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changes after the initial inductive phase of chemically induced carcinogenesis in mouse embryonic stem cells. The
microenvironment was in series compared in mouse embryonic stem cell (ESC) cultures that had or had not been treated with amutagenic dose of dimethylnitrosamine (DMN) and then transferred to fresh cultures. Microenvironmental changes following
the first exposure to DMN were detected by real-time PCR quantification of octamer-binding transcription factor 3/4 (Oct-3/4),
inducible nitric oxide synthase (iNOS), vascular endothelial growth factor (VEGF), and interleukin-6 (IL-6) gene expression,and nitrite and nitrate concentration in the medium. We found that residual DNA damage and apoptotic cells were detected at
the colony level in the affected colonies, although the level of apoptosis was not always reflected by the level of Oct-3/4 geneexpression. Residual DNA damage was detected in the same colony about 10 h after the first exposure to DMN. This wasassociated with high levels of both IL-6 and iNOS gene expression, as well as with measurable levels of nitrite and nitrate in themedium. Based on these results, we hypothesize that, following initial induction

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