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software.18 2.2 (1) Each Security shall be delivered to or on the order of an Investor or otherperson represented by such Investor at the Stipulated location. in English 2.2 CWS SEE-BACK.2.2 It is no longer a prerequisite to have a TSE accredited institution toqualify for TSE acceptance after 25 January, 2002,. .5 3. I have found thisdocument useful? Yes No (2) 4. I have found this document useful? Yes No Please
reply with comments and /or suggestions.Glucocorticoids mediate sex differences inc-fos induction by endotoxin: role of the hypothalamus and pituitary. Studies wereundertaken to compare hypothalamic and pituitary c-fos responses to LPS in female
and male rats to investigate the role of sex hormones in mediating these responses.
The results indicate that differences between the sexes do not result fromdifferences in the number of responding neurons, or in the extent of the inductionof the c-fos gene, but from the sensitivity of females to glucocorticoids.
Corticosterone significantly blocked the induction of c-fos in the paraventricular(PVN) and supraoptic nuclei (SON) of male rats, but had no effect in females.
Because the ventrolateral hypothalamus (VLH) is present in both sexes, but more
intense c-fos staining was present in the female, c-fos was re-induced in the maleafter lesions of the VLH. Even after these lesions, c-fos was still inhibited in thePVN and SON of female rats. To investigate the role of female sex hormones, adultovariectomized females were primed with 17 beta estradiol and progesterone 24 h
before they were injected with LPS. Although estradiol alone had no effect on c-fosinduction, progesterone significantly increased c-fos in all regions examined inmales. In addition, progesterone alone induced c-fos in the PVN and SON of female
rats. These results indicate that, within certain neuronal subpopulations of the
hypothalamus and pituitary, sex differences in c-fos induction are mediated by sex
hormones, and progesterone exerts its effects by itself or in conjunction with

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(2010), 1–36. Apoptosis in the
spinal cord white matter during
the pathogenesis of multiple
sclerosis. Evidence of apoptosis
in the normal-appearing whitematter (NAWM) of the central
nervous system (CNS) inautoimmune diseases has led tothe suggestion that apoptosis may
play a role in the pathogenesis ofwhite matter (WM) lesions. To
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samples of the spinal cord wereobtained from 10 patients withMS (CIS, n = 6; RRMS, n = 4)
and 7 controls. We identified
apoptotic cells by in situ endlabeling of DNA fragments, using
the terminal deoxynucleotidyltransferase-mediateddeoxyuridine triphosphate nickend-labeling (TUNEL) method.
Apoptotic cells were identified in
the NAWM of both the patients
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