Introduction Historically, most of the patients who suffered cardiac arrest far from hospital setting have experience high deaths and morbidity. Research has also found that the increased temperature resulting from the brain injury or ischemia evidently aggravates the level of damage to the brain and also contributes to prolonged hospital stay. The process and duration of recovery is directly correlated to the temperature of the body. In investigational studies show that the increased temperature (hyperthermia) increases oxygen free radicals and causes cell membrane to become more permeable. This causes the membrane to be unstable and as a result this causes an abnormal electrical depolarization. It’s estimated that even with as little increase in temperature as 0.5°C, a very big zone of the brain is injured or there is significant neuronal loss (Diringer et al, 2004, p. 1492; Zeiner et al, 2001, 2008) Description During the post cardiac arrest period, the elevated temperatures greatly add to the impact of ischemic brain damage. Research has shown that by reducing the brain temperature by few degrees Celsius would alleviate the level of the damage (Zeiner et al, 2001, 2008). This is the induced hypothermia and it greatly improves the recovery of the neurological damage. Since 1950’s medical practitioners have used deep hypothermia in sustaining patients through some selected surgical processes (Abella et al, 2005, p. 182). However, in 2002, there was greater development and some trials in Europe and Australia were conducted where the process of induced hypothermia was applied to lower the temperature of patients who had just suffered cardiac arrest. After publication of the process it was adopted for use for post-cardiac arrest patients. Therapeutic hypothermia is hence described as “the purposeful decreasing of core body temperature to between 32°C and 34°C during the first twelve to twenty-four hours following a cardiac arrest attack” (Nolan et al, 2003, p. 119). The reduction in temperature (hypothermia) lowers the body metabolic rate and as a result the amount of oxygen being consumed is reduced (Abella et al, 2005, p. 185). This reduction of metabolic is very critical in primary care or preventative medicine and in this case it protects the body from active chemical reaction that takes place with reperfusion injury. Reduced temperatures also decreased the production of nitric oxide as well as other neurotoxins hence protecting the body from losing the brain cells (Abella et al, 2005, p. 185). This process also allows the blood brain barrier to experience favorable factors for it to recover its normal function and reduce cerebral edema. Strategic Plan The use of induced hypothermia for cardiac arrest survivors is still new. A particular case study stimulated the researcher’s interest in the subject. I will therefore request permission to do a literature search on the recent research that addressed this topic (McManus et al, 1998, p. 1562). Basically, the peer reviewed journals will form a larger part of the materials for study. I will search internet articles by use of key words typed and logged to the search engines to find sources that address