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produced in humans and many animals, and melatonin receptor agonists and antagonists are currently being developed. Recent
investigations have shown the potential of melatonin for the treatment of skin diseases, such as acne and rosacea. However, very
few studies have reported the effect of melatonin on psoriasis, a chronic immune-mediated inflammatory skin disease. The aimof this study was to examine the effect of melatonin on the development of psoriasis. The effect of melatonin was investigatedon skin lesions, epidermal keratinocyte proliferation, and hyperplasia. Melatonin (2 mg/kg daily for 2 weeks) was given toDBA/1 mice either from the initiation or from the development of 2,4,6-trinitrobenzene sulphonic acid (TNBS)-induced
chronic dermatitis. Quantitative assessment of skin lesions and epidermal thickness were performed histologically and cellproliferation was assessed using the 5-bromo-2'-deoxyuridine (BrdU)-immunohistochemistry technique. Administration ofmelatonin to mice with chronic dermatitis during the initiation of dermatitis induced an increase of skin lesions and epidermal
thickness. Melatonin treatment during the development of the chronic dermatitis significantly reduced skin lesions andepidermal hyperplasia. Melatonin also significantly reduced epidermal proliferation in both models. Melatonin did not improve
the skin lesions induced by chronic dermatitis from the initiation of the disease but provided some beneficial effects on skinlesions and epidermal hyperplasia when given f678ea9f9e
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