In anatomy, the G cell is a type of cell in the stomach and duodenum that secretes gastrin.
 It works in conjunction with gastric chief cells and parietal cells.
 G cells are found deep within the pyloric glands of the stomach antrum, and occasionally in the pancreas and duodenum.
 The vagus nerve innervates the G cells.
 Gastrin-releasing peptide is released by the post-ganglionic fibers of the vagus nerve onto G cells during parasympathetic stimulation.
 The peptide hormone bombesin also stimulates gastrin from G cells.
 Gastrin-releasing peptide, as well as the presence of amino acids in the stomach, stimulates the release of gastrin from the G cells.
 Gastrin stimulates enterochromaffin-like cells to secrete histamine.
 Gastrin also targets parietal cells by increasing the amount of histamine and the direct stimulation by gastrin, causing the parietal cells to increase HCl secretion in the stomach.
Feline hyperaldosteronism is a disease in cats.
 The symptoms are caused by abnormally high concentrations of the hormone aldosterone, which is secreted by the adrenal gland.
 The high concentrations of aldosterone may be due directly to a disorder of the adrenal gland (primary hyperadlosteronism), or due to something outside of the adrenal gland causing it to secrete excessive aldosterone (secondary hyperaldosteronism).
Growth hormone treatment refers to the use of growth hormone (GH) as a prescription medication—it is one form of hormone therapy.
 Growth hormone is a peptide hormone secreted by the pituitary gland that stimulates growth and cell reproduction.
 In the past, growth hormone was extracted from human pituitary glands.
 Growth hormone is now produced by recombinant DNA technology and is prescribed for a variety of reasons.
 GH therapy has been a focus of social and ethical controversies for 50 years.
Aldosterone, the main mineralocorticoid hormone, is a steroid hormone produced by the zona glomerulosa of the adrenal cortex in the adrenal gland.
 It is essential for sodium conservation in the kidney, salivary glands, sweat glands and colon.
 It plays a central role in the regulation of the plasma sodium (Na), the extracellular potassium (K) and arterial blood pressure.
 It does so mainly by acting on the mineralocorticoid receptors in the distal tubules and collecting ducts of the nephron.
 It influences the reabsorption of sodium and excretion of potassium (from and into the tubular fluids, respectively) of the kidney, thereby indirectly influencing water retention or loss, blood pressure and blood volume.
 When dysregulated, aldosterone is pathogenic and contributes to the development and progression of cardiovascular and renal disease.
 Aldosterone has exactly the opposite function of the atrial natriuretic hormone secreted by the heart.
Gonadotropin-releasing hormone (GnRH) also known as gonadoliberin, and by various other names in its endogenous form and as gonadorelin (INN) in its pharmaceutical form, is a releasing hormone responsible for the release of follicle-stimulating hormone (FSH) and luteinizing hormone (LH) from the anterior pituitary.
 GnRH is a tropic peptide hormone synthesized and released from GnRH neurons within the hypothalamus.
 The peptide belongs to gonadotropin-releasing hormone family.
 It constitutes the initial step in the hypothalamic–pituitary–gonadal axis.
Growth hormone (GH), also known as somatotropin (or as human growth hormone [hGH or HGH] in its human form), is a peptide hormone that stimulates growth, cell reproduction, and cell regeneration in humans and other animals.
 It is thus important in human development.
 It is a type of mitogen which is specific only to certain kinds of cells.
 Growth hormone is a 191-amino acid, single-chain polypeptide that is synthesized, stored, and secreted by somatotropic cells within the lateral wings of the anterior pituitary gland.
Angiotensin is a peptide hormone that causes vasoconstriction and a subsequent increase in blood pressure.
 It is part of the renin-angiotensin system, which is a major target for drugs that raises blood pressure.
 Angiotensin also stimulates the release of aldosterone, another hormone, from the adrenal cortex.
 Aldosterone promotes sodium retention in the distal nephron, in the kidney, which also drives blood pressure up.
The adrenal medulla (Latin: "medulla glandulae suprarenalis" ) is part of the adrenal gland.
 It is located at the center of the gland, being surrounded by the adrenal cortex.
 It is the innermost part of the adrenal gland, consisting of cells that secrete epinephrine (adrenaline), norepinephrine (noradrenaline), and a small amount of dopamine in response to stimulation by sympathetic preganglionic neurons.
11-Deoxycorticosterone (DOC), or simply deoxycorticosterone, also known as 21-hydroxyprogesterone, as well as desoxycortone (INN), deoxycortone, and cortexone, is a steroid hormone produced by the adrenal gland that possesses mineralocorticoid activity and acts as a precursor to aldosterone.
 It is an active (Na+-retaining) mineralocorticoid.
 As its names indicate, 11-deoxycorticosterone can be understood as the 21-hydroxy- variant of progesterone or as the 11-deoxy- variant of corticosterone.
The ACTH test (also called the cosyntropin, tetracosactide, or Synacthen test) is a medical test usually ordered and interpreted by endocrinologists to assess the functioning of the adrenal glands stress response by measuring the adrenal response to adrenocorticotropic hormone (ACTH; corticotropin) or another corticotropic agent such as tetracosactide (cosyntropin, tetracosactrin; Synacthen) or alsactide (Synchrodyn).
 ACTH is a hormone produced in the anterior pituitary gland that stimulates the adrenal glands to release cortisol, dehydroepiandrosterone (DHEA), dehydroepiandrosterone sulfate (DHEA-S), and aldosterone.

Question: Angiotensin, a peptide hormone, stimulates the release of the main mineralocorticoid hormone from what part of the adrenal gland?
Answer: adrenal cortex