For patients and medical practitioners alike, the sheer quest to surmount infertility is an obsessive one entailing not only a struggle on the physical dimension but also a struggle on the emotional and spiritual front. Each month without signs of pregnancy engineer another round of frustrating and emotive questions among couples, which soon develops into feelings of resentment, finger-pointing and blame-apportioning. Documented research demonstrates that these negative outcomes have, in more than one occasion, led to marriage breakups in the absence of proper intervention measures such as counseling or medical intervention (Cooper & Glazer 38). Indeed, the panorama associated with conceiving, giving birth, and starting a family may be so momentous that it obliges couples, especially women, to consider every avenue of reinstating fertility, from the simplest to the most bizarre. Pushed to the limits, most individuals opt for fertility drugs. It is the purpose of this paper to critically evaluate the functions and side-effects of fertility-enhancing drugs with a view to demonstrate that their benefits outweigh social and health costs. Fertility drugs, also called ovulation-inducing drugs (OIDs), are medications that are mainly used to influence the biological process of ovulation. According to Ayhan et al., “ovulation-inducing drugs have been widely used for various types of infertility since the beginning of 1960s and their use increases day by day parallel to the success achieved in fertility treatment” [1] (1104). Al-Shawaf et al. asserts that the development and dissemination of OIDs generated the opportunity for a vast proportion of the unproductive population to create children (513). These drugs work by enhancing ovulation by arousing or stimulating the victim’s brain to get an egg ready and discharge it from her ovaries every month (Pendzich para. 9). Some of the most commonly used fertility drugs include Clomiphene Citrate, Human Menopausal Gonadotrophin, Recombinant Gonadotrophin, Repronex, and Bromocriptine (Chavis para 3; Singer 45). One of the most basic uses of fertility drugs is to restore the reproductive capacities of infertile individuals. Clomiphene functions to achieve this purpose by stimulating the pituitary gland to generate adequate follicle-stimulating hormones, which are the hormones that are primarily responsible for triggering ovulation (Mix 89). The medication, taken in pill form daily, triggers the ovaries to prepare some eggs for ovulation. Immediately therapy is discontinued, the hypothalamus gland discharges a luteinizing hormone which responsively triggers the ovaries to discharge a ready egg to the section where fertilization takes pace – the fallopian tubes. According to Pendzich, “…about 70 to 90 percent of women who take Clomiphene will ovulate, and of those who ovulate, 20 to 60 percent will get pregnant” [2] (para. 10). Another commonly used fertility drug is Human Menopausal Gonadotropin (hMG). This drug, according to Pendzich, “…consists of purified follicle stimulating hormone (FSH)…” which occasions a woman to develop egg follicles after being injected with the drug (para. 11). Immediately after completion of the injections, the woman is then injected with another hormone named human chorionic gonadotropin, which functions to arouse the ovaries to discharge th