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Instructions refer this link. Platypneaorthodeoxia. Platypnea-orthodeoxia (PO) is a
syndrome where upright brachial bloodpressure is high in supine position and low inupright position. Its cardinal features are lackof a compensatory increase in heart rate(HR), usually an increase in arterial partialpressure of oxygen (pO2), and orthostatic
hypotension. PO is often associated with
Parkinson's disease and orthostatic
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explain the high prevalence of PO in patientswith Parkinson's disease. PO is commonly
misdiagnosed as obstructive sleep apnea withcompensatory hypoventilation, since low
sitting and supine pO2 is often seen inpatients with obstructive sleep apnea. The"normal response" to hypoxia seen in somepatients with PO could be explained by asleep-related respiratory disturbance, or by
non-respiratory factors such as orthostatic
dysautonomia. In patients with obstructive
sleep apnea, the "normal" response toincreasing oxygen could be explained byventilatory inefficiency, reduced somaticafferent drive, and increased sympathetic
outflow with activation of arterial
chemoreceptors. The combination oforthostatic and hypoxemia in patients with
PO is likely to decrease their ability to "reset"

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chemoreceptor sensitivity and increase their
sympathetic activity. Some patients with PO
have a normal compensatory rise in HR but
increased pO2, which has been attributed toan autonomic cholinergic
hyperresponsiveness. Alternatively, therecould be an increased sensitivity to hypoxia
in the brainstem hypoxia sensing neurons.
Other possible explanations include reduced
arterial chemoreceptor sensitivity, alteredchemoreceptor sensitivity, increased posthypoxic vasodilation, and disturbed
respiratory drive. We need to understand themechanism of the "normal response" tohypoxia in PO in order to develop novel
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