this is reviewer question and my answer: 5. In the third paragraph of the discussion you discuss the reduced structural connectivity of the different networks. Please improve the structure of this part by providing clear transitions when you move from one network to the next. Also, avoid just listing previous findings, but make sure to relate them to your findings.

Thank you for your comments on the third paragraph of our discussion. We have revised this section to improve the structure by providing clear transitions when moving from one network to the next and relating previous findings to our findings.
“Recent developments in neuroscience have highlighted the role of brain networks, despite the localization perspective that has been prevalent in the field (Heuvel). Brain networks are a collection of interconnected brain regions with correlated neural activity; they help in understanding brain function in healthy and clinical populations. Research into sleep disorders, both pharmacological and psychological, should be improved with a focus on abnormal brain network activity.
In our research, we examined the seven standard networks from the Schaefer atlas [1]. We discovered that individuals with insomnia had decreased structural connectivity in the frontoparietal network. This network is in charge of executive control that is goal-oriented [2]. The frontoparietal network is also responsible for working memory and problem-solving abilities, which have been found to be compromised in people with insomnia [2, 3].  
The management of motor movements and bodily sensations falls under the responsibility of the SM network. According to our NBS-Predict analysis, there was a decline in structural connectivity within this network. The SM network is closely related to sleep quality, and its subregions undergo changes [4]. In addition, the functional connectivity between the SM and cingulo-opercular control networks partially mediates the link between psychological stress and sleep quality. This indicates that a person’s psychological stress has a significant impact on their sleep quality and that an increase in functional connectivity between the SM and CO networks explains the relationship between psychological stress and poor sleep quality. [4]. Moreover, reduced functional connectivity in the SM network suggests a possible relationship between structural and functional changes in this network.
Our research showed that the VAN network, which is responsible for paying attention to important stimuli, had reduced structural connectivity in patients with insomnia. The DMN, which is active when the brain is at rest, also had reduced structural connectivity in patients with insomnia. Previous studies have reported abnormalities in functional connectivity in these networks in patients with insomnia [5, 6]. In our study, we found reduced structural connectivity between the pars opercular part of the VAN and the parietal part of the DMN, as well as between the DMN and SM networks. These abnormalities may represent the negative impact of sleep deficits on cognitive functions. Additionally, we found reduced structural connectivity between the right angular gyrus and several regions within the VAN and DMN networks. The right angular gyrus has been suggested to play a central role in insomnia disorder by modulating the interactions between these networks [7, 8]. Our finding supports this hypothesis and highlights the importance of this region in the pathophysiology of insomnia.
Our analysis revealed reduced structural connectivity in the DAN network, which is associated with spatial attention and visual processing. These functions may also be impacted by sleep disturbances. We found decreased connections in these networks in our patient group, indicating a potential disruption of their roles in cognitive performance. Interestingly, these networks have also been implicated in hyperarousal, a core feature of insomnia disorder [9]. Reduced structural [9]. Reduced structural connectivity in these networks may reflect a dysregulation of arousal levels in insomnia patients [9]. Furthermore, these networks have been shown to interact with other networks such as the DMN and VAN during cognitive tasks, suggesting that their structural alterations may also influence inter-network communication [10].
In our patient group, we observed a significant left hemisphere lateralization of reduced structural connectivity. The left hemisphere has already been identified as being affected by insomnia [3, 11], although most studies find asymetric alterations in brain connectivity [12, 13]. However, previous reports have also found left hemisphere alterations related to insomnia symptoms, including a reduced volume of the left orbitofrontal cortex and increased activity of the left amygdala [14]. Our finding of reduced structural connectivity mainly in the left hemisphere may indicate a specific vulnerability of this hemisphere to insomnia disorder. is it enough or should add some parts?