Translate the following text from English to the Tunisian dialect. Please follow these steps carefully:
1. *Step-by-step translation*: Translate the text in chunks, and for each part, explain how the meaning is adapted to the Tunisian dialect, especially focusing on informal expressions and cultural references.
2. *Compare with Standard Arabic*: Highlight differences between the Tunisian dialect and Standard Arabic for any phrase or sentence, particularly noting where Tunisian dialect is distinct.
3. *Selective confidence*: If there are any parts where you're confident about translating into Tunisian dialect, highlight them explicitly. If there are any parts you're unsure about translating directly into dialect, explain the challenge and how you'd adapt them.

Text to be translated:

Introduction {#Sec1} ============ A hallmark of immunity is the intrinsic ability to recognize and eliminate foreign molecules, cells, and organisms. The adaptive immune system is comprised of B and T cells. During T and B cell development these cells express unique heterodimeric receptors that can be used in pathogen recognition. Each of these receptor chains is generated by a somatic rearrangement process that joins different segments of the TCR and BCR genes and creates a novel gene. This joining process is imprecise with insertion of non-templated nucleotides (N nucleotides) in the junction site, as well as 3′- and 5′-nucleotide deletion from the germline genes participating in the rearrangement. This region of random nucleotide insertion or deletion referred to as the third complementarity-determining region (CDR3). The resulting CDR3 have a unique nucleotide sequence that is specific to that particular B or T cell and all its progeny; hence, the clonotypic nature of the receptors. The CDR3 is the portion of these receptors that is most involved in interactions with intact soluble antigens (B cells) or intracellular processed antigens presented as immunogenic peptides loaded in MHC molecules (T cells) The initial phase of the adaptive immune responses involves B and T cell clonal selection on the basis of the structural complementarity of antigen-specific receptors to pathogen-derived epitopes (Davis and Chien [@CR6]; Kolar and Capra [@CR11]). The cells recruited into the immune response execute their effector function role. After pathogen clearance, a proportion of these cells will be retained as memory. Memory provides more rapid and effective immune protection against recurring pathogen present in the environment. The collection of cells that respond to a particular pathogen is referred to as the repertoire. T and B cells can also be implicated in responses to non-pathogenic environmental stimuli (allergies). More serious is the lack of tolerance to self that results in responses to self-antigens giving rise to autoimmune disease. In each case, a repertoire of allergen- or self-specific B or T cells is generated. The repertoire recognizing a molecule would be a sum of the repertoires responding against all the component epitopes of the molecule. The repertoire against an organism would be the sum of all the repertoires against all the molecules from the pathogen. Measuring an immune response at the level of the repertoire is becoming very common (Correia-Neves et al. [@CR4]; La Gruta et al. [@CR12]; Naumov et al. [@CR18]; Pewe et al. [@CR20]; Probert et al. [@CR21]; Venturi et al. [@CR26]). An antigen-specific response can be viewed in the context of how many T cells are recruited and the structure of their antigen receptors. The nature of the naïve and antigen-experienced cells repertoire is of interest in basic and clinical immunology, immune-pharmaceutics, and vaccine development. However, comparison of datasets from similar, or even identical, experiments from different laboratories is cumbersome due to lack of the unified clonal identification procedure where the clonotypic antigen-receptor serves a marker of clonal identity. Having a quick way to assign specific identifiers for specific receptor sequence would