Below is an article I wrote. I need you to come up with 5 keywords, an abstract, introduction/background, and conclusion

Endovenous laser thermal ablation (EVLA) is an effective, minimally invasive treatment option for incompetent truncal varicosities of the lower extremities that has largely eclipsed surgical management. Current devices lead to irreversible transmural vein wall thermal damage and subsequent fibrotic occlusion of targeted vessels by emitting wavelengths ranging from 810 to 1940 nm. Knowledge gaps regarding the underlying physical and biochemical effects of EVLA led to the assumption that intravascular coagulation temperatures are achieved by selective photothermolysis of hemoglobin in red blood cells using shorter wavelengths (810, 940, 980, and 1064 nm) or of water in the vein wall using longer wavelengths (1320, 1470, 1500, and 1940 nm).1-4
	A wealth of recent data from mathematical models of EVLA, however, has completely reworked our understanding of the mechanisms of action responsible for clinical and histologic outcomes. A mathematical model by van Ruijven et al5 found that blood and vein wall temperature is essentially independent of wavelength, conforming with the physiology of blood, which is 15% hemoglobin and over 60% water.6 A similar model by Poluektova et al7 not only showed that absorption of laser light by the inner vein wall is wavelength independent, but that with 1470 nm energy, which should lead to even greater water wall absorption if the previous assumption is correct, virtually no photons reach the inner vein wall. In fact, photothermal effects contributed to no more than 20% of the increase in total energy at the inner vein wall, provided that the vein was totally collapsed. Endovenous laser light is essentially absorbed only by residual intraluminal blood surrounding the fiber, not by the wall itself. With sufficient laser power, coagulation temperatures are achieved regardless of whether hemoglobin or water is the target chromophore.6,7 Wavelengths in the mid-infrared spectrum (e.g. 1940 nm) optimally target water, leading to highly efficient heat transfer within blood plasma or water.8
	Based on the available recent evidence, we can therefore no longer assume that irreversible thermal damage of the inner vein wall, a requirement for permanent vessel occlusion by EVLA, is dependent on competitive direct photothermal absorption by hemoglobin or water. Instead, thermal damage of the vein wall is achieved by heat transfer through a small residual layer of intraluminal blood from a hot (950-1100°C) layer coating the fiber tip in combination with photothermal absorption of emitted light by both chromophores within that blood.7 Both heat flow mechanisms may act on the wall by producing boiling steam bubbles, which form on the hot fiber tip or in the intraluminal space, respectively, and diffuse downstream.5-7,9 The hot layer of coagulated blood around a bare fiber tip can lead to carbonization, which can come in direct contact with the vein wall and lead to thermal damage by conduction.10 Reducing vein diameter during treatment with Trendelenburg positioning and tumescent fluid infiltration keeps the amount of residual intraluminal blood to a necessary minimum, decreasing the distance for heat transfer to the vein wall and enhancing efficacy.6,11