Problemas de Ing. Quìmica I - Ocon y Tojo

1. Ocon The.1. Tojo 236 2. V. Tojo To Quimero 236 2. V. Tojo-To Quimero-Lector Jug. Solucionario Problemas De Ingenieria
Quimica Ocon 236 PDF LIST: Solucionario de Problemas De Ingenieria Quimica Ocon 236.doc 1. Ocon The.1. Tojo 236 2. V.
Tojo To Quimero 236 2. V. Tojo-To Quimero-Lector Jug.ECM1 modulates fibroblast-mediated tissue remodeling and hepatic
sinusoid invasion by cancer cells. The extracellular matrix protein 1 (ECM1) is a regulator of hematopoiesis and also a uniquemember of the ECM protein family, which is ubiquitously expressed in mesenchymal cells. However, the physiological role of
ECM1, as well as the potential role of ECM1 in cancer, remains largely unknown. In the present study, we addressed the role of
ECM1 in cancer-related tissue remodeling. ECM1 deficient fibroblasts failed to secrete angiogenic factor(s) into theconditioned medium, which resulted in slower wound closure in a rat fibroblast-populated collagen gel assay. In addition, ECM1
deficient fibroblasts showed a decreased capacity for three-dimensional collagen gel invasion. Using a mouse cancer model,
which we established, we revealed that ECM1 was strongly expressed in stromal fibroblasts in heterotopic liver tumors thatformed in the mammary fat pads of MMTV-PyMT mice. In vivo analysis revealed that loss of ECM1 decreased the size oftumors, due to suppressed liver angiogenesis and invasion. Interestingly, liver stromal fibroblasts contributed to hepatic
sinusoidal invasion of cancer cells, which was abrogated by loss of ECM1. Collectively, the present data indicate that ECM1
secreted by stromal fibroblasts modulates tumor invasion and angiogenesis by directly interacting with the cancer cells, andregulates the liver microenvironment around metastatic tumors, suggesting that ECM1 is a novel tumor suppressor incancer.Danone Seeks French Approval for Its $12bn Caramel Milk Wave to be a Success French dairy

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