Myogenic Mechanism Renal blood flow autoregulation is an essential homeostatic chemical change that defends the kidney from a delegate in arterial pressure that would be sent to the glomerular capillaries and drive injuries. Two mechanisms impart to renal autoregulation that is, the myogenic, which responds very fast, and the tubuloglomerular feedback (TGF), which is slow in response. Myogenic mechanisms help maintain an invariant blood flow at varied pressure in the artery arterial and protect against renal damage from overpressure. The myogenic reaction is defined by a decrease of the diameter of the vessels in response to the various alteration in the Transmural force and plays an essential function in maintaining the ambient vascular tone and the autoregulation of blood flow of the resistivity vasculature. Myogenic mechanisms are also inbuilt to the smooth muscle blood tubes, particularly in small blood vessels and arterioles (Jackson, 2021). If the force inside a particular vessel is abruptly enlarged, the vessel responds by narrowing, while vasodilation and relaxation are caused by minimized pressure within the vessel. Tubuloglomerular Mechanism On the other hand, the tubuloglomerular mechanism is an accommodative execution that links the rate of glomerular action to the compactness of salt in the tubule liquid at the macula lutea. Tubuloglomerular feedback is designed to regulate the amount of salt entering the lateral nephron. A high sodium chloride(salts) distribution and the resorption rate at this site drive contraction of the corticipetal arteriole, a diminution in glomerular filtration pace (GFR), and a decrease in renal blood flow. This auto regulatory mechanism is built-in to the kidney in that it does not necessitate neuronal or humoral factors. The process of tubuloglomerular response acts as counter feedback regulation mechanism which makes use of the information concerning distal tubular fluid flow rate to control the nephritic blood flow and since the Renal Blood Flow affects the glomerular infiltration rate, and thus the proximal fluid flow value rate, the tubuloglomerular feedback helps hold a comparatively unfluctuating value of renal blood flow and glomerular filtration rate. Tubular secretion is the passage of matter like drugs and creatinine from the blood system into the peritubular capillaries to the renal tubule in the lumen of the kidney. Purpose The main aim of tubular secretion is to reduce liquid waste in the blood. It is also crucial in eradicating poisons, toxins, drugs, and other natural elements found in the blood in their excess. Some of the natural elements include potassium ion, hydrogen ion, ammonia, and urea. The removed substances combine with water to form urine which flows into the bladder through the ureter. The secreted products are usually in small quantities and are generally wasted. Processes Two main processes are involved in tubular secretion: passive dispersion and active conveyance. Passive diffusion is the conveyance of particles from the peritubular tubules to the interstitial fluid within the nephron, while active transport involves the movement of the molecules from the renal epithelium tubul