prompt_front: Now, answer the following question based on the text below, only give me the answer and do not output any other words.

Question: What is the purpose of the baseline in the layout procedure?

Read the following text and answer briefly.

Context 1: Probably one of the most frustrating things about building experimental aircraft, especially when starting with a minimum of pre-fabricated parts, is to start building and ending up with an unexpected result  Every builder starts a new project by wanting it to go "perfectly " So when things aren't going well, especially at the beginning, the frustration can lead to an unfinished airplane 
This is the first article in a series dedicated to helping builders of the Rand Robinson KR series planes build a straight and true fuselage -- the first part of the construction process  Borrowing from modern boatbuliding techniques, focus will be on the KR-2S, but the principles apply to the entire lineup of KR-1 & KR-2 series planes 
While building the KR-2(s) a common surprise is encountered by builders when the completed fuselage sides are laid into position to form the fuselage box section  With many hours spent building the sides flat, finding the once straight longerons that now bow up from the building surface, form a most dissatisfying "banana" shape  Especially when using the preformed fiberglass parts, this curve in the top longeron is not acceptable  The builder is left wondering what went wrong and no amount of clamping or brute force forming will solve the problem to any degree of satisfaction  The problem is not the builder's fault  The solution starts by understanding the three dimensional relationship of the assembled parts being built 
First understand that the plans show the finished form of the plane  They show the "projected" form as you would expect to see it if viewing an actual plane from the top, ends and from the side  Since the sides are sloped (flared) outward, looking from the side, the distances given by measuring the profile drawing are "foreshortened" and don't give the proper shape for building the fuselage with a flat top longeron  What needs to be done is to "develop" the "true" distances and shape of the flat panel so that when it is curved into position, the longerons lay flat 
Second, understand that the dimensions called for in the plans put a twist in the sides that tends to work the panel in two directions of curvature  This twist makes the panel "undevelopable" meaning that that shape cannot be unrolled into an equivalent flat shape  This is important when laying out the side and bottom panels onto flat plywood  To illustrate this, try forming a piece of paper around a soda can  The paper can be formed flat around the can either straight or at a diagonal to it's length  It has only one direction of curvature and is by definition "developable"  Now try to form the same piece of paper around a baseball  It won't lie flat on the surface without some deformation (folding, wrinkling or tearing) of the paper  The ball has curvature in more that one direction and is a "compounded" shape  Paper (or plywood) can only be readily formed in developable shapes as opposed to aluminum or other metal which can accept in plane deformation  A developable surface is needed to lay out a curved surface when the materials used can't be deformed with any degree of in-plane strain 
Initially, the fuselage sides are laid out flat with reference to the top longeron measured to a straight chalk line  The bowing problem starts when the side panels are bent and sloped to form the fuselage box section  If the sides were not sloped (tumbled home), the section formed would be cylindrical and the longerons would lie flat  Since the sides are tumbled home, the section formed is now conical  When a conical shape is cut with a plane (building surface) not perpendicular to it's axis, the shape formed is elliptical -- exactly what happens with the top longeron  When it's built flat, bent to form a cylindrical section, and sloped to form a conical section, it takes on an elliptical shape firewall to tailstock 
This method borrows heavily from proven techniques used in the marine trades  It should be stressed at this point that although the layout procedure is not complicated, it is important to take your time  If the layout is not going well initially, start over! Better to erase layout errors now than to have them built it and cause surprises later 
Layout to ensure a fair and true fuselage starts by drawing a reference line (baseline) on the building surface  Refer to figures 2 & 3 and use a wire guide to draw a very straight baseline  About 500 lbs  Of tension should be adequate  One could use a chalk line, but we're talking airplanes here, not house framing 
The main layout difference is that the baseline isn't used as a reference for the top longeron  The baseline references the mid point of the firewall for the developed (and true dimensioned) side panel  Although the baseline will still be the reference, the top and bottom longerons will be laid separately 
Layout differences don't end there  Each of the stations (vertical members) will be laid out with a calculated separation so that when the panels are formed into position, they land on the spacing called for in the plans  Another major difference is that the bottom & side panels are applied after forming the fuselage box section  This is mainly to obtain the ability to "fair" the side and bottom surfaces and insure a straight and true shape 
Refer to figure 1 for the layout of the new developed side panel  The firewall (station a) is layed out perpendicular to the baseline  Longitudinal (station) measurements are given along the length of the baseline from the firewall  Vertical dimensions are given to reference the angle and breadths of the station at the baseline 
Notice that the top longeron is bowed outward and that the stations are spaced slightly greater than called out in the plans  When the panels are formed into the box frame section ,they will work into the dimensions specified in the plans 
Strike a centerline, longer than is needed on the building surface using a wire guide  Draw off the firewall line perpendicular to the centerline at one end 
Using the distances listed in the balloons, mark them off on the centerline  Distances are measured to the nearest sixteenth of an inch  Take time to mark them off carefully  Don't mark off the distances in a cumulative fashion  Use the firewall as a common reference 
Using the angles listed at each station, mark off a station line longer than is needed  The angles are measured to the nearest hundredth of a degree  Take time to mark them off carefully 
At each station, start by marking off each short (bottom longeron) line distance from the centerline  Use your set of trammels or beam compass for doing this  Mark the intersection of the short line with the station line 
At each station, mark off each long (top longeron) line distance from the intersection of the short line distance and the station line  Again the trammels or beam compass is best for completing this step  Mark the intersection of the long line distance with the station line 
Using the longeron as a batten, trace out the inside and outside curves of the longeron  After the batten is secure, in between each station, fasten a keeper block inside and outside to preserve the shape of the longeron taking care to avoid potential future interference with the diagonal members to be installed later  The fairing blocks can be removed or left in place if they won't interfere with building  The vertical station members and their diagonals can now be measured and positioned  Remember to refer to the plans for the material thickness direction 
After vertical and diagonal members are cut and fitted, take time to draw their outlines on the building surface to cut down on time and confusion when laying out the opposite side 
Finishing the side panel is accomplished in a manner similar to that called for in the handbook with the exception that the side and bottom skin panels will be attached later 
The next article in the series will discuss jigging and building techniques to ensure alignment and straightness of the flat built side panels  Also covered will be building a "strongback" jig to assure alignment of the side panels when they are formed into their final shape 
Part 3 in the series will cover assembly of the side panels using the jigs  Some joint details will be discussed that will ensure a stronger and more fair fuselage assembly  Also covered will be the layout & attachment of the side and bottom ply skins 
U S  Mail: Densmore Associates, inc 
ANSI "D" size, computer generated plots of all the layout drawings in this series are available from the author for $30 plus postage & handling  Full (true size) scale plots may be made available depending on demand 
"Scarfing" is the practice of splicing plywood so that short pieces of plywood can be used to span long distances  On the KR, it is required on both the fuselage skins and spar webs  The angle of the splice should be 10 to 12 degrees to maintain strength across the joint  Also, joints should coincide with structural members, such as spar webs or fuselage truss members 
This scarfer is made by mating a regular plunge router (this one costs about $50) to a table saw  Obviously, you really only need a table saw to cut the chamfer, but it does make a nice heavy table for scarfing  You could just as easily use a large work table as the base First, set the table saw for a 5 5 degree cut (for a 1:12 joint, or 6 5 degree cut for a 10:1 joint), and run a 1 x 6 through on edge to chamfer a corner on the board  Then drill the board for three router mounting holes (two are countersunk) and connect the assembly to the table saw with two 1/4 inch bolts  Use a long (2-3 inch) straight cutting bit to do the cutting  Adjust the bit so it doesn't interfere with your table top, and go to town  Keep pressure on the plywood to ensure contact with the table while you're scarfing  Make sure you feed your material from the same end as you would if you were sawing, or the router will take your plywood away from you and put a big dent in your garage door 
In the late 60's Ken Rand and Stuart Robinson were working as flight system engineers for Douglas Avionics  Ken was working as an electrical engineer, having previously worked for Sperry as an autopilots project engineer, while Stu's degree was in aeronautical engineering from Northrop University  They were two of the guys at the end of the DC-8,9, and 10 assembly lines responsible for correcting some of the nits and picks in various systems before delivery to the customer 
They both wanted to build a fast, inexpensive airplane which was also economical to maintain  Several designs were considered, and plans were bought first for the Jeanie's Teenie and then the Taylor Monoplane  The Monoplane was more to their liking, but would require some modification to fit their needs  A cooperative redesign effort ensued, with virtually no dimensions left untouched  Only the basic fuselage structure, airfoil, and powerplant were retained  The tail shape was Stu's, and came directly from the big DC-8s parked on the ramp outside his office window  The landing gear was designed by Ken, after seeing the gear on a Dewey Bird at Santa Paula airport 
Ken was killed in his KR2 a short time later while flying over Cajon Pass in what was apparently a bad weather / low fuel accident  Ken's wife Jeanette became owner of RR overnight, and stepped up to keep the plans and parts coming  Much of the engineering needs are handled by Bill Marcy of Denver, who's been helping out since early '79 
To date, almost 6000 KR1, 9200 KR2, and 760 KR2S plan sets have been sold  1200 KR2s are estimated to be flying, with 5 KR2Ss now in the air  Much of the development work done on KR's is now done by the builders themselves  KR builders tend to be innovative, which leads to some interesting modifications  Some of the mods that work eventually creep into the plans  The KR2S is a case in point  Many builders who'd heard of the pitch sensitivity and tight cabin of the KR2 began to build an enlarged version, with the length determined by the most commonly available longeron material  The result is a KR2 that is stretched 2" between firewall and main spar, and 14" behind the main spar  Higher gross weights dictated more wing area, with the new standard becoming the Diehl wing skin  Those who plan to carry passengers commonly stretch the cabin width a few inches, although 1 5 inches is the limit if you still want to use RR's premolded parts 
Mike Stearns addresses the KR Forum crowd 
This year's KR Forum featured guest speakers Mike Stearns, Steve Trentman, and Bill Marcey  Mike Stearns spoke on several topics, including the many sources for KR and homebuilding information available on the Internet  He also mentioned KRNet, the list server devoted entirely to KR aircraft, as well as several notable World Wide Web home pages  He also brought a sample of the new Rand Robinson wing skins with him, and discussed their high temperature core prepreg construction  His KR2S will receive the first set, which is currently being installed at Hinson Composites 
Steve Trentman spoke on his turbine installation  It uses a turbine engine which saw duty as an A7 attack jet starter engine  Total weight is about 85 pounds, while putting out around 90 horsepower  There is a small stockpile of these engines available from government surplus  sources  This engine can only be throttled back to 52% power, which leads to some pretty interesting landings  One inflight failure has been logged so far, with very little damage to the aircraft  More on this exciting development in next month's issue of KROnline 
Les Palmer's KR2 N202LP won Best KR2, Best Engine Installation, and People's Choice awards at the 1995 KR Gathering at Columbia, TN  After researching the KR series, and reading Neil Bingham's "A Critical Analysis of the KR2" (Jan 88 Sport Aviation), Les decided to build his as a single seater, stretched 24" in the tail, while maintaining a stock width firewall  His fuselage is made from Douglas fir, which weighs in at 4 lbs heavier than if constructed from spruce  It is skinned with 1/8" birch plywood  Spars are covered with plywoood on both fore and aft sides, ala KR2S  Diehl wing skins provide the lift  Horizontal stabilizer and elevator were stretched 7" longer on each side, while the vertical stabilizer and rudder were stretched 8" taller    The fuselage to cowling junction was made more graceful by adding 1 5 inches to the height of the firewall end of the fuselage sides 
Les's canopy is a Dragonfly, using a four linkage system to swing forward when opening  The canopy frame fits snugly into a recess in the foward deck, providing an excellent wind and water seal  The fiberglass work is exemplary 
Seating is luxurious for one 
The cowling is also a work of art, and uses NACA ducts for efficiency  Female molds were made for all the fiberglass parts on Les's plane, so he could proabably be persuaded to make more, if demand dictates  Les also machines a multitude of KR aluminum and steel parts which he now offers for sale 
The firewall was reinforced with aluminum brackets and angles bolted between the longerons in anticipation of the 200 lb Subaru EA-81 engine installation  His 100 HP Asian version is outfitted with an American Holley 5200 caburetor and manifold  It uses a PSRU of Les's own design, featuring two spur gears with a 1 69:1 reduction ratio and a toothed belt  Other than tapping the crank for larger bolts to mount the redrive, no other engine modifications were required  Also, this is probably the only air conditioned KR2 on the planet  The prop is a 60/63 Hegy 
Originally built as a taildragger, the fixed gear is made from 4130 steel tubing  Custom cast 6 00x6 aluminum wheels and steel rotors are mated with 6" Cleveland calipers for braking  An early taxi test accident damaged the main gear, and prompted Les to change to tricycle gear  Again, he designed his own fiberglass main gear, and uses a Diehl nose wheel fork with a 4130 strut and 6" wheel up front 
Early tests revealed cooling problems, which prompted a radiator move from the firewall to a lower cowling location 
The first flight was almost a disaster, as test pilot Randy Smith lost power right after takeoff  He managed a 180 with a safe downwind landing with only minor nosewheel pant damage  The culprit proved to be a spark plug with too much reach, which was quickly remedied  Subsequent flights have shown water temp to be about 210 degrees, oil temp is 220-230, and airspeed is about 180 mph 
Shopping for the Partially Built KR 
This story starts about twenty years ago when I first started looking at the KR-2 as the plane I'd like to build  The only problem at that time was a lack of money, lack of knowledge, and a lack of job stability  I liked the design, except for the low ground clearance of the retractable gear and that a KR was going to be a tight fit for me to fly 
Over the past twenty years I've owned a number of planes, but still always wanted to build my own  I needed one that would fit me, my budget requirements, and have the speed and performance that I wanted  When "KITPLANES" published the article featuring Roy Marsh's new KR-2S, it was the first I had heard of any major modifications or improvements to the same old KR design  I believe that article and Roy Marsh's workmanship have probably been the greatest boon to Rand Robinson (RR) in the last twenty years  It certainly caught my eye! Here was the same design I had decided I wanted to build twenty years ago, with all of the improvements I wanted  It was sitting on fixed gear with some reasonable ground clearance  It had the capability to be built large enough to accommodate me  It has enough prefab parts available that it didn't have to be 100% scratch built if I decided to hurry the project along  And it had the speed I wanted  I knew that Roy's published speeds were probably not realistic expectations for the average KR, but after knocking around for the last three years in my Champ, anything over 90 mph seems pretty fast to me 
After purchasing the info kit and the sales video from Rand Robinson, the next step after deciding for sure to build this plane was to order the KR-2 plans and the KR-2S addendum  I finally got my plans and was putting together my first order to start the plane, when my partner in the Champ pointed out that there was a partially completed KR-2S for sale in Trade-a-plane  My initial answer was "No, I don't even want to look at it  I want to build my own from scratch " My partner insisted that for the advertised price and the fact that it wasn't too far away, I ought to at least give the guy a call and investigate it  "No, I don't think I want to buy someone else's problems," I persisted  That night I went home and crunched up some numbers on the calculator and finally came to the conclusion that for the sake of my budget for the next several years, I really should give this guy a call 
Three days later, I flew to his place about 400 miles away to take a look at his project  At this point I should probably mention that I consider myself to be fairly knowledgeable about airplane construction, although the vast majority of my experience is with tube and fabric  The rest of this article deals with what I looked for and more importantly what I missed and have had to repair in the last year since I purchased the project 
When we went to the seller's house, I found that the left wing was built using the Dan Diehl wing skins and the right wing skins were leaning against the wall inside the house  Also the canopy was in the house with the canopy covered with paper and tape  I wanted to inspect the fuselage first, so off we went to the shop 
There I found a fuselage sitting on it's gear painted in primer gray  The first step was to inspect the quality of workmanship of what could be seen as it sat  The interior of the fuselage looked as if it had been built with a great deal of care  The fit and finish of all of the interior wood was very nice  Even the gussets looked like they had been painstakingly perfectly fitted  The glass work on the turtle back also looked very precise and clean  It was evenly faired into the vertical and horizontal stabs  The tail also appeared to be well built with the exception of a depression directly over the front and rear spars in the horizontal stabs  He explained that when he moved recently, that he had shot the plane with gray primer to protect it from the weather since he wouldn't have ready access to a shop to put it in right away  It ended up sitting out in the hot south Texas summer sun for a few weeks before he got a shop rented to work in  That caused the glass (or possibly the foam inside the horizontal stab) to swell, except that it held onto the spar, so it was slightly ballooned in front of and behind the spars  His recommendation was to fill it back smooth with micro 
I also found a small linear crack in the lower left wing spar cap on the left wing stub  It appeared to be from over tightening the rear spar wing attach fitting bolts  His explanation was that the crack wasn't important because the rear spars only job is to keep the wings from folding back  I also noticed that the holes for attaching the outer wing to the wing stub were badly rounded out on the rear spar  He explained that the Diehl wing skins require the rear spar to be swept slightly more forward than the stock wings  This won't allow you to use the rear spar attach fittings from RR and that I would need to fabricate a new set of rear spar attach fittings 
I also found that the aileron bellcranks were not built or installed as per plans, but found that they looked professional  I couldn't check for function since the right bellcrank and sheeve wasn't installed, the left wing also wasn't installed, and the right wing didn't exist yet 
Next we pulled the inspection panels off of the fuselage and tail and looked at everything I could see with a good flashlight  I didn't find anything else that might be questionable about the fuselage except for a cracked elevator trim tab that was damaged when it fell off it's hanging place on the wall 
Next we spent some time going over his builders log and builders photo album  I still hadn't seen anything that would dissuade me from buying this project 
At this point it was starting to get late and my ride down needed to get airborne for the flight home  I needed to make a decision about whether I wanted this project or not, but I hadn't inspected the wings and canopy yet  I took a cursory look at the left wing and saw lots on micro built up on it and some bubbles in the leading edge, but nothing that looked seriously wrong to my amateur eye  The right wing was only a set of spars in the shop and the Diehl wing skins in the house, so there wasn't much to look at there  The canopy was wrapped in paper and tape, so there wasn't much to look at there either  I decided that even if there were serious problems in the wing that was built, I would be money ahead to go ahead and buy the project  For the advertised price, I could build a new set of wings and still be way ahead financially  We negotiated a final price, shook hands, took my ride to the airport, and started off in search of a U-haul to haul the project home 
Now, at this point, some of you are thinking about what I surely must have forgotten to inspect and why didn't I take a local A & P or EAA member along for the ride  First of all, I don't know any mechanics locally that have any experience with glass and our EAA chapter of which I am VP is woefully lacking in fiberglass knowledge  Secondly, as you will see, I missed plenty  Some by ignorance, some by just not looking close enough 
Now for a list of the problems that I found over the last year and a few of the fixes that I came up with 
I found that the lower set of rear spar attach fittings on the left rear spar were installed backwards with the longer spaced hole towards the fuselage  Since this is the same place that also had the cracked spar cap, it required a major change  Also in the same area he had drilled through the rear spar with a hole saw to create a place for the aileron cable to pass through and managed to cut out the second from the outside vertical brace in the spar  Then he chose to install the aileron bellcranks in front of the rear spar, and cut another hole through the rear spar for the aileron push rod  He also managed to cut out the outside vertical brace in the spar  Since the holes were already drilled through the spar, the choices were to either cut out that section of spar cap and scarf a new piece in, cut the whole rear spar carrythrough out of the fuselage including ruining the left lower wing skin, or do something else creative to reinforce the spar cap and install a custom built set of attach fittings 
I also found that after I built and installed the right side wing stub ribs and skin that the aileron bellcrank setup would not work as installed  The cable that crosses between the two bellcranks had a sharp uphill from the sheeve to the bellcrank in the last 12 inches on either side  This combined with the radius that the bellcranks turn caused the cross cable to pull up tight when the ailerons were pushed to either end of their travel, but allowed the cables to go very slack when the ailerons were centered  Also the Aileron pushrods needed to pass directly through the lower set of rear wing attach fittings to attach to the aileron  This whole rear spar and aileron bellcrank setup was going to either have to be redesigned or cut out and built to plans  The bottom line is that the problems I observed when I inspected this part were much more serious than expected when I had to fix it 
I decided that I had to remove the rear fittings from the left wing to be replaced with the new set that my neighborhood machinist was cutting out for me  When I put the wing on the work bench to start removing the rear fittings, I thought I had better take a closer look at the bubbles in the leading edge  I found that as I pushed on the leading edge, it delaminated between the glass lay-up on top and the upper and lower wing skin edges that were floxed together underneath  I concluded that that area had to come apart and took a belt sander to the leading edge  What I found was that the leading edge had been floxed together and glassed over, but the mold release had never been scrubbed off the leading edge of the wing  It peeled apart for rebuild quite easily 
When I got back to removing the rear spar attach fittings, I noticed that the woodwork inside the wing looked awfully dull  The reason was that the wing had been closed up without varnishing any of the woodwork  This was rectified with a small hole saw, a number of extensions and a modified undercoating sprayer 
I also found that the aluminum drain fitting in the bottom of the left wing tank had been glassed into place upside down  The tapered pipe threads were tapered the wrong way to install the draincock into the tank  Retapping the fitting the right direction seemed to be a good fix for that problem 
When I finally got around to attaching the wing to the fuselage, I found that the front spar attach fittings were badly misaligned  Although they could be forced into alignment, I didn't think I needed that kind of preload on the main spar fittings  This problem was fixed by calling on my local neighborhood machinist to build me an aligning fixture and reaming the attach holes to the next larger size and ordering the new sized bolts 
On the fuselage I found that although it had new Cleveland wheels and brakes on it, one of the brakes had a severe wobble to it  I must complement the manufacturers for taking care of that problem  One call to the Cleveland factory and they shipped me a new set of wheels and brakes even though the receipt for this set was over four years old and in the original builders name  Their only concern was that this set had never been placed in service yet 
I chose to sand the load of micro off the left wing to see what it was covering  When I got down to the glass, I found that there was no glass for the aft inch and a half of the underside of the wing in front of the aileron hinge  With the Diehl wing skins, you build the wings, then cut the ailerons out of trailing edge of the wing  He had mismeasured and cut too much material off the bottom side of the trailing edge in front of the aileron  It was filled by floxing a piece of spruce into the gap to fill the space between the back edge of the fiberglass and the aileron mount  I chose to wrap the trailing edge of that wing, and the other wing to match with a couple of lay-ups of glass 
When I sanded the primer off the aforementioned damaged trim tab, I found that the hinge was floxed to the leading edge of the foam insides of the tab, but not the glass  I also chose to wrap the front of the trim tab with a lay-up of glass 
I decided to pull the paper off the canopy and take a look at it before I'm ready to bolt it on and fly  The original builder had blown his own canopy and after some of the previous problems, I was beginning to have some concerns about not having looked it over closely enough  The canopy turned out to have been blow a little too large  It ended up with a little larger bubble for headroom, which I didn't object to  However, it had more headroom on the right side than the left  Yes, it was just a little bit lopsided  The main problem was that the canopy is stretched thin enough that it can be easily pushed in with one hand when the weather is warm   My fear was that this is just thin enough that it may decide to lay on my head or in my lap when flying on a warm day  It will have to be replaced 
I'm sure that many that are reading this could see several of the potential problems before I mentioned them, but some others may not have and I'm sure that there could have been many other problems that didn't but could have existed on this project  This is also not intended to be critical of the gentleman that started this project as many parts of it, especially the wood work are better than I could have done and much of his work is outstanding  I prefer to think that I'll end up with a better plane with his woodwork combined with my glasswork  This article is intended to feature some of the problems that you may run into in buying someone else's project 
The final question is, knowing what I have found over the past year, would I have still purchased this project  The answer is yes, but primarily because the price was right in that I am still money and work ahead of where I would be if I had started the project from scratch  There are a few things that I would have done differently, but nothing that I can't live with  Although I won't be able to say that I built it all from scratch, I have built and rebuild enough of the plane that I should have no problem qualifying under the 51% rule 
You can send comments directly to the author via e-mail at "jscott@LANL GOV" 
Here is an brief explanation of how I built my turtledecks  The jig was constructed from scrap plywood and a few 1x4s that I ripped into stringers  I made two temporary bulkheads from the plywood, one for each end  Remember the forward bulkhead needs to be shaped in a way that will closely match the aft end of your canopy frame  Make an aft bulkhead by placing a straight edge at the top of your forward bulkhead and the trailing edge of your horizontal stabilizer  This will give you an idea of how tall your aft bulkhead needs to be  As far as location, I placed my aft bulkhead just forward of the lower/front of my vertical fin  I constructed the jig on the fuselage, it is glued together with automotive bondo 
After the bulkheads were bondoed to the fuselage I used the stringers that I ripped from the 1x4s and bondoed them to the bulkheads  This gave me a male form to cover with thin plastic or posterboard  I stapled two layers of posterboard to the jig(thin plastic would work better)  The posterboard wraps down two inches onto the fuselage  After I was satisfied with the way it looked, I then covered the entire thing with duct tape (fiberglass will not stick to duct tape) On top of this I wetout one layer of tri-ply cloth (22oz) that I had left over from an earlier project, and one layer of 8oz  bid  Remember to mask off your fuselage so you don't get epoxy on it  If you are not familiar with composite lay-ups, you should plan on razor cutting your lay-ups 4 to 6 hours after wetout while the lay-up is still soft enough to cut with a razorblade 
After the lay-up cured (2 or 3 days) it was removed from the jig, and the jig was removed from the fuselage and discarded  (be careful, the bondo sticks very well to the spruce, you could splinter your wood during removal) I now have a fiberglass skin that tends to hold the shape of the jig but is still flexible enough to work with  I made two bulkheads out of 1/4 last-a-foam (AS&S) using the plywood formers from the jig as a guide  I covered these foam bulkheads with one 8oz layer of glass on each side, with a glass to glass edge on the bottom  After cure these bulkheads were bondoed into place (to the fuselage)and the fiberglass skin was pulled down tight and floxed to the bulkheads  When the flox cured the bondo joints were broken, again being careful not to harm the wood  The turtledeck was removed from the fuselage and 2 inch tapes added to the bulkheads inside and out 
At this point the turtledeck looked great and only weighed about 5lbs  but I noticed you could deform the skin by pushing hard on the outside  So I flipped the turtledeck over and from 1/4 inch last-a-foam, I cut two inch wide strips that would run the entire length, forward and aft inside the turtledeck  In effect these would act as composite stringers, I made enough of these two inch wide strips to make up three stringers  One down the center (sort of a backbone) and one on each side of the "backbone" half the distance to the edge of the turtledeck  I sanded the edge of the foam so that when covered with a layer of bid @ 45degrees there would be a nice transition from the turtledeck skin up onto the foam and then back onto the turtledeck I scuff sanded and glued the foam stringers in with micro  I covered the foam stringers with one layer of 8oz bid @ 45degrees 
You can also send me email at: mikemims@pacbell net if you have any questions or want to share your ideas 
KROnline is an online KR Newsletter devoted to sharing KR information with other builders and pilots in a timely manner  The first issue (September 96) is now available as a zipped MicroSoft Word file at http://members aol com/bshadr or as an html document at kronline9 html  If you'd like to submit articles or photos, email Randy Stein at BSHADR@aol com ------------------------------------------------------------ Don't bother to email Randy though  KROnline has been retired since the KR Newsletter has improved 
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(b) Agency Spotter will use commercially reasonable efforts to make the Services available on a 24 hours a day, 7 days a week, and 365 days a year basis, subject to Section 23 below and to downtime for maintenance purposes 
(c) Agency Spotter may from time to time modify the Services and add, change, or delete features of the Services in its sole discretion, without notice to you  Your continued use of the Service after any such changes to the Service constitutes your acceptance of these changes  Agency Spotter will use commercially reasonable efforts to post information on the Site regarding material changes to the Services 
(d) The contents of the Site, such as text, graphics, images, logos, user interfaces, visual interfaces, photographs, button icons, software, trademarks, sounds, music, artwork and computer code, and other Agency Spotter content (collectively, “Agency Spotter Content”), are protected under both United States and foreign copyright, trademark and other laws  All Agency Spotter Content is the property of Agency Spotter or its content suppliers or clients  The compilation (meaning the collection, arrangement and assembly) of all content on the Site is the exclusive property of Agency Spotter and is protected by United States and foreign copyright, trademark, and other laws  Unauthorized use of the Agency Spotter Content may violate these laws, and is strictly prohibited  You must retain all copyright, trademark, service mark and other proprietary notices contained in the original Agency Spotter Content on any authorized copy You make of the Agency Spotter Content 
(e) You agree not to sell or modify the Agency Spotter Content or reproduce, display, publicly perform, distribute, or otherwise use the Agency Spotter Content in any way for any public or commercial purpose, in connection with products or services that are not those of the Site, in any other manner that is likely to cause confusion among consumers, that disparages or discredits Agency Spotter or its licensors, that dilutes the strength of Agency Spotter’s or its licensor’s property, or that otherwise infringes Agency Spotter’s or its licensor’s intellectual property rights  You further agree to in no other way misuse Agency Spotter Content that appears on this Site  Any code that Agency Spotter creates to generate or display any Agency Spotter Content or the pages making up the Website is also protected by Agency Spotter’s copyright and You may not copy or adapt such code 
2  Site Restrictions  You may not use the Site in order to transmit, post, distribute, store or destroy material, including without limitation, the Agency Spotter Content, (a) in violation of any applicable law or regulation, (b) in a manner that will infringe the copyright, trademark, trade secret or other intellectual property rights of others or violate the privacy, publicity or other personal rights of others, (c) that is defamatory, obscene, threatening, abusive or hateful, or (d) that is in furtherance of criminal, fraudulent, or other unlawful activity  You are also prohibited from violating or attempting to violate the security of the Site and Services, including without limitation, the following activities: (a) accessing or attempting to access data not intended for You or logging into a server or account which You are not authorized to access; (b) attempting to probe, scan or test the vulnerability of a system or network or to breach security or authentication measures without proper authorization; (c) attempting to interfere with service to any other user of the Site or Services, host or network, including, without limitation, via means of submitting a virus to the Website, overloading, “flooding”, “spamming”, “mailbombing” or “crashing”; or (d) forging any TCP/IP packet header or any part of the header information in any e-mail or newsgroup posting  Violations of system or network security may result in civil and/or criminal liability 
3  Specific Prohibited Uses  The Agency Spotter Content and other features of the Site may be used only for lawful purposes  Agency Spotter specifically prohibits any other use of the Site, and You agree not to do any of the following: (a) use the Site for any purpose other than as a platform for connecting businesses and agencies, including but not limited to using the information in the Website to sell or promote any products or services; (b) post or submit to the Website any incomplete, false or inaccurate biographical information or information which is not Your own; (c) post on the Website any franchise, pyramid scheme or “club membership”; (d) send unsolicited mail or e-mail, make unsolicited phone calls or send unsolicited faxes regarding promotions and/or advertising of products or services to any other user(s) of the Website; (e) delete or revise any material posted by any other person or entity; (f) take any action that imposes an unreasonable or disproportionately large load on the Website’s infrastructure; (g) notwithstanding anything to the contrary contained herein, use or attempt to use any engine, software, tool, agent or other automatic device, program, algorithm, methodology or mechanism (including without limitation browsers, spiders, robots, avatars or intelligent agents) to navigate or search the Website other than the search engine and search agents available from Agency Spotter on the Website and other than through generally available third party web browsers (e g , Internet Explorer, Firefox, Safari); (h) decipher, decompile, disassemble or reverse engineer any of the software comprising or in any way making up a part of the Website; or (i) aggregate, copy or duplicate in any manner any of the Agency Spotter Content or information available from the Website, without express written consent from Agency Spotter 
(a) Certain features or services offered on or through the Site to users or agencies may require you to open a user or agency account (“Agency Account”) (including setting up a user ID and password)  You are entirely responsible for maintaining the confidentiality of the information you hold for your account, including your password, and for any and all activity that occurs under your account until you close down your account or prove that your account security was compromised due to no fault of your own  To close your account, please email us at [email protected] You agree to notify Agency Spotter immediately of any unauthorized use of your account or password, or any other breach of security  You may be held liable for losses incurred by Agency Spotter or any other user of or visitor to the Site due to someone else using your Agency Spotter ID, password or account as a result of your failing to keep your account information secure and confidential  You may not use anyone else’s Agency Spotter ID, password or account at any time without the express permission and consent of the holder of that Agency Spotter ID, password or account  Agency Spotter cannot and will not be liable for any loss or damage arising from your failure to comply with these obligations  Agency Spotter may verify Agency Accounts to confirm that such accounts meet Agency Spotter’s minimum requirements to be an agency, as the same may be modified or amended from time to time, and may assign an administrator to such verified Agency Account 
(b) To eligible to use the Site and the Services, you must meet the following criteria and represent and warrant that you: (i) are at least 18 years of age; (ii) are not currently restricted from the Site or Services, and are not otherwise prohibited from having an Agency Spotter account, (iii) are not a competitor of Agency Spotter or are not using the Site or Services for reasons that are in competition with Agency Spotter, (iv) will only maintain one Agency Spotter account at any given time, (v) have full power and authority to enter into this Agreement and doing so will not violate any other agreement to which you are bound, (vi) will not violate any rights of Agency Spotter, including intellectual property rights such as copyright and trademark rights, and (vii) agree to provide at your cost all equipment, software and internet access necessary to use the Site or Services 
6  User Content and Submissions  You understand that all information, data, text, software, music, sound, photographs, graphics, video, advertisements, messages or other materials submitted, posted or displayed by You on or through the Website (“User Content”) is the sole responsibility of the person from which such User Content originated  Agency Spotter claims no ownership or control over any User Content  You or a third party licensor, as appropriate, retain all patent, trademark and copyright to any User Content You submit, post or display on or through Agency Spotter and You are responsible for protecting those rights, as appropriate  By submitting, posting or displaying User Content on or through Agency Spotter, You grant Agency Spotter a worldwide, non-exclusive, royalty-free license to reproduce, adapt, distribute and publish such User Content through Agency Spotter  In addition, by submitting, posting or displaying User Content which is intended to be available to the general public, You grant Agency Spotter a worldwide, non-exclusive, royalty-free license to reproduce, adapt, distribute and publish such User Content for the purpose of promoting Agency Spotter Services  Agency Spotter will discontinue this licensed use within a commercially reasonable period after such User Content is removed from the Site  Agency Spotter reserves the right to refuse to accept, post, display or transmit any User Content in its sole discretion 
You also represent and warrant that You have the right to grant, or that the holder of any rights has completely and effectively waived all such rights and validly and irrevocably granted to You the right to grant, the license stated above  If You post User Content in any public area of the Website, You also permit any user of the Website to access, display, view, store and reproduce such User Content for personal use  Subject to the foregoing, the owner of such User Content placed on the Website retains any and all rights that may exist in such User Content 
Agency Spotter does not represent or guarantee the truthfulness, accuracy, or reliability of User Content or endorse any opinions expressed by users of the Website  You acknowledge that any reliance on material posted by other users will be at Your own risk 
The following is a partial list of User Content that is prohibited on the Website  Prohibited Content includes, but is not limited to, Content that: is implicitly or explicitly offensive, such as User Content that engages in, endorses or promotes racism, bigotry, discrimination, hatred or physical harm of any kind against any group or individual; harasses, incites harassment or advocates harassment of any group or individual; involves the transmission of “junk mail”, “chain letters,” or unsolicited mass mailing or “spamming”; promotes or endorses false or misleading information or illegal activities or conduct that is abusive, threatening, obscene, defamatory or libelous; promotes or endorses an illegal or unauthorized copy of another person’s copyrighted work, such as providing or making available pirated computer programs or links to them, providing or making available information to circumvent manufacture-installed copy-protect devices, or providing or making available pirated music or other media or links to pirated music or other media files; contains restricted or password only access pages, or hidden pages or images; displays or links to pornographic, indecent or sexually explicit material of any kind; provides or links to material that exploits people under the age of 18 in a sexual, violent or other manner, or solicits personal information from anyone under 18; or provides instructional information about illegal activities or other activities prohibited by these Terms and Conditions, including without limitation, making or buying illegal weapons, violating someone’s privacy, providing or creating computer viruses or pirating any media; and/or solicits passwords or personal identifying information from other users 
It is your responsibility to keep your Agency Spotter profile information accurate and updated 
7  User-to-User Communications and Sharing (Agency Spotter Groups, Ratings, Reviews, Updates, Agency Pages, etc )  Agency Spotter offers various forums such as Agency Spotter Groups, Ratings, Reviews, and Updates, where you can post your observations and comments on designated topics  Agency Spotter also enables sharing of information by allowing users to post updates, including links to news articles and other information such as product recommendations, job opportunities, and other content to their profile and other parts of the Site, such as Agency Spotter Groups and Agency Pages  Agency Spotter members can create Agency Spotter Groups and Agency Pages for free; however, Agency Spotter may close or transfer Agency Spotter Groups or Agency Pages, or remove content from them if the content violates these Terms or others’ intellectual property rights  To create an Agency Spotter Agency Page, the Agency must be a company or legal entity that meets Agency Spotter’s minimum requirements for an Agency, and you must have the authority to create the Agency Page on behalf of the third party Agency 
For clarity, only DMCA Notices should go to the Copyright Agent; any other feedback, comments, requests for technical support, and other communications should be directed to: [email protected] You acknowledge that if you fail to comply with all of the requirements of this Section, your DMCA Notice may not be valid 
Upon receipt of a Notice, Agency Spotter will take whatever action, in its sole discretion, it deems appropriate, including removal of the challenged material from the Site and/or termination of the User’s account in appropriate circumstances  Please note that a Complainant may be liable for damages (including costs and attorneys’ fees) if he or she knowingly makes a material misrepresentation that content is infringing 
(i) If you have posted material subject to a DMCA Notice that allegedly infringes a copyright (the “Counterclaimant”), you may send Agency Spotter a written Counter Notice pursuant to Section 512(g), (ii) and 512(g), (iii) of the DMCA  When Agency Spotter receives a Counter Notice, it may, in its discretion, reinstate the material in question not less than ten (10) nor more than fourteen (14) days after receiving the Counter Notice unless Agency Spotter first receives notice from the Claimant that he or she has filed a legal action to restrain the allegedly infringing activity  Please note that Agency Spotter will send a copy of the Counter Notice to the address provided by the Claimant  A Counterclaimant may be liable for damages (including costs and attorneys’ fees) if he or she knowingly makes a material misrepresentation that that material or activity was removed or disabled by mistake or misidentification 
1  Identification of the material that has been removed or to which access has been disabled and the location at which the material appeared before it was removed or access to it was disabled 
2  A statement under penalty of perjury that you have a good faith belief that the material was removed or disabled as a result of mistake or misidentification of the material to be removed or disabled 
3  Your name, address, and telephone number, and a statement that you consent to the jurisdiction of Federal District Court for the judicial district in which the address is located, or if your address is outside of the United States, for any judicial district in which Agency Spotter may be found, and that you will accept service of process from the person who provided notification under subsection (c)(1)(C) of the DMCA or an agent of such person 
(c) AGENCY SPOTTER HAS NO OBLIGATION TO ADJUDICATE CLAIMS OF INFRINGEMENT – EACH USER’S AGREEMENT TO HOLD AGENCY SPOTTER HARMLESS FROM CLAIMS  Claimants, Counterclaimants, and users understand that Agency Spotter is not an intellectual property tribunal  While Agency Spotter may, in its discretion, use the information provided in a DMCA Notice and Counter Notice in order to decide how to respond to infringement claims, Agency Spotter is not responsible for determining the merits of such claims  If a Counterclaimant responds to a claim of infringement by providing a Counter Notice, the Counterclaimant agrees that if Agency Spotter restores or maintains the content, the Counterclaimant will defend and hold Agency Spotter harmless from any resulting claims of infringement against Agency Spotter 
10  Advertisements and Other Potential Sources Of Revenue  Some of the Services may now or in the future be supported by advertising revenue, pay-per-click mechanisms, or other funding, and the Site may display advertisements and promotions  These advertisements may be targeted to the content of information stored via the Site, queries made through the Services, or other criteria  The manner, mode and extent of advertising on the Site are subject to change without specific notice to you  In consideration for Agency Spotter granting you access to and use of the Site and the Services, you agree that the Agency Spotter may place such advertising on the Site and/or incorporate such advertisements into the Services 
11  DISCLAIMERS  THE SITE AND ITS CONTENT AND THE SERVICES ARE PROVIDED “AS IS” AND AGENCY SPOTTER MAKES NO REPRESENTATIONS OR WARRANTIES OF ANY KIND, EITHER EXPRESS OR IMPLIED, ABOUT THE IMAGES OR SITE INCLUDING, WITHOUT LIMITATION, WARRANTIES OF MERCHANTABILITY, FITNESS FOR A PARTICULAR PURPOSE, OR NON-INFRINGEMENT, TO THE FULLEST EXTENT PERMISSIBLE UNDER APPLICABLE LAW  AGENCY SPOTTER DOES NOT WARRANT THAT ACCESS TO THE SITE OR ITS CONTENTS OR THE SERVICES WILL BE UNINTERRUPTED OR ERROR-FREE, THAT DEFECTS WILL BE CORRECTED, OR THAT THIS SITE OR THE SERVERS THAT MAKE IT AVAILABLE ARE FREE OF VIRUSES OR OTHER HARMFUL COMPONENTS  AGENCY SPOTTER DOES NOT WARRANT OR MAKE ANY REPRESENTATIONS REGARDING THE USE OR THE RESULTS OF THE USE OF ANY CONTENT ON THE SITE IN TERMS OF ITS CORRECTNESS, ACCURACY, RELIABILITY, OR OTHERWISE  ACCORDINGLY, YOU ACKNOWLEDGE THAT YOUR USE OF THE SITE IS AT YOUR OWN RISK  YOU (AND NOT AGENCY SPOTTER) ASSUME THE ENTIRE COST OF ALL NECESSARY SERVICING, REPAIR, OR CORRECTION RESULTING FROM COMPUTER MALFUNCTION, VIRUSES OR THE LIKE  APPLICABLE LAW MAY NOT ALLOW THE EXCLUSION OF IMPLIED WARRANTIES, SO THE ABOVE EXCLUSION MAY NOT APPLY TO YOU 
12  Limitation on Liability  Neither Agency Spotter, nor its licensors, representatives, affiliates, employees, shareholders or directors (collectively, “Agency Spotter Affiliates”), shall be cumulatively responsible or liable for (a) any damages in excess of three (3) times the most recent monthly fee that you paid for a Premium Service, if any, or US $100, whichever amount is greater, or (b) any damages of any kind including, without limitation, lost business, profits or data (or the cost to recreate such data), direct, indirect, incidental, consequential, compensatory, exemplary, special or punitive damages that may result from Your access to or use of Website, the Agency Spotter Content, or the Services, or any content or other materials on, accessed through or downloaded from the Site  The allocations of liability in this Section represent the agreed and bargained-for understanding of the parties and the fees herein reflects such allocation  These limitations of liability will apply notwithstanding any failure of essential purpose of any limited remedy, whether your claim is based in contract, tort, statute or any other legal theory, and whether we knew or should have known about the possibility of such damages; provided, however, that this limitation of liability shall not apply if you have entered into a separate written agreement to purchase Premium Services with a separate Limitation of Liability provision that expressly supersedes this Section in relation to those Premium Services 
13  Indemnification  In the event that You use the Website, the Agency Spotter Content, or any portion thereof, in any manner not authorized by Agency Spotter, or if You otherwise infringe any intellectual property rights or any other rights relating to other users, You agree to indemnify and hold Agency Spotter, its subsidiaries, affiliates, licensors and representatives, harmless against any losses, expenses, costs or damages, including reasonable attorneys’ fees, incurred by them as a result of unauthorized use of the Website or the Agency Spotter Content and/or Your breach or alleged breach of these Terms and Conditions 
(a) You agree that Agency Spotter and its licensors own all intellectual property rights in and to the Services, the Site and related Software, including but not limited to the look and feel, structure, organization, design, algorithms, templates, data models, logic flow, text, graphics, logos, and screen displays associated therewith 
(b) You will not reverse engineer, decompile or disassemble the Software, or otherwise attempt to reconstruct or discover the source code for the Software  You further agree not to resell, lease, assign, distribute, time share or otherwise commercially exploit or make the Services available to any third party for such third party’s benefit 
(c) You may make a single copy of the Downloaded Software for backup purposes only; provided that any such copies contain the same proprietary rights notices that appear on the Downloaded Software  Agency Spotter reserves all rights in the Services and Software not expressly granted to you hereunder  As used herein, “Software” means Agency Spotter’s proprietary software used to deliver the Services, made available to you as part of the Site and/or Services, and all updates and associated documentation thereto made available as a part of the Site or Services pursuant to these Terms, including Downloadable Software  The term “Downloadable Software” means client software downloaded by you from the Site that augments your use of the Site and/or Services, including add-ins, sample code, APIs and ancillary programs 
(d) Agency Spotter shall have a perpetual, royalty-free, worldwide, and transferable license to use or incorporate into the Site and Services any suggestions, ideas, enhancements, feedback, or other information provided by you related to the Site or Services 
(e) Agency Spotter may derive and compile aggregated and/or analytical information from your usage of the Site and Services  Such aggregated data and metadata may be used for Agency Spotter’s own purposes without restriction, including, but not limited to, using such data in conjunction with data from other sources to improve Agency Spotter’s products and services and to create new products 
15  Third Party Software and Features; Agency Spotter Applications  (a) Agency Spotter may make software from third-party companies available to You  To download such software, You may be required to agree to the respective software licenses and/or warranties of such third-party software  Each software product is subject to the individual company’s terms and conditions, and the agreement will be between You and the respective company  This means that Agency Spotter does not guarantee that any software You download will be free of any contaminating or destructive code, such as viruses, worms or Trojan horses  Agency Spotter does not offer any warranty on any third-party software You download using the Site  Further, the Site and/or Service may contain features, functionality and information that are provided through or by third-party content, software, websites, and/or system (“Third Party Materials”)  Your use and access of these features and functionality are subject to the terms published or otherwise made available by the third-party providers of Third Party Materials  Agency Spotter has no responsibility for any Third-Party Materials, and you irrevocably waive any claim against Agency Spotter with respect to such Third-Party Materials 
(b) Agency Spotter may also offer the Services through applications built using Agency Spotter’s platform (“Agency Spotter Applications”), including smart phone applications, “Share” and other similar buttons and other interactive plugins distributed on websites across the Internet  Agency Spotter Applications are distinct from Third-Party Materials and applications address in Section 14(a), above  If you use an Agency Spotter application or interact with a website that has deployed a plugin, you agree that information about you and your use of the Services, including, but not limited to, your device, your mobile carrier, your internet access provider, your physical location, and/or web pages containing Agency Spotter plugins that load in your browser may be communicated to us  You acknowledge that you are responsible for all charges and necessary permissions related to accessing Agency Spotter through your mobile access provider  You should therefore check with your provider to find out if the Services are available and the terms for these services for your specific mobile devices  Finally, by using any downloadable application to enable your use of the Services, you are explicitly confirming your acceptance of the terms of the End User License Agreement associated with the application provided at download or installation, or as may be updated from time to time 
16  International Use  Agency Spotter makes no representation that materials on this site are appropriate or available for use in locations outside the United States, and accessing them from territories where their contents are illegal is prohibited  Those who choose to access this site from other locations do so on their own initiative and are responsible for compliance with local laws 
17  Dispute Resolution  These Terms and any claim, cause of action or dispute (“claim”) arising out of or related to these Terms shall be governed by the laws of the State of Georgia, regardless of your country of origin or where you access Agency Spotter, and notwithstanding any conflicts of law principles and the United Nations Convention for the International Sale of Goods  You and Agency Spotter agree that all claims arising out of or related to these Terms must be resolved exclusively by a state or federal court located in Fulton County, Georgia, except as otherwise mutually agreed in writing by the parties or as described in the Arbitration option in Section 16(b), below  You and Agency Spotter agree to submit to the personal jurisdiction of the courts located within Fulton County, Georgia, for the purpose of litigating all such claims  Notwithstanding the foregoing, you agree that Agency Spotter shall still be allowed to seek injunctive remedies (or an equivalent type of urgent legal relief) in any jurisdiction 
18  Arbitration  You agree that any dispute, claim or controversy arising hereunder or relating in any way to the Terms, shall be settled by binding arbitration in Fulton County, Georgia, in accordance with the commercial arbitration rules of Judicial Arbitration and Mediation Services (“JAMS”)  The arbitrator shall issue a written decision specifying the basis for the award made  The party filing a claim or counterclaim in the arbitration proceeding shall pay the deposit(s) determined by JAMS with respect to such claim or counterclaim  All other costs associated with the arbitration and imposed by JAMS shall be paid as determined by the arbitrator(s) and, in absence of such determination, equally by each party to the arbitration  In addition, unless the arbitrator awards payment of reasonable attorney and other fees to a party, each party to the arbitration shall be responsible for its own attorneys’ fees and other professional fees incurred in connection with the arbitration  Determinations of the arbitrator will be final and binding upon the parties to the arbitration, and judgment upon the award rendered by the arbitrator may be entered in any court having jurisdiction, or application may be made to such court for a judicial acceptance of the award and an order of enforcement, as the case may be  The arbitrator shall apply the substantive law of the State of Georgia, without giving effect to its conflict of laws rules 
19  Export Control  You agree to comply with all relevant export laws and regulations, including, but not limited to, the U S  Export Administration Regulations and Executive Orders (“Export Controls”)  You warrant that you are not a person, company or destination restricted or prohibited by Export Controls (“Restricted Person”)  You will not, directly or indirectly, export, re-export, divert, or transfer the Site or Service or any related software, any portion thereof or any materials, items or technology relating to Agency Spotter’s business or related technical data or any direct product thereof to any Restricted Person, or otherwise to any end user and without obtaining the required authorizations from the appropriate governmental entities 
(a) These Terms will continue until terminated in accordance with this Section 
(b) You may cancel your legal agreement with Agency Spotter at any time by (i) notifying Agency Spotter in writing, (ii) ceasing to use the Services, and (iii) closing your accounts for all of the Services which you use, if we have made this option available to you  Your cancellation of the Services will not alter your obligation to pay all charges incurred prior to your effective date of termination 
Agency Spotter may terminate its legal agreement with you if, (i) you have breached any provision of the Terms (or have acted in manner which clearly shows that you do not intend to, or are unable to comply with the provisions of the Terms), or (ii) Agency Spotter is required to do so by law (for example, where the provision of the Services to you is, or becomes, unlawful), or (iii) Agency Spotter is transitioning to no longer providing the Services to users in the country in which you are resident or from which you use the service, or (iv) the provision of the Services to you by Agency Spotter is, in Agency Spotters’ opinion, no longer commercially viable 
(c) The terms provided in Sections 2, 3, 6, 11, 12, 13, 14, 17, 19, 20, 21 and 22 of these Terms shall survive any termination of these Terms 
21  Independent Contractors  The parties are and intend to be independent contractors with respect to the Services contemplated hereunder  You agree that neither you nor any of your employees or contractors shall be considered as having an employee status with Agency Spotter  No form of joint employer, joint venture, partnership, or similar relationship between the parties is intended or hereby created 
22  Assignment and Delegation  You may not assign or delegate any rights or obligations under these Terms  Any purported assignment or delegation shall be ineffective  We may freely assign or delegate all rights and obligations under these Terms, fully or partially, without notice to you  We may also substitute, by way of unilateral novation, effective upon notice to you, Agency Spotter Inc  for any third party that assumes our rights and obligations under these Terms 
The personally identifiable information we collect from you allows us to provide you with the Services and to enable users to navigate and enjoy using the Site  We will also use your personally identifiable information to develop, improve and advertise the Site and Services  We may also use your personally identifiable information for internal purposes such as auditing, data analysis and research to improve our Services and customer communications  We do not rent, sell or otherwise provide your personally identifiable information to third parties without your consent, except as described in this policy or as required by law 
When you register with us through the Site or Services and become a Registered User, or when you wish to contact another Registered User, we will ask you for personally identifiable information  This refers to information about you that can be used to contact or identify you (“Personally Identifiable Information“)  Personally Identifiable Information includes, but is not limited to, your name, phone numbers, email address, home postal address, business address, social media user names, employer/affiliated organization, reasons for accessing the Site, and intended usage of requested information, but does not include your credit card number or billing information  We may also use your email address or phone number (if provided by you) to contact you regarding changes to the Services; system maintenance and outage issues; account issues; or otherwise to troubleshoot problems  In order to process some of your transactions through the Site and Services, we may also ask for your credit card number and other billing information (“Billing Information“; and, together with Personally Identifiable Information, “Personal Information“) 
Information you provide to us also includes your account profile and your contributions to discussion groups and community features Agency Spotter may offer  Do not upload or insert any information to or into the Site or Services that you do not want to be shared or used in the manner described in this section 
In addition, when you use the Site, our servers automatically record certain information that your web browser sends whenever you visit any website  These server logs may include information such as your web request, Internet Protocol address, browser type, browser language, referring/exit pages and URLs, platform type, number of clicks, domain names, landing pages, pages viewed and the order of those pages, the amount of time spent on particular pages, the date and time of your request, and one or more cookies that may uniquely identify your browser 
Information from third party services and other websites 
Do not upload or insert any information to or into the Site or Services that you do not want to be shared or used in the manner described in this section 
Advertisements  Advertisers who present ads on the Site may use technological methods to measure the effectiveness of their ads and to personalize advertising content  You may use your browser cookie settings to limit or prevent the placement of cookies by advertising networks  Agency Spotter does not share personally identifiable information with advertisers unless we get your permission 
Links  When you click on links on Agency Spotter you may leave our site  We are not responsible for the privacy practices of other sites, and we encourage you to read their privacy statements 
If we are requested to disclose your information to a government agency or official, we will do so if we believe in good faith, after considering your privacy interests and other relevant factors, that such disclosure is necessary to: (i) conform to legal requirements or comply with a legal process with which we are involved; (ii) protect our rights or property or the rights or property of our affiliated companies; (iii) prevent a crime or protect national security; or (iv) protect the personal safety of Site users or the public  Because Agency Spotter is a United States limited liability company and information collected on our Site is stored in whole or in part in the United States, your information may be subject to U S  law 
We also reserve the right to disclose Personally Identifiable Information and/or other information about users that Agency Spotter believes, in good faith, is appropriate or necessary to enforce our agreements, take precautions against liability, investigate and defend itself against ay third-party claims or allegations, assist government enforcement agencies, protect the security or integrity of our Site or Services, and protect the rights, property or personal safety of Agency Spotter, our users and others 
Cookies allow us to (i) manage, present and keep track of temporary information, such as data you upload onto the Site for use with the Services; (ii) register you as a Registered User on the Site or in other various programs associated with the Site; (iii) remember you when you log in to the places on the Site that require you to be a Registered User of the Site; (iv) help us understand the size of our audience and traffic patterns; (v) collect and record information about what you viewed on the Site; and (vi) deliver specific information to you based on your interests 
When you access the Site, the Site automatically collects certain non-personally identifiable information through the use of electronic images known as web beacons (sometimes called single-pixel gifs) and log files  Such information may include your IP address, browser type, the date, time and duration of your access and usage of the Site and whether you opened emails You received from us 
This information is collected for all visits to the Site and then analyzed in the aggregate  This information is useful for, among other things, tracking the performance of our online advertising, such as online banner ads, and determining where to place future advertising on other websites 
Editing your profile  You may review and change or remove your personal information or the settings for your Agency Spotter account at any time by going to your account profile  You can edit your name, email address, password and other account information here  Please be aware that even after your request for a change is processed, Agency Spotter may, for a time, retain residual information about you in its backup and/or archival copies of its database 
Deactivating or deleting your account  If you want to stop using your account you may deactivate it or delete it  When you deactivate an account, no user will be able to see it, but it will not be deleted  We save your profile information in case you later decide to reactivate your account  Many users deactivate their accounts for temporary reasons and in doing so are asking us to maintain their information until they return to Agency Spotter  You will still have the ability to reactivate your account and restore your profile in its entirety  When you delete an account, it is permanently deleted from Agency Spotter  You should only delete your account if you are certain you never want to reactivate it  You may deactivate your account or delete your account within your account profile 
Limitations on removal  Even after you remove information from your profile or delete your account, copies of that information may remain viewable elsewhere to the extent it has been shared with others, it was otherwise distributed pursuant to your privacy settings, or it was copied or stored by other users  However, your name will no longer be associated with that information on Agency Spotter  (For example, if you post something to another user’s or Agency’s profile or Agency’s portfolio and then you delete your account, that post may remain, but be attributed to an “Anonymous Agency Spotter User ”) Additionally, we may retain certain information to prevent identity theft and other misconduct even if deletion has been requested  If you have given third party applications or websites access to your information, they may retain your information to the extent permitted under their terms of service or privacy policies  But they will no longer be able to access the information through our platform after you disconnect from them 
Default Settings  Because the mission of Agency Spotter is to connect businesses and agencies, enabling them to save time, be more productive and successful, we have established what we believe are reasonable default settings that we have found most agencies and professionals desire  Because Registered Users may use and interact with Agency Spotter in a variety of ways, and because those uses may change over time, we designed our settings to provide our users control over the information they share  We encourage our Registered Users to review their account settings and adjust them in accordance with their preferences 
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Context 3: Thalassaemia minor | definition of Thalassaemia minor by Medical dictionary
Thalassaemia minor | definition of Thalassaemia minor by Medical dictionary
https://medical-dictionary thefreedictionary com/Thalassaemia+minor
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Related to Thalassaemia minor: thalassaemia major
Thalassemia describes a group of inherited disorders characterized by reduced or absent amounts of hemoglobin, the oxygen-carrying protein inside the red blood cells  There are two basic groups of thalassemia disorders: alpha thalassemia and beta thalassemia  These conditions cause varying degrees of anemia, which can range from insignificant to life threatening 
All types of thalassemias are considered quantitative diseases of hemoglobin, because the quantity of hemoglobin produced is reduced or absent  Usual adult hemoglobin is made up of three components: alpha globin, beta globin, and heme  Thalassemias are classified according to the globin that is affected, hence the names alpha and beta thalassemia  Although both classes of thalassemia affect the same protein, the alpha and beta thalassemias are distinct diseases that affect the body in different ways 
Beta thalassemia may be the most best-known type of thalassemia and is also called Cooley's anemia  It is caused by a change in the gene for the beta globin component of hemoglobin  Beta thalassemia causes variable anemia that can range from moderate to severe, depending in part on the exact genetic change underlying the disease  Beta thalassemia can be classified based on clinical symptoms  Beta thalassemia major usually causes severe anemia that can occur within months after birth  If left untreated, severe anemia can result in insufficient growth and development, as well as other common physical complications that can lead to a dramatically decreased life-expectancy  Fortunately, in developed countries beta thalassemia is usually identified by screening in the newborn period, before symptoms have developed  Children who are identified early can be started on ongoing blood transfusion therapy as needed  Although transfusion therapy prevents many of the complications of severe anemia, the body is unable to eliminate the excess iron contained in the transfused blood  Over time, the excess iron deposits in tissues and organs, resulting in damage and organ failure  Another medication must be administered to help the body eliminate the excess iron and prevent iron-over-load complications  Beta thalassemia intermedia describes the disease in individuals who have moderate anemia that only requires blood transfusions intermittently, if at all 
Alpha thalassemia is the result of changes in the genes for the alpha globin component of hemoglobin  There are two main types of alpha thalassemia disease: hemoglobin H disease and alpha thalassemia major  The two diseases are quite different from beta thalassemia as well as from one another  Individuals with hemoglobin H disease can experience events of hemolytic anemia—anemia caused by the rapid breakdown of the red blood cells  These events are thought to be triggered by various environmental causes, such as infection and/or exposure to certain chemicals  Hemoglobin H disease is in most cases milder than beta thalassemia  It does not generally require transfusion therapy  Alpha thalassemia major is a very serious disease that results in severe anemia that begins even before birth  Most affected babies do not survive to be born or die shortly after birth 
The thalassemias are among the most common genetic diseases worldwide  Both alpha and beta thalassemia have been described in individuals of almost every ancestry, but the conditions are more common among certain ethnic groups  Unaffected carriers of all types of thalassemia traits do not experience health problems  In fact, the thalassemia trait is protective against malaria, a disease caused by blood-borne parasites transmitted through mosquito bites  According to a widely accepted theory, most genetic changes—mutations—that cause thalassemia occurred multiple generations ago  Coincidentally, these mutations increased the likelihood that carriers would survive malaria infection  Survivors passed the mutation onto their offspring, and the trait became established throughout areas where malaria is common  As populations migrated, so did the thalassemia traits 
Beta thalassemia trait is seen most commonly in people with the following ancestry: Mediterranean (including North African, and particularly Italian and Greek), Middle Eastern, Indian, African, Chinese, and Southeast Asian (including Vietnamese, Laotian, Thai, Singaporean, Filipino, Cambodian, Malaysian, Burmese, and Indonesian)  Alpha-thalassemia trait is seen with increased frequency in the same ethnic groups  However, there are different types of alpha thalassemia traits within these populations  The frequency of hemoglobin H disease and alpha thalassemia major depends on the type of alpha thalassemia trait  The populations in which alpha thalassemia diseases are most common include Southeast Asians and Chinese (particularly Southern Chinese) 
It is difficult to obtain accurate prevalence figures for various types of thalassemia within different populations  This difficulty arises due to testing limitations in determining exact genetic diagnoses, as well as the fact that many studies have focused on small, biased hospital populations 
Two studies reflect prevalence figures that can be helpful counseling families and determining who to screen for beta thalassemia  Between the years of 1990 and 1996, the State of California screened more than 3 1 million infants born in the state for beta thalassemia  Approximately 1 in 114,000 infants had beta thalassemia major, with prevalence rates being highest among Asian Indians (about one in 4,000), Southeast Asians (about one in 10,000), and Middle Easterners (about one in 7,000)  Another type of beta thalassemia disease, E/beta thalassemia, was represented in approximately one in 110,000 births, all of which occurred in families of Southeast Asian ancestry  Among Southeast Asians, the prevalence of E/beta thalassemia was approximately one in 2,600 births  This is in keeping with the observation that hemoglobin E trait carrier rates are relatively high within the Southeast Asian population: 16% in a study of 768 immigrants to California, and up to 25% in some specific Southeast Asian populations such as Cambodians  While these California studies address some of the limitations of earlier population studies, the pattern observed in California is expected to be different in other areas of the United States and the world  For example, Italians are underrepresented in this population when compared to the population of the East Coast of the United States 
Determining prevalence figures for alpha thalassemia is even more difficult due to increased limitations in diagnostic testing  All types of alpha thalassemia disease are most common among people of Southeast Asian and Chinese descent, for reasons that become clearer with an understanding of the underlying genetics of alpha thalassemia  One study of 500 pregnant women in Northern Thailand estimated a frequency of one in 500 pregnancies affected by alpha thalassemia major, for example  Prevalence of alpha thalassemia disease is significantly lower in the United States primarily because of immigration patterns; although at least one state, California, has observed growing hemoglobin H disease incidence rates that are high enough to justify universal newborn screening for the condition 
Humans normally make several types of the oxygen-carrying protein hemoglobin  An individual's stage in development determines whether he or she makes primarily embryonic, fetal, or adult hemoglobins  All types of hemoglobin are made of three components: heme, alpha (or alpha-like) globin, and beta (or beta-like) globin  All types of thalassemia are caused by changes in either the alpha- or beta-globin gene  These changes cause little or no globin to be produced  The thalassemias are, therefore, considered quantitative hemoglobin diseases  All types of thalassemias are recessively inherited, meaning that a genetic change must be inherited from both the mother and the father  The severity of the disease is influenced by the exact thalassemia mutations inherited, as well as other genetic and environmental factors  There are rare exceptions, notably with beta thalassemia, where globin gene mutations exhibit a dominant pattern of inheritance in which only one gene needs to be altered in order to see disease expression  Scientists continue to study the causes  For instance, a new mutation for alpha-thalassemia was discovered for the first time among Iranian patients in 2004 
BETA-THALASSEMIA  Most individuals have two normal copies of the beta globin gene, which is located on chromosome 11 and makes the beta globin component of normal adult hemoglobin, hemoglobin A  There are approximately 100 genetic mutations that have been described that cause beta thalassemia, designated as either beta0 or beta + mutations  No beta globin is produced with a beta0 mutation, and only a small fraction of the normal amount of beta globin is produced with a beta + mutation 
When an individual has one normal beta globin gene and one with a beta thalassemia mutation, he or she is said to carry the beta thalassemia trait  Beta thalassemia trait, like other hemoglobin traits, is protective against malaria infection  Trait status is generally thought not to cause health problems, although some women with beta thalassemia trait may have an increased tendency toward anemia during pregnancy 
When two members of a couple carry the beta thalassemia trait, there is a 25% chance that each of their children will inherit beta thalassemia disease by inheriting two beta thalassemia mutations, one from each parent  The clinical severity of the beta thalassemia disease—whether an individual has beta thalassemia intermedia or beta thalassemia major—will depend largely on whether the mutations inherited are beta0 thalassemia or beta + thalassemia mutations  Two beta0 mutations generally lead to beta thalassemia major, and two beta+ thalassemia mutations generally lead to beta thalassemia intermedia  Inheritance of one beta0 and one beta + thalassemia mutation tends to be less predictable 
Although relatively uncommon, there are other thalassemia-like mutations that can affect the beta globin gene  Hemoglobin E is the result of a substitution of a single nucleotide  This change results in a structurally altered hemoglobin that is produced in decreased amounts  Therefore, hemoglobin E is unique in that it is both a quantitative (i e  thalassemia-like) and qualitative trait  When co-inherited with a beta thalassemia trait, it causes a disease that is almost indistinguishable from beta thalassemia disease  Large deletions around and including the beta globin gene can lead to delta/beta thalassemia or hereditary persistence of fetal hemoglobin (HPFH)  Interestingly, delta/beta thalassemia trait behaves very similarly to beta thalassemia trait in its clinical manifestations  However, HPFH trait does not tend to cause hemoglobin disease when co-inherited with a second thalassemia or other beta globin mutation 
ALPHA-THALASSEMIA  Most individuals have four normal copies of the alpha globin gene, two copies on each chromosome 16  These genes make the alpha globin component of normal adult hemoglobin, which is called hemoglobin A  Alpha globin is also a component of fetal hemoglobin and the other major adult hemoglobin called hemoglobin A2  Mutations of the alpha globin genes are usually deletions of the gene, resulting in absent production of alpha globin  Since there are four genes (instead of the usual two) to consider when looking at alpha globin gene inheritance, there are several alpha globin types that are possible 
Absence of one alpha globin gene leads to a condition known as silent alpha thalassemia trait  This condition causes no health problems and can be detected only by special genetic testing  Alpha thalassemia trait occurs when two alpha globin genes are missing  This can occur in two ways  The genes may be deleted from the same chromosome, causing the 'cis' type of alpha thalassemia trait  Alternately, they may be deleted from different chromosomes, causing the 'trans' type of alpha thalassemia trait  In both instances, there are no associated health problems, although the trait status may be detected by more routine blood screening 
Hemoglobin H disease results from the deletion of three alpha globin genes, such that there is only one functioning gene  Typically, this can occur when one parent carries the silent alpha thalassemia trait, and the other parent carries the 'cis' type of the alpha thalassemia trait  In this situation, there is a 25% chance for hemoglobin H disease in each of such a couple's children 
Hemoglobin H disease-like symptoms can also be a part of a unique condition called alpha thalassemia mental retardation syndrome  Alpha thalassemia mental retardation syndrome can be caused by a deletion of a significant amount of chromosome 16, affecting the alpha globin genes  This is usually not inherited, but rather occurs sporadically in the affected individual  Affected individuals have mild hemoglobin H disease, mild-to-moderate mental retardation, and characteristic facial features  This syndrome can also occur as a sex-linked form in which a mutation is inherited in a particular gene on the X-chromosome  This gene influences alpha globin production, as well as various other developmental processes  Individuals affected with this form of the syndrome tend to have more severe mental retardation, delayed development, nearly absent speech, characteristic facial features, and genital-urinary abnormalities  The remaining discussion will focus only on aspects of hemoglobin H disease 
Alpha thalassemia major results from the deletion of all four alpha globin genes, such that there are no functioning alpha globin genes  This can occur when both parents carry the 'cis' type of the alpha thalassemia trait  In this situation, there is a 25% chance for alpha thalassemia major in each of such a couple's children 
Beta thalassemia major is characterized by severe anemia that can begin months after birth  In the United States and other developed countries beta thalassemia is identified and treated early and effectively  Therefore, the following discussion of symptoms applies primarily to affected individuals in the past and unfortunately in some underdeveloped countries now  If untreated, beta thalassemia major can lead to severe lethargy, paleness, and delays in growth and development  The body attempts to compensate by producing more blood, which is made inside the bones in the marrow  However, this is ineffective without the needed genetic instructions to make enough functioning hemoglobin  Instead, obvious bone expansion and changes occur that cause characteristic facial and other changes in appearance, as well as increased risk of fractures  Severe anemia taxes other organs in the body—such as the heart, spleen, and liver—which must work harder than usual  This can lead to heart failure, as well as enlargement and other problems of the liver and spleen  When untreated, beta thalassemia major generally results in childhood death, usually due to heart failure  In 2004, the first known heart attack associated with beta thalassemia major was reported  Fortunately, in developed countries diagnosis is usually made early, often before symptoms have begun  This allows for treatment with blood transfusion therapy, which can prevent most of the complications of the severe anemia caused by beta thalassemia major  Individuals with beta thalassemia intermedia have a more moderate anemia that may only require treatment with transfusion intermittently, such as when infections occur and stress the body  As a person with beta thalassemia intermedia gets older, however, the need for blood transfusions may increase to the point that they are required on a regular basis  When this occurs their disease becomes more similar to beta thalassemia major  Other genetic and environmental factors can influence the course of the disease as well  For example, co-inheritance of one or two alpha thalassemia mutations can tend to ameliorate some of the symptoms of beta thalassemia disease, which result in part from an imbalance in the amount of alpha- and beta-globin present in the red blood cells 
Hemoglobin h disease
Absence of three alpha globin genes causes an imbalance of alpha and beta globin proteins in the red blood cells  The excess beta globin proteins tend to come together to form hemoglobin H, which is unable to release oxygen to the tissues  In addition, hemoglobin H tends to precipitate out in the cells, causing damage to the red blood cell membrane  When affected individuals are exposed to certain drugs and chemicals known to make the membrane more fragile, the cells are thought to become vulnerable to breakdown in large numbers, a complication called hemolytic anemia  Fever and infection are also considered to be triggers of hemolytic anemia in hemoglobin H disease  This can result in fatigue, paleness, and a yellow discoloration of the skin and whites of eyes called jaundice  Usually, the anemia is mild enough not to require treatment  Severe anemia events may require blood transfusion, however, and are usually accompanied by such other symptoms as dark feces or urine and abdominal or back pain  These events are uncommon in hemoglobin H disease, although they occur more frequently in a more serious type of hemoglobin H disease called hemoglobin H/Constant Spring disease  Individuals effected with this type of hemoglobin H disease are also more likely to have enlargement of and other problems with the spleen 
Alpha thalassemia major
Because alpha globin is a necessary component of all major hemoglobins and some minor hemoglobins, absence of all functioning alpha globin genes leads to serious medical consequences that begin even before birth  Affected fetuses develop severe anemia as early as the first trimester of pregnancy  The placenta, heart, liver, spleen, and adrenal glands may all become enlarged  Fluid can begin collecting throughout the body as early as the start of the second trimester, causing damage to developing tissues and organs  Growth retardation is also common  Affected fetuses usually miscarry or die shortly after birth  In addition, women carrying affected fetuses are at increased risk of developing complications of pregnancy and delivery  Up to 80% of such women develop toxemia, a disturbance of metabolism that can potentially lead to convulsions and coma  Other maternal complications include premature delivery and increased rates of delivery by cesarean section, as well as hemorrhage after delivery 
Thalassemia may be suspected if an individual shows signs that are suggestive of the disease  In all cases, however, laboratory diagnosis is essential to confirm the exact diagnosis and to allow for the provision of accurate genetic counseling about recurrence risks and testing options for parents and affected individuals  Screening is likewise recommended to determine trait status for individuals of high-risk ethnic groups 
The following tests are used to screen for thalassemia disease and/or trait:
hemoglobin electrophoresis with quantitative hemoglobin A2 and hemoglobin F
free erythrocyte-protoporphyrin (or ferritin or other studies of serum iron levels)
A complete blood count will identify low levels of hemoglobin, small red blood cells, and other red blood cell abnormalities that are characteristic of a thalassemia diagnosis  Since thalassemia trait can sometimes be difficult to distinguish from iron deficiency, tests to evaluate iron levels are important  A hemoglobin electrophoresis is a test that can help identify the types and quantities of hemoglobin made by an individual  This test uses an electric field applied across a slab of gel-like material  Hemoglobins migrate through this gel at various rates and to specific locations, depending on their size, shape, and electrical charge  Isoelectric focusing and high-performance liquid chromatography (HPLC) use similar principles to separate hemoglobins and can be used instead of or in various combinations with hemoglobin electrophoresis to determine the types and quantities of hemoglobin present  Hemoglobin electrophoresis results are usually within the normal range for all types of alpha thalassemia  However, hemoglobin A2 levels and sometimes hemoglobin F levels are elevated when beta thalassemia disease or trait is present  Hemoglobin electrophoresis can also detect structurally abnormal hemoglobins that may be co-inherited with a thalassemia trait to cause thalassemia disease (i e , hemoglobin E) or other types of hemoglobin disease (i e , sickle hemoglobin)  Sometimes DNA testing is needed in addition to the above screening tests  This can be performed to help confirm the diagnosis and establish the exact genetic type of thalassemia 
Diagnosis of thalassemia can occur under various circumstances and at various ages  Several states offer thalassemia screening as part of the usual battery of blood tests done for newborns  This allows for early identification and treatment  Thalassemia can be identified before birth through the use of prenatal diagnosis  Chorionic villus sampling (CVS) can be offered as early as 10 weeks of pregnancy and involves removing a sample of the placenta made by the baby and testing the cells  CVS carries a risk of causing a miscarriage that is between 0 5%-1%  Amniocentesis is generally offered between 15 and 22 weeks of pregnancy, but can sometimes be offered earlier  Two to three tablespoons of the fluid surrounding the baby is removed  This fluid contains fetal cells that can be tested  The risk of miscarriage associated with amniocentesis ranges from 0 33-0 5%  Pregnant woman and couples may choose prenatal testing in order to prepare for the birth of a baby that may have thalassemia  Alternately, knowing the diagnosis during pregnancy allows for the option of pregnancy termination  Preimplantation genetic diagnosis (PGD) is a relatively new technique that involves in-vitro fertilization followed by genetic testing of one cell from each developing embryo  Only the embryos unaffected by sickle cell disease are transferred back into the uterus  PGD is currently available on a research basis only and is relatively expensive 
Individuals with beta thalassemia major receive regular blood transfusions, usually on a monthly basis  This helps prevent severe anemia and allows for more normal growth and development  Transfusion therapy does have limitations, however  Individuals can develop reactions to certain proteins in the blood—called a transfusion reaction  This can make locating appropriately matched donor blood more difficult  Although blood supplies in the United States are very safe, particularly relative to the past and to other areas of the world, there remains an increased risk of exposure to such blood-borne infections as hepatitis  Additionally, the body is not able to get rid of the excess iron that accompanies each transfusion  An additional medication called desferoxamine is administered, usually five nights per week over a period of several hours, using an automatic pump that can be used during sleep or taken anywhere the person goes  This medication is able to bind to the excess iron, which can then be eliminated through urine  If desferoxamine is not used regularly or is unavailable, iron overload can develop and cause tissue damage and organ damage and failure  The heart, liver, and endocrine organs are particularly vulnerable  Desferoxamine itself may rarely produce allergic or toxic side effects, including hearing damage  Signs of desferoxamine toxicity are screened for and generally develop in individuals who overuse the medication when body iron levels are sufficiently low  Overall, however, transfusion and desferoxamine therapy have increased the life expectancy of individuals with the most severe types of beta thalassemia major to the 4th or 5th decade  This can be expected to improve with time and increased developments in treatment, as well as for those with more mild forms of the disease 
New treatments offer additional options for some individuals with beta thalassemia major  There are various medications that target the production of red blood cells (i e  erythropoeitin) or fetal hemoglobin (i e  hydroxyurea and butyrate)  Their effectiveness in ameliorating the severity of beta thalassemia is currently being investigated  Another promising new treatment is bone marrow transplantation, in which the bone marrow of an affected individual is replaced with the bone marrow of an unaffected donor  If successful, this treatment can provide a cure  However, there is an approximately 10-15% chance the procedure could be unsuccessful (i e  the thalassemia returns); result in complications (i e  graft-versus-host disease); or result in death  The risk for specific individuals depends on current health status, age, and other factors  Because of the risks involved and the fact that beta thalassemia is a treatable condition, transplant physicians require a brother or sister donor who has an identically matched tissue type, called HLA type  HLA type refers to the unique set of proteins present on each individual's cells, which allows the immune system to recognize "self" from "foreign " HLA type is genetically determined, so there is a 25% chance for two siblings to be a match  Transplant physicians and researchers are also investigating ways to improve the safety and effectiveness of bone marrow transplantation  Using newborn sibling umbilical cord blood—the blood from the placenta that is otherwise discarded after birth but contains cells that can go on to make bone marrow—seems to provide a safer and perhaps more effective source of donor cells  Donors and recipients may not have to be perfect HLA matches for a successful transplant using cord blood cells  Trials are also underway to determine the effectiveness of "partial transplants," in which a safer transplant procedure is used to replace only a percentage of the affected individual's bone marrow  Other possible treatments on the horizon may include gene therapy techniques aimed at increasing the amount of normal hemoglobin the body is able to make 
Hemoglobin H disease is a relatively mild form of thalassemia that may go unrecognized  It is not generally considered a condition that will reduce one's life expectancy  Education is an important part of managing the health of an individual with hemoglobin H disease  It is important to be able to recognize the signs of severe anemia that require medical attention  It is also important to be aware of the medications, chemicals, and other exposures to avoid due to the theoretical risk they pose of causing a severe anemia event  When severe anemia occurs, it is treated with blood transfusion therapy  For individuals with hemoglobin H disease, this is rarely required  For those with the hemoglobin H/Constant Spring form of the disease, the need for transfusions may be intermittent or ongoing, perhaps on a monthly basis and requiring desferoxamine treatment  Individuals with this more severe form of the disease may also have an increased chance of requiring removal of an enlarged and/or overactive spleen 
Anemia — A blood condition in which the level of hemoglobin or the number of red blood cells falls below normal values  Common symptoms include paleness, fatigue, and shortness of breath 
Bilirubin — A yellow pigment that is the end result of hemoglobin breakdown  This pigment is metabolized in the liver and excreted from the body through the bile  Bloodstream levels are normally low; however, extensive red cell destruction leads to excessive bilirubin formation and jaundice 
Bone marrow — A spongy tissue located in the hollow centers of certain bones, such as the skull and hip bones  Bone marrow is the site of blood cell generation 
Bone marrow transplantation — A medical procedure used to treat some diseases that arise from defective blood cell formation in the bone marrow  Healthy bone marrow is extracted from a donor to replace the marrow in an ailing individual  Proteins on the surface of bone marrow cells must be identical or very closely matched between a donor and the recipient 
Desferoxamine — The primary drug used in iron chelation therapy  It aids in counteracting the life-threatening buildup of iron in the body associated with long-term blood transfusions 
Globin — One of the component protein molecules found in hemoglobin  Normal adult hemoglobin has a pair each of alpha-globin and beta-globin molecules 
Heme — The iron-containing molecule in hemoglobin that serves as the site for oxygen binding 
Hemoglobin — Protein-iron compound in the blood that carries oxygen to the cells and carries carbon dioxide away from the cells 
Hemoglobin A — Normal adult hemoglobin that contains a heme molecule, two alpha-globin molecules, and two beta-globin molecules 
Hemoglobin electrophoresis — A laboratory test that separates molecules based on their size, shape, or electrical charge 
Hepatomegaly — An abnormally large liver 
HLA type — Refers to the unique set of proteins called human leukocyte antigens  These proteins are present on each individual's cell and allow the immune system to recognize 'self' from 'foreign'  HLA type is particularly important in organ and tissue transplantation 
Hydroxyurea — A drug that has been shown to induce production of fetal hemoglobin  Fetal hemoglobin has a pair of gamma-globin molecules in place of the typical beta-globins of adult hemoglobin  Higher-than-normal levels of fetal hemoglobin can ameliorate some of the symptoms of thalassemia 
Iron overload — A side effect of frequent blood transfusions in which the body accumulates abnormally high levels of iron  Iron deposits can form in organs, particularly the heart, and cause life-threatening damage 
Jaundice — Yellowing of the skin or eyes due to excess of bilirubin in the blood 
Mutation — A permanent change in the genetic material that may alter a trait or characteristic of an individual, or manifest as disease, and can be transmitted to offspring 
Placenta — The organ responsible for oxygen and nutrition exchange between a pregnant mother and her developing baby 
Red blood cell — Hemoglobin-containing blood cells that transport oxygen from the lungs to tissues  In the tissues, the red blood cells exchange their oxygen for carbon dioxide, which is brought back to the lungs to be exhaled 
Screening — Process through which carriers of a trait may be identified within a population 
Splenomegaly — Enlargement of the spleen 
Because alpha thalassemia major is most often a condition that is fatal in the prenatal or newborn period, treatment has previously been focused on identifying affected pregnancies in order to provide appropriate management to reduce potential maternal complications  Pregnancy termination provides one form of management  Increased prenatal surveillance and early treatment of maternal complications is an approach that is appropriate for mothers who wish to continue their pregnancy with the knowledge that the baby will most likely not survive  In recent years, there have been a handful of infants with this condition who have survived long-term  Most of these infants received experimental treatment including transfusions before birth, early delivery, and even bone marrow transplantation before birth, although the latter procedure has not yet been successful  For those infants that survive to delivery, there seems to be an increased risk of developmental problems and physical effects, particularly heart and genital malformations  Otherwise, their medical outlook is similar to a child with beta thalassemia major, with the important exception that ongoing, life-long blood transfusions begin right at birth 
As discussed above, the prognosis for individuals with the most serious types of thalassemia has improved drastically in the last several years following recent medical advances in transfusion, chemo-, and transplantation therapy  Advances continue and promise to improve the life expectancy and quality of life further for affected individuals 
"First Known Heart Attack Associated With Beta-thalassemia Major Reported " Heart Disease Weekly February 22, 2004: 10 
"Novel Alpha-thalassemia Mutations Identified " Hematology Week January 26, 2004: 19 
Children's Blood Foundation  333 East 38th St , Room 830, New York, NY 10016-2745  (212) 297-4336  cfg@nyh med cornell edu 
Cooley's Anemia Foundation, Inc  129-09 26th Ave  #203, Flushing, NY 11354  (800) 522-7222 or (718) 321-2873  http://www thalassemia org 
March of Dimes Birth Defects Foundation  1275 Mamaroneck Ave , White Plains, NY 10605  (888) 663-4637  resourcecenter@modimes org  http://www modimes org 
National Heart, Lung, and Blood Institute  PO Box 30105, Bethseda, MD 20824-0105  (301) 592-8573  nhlbiinfo@rover nhlbi nih gov  http://www nhlbi nih gov 
National Organization for Rare Disorders (NORD)  PO Box 8923, New Fairfield, CT 06812-8923  (203) 746-6518 or (800) 999-6673  Fax: (203) 746-6481  http://www rarediseases org 
Bojanowski J  "Alpha Thalassemia Major: The Possibility of Long-Term Survival " Pamphlet from the Northern California Comprehensive Thalassemia Center  (1999) 
Children's Hospital Oakland, Northern California Comprehensive Thalassemia Center website  http://www thalassemia com 
Cooley's Anemia Foundation, Inc  website  http://www thalassemia org/gohome html 
Joint Center for Sickle Cell and Thalassemic Disorders website  http://cancer mgh harvard edu/medOnc/sickle htm 
[thal″ah-se´me-ah]
a heterogeneous group of hereditary hemolytic anemias marked by a decreased rate of synthesis of one or more hemoglobin polypeptide chains, classified according to the chain involved (α, β, δ); the two major categories are α- and β-thalassemia 
α-thalassemia (alpha-thalassemia) that caused by diminished synthesis of alpha chains of hemoglobin  The homozygous form is incompatible with life, the stillborn infant displaying severe hydrops fetalis  The heterozygous form may be asymptomatic or marked by mild anemia 
β-thalassemia (beta-thalassemia) that caused by diminished synthesis of beta chains of hemoglobin  The homozygous form is called t  major and the heterozygous form is called t  minor 
thalassemia ma´jor the homozygous form of β-thalassemia, in which hemoglobin A is completely absent; it appears in the newborn period and is marked by hemolytic, hypochromic, microcytic anemia; hepatosplenomegaly; skeletal deformation; mongoloid facies; and cardiac enlargement 
thalassemia mi´nor the heterozygous form of β-thalassemia; it is usually asymptomatic, but there may be mild anemia 
sickle cell–thalassemia a hereditary anemia involving simultaneous heterozygosity for hemoglobin S and thalassemia 
thal·as·se·mi·a
, thalassanemia (thal'ă-sē'mē-ă, thă-las-ă-nē'mē-ă),
Any of a group of inherited disorders of hemoglobin metabolism in which there is impaired synthesis of one or more of the polypeptide chains of globin; several genetic types exist, and the corresponding clinical picture may vary from barely detectable hematologic abnormality to severe and fatal anemia 
[G  thalassa, the sea, + haima, blood]
/thal·as·se·mia/ (thal″ah-se´me-ah) a heterogeneous group of hereditary hemolytic anemias marked by a decreased rate of synthesis of one or more hemoglobin polypeptide chains, classified according to the chain involved (α, β, δ); the two major categories are α- and β-thalassemia 
α-thalassemia that caused by diminished synthesis of alpha chains of hemoglobin  The homozygous form is incompatible with life, the stillborn infant displaying severe hydrops fetalis  The heterozygous form may be asymptomatic or marked by mild anemia 
β-thalassemia that caused by diminished synthesis of beta chains of hemoglobin  The homozygous form is called t  major and the heterozygous form is called t  minor 
thalassemia ma´jor the homozygous form of &#x03B2;, in which hemoglobin A is completely absent; it appears in the newborn period and is marked by hemolytic, hypochromic, microcytic anemia, hepatosplenomegaly, skeletal deformation, mongoloid facies, and cardiac enlargement 
thalassemia mi´nor the heterozygous form of &#x03B2;, usually asymptomatic, although there is sometimes mild anemia 
(thăl′ə-sē′mē-ə)
An inherited form of anemia occurring chiefly among people of Mediterranean descent, caused by faulty synthesis of part of the hemoglobin molecule  Also called Mediterranean anemia 
thal′as·se′mic adj 
[thal′əsē′mē·ə]
Etymology: Gk, thalassa, sea, a + haima, without blood
production and hemolytic anemia characterized by microcytic, hypochromic red blood cells  Thalassemia is caused by inherited deficiency of alpha- or beta-globin synthesis  See also hemochromatosis, hemosiderosis 
Beta thalassemia, clinical thalassemia, Cooley's anemia, Mediterranean anemia, thalassemia major Hematology A group of genetic diseases by underproduction of hemoglobin due to mutations in the beta globin gene, which is more common in Mediterraneans Heredity Parents are carriers–heterozygotes; one in 4 children is homozygous for the mutation and thus has full-blown disease Clinical See Anemia  Cf Sickle cell anemia 
α-thalassemia
Hemoglobin Barts Hematology An inherited condition caused by a defect in the synthesis of the Hb α chain; Hb Barts hemoglobinopathy is characterized by the presence of 4 gamma chains; it is more common in southeast Asians; the most severe form of alpha thalassemia causes stillbirth due to hydrops fetalis Heredity Parents are carriers–heterozygotes; one in 4 children is homozygous for the mutation and thus has full-blown disease Clinical Pallor, fatiguability, FTT, fever, infections, diarrhea Management Transfusions
Thalassemia major Hematology A hemoglobinopathy caused by a defect in the synthesis of Hb β chain Clinical Pallor, fatigability, FTT, fever due to infections, diarrhea, bone deformities, hepatosplenomegaly Management Transfusions, but iron overload can damage the heart, liver, and endocrine systems, ergo iron chelation–early use of deferiprone, deferoxamine ↓ transfusion-related iron overload and may protect against DM, cardiac disease, early death
δ-thalassemia
Hematology A condition characterized by a defect of Hb A2–α2δ2; because Hb A2 comprises only 3% of the circulating Hb, even its complete absence; δ-thalassemia has little clinical or hematologic impact
γ-thalassemia
Hematology A condition characterized by a defect of gamma–γ Hb chains found in Hb F–α2γ2; because Hb F is present primarily in the fetus and newborns, it is rarely seen outside of the neonatal period, but may cause transient neonatal hemolytic anemia 
, thalassanemia (thal'ă-sē'mē-ă, -ă-să-nē'mē-ă)
Any of a group of inherited disorders of hemoglobin metabolism in which there is impaired synthesis of one or more of the polypeptide chains of globin; several genetic types exist, and the corresponding clinical picture may vary from barely detectable hematologic abnormality to severe and fatal anemia  People of Mediterranean, extraction are more often affected than others by this type of anemia 
Synonym(s): thalassaemia, thalassanaemia 
Any of a group of inherited disorders of hemoglobin metabolism with impaired synthesis of one or more polypeptide chains of globin; several genetic types exist 
<a href="https://medical-dictionary thefreedictionary com/Thalassaemia+minor">thalassemia</a>
Barts hemoglobin
beta hemoglobinopathy
beta-delta thalassemia
beta-thalassaemia
Bite Cell
black gallstone
I know of a couple, totally unrelated and unbeknownst to them, who are silent carriers of Thalassaemia minor 
Pakistan: Genetic factor: All in the genes
But, unfortunately, when one person with thalassaemia minor carrier happens to marry another with the same diagnosis, there is a strong possibility that their child would be thalassaemia major, as happened in the case of Taneja 
' My life depends upon a monthly blood transfusion '
0] thalassaemia demonstrates variable severity, ranging from a condition similar to [beta] thalassaemia minor to something approaching thalassaemia major 
A retrospective review of homozygous haemoglobin E patients
Thal, Alan P 
thalame
thalamencephalic
thalamencephalon
thalamic
thalamic fasciculus
thalamic nucleus
thalamic pain syndrome
thalamic peduncle
thalamic radiation
thalamo-
thalamocoele
thalamocortical
thalamocortical fibers
thalamogeniculate artery
thalamolenticular
thalamoperforating artery
thalamostriate radiation
thalamotuberal artery
Thalassaemia minor
thalassaemiaor Cooley's disease
thalassemic facies
thalasso-
Thalassobacter
Thalassobacter utilis
thalassoplankton
thalassoposia
thalidomide neuropathy
Thalidomider
thallium poisoning
Thalarctos
THALAS
Thalasaemia
Thalassaemia Association of Malaysia
thalassaemia major
Thalassaemias
thalassaemic
thalassanaemia
Thalassemia Action Group
Thalassemia Clinical Research Network
thalassemia syndrome



ground_truth: No.

prompt_front: Now, answer the following question based on the text below, only give me the answer and do not output any other words.

Question: How are thalassemias classified?

Read the following text and answer briefly.

Context 4: [49]
In the spring of 1774, William Hewson died unexpectedly of septicemia two weeks after cutting himself while dissecting a cadaver  Polly was left to care for their two young sons and was pregnant with a daughter she would give birth to in August of the same year  Is it possible that Margaret and Benjamin moved back into No  27 to assist Polly after the death of her husband as suggested in The Americanization of Benjamin Franklin?[50]
If the Westminster Rate Books are to be believed, the answer is no  For the year 1774, the Rate Books list Margaret Stevenson at No  1 and William Hewson at No  27  For the year 1775, they list Margaret Stevenson at No  1 and Magnus Falkner (Falconer/Falconar) at No  27  Magnus was William’s assistant at the anatomy school and fiancé to William’s sister, Dorothy  On his death bed, William instructed Polly, “let Mr  Falconar be my successor ”[51] Magnus would immediately take over the running of the anatomy school and continue William’s unfinished research  Four months later, he and Dorothy would marry [52] Essentially only two things changed at 27 Craven Street after William’s death: Polly gave birth to her daughter, and Magnus replaced William as the lease holder, so even if Margaret and Benjamin had wished to move back into No  27, there would have been no room for them  It is also interesting to note that considering the multiple times Benjamin wrote of his move out of No  27 (and complained of it), he never once mentioned moving back into No  27 in any of his correspondence after Mr  Hewson’s death 
Figure 6  No  36 Craven Street  (Photo courtesy of David Ross, britainexpress com)
In sum, based on the Westminster Rate Books[53] and Franklin’s correspondence, Mrs  Stevenson is known to have resided at No  27 (36) Craven Street from 1748 to 1772  It follows that, aside from the two years Franklin spent in Philadelphia from 1762 to 1764, he resided there from 1757 to 1772  Franklin’s correspondence also reveals that in the autumn of 1772, he and Mrs  Stevenson moved to another house on Craven Street  The 1773 Westminster Rate Books show her name crossed off at No  27 and William Hewson’s inserted  The following year the Rate Books list her at No  1 Craven Street  Evidence for Mrs  Stevenson and Benjamin remaining at No  1 after William’s death appears in the Westminster Rate Books for 1775 which have Mrs  Stevenson still residing at No  1 and Magnus Falkner residing at No  27  Further evidence can be construed from the lack of any mention of a move back into No  27 in Franklin’s correspondence  Despite the many theories one could devise as to why Franklin was thought to have lived at No  7 Craven Street by so many guide books and Franklin biographers of the nineteenth century, one thing is certain; at some point after Franklin’s departure to America in March of 1775, and no later than 1807, someone mistakenly associated him with No  7 on the west side of Craven Street, and it soon became his de facto residence  Credit must go to D  H  Montgomery in 1896 and Sir George in 1913 for setting the record partially straight by placing Franklin at No  27(36)  In 1937, the London County Council gave us the first accurate account of Franklin’s residences on Craven Street in the Survey of London at No  27(36) and No  1  It has been shown conclusively that No  27 was never previously numbered 7  It was, however, renumbered 36 in 1792 after ten additional houses were built at the southern end of the street and remains No  36 to this day 
[1] “Craven Street and Hungerford Lane”, in Survey of London: Volume 18, St Martin-in-the-Fields II: the Strand, ed  G H Gater and E P Wheeler (London, 1937), 27-39, Early History of the Site 
http://www british-history ac uk/survey-london/vol18/pt2/pp27-39
[2] “England, Westminster Rate Books, 1634-1900,” from database with images, Craven Street – 1735, FamilySearch from database by FindMyPast and images digitized by FamilySearch; citing Westminster City Archives, London 
[3] Ibid , Craven Street – 1748 
[4] The Statutes at Large, From Magna Charta to the End of the Eleventh Parliament of Great Britain  Anno 1761 Continued, Vol  XXVII, ed  Danby Pickering, (Cambridge, John Archdeacon, 1767), 96 
[6] James Raven, Publishing Business in Eighteenth-Century England, (Woodbridge: The Boydell Press, 2014), 201 
[7] The London Directory For the Year 1776, Ninth Edition, (London: T  Lowndes, 1776), title page 
[8] Kent’s Directory For the Year 1778, Forty-Sixth Edition, (London: Richard and Henry Causton, 1778), title page 
[9] A listing in Kent’s Directory for the Year 1882 on p  28 reveals, “Brown Sarah, Leather-seller, 1, Westmoreland-buildings, Aldersgate-street”, and in Kent’s Directory for the Year 1883 on p  175, “Whiteland Mary, Wine & Brandy Mercht  Jermyn-str  St  James ”
[10] “The Papers of Benjamin Franklin,” Sponsored by The American Philosophical Society and Yale University, Digital Edition by The Packard Humanities Institute, 22:263a 
http://franklinpapers org/franklin
Mrs  Stevenson wrote to Benjamin Franklin a letter from her new home at 75 Northumberland Court on November 16, 1775: “In this Court I have a kind friend, Mr  Lechmoen he comes and seats with me and talks of you with a hiy regard and friendship ”
[11] Survey of London, Early History of the Site 
[12] Survey of London, Footnotes/n 10 
[13] Survey of London, Historical Notes/No  31 
[14] David Hughson, LL D , London; Being An Accurate History And Description Of The British Metropolis And Its Neighbourhood, To Thirty Miles Extent, From An Actual Perambulation, Vol  IV, (London: W  Stratford, 1807), 227 
[15] The Reverend Joseph Nightingale, The Beauties of England and Wales: Or, Original Delineations, Topographical, Historical, and Descriptive, of Each County, Vol  X, Part III, Vol  II (London: J  Harris; Longman and Co ; J  Walker; R  Baldwin; Sherwood and Co ; J  and J  Cundee; B  and R  Crosby and Co ; J Cuthell; J  and J  Richardson; Cadell and Davies; C  and J  Rivington; and G  Cowie and Co , 1815), 245 
[16] John Britton, F S A  & Co , ed , The Original Picture of London, Enlarged and Improved: Being A Correct Guide For The Stranger, As Well As For the Inhabitant, To The Metropolis Of The British Empire Together With A Description Of The Environs, The Twenty-Fourth Edition (London: Longman, Rees, Orme, Brown, and Green, 1826), 479 
[17] Jared Sparks, The Works of Benjamin Franklin, Vol  VII, (Philadelphia: Childs & Peterson, 1840), 151 
[18] George Gulliver, F R S , The Works of William Hewson, F  R  S , (London: Printed for the Sydenham Society, MDCCCXLVI), xx 
[19] Peter Cunningham, Handbook for London; Past and Present, Vol  I, (London: John Murray, 1849), 245 
[20] F  Saunders, Memories of the Great Metropolis: or, London, from the Tower to the Crystal Palace, (New York: G P  Putnam, MDCCCLII), 138 
[21] Leigh Hunt, The Town; Its Memorable Characters and Events, (London: Smith, Elder and Co , 1859), 185 
[22] K  Baedeker, London and Its Environs, Including Excursions To Brighton, The Isle of Wight, Etc : Handbook For Travelers, Second Edition, (London: Dulau and Co , 1879), 133 
[23] Herbert Fry, London In 1880 Illustrated With Bird’s-Eye Views of the Principal Streets, Sixth Edition, (New York: Scribner, Welford, & Co , 1880), 50 
[24] Herbert Fry, London  Illustrated By Eighteen Bird’s-Eye Views of the Principal Streets, (London: W  H  Allen and Co , 1886), 40 
[25] Henry B  Wheatley, F S A , London Past and Present; Its History, Associations, and Traditions, Vol  1, (London: John Murray, New York: Scribner & Welford, 1891), 473 
[26] The Journal of the Society of Arts, Vol  XV, No  778, (October 18, 1867): 717 
[27] D  G  C  Allen, “Dear and Serviceable to Each Other: Benjamin Franklin and the Royal Society of Arts,” American Philosophical Society, Vol  144, No  3, (September 2000): 248-249 
Franklin was a corresponding member in 1756 because he was still residing in Philadelphia  He became an active member the following year when he moved to London 
[28] The Journal of the Society of Arts, Vol  XVIII, No  894, (Jan  7, 1870): 137 
“Since the last announcement, the following tablets have been affixed on houses formerly occupied by – Benjamin Franklin, 7 Craven-street, Strand, W C ”
[29] Franklin in His Own Time, eds  Kevin J  Haytes and Isabelle Bour, (Iowa City, University of Iowa Press, 2011), xxxvii 
“Takes lodgings with Margaret Stevenson at No  7 Craven Street ” It is unknown if the editors are referring to No  7 on the west side of Craven Street or No  36 on the east side using Sir George’s explanation of No  36 being previously numbered 7 
[30] Henry C  Shelly, “American Shrines on English Soil, III  In the Footprints of Benjamin Franklin,” in The Book News Monthly, September, 1913 to August, 1914, (Philadelphia: John Wanamaker, 1914), 325 
[31] The Journal of the Royal Society of Arts, Vol  LVI, No  2,880, (Jan  31, 1908): 245 
http://babel hathitrust org/cgi/pt?id=mdp 39015058423073;view=1up;seq=251
“His Majesty the King, who is Patron of the Society, has granted permission to the Society to prefix to its title the term ‘Royal,’ and the Society will consequently be known in future as the ‘Royal Society of Arts ’”
[32] Nineteenth Annual Report, 1914, of the American Scenic and Historic Preservation Society, (Albany: J  B  Lyon Company, 1914), 293 
http://babel hathitrust org/cgi/pt?id=wu 89072985302;view=1up;seq=4;size=150
[33] The Journal of the Society of Arts, Vol  LXII, No  3,183, (Nov  21, 1913): 18 
http://babel hathitrust org/cgi/pt?id=mdp 39015058422968;view=1up;seq=26
[36] Allen, “Dear and Serviceable,” 263-264 
[37] Papers of Benjamin Franklin, 19:20 
[38] Thomas Joseph Pettigrew, F  L  S , Memoirs of the Life and Writings of the Late John Coakley Lettsom With a Selection From His Correspondence, Vol  I, (London: Nichols, Son, and Bentley, 1817), 144 of Correspondence 
[39] Papers of Benjamin Franklin, 19:321b 
[40] Ibid , 19:314 
[41] Ibid , 19:353a 
[43] Simon David John Chaplin, John Hunter and the ‘museum oeconomy’, 1750-1800, Department of History, King’s College London  Thesis submitted for the degree of Doctor of Philosophy of the University of London , 202 
“Following Falconar’s death [1778] the lease [27 Craven Street] was advertised, and the buildings were described as:
A genteel and commodious house, in good Repair, with Coach-house and Stabling for two Horses…consisting of two rooms and light closets on each floor, with outbuildings in the Yard, a Museum, a Compleat Theatre, and other conveniences  (Daily Advertiser, 27 August 1778)”
[44] Simon Chaplin, “Dissection and Display in Eighteenth-Century London,” in Anatomical Dissection in Enlightenment England and Beyond: Autopsy, Pathology and Display, ed  Dr  Piers Mitchell, (Burlington: Ashgate Publishing Company, 2012), 108 
“Given that a nearby building at 35 [ No  26 in Franklin’s time] was occupied by the man-midwife John Leake, who advertised lectures – including lessons in the art of making preparations – at his ‘theatre’ between 1764 and 1788, it is possible that some facilities were shared  In both cases, however, the buildings [Leake’s residence at No  26 and Hewson’s residence next door at 27] served a dual function as domestic accommodation and as sites for lecturing and dissection ”
[45] George Gulliver, F R S , The Works of William Hewson, F  R  S , (London: Printed for the Sydenham Society, MDCCCXLVI), xviii 
[46] Westminster Rate Books, Craven Street – 1773, courtesy of the City of Westminster Archives 
[47] S W  Hillson et al , “Benjamin Franklin, William Hewson, and the Craven Street Bones,” Archaeology International, Vol  2, (Nov  22, 1998): 14-16 
http://dx doi org/10 5334/ai 0206
[48] Westminster Rate Books, Craven Street – 1774, 1775, courtesy of the City of Westminster Archives 
[49] Survey of London, Historical Notes/No  36, Craven Street (not sourced) 
[50] Gordon S  Wood, The Americanization of Benjamin Franklin, (New York: The Penguin Press, 2004), 261 
[51] Pettigrew, Memoirs, 146 of Correspondence 
[52] http://founders archives gov/documents/Franklin/01-22-02-0178, note 7  “Falconar married Hewson’s sister five months after the Doctor’s death; most of the Craven Street circle attended the wedding, and BF gave away the bride: Polly to Barbara Hewson, Oct  4, 1774, APS” (American Philosophical Society); “England Marriages, 1538–1973 ,” database, FamilySearch (https://familysearch org/ark:/61903/1:1:V52W-TGS : accessed September 15, 2015), Magnus Falconar and Dorothy Hewson, September 12, 1774; citing Saint Martin In The Fields, Westminster, London, England, reference ; FHL microfilm 561156, 561157, 561158, 942 B4HA V  25, 942 B4HA V  66 
[53] I chose to rely on the Westminster Rate Books for the numbering system on Craven Street  The books were consistent throughout the eighteenth century in the ordering of residents on the street and were used as the basis for the 1792 re-numbering  For the most part, commercial directories aligned with them as well  If by chance a directory didn’t initially align, it would inevitably produce future editions that did 
Benjamin Franklin, Benjamin Franklin House, London
More from David Turnquist
If one looked into Benjamin Franklin’s time on Craven Street, they might   
I think it’s very ironic that on the street maps included in your excellent article, Craven Street is so close to Scotland Yard  Because following the back and forth juxtapositions of numbers 7, 27 and 36 Craven Street (throw in 75 Northumberland Court and 1 Craven Street, too) was a case that could confound Sherlock Holmes 
Excellent job of deciphering street renumbering material spanning sixty years, including that of a wrong house number (# 7) being erroneously identified and then perpetuated in subsequent street map printings  It’s gratifying at least to know that the present day #36 Craven Street is the correct house for Ben Franklin tourists to visit  Except for #1 Craven Street for the last three years Franklin was in London, but we won’t get into that 
Again, excellent article, David!



ground_truth: According to the globin that is affected (alpha or beta).