30E+00

4.10E+01

Subchronic R F D

(mg/kg/day )

1.14E-04

5.70E-06.

1.14E-04

Chronic R F D

(mg/kg/day )

1.14E-04

5.70E-06

1.14E-04

Carcinogenic Risk

2.49E-10

9.87E-13

6.28E-12

1.04E-11

Hazard

Quot i ent

6.47E-08

3.46E-07

3.04E-06

Hazard

Quotien t

6.47E-08

3.46E-07

3.04E-06

Tota l

Risk

2.66E-10

Hazard

Index

3.45E-06

Hazard

Index

3.45E-06
Dirt Biker Inhalatio n of Particulates Reasonable Maximum Exposure

1

| Carcinogeni c Risk

i

Chemical

Arseni c Berylliu m Cadmium (food ) Chromium V I

I

| Hazard Index -- Subchronic

i

Chemical

Antimon y Chromium V I

Manganese

i

| Hazard Index -- Chronic

i

Chemical

Antimon y Chromium V I

Manganese

1

1

1

Chemical

Concentratio n

(mg/m3 )

1.34E-07

1.01E-09

9.09E-09

2.35E-09

I

II

Chemical

Concentratio n

(mg/m3 )

6.54E-09

2.35E-09

4.45E-07

I

I| •••••••:.: .

Chemical

Concentratio n

(mg/m3 )

6.54E-09

2.35E-09

4.45E-07

Inhalatio n Intak e Facto r

<m3/kg/day )

1.93E-03

1.93E-03

1.93E-03

1.93E-03

Inhalatio n Intak e Facto r

(m3/kg/day )

1.50E-02

1.50E-02

i.50E-02

Inhalatio n Intak e Factor

(m3/kg/day )

1.50E-02

1.50E-02

1.50E-02

D ai ly Intak e

(mg/kg/day )

2.59E-10

1.95E-12

1.75E-11

4.54E-12

Dail y Intake

(mg/kg/day )

9..82E-11

3.53E-11

6.68E-09

D ai ly Intak e

(mg/kg/day )

9.82E-11

3.53E-11

6.68E-09

Slop e Facto r

(mg/kg/day ) -1

1.50E+01

8.40E+00

6.30E+00

4.10E+01

Subchronic R F D

<mg/kg/day )

1.14E-04

5.70E-06 .

1.14E-04

Chronic R F D

(mg/kg/day )

1.14E-04

5.70E-06

1.14E-04

Carcinogenic Risk

3.88E-09

1.64E-11

1.11E-10

1.86E-10

Hazard

Quotien t

8.61E-07

6.19E-06

5.86E-05

Hazard

Quotien t

8.61E-07

6.19E-06

5.86E-05

Tota l

Risk

4.19E-09

Hazard

Index

6.57E-05

Hazard

Index

6.57E-05
Intake Factor: Impounded Tailin g January 28, 1993

16:58

Description : Adul t Visitor-Tailin g Ingestio n Abbreviation : ART G

Pathwa y Type: Ingestio n o f Soi l

Intak e Facto r =

( IR x EF x ED x ME x FI x CF x SS )

( BW x AT )

Parameters:

Carcinogenic Abbreviatio n

I R

EF

ED

ME

F I

CF

- ss

BW

AT

Descriptio n

Ingestio n Rate(mg/day )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Fractio n Contaminated

Conversion Facto r (kg/mg ) Site-specifi c Facto r Body Weigh t (kg ) Averagin g Tim e (dys )

A ve

10

29

3

.5

1

.000001

1

25

25550

R ME

36

87

24

.5

1

.000001

1

57

25550

Non - Carcinogenic A ve

10

29

3

.5

r

.000001

1

25

1095

R ME

36

87

24

.5

1

.000001

1

57

8760

Intake Facto r Calculations :

Carcinogenic: Average Exposure

< 1.00E+01 x 2.90E+01 x 3.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

6.81E-10 =

< 2.50E+01 x 2.56E+04 )

Carcinogenic: Reasonable Maximum Exposure

( 3.60E+01 x 8.70E+01 X 2.40E+01 x 5.00E-01 x 1 x 1.00E-06 x 1 )

2.58E-08 =

< 5.70E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 1.00E+01 x 2.90E+01 x 3.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

1.59E-08 =

( 2.50E+01 x 1.10E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 3.60E+01 x 8.70E+01 x 2.40E+01 x 5.00E-01 x 1 x 1.00E-06 x 1 )

7.53E-08 =

( 5.70E+01 x 8.76E+03 )
Intake Factor: Impounded Tailin g January 28, 1993

16:58

Description : ChiI d Visitor-Tailin g Ingestio n Abbreviation : CRT G

Pathwa y Type: Ingestio n o f Soi l

Intak e Facto r =

( IR x EF x ED x ME x FI x CF x SS )

( BW x AT )

Parameters:

Abbreviatio n

I R

EF

ED

ME F I

CF

- ss

BW

AT

Descriptio n

Carcinogenic A ve RME

Ingestio n Rate(mg/day )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Fractio n Contaminated

36

29

6

.5

1

55

87

6

.5

1

Non-Carcinogeni c A ve RME

36

29

6

•5

1:

55

87

6

.5

1

Conversion Facto r (kg/mg ) .000001 .000001 .000001 .000001

Site-specifi c Factor 1 1 1 1

Body Weigh t (kg ) 16 16 16 16

Averagin g Tim e (dys ) 25550 25550 2190 2190

Intak e Facto r Calculations :

Carcinogenic: Average Exposure

< 3.60E+01 x 2.90E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

7.66E-09 =

( 1.60E+01 x 2.56E+04 )

Carcinogenic: Reasonable Maximum Exposure

( 5.50E+01 x .8.70E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

3.51E-08 =

( 1.60E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 3.60E+01 x 2.90E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

8.94E-08 =

( 1.60E+01 x 2.19E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 5.50E+01 x 8.70E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

4.10E-07 =

( 1.60E+01 x 2.19E+03 )
Intake Factor: Impounded Tailin g January 28, 1993

16:58

Description : Dir t Biker-Tailin g Ingestio n Abbreviation : TUT G

Pathwa y Type : Ingestio n o f Soi l

Intak e Facto r =

IR x EF x ED x ME x FI x CF x SS )

( BU x AT )

Parameters:

Abbreviatio n

I R

EF

ED

HE

F I

CF

- SS

BU

AT

Descriptio n

Carcinogenic A ve RME

Ingestio n Rate(mg/day )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Fractio n Contaminated

25

29

9

.5

1

50

87

9

.5

1.,

Non-Carcinogeni c A ve RME

25

29

9

.5

r

50

87

9

.5

1

Conversion Facto r (kg/mg> .000001 .000001 .000001 .000001

Site-specifi c Facto r 1 1 ; '\ 1

Body Weigh t (kg ) 50 50 50 50

Averagin g Time (dys ) 25550 25550 3285 3285

Intak e Facto r Calculations :

Carcinogenic: Average Exposure

( 2.50E+01 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

2.55E-09 =

( 5.00E+01 x 2.56E+04 )

Carcinogenic: Reasonable Maximum Exposure

( 5.00E+OT x 8.70E*01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 }

1.53E-08 =

( 5.00E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 2.50E+01 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

1.99E-08 =

( 5.00E+01 x 3.29E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 5.00E+01 x 8.70E+01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

1.19E-07 =

( 5.00E+01 x 3.29E+03 )
Intake Factor: Impounded Tailin g January 28, 1993

16:58

Description : Dir t Biker-Tailin g Inhalatio n Abbreviation : TUT !

Pathwa y Type: Inhalatio n

( IR x ET x EF x ED x HE x DF x CC x FI x SS )

Parameters:

( BW x AT >

Carcinogeni c Abbreviatio n

I R

ET

EF

ED

HE

DF

- cc

F I

SS

BW

AT

Descriptio n

Inhalatio n Rate(m3/hr )

Exposure Time(hrs/dy )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Depositio n Factor C i liar y Clearanc e Fractio n Contaminated

Site-Specifi c Facto r Body Weigh t (kg ) Averagin g Time(dys )

A ve

1.2

2

29

9

.5

.75

1

1

...1.

50

25550

R ME

2.1

2

87

:9 '

r; :

 .75

1

1

1

50

25550

Non-Carcinogeni c Ave RME

1.2

2

29

9

.5

.75

.1

r

1.

50

3285

2.1

2

87

9

1

.75

1

1

1

50

3285

Intake Facto r Calculations :

Carcinogenic: Average Exposure

( 1.20E+00 x 2.00E+00 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

1.84E-04 =

( 5.00E+01 x 2.56E+04 )

Carcinogenic: Reasonable Haximum Exposure

( 2.10E+00 x 2.00E+00 x 8.70E+01 x 9.00E+00 x 1 x 7.50E-01 x 1 x 1 x 1 )

1.93E-03 =

( 5.00E+01 x 2.56E+04 >

Non-Carcinogenic: Average Exposure

( 1.20E+00 x 2.00E+00 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

1.43E-03 =

( 5.00E+01 x 3.29E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 2.10E+00 x 2.00E+00 x 8.70E+01 x 9.00E+00 x 1 x 7.50E-01 x 1 x 1 x 1 )

1.50E-02 =

( 5.00E+01 x 3.29E+03 )
Intake Factor: Impounded Tailin g January 28, 1993

16:58

Description : Adul t Visitor-Tailin g Inhalatio n Abbreviation : ARU I

Pathwa y Type: Inhalatio n

C IR x ET x EF x ED x HE x OF x CC x FI x SS )

< BW x AT )

imeters:

Carcinogeni.c Non-Carcinogeni c Abbreviatio n

I R

ET

EF

ED

ME

D F

- cc

F I

S S

BW

AT

Descriptio n

Inhalatio n Rate(m3/hr>

Exposure Time(hrs/dy >

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Depositio n Facto r C i liar y Clearanc e Fractio n Contaminated

Site-Specifi c Facto r Body Weight(kg ) Averagin g Time(dys )

A ve RME Ave

.63 .83 .63

2 2 2

29 87 29

9 30 9

.5

 :

 1/

 :

 .5

.75 .75 .75

1 1 ,

:

 1

1 1 1

1 1 1

70 70. 70

25550 25550 3285

:

"

:

R ME

.83

2

87

30

1

.75

1

1

1

70

10950

i ke Facto r Calculations :

Carcinogenic: Average Exposure

( 6.30E-01 x 2.00E+00 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

( 7.00E+01 x 2.56E+04 )

Careinogenic: Reasonable Maximum Exposure

( 8.30E-01 x 2.00E+00 x 8.70E+01 x 3.00E+01 x 1 x 7.50E-01 x 1 x 1 x 1

1.82E-03 =

( 7.00E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 6.30E-01 x 2.00E+00 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

5.36E-04 =

< 7.00E+01 x 3.29E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 8.30E-01 x 2.00E+00 x 8.70E+01 x 3.00E+01 x 1 x 7.50E-01 x 1 x 1 x 1 )

4.24E-03 =

( 7.00E+01 x 1.10E+04 )
Media Chemical Concentrations: Impounded Tailin g January 28, 1993

16:58

Medium: Tailin g Abbreviation : Tailin g Medium Type: Soi l

Subchronic

Chemical

Antimon y Arseni c Berylliu m

Cadmium (food ) Chromium V I

Copper Manganese Zin c

Average

(mg/kg )

2.39E+01

4.17E+02

2.96E+00

2.51E+01

6.41E+00

4.94E+02

1.12E+03

3.50E+03

R.M.E .

(mg/kg )

3.03E+01

6.21E+02

4.69E+00

4.21E+01

1.09E+01

7.83E+02

2.06E+03

6.28E+03

Chronic/Carcinogenic

Average R.M.E.

(mg/kg ) (mg/kg )

2.39E+01

4.17E+02

2.96E+QO

2.51E+01

6;41E+00

4.94E+02

1.12E+03

3.50E+03

3.03E+01

6,21E+02

4.69E+00

4.21E+01

1.09E+01

7.83E+02

2.06E+03

6.28E+03
Media Chemical Concentrations: Impounded Tailin g January 28, 1993

16:58

Medium: Particulate s

Abbreviation : Dust

Medium Type: Particulat e / VOCs

Subchronic

Chemical

Antimon y Arseni c Beryllium

Cadmium (food ) Chromium V I

Copper Manganese Zin c

Average

Cmg/m3 )

5.16E-09

9.01E-08

6.39E-10

5.42E-09

1.38E-09

1.07E-07

2.42E-07

7.56E-07

R.M.E .

(mg/m3 )

6.54E-09

1.34E-07

1.01E-09

9.09E-09

2.35E-09

1.69E-07

4.45E-07

1.36E-06

Chronic/Carcinogeni c

Average R.M.E .

(mg/m3 ) (mg/m3>

5.16E-09

9.01E-08

6.39E-10

5.42E-09

1.38E-09

1.07E-07

2.42E-07

7.56E-07

6.54E-09

1.34E-07

1.01E-09

9.09E-09

2.35E-09

1.69E-07

4.45E-07

1.36E-06
Chemical List: Fluvial Tailin g January 28, 1993

16:57

Chemical

Antimon y Arsenic Beryl lium

Cadmium (food ) Chromium VI

Copper Manganese Mercury Thalliu m (i n soluble salts ) Zin c

————— — innat e

Slop e Facto r

(mg/kg/day ) -1

1.50E+01

8.40E+00

6.30E+00

4.10E+01

I 1 Ul 1 ———————————— Subchronic R F D

mg/kg/da y

1.14E-04

5.70E-06

1.14E-04

8.60E-05

Chronic R F D

mg/kg/da y

1. 14E-04

5.70E-06

1.14E-04

8.60E-05

Slop e Subchroni c Facto r RF D

(mg/kg/day ) - 1 mg/kg/da y

4.00E-04

1.75E+00 3.00E-04

4.30E+00 5.00E-03

2.00E-02

3.70E-02

3.00E-04

7.00E-04

2.00E-01

Chronic R F D

mg/kg/da y

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-04

7.00E-04

3.00E-01
Receptor/Pathwa y Summary: Fluvial Tailin g January 28, 1993

16:57

Receptor / Pathwa y

Average Exposure

Cancer Subchronic Chronic Risk H.I . H.I .

Reasonable Maximum Exposure

Cancer Subchronic Chronic Risk H.I . H.I .

Adul t Visito r

-- Ingestio n of Tailin g

-- Inhalatio n of Particulate s

2.29E-07 1.18E-02 1.22E-02 1.07E-05 7.15E-02 7.39E-02

4.79E-11 2.10E-06 2.10E-06 1.58E-09 2.87E-05 2.87E-05

2.29E-07 1.18E-02 1.22E-02 1.07E-05 7.15E-02 7.40E-02

Chil d Visito r

-- Ingestio n of Tailin g 2.58E-06 6.64E-02 6.85E-02 1.46E-05 3.89E-01 4.02E-01

2.58E-06 6.64E-02 6.85E-02 1.46E-05 3.89E-01 4.02E-01

Dir t Biker

-- Ingestio n of Tailin g

-- Inhalatio n of Particulate s

8.59E-07 1.48E-02 1.52E-02 6.36E-06 1.13E-01 1.17E-01

1.28E-10 5.59E-06 5.59E-06 1.68E-09 1.02E-04 1.02E-04

8.59E-07 1.48E-02 1.52E-02 6.36E-06 1.13E-01 1.17E-01
Adul t Visitor Ingestio n of Tailin g Average Exposure

1

| Carcinogeni c Risk

i

Chemical

Arseni c Beryl 1 inn

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Arsenic" Berylliu m

Chromium VI

Copper Mercur y Thalliu m (i n solubl e salts ) Zin c

i

| Hazard Inde x -- Chroni c

i

Chemical

Antimon y Arsenic Berylliu m

Cadmium (food ) Chromium V I

Copper Mercury Thalliu m (i n soluble salts ) Zin c

1

1

1

Chemical

Concentratio n

(mg/kg )

1 .89E+02

1.28E+00

Chemical

Concentratio n

(mg/kg )

2.54E+01

1.89E+02

1.28E+00

3.80E+00

3.13E+02

1.52E+00

3.06E+00

6.16E+03

I

I , I

Chemical

Concentratio n

(mg/kg )

2.54E+01

1.89E+02

1.28E+00

3.38E+01

3.80E+00

3.13E+02

1.52E+00

3.06E+00

6.16E+03

Ingestio n Intak e Facto r

(kg/kg/day )

6.81E-10

6.81E-10

Ingestio n Intake Facto r

(kg/kg/day )

1.59E-08

1.59E-08

1.59E-08

1.59E-08

1 .59E-08

1.59E-08

1.59E-08

1.59E-08

""''•'•• •

Ingestio n Intake Facto r

(kg/kg/day )

1.59E-08

1.59E-08

1.59E-08

1.59E-08

1.59E-08

1.59E-08

1.59E-08

1.59E-08

1.59E-08

D ai l y Intak e

(mg/kg/day )

1.29E-07

8.72E-10

: : .

Dail y Intak e

(mg/kg/day )

4.04E-07

3.00E-06

2.03E-08

6.04E-08

4.97E-06

2.42E-08

4.86E-08

9.79E-05

Dail y Intak e

(mg/kg/day )

4.04E-07

3.00E-06

2.03E-08

5.37E-07

6.04E-08

4.97E-06

2.42E-08

4.86E-08

9.79E-05

Slop e Facto r

(mg/kg/day ) -1

1 . 75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

;• 2.00E-02 .

3.70E-02

3.00E-04

7.00E-04

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-04

7.00E-04

3.00E-01

Carcinogeni c Risk

2.25E-07

3.75E-09

Hazard

Quotien t

1.01E-03

1.00E-02

4.07E-06

3.02E-06

1.34E-04

8.05E-05

6.95E-05

4.89E-04

Hazard

Quotien t

1.01E-03

1.00E-02

4.07E-06

5.37E-04

1.21E-05

1.34E-04

8.06E-05

6.95E-05

3.26E-04

Tota l

Risk

2.29E-07

Hazard

Index

1.18E-02

Hazard

Index

1.22E-02
Adul t Visitor Ingestio n of Tailin g Reasonable Maximum Exposure

1

| Carcinogenic Risk

i _

Chemical

Arseni c Beryllium

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Arsenic" Berylliu m

Chromium V I

Copper Mercury Thalliu m (i n soluble salts ) Zin c

i

| Hazard Index -- Chronic

i

Chemical

Antimon y Arsenic Beryllium

Cadmium (food ) Chromium V I

Copper Mercury Thalliu m (i n soluble salts ) Zin c

\

\1

Chemical

Concentration

(mg/kg )

2.33E+02

1 .67E+00

I

II

Chemical

Concentratio n

(mg/kg )

3.79E+01

2.33E+02

1.67E+00

5.54E+00

3.88E+02

3.65E+00

6.57E+00

9.20E+03

I

 : i

i

Chemical

Concentration

(mg/kg )

3.79E+01

2.33E+02

1.67E+00

4.67E+01

5.54E+00

3.88E+02

3.65E+00

:

 6.57E+00

9.20E+03

Ingestio n Intake Facto r

(kg/kg/day )

2.58E-08

2.58E-08

Ingestio n Intak e Facto r

(kg/kg/day )

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

" • : ••::: . Ingestio n Intake Factor

(kg/kg/day )

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

7.53E-08

D ai ly Intake

(mg/kg/day )

6.01E-06

4.31E-08

Dail y Intake

(mg/kg/day )

2.85E-06

1.75E-05

1.26E-07

4.17E-07

2.92E-05

2.75E-07

4.95E-07

6.92E-04

Dail y Intake

(mg/kg/day )

2.85E-06

1.75E-05

1.26E-07

3.52E-06

4.17E-07

2.92E-05

2.75E-07

4.95E-07

6.92E-04

Slop e Facto r

(mg/kg/day) - 1

1.75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

3.00E-04

7.00E-04

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-04

7.00E-04

3.00E-01

Carcinogenic Risk

1.05E-05

1.85E-07

Hazard

Quotien t

7.13E-03

5.85E-02

2.51E-05

2.08E-05

7.89E-04

9.16E-04

7.06E-04

3.46E-03

Hazard

Quotient

7.13E-03

5.85E-02

2.51E-05

3.52E-03

8.34E-05

7.89E-04

9.16E-04

7.06E-04

2.31E-03

Tota l

Risk

1.07E-05

Hazard

Index

7.15E-02

Hazard

Index

7.39E-02
Adul t Visitor Inhalatio n of Particulates Average Exposure

Carcinogeni c Risk

Chemical

^senic

Serylliu m

:admium (food )

:hromium V I

Hazard Index -- Subchronic

Chemical

\ntimony

:hromium V I

Manganese Mercur y

Hazard Index -- Chronic

Chemical

Antimon y Chromium V I

Manganese Mercur y

1

1

1

Chemical

Concentratio n

(mg/m3 )

4.08E-08

2.76E-10

7.30E-09

8.21E-10

I

II

Chemical

Concentratio n

(mg/m3 )

5.49E-09

8.21E-10

4.23E-07

3.28E-10

I

II

Chemi ca I

Concentratio n

(mg/m3 )

5.49E-09

8.21E-10

4.23E-07

3.28E-10

Inhalatio n Intak e Facto r

(m3/kg/day )

6.90E-05

6.90E-05

6.90E-05

6.90E-05

Inhalatio n Intak e Facto r

(m3/kg/day )

5.36E-04

5.36E-04

5.36E-04

5.36E-04

Inhalatio n Intake Factor

(m3/kg/day )

5.36E-04

5.36E-04

5.36E-04

5.36E-04

Dail y Intake

(mg/kg/day )

2.81E-12

1.90E-14

5.03E-13

5.66E-14

Dail y Intake

tmg/kg/day )

2.94E-12

4.40E-13

2.27E-10

T.76E-13

D ai l y Intak e

(mg/kg/day )

2.94E-12

4.40E-13

2.27E-10

1.76E-13

Slop e Facto r

(mg/kg/day)-1

1.50E+01

8.40E+00

6.30E+00

4.10E+01

Subchroni c R F D

(mg/kg/day )

1.14E-04

5.70E-06 .

1.14E-04

8.60E-05

Chronic R F D

(mg/kg/day )

1.14E-04

5.70E-06

1.14E-04

8.60E-05

Carcinogeni c Risk

4.22E-11

1.60E-13

3.17E-12

2.32E-12

Hazard

Quot i ent

2.58E-08

7.72E-08

1.99E-06

2.05E-09

Hazard

Quotient

2.58E-08

7.72E-08

1.99E-06

2.05E-09

Tota l

Risk

4.79E-11

Hazard

Index

2.10E-06

Hazard

Index

2.10E-06
Adul t Visitor Inhalatio n of Particulates Reasonable Maximum Exposure

1

| Carcinogenic Risk

i

Chemical Inhalatio n Dail y Concentratio n Intake Facto r Intak e Chemical

Arsenic Berylliu m

Cadmium (food ) Chromium V I

i

| Hazard Index -- Subchronic

i

(mg/m3 ) <m3/kg/day ) (mg/kg/day )

5.03E-08 1.82E-03 9.14E-11

3.61E-10 1.82E-03 6.56E-13

1.01E-08 1.82E-03 1.84E-11

1.20E-09 1.82E-03 2.18E-12

Chemical Inhalatio n Dail y Concentratio n Intake Facto r Intak e Chemical

Antimon y

Chromium V I

Manganese Mercur y

i

| Hazard Index -- Chronic

i

(rng/m3 ) (m3/kg/day ) (mg/kg/day )

8.19E-09 4.24E-03 3.47E-11

1.20E-09 4.24E-03 5.09E-12

7.39E-07 4.24'E-'03 3.13E-09

7.88E-10 4.24E-03 3.34E-12

•" "" ": ••...

••" ...

..."

 :

 .. Chemical Inhalatio n Dail y Concentratio n Intak e Factor Intak e Chemical

Antimon y Chromium V I

Manganese ,.•••" --'-•"••.

Mercury

(mg/m3 ) (m3/kg/day ) (mg/kg/day )

8.19E-09 4.24E-03 3.47E-11

1.20E-09 4.24E-03 5.09E-12

7.39E-07 4.24E-03 3.13E-09

7.88E-10 4.24E-03 3.34E-12

Slop e Facto r

( mg/kg/day ) -1

1.50E+01

8.40E+00

6.30E+00

4.10E+01

Subchronic R F D

(mg/kg/day )

1.14E-04

5.70E-06.

1. HE-04

8.60E-05

Chronic R F D

(mg/kg/day )

1.14E-04

5.70E-06

1.14E-04

8.60E-05

Carcinogenic Risk

1.37E-09

5.51E-12

1.16E-10

8.94E-11

Hazard

Quotien t

3.05E-07

8.92E-07

2.75E-05

3.88E-08

Hazard

Quotien t

3.05E-07

8.92E-07

2.75E-05

3.88E-08

Tota l

Risk

1.58E-09

Hazard

Index

2.87E-05

Hazard

Index

2.87E-05
Child Visitor

Ingestio n of Tailin g Average Exposure

1

| Carcinogenic Risk

i

Chemical

Arseni c Berylliu m

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Arsenic" Beryllium Chromium V I

Copper Mercury Thalliu m (i n solubl e salts ) Zin c

i

| Hazard Index -- Chroni c

i

Chemical

Antimon y Arseni c Berylliu m

Cadmium (food ) Chromium V I

Copper Mercury Thalliu m (i n solubl e salts ) Zin c

1

1I

Chemical

Concentratio n

(mg/kg )

1.89E+02

1.28E+00

I

II

Chemical

Concentratio n

(mg/kg )

2.54E+01

1.89E+02

1.28E+00

3.80E+00

3.13E+02

1.52E+00

3.06E+00

6.16E+03

I

I : I

Chemical

Concentratio n

(mg/kg )

2.54E+01

1.89E+02

1.28E+00

3.38E+01

3.80E+00

3.13E+02

1.52E+00

3.06E+00

6.16E+03

Ingestio n Intak e Facto r

(kg/kg/day )

7.66E-09

7.66E-09

Ingestio n Intak e Facto r

(kg/ kg/day )

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8,94E-08

8.94E-08

8.94E-08

• ' •'•• • :...,. . .

Ingestio n Intak e Facto r

(kg/kg/day )

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

8.94E-08

D ai l y Intak e

(mg/kg/day )

1.45E-06

9.81E-09

Dail y Intak e

(mg/kg/day )

2.27E-06

1.69E-05

1.14E-07

3.40E-07

2.80E-05

1.36E-07

, 2.74E-07

5.51E-04

Dail y Intak e

(mg/kg/day )

2.27E-06

1.69E-05

1.14E-07

3.02E-06

3.40E-07

2.80E-05

1.36E-07

2.74E-07

5.51E-04

Slop e Facto r

(mg/kg/day ) -1

1..75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

3.00E-04

7.00E-04

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-04

7.00E-04

3.00E-01

Carcinogenic Risk

2.53E-06

4.22E-08

Hazard

Quotien t

5.68E-03

5.63E-02

2.29E-05

1.70E-05

7.56E-04

4.53E-04

3.91E-04

2.75E-03

Hazard

Quotien t

5.68E-03

5.63E-02

2.29E-05

3.02E-03

6.79E-05

7.56E-04

4.53E-04

3.91E-04

1.84E-03

Tota l

Risk

2.58E-06

Hazard

Index

6.64E-02

Hazard

Index

6.85E-02
Child Visitor Ingestio n of Tailin g Reasonable Maximum Exposure

1

| Carcinogenic Risk

i

Chemical

Arseni c Berylliu m

1

| Hazard Index -- Subchronic

i

Chemical

Ant i mony Arsenic Berylliu m Chromium V I

Copper Mercur y Thalliu m (i n solubl e salts ) Zin c

i

| Hazard Index -- Chronic

i

Chemical

Antimon y Arseni c Berylliu m Cadmium (food ) Chromium V I

Copper Mercur y Thalliu m (i n solubl e salts ) Zin c

1

1

1

Chemical

Concentratio n

(rag/kg )

2.33E+02

1.67E+00

II

Chemical

Concentratio n

(mg/kg>

3.79E+01

2.33E+02

1.67E+00

5.54E+00

3.88E+02

3.65E+00

6.57E+00

9.20E+03

I

: |

|

Chemical

Concentratio n

(mg/kg )

3.79E+01

2.33E+02

1.67E+00

4.67E+01

5.54E+00

3.88E+02

3.65E+00

6.57E+00

9.20E+03

Ingestio n Intak e Facto r

(kg/kg/day )

3.51E-08

3.51E-08

Ingestio n Intake Facto r

(kg/kg/day )

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

Ingestio n Intake Facto r

(kg/kg/day )

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

4.10E-07

Dail y Intak e

(mg/kg/day )

8.18E-06

5.86E-08

Dail y Intake

(mg/kg/day )

1.55E-05

9.55E-05

6.84E-07

2.27E-06

1.59E-04

1.50E-06

2.69E-06

3.77E-03

Dail y Intak e

(mg/kg/day )

1.55E-05

9.55E-05

6.84E-07

1.91E-05

2.27E-06

1.59E-04

1.50E-06

2.69E-06

3.77E-03

Slop e Facto r

(mg/ kg/day) - 1

1.75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02.

3.70E-02

3.00E-04

7.00E-04

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-04

7.00E-04

3.00E-01

Carcinogenic Risk

1.43E-05

2.52E-07

Hazard

Quotien t

3.88E-02

3.18E-01

1.37E-04

1.13E-04

4.30E-03

4.98E-03

3.85E-03

1.88E-02

Hazard

Quot i ent

3.88E-02

3.18E-01

1.37E-04

1.91E-02

4.54E-04

4.30E-03

4.98E-03

3.85E-03

1.26E-02

Tota l

Risk

1.46E-05

Hazard

Index

3.89E-01

Hazard

Index

4.02E-01
Dirt Biker

Ingestio n of Tailin g Average Exposure

1

| Carcinogeni c Risk

i

Chemical

Arsenic Berylliu m

i

| Hazard Index -- Subchronic

i

Chemical

Antimon y Arsenic- Berylliu m Chromium V I

Copper Mercury Thalliu m (i n solubl e salts ) Zin c

i

| Hazard Index -- Chronic

i

Chemical

.,...;..-.. . . .

Antimon y Arsenic Berylliu m Cadmium (food ) Chromium V I

Copper Mercur y Thalliu m (i n solubl e salts ) Zin c

1

1I

Chemi ca I

Concentratio n

(mg/kg )

1.89E+02

1.28E+00

I

II

Chemical

Concentratio n

< mg/kg )

2.54E+01

1 .89E+02

1.28E+00

3.80E+00

3.13E+02

1.52E+00

3.06E+00

6.16E+03

:::.":> . .

I

I

|

Chemical

Concentratio n

(mg/kg )

2.54E+01

1.89E+02

1.28E+00

3.38E+01

3.80E+00

3.13E+02

1.52E+00

3.06E+00

6.16E+03

Ingestio n Intake Facto r

(kg/kg/day )

2.55E-09

2.55E-09

Ingestio n Intak e Facto r

(kg/kg/day )

1 .99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

Inges t i on Intake Facto r

(kg/kg/day )

1.99E-08

1.99E-08

1.99E-08

1 .99E-08

1 .99E-08

1.99E-08

1.99E-08

1.99E-08

1.99E-08

D ai ly Intak e

(mg/kg/day )

4.83E-07

3.27E-09

Dail y Intak e

(mg/kg/day )

5.05E-07

3.75E-06

2.54E-08

7.55E~08

6.22E-06

3.02E-08

6.08E-08

1.22E-04

D ai ly Intak e

(mg/kg/day )

5.05E-07

3.75E-06

2.54E-08

6.71E-07

7.55E-08

6.22E-06

3.02E-08

6.08E-08

1.22E-04

Slop e Facto r

(mg/kg/day)-1

1.75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 .

3.70E-02

3.00E-04

7.00E-04

2.00E-01

Chronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-04

7.00E-04

3.00E-01

Carcinogenic Risk

8.45E-07

1.41E-08

Hazard

Quotien t

1.26E-03

1.25E-02

5.08E-06

3.77E-06

1.68E-04

1.01E-04

8.68E-05

6.12E-04

Hazard

Quotien t

1.26E-03

1.25E-02

5.08E-06

6.71E-04

1.51E-05

1.68E-04

1.01E-04

8.68E-05

4.08E-04

Tota l

Risk

8.59E-07

Hazard

Index

1.48E-02

Hazard

Index

1.52E-02
Dirt Biker

Ingestio n of Tailin g Reasonable Maximum Exposure

1

| Carcinogenic Risk

i

Chemical

Arseni c Berylliu m

i] Hazard Index -- Subchronic

i

Chemical

Antimon y Arsenic" Berylliu m Chromium V I

Copper Mercur y Thalliu m (i n solubl e salts ) Zin c

i

| Hazard Index -- Chronic

i

Chemical

Antimon y Arsenic Berylliu m Cadmium (food ) Chromium VI

Copper Mercury Thalliu m (i n solubl e salts ) Zin c

1

1

1

Chemical

Concentratio n

(mg/kg )

2.33E+02

1.67E+00

I

II

Chemical

Concentratio n

(mg/kg )

3.79E+01

2.33E+02

1.67E+00

5.54E+00

3.88E+02

3.65E+00

6.57E+00

9.20E+03

I

I

I : Chemical Concentratio n

(mg/kg )

3.79E+01

2.33E+02

1.67E+00

4.67E+01

5.54E+00

3.88E+02

3.65E+00

6.57E+00

9.20E+03

Ingestio n Intake Facto r

(kg/kg/day )

1.53E-08

1.53E-08

Ingestio n Intak e Facto r

(kg/ kg/day )

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

Ingestio n Intak e Facto r

(kg/kg/day )

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

1.19E-07

D ai ly Intak e

(mg/kg/day )

3.57E-06

2.56E-08

O ai ly Intak e

(mg/kg/day )

4.52E-06

2.78E-05

1.99E-07

6.60E-07

4.62E-05

4.35E-07

7.83E-07

1.10E-03

D ai l y Intake

(mg/kg/day )

4.52E-06

2.78E-05

1 .99E-07

5.57E-06

6.60E-07

4.62E-05

4.35E-07

7.83E-07

1.10E-03

Slop e Facto r

(mg/kg/day ) -1

1.75E+00

4.30E+00

Subchronic R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

2.00E-02 -

3.70E-02

3.00E-04

7.00E-04

2.00E-01

Chroni c R F D

(mg/kg/day )

4.00E-04

3.00E-04

5.00E-03

1.00E-03

5.00E-03

3.70E-02

3.00E-04

7.00E-04

3.00E-01

Carcinogeni c Risk

6.25E-06

1.10E-07

Hazard

Quotien t

1.13E-02

9.26E-02

3.98E-05

3.30E-05

1.25E-03

1.45E-03

1.12E-03

5.48E-03

Hazard

Quotien t

1.13E-02

9.26E-02

3.98E-05

5.57E-03

1.32E-04

1.25E-03

1.45E-03

1.12E-03

3.65E-03

Tota l

Risk

6.36E-06

Hazard

Index

1.13E-01

Hazard

Index

1.17E-01
Dirt Biker

Inhalatio n of Particulates Average Exposure

Carcinogeni c Risk

Chemical

Arsenic Jerylliu m Cadmium (food )

:hromiura V I

Hazard Index -- Subchronic

Chemical

\ntimon y

Chromium V I

langanese

lercury

Hazard Index -- Chroni c

Chemical

\ntimon y

:hromium V I

langanese

lercury

I

1I

Chemical

Concentratio n

<mg/m3)

4.08E-08

2.76E-10

7.30E-09

8.21E-10

I

II

Chemical

Concentratio n

(mg/m3 )

5.49E-09

8.21E-10

4.23E-07

3.28E-10

I

| -"-'"• ,

I

Chemical

Concentratio n

<mg/m3)

5.49E-09

8.21E-10

4.23E-07

3.28E-10

Inhalatio n Intak e Facto r

(m3/kg/day )

1.84E-04

1.84E-04

1.84E-04

1.84E-04

Inhalatio n Intak e Facto r

(m3/kg/day )

1.43E-03

1.43E-03

1743E"03

1.43E-Q3

Inhalatio n Intak e Facto r

(hfi/kg/day )

1.43E-03

1.43E-03

1.43E-03

1.43E-03

D ai l y Intak e

(mg/kg/day )

7.50E-12

5.07E-14

1.34E-12

1.51E-13

Dail y Intake

(ing/kg/day )

7.85E-12

1.17E-12

6.05E-10

4.69E-13

"•••'I.. .

D ai ly Intak e

(mg/kg/day )

7.85E-12

1.17E-12

6.05E-10

4.69E-13

Slop e Facto r

(mg/kg/day ) -1

1.50E+01

8.40E+00

6.30E+00

4.10E+01

Subchronic R F D

(mg/kg/day )

1.14E-04

5.70E-06 .

1.14E-04

8.60E-05

Chronic R F D

(mg/kg/day )

1.14E-04

5.70E-06

1.14E-04

8.60E-05

Carcinogenic Risk

1.13E-10

4.26E-13

8.46E-12

6.19E-12

Hazard

Quotien t

6.89E-08

2.06E-07

5.31E-06

5.45E-09

Hazard

Quotient

6.89E-08

2.06E-07

5.31E-06

5.45E-09

Tota l

Risk

1.28E-10

Hazard

Index

5.59E-06

Hazard

Index

5.59E-06
Dirt Biker Inhalatio n of Particulates Reasonable Maximum Exposure

Carcinogenic Risk

Chemical

\rsenic

Jerylliu m

:admium (food )

:hromium VI

Hazard Index -- Subchronic

Chemical

\ntimony

Chromium V I

Manganese Mercury

Hazard Index -- Chronic

Chemical

Antimon y

Chromium V I

Manganese

 : •lercury

I

1I

Chemical

Concentratio n

Cmg/m3 )

5.03E-08

3.61E-10

1.01E-08

1.20E-09

1

1I

Chemical

Concentratio n

(mg/m3 )

8.19E-09

1.20E-09

7.39E-07

7.88E-10

-I

I • ••••'••• . 1

Chemical

Concentratio n

<mg/m3)

8.19E-09

1 .20E-09

7.39E-07

7.88E-10

Inhalatio n Intake Facto r

(m3/kg/day >

1.93E-03

1.93E-03

1.93E-03

1 .93E-03

Inhalatio n Intak e Facto r

(m3/kg/day )

1.50E-02

1.50E-02

1.50E-02

1.50E-02

Inhalatio n Intak e Facto r

(m3/kg/day )

1.50E-02

1.50E-02

1.50E-02

1.50E-02

D ai l y Intak e

(mg/kg/day )

9.71E-11

6.97E-13

1.95E-11

2.32E-12

Dail y Intake

(rag/ kg/day )

1.23E-10

1.80E-11

1.11E-08

1.18E-11

Dail y Intak e

(mg/kg/day )

1.23E-10

1.80E-11

1.11E-08

1.18E-11

Slop e Facto r

(mg/kg/day ) -1

1.50E+01

8.40E+00

6.30E+00

4.10E+01

Subchronic R F D

(mg/kg/day )

1. HE-04

5.70E-06 .

1. HE-04

8.60E-05

Chronic R F D

(mg/kg/day )

1. HE-04

5.70E-06

1. HE-04

8.60E-05

Carcinogenic Risk

1.46E-09

5.85E-12

1.23E-10

9.50E-11

-

Hazard

Quotient

1.08E-06

3.16E-06

9.73E-05

1.38E-07

Hazard

Quotien t

1.08E-06

3.16E-06

9.73E-05

1.38E-07

Tota l

Risk

1.68E-09

Hazard

Index

1.02E-04

Hazard

Index

1.02E-04
Intake Factor: Fluvial Tailin g January 28, 1993

16:57

Description : Adul t Visitor-Tailin g Ingestio n Abbreviation : ART G

Pathwa y Type: Ingestio n o f Soi l

Intak e Facto r =

< IR x EF x ED x HE X FI x CF x SS )

( BU x AT )

Parameters:

Abbreviatio n

I R

EF

ED

ME

F I

CF

- SS

BU

AT

Descriptio n

Carcinogeni c A ve RME

Ingestio n Rate(mg/day )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Fractio n Contaminated

10

29

3

.5

1

36

87

24

.5

1

Non-Carcinogeni c A ve RME

10

29

3

.5

1

36

87

24

.5

1

Conversion Facto r (kg/mg ) .000001 .000001 .000001 .000001

Site-specifi c Facto r 1 1 1 1

Body Weigh t (kg ) 25 57 25 57

Averagin g Time (dys ) 25550 25550 1095 8760

Intak e Facto r Calculations :

Carcinogenic: Average Exposure

( 1.00E+01 x 2.90E+01 x 3.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

6.81E-10 =

( 2.50E+01 x 2.56E+04 )

Carcinogenic: Reasonable Maximum Exposure

( 3.60E+OT x 8.70E+01 x 2.40E+01 x 5.00E-01 x 1 x 1.00E-06 x 1 )

2.58E-08 =

( 5.70E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 1.00E+01 x 2.90E+01 x 3.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

1.59E-08 =

( 2.50E+01 x 1.10E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 3.60E+01 x 8.70E+01 x 2.40E+01 x 5.00E-01 x 1 x 1.00E-06 x 1 )

7.53E-08 =

( 5.70E+01 x 8.76E+03 )
Intake Factor: Fluvial Tailin g January 28, 1993

16:57

Description : ChiI d Visitor-Tailin g Ingestio n Abbreviation : CRT G

Pathwa y Type : Ingestio n o f Soi l

Intak e Facto r =

Parameters:

< IR x EF x ED x ME x FI x CF x SS )

( BW x AT )

Carcinogenic Abbreviatio n

I R

EF

ED

ME

F I

CF

- ss

BW

AT

Descriptio n

Ingestio n Rate(mg/day )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Fractio n Contaminated

Conversion Facto r (kg/mg ) Site-specifi c Facto r Body Weigh t (kg ) Averagin g Tim e (dys )

A ve

36

29

6

.5

1

.000001

1

16

25550

R ME

55

87

6

,

:

.5 .

1

.000001

1

16

25550

Non - Carcinogenic A ve

36

29

6

.5

1

.000001

1

16

2190

R ME :

55

87

6

.5

1

.000001

1

16

2190

Intak e Facto r Calculations :

 :

Carcinogenic: Average Exposure

( 3.60E+01 x 2.90E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

7.66E-09 =

( 1.60E+01 x 2.56E+04 )

Carcinogenic: Reasonable Maximum Exposure

( 5.50E+OT x 8.70E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 >

3.51E-08 =

( 1.60E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 3.60E+01 x 2.90E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

8.94E-08 =

( 1.60E+01 x 2.19E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 5.50E+01 x 8.70E+01 x 6.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

4.10E-07 =

C 1.60E+01 x 2.19E+03 )
Intake Factor: Fluvial Tailin g January 28, 1993

16:57

Description : Dir t Biker-Tailin g Ingestio n Abbreviation : TUT G

Pathwa y Type: Ingestio n o f Soi l

Intak e Facto r =

( IR x EF x ED x HE x FI x CF x SS )

( BW x AT )

Parameters:

Abbreviatio n

I R

EF

ED

ME F I

CF

- SS

BW

AT

Descriptio n

Carcinogenic A ve RME

Ingestio n Rate(mg/day )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Fractio n Contaminated

25

29

9

.5

1

50

87

9

• 5

1

Won-Carcinogeni c A ve RME

Conversion Facto r (kg/mg ) .000001 .000001

Site-specifi c Facto r 1 1

Body Heigh t (kg ) 50 50

Averagin g Tim e (dys ) 25550 25550

25 50

29 87

9 9

.5 .5

1 1

.000001 .000001

1

50

3285

1

50

3285

Intak e Facto r Calculations :

Carcinogenic: Average Exposure

( 2.50E+01 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1

2.55E-09 =

C 5.00E+01 x 2.56E+04 )

Carcinogenic: Reasonable Maximum Exposure

( 5.00E*01 x 8.70E+01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

1.53E-08 =

( 5.00E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 2.50E+01 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1

1.99E-08 =

( 5.00E+01 x 3.29E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 5.00E+01 x 8.70E+01 x 9.00E+00 x 5.00E-01 x 1 x 1.00E-06 x 1 )

1.19E-07 =

( 5.00E+01 x 3.29E+03 )
Intake Factor: Fluvial Tailin g January 28, 1993

16:57

Description : Dir t Biker-Tailin g Inhalatio n Abbreviation : TUT I

Pathwa y Type: Inhalatio n

( IR x ET x EF x ED x HE x DF x CC x FI x SS

Intak e Facto r =

Parameters:

Abbreviatio n

I R

ET

EF

ED

HE

D F

- CC

F I

SS

BW

AT

( BW x AT )

Carcinogenic Descriptio n

Inhalatio n Rate(m3/hr )

Exposure Time(hrs/dy ) Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Hatri x Effec t

Depositio n Facto r Ciliar y Clearance Fractio n Contaminated

Site-Specifi c Facto r Body Weight(kg ) Averagin g Time(dys )

A ve

1.2

2

29

9

.5

.75

1

1

1

50

25550

R HE

2.1

2 ,:

:

 '

87

9

1

.75

1 ^

1

1

50

25550

Non-Carcinogeni c A ve RHE

1.2

2

29

.5

.75

1

1

Y ,

50

3285

2.1

2

87

9

1

.75

1

1

1

50

3285

Intak e Facto r Calculations :

Carcinogenic: Average Exposure

( 1.20E+00 x 2.00E+00 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

< 5.00E+01 x 2.56E+04 )

Carcinogenic: Reasonable Haximum Exposure

( 2.10E+00 x 2.00E+00 x 8.70E+01 x 9.00E+00 x 1 x 7.50E-01 x 1 x 1 x 1 )

1.93E-03 =

( 5.00E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

< 1.20E+00 x 2.00E+00 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

1.43E-03 =

< 5.00E+01 x 3.29E+03 )

Non-Carcinogenic: Reasonable Haximum Exposure

( 2.10E+00 x 2.00E+00 x 8.70E+01 x 9.00E+00 x 1 x 7.50E-01 x 1 x 1 x 1 )

1.50E-02 =

( 5.00E+01 x 3.29E+03 )
Intake Factor: Fluvial Tailin g January 28, 1993

16:57

Description : Adul t Visitor-Tailin g Inhalatio n Abbreviation : ARU I

Pathway Type: Inhalatio n

( IR x ET x EF x ED x HE x DF x CC x FI x SS )

iiiua^cr rat-tin —

Parameters:

( BW x AT >

Carcinogenic Abbreviatio n

I R

ET

EF

ED

ME

D F

- cc

F I

SS

BW

AT

Descriptio n

Inhalatio n Rate(m3/hr )

Exposure Time(hrs/dy )

Exposure Freq.(dys/yr )

Exposure Duration(yrs ) Matri x Effec t

Depositio n Facto r Ciliar y Clearance Fractio n Contaminated

Site-Specifi c Facto r Body Height(kg ) Averagin g Time(dys )

A ve

.63

2

29

9

.5

.75

1

1

1

70

25550

R ME

.83

2

87

30

1

'••:.75 : : -

:

1

1

1

70

25550

Non-Carcinogeni c A ve RME

.63

2

29

9

.5

.75

1

70

3285

.83

2

87

30

1

.75

1

1

1

70

10950

Intak e Facto r Calculations :

Carcinogenic: Average Exposure :

( 6.30E-01 x 2.00E+00 x 2.90E+OT x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

6.90E-05 =

( 7.00E+01 x 2.56E+04 )

Carcinogenic: Reasonable Maximum Exposure

( 8.30E-01 x 2.00E+00 x 8.70E+01 x 3.00E+01 x 1 x 7.50E-01 x 1 x 1 x 1 )

1.82E-03 =

< 7.00E+01 x 2.56E+04 )

Non-Carcinogenic: Average Exposure

( 6.30E-01 x 2.00E+00 x 2.90E+01 x 9.00E+00 x 5.00E-01 x 7.50E-01 x 1 x 1 x 1 )

5.36E-04 =

( 7.00E+01 x 3.29E+03 )

Non-Carcinogenic: Reasonable Maximum Exposure

( 8.30E-01 x 2.00E+00 x 8.70E+01 x 3.00E+01 x 1 x 7.50E-01 x 1 x 1 x 1 )

4.24E-03 =

( 7.00E+01 x 1.10E+04 )
Media Chemical Concentrations: Fluvial Tailin g January 28, 1993

16:57

Medium: Particulate s

Abbreviation : Dust

Medium Type : Particulat e / VOCs

Subchronic

Chemical

Average R.M.E . Cmg/m3> (mg/m3 )

Chronic/Carcinogeni c

Average R.M.E .

(mg/m3 ) <mg/m3)

Antimon y Arseni c Berylliu m Cadmium (food ) Chromium V I

Copper Manganese Mercury Thalliu m (i n solubl e salts ) Zin c

5.49E-09

4.08E-08

2.76E-10

7.30E-09

8.21E-10

6.76E-08

4.23E-07

3.28E-10

2.94E-10

1.33E-06

8.19E-09

5.03E-08

3.61E-10

1.01E-08

1.20E-09

8.38E-08

7.39E-07

7.88E-10

3.41E-11

1.99E-06

5.49E-09

4.08E-08

2.76E-10

7.30E-09

8.21E-10

6.76E-08

4.23E-07

3.28E-10

2.94E-10

1.33E-06

8.19E-09

5.03E-08

3.61E-10

1.01E-08

1.20E-09

8.38E-08

7.39E-07

7.88E-10

3.41E-11

1.99E-06
Media Chemical Concentrations: Fluvial Tailin g

Medium: Tailin g Abbreviation : Tailin g Medium Type: Soi I

Subchronic

Chemical

Antimon y Arseni c Beryllium

Cadmium (food ) Chromium V I

Copper Manganese Mercury Thalliu m (i n solubl e salts ) Zin c

Average

(mg/kg )

2.54E+01

1.89E+02

1.28E+00

3.38E+01

3.80E+00

3.13E+02

1.96E+03

1.52E+00

3.06E+00

6.16E+03

R.M.E .

(mg/kg )

3.79E+01

2.33E+02

1.67E+00

4.67E+01

5.54E+00

3.88E+02

3.42E+03

3.65E+00

6.57E+00

9.20E+03

Chronic/Carcinogeni c

Average

(mg/kg )

2.54E+01

1.89E+02

1.28E+00

3.38E+01

3.80E+00

3.13E+02

1.96E+03

1.52E+00

3.06E-I-00

6.16E+03

R.M.E .

(mg/kg )

3.79E+01

2.33E+02

1.67E+00

4.67E+01 : 5.54E+00

3.88E+02

3.42E+03

3.65E+00

6.57E+00

9.20E+03

January 28, 1993

16:57
APPENDIX G

SUMMARY OF THE CHEMICA L DATA QUALITY ASSURANCE PROCEDURES

22909C-22327/R22.TS 01-28-93(7:42pm)/RPT/10
APPENDIX G

SUMMAR Y OF THE CHEMICA L DATA QUALITY ASSURANCE PROCEDURES

T he Tailin g Disposal Area Remedial Investigation (RI) was performed in accordance

with the requirements of the Administrative Consent Order 91-19 (AGO) .

T he approved Tailin g Disposal Area Samplin g and Analysis Plan (SAP) incorporated by

reference Samplin g and Analysis Plan, Revision 3.1, Yak Tunnel Operable Unit , California Gulch, Leadville, Colorado (Res-ASARCO 1990), with modification s to the

Y ak Tunnel SAP described in Section 3.0 of the Tailin g Disposal Area work plan. The

Tailin g Disposal Area Work Plan included the Field Samplin g Plan (FSP), which

describes the sampling tasks required for the remedial investigation of tailings disposal

sites and potential fluvial tailings deposit s located within the California Gulch Site. Standard Operating Procedures (SOPs) incorporated those described in Sections 8.0 and

9.0 of the Yak Tunnel QAPP, with modifications to the procedures described in the

Appendix A of the Tailin g Disposal Area Work Plan. Collection and handlin g of the

samples was conducted in accordance with the FSP, and ASARCO was responsible for

t he management of the field samplin g program.

Chemical analyses were performed by CLP methods from the CLP Statement of Work

S OW 7/88 (EPA 1988) and SOW ILM01.0 (EPA 1990), and Methods for Chemical

Analysis of Water and Wastes (EPA 1983). Coordination and oversight of the

laboratories and analytical program was undertaken by ASARCO.

T he laboratory responsibilities , the analytical parameters and analytical procedures, and

the QA/QC requirements were those specified in Section 5.0 of the Yak Tunnel QAPP. Minor changes to these specification s were described in Section 3.2 of the Tailin g Disposal Area Work Plan. These requirements are designed to assure that the quality

of analytical measurements is known and that a determination can be made as to the

acceptability of the data for intended uses.

Reporting of results by the laboratories was in compliance with appropriate CLP

protocol s for CLP analyses, and provision of analytical results and QA/QC summary

22909C-22327/R22.G 01-28-93(8:44pm)/RPT/16 G-l
sheets for non-CLP methods. Raw data packages were supplied for all analyses,

including non-CLP methods, for reference during data validation. Solid samples were

analyzed for total metals, or the short metals list comprising arsenic, lead, cadmium, and

zinc. Selected solids samples were evaluated for leachable metals. These samples were

extracted followin g the SW-846 Method 1312 leaching procedure, and the leachate

analyzed for the target metals. Water samples were analyzed for dissolved and total

metals, and major water quality constituents.

Data validation was conducted by data validation companies contracted by ASARCO. To establish consistent validation procedures, the companies conducting the data

validation followed the checklist 'Data Validatio n Forms, Part III, Attachment A of the

QAPP, from the Yak Tunnel SAP. Each stage of the data analysis, data reporting, and

data validation was monitored by ASARCO,

The purpose of the validation was to assess the data quality, and was a process to review

t he analytical data generated through the overall analytical program. After performin g

the validation steps, some data failed the acceptance criteria, and in accordance with the

QAPP such data are qualified as estimated, or not detected, and in extreme cases data

are rejected as unusable. Data that have been qualified as estimated, and flagged T,

denote that the presence of the analyte was positively identified, but the associated

numerical value is the approximate concentration of the analyte in the sample. Data

that were flagged 'R denote that these sample results are rejected due to serious

deficiencies in the ability to analyze the sample, and that the presence or absence of the

analyte cannot be verified.

F or analyses conducted under the Tailin g Disposal Area Work Plan, positive results

reported between the Instrument Detection Limit (IDL) and the Contract Required

Detection Limit (CRDL) were reported as non-detect at the CRDL value, as specified

in the approved data validation protocols. For analyses conducted under the Surface

Water Work Plan (Surface Water Work Plan, California Gulch Site, Leadville, Colorado, WCC, March 1991), positive results reported between the IDL and the CRDL were

qualified as estimated, and flagged M, and non-detect data were reported at the IDL,

a n d flagged 'U, as specified in the validation protocols.

22909C-22327/R22.G 01-28-93(8:44pm)/RPT/16 G-2
T he quality assurance objectives of accuracy, precision, completeness, comparability, and

representativeness were described in Sections 6.2 and 6.3 of the Yak Tunnel SAP and

were reviewed as part of the data validation process. Minor modification s to the

analytical program from the Yak Tunnel SAP are noted in the Section 3.2 of the Tailin g Disposal Area Work Plan.

Accuracy was assessed by evaluation of the percent recoveries of spiked analytes in the

laboratory control samples, CRDL standard analyses, matrix spike samples, and for

graphite furnace atomic absorption analyses, the post digestion spike samples. Precision

w as assessed by evaluating the relative percent difference (RPD) obtained from results

of laboratory control samples, duplicate samples, and field duplicate samples. The data

validation process provides an assessment of accuracy and precision through the

evaluation of field and laboratory QC sample results and a comparison to acceptance

limits specified in the QAPP.

Analytical representativeness is the degree to which data accurately and precisely

represent the concentration of target compounds in the samples, and was evaluated by

analyzing two split s from each homogenized field sample assigned as a field duplicate

sample. The data validation process provided an assessment of representativeness

through the comparison and RPDs for each analyte of the field duplicate pair.

Data outside of the acceptance limits for accuracy, precision, and representativeness

were assigned T or 'R qualifiers in accordance with the validation protocols.

The overall completeness of the collected and analyzed data was assessed on a regular

basis in the monthly, quarterly, and annual reports, in accordance with the Yak Tunnel

SAP. Laboratory completeness was assessed as part of the validation process.

Comparability expresses the confidence with which one data set can be compared with

another. Qualitatively , differen t data sets can be considered to be comparable as the

data were analyzed followin g the same analytical protocols, and were validated followin g

the same validation procedures.

22909C-22327/R22.G 01-28-93(8:44pm)/RPT/16 G~3
ASARCO was responsible for the data management, including verification of data, data

entry, data storage, and reporting and transmittal of data. The database was maintained

at the ASARCO office s in Golden. The validated data, including assigned qualifiers ,

were manually entered or electronically loaded into the database. Followin g entry of the

data in the ASARCO database, hard copy or disk copies of the validated data were

supplied to WCC on an ongoing basis. WCC then entered the ASARCO-supplied data

into the WCC database system for use in reporting and interpretation. Where required,

changes to the initially reported data were taken from corrected data reports supplied

by ASARCO.

T he data reports extracted from the WCC database formed the basis for data

interpretation during compilation of the Remedial Investigation (RI) Reports, and data

use was based on this data, including estimated data. As the validation process

evaluated the data quality, and appropriate data qualifiers were assigned during the

validation , the data were used as reported from the database . With the exception of

data rejected during the validation process, all data, including estimated data, were

considered to meet the quality assurance objectives and to be usable for the purposes

of compiling the Tailin g Disposal Area RI. The data sufficiency and usability to meet

the specific project objectives under the RI is discussed in Section 13.

22909C-22327/R22.G 01-28-93(8:44pm)/RPT/16 G-4