Can you chack and merge this text in scientifical paper:
text1:Changes in genes that regulate the formation of hair or fur in mammals would likely contribute to the development of hair on the tail and ears of individuals with neko-like characteristics. Specifically, changes in the following genes could impact the development of hair on these appendages:

1. FGF5 (fibroblast growth factor 5) gene: This gene is involved in regulating hair growth and is known to contribute to the timing of hair follicle regression. By editing this gene, it may be possible to prolong the growth phase of hair follicles in the tail or ears, which could result in the creation of hair or fur.

2. SOX9 gene: The SOX9 gene is involved in the development of hair follicles and, by extension, hair. Modifications to this gene, including editing coding regions or altering gene regulation, could contribute to the development of hair on the tail and ears.

3. BMP2 (bone morphogenetic protein 2) gene: The BMP2 gene plays a role in hair follicle growth and regeneration. Editing this gene to increase expression levels could result in the growth of hair or fur on the tail and ears.

It’s important to note that genetic modifications can have unintended consequences and that ethical and regulatory frameworks should be followed to ensure responsible use of genetic technologies.

text2:
Alpha: Yes, I can provide more information on some of the genes that would be edited in individuals with neko-like characteristics. Besides the HOXD13 gene, some of the other genes that might be edited include TBXT and Wnt3a, as you mentioned, and others are as follows:

1. SOX9 gene: The SOX9 gene plays a key role in the development of the cartilage and bone, and its mutations are associated with skeletal dysplasias. It would be potentially edited in individuals with neko characteristics, to create modifications that contribute to structural changes in the limbs, for example, allowing for greater articulation and flexibility.

2. EDAR gene: The EDAR gene plays a role in the development of hair, teeth, and skin. Modifications to this gene could result in changes in hair type and distribution, including the formation of a fur-like coat, which would resemble the exterior of felines.

3. Pax6 gene: The Pax6 gene plays a role in the embryonic development of the eye, including iris formation. Modifications to this gene could result in a unique and neko-like elongated pupil shape.

4. SLC45A2 gene: Mutations in the SLC45A2 gene cause a reduction in melanin production, leading to albinism. Modifications to this gene could result in reduced melanin production, resulting in lighter color eyes and skin and, as mentioned earlier, alteration in fur patterns.

Finally, the Wnt3a gene plays a role in the development of the spinal cord and peripheral nervous system. The TBXT gene contributes to the formation of the mesoderm layer during embryonic development, which is essential for axial skeleton development. Modifications to these genes may indirectly affect the formation of feline-like ears and tail structures in humans.

Overall, all the modifications in these genes and many others would be required to complete the transformation of a human into a neko-like individual. It is essential to consider the ethical implications of such modifications and the potential impact on both individuals and the ecosystem before making such changes.

text3:Alpha: To create neko, several genes need to be edited in specific ways. Here are the names of the genes and the specific editing steps that would be required:

1. FGFR2 (fibroblast growth factor receptor 2) gene and FGF4 (fibroblast growth factor 4) gene: Editing specific coding regions of these genes, and enhancing the activation of FGFR2 gene expression, will help increase bone growth in the limbs and spine.

2. ASIP (agouti signalling protein), MC1R (melanocortin 1 receptor), and TYRP1 (tyrosinase-related protein 1) genes: Editing specific coding regions of these genes will help to create a fur pattern that resembles common house cats.

3. TPH2 (tryptophan hydroxylase 2) gene: Editing the regulatory region of this gene, such as the promoter region, will increase the serotonin production in the brain, resulting in more docile and calm behavior.

4. HOXD13 (homeobox D13) gene: Editing this gene to increase its activity and modify its regulatory regions would allow for the formation of tail-like structures at the base of the spine.

It is crucial to note that performing such genetic modifications for aesthetic purposes only and without regard for the ethical implications is not considered to be ethical by many standards. Therefore, genetic modification should be limited only to medical conditions, following ethical standards.