Task: Question Answering
Input (English): Background Paragraph: Excess intake of calcium may cause hypercalcaemia. However, because calcium is absorbed rather inefficiently by the intestines, high serum calcium is more likely caused by excessive secretion of parathyroid hormone (PTH) or possibly by excessive intake of vitamin D, both which facilitate calcium absorption. It may also be due to bone destruction that occurs when tumours metastasize within bone. All these conditions result in excess calcium salts being deposited in the heart, blood vessels, or kidneys. Symptoms include anorexia, nausea, vomiting, memory loss, confusion, muscle weakness, increased urination, dehydration, and metabolic bone disease. Chronic hypercalcaemia typically leads to calcification of soft tissue and its serious consequences: for example, calcification can cause loss of elasticity of vascular walls and disruption of laminar blood flow—and thence to plaque rupture and thrombosis. Conversely, inadequate calcium or vitamin D intakes may result in hypocalcaemia, often caused also by inadequate secretion of parathyroid hormone or defective PTH receptors in cells. Symptoms include neuromuscular excitability, which potentially causes tetany and disruption of conductivity in cardiac tissue. 
Story: Two brothers went to the doctor's office for a medical exam and blood tests. Dan found out he is healthy and had normal blood cholesterol and calcium levels. Ben however, was told that he had a disease called hypercalcaemia and had to start a treatment for that. 
Question: Which brother didn't have excess calcium salts deposited in the kidneys?
Output (English): Dan.